Relevant bibliographies by topics / Ankylosis spondylitis

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Academic literature on the topic 'Ankylosis spondylitis'

Author: Grafiati
Published: 9 March 2023
Last updated: 10 March 2023

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Journal articles on the topic "Ankylosis spondylitis"

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Roth, RD. "Tarsal ankylosis in juvenile ankylosing spondylitis." Journal of the American Podiatric Medical Association 76, no. 9 (September 1, 1986): 514–18. http://dx.doi.org/10.7547/87507315-76-9-514.

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Haroon, Nigil. "Ankylosis in ankylosing spondylitis: current concepts." Clinical Rheumatology 34, no. 6 (May 3, 2015): 1003–7. http://dx.doi.org/10.1007/s10067-015-2956-4.

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Reinders, Antoinette, and Matthys J. Van Wyk. "Bamboo spine – X-ray findings of ankylosing spondylitis revisited." South African Journal of Radiology 16, no. 3 (September 10, 2012): 111–13. http://dx.doi.org/10.4102/sajr.v16i3.294.

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Ankylosing spondylitis is a debilitating disease that is one of the seronegative spondylarthropathies, affecting more males than females in the proportion of about 6:1 in the age group 15 - 35 years of age. Early radiographic findings include bilateral sacro-iliitis and early axial (lower lumbar spine) ankylosis. Typical X-ray findings are florid spondylitis (Romanus lesions), florid diskitis (Andersson lesions), early axial ankylosis, enthesitis, syndesmophytes and insufficiency fractures. Typical radiological abnormalities are pointed out on conventional X-rays and reviewed for early diagnosis and prompt treatment of patients at risk.
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Felstead, Andrew M., and Peter J. Revington. "Surgical Management of Temporomandibular Joint Ankylosis in Ankylosing Spondylitis." International Journal of Rheumatology 2011 (2011): 1–5. http://dx.doi.org/10.1155/2011/854167.

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Relatively few patients develop such severe degenerative temporomandibular joint (TMJ) disease that they require total joint replacement. Current indications include those conditions involving condylar bone loss such as degenerative (osteoarthritis) or inflammatory joint disease (ankylosing spondylitis, rheumatoid, and psoriatic). Ankylosis of the temporomandibular joint (TMJ) secondary to ankylosing spondylitis remains an under investigated entity. We aim to provide an overview of treatment objectives, surgical procedures, and our experience with total TMJ replacement for this condition.
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Khudaverdyan, Anahit Yu, Seda H. Devejyan, Ruben H. Davtyan, Azat A. Yengibaryan, Arshak A. Hovhanesyan, and Shota A. Vardanyan. "Female with Ankylosing Spondylitis from the 7th–6th century BCE Lori Berd burial (Armenia)." Anthropological Review 84, no. 1 (March 1, 2021): 85–100. http://dx.doi.org/10.2478/anre-2021-0005.

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Abstract A female skeleton from the Lori Berd archaeological cemetery, located near the city of Stepanavan (Lori Province of Armenia) is described. Palaeopathological analysis revealed a variety pathology (ankylosis of the sacroiliac joints, ankylosis of the vertebrae, syndesmophytes, ankylosed of the costovertebral and costotransverse joint fusions, kyphosis, lordosis, fracture of the anterior inferior iliac spine and traumatic lesions). This paper reports a new case of ankylosing spondylitis in a skeleton and a differential diagnosis performed to determine the etiology of the condition. The vertebral bodies remodel and together with the associated syndesmophytes form a continuous, smooth bone surface that is sometimes referred to as “bamboo spine”. In this skeleton changes in the spine, ribs, the sacrum, acetabulum, head of the femur and greater trochanter, as well as the anterior inferior iliac spine are typical of ankylosing spondylitis in advanced stage. Addtionally, there were signs of a traumatic death with injuries sustained to the scapula and vertebra. Using osteological markers in combination with the reconstruction of the archaeological context, the burial pattern suggests that the pathology the female suffered was likely due to her physical deficiencies.
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Xu, Bo, Fuqin Bao, Juan Ma, and Zhiqi Li. "Comparative Analysis of Computed Tomography and Magnetic Resonance Imaging in Clinically Examining Sacroiliac Joint Complications of Ankylosing Spondylitis." Journal of Medical Imaging and Health Informatics 10, no. 12 (December 1, 2020): 2940–43. http://dx.doi.org/10.1166/jmihi.2020.3236.

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Background: To analyze computed tomography (CT) and magnetic resonance Imaging (MRI) in examination diagnosis of sacroiliac joint disease of ankylosing spondylitis. Methods: Eighty patients with ankylosing spondylitis were randomly selected our experimental subjects, and CT and MRI examinations were performed on them to observe and analyze the diagnosis results of the two examination methods. Results: The detection rates of I and II for ankylosing spondylitis sacroiliac joint disease were prominently higher than those of CT (P < 0.05). Note that detection rates pertaining to MRI on ankylosing spondylitis, sacroiliac joint disease, erosion of the articular surface, and bone cysts under the articular surface were significantly higher than those of CT (P < 0.05). There exists no significant differences in the detection rates of widening or narrowing and joint ankylosis (P > 0.05). Conclusions: The detection rate of MRI in clinical diagnosis of sacroiliac joint disease of ankylosing spondylitis is higher than that of CT. It possesses excellent clinical promotion and application.
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Carter, Shea, Kirsten Braem, and Rik J. Lories. "The role of bone morphogenetic proteins in ankylosing spondylitis." Therapeutic Advances in Musculoskeletal Disease 4, no. 4 (April 18, 2012): 293–99. http://dx.doi.org/10.1177/1759720x12444175.

