Books: 'Anorexic Studies'

1

Bruch, Hilde. Conversationswith anorexics. New York: Basic Books, 1988.

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2

Bruch, Hilde. Conversations with anorexics. New York: Basic Books, 1988.

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3

Bruch, Hilde. Conversations with Anorexics. New York: Basic Books, 1989.

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4

Manara, Fausto. Anoressia nervosa: Tra psichiatria, psicologia e medicina. Milano, Italy: F. Angeli, 1991.

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5

Li-heyot mushlemet. [Tel-Aviv]: ha-Ḳibuts ha-meʾuḥad, 2002.

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6

Sacker, Ira M. Dying to be thin. New York, NY: Warner Books, 1987.

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7

Essstörungen und Depression: Psychobiologische Studien bei Anorexia nervosa und Bulimie. Frankfurt am Main: P. Lang, 1987.

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8

Heilpädagogisches Voltigieren bei Anorexia Nervosa: Eine Studie über die Wirksamkeit von Reittherapie auf das Körperbild und spezifische Persönlichkeitsmerkmale bei Anorexia Nervosa. Frankfurt am Main: P. Lang, 2004.

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9

Anorexia, murder, and suicide: What can be learned from the stories of three remarkable patients. Oxford: Butterworth-Heinemann, 1997.

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10

1951-, Lewis Gregg, ed. Dark marathon: The Mary Wazeter story : the ongoing struggles of a world-class runner. Grand Rapids, Mich: Zondervan, 1989.

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11

Magersüchtige und bulimische Patientinnen erzählen: Eine narrative Studie der Psychodynamik bei Essstörungen. Bern: Peter Lang, 2000.

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12

Grahl, Gary A. Skinny Boy. Chicago: American Legacy Media, 2007.

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13

Bauer, Barbara G. Bulimia: Book for therapist and client. Muncie, Ind: Accelerated Development, 1986.

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14

Bauer, Barbara G. Bulimia: Book for client and therapist. Muncie, Ind: Accelerated Development, 1986.

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15

Victorian Literature and the Anorexic Body (Cambridge Studies in Nineteenth-Century Literature and Culture). Cambridge University Press, 2006.

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16

Way, Karen. Anorexia Nervosa and Recovery: A Hunger for Meaning (Haworth Women's Studies) (Haworth Women's Studies). Haworth Press, 1996.

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17

E, Herzog, Deter Hans-Christian, and Vandereycken Walter 1949-, eds. The Course of eating disorders: Long-term follow-up studies of anorexia and bulimia nervosa. Berlin: Springer-Verlag, 1992.

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18

(Editor), W. Herzog, H. C. Deter (Editor), and W. Vandereycken (Editor), eds. Course of Eating Disorders: Long Term Follow-Up Studies of Anorexia & Bulimia Nervosa. Springer, 1992.

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19

The Long Road Back, A Survivors Guide to Anorexia. North Star Publications (MA), 1999.

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20

Pieters, Guido. Psychomotor Functioning in Anorexia Nervosa: Neuropsychological & Electrophysiological Studies (Acta Biomedica Lovaniensia). Leuven Univ Pr, 2006.

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21

Understanding Dunblane And Other Massacres Forensic Studies Of Homicide Paedophilia And Anorexia. Karnac Books, 2012.

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22

Grahl, Gary A. Skinny Boy: A Young Man's Battle and Triumph Over Anorexia. American Legacy Media, 2007.

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23

When the Mirror Lies: Anorexia, Bulimia, And Other Eating Disorders (Social Studies: Teen Issues). Franklin Watts, 2007.

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24

Reading Eating Disordrs: Writings on Bulimia and Anorexia As Confessions of American Culture (Neue Studien Zur Anglistik Und Amerikanistik, Bd. 87). Peter Lang Publishing, 2003.

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25

Olson, Greta. Reading Eating Disordrs: Writings On Bulimia And Anorexia As Confessions Of American Culture (Neue Studien Zur Anglistik Und Amerikanistik, Bd. 87). Peter Lang Publishing, 2003.

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26

Jacobi, Corinna, Kristian Hütter, and Eike Fittig. Psychosocial Risk Factors for Eating Disorders. Edited by W. Stewart Agras and Athena Robinson. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190620998.013.6.

