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Journal articles on the topic 'Anti-arthritis'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Chaabo, Khaldoun, and Bruce Kirkham. "Rheumatoid Arthritis — Anti-TNF." International Immunopharmacology 27, no. 2 (2015): 180–84. http://dx.doi.org/10.1016/j.intimp.2015.04.051.

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2

KAKUMANU, PRASANTHI, ERIC S. SOBEL, SONALI NARAIN, et al. "Citrulline Dependence of Anti-Cyclic Citrullinated Peptide Antibodies in Systemic Lupus Erythematosus as a Marker of Deforming/Erosive Arthritis." Journal of Rheumatology 36, no. 12 (2009): 2682–90. http://dx.doi.org/10.3899/jrheum.090338.

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Objective.Anti-cyclic citrullinated peptide (CCP) antibodies are a serological marker for rheumatoid arthritis (RA); up to 10%–15% of patients with systemic lupus erythematosus (SLE) are also positive. While anti-CCP in RA is citrulline-dependent, anti-CCP in some other diseases is citrulline-independent and reacts with both CCP and the unmodified (arginine-containing) cyclic arginine peptide (CAP). We investigated the citrulline dependence of anti-CCP and its significance in the arthritis of SLE.Methods.IgG anti-CCP was compared by ELISA to anti-CAP in sera from patients with SLE (n = 335) an

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Inanc, N., E. Dalkılıc, S. Kamalı, et al. "Anti-CCP antibodies in rheumatoid arthritis and psoriatic arthritis." Clinical Rheumatology 26, no. 1 (2006): 17–23. http://dx.doi.org/10.1007/s10067-006-0214-5.

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4

Stewart, I., and J. C. Ridway. "Anti-Ro positive rheumatoid arthritis." Annals of the Rheumatic Diseases 45, no. 1 (1986): 86–87. http://dx.doi.org/10.1136/ard.45.1.86-b.

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5

HERZOG, C. "MONOCLONAL ANTI-CD4 IN ARTHRITIS." Lancet 330, no. 8573 (1987): 1461–62. http://dx.doi.org/10.1016/s0140-6736(87)91158-5.

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6

Larkin, Marilynn. "Nutraceuticals' anti-arthritis benefits “exaggerated”." Lancet 355, no. 9208 (2000): 993. http://dx.doi.org/10.1016/s0140-6736(05)74734-6.

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7

Mergaert, Aisha M., Mandar Bawadekar, Thai Q. Nguyen, et al. "Reduced Anti-Histone Antibodies and Increased Risk of Rheumatoid Arthritis Associated with a Single Nucleotide Polymorphism in PADI4 in North Americans." International Journal of Molecular Sciences 20, no. 12 (2019): 3093. http://dx.doi.org/10.3390/ijms20123093.

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Autoantibodies against citrullinated proteins are a hallmark of rheumatoid arthritis, a destructive inflammatory arthritis. Peptidylarginine deiminase 4 (PAD4) has been hypothesized to contribute to rheumatoid arthritis by citrullinating histones to induce neutrophil extracellular traps (NETs), which display citrullinated proteins that are targeted by autoantibodies to drive inflammation and arthritis. Consistent with this theory, PAD4-deficient mice have reduced NETs, autoantibodies, and arthritis. However, PAD4′s role in human rheumatoid arthritis is less clear. Here, we determine if single

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8

MOHAMMED, KHULOOD, JANET POPE, NICOLE LE RICHE, et al. "Association of Severe Inflammatory Polyarthritis in Primary Sjögren’s Syndrome: Clinical, Serologic, and HLA Analysis." Journal of Rheumatology 36, no. 9 (2009): 1937–42. http://dx.doi.org/10.3899/jrheum.080234.

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Objective.To evaluate the clinical, serologic, and MHC class II antigen characteristics of a group of patients with primary Sjögren’s syndrome (SS) and severe arthritis.Methods.A case-control study comparing 35 patients with primary SS: 17 with inflammatory arthritis, 18 without arthritis.Results.All patients fulfilled criteria for primary SS. There were no demographic or clinical features other than inflammatory arthritis, often erosive, that distinguished patients with arthritis from those without. All patients had anti-Ro/SSA autoantibodies, most had anti-La/SSB autoantibodies, and a high p

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De Vries, Charlotte, Alexandra Cîrciumaru, Linda Mathsson-Alm, et al. "Porphyromonas gingivalisassociates with the presence of anti-citrullinated protein antibodies, but not with the onset of arthritis: studies in an at-risk population." RMD Open 11, no. 1 (2025): e005111. https://doi.org/10.1136/rmdopen-2024-005111.

