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Journal articles on the topic 'Anisomysis'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Punchihewa, Neetha N. "A systematic study on two new species of mysids (Crustacea: Mysida: Mysidae) and two common mysid species in Sri Lanka." Ceylon Journal of Science 54, no. 3 (2025): 757–69. https://doi.org/10.4038/cjs.v54i3.8503.

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The material examined was collected from nine major estuarine bodies from South-west to North-west of Sri Lanka. Four species of mysids belong to three genera, Mesopodopsis Czerniavsky, Anisomysis Hansen and Siriella Dana, were identified: Mesopodopsis orientalis Tattersall, 1908 Mesopodopsis zelanica Nouvel, 1954 and another two new species referred to as Anisomysis srilankensis nov. and Siriella srilankensis nov. herein. The new A. srilankensis nov. is presently known from Kalpitiya lagoon, and the second species S. srilankensis nov. is presently known from Negombo, Chilaw, Puttalam, Kalpiti

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2

Moriya, Mitsuyasu, Khwanruan Srinui, and Shozo Sawamoto. "Two new species of the genus Anisomysis (Anisomysis) (Crustacea, Mysida, Mysidae) from coral reef waters in Thailand." ZooKeys 525 (October 6, 2015): 129–45. https://doi.org/10.3897/zookeys.525.5958.

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Two new species of Anisomysis Hansen, 1910 (Mysida, Mysidae), Anisomysis (Anisomysis) spinaintus sp. n. and A. (A.) phuketensis sp. n., from coral-reef waters in Thailand are described. Anisomysis (A.) spinaintus, collected in the Chaolao Beach, Chanthaburi Province, is distinguished from the closely allied species A. (A.) incisa Tattersall, 1936, and A. (A.) hawaiiensis Murano, 1995, by the presence of 6–9 spines on the apical cleft of telson, which are absent in the latter two allied species. The new species can also be distinguished from A. (A.) aikawai Ii, 1964, by the presence of a deep t

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3

Sawamoto, Shozo, Khwanruan Srinui, and Mitsuyasu Moriya. "Re-definition of the genus Javanisomysis Băcescu, 1992 as a subgenus in the genus Anisomysis Hansen, 1910 (Mysida, Mysidae) and a new species of the subgenus from coastal waters in Phuket, Thailand." Crustaceana 88, no. 7-8 (2015): 809–38. http://dx.doi.org/10.1163/15685403-00003455.

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Anisomysis (Javanisomysis) gutzui Băcescu, 1992 was reported as a characteristic species, of which the fourth male pleopod possesses an un-segmented exopod and no endopod. The species is placed in the valid genus Javanisomysis on the basis of the characteristics of the fourth male pleopod; however, the definition of the genus was insufficient. In the present paper, re-examination of the genus Javanisomysis is carried out on the basis of paratypes loaned from the depository in Romania. We found that major morphological characteristics of the types are common to those of the species of the genus

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4

Moriya, Mitsuyasu, Khwanruan Srinui, and Shozo Sawamoto. "Two new species of the genus Anisomysis (Anisomysis) (Crustacea, Mysida, Mysidae) from coral reef waters in Thailand." ZooKeys 525 (October 6, 2015): 129–45. http://dx.doi.org/10.3897/zookeys.525.5958.

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5

Panampunnayil, S. U. "Two new species of Anisomysis (Crustacea-Mysidacea) from the Lakshadweep archipelago." Journal of Plankton Research 15, no. 10 (1993): 1141–48. http://dx.doi.org/10.1093/plankt/15.10.1141.

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6

Murano, Masaaki. "Two New Species of the Genus Anisomysis (Crustacea: Mysidacea) from Northern Australia." Beagle : Records of the Museums and Art Galleries of the Northern Territory 12 (December 1995): 145–50. http://dx.doi.org/10.5962/p.264282.

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7

Murano, Masaaki. "A New Species of the Genus Anisomysis From the Great Barrier Reef (Mysidacea)." Crustaceana 52, no. 1 (1987): 47–52. http://dx.doi.org/10.1163/156854087x00051.

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8

Shaw, Arun, and Sama Ajay. "Studies Towards the Synthesis of (+)- and (–)-Anisomycin and Their Analogues." Synthesis 50, no. 01 (2017): 17–34. http://dx.doi.org/10.1055/s-0036-1588569.