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Ankylosing spondylitis (AS), the best-known form of spondyloarthritis (SpA), is a remodelling arthritis characterized by chronic inflammation and bone formation. Ankylosis of the axial skeleton and sacroiliac joints leads to an impairment of spinal mobility, progressive spinal fusion and an increased risk of spinal fractures. The nature of the relationship between inflammation and new bone formation in AS has been controversial and questions remain as to whether there is a direct relationship between inflammation and new bone formation. Like others, we have hypothesized that the molecular pathways underlying ankylosis recapitulate the process of endochondral bone formation and that bone morphogenetic proteins (BMPs) play a key role in this process in AS. Furthermore, we discuss the entheseal stress hypothesis, which proposes that inflammation and ankylosis are linked but largely independent processes, and consider observations from mouse models and other human diseases which also imply that biomechanical factors contribute to the pathogenesis of AS. As current therapeutics, such as tumour necrosis factor inhibitors do not impede disease progression and ankylosis in AS, it is the pathways discussed in this review that are the now the focus for the identification of future drug targets.
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Stal, Rosalinde, Floris van Gaalen, Alexandre Sepriano, Juergen Braun, Monique Reijnierse, Rosaline van den Berg, Désirée van der Heijde, and Xenofon Baraliakos. "Facet joint ankylosis in r-axSpA: detection and 2-year progression on whole spine low-dose CT and comparison with syndesmophyte progression." Rheumatology 59, no. 12 (May 17, 2020): 3776–83. http://dx.doi.org/10.1093/rheumatology/keaa155.

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Abstract Objectives To evaluate the occurrence and progression of facet joint ankylosis in the whole spine using low-dose CT (ldCT) in radiographic axial spondyloarthritis (r-axSpA) and compare progression of facet joint ankylosis and syndesmophytes. Methods Patients with r-axSpA from the Sensitive Imaging in Ankylosing Spondylitis (SIAS) cohort underwent ldCT at baseline (n = 60) and 2 years (n = 53). Facet joints (right and left, levels C2-S1) were scored as ankylosed, not ankylosed or unable to assess. Joints that were frequently poorly visible (&gt;15% missing), were excluded. Inter-reader reliability on the patient level was assessed with intraclass correlation coefficients (ICCs) and smallest detectable change (SDC). Ankylosis was assessed at joint level and patient level for both timepoints. Syndesmophytes were assessed with CT syndesmophyte score. Results Levels C5-T2 were difficult to assess and excluded from all further analyses. Facet joint ICCs were good to excellent for status scores (0.72–0.93) and poor to excellent for progression scores (0.10–0.91). Facet joint ankylosis was detected at every level but most frequently in the thoracic joints. In total, 48% of patients showed 2-year progression. Most progression occurred in the thoracic segment. Using SDCs as cutoff, 18% of patients had progression of facet joint ankylosis only, whereas 20% of patients had progression of syndesmophytes only. Conclusion This is the first study evaluating facet joints in the whole spine by ldCT in r-axSpA. Facet joint ankylosis was detected most often in the thoracic spine. Assessing facet joints in addition to syndesmophytes detected substantially more patients with damage progression over two years.
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Сергей Франк, Михаил Франк, and Георгий Франк. "ИСПОЛЬЗОВАНИЕ МАНУАЛЬНОЙ ТЕРАПИИ В СОЧЕТАНИИ С УДАРНО-ВОЛНОВОЙ ТЕРАПИЕЙ У ПАЦИЕНТОВ С АНКИЛОЗИРУЮЩИМ СПОНДИЛИТОМ." World Science 2, no. 9(49) (September 30, 2019): 28–36. http://dx.doi.org/10.31435/rsglobal_ws/30092019/6708.

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Ankylosing spondylitis is a chronic systemic disease of the connective tissue which affects primarily joints and ligaments of the spine, peripheral joints and internal organs, causing progressive development of limited spinal mobility due to ankylosing of epiphyseal joints, formation of syndesmosis and calcification of vertebral ligaments. There are currently no methods that can completely cure or stop the progression of ankylosing spondylitis, however, manual therapy combined with shock- wave therapy is able to quickly, non-invasively, effectively, economically, safely and without side effects support patients, improving their quality of life, prolonging the duration of the remissions, reducing the frequency of relapses, significantly delaying the development of structural damage and the progression of bone ankylosis. This study discusses the effect of manual therapy combined with shock wave therapy on ankylosing spondylitis resistant to other treatments.
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Bouchrane, Ramadhane, Adama Bah, Harine Abdel Aziz Garba, Vanessa Lienou Tagne, Anne Stéphanie Elodie Doun Fouda, Moustapha Niasse, and Saïdou Diallo. "Ankylosing Spondylitis and Right Lower Limb Lymphoedema A Rare Association: A Case Report and Review of the Literature." SAS Journal of Medicine 9, no. 1 (January 14, 2023): 40–46. http://dx.doi.org/10.36347/sasjm.2023.v09i01.009.

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Introduction: Ankylosing spondylitis or rheumatic pelvispondylitis is a chronic inflammatory rheumatism characterized by an axial localization predominantly in the spine and sacroiliac joints, an involvement of the entheses and a tendency to bone ankylosis by ossification of these entheses. We report an observation of right lower limb lymphedema associated with ankylosing spondylitis. Observation: This was a 63-year-old patient with a history of inflammatory rheumatism (his older sister) and osteoporosis (his mother), He was diagnosed with ankylosing spondylitis based on enthesis involvement (bilateral tilted gluteal pain and low inflammatory back pain) with a positive HLA-B27 antigen by the 8-point Amor criteria for spondyloarthritis (right unilateral sacroiliitis, low inflammatory back pain, bilateral tilted gluteal pain, HLA-B27+ and NSAID sensitivity). The unilateral lymphedema with positive Stemmer's sign affected the lower 2/3 of the right lower limb. It occurred one year after the onset of ankylosing spondylitis. The echo-Doppler scan showed no signs of thrombosis or venous infiltration of the lower limbs, and diffuse infiltration of the subcutaneous tissue of the right leg without inflammatory character. Treatment of ankylosing spondylitis gradually improved the lymphedema. Conclusion: Lymphedema is a rare complication of ankylosing spondylitis. Dermatologists should be alert to early swelling of the extremities in patients with ankylosing spondylitis so as not to delay specific multidisciplinary treatment and measures to avoid irreversible lymphatic damage.
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Dissertations / Theses on the topic "Ankylosis spondylitis"

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Moz, Stefania. "Emerging role of monocytes and of their intracellular calcium content in spondyloarthritis." Doctoral thesis, Università degli studi di Padova, 2018. http://hdl.handle.net/11577/3425393.