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ObjectiveThe antibody response againstPorphyromonas gingivalis(Pg) is elevated in rheumatoid arthritis (RA), especially in patients with anti-citrullinated protein antibodies (ACPA). Here, we investigated whether antibodies against thePgvirulence factor arginine gingipain (Rgp) are associated with the RA-risk phase and development of arthritis.MethodsAt-risk individuals were included in a prospective study (Risk-RA) based on a positive anti-cyclic citrullinated peptide 2 (CCP2) antibody test, and having musculoskeletal complaints but no signs of arthritis. Study participants were followed for

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10

Dong, Baoqi, Wenqing Wu, Jingbao Chen, and Xiao-qin Chu. "The Anti-Arthritis Effect of Cinnamaldehyde on Adjuvant Arthritis Rats." International Journal of Pharmacology 18, no. 3 (2022): 551–61. http://dx.doi.org/10.3923/ijp.2022.551.561.

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11

Faheem, Muhammad Asif, Tasleem Akhtar, Nadia Naseem, et al. "Chrysin Is Immunomodulatory and Anti-Inflammatory against Complete Freund’s Adjuvant-Induced Arthritis in a Pre-Clinical Rodent Model." Pharmaceutics 15, no. 4 (2023): 1225. http://dx.doi.org/10.3390/pharmaceutics15041225.

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Chrysin (5,7-dihydroxyflavone) has many pharmacological properties including anti-inflammatory actions. The objective of this study was to evaluate the anti-arthritic activity of chrysin and to compare its effect with the non-steroidal anti-inflammatory agent, piroxicam, against complete Freund’s adjuvant (CFA)-induced arthritis in a pre-clinical model in rats. Rheumatoid arthritis was induced by injecting CFA intra-dermally in the sub-plantar region of the left hind paw of rats. Chrysin (50 and 100 mg/kg) and piroxicam (10 mg/kg) were given to rats with established arthritis. The model of art

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12

Cooper, S. M., S. Sriram, and G. E. Ranges. "Suppression of murine collagen-induced arthritis with monoclonal anti-Ia antibodies and augmentation with IFN-gamma." Journal of Immunology 141, no. 6 (1988): 1958–62. http://dx.doi.org/10.4049/jimmunol.141.6.1958.

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Abstract Collagen-induced arthritis (CIA) is an experimental model in which a specific immune response to type II collagen (CII) is associated with the development of inflammatory arthritis. In this study, we evaluated the effects of early and delayed treatments with anti-Ia mAb and IFN-gamma on murine CIA. Administration of anti-Ia mAb at the time of immunization with CII decreased the incidence and delayed the onset of arthritis, whereas anti-Ia treatments begun 2 wk after immunization had no effect upon either arthritis incidence or onset. Neither treatment protocol resulted in a significan

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13

Khaleel, Khoshawi, and Saeed Hussain. "The prevalence of thyroid dysfunction and thyroid autoantibodies among patients with rheumatoid arthritis." Zanco Journal of Medical Sciences 26, no. 1 (2022): 78–87. http://dx.doi.org/10.15218/zjms.2022.009.

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Background and objective: Rheumatoid arthritis is a chronic and inflammatory autoimmune systemic disease of unknown cause that may affect many tissues and organs. Rheumatoid arthritis and thyroid disorders may occur in the patient simultaneously. The main antigens that give rise to thyroid antibodies are thyroglobulin, thyroid peroxidase, anti-cyclic citrullinated protein, and thyroid hormone receptor. This study aimed to determine the frequency of thyroid dysfunction and seroprevalence of anti-thyroid antibody in patients with rheumatoid arthritis and its association with inflammatory marker

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14

Khaleel, Khoshawi, and Saeed Hussain. "The prevalence of thyroid dysfunction and thyroid autoantibodies among patients with rheumatoid arthritis." Zanco Journal of Medical Sciences 26, no. 1 (2022): 78–87. http://dx.doi.org/10.15218/zjms.2022.009.