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This chapter provides an updated overview of risk factors for eating disorders, on the basis of the risk factor taxonomy described by (Kraemer et al., 1997). It summarizes risk factors identified in longitudinal studies and markers and retrospective correlates from cross-sectional studies through April 2002 for anorexia nervosa, bulimia nervosa, and binge eating disorder, identifies new studies published between May 2002 and June 2015, and integrates them into the earlier review. The updated review confirms that longitudinal evidence on risk factors is strongest for nonspecific eating disorder diagnoses including subclinical forms and weakest for participants with diagnoses of anorexia nervosa. When strict criteria for caseness are applied, the majority of risk factors were not able to predict distinct diagnoses and only very few risk factors were confirmed in more than one sample. Case prediction, specificity, and replication therefore remain the biggest challenges in risk factor research for eating disorders.

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27

Steinhausen, Hans-Christoph. Eating Disorders in Adolescence: Anorexia and Bulimia Nervosa (International Studies on Childhood and Adolescence, No 3). Walter de Gruyter, 1996.

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28

Le Grange, Daniel, and Renee Rienecke. Family Therapy. Edited by W. Stewart Agras and Athena Robinson. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190620998.013.17.

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Family therapy is increasingly recommended as the treatment of choice for eating disorders among adolescents. The shift from blaming parents for causing an ED to seeing them as a necessary part of the recovery process was set in motion by Salvador Minuchin and colleagues, and then reinforced and expanded on by researchers at the Maudsley Hospital in London, UK, and in the United States and Australia. Data supporting the efficacy of family therapy for adolescent anorexia nervosa has been solidified, while family-based approaches in the treatment of adolescents with bulimia nervosa show promise. Further research is needed to replicate the findings of existing studies and to further clarify the utility of parental involvement in the treatment of older adolescents, or transition age youth, with anorexia nervosa and bulimia nervosa.

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29

Wendt, Julie, Anna Rotkiewicz, and Alice Berg. Geriatric Nutrition. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190466268.003.0002.

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The practice of integrative nutrition therapy aims to use food therapeutically in order to stimulate the body’s innate healing mechanisms and optimize health. Studies exploring the link between longevity and diet have reinforced the idea that health-promoting lifestyle habits delay the onset of age-related illness and death. As inflammatory processes drive chronic disease, any dietary intervention should aim to mitigate inflammation and promote the anti-inflammatory cascade. Practitioners can personalize the dietary interventions through the use of nutrigenomics and ancestral diets. This chapter covers nutrition assessment methods and key interventions to prevent weight loss, anorexia of aging, and malnutrition.

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30

Bulimia. Taylor & Francis Group, 2013.

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31

A Starving Madness: Tales of Hunger, Hope, and Healing in Psychotherapy. Gurze Books, 2002.

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32

Bauer, Barbara G., Wayne P. Anderson, and Robert W. Hyatt. Bulimia. Taylor & Francis Group, 2013.

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33

Dillon, Stephanie K., (Stephanie Kathryn). Girl lost and found: Through metaphor and drama : a narrative journey. $c2002, 2002.

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34

Boys Get Anorexia Too: Coping with Male Eating Disorders in the Family (Lucky Duck Books). Paul Chapman Educational Publishing, 2006.

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35

Langley, Jenny. Boys Get Anorexia Too: Coping with Male Eating Disorders in the Family (Lucky Duck Books). Paul Chapman Educational Publishing, 2006.

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36

Keel, Pamela K., and Lauren A. Holland. Eating Disorders. Edited by C. Steven Richards and Michael W. O'Hara. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.017.

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This chapter examines patterns of comorbidity between eating disorders and mood, anxiety, and substance use disorders along with evidence regarding support for different theoretical models that may account for these patterns. Although comorbidity estimates may be inflated by reliance on treatment-seeking samples and double counting of symptoms that overlap between syndromes, evidence supports elevated risk of mood, anxiety, and substance use disorders in anorexia nervosa, bulimia nervosa, and binge eating disorder. Data from family and twin studies support that eating and anxiety disorders may have a shared diathesis, consistent with the common cause model. Data from longitudinal studies suggest that eating disorders may increase vulnerability for developing a substance use disorder, consistent with the predisposition model. In contrast, comorbidity between eating and mood disorders, such as depression, remains poorly understood. Clinical issues regarding comorbidity of depression and eating disorders along with guidelines for clinicians treating patients with comorbid depression and eating disorders are discussed.

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37

Pike, Kathleen M., Loren M. Gianini, Katharine L. Loeb, and Daniel Le Grange. Treatments for Eating Disorders. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780199342211.003.0020.