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Background. The Spondyloarthritis (SpA) are a group of a multifactorial diseases characterised by a complex interplay between an inherited background and environmental factors that lead to immune response dysregulation and inflammation of the joints, mainly the sacro-ileal. Different from rheumatoid arthritis, there are no specific biomarkers for disease activity in the SpA that could be used in clinical practice. New biomarkers discovery could be helpful for early diagnosis, monitoring of disease activity, as well as for prognosis, outcome measures, and for assessing treatment efficacy. In SpA patients, macrophages infiltrating the inflamed joints, derive from circulating monocytes, express not only inflammatory cytokines, like TNF-α, IL-1β or TGF-β, but also enzymes causing tissue destruction and remodelling, like metalloproteinases. Metalloproteinases (MMPs), MMP3 in particular, have been reported to be highly expressed in synovial tissue and in peripheral blood of SpA patients. Recent studies have showed that MMP8 and MMP9, in particular, are produced by peripheral blood mononuclear cells (PBMCs) if they are stimulated by calprotectin (S100A8/S100A9 heterodimer). The SpA synovial tissue is characterized by an increased vascularization and an infiltrate composed of nucleated polymorphs, macrophages and lymphocytes. In these cells calcium signals are essential for various cellular functions, including proliferation, differentiation, apoptosis, and gene transcription. The aims of this work are to investigate whether the TNF-α, IL-1β, TGF-β, S100A8, S100A9, MMP3, MMP8 and MMP9 mRNA expression levels and intracellular calcium ([Ca2+]i) fluxes variations in PBMCs might be associated with SpA. Methods. The study population comprised 64 patients with a diagnosis of SpA (39 males and 25 females; mean age±standard deviation: 39.5±13.2 years) and 100 healthy controls (58 males and 42 females; mean age±standard deviation: 46.68.5). Among patients, 26 (40.6%) had diagnosis of Ankylosing Spondylitis (AS), (modified New York criteria) and 38 (59.3%) had a diagnosis of Psoriatic Arthritis (PsA) (CASPAR criteria). Blood samples were collected and complete blood count, CRP, ESR, uric acid, ALT and glucose were evaluated. Relative quantification (Real Time PCR) of TNF-α, IL-1β, TGF-β, S100A8, S100A9, MMP3, MMP8 and MMP9 mRNA were performed. Intracellular calcium ([Ca2+]i) fluxes were studied in patients and controls monocyte cells by a fluorescent microscope. Results. The mRNA expression levels in PBMCs of TNF-α, IL-1β, TGF-β were similar in AS and PsA patients when compared to controls. The variations of TNF-α, TGF-β and IL-1β were correlated each other. TNF-α mRNA expression levels also show a significant correlation if patient’s relatives with SpA where found (t=-2.5386, p=0.013). MMP8 and MMP9 mRNA expression levels did not vary between controls and patients, nor they were related to disease clinical activity indices. S100A9 mRNA expression did not vary, the expression of S100A8 (F=3.29, p=0.039) was reduced in PsA patients. S100A8 and S100A9 expression levels were significantly correlated with circulating inflammatory cells and S100A8 was correlated with CRP and ESR. Monocytes from healthy controls had evident and frequent ([Ca2+]i) oscillations, while SpA patients monocytes did not. The percentage of cells exhibiting ([Ca2+]i) oscillations profile was significantly lower in AS with respect to controls (F=6.15, p=0.003). The percentage of monocytes with intracellular calcium oscillations and the studied molecules were not correlated with the type of therapy or of drug used. Conclusions. SpA associates with a reduced expression of the inflammatory S100A8 calcium binding protein and with a decreased intracellular calcium fluxes in patients' cells compared to healthy subjects, suggesting that the presence of the disease affects the "on-off" mechanisms that regulate the concentration of intracellular calcium.
Introduzione. Le Spondiloartriti (SpA) sono un gruppo di malattie multifattoriali caratterizzate da una complessa interazione tra fattori genetici ed ambientali che determinano una disregolazione del sistema immunitario e l’attivazione di processi infiammatori a livello articolare, in particolar modo nelle articolazioni sacro-iliache. A differenza dell’artrite reumatoide, nelle SpA non esistono dei biomarcatori specifici di attività di malattia che vengono utilizzati nella pratica clinica. Pertanto, la ricerca di nuovi biomarcatori potrebbe essere d’ausilio per una diagnosi precoce e per un adeguato monitoraggio dell’attività di malattia, oltre che essere impiegati come fattori prognostici, misure di outcome e strumenti di valutazione dell’efficacia di trattamento.Nei pazienti con SpA, i macrofagi che infiltrano le articolazioni e che derivano principalmente dai monociti circolanti non esprimono solo citochine infiammatorie come TNF-α, IL-1β o TGF-β ma anche enzimi coinvolti nel rimodellamento tissutale come le metallo proteinasi di matrice (MMPs). La metalloproteinasi di matrice 3 (MMP-3), infatti, è riconosciuta come una molecola altamente espressa nel tessuto sinoviale e nel sangue periferico dei pazienti con SpA. Studi recenti hanno evidenziato che le metallo proteinasi di matrice 8 e 9 (MMP8 e MMP9) vengono prodotte dalle cellule mononucleate derivate da sangue periferico (PBMCs) quando vengono stimolate da calprotectina (eterodimero formato dalle proteine S100A8 e S100A9).Vi è poi una crescente evidenza del ruolo patogenetico nelle Spa svolto dalle cellule appartenenti all’immunità innata quali macrofagi, mastociti e neutrofili; il tessuto sinoviale dei pazienti con SpA infatti è caratterizzato da una elevata vascolarizzazione e quindi da una forte infiltrazione delle cellule immunitarie. In queste cellule i segnali di calcio intracellulare sono essenziali nella regolazione di numerose funzioni cellulari incluse proliferazione, differenziazione, apoptosi e trascrizione genica. Lo scopo di questo lavoro è stato quello di analizzare i livelli di espressione genica delle molecole TNF-α, IL-1β, TGF-β, S100A8, S100A9, MMP3, MMP8 e MMP9 e di valutare se le variazione dei flussi di calcio intracellulare ([Ca2+]i) nei PBMCs potrebbero essere associati alla presenza di SpA. Metodi.La popolazione studiata comprendeva 64 pazienti con diagnosi di SpA (età media ± deviazione standard: 39.5 ± 13.2 anni; 39 maschi, 25 femmine) e 100 controlli sani (età media ± deviazione standard: 46.6 ± 8.5 anni; 58 maschi, 42 femmine). Tra i pazienti, 26 (40.6%) presentavano spondilite anchilosante (AS) e 38 (59.3%) artrite psoriasica (PsA), con diagnosi formulata sulla base dei criteri rispettivamente di New York e CASPAR. Per ciascun soggetto arruolato, sono stati raccolti i dati demografici e fisiologici, la storia clinica e familiare. Sono stati raccolti poi, campioni di sangue, al fine di valutare l’emocromo e la VES, e di determinare i livelli di PCR, acido urico, prealbumina, alanina aminotransferasi (ALT) e glucosio. Per ciascun soggetto è stata effettuata un’analisi di espressione genica relativa (Real Time PCR) di TNF-α, IL-1β, TGF-β, S100A8, S100A9, MMP3, MMP8 e MMP9. La determinazione dei flussi di calico intracellulare ([Ca2+]i) nei pazienti e nei controlli è stata effettuate mediante microscopio ad epifluorescenza. Risultati. I livelli di espressione genica relativa nei PBMCs delle citochine infiammatorie of TNF-α, IL-1β, TGF-β erano simili nei pazienti con AS e PsA se comparati ai livelli di espressione nei controlli sani. Le variazioni dei livelli di espressione di TNF-α, TGF-β e IL-1β correlavano tra di loro. I livelli di espressione di TNF-α però risultavano correlati in maniera diretta, nei pazienti, con la presenza di una familiarità per la malattia (t=-2.5386, p=0.013). I livelli di espressione di MMP8 e MMP9 non risultavano essere associati con la diagnosi di SpA e non correlavano con gli indici clinici di attività di malattia.Anche i livelli di espressione di S100A9 non risultavano essere associati con la diagnosi di SpA mentre i livelli di espressione di S100A8 (F=3.29, p=0.039) erano ridotti nei pazienti con PsA.I livelli di espressione di S100A8 e S100A9 correlavano in maniera significativa con il numero di cellule infiammatorie circolanti e S100A8 correlava con i valori di PCR e VES. Dall’analisi dei dati ottenuti dai controlli sani e dai pazienti risultava evidente come la maggior parte dei monociti dei soggetti di controllo presentassero pulsazioni regolari di calcio intracellulare a differenza delle cellule ottenute da pazienti.I pazienti affetti da AS presentavano una ridotta percentuale di monociti con oscillazioni dei flussi di calcio intracellulare rispetto ai controlli sani (F=6.15, p=0.003). La percentuale di monociti con variazione di calcio intracellulare e l’ espressione delle molecole studiate non risultavano essere correlati ne con il tipo di terapia ne con il tipo di farmaco utilizzato. Conclusioni. In conclusione, I risultati di questo studio hanno evidenziato che nei pazienti con SpA vi è una ridotta espressione della proteina legante calcio S100A8 e vi è un decremento dei flussi di calcio intracellulare rispetto ai controlli sani, suggerendo che la presenza della malattia influenza i meccanismi "on-off" che regolano la concentrazione di calcio intracellulare.
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Karaderi, Tugce. "Genetics of ankylosing spondylitis." Thesis, University of Oxford, 2012. http://ora.ox.ac.uk/objects/uuid:8c0e848a-e712-4603-b923-a96a2f1644ac.