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Background and objective: Rheumatoid arthritis is a chronic and inflammatory autoimmune systemic disease of unknown cause that may affect many tissues and organs. Rheumatoid arthritis and thyroid disorders may occur in the patient simultaneously. The main antigens that give rise to thyroid antibodies are thyroglobulin, thyroid peroxidase, anti-cyclic citrullinated protein, and thyroid hormone receptor. This study aimed to determine the frequency of thyroid dysfunction and seroprevalence of anti-thyroid antibody in patients with rheumatoid arthritis and its association with inflammatory marker

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15

Yoshino, Shin. "Disease-specific regulation of antibody-mediated immune diseases by the pathogenic antibody Fab fragments (107.15)." Journal of Immunology 186, no. 1_Supplement (2011): 107.15. http://dx.doi.org/10.4049/jimmunol.186.supp.107.15.

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Abstract Ag-specific immunotherapies for Ab-mediated diseases including asthma and rheumatoid arthritis have been unavailable. In the present study, we investigated whether Ab-mediated asthmatic responses as well as arthritis in mice were able to be Ag-specifically regulated by the pathogenic Fabs. Asthmatic responses were induced in BALB/c mice by i.p. injection of anti-OVA Abs followed by intratracheal (i.t.) challenge with OVA. Arthritis was induced in DBA/1J mice by i.v. injection of anti-OVA Abs and then by intraarticular injection of OVA. Fabs from anti-OVA Abs were prepared by digestion

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16

Gaurav, Chaudhary Lovy Sharma. "Synthesis and Bioactivity Studies of Curcumin-NSAID Conjugates." International Journal of Pharmaceutical Sciences 2, no. 8 (2024): 3220–35. https://doi.org/10.5281/zenodo.13333231.

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In the current study, four different curcumin-NSAID conjugates are synthesized, and their biological screening for rheumatoid arthritis, anti-inflammatory, and anti-ulcer properties follows. One phytochemical with a wide range of therapeutic applications is curcumin. In the sense that it may aid in achieving the goal of incorporating specific desired properties in curcumin, such as enhanced bioavailability, reduction in side effects, and also offering good therapeutic potential in terms of determination of rheumatoid arthritis, anti-inflammatory and anti-ulcer activities of synthesized conjuga

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17

Neeraj, Srivastava, Dashora Nipun, Menaria Jyoti, and Kumar Neeraj. "Evaluation of Anti-inflammatory and Anti-arthritic Activity of Ajmodadi Churna- A Polyherbal Formulation." International Journal of Pharmaceutical and Phytopharmacological Research 6, no. 5 (2016): 72. http://dx.doi.org/10.24896/eijppr.2016651.

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Anti-inflammatory and anti- arthritic activity of aqueous extract of Ajmodadi Churna (AJM) were evaluated by three methods namely, Carrageenan paw edema, Carrageenan induced Air Pouch Model in Rats and Freunds’ complete adjuvant Arthritis. The Carrageenan paw edema was carried out to test the effect of the extract on acute phase of inflammation. Carrageenan induced Air Pouch Model was used for local inflammation and Freunds’ complete adjuvant Arthritis was used for evaluation of chronic inflammation. Results showed that AJM have significant anti-inflammatory activity and Anti-arthritic Activit

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18

Gejalakshmi S, Harikrishnan N, Rehka V, et al. "Complementary Review Of Anti Arthritis Activity Of Pithcellobium Dulce Plant Extract." International Journal of Research in Pharmaceutical Sciences 11, SPL4 (2020): 1377–85. http://dx.doi.org/10.26452/ijrps.v11ispl4.4309.

