Relevant bibliographies by topics / Anti-IL-17

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Anti-IL-17'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Anti-IL-17"

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Ly, Karen, Mary P. Smith, Quinn Thibodeaux, Vidhatha Reddy, Wilson Liao, and Tina Bhutani. "Anti IL-17 in psoriasis." Expert Review of Clinical Immunology 15, no. 11 (2019): 1185–94. http://dx.doi.org/10.1080/1744666x.2020.1679625.

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Buckland, Jenny. "Anti-TNF and anti-IL-17 antibodies—better together!" Nature Reviews Rheumatology 10, no. 12 (2014): 699. http://dx.doi.org/10.1038/nrrheum.2014.183.

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Daude, M., and S. Barbarot. "Maladie de Darier et anti IL 17." Annales de Dermatologie et de Vénéréologie - FMC 3, no. 8 (2023): A282. http://dx.doi.org/10.1016/j.fander.2023.09.448.

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Ndongo-Thiam, Ndieme, Alice Clement, Jean-Jacques Pin, Diane Razanajaona-Doll, and Pierre Miossec. "Negative association between autoantibodies against IL-17, IL-17/anti-IL-17 antibody immune complexes and destruction in rheumatoid arthritis." Annals of the Rheumatic Diseases 75, no. 7 (2016): 1420–22. http://dx.doi.org/10.1136/annrheumdis-2016-209149.

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Sales-Campos, Helioswilton, Chamberttan Souza Desidério, Rafael Obata Trevisan, et al. "Anti-IL-4, Anti-IL-17, and Anti-IFN-Gamma Activity in the Saliva of Amblyomma sculptum Ticks." International Journal of Molecular Sciences 26, no. 10 (2025): 4734. https://doi.org/10.3390/ijms26104734.

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The saliva of hematophagous arthropods, such as ticks and triatomines, contains bioactive ligands capable of modulating immune molecules, including cytokines. Cytokines play a critical role in immune regulation and have therapeutic relevance in inflammatory and immune-mediated diseases. Despite recent advances, identifying cytokine-binding molecules remains a significant challenge. Interferon-gamma (IFN-γ), interleukin-4 (IL-4), and interleukin-17 (IL-17) are key cytokines involved in inflammation, adaptive immunity, and host defense. This study evaluated the ability of salivary components fro
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Selimov, Pavel, Rositsa Karalilova, Ljubinka Damjanovska, et al. "Rheumatoid arthritis and the proinflammatory cytokine IL-17." Folia Medica 65, no. 1 (2023): 53–59. http://dx.doi.org/10.3897/folmed.65.e72448.

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Introduction: Rheumatoid arthritis (RA) is the most common inflammatory joint disease. Various proinflammatory cytokines are involved in the pathogenesis of this chronic disorder. It is characterized by the presence of autoantibodies, such as rheumatoid factor and antibodies against citrullinated peptides. The present study focuses on investigation of possible association between the proinflammatory cytokine interleukin 17 and anti-CCP, anti-MCV, and anti-CarP antibodies seropositivity in RA patients. Aim: To assess serum levels of interleukin 17 (IL-17) in patients with rheumatoid arthritis a
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Burchill, Matthew A., Dean T. Nardelli, Douglas M. England, et al. "Inhibition of Interleukin-17 Prevents the Development of Arthritis in Vaccinated Mice Challenged with Borrelia burgdorferi." Infection and Immunity 71, no. 6 (2003): 3437–42. http://dx.doi.org/10.1128/iai.71.6.3437-3442.2003.