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Ankylosing spondylitis (AS) is a common inflammatory arthritis of the spine and other affected joints, which is highly heritable, being strongly influenced by the HLA-B27 status, as well as hundreds of mostly unknown genetic variants of smaller effect. The aim of my research was to confirm some of the previously observed genetic associations and to identify new associations, many of which are in biological pathways relevant to AS pathogenesis, most notably the IL-23/TH17 axis (IL23R) and antigen presentation (ERAP1 and ERAP2). Studies presented in this thesis include replication and refinement of several potential associations initially identified by earlier GWAS (WTCCC-TASC, 2007 and TASC, 2010). I conducted an extended study of IL23R association with AS and undertook a meta-analysis, confirming the association between AS and IL23R (non-synonymous SNP rs11209026, p=1.5 x 10-9, OR=0.61). An extensive re-sequencing and fine mapping project, including a meta-analysis, to replicate and refine the association of TNFRSF1A with AS was also undertaken; a novel variant in intron 6 was identified and a weak association with a low frequency variant, rs4149584 (p=0.01, OR=1.58), was detected. Somewhat stronger associations were seen with rs4149577 (p=0.002, OR=0.91) and rs4149578 (p=0.015, OR=1.14) in the meta-analysis. Associations at several additional loci had been identified by a more recent GWAS (WTCCC2-TASC, 2011). I used in silico techniques, including imputation using a denser panel of variants from the 1000 Genomes Project, conditional analysis and rare/low frequency variant analysis, to refine these associations. Imputation analysis (1782 cases/5167 controls) revealed novel associations with ERAP2 (rs4869313, p=7.3 x 10-8, OR=0.79) and several additional candidate loci including IL6R, UBE2L3 and 2p16.3. Ten SNPs were then directly typed in an independent sample (1804 cases/1848 controls) to replicate selected associations and to determine the imputation accuracy. I established that imputation using the 1000 Genomes Project pilot data was largely reliable, specifically for common variants (genotype concordence~97%). However, more accurate imputation of low frequency variants may require larger reference populations, like the most recent 1000 Genomes reference panels. The results of my research provide a better understanding of the complex genetics of AS, and help identify future targets for genetic and functional studies.
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Dean, Linda E. "Epidemiology of ankylosing spondylitis utilizing the Scotland and Ireland Registry for Ankylosing Spondylitis." Thesis, University of Aberdeen, 2015. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=225705.