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Rheumatoid arthritis (RA) is a chronic autoimmune disorder. It generates joint inflammation and synovitis. Which influence more or less 20 % of the population in India. The arthritis symptoms are created step by step. The major symptom of RA is a pain in the joint. The high level of rheumatoid factor ( RF ) in the blood is most often associated with autoimmune disease such as rheumatoid arthritis & sjogren's syndrome. The Normal range of RF is from 0-20 IU / ml. The anti-arthritis activity evaluation can be studied by using an animal model. The anti-arthritis activity can be done using pit

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19

P. Muralidharan, M. Vignesh, P. Madhu maya devi, S. Swetha, L. Gop, and K. Yuvaraj. "Exploring the anti-arthritic and anti- inflammatory activity of ethanol extract of Mimosa pudica leaves against complete Freund’s adjuvant (CFA) induced arthritis in Wistar albino rats." International Journal of Science and Research Archive 13, no. 2 (2024): 1918–29. https://doi.org/10.30574/ijsra.2024.13.2.2280.

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The study explores the potential anti-arthritic and anti-inflammatory effects of an ethanol extract from Mimosa pudica leaves in a rat model of arthritis induced by Complete Freud’s Adjuvant (CFA). The extract’s efficacy was evaluated by measuring key markers associated with inflammation and arthritis. The results revealed a significant reduction in inflammation and arthritis-related symptoms in the treatment group, suggesting the extract's strong anti-inflammatory properties. The findings highlight the therapeutic potential of Mimosa pudicas a natural remedy for arthritis, which could be attr

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20

Avnish Kumar, Swati Tiwari, Ramdarshan Parashar, Ajay Thakur, and Yogesh Sharma. "Assessment of anti-arthritic efficacy of the ethanolic extract of the entire Morinda citrifolia Plant." World Journal of Biology Pharmacy and Health Sciences 20, no. 3 (2024): 352–57. https://doi.org/10.30574/wjbphs.2024.20.3.1024.

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The study investigates the anti-arthritic and anti-inflammatory properties of an ethanol extract of the entire Morinda citrifolia plant in a rat model of arthritis caused by Complete Freud's Adjuvant (CFA). The extract's effectiveness was assessed by assessing key indicators of inflammation and arthritis. The results showed a substantial reduction in inflammation and arthritis-related symptoms in the treatment group, indicating that the extract had potent anti-inflammatory capabilities. The findings point out the therapeutic potential of Morinda citrifolia, a natural arthritis treatment, which

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21

Seymour, H. E., A. Worsley, J. M. Smith, and S. H. L. Thomas. "Anti-TNF agents for rheumatoid arthritis." British Journal of Clinical Pharmacology 51, no. 3 (2001): 201–8. http://dx.doi.org/10.1046/j.1365-2125.2001.00321.x.

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22

Maini, R. N., and P. C. Taylor. "Anti-Cytokine Therapy for Rheumatoid Arthritis." Annual Review of Medicine 51, no. 1 (2000): 207–29. http://dx.doi.org/10.1146/annurev.med.51.1.207.

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23

MUSCAT, J. E., and E. L. WYNDER. "Anti-inflammatory Drugs and Rheumatoid Arthritis." JNCI Journal of the National Cancer Institute 85, no. 11 (1993): 921–22. http://dx.doi.org/10.1093/jnci/85.11.921-a.

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24

Yavasoglu, Irfan, Taskin Senturk, Adil Coskun, and Zahit Bolaman. "Rheumatoid arthritis and anti-thyroid antibodies." Autoimmunity 42, no. 2 (2009): 168–69. http://dx.doi.org/10.1080/08916930802428114.

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25

Cabezas-Rodríguez, Iván, Isla Morante-Bolado, Anahy Brandy-García, Rubén Queiro-Silva, Lourdes Mozo, and Francisco Javier Ballina-García. "Anti-MDA5 dermatomyositis mimicking psoriatic arthritis." Reumatología Clínica 14, no. 4 (2018): 224–26. http://dx.doi.org/10.1016/j.reuma.2016.10.010.

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26

Cabezas-Rodríguez, Iván, Isla Morante-Bolado, Anahy Brandy-García, Rubèc)n Queiro-Silva, Lourdes Mozo, and Francisco Javier Ballina-García. "Anti-MDA5 dermatomyositis mimicking psoriatic arthritis." Reumatología Clínica (English Edition) 14, no. 4 (2018): 224–26. http://dx.doi.org/10.1016/j.reumae.2016.10.011.

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27

Elliott, Michael J., and Ravinder N. Maini. "Anti-cytokine therapy in rheumatoid arthritis." Baillière's Clinical Rheumatology 9, no. 4 (1995): 633–52. http://dx.doi.org/10.1016/s0950-3579(05)80306-1.