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Anisomycin shows potent biological activity and it has attracted much attention since its isolation in 1954, with around 13 total syntheses and 20 formal syntheses, and also two reports concerning analogues of anisomycin, reported to date. The present review highlights all of these synthetic approaches (around 35) to the total or formal synthesis of anisomycin along with its isomers and analogues.1 Introduction2 Isolation and Therapeutic Importance3 Total Synthesis of (+)-, (–)-, and (±)-Anisomycins and Their Analogues4 Formal Synthesis of (+)- and (–)-Anisomycins and Their Analogues5 Conclusi

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9

Yudhatama, Bayu Khrisna, Sri Redjeki, and Chrisna Adhi Suryono. "Distribusi Horizontal Zooplankton Berdasarkan Salinitas Di Perairan Bonang Kabupaten Demak Indonesia." Journal of Marine Research 8, no. 3 (2019): 322–27. http://dx.doi.org/10.14710/jmr.v8i3.24988.

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ABSTRAK: Zooplankton merupakan salah satu biota yang sensitif akan perubahan karakteristik perairan. Zooplankton sering dijadikan indikator terhadap kondisi ekologis suatu perairan. Tujuan dalam penelitian ini yaitu untuk mengetahui Distribusi zooplankton dan pengaruh perbedaan salinitas pada distribusi zooplankton di Perairan Bonang Kabupaten Demak. Lokasi penelitian dibagi menjadi tiga stasiun yaitu Stasiun A (Sungai), Stasiun B (Muara), Stasiun C (Laut). Sampel diambil dengan plankton net di ketiga stasiun tersebut dengan tiga kali pengulangan dan tiga waktu pengulangan di setiap stasiunnya

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10

Murano, Masaaki. "New and Already Known Species of the Genus Anisomysis (Mysidacea) from Hawaii and the Society Islands." Journal of Crustacean Biology 15, no. 2 (1995): 355. http://dx.doi.org/10.2307/1548962.

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11

Johnston, N. M., D. A. Ritz, and G. E. Fenton. "Larval development in the Tasmanian mysids Anisomysis mixta australis , Tenagomysis tasmaniae and Paramesopodopsis rufa (Crustacea: Mysidacea)." Marine Biology 130, no. 1 (1997): 93–99. http://dx.doi.org/10.1007/s002270050228.

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12

Johnston, Nadine M., and David A. Ritz. "Kin recognition and adoption in mysids (Crustacea: Mysidacea)." Journal of the Marine Biological Association of the United Kingdom 85, no. 6 (2005): 1441–47. http://dx.doi.org/10.1017/s0025315405012622.

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The phenomenon of adoption, i.e. replacement of larvae prematurely liberated from the brood pouch of conspecifics, and kin recognition was examined in three mysid species found commonly in Tasmanian waters: Anisomysis mixta australis, Paramesopodopsis rufa and Tenagomysis tasmaniae. A new method for testing the incidence of adoption using fluoroscein dye, which enables the determination of ‘same stage’ adoptions, is described. Under field and laboratory conditions, all species practised intraspecific adoption into their own brood pouches. Under laboratory conditions the incidence of adoption w

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13

YOLANDA, ROFIZA, RENI AMBARWATI, DWI ANGGOROWATI RAHAYU, et al. "An annotated checklist of the species of Lopogastrida and Mysida (Crustacea: Peracarida) from Thailand and its adjacent waters." Zootaxa 5244, no. 3 (2023): 201–32. http://dx.doi.org/10.11646/zootaxa.5244.3.1.

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A compilation of mysid shrimps species found in brackish water to seawater has been carried out based on published records. The checklist includes the taxonomic position of each species, local distribution and depth range, the geographic zones and also the bibliographic references where they were recorded. Altogether 3 Lophogastrida and 52 Mysida species and 1 subspecies are listed. Most of them are distributed in the Andaman Sea (41 species), some in the Gulf of Thailand (13 species) and a few in the Songkhla Lagoon System (5 species). Mysids from the tribe Erythropini and Neomysini are the m

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14

Fenton, Gwen Elizabeth. "Population dynamics of Tenagomysis tasmaniae Fenton, Anisomysis mixta australis (Zimmer) and Paramesopodopsis rufa Fenton from south-eastern Tasmania (Crustacea:Mysidacea)." Hydrobiologia 246, no. 3 (1992): 173–93. http://dx.doi.org/10.1007/bf00005696.