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Background: For effective provision of healthcare, knowledge of disease epidemiology is of paramount importance. Within Ankylosing Spondylitis (AS), however, several areas are poorly understood, including information regarding disease prevalence and natural history, and the quality of life (QoL) of patients. This thesis aimed to address these areas, specifically to: a) determine the prevalence of AS, b) identify determinants of poor QoL, and c) describe the natural history of the disease. Methods: The Scotland and Ireland Registry for Ankylosing Spondylitis (SIRAS) was used to fulfill these aims and collects clinical and patient-reported information on all AS patients seen within Scottish secondary care. AS prevalence was calculated as the number of patients, per 10,000 adults, while QoL was described using the Ankylosing Spondylitis Quality of Life (ASQoL) questionnaire and factors associated with poor QoL assessed using Poisson regression. To assess the natural history of AS, the mean scores of commonly collected disease measures (including disease activity) were plotted yearly, from diagnosis. Results: The prevalence of AS was calculated to be 4.7 per 10,000. Poor QoL was associated with being retired/unemployed due to ill health, poor physical function, high disease activity, pain, fatigue, poor spinal mobility and being prescribed a biologic therapy. Regarding the natural history of AS; of the clinical measures studied, all remained relatively stable over time at the group level; however differences were demonstrated, when stratifying by gender, initial disease activity and by biologic use. Discussion/Conclusions: This study has added to the knowledge of disease in three main ways: accurate prevalence estimates aid the distribution of healthcare, the factors associated with reporting poor QoL may provide novel targets for future intervention and the reported consistency in the natural history of the disease may be used to reassure patients who present with low disease activity while initiating aggressive therapy for those with initial high activity.
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Sundström, Björn. "On diet in ankylosing spondylitis." Doctoral thesis, Umeå universitet, Reumatologi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-48557.

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The aim of this thesis was to examine the role of diet in ankylosing spondylitis (AS). Patients were examined in: i) a postal questionnaire survey of dietary habits and gastrointestinal (GI) symptoms; ii) a study on biomarkers of diet and disease activity; iii) a comparison of cardiovascular risk factors with the general population using data from the Västerbotten Intervention Programme (VIP), and; iv) a 21-week omega-3 fatty acid supplementation study regarding the effects on disease activity. The postal survey (111 respondents) revealed no correlation between dietary habits and disease activity measured by the Bath Ankylosing Spondylitis Disease Activity Index (BASDAI). However, GI problems, and in particular GI pain, were prevalent in patients with AS irrespective of NSAID usage.Gastrointestinal pain was predicted by higher BASDAI and a higher consumption of vegetables. Overall, 30 (27%) of the patients experienced an aggravation of gastric symptoms when consuming certain foods. In the study of biomarkers (n=66) no correlation was found between diet and disease activity as assessed by BASDAI. There were, however, positive correlations between BASDAI and the content of arachidonic acid (AA) in plasma phospholipids (rs=0.39, p<0.01) and the estimated activity of the enzyme delta-5-desaturase (rs=0.37, p<0.01). This may reflect a process involved in the inflammation associated with AS that requires further investigation. Comparing data from the VIP for patients (n=89) and controls showed no significant differences regarding diet, physical activity or smoking. Nonetheless, more pronounced correlations between blood lipids and diet were identified among patients than in controls. Furthermore, the levels of cholesterol and triglycerides were lower in patients compared with controls. Lastly, in the supplementation study, a high-dose of long-chain omega-3 fatty acids (4.55 grams/day) was found to lower disease activity, as measured by BASDAI, whereas low-dose treatment (1.95 grams/day) caused no change. In conclusion, within a group of Swedish AS patients we found no correlation between ordinary dietary habits and disease activity. Diet in western populations of patients with AS may, however, be of importance for gastric symptoms and for cardiovascular risk factors. The finding of a lowered disease activity in patients on high-dose supplementation with long-chain omega-3 fatty acids indicates that a radical dietary shift may influence disease activity. The findings of a positive correlation between disease activity and plasma AA, and the decreased levels of blood lipids imply the need for further studies into fatty acid metabolism in AS.
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Wright, Pamela Burnby. "The immunopathogenesis of ankylosing spondylitis." Thesis, University of Glasgow, 2013. http://theses.gla.ac.uk/4882/.

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The Spondyloarthritides (SpA) are a group of genetically and pathophysiologically related diseases. Ankylosing spondylitis (AS), the prototypic SpA family member, is a systemic inflammatory disease primarily affecting the axial skeleton, characterised by sacroiliitis and bone formation, promoting joint inhibition. AS is highly heritable; approximately 90% of AS susceptibility is defined by an individuals’ genetic background, to which the MHC class I molecule HLA-B27 contributes approximately 30%. This association was discovered 40 years ago, yet the pathogenic role of HLA-B27 remains elusive. Dendritic cells (DCs) belong to the myeloid lineage and, the principal antigen presenting cells (APCs) of the immune system, activate naïve T cells and contribute to the balance between activation and suppression of the immune response. If affected by HLA-B27, DCs are therefore likely to contribute to the T cell-mediated aspects of AS pathogenesis. Studies in our laboratory, using HLA-B27 transgenic (HLA-B27 TG) rats, have revealed HLA-B27-mediated effects on DC populations. The affected DCs induce abnormally high levels of IL-17 production from T cells; CCR6+ IL-17-secreting cells appear to be important in driving pathology both in the HLA-B27 TG rats and in AS patients. We therefore aimed to perform the first characterisations of the phenotype and functions of DCs and other myeloid populations purified directly from AS patients, to understand their role in AS pathogenesis. Analyses of circulating myeloid populations revealed that AS patients have a reduced proportion of the CD1c-expressing blood DCs, offset by an increase in CD14- CD16+ mononuclear cells. Interactions between CD14- CD16+ mononuclear cells and CD4+ T cells generated high levels of IL-6 secretion, required for the generation of Th17 cells. CD14- CD16+ mononuclear cells also induced T cells to express CCR6, and may therefore contribute to pathology by promoting Th17 responses. Interestingly, our data also indicate that APCs of mucosal origin may make a significant contribution to the systemic inflammation observed in AS patients. These observations give new insights into the pathogenic mechanisms in AS.
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Porter, Stuart B. "Exercise behaviour in ankylosing spondylitis." Thesis, University of Central Lancashire, 2009. http://clok.uclan.ac.uk/8575/.