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ABSTRACT We showed that Borrelia burgdorferi-vaccinated interferon gamma-deficient (IFN-γ0) mice challenged with the Lyme spirochete developed a prominent chronic severe destructive osteoarthropathy. The immune response underlying the development of the severe destructive arthritis involves interleukin-17 (IL-17). Treatment of vaccinated IFN-γ0 mice challenged with B. burgdorferi with anti-IL-17 antibody delayed the onset of swelling of the hind paws but, more importantly, inhibited the development of arthritis. Histopathologic examination confirmed that treatment with anti-IL-17 antibody prev
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Maniati, Eleni, and Thorsten Hagemann. "IL-17 mediates resistance to anti-VEGF therapy." Nature Medicine 19, no. 9 (2013): 1092–94. http://dx.doi.org/10.1038/nm.3333.

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Lux, Sebastian. "Psoriasis: Anti-IL-17-Inhibitor als Rescue-Therapie?" hautnah dermatologie 35, no. 2 (2019): 30. http://dx.doi.org/10.1007/s15012-019-3036-2.

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Cooper, Andrea M. "IL-17 and anti-bacterial immunity: Protectionversustissue damage." European Journal of Immunology 39, no. 3 (2009): 649–52. http://dx.doi.org/10.1002/eji.200839090.

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Dissertations / Theses on the topic "Anti-IL-17"

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Milanez, Fernanda Manente. "O uso de medicações anti-TNF não influencia o eixo IL-23/IL-17 em pacientes com espondilite anquilosante." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/5/5164/tde-23082017-111419/.

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Introdução: Apesar dos recentes avanços no entendimento da fisiopatologia e no tratamento da espondilite anquilosante (EA), pouco se sabe acerca da influência das medicações anti-fator de necrose tumoral (anti-TNF) sobre as novas vias inflamatórias descritas na patogênese das espondiloartrites. Objetivo: Dessa forma, o objetivo desse estudo é investigar e descrever a influência a longo prazo das medicações anti-TNF sobre o eixo da IL-23/IL-17 em pacientes com EA e sua possível correlação com o tratamento, parâmetros clínicos, laboratoriais e radiológicos. Métodos: Oitenta e seis pacientes com
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Cruz, da Silva Juliana. "Síntese e Avaliação Biológica de Novos Derivados 5-Benzilideno-3-Benzil- Tiazolidina-2,4-diona: Futuros Candidatos a Fármacos na Terapêutica do Tratamento de Doenças Autoimunes; como Lúpus Eritematoso Sistêmico e Artrite Reumatoide." Universidade Federal de Pernambuco, 2012. https://repositorio.ufpe.br/handle/123456789/2262.

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Made available in DSpace on 2014-06-12T15:55:53Z (GMT). No. of bitstreams: 2 arquivo9638_1.pdf: 1415158 bytes, checksum: 99d72178b3b8199614795e307f81954f (MD5) license.txt: 1748 bytes, checksum: 8a4605be74aa9ea9d79846c1fba20a33 (MD5) Previous issue date: 2012<br>Entre os fármacos que são utilizados na terapêutica do lúpus eritematoso sistêmico (LES) e artrite reumatoide (AR) destacam-se os anti-maláricos como a cloroquina (CQ) e hidroxicloroquina (HCQ) e os glicocorticoides que exercem graves efeitos colaterais. Entre os efeitos biológicos da CQ e da HCQ que são descritos na literatura
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Silva, Juliana Cruz da. "Síntese e avaliação biológica de novos derivados 5-benzilideno-3- benzil-tiazolidina-2,4-diona: futuros candidatos a fármacos na terapêutica do tratamento de doenças autoimunes, como lúpus eritematoso sistêmico e artrite reumatoide." Universidade Federal de Pernambuco, 2012. https://repositorio.ufpe.br/handle/123456789/12740.

