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Journal articles on the topic 'Anti-Calbindin'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Pasteels, Brigitte, John Rogers, François Blachier, and Roland Pochet. "Calbindin and calretinin localization in retina from different species." Visual Neuroscience 5, no. 1 (1990): 1–16. http://dx.doi.org/10.1017/s0952523800000031.

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AbstractCalbindin-D28K and calretinin are homologous calcium-binding proteins localized in many neurons of the central nervous system. We have compared polyclonal antibodies against calbindin and calretinin and have shown by western blots using purified calbindin and calretinin from rat that (1) anti-calretinin does not recognize calbindin and (2) anti-calbindin presents some cross-reactivity with calretinin.In this report, we have compared by immunohistochemistry the localization of both calcium-binding proteins in the retina of monkey, pig, sheep, rat, cat, pigeon, and salamander. These resu

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2

Sourial, Samer, Maritha Marcusson-Ståhl, and Karin Cederbrant. "Meso Scale Discovery and Luminex Comparative Analysis of Calbindin D28K." Journal of Biomedicine and Biotechnology 2009 (2009): 1–5. http://dx.doi.org/10.1155/2009/187426.

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The sensitivity of different renal regions to xenobiotics requires the development of a multiplex immunoassay for the simultaneous analysis of kidney biomarkers. Calbindin D28K is a distal tubule-specific protein that can be detected in urine under pathological conditions. In this study, a pair of anti-calbindin D28K antibodies was used in an immunoassay for the detection of calbindin D28K expression in rat and human kidney and urine. Comparative analysis of the immunoassay was performed on the Meso Scale Development (MSD) and Luminex platforms. Analysis on both platforms detected calbindin D2

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3

Haley, Tammie L., Roland Pochet, Larry Baizer, et al. "Calbindin D-28K immunoreactivity of human cone cells varies with retinal position." Visual Neuroscience 12, no. 2 (1995): 301–7. http://dx.doi.org/10.1017/s0952523800007987.

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AbstractCalbindin D-28K is a calcium-binding protein found in the cone but not rod photoreceptor cells in the retinas of a variety of species. Recent studies of the monkey retina indicated that calbindin D-28K may be expressed preferentially in non-foveal regions of the retina. In the current studies of human retinas, immunohistochemical experiments demonstrated that calbindin D-28K is reduced or absent in the fovea and parafovea, but prevalent in the perifovea and periphery. These findings were supported by the quantification of calbindin D-28K in 1-mm trephine punches obtained from different

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4

Ferrer, Isidro, Fabián Isamat, Luis López-Obarrio, et al. "Parvalbumin and calbindin D-28K immunoreactivity in central ganglioglioma and dysplastic gangliocytoma of the cerebellum." Journal of Neurosurgery 78, no. 1 (1993): 133–37. http://dx.doi.org/10.3171/jns.1993.78.1.0133.

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✓ Calbindin D-28K and parvalbumin immunocytochemistry were used in the study of central ganglionic cell tumors. Most neurons in the ganglioglioma were immunoreactive to calbindin D-28K, but a few cells were labeled with antibodies against parvalbumin. In contrast, most cells in dysplastic gangliocytoma of the cerebellum were parvalbumin immunoreactive, but fewer reacted with anti-calbindin antibodies. These latter cells had two or three dendrites with claw-shaped terminals and axons with recurrent collateral branches and varicose terminals filled with strings and buttons. These observations su

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RIZZO, MARIA, GIOVAMBATTISTA CAPASSO, MARKUS BLEICH, et al. "Effect of Chronic Metabolic Acidosis on Calbindin Expression along the Rat Distal Tubule." Journal of the American Society of Nephrology 11, no. 2 (2000): 203–10. http://dx.doi.org/10.1681/asn.v112203.

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Calbindin D28k has been reported to be involved in the transcellular calcium transport along the rat distal tubule. It has also been shown that chronic metabolic acidosis (CMA) induces significant hypercalciuria. The present study investigated whether CMA affects the mRNA and the protein expression of calbindin D28k along isolated distal tubule (DT) of rats. The animals were made acidotic by adding 0.28 mol/L NH4Cl to the drinking water for 7 d. This maneuver was associated with an increase in plasma ionized calcium. Inulin clearance experiments demonstrated that metabolic acidosis did not aff

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Merico, Valeria, Gabriela Diaz de Barboza, Chiara Vasco, et al. "A mitochondrial mechanism is involved in apoptosis of Robertsonian mouse male germ cells." REPRODUCTION 135, no. 6 (2008): 797–804. http://dx.doi.org/10.1530/rep-07-0466.