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15

Johnston, NM, and DA Ritz. "Synchronous development and release of broods by the swarming mysids Anisomysis mixta australis, Paramesopodopsis rufa and Tenagomysis tasmaniae (Mysidacea: Crustacea)." Marine Ecology Progress Series 223 (2001): 225–33. http://dx.doi.org/10.3354/meps223225.

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Fenton, Gwen Elizabeth. "Breeding biology of Tenagomysis tasmaniae Fenton, Anisomysis mixta australis (Zimmer) and Paramesopodopsis rufa Fenton from south-eastern Tasmania (Crustacea: Mysidacea)." Hydrobiologia 287, no. 3 (1994): 259–76. http://dx.doi.org/10.1007/bf00006375.

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17

Fenton, Gwen Elizabeth. "Diet and predators of Tenagomysis tasmaniae Fenton, Anisomysis mixta australis (Zimmer) and Paramesopodopsis rufa Fenton from south-eastern Tasmania (Crustacea:Mysidacea)." Hydrobiologia 323, no. 1 (1996): 31–44. http://dx.doi.org/10.1007/bf00020545.

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18

Fenton, Gwen Elizabeth. "Production and biomass of Tenagomysis tasmaniae Fenton, Anisomysis mixta australis (Zimmer) and Paramesopodopsis rufa Fenton from south-eastern Tasmania (Crustacea: Mysidacea)." Hydrobiologia 323, no. 1 (1996): 23–30. http://dx.doi.org/10.1007/bf00020544.

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19

Sawamoto, Shozo, Yukio Hanamura, Rose O. S. E. Mantiri, and Susumu Ohtsuka. "A new species in the subgenus Javanisomysis in the genus Anisomysis (Crustacea: Mysida: Mysidae) for specimens collected from Lombok Island, Indonesia." Plankton and Benthos Research 15, no. 3 (2020): 238–49. http://dx.doi.org/10.3800/pbr.15.238.

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20

Kanwilyanti, Soty, Supriharyono, and Agung Suryanto. "KELIMPAHAN LARVA UDANG DI SEKITAR PERAIRAN PT. KAYU LAPIS INDONESIA, KALIWUNGU, KENDAL." Management of Aquatic Resources Journal (MAQUARES) 2, no. 4 (2013): 71–80. http://dx.doi.org/10.14710/marj.v2i4.4270.

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Pembuangan limbah dari pabrik atau industri, pertanian, maupun limbah domestik dari suatu pemukiman penduduk ke dalam badan air suatu perairan dapat menyebabkan terjadinya degradasi kualitas air, dimana terjadi perubahan parameter kualitas air yang dikarenakan adanya pencemaran yang dapat mempengaruhi sifat kimia, fisika, dan biologi perairan yang memiliki potensi mencemari lingkungan perairan dan yang pertama kali merasakan dampak tersebut adalah organisme-organisme akuatik. Penelitian ini bertujuan untuk mengetahui kondisi parameter fisika dan kimia air yang berpengaruh terhadap kelimpahan l

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21

Chen, Wei, Wenjing Yang, Chunyan Zhang, et al. "Modulation of the p38 MAPK Pathway by Anisomycin Promotes Ferroptosis of Hepatocellular Carcinoma through Phosphorylation of H3S10." Oxidative Medicine and Cellular Longevity 2022 (November 24, 2022): 1–20. http://dx.doi.org/10.1155/2022/6986445.

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Hepatocellular carcinoma (HCC) is a prevalent malignant tumor worldwide. Ferroptosis is emerging as an effective target for tumor treatment as it has been shown to potentiate cell death in some malignancies. However, it remains unclear whether histone phosphorylation events, an epigenetic mechanism that regulates transcriptional expression, are involved in ferroptosis. Our study found that supplementation with anisomycin, an agonist of p38 mitogen-activated protein kinase (MAPK), induced ferroptosis in HCC cells, and the phosphorylation of histone H3 on serine 10 (p-H3S10) was participated in

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22

Hazzalin, Catherine A., Rozen Le Panse, Eva Cano, and Louis C. Mahadevan. "Anisomycin Selectively Desensitizes Signalling Components Involved in Stress Kinase Activation and fos andjun Induction." Molecular and Cellular Biology 18, no. 4 (1998): 1844–54. http://dx.doi.org/10.1128/mcb.18.4.1844.