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28

Robinson, William, Wolfgang Hueber, Peggy P. Ho, and Xiaoyan Zhao. "Anti-fibrinbogen Autoimmunity in Rheumatoid Arthritis." Clinical Immunology 123 (2007): S91—S92. http://dx.doi.org/10.1016/j.clim.2007.03.442.

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29

Roudier, Jean, Nathalie Balandraud, and Isabelle Auger. "Anti PAD autoimmunity and rheumatoid arthritis." Joint Bone Spine 85, no. 6 (2018): 659–61. http://dx.doi.org/10.1016/j.jbspin.2018.03.017.

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30

Dong, Lei, Suhua Xia, Huan Chen, Jiangning Chen, and Junfeng Zhang. "Anti-arthritis activity of cationic materials." Journal of Cellular and Molecular Medicine 14, no. 7 (2010): 2015–24. http://dx.doi.org/10.1111/j.1582-4934.2009.00806.x.

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31

Geiler, Janina, Maya Buch, and Michael F. McDermott. "Anti-TNF Treatment in Rheumatoid Arthritis." Current Pharmaceutical Design 17, no. 29 (2011): 3141–54. http://dx.doi.org/10.2174/138161211798157658.

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32

Bosworth, Ailsa. "Anti-TNF drugs for rheumatoid arthritis." British Journal of Healthcare Management 11, no. 5 (2005): 148–51. http://dx.doi.org/10.12968/bjhc.2005.11.5.23994.

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33

Roll, P., and H. P. Tony. "Anti-CD20-Therapie bei rheumatoider Arthritis." Zeitschrift für Rheumatologie 68, no. 5 (2009): 370–79. http://dx.doi.org/10.1007/s00393-009-0437-3.

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34

Karami, Pari, Danial Gholamin, Farzaneh Fathi, Taha Afsar, and Mohammad Johari-Ahar. "Anti-CCP biosensors in rheumatoid arthritis." Clinica Chimica Acta 561 (July 2024): 119767. http://dx.doi.org/10.1016/j.cca.2024.119767.

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35

Faustino, Célia, Noélia Duarte, and Lídia Pinheiro. "Triterpenes Drug Delivery Systems, a Modern Approach for Arthritis Targeted Therapy." Pharmaceuticals 17, no. 1 (2023): 54. http://dx.doi.org/10.3390/ph17010054.

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Arthritis is a major cause of disability. Currently available anti-arthritic drugs, such as disease-modifying anti-rheumatic drugs (DMARDs), have serious side-effects associated with long-term use. Triterpenoids are natural products with known anti-inflammatory properties, and many have revealed efficiency against arthritis both in vitro and in vivo in several animal models, with negligible cytotoxicity. However, poor bioavailability due to low water solubility and extensive metabolism upon oral administration hinder the therapeutic use of anti-arthritic triterpenoids. Therefore, drug delivery

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36

Kim, Hwan Mook, Jong Soon Kang, Ki Hoon Lee, Chang Woo Lee, Kiho Lee, and Song-Kyu Park. "Anti-inflammatory and anti-arthritic effect of essential oil isolated from Curcuma wenyujin (35.7)." Journal of Immunology 184, no. 1_Supplement (2010): 35.7. http://dx.doi.org/10.4049/jimmunol.184.supp.35.7.

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Abstract Curcuma species has been reported to exert anti-inflammatory and anti-tumor effects. Curcuma wenyujin is mainly cultivated in wenzhou region of China and it was reported that the main constituents of Curcuma wenyujin is different from that of other Curcuma species. In this study, we investigated the effect of essential oil isolated from Curcuma wenyujin (CW) on inflammation and arthritis in rodents. CW dose-dependently suppressed lipopolysaccharide (LPS)-induced TNF-α production in mice. CW also inhibited zymosan-induced inflammation in concentration-dependent manner. Further studies

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Al-Rubaye, Mahmood R. "Anti-neutrophlic cytoplasmic antibody Elastase, Lactoferrin, Cathapsin G,and Lysozyme in a sample of Iraqi patients with Rheumatoid Arthritis." Journal of the Faculty of Medicine Baghdad 57, no. 1 (2015): 68–74. http://dx.doi.org/10.32007/jfacmedbagdad.571312.