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Background: Ankylosing spondylitis (AS) is an incurable, fluctuating, long-term condition for which prescribed exercise is central to management. However, many people with AS do not undertake prescribed exercises, the reasons for which are poorly-understood. Aims: The project sought to develop a grounded understanding of the exercise beliefs of people with AS, the exercise behaviours they adopt, and the decision making processes they undergo when choosing exercise behaviours. Design: Adopting a constructivist approach, 23 semi-structured interviews and 7 focus groups were undertaken with people with AS in the North West of England. In-depth data were analysed using open, axial and selective coding to inform the development of a conceptual model of exercise behaviour in AS. Ethical approval and informed consent were obtained. Key findings: 48 people participated in interviews and focus groups, with a disease duration ranging from 6 months to 29 years, and age range 19-62 (mean age 44.8 years ) there were 37 men and 11 women. Participants described the need to predict and respond to a changing disease trajectory and utilised a number of informed strategies for long- and short-term exercise management. Participants described a process of ongoing appraisal of their AS status and used approaches similar to cost—benefit analyses to make decisions about exercise behaviour. In the context of the patient journey, four discrete exercise behaviours (no prescribed exercise, other exercise, two tier behaviour, prescribed exercise), and four responses to changing disease status (no behaviour change, transient change, goal oriented change, non exercise change) were identified. Some key determinants of exercise behaviour were also revealed. Conclusion: This study has led to the grounding of participant perspectives on exercise in AS, rendering the proposed model both relevant and understandable to people with AS, and may have use in helping people understand and plan their future exercise behaviour in AS and forms a baseline for future study.
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Hidding, Alita. "Group physical therapy in ankylosing spondylitis." [s.l. : Maastricht : s.n.] ; University Library, Maastricht University [Host], 1993. http://arno.unimaas.nl/show.cgi?fid=5755.

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Jaakkola, Elisa Johanna. "Investigating genetic determinants of ankylosing spondylitis." Thesis, Open University, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.402693.

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Heuft-Dorenbosch, Elisabeth Louise Johanna. "From inflammatory back pain to ankylosing spondylitis." Maastricht : Maastricht : Universitaire Pers Maastricht ; University Library, Universiteit Maastricht [host], 2006. http://arno.unimaas.nl/show.cgi?fid=5379.

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Nemes, Lewis. "Problem specification of psychosocial and physical sequelae in ankylosing spondylitis : phase I of a competency-based model of coping." Virtual Press, 1989. http://liblink.bsu.edu/uhtbin/catkey/720132.

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The purpose of this two stage exploratory study was to investigate the psychosocial, and physical problems associated with a chronic form of arthritis, Ankylosing Spondylitis (AS), following the format of `Problem Specification' suggested by the competency-based model of coping (Goldfried and D'Zurilla, 1969). The first stage goal identified problem thoughts, feelings, and situations associated with AS, by thirteen AS sufferers, two physicians, two physical therapists, and ten significant others. The method for obtaining first stage information was an author constructed structured telephone interview. The second stage goal was the development of an author constructed survey instrument, the Inventory of Problems for Ankylosing Spondylitis (IPAS) completed by Iii individuals with AS, to identify problem frequency, and severity, both for the past twelve months, and for the total time a problem had been present. The second stage included a measure of social support, the Family Relationship Index, consisting of three scales from the Family Environmental Scales (Moos and Moos, 1986).Four areas of exploration were undertaken by this study which included: 1) Taxonomy of AS Problems - identification of AS problems 2) Frequency and Severity - how are identified problems rated and rank ordered 3) Social Support - how does social support affect responses to identified problems, and demographics 4) Demographics - how do such factors as age or gender affect responses to identified problems.The descriptive analysis confirmed that problems in the IPAS are problems experienced by the sample of AS participants. Items sorted into three categories of physical, relationship, and intrapersonal content areas were subjected to post hoc principle component analyses, yielding forty-five factors. Principle component analyses were conducted on first order factors within the three content areas yielding 13 second order orthogonal factors of AS problem themes which included work discrimination, and relationship difficulties with physicians. Significant findings were found for health ratings and second order factors `uncontrollability' and `stress'ratings of health status associated with lower levels of `uncontrollability' and `stress'. No significant results were found for social support and second order factors. The validity and reliability of the IPAS are suggested for future study, and current findings must be interpreted cautiously. Further research is recommended.
Department of Counseling Psychology and Guidance Services
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Books on the topic "Ankylosis spondylitis"

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Sieper, Joachim. Ankylosing spondylitis: In clinical practice. [London]: Springer, 2011.

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Weisman, Michael H. Ankylosing spondylitis. Oxford: Oxford University Press, 2011.

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van, Royen Barend J., and Dijkmans B. A. C, eds. Ankylosing spondylitis: Diagnosis and management. New York: Taylor & Francis, 2006.

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Ankylosing spondylitis. Oxford: Oxford University Press, 2008.

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Khan, Muhammad Asim. Ankylosing spondylitis. Oxford: Oxford University Press, 2002.

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Calabro, John J., and W. Carson Dick, eds. Ankylosing Spondylitis. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3231-9.

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Sieper, Joachim, and Jurgen Braun. Ankylosing Spondylitis. London: Springer London, 2011. http://dx.doi.org/10.1007/978-0-85729-180-6.

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Peer, Karen M. Living with ankylosing spondylitis. London: NASS, 1985.

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Nicknam, Mohammad Hossein, ed. Ankylosing Spondylitis - Axial Spondyloarthritis. Singapore: Springer Singapore, 2022. http://dx.doi.org/10.1007/978-981-16-4733-8.

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Ebringer, Alan. Ankylosing spondylitis and Klebsiella. London: Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-4300-0.

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Book chapters on the topic "Ankylosis spondylitis"

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Freemont, A. J. "Pathology of Ankylosing Spondylitis." In Ankylosing Spondylitis, 1–22. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3231-9_1.

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Khan, M. A. "HLA and Ankylosing Spondylitis." In Ankylosing Spondylitis, 23–44. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3231-9_2.

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Calabro, J. J. "Ankylosing Spondylitis: Early Diagnosis Based on the Natural History." In Ankylosing Spondylitis, 45–78. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3231-9_3.

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Husby, G., and J. T. Gran. "Ankylosing Spondylitis: Clinical Aspects, Comparisons, Men versus Women, Hospitalized versus Epidemiological Patients." In Ankylosing Spondylitis, 79–108. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3231-9_4.

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Jimenez, J., and G. Mintz. "The Onset, Evolution and Final Stages of Juvenile Ankylosing Spondylitis are Different from those of Adult Ankylosing Spondylitis." In Ankylosing Spondylitis, 109–16. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3231-9_5.

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Calabro, J. J. "Management of Ankylosing Spondylitis." In Ankylosing Spondylitis, 117–40. Dordrecht: Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3231-9_6.