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ABSTRACT Anisomycin, a translational inhibitor secreted byStreptomyces spp., strongly activates the stress-activated mitogen-activated protein (MAP) kinases JNK/SAPK (c-Jun NH2-terminal kinase/stress-activated protein kinase) and p38/RK in mammalian cells, resulting in rapid induction of immediate-early (IE) genes in the nucleus. Here, we have characterized this response further with respect to homologous and heterologous desensitization of IE gene induction and stress kinase activation. We show that anisomycin acts exactly like a signalling agonist in eliciting highly specific and virtually c

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23

Menezes, Jefferson, Niége Alves, Sidnei Borges, et al. "Facilitation of fear extinction by novelty depends on dopamine acting on D1-subtype dopamine receptors in hippocampus." Proceedings of the National Academy of Sciences 112, no. 13 (2015): E1652—E1658. http://dx.doi.org/10.1073/pnas.1502295112.

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Extinction is the learned inhibition of retrieval. Recently it was shown that a brief exposure to a novel environment enhances the extinction of contextual fear in rats, an effect explainable by a synaptic tagging-and-capture process. Here we examine whether this also happens with the extinction of another fear-motivated task, inhibitory avoidance (IA), and whether it depends on dopamine acting on D1 or D5 receptors. Rats were trained first in IA and then in extinction of this task. The retention of extinction was measured 24 h later. A 5-min exposure to a novel environment 30 min before extin

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24

Sun, Manman, Feiyue Xing, Shan Pan, Jingfang Di, Shan Zeng, and Jing Liu. "Low-dose anisomycin is sufficient to alter the bio-behaviors of Jurkat T cells." Open Life Sciences 8, no. 12 (2013): 1230–40. http://dx.doi.org/10.2478/s11535-013-0235-4.

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AbstractAnisomycin is a pyrrolidine antibiotic isolated from Streptomyces griseolus. It has been found that a quite low dose of anisomycin is sufficient to block proliferation of primary T lymphocytes. The focus of this study is to explore the possibility of anisomycin to treat human acute leukemia Jurkat T cells in vitro. The results indicated that the low dose of anisomycin could significantly inhibit the colony formation of Jurkat T cells and elevate the inhibition rate of Jurkat T cell growth along with its increasing concentrations. Jurkat T cell cycle was blocked into S-phase by anisomyc

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25

Zhao, Ting C., Ling Zhang, Jun T. Liu, and Tai L. Guo. "Disruption of Nox2 and TNFRp55/p75 eliminates cardioprotection induced by anisomycin." American Journal of Physiology-Heart and Circulatory Physiology 303, no. 10 (2012): H1263—H1272. http://dx.doi.org/10.1152/ajpheart.00306.2012.

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Transient activation of p38 through anisomycin is demonstrated to precondition the heart against myocardial injury. However, it remains unknown whether specific TNF-α receptor (TNFR) p55/p75 and Nox2, a subunit of NADPH oxidase, are involved in this event. We sought to investigate whether the genetic disruption of TNFRp55/p75 and Nox2 eliminated cardioprotection elicited by anisomycin and whether p38-dependent activation of Nox2 stimulated TNFR to ultimately achieve protective effects. Adult wild-type and TNFR p55/p75−/−and Nox2−/−mice received intraperitoneal injections of anisomycin (0.1 mg/

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26

Kočović, Dušica M., Danica Bajuk-Bogdanović, Ilinka Pećinar, Biljana Božić Nedeljković, Marko Daković, and Pavle R. Andjus. "Assessment of cellular and molecular changes in the rat brain after gamma radiation and radioprotection by anisomycin." Journal of Radiation Research 62, no. 5 (2021): 793–803. http://dx.doi.org/10.1093/jrr/rrab045.

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Abstract The objective of the study was to describe cellular and molecular markers of radioprotection by anisomycin, focusing on the changes in rat brain tissue. Two-month-old Wistar rats were exposed to a 60Co radiation source at a dose of 6 Gy, with or without radioprotection with anisomycin (150 mg/kg) administered subcutaneously 30 min before or 3 or 6 h after irradiation. Survivors were analyzed 30 days after treatment. Astroglial and microglial responses were investigated based on the expression of glial markers assessed with immunohistochemistry, and quantitative changes in brain biomol

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27

Gould, G. W., A. Cuenda, F. J. Thomson, and P. Cohen. "The activation of distinct mitogen-activated protein kinase cascades is required for the stimulation of 2-deoxyglucose uptake by interleukin-1 and insulin-like growth factor-1 in KB cells." Biochemical Journal 311, no. 3 (1995): 735–38. http://dx.doi.org/10.1042/bj3110735.