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mucosal secretions as well as in secondary granules of polymorphonuclear leukocytes. Anti-Elastase antibodies, anti-Lactoferrin antibodies,anti-Cathapsin G antibodies and anti-Lysozyme antibodies, which belong to Perinuclear Anti-Neutrophil Cytoplasmic Antibodies(pANCA) have been described in several immunomediated diseases, including Rheumatoid Arthritis .Objectives: Investigate the prevalence of anti-Elastase antibodies, anti-Lactoferrin antibodies,anti-Cathapsin G antibodies , anti- Lysozyme antibodies and rheumatoid factor in patients with rheumatoid arthritis in comparison to healthy cont

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Sousa, Bárbara, Joana Mendes, and Ana Sofia Sousa. "Anti-inflammatory diet and rheumatoid arthritis: overview on the current evidence." Acta Portuguesa de Nutrição 34 (September 30, 2023): 40–44. http://dx.doi.org/10.21011/apn.2023.3408.

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INTRODUCTION: Rheumatoid arthritis is a chronic autoimmune disease, with symptoms that are characterized by inflammation of the joints and requires pharmacological treatment. However, pharmacological approach is not always enough to eliminate the symptoms related to rheumatoid arthritis, and nutritional intervention is necessary as a complementary treatment to obtain a significant improvement in symptoms. Several nutritional approaches have been studied to understand which diet would provide more benefits to patients with rheumatoid arthritis, and anti-inflammatory diet described as rich in nu

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Nasuti, Cinzia, Donatella Fedeli, Laura Bordoni, et al. "Anti-Inflammatory, Anti-Arthritic and Anti-Nociceptive Activities of Nigella sativa Oil in a Rat Model of Arthritis." Antioxidants 8, no. 9 (2019): 342. http://dx.doi.org/10.3390/antiox8090342.

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This study investigated the preventive efficacy of the crude oil extracted from Nigella sativa seeds in a rat model of arthritis induced by using complete Freund’s adjuvant (CFA). Nigella sativa oil at 1.82 mL/kg or 0.91 mL/kg (corresponding to 1596 and 798 mg/kg, respectively) was orally administered for 25 days from the day of immunization. One immunized group was treated orally with indomethacin (3 mg/kg) as a reference drug. Body weight growth rate, paw swelling, arthritis score, mechanical allodynia, locomotor activity and anxiety-like behavior were observed, and the levels of Interleukin

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Jezernik, Gregor, Mario Gorenjak, and Uroš Potočnik. "Gene Ontology Analysis Highlights Biological Processes Influencing Non-Response to Anti-TNF Therapy in Rheumatoid Arthritis." Biomedicines 10, no. 8 (2022): 1808. http://dx.doi.org/10.3390/biomedicines10081808.

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Anti-TNF therapy has significantly improved disease control in rheumatoid arthritis, but a fraction of rheumatoid arthritis patients do not respond to anti-TNF therapy or lose response over time. Moreover, the mechanisms underlying non-response to anti-TNF therapy remain largely unknown. To date, many single biomarkers of response to anti-TNF therapy have been published but they have not yet been analyzed as a system of interacting nodes. The aim of our study is to systematically elucidate the biological processes underlying non-response to anti-TNF therapy in rheumatoid arthritis using the ge

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Mammadova, U. "Using Secukinumabin Spondyloarthritis : a case report." REVMATOLOGİYA VƏ İLTİHAB JURNALI 1, no. 1 (2024): 1–3. https://doi.org/10.28942/jri.v1i1y2024.99.

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Spondyloarthritis is a progressive inflammatory disease that typically causes lower back pain and stiffness.Spondyloarthropathies (SpA) are a group of inflammatory arthritis which consist of ankylosing spondylitis (AS), reactive arthritis, arthritis/spondylitis associated with psoriasis (PsA), and arthritis/spondylitis associated with inflammatory bowel diseases.A strong link between human leukocyte antigen B27 and axSpA, however, has been identified, and the success of anti-tumour necrosis factor and anti-interleukin (IL)-17A therapy has highlighted some of the key pro- inflammatory cytokines

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Zhou, Qian, Tian Li, Gang Fang, Yuzhou Pang, and Xueni Wang. "Bioactive Molecules against Rheumatoid Arthritis by Suppressing Pyroptosis." Pharmaceuticals 16, no. 7 (2023): 952. http://dx.doi.org/10.3390/ph16070952.