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Sieper, Joachim, and Jürgen Braun. "Introduction." In Ankylosing Spondylitis, 1–3. London: Springer London, 2010. http://dx.doi.org/10.1007/978-0-85729-180-6_1.

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Sieper, Joachim, and Jürgen Braun. "Overview of ankylosing spondylitis." In Ankylosing Spondylitis, 5–12. London: Springer London, 2010. http://dx.doi.org/10.1007/978-0-85729-180-6_2.

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Sieper, Joachim, and Jürgen Braun. "Clinical manifestations of ankylosing spondylitis." In Ankylosing Spondylitis, 13–23. London: Springer London, 2010. http://dx.doi.org/10.1007/978-0-85729-180-6_3.

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Sieper, Joachim, and Jürgen Braun. "Diagnosis of ankylosing spondylitis." In Ankylosing Spondylitis, 25–37. London: Springer London, 2010. http://dx.doi.org/10.1007/978-0-85729-180-6_4.

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Conference papers on the topic "Ankylosis spondylitis"

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Kiapour, A., A. M. Kiapour, V. K. Goel, and K. Sairyo. "Effects of Spine Soft Tissue Stiffening on Biomechanics of Lumbar Spine: A FEA Study." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14664.

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Spine stiffening or also called Ankylosing Spondylitis (AS) is a type of progressive arthritis that leads to a chronic inflammation affecting spinal column and sacroiliac joints (SI). Long-term inflammation of the spinal joints (called Spondylitis) leads to calcium deposits forming on the ligaments and around the intervertebral discs. Subsequent tissue inflammation resulted from deposition of calcium, causes the spine to stiffen and potential mobility loss. This may result in clinical sequelae such as long-term disability and in complete fusion of vertebral column (Ankylosis) in severe cases [1]. Current literature on AS and its effects on segmental biomechanics is not known. Hence, this study designed to investigate the biomechanical effects of spine stiffening through changing the stiffness of soft tissue (ligaments & discs) across lumbar spine using finite element (FE) analysis.
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TANDAYA BENDAHAN, LOUISE, THAUANA LUIZA DE OLIVEIRA, ELOY DE AVILA FERNANDES, ARTUR DA ROCHA CORREA FERNANDES, CARLA GONCALVES SCHAHIN SAAD, and MARCELO DE MEDEIROS PINHEIRO. "Could the Total Spine MRI be useful for decision-making in patients with ankylosis spondylitis? Data from a 2-Year prospective study." In SBR 2021 Congresso Brasileiro de Reumatologia. Sociedade Brasileiro de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2021.1841.

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Scheer, Justin K., Jessica A. Tang, Vedat Deviren, Jenni M. Buckley, Murat Pekmezci, R. Trigg McClellan, and Christopher P. Ames. "Biomechanical Analysis of Cervicothoracic Junction Osteototomy in Cadaveric Model of Ankylosing Spondylitis: Effect of Rod Material and Diameter." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19130.

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Ankylosing spondylitis is a genetic condition [1] that frequently results in spinal sagittal plane deformity of thoracolumbar or cervicothoracic junction. Generally, a combination of osteotomy and spinal fixation is used to treat severe cases of ankylosing spondylitis to restore spinal balance and horizontal gaze [2]. This study investigates the biomechanics of opening wedge osteotomy at the cervicothoracic junction. Although surgical techniques for traumatic injury across the cervicothoracic junction have been well characterized in the clinical and biomechanical literature, the specific model of instrumented opening wedge ostetomy in autofused ankylosing spondylitis has not been studied biomechanically.
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Scheer, Justin K., Jessica A. Tang, Vedat Deviren, Jenni M. Buckley, Murat Pekmezci, R. Trigg McClellan, and Christopher P. Ames. "Biomechanical Analysis of Osteotomy Type (OWO, CWO) and Rod Diameter for Treatment of Cervicothoracic Kyphosis." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19128.

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Sagittal imbalance of the cervicothoracic spine often causes severe pain and loss of horizontal gaze. Traditionally, C7 opening wedge osteotomy has been classically performed for patients with ankylosing spondylitis. For patients without ankylosing spondylitis closing wedge osteotomy may be considered for more controlled closure. Biomechanical characteristics of the two osteotomy alternatives have not yet been analyzed. The goal of this study is to characterize the structural stability of the two types of cervicothoracic junction osteotomies and the independent effect of rod diameter.
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Gandaloeva, Zuleykhan, Olga Krichevskaya, and Tatiana Dubinina. "SAT0326 PLANS FOR MOTHERHOOD ARE CHANGED AFTER DIAGNOSING ANKYLOSIS SPONDYLITIS (AS). AS FEMALE PATIENTS’ ATTITUDE TO THE USE OF AS MEDICATIONS DURING PREGNANCY PLANNING AND CONCEPTION." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.3958.

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Ramos, Gabriel Bortoli, Rebecca Ranzani Martins, Júlia Carvalhinho Carlos de Souza, Cesar Castello Branco Lopes, and Guilherme Diogo Silva. "Spinal cord lesion and ischemic stroke after chiropractic: a case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.337.

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Context: Chiropractic is a form of spinal manipulation used to treat cervical pain. This therapy is considered safer than chronic use of anti-inflammatory drugs, opioids or spine surgery. However, chiropractic may cause severe complications such as myelopathy and ischemic stroke. Case report: A 48-year-old woman was admitted to our hospital due to acute tetraparesis. During a chiropractic session for chronic neck pain, patient referred lancinating cervical pain, weakness in the upper and lower limbs, and numbness below the level of the neck. Neurologic examination showed grade two tetraparesis with preserved arm abduction. Pain and vibratory sensation were reduced in trunk, upper and lower limbs. We considered a C5 level spinal cord injury. Cervical spine magnetic resonance imaging revealed a transdiscal fracture of C5-C6 vertebrae. The fracture led to an epidural hematoma and spinal cord compression. Cervical spine displayed ligamenta flava thickening, which may be associated with an undiagnosed ankylosing spondylitis. We also found bilateral vertebral occlusion of V1 and V2 segments. Cerebellar restricted diffusion suggested posterior circulation stroke. We believe that rigidity associated with ankylosing spondylitis favored spinal fracture during chiropractic. Patient was treated with 24mg/day of dexamethasone and 100mg/day of aspirin. Spinal cord decompression surgery was indicated. Unfortunately, in the last followup there was no improvement in patient motor status. Conclusions: Transdiscal C5-C6 fracture led to compressive myelopathy and cerebellar stroke. Safety of chiropractic should be better investigated in specific populations such as ankylosing spondylitis patients.
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Halm, Henry. "Ankylosing Spondylitis (AS)." In eccElearning Postgraduate Diploma in Spine Surgery. eccElearning, 2017. http://dx.doi.org/10.28962/01.3.075.