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Submitted by Amanda Silva (amanda.osilva2@ufpe.br) on 2015-04-08T13:58:16Z No. of bitstreams: 2 TESE_completa_em_PDF.pdf: 1415623 bytes, checksum: 2b87aa820a2d8db28f2ec936e6a6d764 (MD5) license_rdf: 1232 bytes, checksum: 66e71c371cc565284e70f40736c94386 (MD5)<br>Made available in DSpace on 2015-04-08T13:58:17Z (GMT). No. of bitstreams: 2 TESE_completa_em_PDF.pdf: 1415623 bytes, checksum: 2b87aa820a2d8db28f2ec936e6a6d764 (MD5) license_rdf: 1232 bytes, checksum: 66e71c371cc565284e70f40736c94386 (MD5) Previous issue date: 2012-02-27<br>FACEPE<br>Entre os fármacos que são utilizados na ter
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Chraa, Dounia. "Analyse du rôle de l’IL-17 dans la progression du cancer du sein chez différents sous-groupes moléculaires de patientes : effet sur l’expression de PD-L1, sur la prolifération des cellules cancéreuses et sur la survie des patientes." Thesis, Aix-Marseille, 2019. http://www.theses.fr/2019AIXM0629.

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Dans le cancer du sein, de nombreux types de cellules immunitaires se trouvent au sein du microenvironnement tumoral. Des effets immunitaires pro et anti-tumoraux ont été rapportés. Dans la présente étude, nous démontrons que l'IL-17, une cytokine pro-inflammatoire, principalement produite par les cellules Th17, est fortement associée à des marqueurs de mauvais pronostic chez les patientes atteintes d'un cancer du sein. L'IL-17 était fortement exprimée chez les patients ER (-) y compris le sous-groupe triple négatif. De plus, l'expression de l'IL-17 au niveau de la tumeur était étroitement cor
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Machado, Joana Hygino da Silva. "Influência das alterações imunes em gestantes infectadas pelo HIV-1 no perfil funcional das células T de neonatos não infectados." Universidade do Estado do Rio de Janeiro, 2013. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=7981.

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Coordenação de Aperfeiçoamento de Pessoal de Nível Superior<br>O número de mulheres jovens infectadas pelo HIV-1 vem crescendo desde o início da epidemia da aids, principalmente nos países em desenvolvimento, onde a frequência de gravidez é elevada. O desenvolvimento de estratégias para evitar a transmissão vertical tem aumentado o número de recém-nascidos não infectados nascidos de gestantes portadora do vírus. O objetivo da presente tese foi avaliar, in vivo e in vitro, o perfil de citocinas de neonatos não infectados, porém nascidos de mulheres infectadas pelo HIV-1. Os resultados demonstra
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Camargo, Leandro do Nascimento. "O efeito do anti-IL-17 na inflama~ção, remodelamento e estresse oxidativo em modelo experimental de inflamação pulmonar alérgica crônica exacerbado por LPS." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/5/5165/tde-15082018-082634/.

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INTRODUÇÃO: A inflamação desempenha um papel central no desenvolvimento da asma, que é considerada uma doença alérgica com um perfil inflamatório clássico Th2. No entanto, as citocinas de perfil IL-17 tem sido estudadas para melhor compreender sua participação na fisiopatologia desta doença. Pacientes asmáticos graves apresentam exacerbações freqüentes, sendo a causa infecciosa um dos principais desencadeantes e que podem perpetuar as respostas inflamatórias. A resposta Th17 pode estar claramente associada a esses eventos. OBJETIVO: Este estudo avaliou os efeitos da terapia com anticorpo monoc
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Benchetrit, Fabrice. "Utilisation d'un vecteur non réplicatif, la sous-unité B de la toxine de Shiga, et d'une cytokine, l'IL-17, pour l'induction de lymphocytes T-CD8 cytotoxiques à activité anti-tumorale." Paris 6, 2003. http://www.theses.fr/2003PA066020.

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Guillot, Xavier. "Effets thérapeutiques et anti-inflammatoires de la cryothérapie dans les rhumatismes inflammatoires." Thesis, Besançon, 2016. http://www.theses.fr/2016BESA3009/document.