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Rheumatoid arthritis is an inflammatory disease, and pyroptosis is a form of death associated with an inflammatory response. Pyroptosis, which occurs in synovial and osteoblastic cells, can exacerbate the development of rheumatoid arthritis. The inhibition of pyroptosis of these cells can, therefore, clearly be used as a therapeutic strategy against rheumatoid arthritis. Here, we have summarized the current status of progress in the treatment of rheumatoid arthritis by targeting cellular pyroptosis. We have identified seven compounds, including a cyclic RNA, a microRNA, a peptide, and a cytoki

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43

van den Beukel, Michelle D., Tineke J. van Wesemael, Anna Titia W. Hoogslag, et al. "Antibodies against advanced glycation end-products and malondialdehyde-acetaldehyde adducts identify a new specific subgroup of hitherto patients with seronegative arthritis with a distinct clinical phenotype and an HLA class II association." RMD Open 9, no. 4 (2023): e003480. http://dx.doi.org/10.1136/rmdopen-2023-003480.

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ObjectiveIn rheumatoid arthritis (RA) around two-thirds of patients are autoantibody positive for rheumatoid factor, anti-citrullinated protein antibodies and/or anti-carbamylated protein antibodies. The remaining seronegative subgroup of patients is clinically heterogeneous and thus far, biomarkers predicting the disease course are lacking. Therefore, we analysed the value of other autoantibodies in RA directed against malondialdehyde-acetaldehyde adducts (MAA) and advanced glycation end-products (AGE).MethodsIn sera of 648 patients with RA and 538 patients without RA from the Leiden Early Ar

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Oka, Shomi, Takashi Higuchi, Hiroshi Furukawa, et al. "False-positive detection of IgM anti-severe acute respiratory syndrome coronavirus 2 antibodies in patients with rheumatoid arthritis: Possible effects of IgM or IgG rheumatoid factors on immunochromatographic assay results." SAGE Open Medicine 10 (January 2022): 205031212210880. http://dx.doi.org/10.1177/20503121221088090.

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Objectives: The severe acute respiratory syndrome coronavirus 2 causes coronavirus disease 2019. A serological test is conducted to determine prior infection by severe acute respiratory syndrome coronavirus 2. We investigated whether the results of anti-severe acute respiratory syndrome coronavirus 2 antibody tests are modified in patients with rheumatoid arthritis. Methods: Patients in Japan with rheumatoid arthritis were recruited at Sagamihara Hospital from July 2014 to October 2015 ( n = 38; 2014 cohort) and at Tokyo Hospital from June to October 2020 ( n = 93; 2020 cohort). Anti-severe ac

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Kulkarni, Abhay P., and Himanshu A. Bendrey. "Systemic enzyme therapy with trypsin, bromelain and rutoside in the management of arthritis: an overview." International Journal of Research in Orthopaedics 7, no. 5 (2021): 1062. http://dx.doi.org/10.18203/issn.2455-4510.intjresorthop20213392.

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<p class="abstract">Chronic arthritis, including osteoarthritis and rheumatoid arthritis, is a growing major public health problem leading to disability and reduced quality of life. Analgesic and anti-inflammatory drugs form the mainstay of treatment for chronic arthritis. The protracted use of the conventional medications for their management is fraught with shortcomings, including safety concerns. Proteolytic enzymes and antioxidant combinations have been used empirically, since ages, in many of these conditions. There is a growing body of evidence indicating the beneficial effects exe

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Burchill, Matthew A., Dean T. Nardelli, Douglas M. England, et al. "Inhibition of Interleukin-17 Prevents the Development of Arthritis in Vaccinated Mice Challenged with Borrelia burgdorferi." Infection and Immunity 71, no. 6 (2003): 3437–42. http://dx.doi.org/10.1128/iai.71.6.3437-3442.2003.