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The aim of this study was to determine whether the intrinsic mechanism of apoptosis is involved in the death of germ cells in Robertsonian (Rb) heterozygous adult male mice. Testes from 5-month-old Rb heterozygous CD1×Milano II mice were obtained and compared with those from homozygous CD1 (2n=40) and Milano II (2n=24) mice. For histological evaluation of apoptosis, TUNEL labelling and immunohistochemistry were used to localise Bax and cytochrome c. Expression of calbindin D28k (CB), an anti-apoptotic molecule, was also analysed by immunohistochemistry and immunoblotting. Testicular ultrastruc

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7

Rodriguez, Valeria, Gabriela Diaz de Barboza, Ruben Ponce, Valeria Merico, Silvia Garagna, and Nori Tolosa de Talamoni. "Spermatocyte apoptosis, which involves both intrinsic and extrinsic pathways, explains the sterility of Graomys griseoflavus × Graomys centralis male hybrids." Reproduction, Fertility and Development 22, no. 2 (2010): 478. http://dx.doi.org/10.1071/rd09106.

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Spermatogenic impairment and the apoptotic pathways involved in establishing sterility of male hybrids obtained from crossing Graomys griseoflavus females with Graomys centralis males were studied. Testes from G. centralis, G. griseoflavus and hybrids were compared at different ages. Terminal transferase-mediated dUTP nick-end labelling assay (TUNEL), Fas, Bax and cytochrome c labelling were used for apoptosis evaluation, and calbindin D28k staining as an anti-apoptotic molecule. In 1-month-old animals, spermatocytes were positive for all apoptotic markers, but moderate TUNEL (+) spermatocyte

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8

de Barry, J., and G. Gombos. "Immunohistochemistry with anti-calbindin and anti-neurofilament antibodies in the cerebellum of methylazoxymethanol-treated mice." Journal of Neuroscience Research 23, no. 3 (1989): 330–36. http://dx.doi.org/10.1002/jnr.490230313.

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9

Huybers, Sylvie, Ton H. J. Naber, René J. M. Bindels, and Joost G. J. Hoenderop. "Prednisolone-induced Ca2+ malabsorption is caused by diminished expression of the epithelial Ca2+ channel TRPV6." American Journal of Physiology-Gastrointestinal and Liver Physiology 292, no. 1 (2007): G92—G97. http://dx.doi.org/10.1152/ajpgi.00317.2006.

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Glucocorticoids, such as prednisolone, are often used in clinic because of their anti-inflammatory and immunosuppressive properties. However, glucocorticoids reduce bone mineral density (BMD) as a side effect. Malabsorption of Ca2+ in the intestine is supposed to play an important role in the etiology of low BMD. To elucidate the mechanism of glucocorticoid-induced Ca2+ malabsorption, the present study investigated the effect of prednisolone on the expression and activity of proteins responsible for active intestinal Ca2+ absorption including the epithelial Ca2+ channel TRPV6, calbindin-D9K, a

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10

Milam, Ann H., Dennis M. Dacey, and Alexander M. Dizhoor. "Recoverin immunoreactivity in mammalian cone bipolar cells." Visual Neuroscience 10, no. 1 (1993): 1–12. http://dx.doi.org/10.1017/s0952523800003175.

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AbstractHuman, macaque monkey, and rat retinas were immunostained with a polyclonal antibody preparation against purified recoverin, a 23-kD calcium-binding protein isolated from bovine retina that localizes to rods and cones (Dizhoor et al., 1991). In addition to immunoreactive photoreceptors, we have identified subpopulations of recoverin-positive bipolar cells in all three species. Results from immunostaining with progressive dilutions of anti-recoverin and preadsorption of the antibody with a dilution series of purified recoverin showed that photoreceptors and bipolar cells had similar aff

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11

Ji, Youn-Kyu, Geun-Shik Lee, Kyung-Chul Choi, and Eui-Bae Jeung. "Anti-progestogenic effect of flutamide on uterine expression of calbindin-D9k mRNA and protein in immature mice." Reproductive Toxicology 22, no. 4 (2006): 694–701. http://dx.doi.org/10.1016/j.reprotox.2006.04.015.