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Substantial progress in advancing evidence-based treatments for eating disorders has been made. Many well-designed studies provide cumulative support for cognitive-behavioral therapy (CBT) as the treatment of choice for bulimia nervosa. Interpersonal psychotherapy (IPT) and pharmacotherapy are considered appropriate alternative treatments for bulimia nervosa. While CBT, IPT and pharmacotherapy often produce significant reductions in binge eating and compensatory behaviors, these treatment options need to be improved to help more individuals achieve full and lasting recovery. In the treatment of binge eating disorder, CBT and IPT have been shown to be the most efficacious in reducing symptoms and improving psychological outcomes. Weight loss is often an additional goal of those entering treatment for binge eating disorder; however, existing treatments have generally been unsuccessful in producing significant maintainable weight loss. Initial studies suggest that CBT may be associated with improved outcome both in the acute and maintenance phases of treatment for anorexia nervosa.

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38

Whisman, Mark A., and Briana L. Robustelli. Intimate Relationship Functioning and Psychopathology. Edited by Erika Lawrence and Kieran T. Sullivan. Oxford University Press, 2016. http://dx.doi.org/10.1093/oxfordhb/9780199783267.013.13.

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This chapter explores the association between intimate relationship functioning and psychopathology. It begins with a review of the literature on intimate relationship functioning and how it is correlated with the prevalence and incidence of psychopathology, focusing on the results of cross-sectional and longitudinal studies. It then considers the literature on couple-based interventions as treatments for specific forms of psychopathology such as mood disorders, substance use disorders, posttraumatic stress disorder (PTSD), obsessive-compulsive disorder, and anorexia nervosa. The chapter focuses on three couple-based interventions, namely, cognitive-behavioral couple therapy, emotion-focused couple therapy, and strategic approaches to couple therapy. It concludes by outlining directions for future research to better understand the link between intimate relationship functioning and psychopathology.

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39

Halmi, Katherine A. Psychological Comorbidities of Eating Disorders. Edited by W. Stewart Agras and Athena Robinson. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190620998.013.13.

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Psychological comorbidity of eating disorders may be conceptualized in varying facets including psychiatric diagnosis, specific behaviors, traits, affect regulation, and cognitive characteristics. Although the Diagnostic and Statistical Manual, fifth edition (DSM-5) modified some criteria for psychiatric diagnoses, these modifications should have little effect over the previous rates of DSM-IV comorbidities and thus do not necessitate repeat large sample comorbidity studies. This chapter presents facets of psychological comorbidities of the three major eating disorders: anorexia nervosa (AN), bulimia nervosa (BN), and binge eating disorder (BED). The most comprehensive comorbid psychiatric diagnosis study from the US national comorbidity survey replication revealed at least one lifetime comorbid psychiatric DSM-IV disorder was present in 56.2% AN, 94.5% BN, and 78.9% BED. Affect regulation, negative affect, perfectionism, cognitive-behavioral flexibility, and impulse control are common comorbid features present in these disorders.

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40

Allison, Kelly C., and Jennifer D. Lundgren. Emerging Syndromes. Edited by W. Stewart Agras and Athena Robinson. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780190620998.013.24.

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The Diagnostic and Statistical Manual, fifth edition, of the American Psychiatric Association (2013) has designated several disorders under the diagnosis of otherwise specified feeding and eating disorder (OSFED). This chapter evaluates three of these, night eating syndrome (NES), purging disorder (PD), and atypical anorexia nervosa (atypical AN). It also reviews orthorexia nervosa, which has been discussed in the clinical realm as well as the popular press. The history and definition for each is reviewed, relevant theoretical models are presented and compared, and evidence for the usefulness of the models is described. Empirical studies examining the disorders’ independence from other disorders, comorbid psychopathology, and, when available, medical comorbidities, are discussed. Distress and impairment in functioning seem comparable between at least three of these emerging disorders and threshold eating disorders. Finally, remaining questions for future research are summarized.

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41

Morris, Katherine J. Body Image Disorders. Edited by K. W. M. Fulford, Martin Davies, Richard G. T. Gipps, George Graham, John Z. Sadler, Giovanni Stanghellini, and Tim Thornton. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199579563.013.0037.

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This chapter examines so-called body image disorders, focusing on body dysmorphic disorder, anorexia nervosa, bulimia nervosa, and binge eating disorder. These disorders have been studied extensively by psychologists and psychiatrists from both the "body image" and "body shame" research orientations. Body image disorders have also proved, for feminist thinkers mindful of the gender imbalance in many of these disorders, to be an important locus for cultural criticism, including criticism of psychological and psychiatric perspectives. Those philosophers and anthropologists with a phenomenological bent, particularly those with an interest in the lived body and embodiment, have also found a fruitful terrain in body image disorders. These different disciplines and approaches provide multiple perspectives which are often complementary, occasionally in some tension with one another, but always mutually enriching, and all of them are sketched here.

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42

Feldman, Talya, Cristin D. Runfola, and James Lock. Feeding and Eating Disorders. Edited by Thomas H. Ollendick, Susan W. White, and Bradley A. White. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780190634841.013.23.