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Lee, So Yun, Seong Jong Yun, Ran Song, and Sang-Hoon Lee. "AB0734B BONY BRIDGE ON FACET JOINT OR LATERAL SIDE OF LUMBAR VERTEBRA IS MORE FREQUENT AND CORRELATED WITH LUMBAR FLEXION FUNCTION THAN THAT ON ANTERIOR SIDE OF LUMBAR VERTEBRA IN ANKYLOSING SPONDYLITIS; ANALYSIS OF ANKYLOSIS REGISTRY OF AXIAL SPONDYLOARTHRITIS (ANKAS)." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.2020.

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White, Allison, Hannah Abbott, Alfonse Masi, and Kalyani Nair. "Viscoelastic Properties of the L3-L4 Myofascial Tissue in Ankylosing Spondylitis Patients." In ASME 2018 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2018. http://dx.doi.org/10.1115/imece2018-87906.

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Ankylosing spondylitis (AS) is a degenerative rheumatological disorder that mainly affects the spine. It has been reported that different degrees of human resting myofascial tone (HRMT) would affect spinal stability and may predispose to the respective curvature deformities of adolescent idiopathic scoliosis (AIS) and the enthesopathy of ankylosing spondylitis (AS). Although osteoligamentous impacts are prominently recognized in many chronic spine and low back conditions, no research has been performed on the possible role of passive axial (spinal) myofascial tone as a causative factor. In this particular study, the passive muscle properties of the lower lumbar regions of 24 healthy adults and 24 adult AS subjects were examined. Our recent publications examined the linear elastic properties among normal and AS subjects. In this study, those analyses are expanded to include detailed analysis and correlations of the linear elastic property of stiffness to two viscoelastic properties: stress relaxation time (SRT) and creep. Analyzed data supports the hypothesis that resting muscle properties of the lower lumbar muscles hold significance in differentiation of human back health between healthy and diseased subjects, but more testing should be performed to support this study’s results.
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Borman, P., U. Seckin, H. Gunduz, N. Barca, and L. Damgaci. "SAT0045 Spondylodiscitis in ankylosing spondylitis." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.397.

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Reports on the topic "Ankylosis spondylitis"

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Tian, Cong, Jianlong Shu, Wenhui Shao, Zhengxin Zhou, Huayang Guo, and Jingang Wang. The efficacy and safety of IL Inhibitors, TNF-α Inhibitors, and JAK Inhibitor on ankylosing spondylitis: A Bayesian network meta-analysis of a “randomized, double-blind, placebo-controlled” trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0117.

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Review question / Objective: In this study, we conducted a Bayesian network meta-analysis to evaluate the efficacy and safety of interleukin (IL) inhibitors, tumor necrosis factor-alpha (TNF-α) inhibitors, and Janus kinase (JAK) inhibitors on ankylosing spondylitis (AS).The purpose of this study is to compare the effectiveness and safety of different interventions for treating AS to provide insights into the decision-making in clinicalpractice. Condition being studied: Ankylosing spondylitis. Based on the Bayesian hierarchical model, we conducted a network meta-analysis using the gemtc package in R software (version 4.1.3) and Stata software (version 15.1). Cong Tian and Jianlong Shu contributed to the conception and design of the study and supervised the tweet classification. All authors drafted the manuscript. Wenhui Shao, Zhengxin Zhou, Huayang Guo and Jingang Wang contributed to data management and tweet classification. Cong Tian, Jianlong Shu and Zhengxin Zhou performed the statistical analysis. Cong Tian, Jianlong Shu, Wenhui Shao and Zhengxin Zhou reviewed the manuscript.
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Chen, Menglu, Wen Li, Lailai Li, Yihui Chai, Yuqi Yang, and Xiang Pu. Ankylosing spondylitis disease activity and serum vitamin D levels: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2022. http://dx.doi.org/10.37766/inplasy2022.9.0030.

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Li, Jiaxiao, Suling Liu, and Yang Cui. Oxidative and Anti-oxidative stress-linked biomarkers in Ankylosing Spondylitis: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2020. http://dx.doi.org/10.37766/inplasy2020.5.0066.

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Zhao, Yu. Efficacy of alendronate for the treatment of ankylosing spondylitis: a protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review Protocols, April 2020. http://dx.doi.org/10.37766/inplasy2020.4.0153.

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Zhao, Pan-Pan, Yan-Hua Li, and Dong-Qin Xia. Efficacy of Biqi Capsule in the Adjuvant Treatment of Ankylosing Spondylitis : A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2022. http://dx.doi.org/10.37766/inplasy2022.3.0009.

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Liu, Hong-fei. Efficacy and safety of sulfasalazine for the treatment of ankylosing spondylitis: A systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0053.

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Yang, Lei, Lu Ma, Chao Zheng, Zhao-Wen Zeng, Fu Sheng, and Ying Nie. Traditional Chinese medicine formulations for treating ankylosing spondylitis: A protocol for systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0147.

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Dang, Sha, YuanYuan Ren, BoYi Zhao, XiangWei Meng, and ChaoYang Zhang. Efficacy and safety of warm acupuncture in the treatment of ankylosing spondylitis: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2020. http://dx.doi.org/10.37766/inplasy2020.11.0096.

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Chen, Jun, Lun-Bin Lu, Geng-Hua Tang, and Jun Xiong. The efficacy of moxibustion and acupuncture therapy for Ankylosing spondylitis: a protocol for an overview of systematic reviews and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2020. http://dx.doi.org/10.37766/inplasy2020.8.0035.

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Liu, Miao, Xiaolei Deng, and Jing Yu. Effectiveness and safety of Chinese herbal formula combined with western medicine for ankylosing spondylitis: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2021. http://dx.doi.org/10.37766/inplasy2021.5.0089.

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