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12

Kumar, Deepak, Aditi Sharma, and Lalit Sharma. "A Comprehensive Review of Alzheimer’s Association with Related Proteins: Pathological Role and Therapeutic Significance." Current Neuropharmacology 18, no. 8 (2020): 674–95. http://dx.doi.org/10.2174/1570159x18666200203101828.

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: Alzheimer’s is an insidious, progressive, chronic neurodegenerative disease which causes the devastation of neurons. Alzheimer's possesses complex pathologies of heterogeneous nature counting proteins as one major factor along with enzymes and mutated genes. Proteins such as amyloid precursor protein (APP), apolipoprotein E (ApoE), presenilin, mortalin, calbindin-D28K, creactive protein, heat shock proteins (HSPs), and prion protein are some of the chief elements in the foremost hypotheses of AD like amyloid-beta (Aβ) cascade hypothesis, tau hypothesis, cholinergic neuron damage, etc. Distur

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13

Zacharko-Siembida, A., J. L. Valverde Piedra, and M. B. Arciszewski. "Changes in expression of calbindin 28 kDa in the small intestine of red kidney bean (Phaseolus vulgaris) lectin-treated suckling piglets." Polish Journal of Veterinary Sciences 16, no. 2 (2013): 201–9. http://dx.doi.org/10.2478/pjvs-2013-0029.

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Abstract Calbindin (CB) is a calcium binding protein playing a role in calcium uptake and anti-apoptotic cellular protection. The expression of CB was immunohistochemically studied in the small intestine of normal and red bean kidney lectin-treated suckling piglets. In the duodenum and jejunum (but not ileum) of lectin-treated animals overexpression of CB was noted in chromogranin A-immunoreactive (CgA-IR) neuroendocrine (NE) cells. In both control and experimental group a small population of CB-IR NE cells exhibited the presence of somatostatin (but not serotonin, histamine or CRF). After the

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14

D’Alessio, Luciana, Hector Konopka, Patricia Solís, et al. "Depression and Temporal Lobe Epilepsy: Expression Pattern of Calbindin Immunoreactivity in Hippocampal Dentate Gyrus of Patients Who Underwent Epilepsy Surgery with and without Comorbid Depression." Behavioural Neurology 2019 (May 2, 2019): 1–12. http://dx.doi.org/10.1155/2019/7396793.

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Purpose. Changes in calbindin (CB) expression have been reported in patients with temporal lobe epilepsy (TLE) with controversial implications on hippocampal functions. The aim of this study was to determine the CB immunoreactivity in hippocampal dentate gyrus of patients who underwent epilepsy surgery for drug-resistant TLE with and without comorbid depression and/or memory deficits. Methods. Selected hippocampal samples from patients with TLE who underwent epilepsy surgery were included. Clinical and complementary assessment: EEG, video-EEG, MRI, psychiatric assessment (structured clinical i

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15

Elms, T. N., and A. N. Taylor. "Calbindin-D 28K Localization in Rat Molars During Odontogenesis." Journal of Dental Research 66, no. 9 (1987): 1431–34. http://dx.doi.org/10.1177/00220345870660090401.

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Specific antiserum raised against Calbindin-D28K, a vitamin D-dependent calcium-binding protein (CaBP) isolated from chick intestine, was used for localization of the protein in developing rat molars. Previously, CaBP had been localized in specific cells associated with the continuously erupting rat incisor: late pre-secretory ameloblasts, secretory and maturation zone ameloblasts, stratum intermedium cells adjacent to ameloblasts in the late zone of enamel secretion, and papillary cells underlying maturation zone ameloblasts. In this study, the peroxidase anti-peroxidase technique was used fo

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16

Wang, Guo-Du, Xi-Yu Wang, Hong-Zhen Hu, et al. "Platelet-activating factor in the enteric nervous system of the guinea pig small intestine." American Journal of Physiology-Gastrointestinal and Liver Physiology 291, no. 5 (2006): G928—G937. http://dx.doi.org/10.1152/ajpgi.00153.2006.

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Platelet-activating factor (PAF) is a proinflammatory mediator that may influence neuronal activity in the enteric nervous system (ENS). Electrophysiology, immunofluorescence, Western blot analysis, and RT-PCR were used to study the action of PAF and the expression of PAF receptor (PAFR) in the ENS. PAFR immunoreactivity (IR) was expressed by 6.9% of the neurons in the myenteric plexus and 14.5% of the neurons in the submucosal plexus in all segments of the guinea pig intestinal tract as determined by double staining with anti-human neuronal protein antibody. PAFR IR was found in 6.1% of the n

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17

Wittmann, Walter, and Ian S. McLennan. "Anti-Müllerian hormone may regulate the number of calbindin-positive neurons in the sexually dimorphic nucleus of the preoptic area of male mice." Biology of Sex Differences 4, no. 1 (2013): 18. http://dx.doi.org/10.1186/2042-6410-4-18.