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The uptake of 2-deoxyglucose into KB cells was stimulated about 2-fold by interleukin-1 (IL1), anisomycin or insulin-like growth factor-1 (IGF1). Stimulation by IL1 and anisomycin was prevented by SB 203580, a specific inhibitor of the mitogen-activated protein (MAP) kinase homologue termed ‘re-activating kinase’ [RK; also known as p38, p40 and CSBP (cytokine synthesis anti-inflammatory-drug-binding protein)], but was unaffected by PD 98059, a specific inhibitor of the activation of the classical MAP kinase pathway. In contrast, the stimulation of 2-deoxyglucose uptake by IGF1 was blocked by P

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28

Zinck, R., M. A. Cahill, M. Kracht, C. Sachsenmaier, R. A. Hipskind, and A. Nordheim. "Protein synthesis inhibitors reveal differential regulation of mitogen-activated protein kinase and stress-activated protein kinase pathways that converge on Elk-1." Molecular and Cellular Biology 15, no. 9 (1995): 4930–38. http://dx.doi.org/10.1128/mcb.15.9.4930.

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Inhibitors of protein synthesis, such as anisomycin and cycloheximide, lead to superinduction of immediate-early genes. We demonstrate that these two drugs activate intracellular signaling pathways involving both the mitogen-activated protein kinase (MAPK) and stress-activated protein kinase (SAPK) cascades. The activation of either pathway correlates with phosphorylation of the c-fos regulatory transcription factor Elk-1. In HeLa cells, anisomycin stabilizes c-fos mRNA when protein synthesis is inhibited to only 50%. Under these conditions, anisomycin, in contrast to cycloheximide, rapidly in

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29

Chen, Shui-Tein, Meng-Yang Chang, and Nein-Chen Chang. "Formal Synthesis of Anisomycin." HETEROCYCLES 60, no. 5 (2003): 1203. http://dx.doi.org/10.3987/com-03-9723.

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30

Shono, Tatsuya, and Naoki Kise. "Total Synthesis of (−)-Anisomycin." Chemistry Letters 16, no. 4 (1987): 697–700. http://dx.doi.org/10.1246/cl.1987.697.

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31

Li, Ji, Yan Hua Feng, Xin Bai Li, Wei Han, Huan Qiu Liu, and Guo Guang Shao. "Concise synthesis of (−)-anisomycin." Chinese Chemical Letters 23, no. 6 (2012): 647–49. http://dx.doi.org/10.1016/j.cclet.2012.03.029.

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32

Scavuzzo, C. J., M. J. LeBlancq, F. Nargang, H. Lemieux, T. J. Hamilton, and C. T. Dickson. "The amnestic agent anisomycin disrupts intrinsic membrane properties of hippocampal neurons via a loss of cellular energetics." Journal of Neurophysiology 122, no. 3 (2019): 1123–35. http://dx.doi.org/10.1152/jn.00370.2019.

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The nearly axiomatic idea that de novo protein synthesis is necessary for long-term memory consolidation is based heavily on behavioral studies using translational inhibitors such as anisomycin. Although inhibiting protein synthesis has been shown to disrupt the expression of memory, translational inhibitors also have been found to profoundly disrupt basic neurobiological functions, including the suppression of ongoing neural activity in vivo. In the present study, using transverse hippocampal brain slices, we monitored the passive and active membrane properties of hippocampal CA1 pyramidal ne

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33

Zhu, Chong-Bin, Ana M. Carneiro, Wolfgang R. Dostmann, William A. Hewlett, and Randy D. Blakely. "p38 MAPK Activation Elevates Serotonin Transport Activity via a Trafficking-independent, Protein Phosphatase 2A-dependent Process." Journal of Biological Chemistry 280, no. 16 (2005): 15649–58. http://dx.doi.org/10.1074/jbc.m410858200.