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La cryothérapie est utilisée de manière large et empirique à visée adjuvante dans les rhumatismes inflammatoires, avec un niveau de preuve faible. Dans une revue systématique de la littérature, en poolant les données de 6 études non contrôlées, nous avons pu démontrer que la cryothérapie (locale ou corps entier) appliquée deux fois par jour pendant 7 à 15 jours réduisait significativement l'EVA douleur et le score d'activité DAS25 dans la polyarthrite rhumatoïde. La cryothérapie locale (glace ou gaz froide) montrait par ailleurs des effets taille intra-classes supérieurs à ceux obtenus en util
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Aymeric, Laetitia. "Immunogénicite de la mort cellulaire induite par les chimiothérapies anti-cancéreuses." Phd thesis, Université Paris Sud - Paris XI, 2011. http://tel.archives-ouvertes.fr/tel-00634183.

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Le développement d'un cancer chez un individu immunocompétent témoigne del'inefficacité de ses défenses immunitaires naturelles à entraver la progression tumorale. Larestauration ou l'induction de réponses immunitaires anti-tumorales efficaces sont des enjeuxmajeurs des stratégies thérapeutiques actuelles. En plus des immunothérapies classiques,visant à activer le système immunitaire des patients contre leurs tumeurs, les thérapiesconventionnelles, telles que les chimiothérapies et la radiothérapie, peuvent avoir des effetsimmunostimulants, en parallèle de leur effet directement cytotoxique su
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Yuting, Ma. "The crosstalk between dying tumor cells and immune effectors within tumor microenvironment elicited by anti-cancer therapies dictates the therapeutic outcome." Thesis, Paris 11, 2011. http://www.theses.fr/2011PA11T033/document.

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En dehors des effets cytostatiques ou cytotoxiques sur les cellules tumorales, certaines thérapies anti-cancéreuses peuvent déclencher la mort cellulaire immunogénique, libérant les signaux de danger pour alerter le système immunitaire. Les cellules T CD8+ T (Tc1) productrices d’IFN- et spécifiques de la tumeur sont nécessaires pour le succès de la chimiothérapie et la diminution de la croissance tumorale. L’amorçage d’une réponse bénéfique Tc1 dépend de la sécrétion d'IL-1β par les cellules dendritiques confrontées à des cellules tumorales traitées avec de l’anthracycline libérant de l’ATP.
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Books on the topic "Anti-IL-17"

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Gaston, J. S. Hill. Reactive arthritis and enteropathic arthropathy. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0115.

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Reactive arthritis (ReA), and enteropathic arthritis secondary to inflammatory bowel disease, are forms of spondyloarthritis, all of which share an association with HLA B27 and can involve both axial and peripheral joints. Genetic studies strongly implicate the cytokines IL-17 and IL-23 in their pathogenesis, and evidence for autoimmunity is lacking. ReA is triggered by particular bacteria, mainly affecting the gut and genitourinary tract, though infections are sometimes asymptomatic. Classically an acute oligo- or monoarthritis with enthesitis occurs, often with inflammatory back pain, though
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Gaston, J. S. Hill. Reactive arthritis and enteropathic arthropathy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0115_update_002.

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Reactive arthritis (ReA), and enteropathic arthritis secondary to inflammatory bowel disease, are forms of spondyloarthritis, all of which share an association with HLA B27 and can involve both axial and peripheral joints. Genetic studies strongly implicate the cytokines IL-17 and IL-23 in their pathogenesis, and evidence for autoimmunity is lacking. ReA is triggered by particular bacteria, mainly affecting the gut and genitourinary tract, though infections are sometimes asymptomatic. Classically an acute oligo- or monoarthritis with enthesitis occurs, often with inflammatory back pain, though
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Braun, Juergen, and Irene E. van der Horst-Bruinsma. Treatment: biologics. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0022.