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18

Oyagbemi, Ademola A., Olamide E. Adebiyi, Kabirat O. Adigun та ін. "Clofibrate, a PPAR‐α agonist, abrogates sodium fluoride‐induced neuroinflammation, oxidative stress, and motor incoordination via modulation of GFAP/Iba‐1/anti‐calbindin signaling pathways". Environmental Toxicology 35, № 2 (2019): 242–53. http://dx.doi.org/10.1002/tox.22861.

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19

Nam, Sung Min, Sun-Hye Choi, Hee-Jung Cho, et al. "Ginseng Gintonin Attenuates Lead-Induced Rat Cerebellar Impairments during Gestation and Lactation." Biomolecules 10, no. 3 (2020): 385. http://dx.doi.org/10.3390/biom10030385.

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Gintonin, a novel ginseng-derived lysophosphatidic acid receptor ligand, improves brain functions and protects neurons from oxidative stress. However, little is known about the effects of gintonin against Pb-induced brain maldevelopment. We investigated the protective effects of gintonin on the developing cerebellum after prenatal and postnatal Pb exposure. Pregnant female rats were randomly divided into three groups: control, Pb (0.3% Pb acetate in drinking water), and Pb plus gintonin (100 mg/kg, p.o.). Blood Pb was increased in dams and pups; gintonin treatment significantly decreased blood

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20

Lee, Myung-Jun, Won-Tae Lee, and Chang-Jin Jeon. "Organization of Neuropeptide Y-Immunoreactive Cells in the Mongolian gerbil (Meriones unguiculatus) Visual Cortex." Cells 10, no. 2 (2021): 311. http://dx.doi.org/10.3390/cells10020311.

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Neuropeptide Y (NPY) is found throughout the central nervous system where it appears to be involved in the regulation of a wide range of physiological effects. The Mongolian gerbil, a member of the rodent family Muridae, is a diurnal animal and has been widely used in various aspects of biomedical research. This study was conducted to investigate the organization of NPY-immunoreactive (IR) neurons in the gerbil visual cortex using NPY immunocytochemistry. The highest density of NPY-IR neurons was located in layer V (50.58%). The major type of NPY-IR neuron was a multipolar round/oval cell type

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21

Lee, Geun-Shik, Kyung-Chul Choi, and Eui-Bae Jeung. "Glucocorticoids differentially regulate expression of duodenal and renal calbindin-D9k through glucocorticoid receptor-mediated pathway in mouse model." American Journal of Physiology-Endocrinology and Metabolism 290, no. 2 (2006): E299—E307. http://dx.doi.org/10.1152/ajpendo.00232.2005.

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Dexmethasone (Dex) is a member of the glucocorticoids (GCs), and is broadly used as an anti-inflammatory medication. Continuous administration with GCs induces adverse effects and suffering in humans (i.e., osteoporosis) due to negative calcium balance derived from low re- and absorption in the duodenum and kidney. A cytosolic calcium-binding protein, calbindin-D9k (CaBP-9k), is dominantly expressed in the renal and intestinal tissues involved in calcium re- and absorption and plays an active role in calcium transport. In the present study, we employed adrenalectomized (ADX) and sham-treated (

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22

DE LIMA, S. M. A., P. K. AHNELT, T. O. CARVALHO, et al. "Horizontal cells in the retina of a diurnal rodent, the agouti (Dasyprocta aguti)." Visual Neuroscience 22, no. 6 (2005): 707–20. http://dx.doi.org/10.1017/s0952523805226032.

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The morphology and distribution of normally placed and displaced A horizontal cells were studied in the retina of a diurnal hystricomorph rodent, the agouti Dasyprocta aguti. Cells were labeled with anti-calbindin immunocytochemistry. Dendritic-field size reaches a minimum in the visual streak, of about 9000 μm2, and increases toward the retinal periphery both in the dorsal and ventral regions. There is a dorsoventral asymmetry, with dorsal cells being larger than ventral cells at equal distances from the streak. The peak value for cell density of 281 ± 28 cells/mm2 occurs in the center of the

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Liu, Cuiping, Guoli Shao, Yirong Lu та ін. "Parathyroid Hormone-Related Protein (1-40) Enhances Calcium Uptake in Rat Enterocytes Through PTHR1 Receptor and Protein Kinase Cα/β Signaling". Cellular Physiology and Biochemistry 51, № 4 (2018): 1695–709. http://dx.doi.org/10.1159/000495674.