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ABSTRACT We showed that Borrelia burgdorferi-vaccinated interferon gamma-deficient (IFN-γ0) mice challenged with the Lyme spirochete developed a prominent chronic severe destructive osteoarthropathy. The immune response underlying the development of the severe destructive arthritis involves interleukin-17 (IL-17). Treatment of vaccinated IFN-γ0 mice challenged with B. burgdorferi with anti-IL-17 antibody delayed the onset of swelling of the hind paws but, more importantly, inhibited the development of arthritis. Histopathologic examination confirmed that treatment with anti-IL-17 antibody prev

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Lin, Chun-Ching, Ming-Feng Chen, and Chieh-Fu Chen. "The Anti-inflammatory Effects of Chinese Crude Drug Prescriptions on Experimental Arthritis." American Journal of Chinese Medicine 23, no. 02 (1995): 145–52. http://dx.doi.org/10.1142/s0192415x95000195.

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San-Miao-Warn (SMW), Tuhwo-Jih-Shen-Tang (TJS) and Dang-Quei-Nian-Tong-Tang (DGT) are Chinese traditional prescriptions. In this study, we evaluated the anti-inflammatory activities of these crude drug prescriptions in carrageenan-induced acute arthritis and complete Freund's adjuvant (CFA)-induced chronic arthritis in rats. It was found that pretreatment with SMW, TJS or DGT at a dosage of 100 mg/kg or 300 mg/kg, significantly inhibited carrageenan-induced acute arthritis. Moreover, these crude drugs also significantly suppressed the development of chronic arthritis induced by CFA. These resu

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Papadopoulos, Theodore, Francois Santinon, Carolina Lopez Naranjo, Marie Hudson, and Sonia Victoria Del Rincon. "Abstract A089: Methotrexate preserves anti-PD1 anti-tumor benefits while reducing arthritic inflammation in a tumor and ICI-arthritis combination mouse model." Cancer Immunology Research 13, no. 2_Supplement (2025): A089. https://doi.org/10.1158/2326-6074.io2025-a089.

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Abstract Immune checkpoint inhibitors (ICIs) have revolutionized the treatment of cancer and it is now estimated that up to 50% of cancer patients are eligible for ICIs. ICIs are associated with high rates of immune toxicities known as immune related adverse events (irAEs) which in up to 7% of patients occur as ICI-arthritis. ICI-arthritis is empirically treated with steroids and drugs such as methotrexate (MTX), but evidence is mounting that steroids may diminish the survival benefit conferred by ICIs. With the increasing use of ICIs in cancer, it is critical to determine how immune suppressi

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Papadopoulos, Theodore, Francois Santinon, Carolina Lopez Naranjo, Marie Hudson, and Sonia Victoria Del Rincon. "Abstract A089: Methotrexate preserves anti-PD1 anti-tumor benefits while reducing arthritic inflammation in a tumor and ICI-arthritis combination mouse model." Cancer Immunology Research 13, no. 2_Supplement (2025): A0. https://doi.org/10.1158/2326-6074.io2025-a0.

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Abstract Immune checkpoint inhibitors (ICIs) have revolutionized the treatment of cancer and it is now estimated that up to 50% of cancer patients are eligible for ICIs. ICIs are associated with high rates of immune toxicities known as immune related adverse events (irAEs) which in up to 7% of patients occur as ICI-arthritis. ICI-arthritis is empirically treated with steroids and drugs such as methotrexate (MTX), but evidence is mounting that steroids may diminish the survival benefit conferred by ICIs. With the increasing use of ICIs in cancer, it is critical to determine how immune suppressi

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Austin, Publishing Group. "Synthesis and Biological Evaluation of Curcumin-NSAIDs Conjugates." Austin Journal of Analytical and Pharmaceutical Chemistry 10, no. 1 (2023): 1153. https://doi.org/10.26420/austinjanalpharmchem.2023.1153.

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Abstract The present research work involves the synthesis of a total of four Curcumin-NSAIDs conjugates and their subsequent biological screening for Rheumatoid Arthritis, Anti-inflammatory, and Anti-ulcer activities. Curcumin is a phytochemical having versatile therapeutic potential. The idea of conjugating curcumin with various NSAIDs is somewhat novel in the sense that it helps in obtaining the target of incorporating certain desired properties in the curcumin like enhanced bioavailability, reduction in the side effects and also giving good therapeutic potential in terms of determination of

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