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According to classification criteria, the spectrum of spondyloarthritis (SpA) covers axial SpA (axSpA), which includes non-radiographic axSpA and ankylosing spondylitis, and peripheral SpA, which overlaps with psoriatic arthritis. Management recommendations for many forms of SpA have been recently published. Treatment of patients with axSpA with active disease starts with a sufficient dose of non-steroidal anti-inflammatory drugs (NSAIDs) for at least 4 weeks and preferably longer, in combination with exercise. In case of peripheral SpA, several disease-modifying antirheumatic drugs can be giv
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Book chapters on the topic "Anti-IL-17"

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Evans Adunyah, Samuel, Richard Akomeah, Fareed K.N. Arthur, Roland S. Cooper, and Joshua C.M. Williams. "IL-17 Biological Effects and Signaling Mechanisms in Human Leukemia U937 Cells." In Interleukins - The Immune and Non-Immune Systems’ Related Cytokines. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96422.

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Human Interlekin-17 is produced by memory activated CD4+ T cells and other cells. It was initially considered unique in that its specific receptor is distinct from other cytokine receptors. IL-17 receptor is ubiquitously expressed by different cells including T cells. IL-17 plays a role in regulating growth, immune response and pro-inflammatory responses. It regulates differentiation of a subset of Th0 cells into Th-17 cells, which produce IL-17-induced cytokines. The IL-17R belongs to type 1 cytokine receptors. IL-17 belongs to a superfamily of its own, which includes IL-17A, IL-17B, IL-17C,
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D. Radosavljevic, Gordana, Jelena Pantic, Bojana Simovic Markovic, and Nebojsa Arsenijevic. "Modulators of Tumor Angiogenesis: Insights into the Role of Galectin-3 and IL-17 Signaling." In Tumor Angiogenesis [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.102893.

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Angiogenesis is a pivotal point in tumor progression driven by firmly orchestrated process of forming the new blood vessels relying on the complex signaling network. Here, the pleiotropic functions of Galectin-3 and IL-17 in tumor progression have been overviewed through their impacts on angiogenesis. As a key player in tumor microenvironment, Galectin-3 orchestrates practically all critical events during angiogenic cascade through interaction with various ligands and their downstream signaling pathways. Galectin-3 shapes chronic inflammatory tumor microenvironment that is closely related to a
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Hamzaoui, Agnès, Sabrine Louhaichi, and Kamel Hamzaoui. "The Potential Value of Sputum Level Interleukin38 and NLRP3 Inflammasome in Severe Childhood Asthma." In The NLRP3 Inflammasome: An Attentive Arbiter of Inflammatory Response. BENTHAM SCIENCE PUBLISHERS, 2024. http://dx.doi.org/10.2174/9789815223941124010010.

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Asthma in children is associated with serious exacerbations that are modulated by inflammation. The expression of inflammatory cytokines varies according to the severity of the disease. The transition from the state of exacerbation of the disease to the state of cure always passes through a relationship between inflammatory and anti-inflammatory mediators. This study looks at the expression of IL-38 and NLRP3 inflammasome in severe childhood asthma. NLRP3 inflammasome is upregulated in severe asthma, contrasting with low levels of IL-38. The inflammatory pattern of severe asthma in children is
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Mukherjee, Saikat, Soubhik Ghosh, and Arindam Bhattacharyya. "Regulation of T-reg/Th-17 Balance: One Step Closer Towards Immunotherapy Against Malaria Infection." In Plasmodium Species and Drug Resistance [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.97045.

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According to World Malaria Report 2020, the rate of decline in malaria case incidence and deaths caused by malaria has ceased in latter half of the past decade. Though Artemisinin Combination Therapy (ACT) is still the major therapeutic approach globally to treat malaria patients, increased resistance of Plasmodium sp. to artemisinin can be looked upon as a major factor responsible for the rate of decline. In the present world, immunotherapeutic approaches are in the limelight to treat several infections, autoimmune disorders, cancers but application of such therapeutic measures in case of mal
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Ponist, Silvester, Katarina Pruzinska, and Katarina Bauerova. "Inflammation in the Pathogenesis of Rheumatoid Arthritis and in Experimental Arthritis: Evaluation of Combinations of Carnosic Acid and Extract of Rhodiola rosea L. with Methotrexate." In Inflammation [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.99073.