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Background/Aims: Parathyroid hormone-related protein (PTHrP) is implicated in regulating calcium homeostasis in vertebrates, including sea bream, chick, and mammals. However, the molecular mechanism underlying the function of PTHrP in regulating calcium transport is still not fully investigated. This study aimed to investigate the effect of PTHrP on the calcium uptake and its underlying molecular mechanism in rat enterocytes. Methods: The rat intestinal epithelial cell line (IEC-6) was used. Calcium uptake was determined by using the fluo-4 acetoxymethyl ester fluorescence method. The expressi

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OHTSUKA, Aiji, Takehito TAGUCHI, Ramadan SAYED, and Takuro MURAKAMI. "The Spatial Relationship between the Perineuronal Proteoglycan Network and the Synaptic Boutons as Visualized by Double Staining with Cationic Colloidal Iron Method and Anti-Calbindin-D-28K Immunohistochemistry in Rat Cerebellar Nuclei." Archives of Histology and Cytology 63, no. 4 (2000): 313–18. http://dx.doi.org/10.1679/aohc.63.313.

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25

R Butler, Logan. "Computational Design of Cyclic Peptide Inhibitors of the Anti-Apoptotic Protein Calbindin-D28K." Biochemistry & Pharmacology: Open Access 03, no. 04 (2014). http://dx.doi.org/10.4172/2167-0501.1000142.

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26

Seele, Jana, Melissa Ballüer, Simone C. Tauber, et al. "Neural Injury and Repair in a Novel Neonatal Mouse Model of Listeria Monocytogenes Meningoencephalitis." Journal of Neuropathology & Experimental Neurology, September 6, 2021. http://dx.doi.org/10.1093/jnen/nlab079.

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Abstract To improve the therapy of neonatal central nervous system infections, well-characterized animal models are urgently needed. The present study analyzes neuropathological alterations with particular focus on neural injury and repair in brains of neonatal mice with Listeria monocytogenes (LM) meningitis/meningoencephalitis using a novel nasal infection model. The hippocampal formation and frontal cortex of 14 neonatal mice with LM meningitis/meningoencephalitis and 14 uninfected controls were analyzed by histology, immunohistochemistry, and in situ tailing for morphological alterations.

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27

Keilhoff, Gerburg, Maximilian Titze, and Uwe Ebmeyer. "Immuno-histological detection of resistant columnar units and vulnerable networks in the rat retina after asphyxia-induced transient cardiac arrest." Restorative Neurology and Neuroscience, July 28, 2021, 1–23. http://dx.doi.org/10.3233/rnn-211174.

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Background: Stroke-related loss of vision is one of the residual impairments, restricting the quality of life. However, studies of the ocular manifestations of asphyxia cardiac arrest/resuscitation (ACA/R) have reported very heterogeneous results. Objective: We aimed to evaluate the ACA/R-induced degeneration pattern of the different retinal cell populations in rats using different immuno-histological stainings. Methods: The staining pattern of toluidine blue and the ganglion cell markers β-III-tubulin and NeuN; the calcium-binding protein parvalbumin, indicating ganglion, amacrine, and horizo

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28

Quillinan, Nidia, Kaori Shimizu, Guiying Deng, Richard Traystman, and Paco Herson. "Abstract W P94: Recovery From Plasticity Impairments in Pediatric But Not Adult Mice Following Cardiac Arrest and Cardiopulmonary Resuscitation." Stroke 46, suppl_1 (2015). http://dx.doi.org/10.1161/str.46.suppl_1.wp94.

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Objectives: The goal of this study was to assess neuronal injury and synaptic plasticity impairments in Purkinje cells in pediatric and adult mice following cardiac arrest and cardiopulmonary resuscitation. We hypothesized that synaptic impairments in pediatrics would be less severe than in adult mice. Methods: Pediatric ( 21-25 day old) and adult (8-12 week) male mice were subjected to 8 minutes cardiac arrest followed by cardiopulmonary resuscitation or sham surgery. Purkinje cells were labeled using anti-calbindin antibody and cell density was analyzed to examine neuronal injury. To examine

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