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Presynaptic, plasma membrane serotonin (5-hydroxytryptamine; 5-HT) transporters (SERTs) clear 5-HT following vesicular release and are regulated through trafficking-dependent pathways. Recently, we (Zhu, C.-B., Hewlett, W. A., Feoktistov, I., Biaggioni, I., and Blakely, R. D. (2004)Mol. Pharmacol.65, 1462–1474) provided evidence for a trafficking-independent mode of SERT regulation downstream of adenosine receptor (AR) activation that is sensitive to p38 MAPK inhibitors. Here, we probe this pathway in greater detail, demonstrating elevation of 5-HT transport by multiple p38 MAPK activators (an

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34

Kornhuber, Johannes, and Iulia Zoicas. "Social Fear Memory Requires Two Stages of Protein Synthesis in Mice." International Journal of Molecular Sciences 21, no. 15 (2020): 5537. http://dx.doi.org/10.3390/ijms21155537.

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It is well known that long-term consolidation of newly acquired information, including information related to social fear, require de novo protein synthesis. However, the temporal dynamics of protein synthesis during the consolidation of social fear memories is unclear. To address this question, mice received a single systemic injection with the protein synthesis inhibitor, anisomycin, at different time-points before or after social fear conditioning (SFC), and memory was assessed 24 h later. We showed that anisomycin impaired the consolidation of social fear memories in a time-point-dependent

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35

Haas, Daniel A., William C. Sturtridge, and Susan R. George. "Effect of protein synthesis Inhibition on brain corticotropin-releasing factor and plasma adrenocorticotropin." Canadian Journal of Physiology and Pharmacology 68, no. 12 (1990): 1595–600. http://dx.doi.org/10.1139/y90-243.

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The effect of inhibiting protein synthesis on concentrations of corticotropin-releasing factor (CRF) in rat brain and plasma adrenocorticotropin (ACTH) was assessed following the administration of the general protein synthesis inhibitor anisomycin. Compared with vehicle-injected controls, protein synthesis inhibition resulted in significantly reduced CRF immunoreactivity (CRF-ir) in median eminence within 1 h (p < 0.01), remained decreased after 4 h (p < 0.025), and was nonsignificantly decreased after 24 h. Plasma ACTH levels were greatly increased within 1 h posttreatment (p < 0.000

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Qu, Yu-Juan, Xiao Zhang, Zhen-Zhen Fan, et al. "Effect of TRPV4-p38 MAPK Pathway on Neuropathic Pain in Rats with Chronic Compression of the Dorsal Root Ganglion." BioMed Research International 2016 (2016): 1–12. http://dx.doi.org/10.1155/2016/6978923.

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The aim of this study was to investigate the relationships among TRPV4, p38, and neuropathic pain in a rat model of chronic compression of the dorsal root ganglion. Mechanical allodynia appeared after CCD surgery, enhanced via the intrathecal injection of 4α-phorbol 12,13-didecanoate (4α-PDD, an agonist of TRPV4) and anisomycin (an agonist of p38), but was suppressed by Ruthenium Red (RR, an inhibitor of TRPV4) and SB203580 (an inhibitor of p38). The protein expressions of p38 and P-p38 were upregulated by 4α-PDD and anisomycin injection but reduced by RR and SB203580. Moreover, TRPV4 was upre

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Nguyen, Anh Minh Thao, Moran Shalev-Benami, Chloé Rosa-Teijeiro, et al. "Systematic Exploration of Functional Group Relevance for Anti-Leishmanial Activity of Anisomycin." Biomedicines 11, no. 9 (2023): 2541. http://dx.doi.org/10.3390/biomedicines11092541.

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Assessment of structure–activity relationships for anti-protozoan activity revealed a strategy for preparing potent anisomycin derivatives with reduced host toxicity. Thirteen anisomycin analogs were synthesized by modifying the alcohol, amine, and aromatic functional groups. Examination of anti-protozoal activity against various strains of Leishmania and cytotoxicity against leucocytes with comparison against the parent natural product demonstrated typical losses of activity with modifications of the alcohol, amine, and aromatic meta-positions. On the other hand, the para-phenol moiety of ani

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38

Geiger, Paige C., David C. Wright, Dong-Ho Han, and John O. Holloszy. "Activation of p38 MAP kinase enhances sensitivity of muscle glucose transport to insulin." American Journal of Physiology-Endocrinology and Metabolism 288, no. 4 (2005): E782—E788. http://dx.doi.org/10.1152/ajpendo.00477.2004.