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The host immune response generates the pro-inflammatory immune response as a protective measure against invading pathogens, allergens, and/or trauma. However, dysregulated and chronic inflammation may result in secondary damage to tissues and immune pathology to the host. Rheumatoid arthritis (RA) is a chronic systemic autoimmune disease which primarily involves synovial inflammation, joint pain, immobility, and stiffness. Increased infiltration of inflammatory immune cells and fibroblast-like synoviocytes into joints, form pannus and small blood vessels that lead to synovium and cartilage des
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Salazar González, Fernando, Júlia Pardo Pastor, Nuria Meca Casasnovas, et al. "Estudio en vida real de efectividad y calidad de vida en pacientes con psoriasis moderada/grave y anti IL-12/23, IL-17, IL-23." In Libro de comunicaciones 69 Congreso SEFH. Sociedad Española de Farmacia Hospitalaria, 2024. http://dx.doi.org/10.62917/24.69.0391.

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Ji, Cheng’ao. "Anti-Inflammatory Mechanism of Ligustrum Lucidum Ait Based on Network Pharmacology and Molecular Docking." In Studies in Health Technology and Informatics. IOS Press, 2023. http://dx.doi.org/10.3233/shti230862.

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Objective: To investigate possible targets of Ligustrum lucidum and its anti-inflammatory mechanism. Core targets were screened by the established networks of component target and potential protein interaction, accompanied by GO and KEGG analyses. The findings were confirmed using molecular docking. Results: eight active components including Quercetin, luteolin, kaempferol and corresponding 163 potential targets, including CCL2, IL6, CXCL8, TNF, IL1B, were identified with a threshold of OB ≥ 30% and DL ≥ 0.18%. 1018 anti-inflammatory targets were acquired, with 101 common targets identified by
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Uzair, Mohammad, Hammad Qaiser, Muhammad Arshad, Saneela Tahseen, and Shahid Bashir. "Neurobiology of Cannabinoids and Medical Cannabis in Therapeutic Intervention for Multiple Sclerosis." In Medical Cannabis and the Effects of Cannabinoids on Fighting Cancer, Multiple Sclerosis, Epilepsy, Parkinson's, and Other Neurodegenerative Diseases. IGI Global, 2022. http://dx.doi.org/10.4018/978-1-6684-5652-1.ch008.

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Cannabinoids, either phytocannabinoids or synthetic derivatives of medical cannabis, exhibit a variety of potential pharmacotherapeutic effects by acting on the endocannabinoid system, through interactions with the cannabinoid receptors (CB1, CB2, and other receptors). Multiple sclerosis (MS) is an autoimmune demyelinating disease of the central nervous system (CNS) that causes spasticity, neuropathic pain, urinary bladder dysfunction, and sleep disturbance. Treatment with cannabinoids decreases the severity of disease in animal models of experimental autoimmune-Theiler's virus-induced-encepha
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Gaston, J. S. Hill. "Reactive arthritis and enteropathic arthropathy." In Oxford Textbook of Rheumatology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0115_update_003.

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Reactive arthritis (ReA), and enteropathic arthritis secondary to inflammatory bowel disease, are forms of spondyloarthritis, all of which share an association with HLA B27 and can involve both axial and peripheral joints. Genetic studies strongly implicate the cytokines IL-17 and IL-23 in their pathogenesis, and evidence for autoimmunity is lacking. ReA is triggered by particular bacteria, mainly affecting the gut and genitourinary tract, though infections are sometimes asymptomatic. Classically an acute oligo- or monoarthritis with enthesitis occurs, often with inflammatory back pain, though
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Gaston, J. S. Hill. "Reactive arthritis and enteropathic arthropathy." In Oxford Textbook of Rheumatology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0115_update_005.