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Eating disorders are severe, life-threatening psychological disorders that frequently manifest in children and adolescents. This chapter provides an overview of the prevalence, epidemiology, assessment, and treatment of the six child and adolescent feeding and eating disorders covered by the fifth edition of the Diagnostic and Statistical Manual of Mental Disorders: pica, rumination disorder, avoidant/restrictive food intake disorder, anorexia nervosa, bulimia nervosa, binge-eating disorder, and other specified feeding or eating disorder. Existing research is limited, but the most evidenced treatments, depending on disorder, are applied behavioral analysis; individualized behavior plans; family-based therapy; cognitive behavioral therapy; and self-help. Given the limited number of randomized controlled studies and the moderate (at best) recovery rates, future research should investigate possible adjunctive treatments (i.e., cognitive remediation therapy, dialectical behavior therapy); adaptations for specific populations; and dissemination and implementation improvements (i.e., phone or Internet delivered interventions).

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43

Glannon, Walter. Psychiatric Neuroethics I. Edited by John Z. Sadler, K. W. M. Fulford, and Werdie (C W. ). van Staden. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780198732372.013.30.

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Severe psychiatric disorders may be resistant to conventional pharmacological and psychotherapeutic treatments. Invasive interventions such as deep-brain stimulation (DBS) and neurosurgical ablation (lesioning) can modulate dysfunctional neural circuits implicated in these disorders. Yet these two forms of psychiatric neurosurgery are still experimental and investigational and thus their safety and efficacy have yet to be established. This chapter is an examination and discussion of the main ethical issues surrounding the experimental use of DBS and lesioning for treatment-refractory psychiatric disorders. I address questions regarding research subjects’ exposure to risk and informed consent to be enrolled in clinical trials testing these techniques for major depression and obsessive-compulsive disorder. These questions include whether or to what extent the therapeutic misconception influences decisions to enroll in these trials. I then explore similar questions about the use of DBS for schizophrenia and anorexia nervosa. Finally, I discuss the obligations of researchers conducting these studies to research subjects.

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44

Wójcik-Gładysz, Anna. Ghrelin – hormone with many faces. Central regulation and therapy. The Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, 2020. http://dx.doi.org/10.22358/mono_awg_2020.

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Discovered in 1999, ghrelin, is one of the peptides co-creating the hypothalamicgastrointestinal axis, otherwise known as the brain-gut axis. Ghrelin participates in many physiological processes and spectrum of its activity is still being discovered. This 28 amino acid peptide ‒ a product of the ghrl gene, was found in all vertebrates and is synthesized and secreted mainly from enteroendocrine X/A cells located in the gastric mucosa of the stomach. Expression of the ghrelin receptor has been found in many nuclei of the hypothalamus involved in appetite regulation. Therefore it’s presumed that ghrelin is one of the crucial hormones deciphering the energy status required for the maintenance of organism homeostasis. Ghrelin acts as a signal of starvation or energy insufficiency and its level in plasma is reduced after the meal. Neuropeptide Y (NPY) and agouti-related peptide (AgRP; NPY/AgRP) neurons located in the arcuate nucleus (ARC) area are the main target of ghrelin in the hypothalamus. This subpopulation of neurons is indispensable for inducing orexigenic action of ghrelin. Moreover ghrelin acting as a neurohormone, mainly in the hypothalamus area, plays an important role in the regulation of growth and reproduction processes. Indeed, ghrelin action on reproductive processes has been observed in the systemic effects exerted at both hypothalamus-pituitary and gonadal levels. Similarly the GH-releasing ghrelin action was observed both on the hypothalamus level and directly on the somatotrophic cells in the pituitary and this dose-related GH releasing activity was found in in vitro as well as in in vivo experiments. In recent years, numerous studies revealed that ghrelin potentially takes part in the treatment of diseases associated with serious disturbances in the organism energy balance and/or functioning of the gastrointestinal tract. It was underlined that ghrelin may be a hormone with a broad spectrum of therapeutic effect on obesity and anorexia nervosa, as well as may also have protective effect on neurodegenerative diseases, inflammatory disorders or functional changes in the body caused by cancers. In overall, ghrelin treatment has been tested in over 100 preclinical studies with healthy volunteers as well as patients with various types of cancer, eating disorders such as anorexia nervosa and bulimia nervosa. It was observed that ghrelin has an excellent clinical safety profile and emerging side effects occurred only in 3–10% of patients and did not constitute a sufficient premise to discontinue the therapy. In general, it can be concluded that ghrelin may be sufficiently used as a prescription drug.

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Books on the topic 'Anorexic Studies'

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