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Muscle contractile activity is followed by an increase in the sensitivity of glucose transport to insulin. There is evidence suggesting that activation of p38 MAP kinase (p38) is involved in the stimulation of glucose transport by insulin and contractions. Exercise results in an increase in p38 phosphorylation that lasts for hours. In this context, we tested the hypothesis that activation of p38 results in an increase in insulin sensitivity. Muscles were exposed to anisomycin for 30 min to activate p38. Anisomycin increased p38 phosphorylation ∼2.5-fold and glucose transport activity 2- to 3-f

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HELLIWELL, Philip A., Michael RICHARDSON, Julie AFFLECK, and George L. KELLETT. "Regulation of GLUT5, GLUT2 and intestinal brush-border fructose absorption by the extracellular signal-regulated kinase, p38 mitogen-activated kinase and phosphatidylinositol 3-kinase intracellular signalling pathways: implications for adaptation to diabetes." Biochemical Journal 350, no. 1 (2000): 163–69. http://dx.doi.org/10.1042/bj3500163.

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We have investigated the role of the extracellular signal-regulated kinase (ERK), p38 and phosphatidylinositol 3-kinase (PI 3-kinase) pathways in the regulation of intestinal fructose transport. Different combinations of anisomycin, PD98059 and wortmannin had very different effects on fructose transport in perfused isolated loops of rat jejunum. Transport was stimulated maximally by anisomycin (2µM) and blocked by SB203580 (20µM), confirming involvement of the p38 pathway. PD98059 (50µM) alone had little effect on fructose transport. However, it had a dramatic effect on stimulation by aniso

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Huang, Pei Qiang, Shi Li Wang, Yuan Ping Ruan, and Jing Xing Gao. "A New Approach to (−)-Anisomycin." Natural Product Letters 11, no. 2 (1998): 101–6. http://dx.doi.org/10.1080/10575639808041204.

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Rudy, J. W. "Anisomycin and the reconsolidation hypothesis." Learning & Memory 13, no. 1 (2006): 1–3. http://dx.doi.org/10.1101/lm.157806.

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Reddy, J. Santhosh, A. Ravi Kumar, and B. Venkateswara Rao. "A new approach to (+)-anisomycin." Tetrahedron: Asymmetry 16, no. 19 (2005): 3154–59. http://dx.doi.org/10.1016/j.tetasy.2005.08.039.

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Jegham, Samir, and Bhupesh C. Das. "A new synthesis of (−)-anisomycin or (+)-anisomycin starting from D-tyrosine or L-tyrosine." Tetrahedron Letters 29, no. 35 (1988): 4419–22. http://dx.doi.org/10.1016/s0040-4039(00)80511-0.

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Iordanov, M. S., D. Pribnow, J. L. Magun, et al. "Ribotoxic stress response: activation of the stress-activated protein kinase JNK1 by inhibitors of the peptidyl transferase reaction and by sequence-specific RNA damage to the alpha-sarcin/ricin loop in the 28S rRNA." Molecular and Cellular Biology 17, no. 6 (1997): 3373–81. http://dx.doi.org/10.1128/mcb.17.6.3373.

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Inhibition of protein synthesis per se does not potentiate the stress-activated protein kinases (SAPKs; also known as cJun NH2-terminal kinases [JNKs]). The protein synthesis inhibitor anisomycin, however, is a potent activator of SAPKs/JNKs. The mechanism of this activation is unknown. We provide evidence that in order to activate SAPK/JNK1, anisomycin requires ribosomes that are translationally active at the time of contact with the drug, suggesting a ribosomal origin of the anisomycin-induced signaling to SAPK/JNK1. In support of this notion, we have found that aminohexose pyrimidine nucleo

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Hauser, Christoph, Bernd Schuettengruber, Stefan Bartl, Gerda Lagger, and Christian Seiser. "Activation of the Mouse Histone Deacetylase 1 Gene by Cooperative Histone Phosphorylation and Acetylation." Molecular and Cellular Biology 22, no. 22 (2002): 7820–30. http://dx.doi.org/10.1128/mcb.22.22.7820-7830.2002.

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ABSTRACT Histone deacetylase 1 (HDAC1) is a major regulator of chromatin structure and gene expression. Tight control of HDAC1 expression is essential for normal cell cycle progression of mammalian cells. HDAC1 mRNA levels are regulated by growth factors and by changes in intracellular deacetylase activity levels. Stimulation of the mitogen-activated protein kinase cascade by anisomycin or growth factors, together with inhibition of deacetylases by trichostatin A (TSA), leads to stable histone H3 phosphoacetylation and strongly induced HDAC1 expression. In contrast, activation of the nucleosom

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Zheng, Xiaoqing, Qiuxiang Cheng, Fen Yao, et al. "Biosynthesis of the pyrrolidine protein synthesis inhibitor anisomycin involves novel gene ensemble and cryptic biosynthetic steps." Proceedings of the National Academy of Sciences 114, no. 16 (2017): 4135–40. http://dx.doi.org/10.1073/pnas.1701361114.

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The protein synthesis inhibitor anisomycin features a unique benzylpyrrolidine system and exhibits diverse biological and pharmacologic activities. Its biosynthetic origin has remained obscure for more than 60 y, however. Here we report the identification of the biosynthetic gene cluster (BGC) of anisomycin in Streptomyces hygrospinosus var. beijingensis by a bioactivity-guided high-throughput screening method. Using a combination of bioinformatic analysis, reverse genetics, chemical analysis, and in vitro biochemical assays, we have identified a core four-gene ensemble responsible for the syn

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Clark, Jordan, Rania Nasrallah та Richard L. Hébert. "The PPARδLigand GW501516 Reduces Growth but Not Apoptosis in Mouse Inner Medullary Collecting Duct Cells". PPAR Research 2009 (2009): 1–11. http://dx.doi.org/10.1155/2009/706283.

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The collecting duct (CD) expresses considerable amounts of PPARδ. While its role is unknown in the CD, in other renal cells it has been shown to regulate both growth and apoptosis. We thus hypothesized that PPARδreduces apoptotic responses and stimulates cell growth in the mouse CD, and examined the effect of GW501516, a synthetic PPARδligand, on these responses in mouse IMCD-K2 cells. High doses of GW501516 decreased both DNA and protein synthesis in these cells by 80%, but had no overall effect on cell viability. Although anisomycin treatment resulted in an increase of caspase-3 levels of ab

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Inouye, S. T., J. S. Takahashi, F. Wollnik, and F. W. Turek. "Inhibitor of protein synthesis phase shifts a circadian pacemaker in mammalian SCN." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 255, no. 6 (1988): R1055—R1058. http://dx.doi.org/10.1152/ajpregu.1988.255.6.r1055.

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The suprachiasmatic nucleus (SCN) of the hypothalamus contains a circadian pacemaker that regulates many circadian rhythms in mammals. Experimental work in microorganisms and invertebrates suggests that protein synthesis is required for the function of the circadian oscillator, and recent experiments in golden hamsters suggest an acute inhibition of protein synthesis can induce phase shifts in a mammalian circadian pacemaker. To determine whether protein synthesis in the SCN region is involved in the generation of circadian rhythms in mammals, a protein synthesis inhibitor, anisomycin, was mic

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Junghae, Muthoni, and John G. Raynes. "Activation of p38 Mitogen-Activated Protein Kinase Attenuates Leishmania donovani Infection in Macrophages." Infection and Immunity 70, no. 9 (2002): 5026–35. http://dx.doi.org/10.1128/iai.70.9.5026-5035.2002.

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ABSTRACT Leishmania-induced macrophage dysfunctions have been correlated with altered signaling events. In this work, we report that SB203580, a specific inhibitor of p38 mitogen-activated protein kinases (MAPK), increases Leishmania donovani survival in human peripheral blood mononuclear macrophages. Consistent with this finding, activation of p38 and c-jun N-terminal kinase (JNK) MAPK signaling pathways by anisomycin significantly reduced parasite survival within these cells. However, the majority of the effect was seen in a 50% reduction in the percentage of macrophages infected, with littl

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Kardalinou, E., N. Zhelev, C. A. Hazzalin, and L. C. Mahadevan. "Anisomycin and rapamycin define an area upstream of p70/85S6k containing a bifurcation to histone H3-HMG-like protein phosphorylation and c-fos-c-jun induction." Molecular and Cellular Biology 14, no. 2 (1994): 1066–74. http://dx.doi.org/10.1128/mcb.14.2.1066-1074.1994.

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Anisomycin, a translational inhibitor, synergizes with growth factors and phorbol esters to superinduce c-fos and c-jun by a number mechanisms, one of which is its ability to act as a potent signalling agonist, producing strong, prolonged activation of the same nuclear responses as epidermal growth factor or tetradecanoyl phorbol acetate. These responses include the phosphorylation of pp33, which exists in complexed and chromatin-associated forms, and of histone H3 and an HMG-like protein. By peptide mapping and microsequencing, we show here that pp33 is the phosphoprotein S6, present in ribos

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