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Reactive arthritis (ReA), and enteropathic arthritis secondary to inflammatory bowel disease, are forms of spondyloarthritis, all of which share an association with HLA B27 and can involve both axial and peripheral joints. Genetic studies strongly implicate the cytokines IL-17 and IL-23 in their pathogenesis, and evidence for autoimmunity is lacking. ReA is triggered by particular bacteria, mainly affecting the gut and genitourinary tract, though infections are sometimes asymptomatic. Classically an acute oligo- or monoarthritis with enthesitis occurs, often with inflammatory back pain, though
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Conference papers on the topic "Anti-IL-17"

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Felix Nascimento Coelho, Vanessa, María Verónica Russo Macchi, Carolina Pellisson Carvalho, et al. "Ixekizumab (anti - IL 17) triggering intestinal inflammatory." In SBR 2021 Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2021.2019.

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Hernández, R., A. Perez Gil, J. Uceda, and JL Marenco de la Fuente. "P095 Inflammatory bowel disease, during anti IL 17 treatment." In 39th European Workshop for Rheumatology Research, 28 February–2 March 2019, Lyon, France. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2018-ewrr2019.84.

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Kotz, Sebastian, Lisa Pechtold, Barbara Wollenberg, and Adam Chaker. "Side effects of Anti IL-17 A in the ENT domain." In 94th Annual Meeting German Society of Oto-Rhino-Laryngology, Head and Neck Surgery e.V., Bonn. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1767034.

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Kotz, Sebastian, Lisa Pechtold, Barbara Wollenberg, and Adam Chaker. "Nebenwirkungen im HNO-Bereich unter immunmodulatorischer Therapie mit Anti IL-17 A." In 94. Jahresversammlung Deutsche Gesellschaft für Hals-Nasen-Ohren-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1766447.

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Jörns, A., D. Wedekind, and S. Lenzen. "Wiederherstellung einer normoglykämischen Stoffwechsellage durch Kombinationstherapien mit anti-IL-6 und anti-IL-17 im Typ 1 Diabetes Tiermodell der IDDM-Ratte." In Diabetes Kongress 2019 – 54. Jahrestagung der DDG. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1688229.

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Kotliarova, Anastasiia, Anna S. Davydova, Veronika E. Goncharova, et al. "Anti-IL-17 nucleic acid aptamer alleviates motor impairment in mouse spondyloarthritis model." In 2022 IEEE International Multi-Conference on Engineering, Computer and Information Sciences (SIBIRCON). IEEE, 2022. http://dx.doi.org/10.1109/sibircon56155.2022.10016994.

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Camargo, L., T. M. D. Santos, S. Fukuzaki, et al. "Anti-IL-17 Treatment Modulates Pulmonary Responses in Model of Asthma-COPD Overlap." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a3230.

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Almeida, F. M., L. N. Camargo, N. Martins, et al. "Effects of anti-IL 17 on inflammation in Asthma, COPD and Asthma-COPD models." In ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.2570.

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Santos, Tabata, Renato Righetti, Luciana Aristóteles, et al. "Anti IL-17 with or without Rho kinase inhibitor in mice with chronic allergic pulmonary inflammation." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa1051.

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Hara, Hiromichi, Hanae Miyagawa, Jun Araya, et al. "Effect of anti-IL-17 biologics on KL-6 level and coexisting interstitial pneumoniain patients with psoriasis." In ERS International Congress 2020 abstracts. European Respiratory Society, 2020. http://dx.doi.org/10.1183/13993003.congress-2020.3398.

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Reports on the topic "Anti-IL-17"

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Palaga, Tanapat, Nattiya Hirankarn, and Hathaipat Phuwapirom. Level of IL-17 in Thai SLE patients. Chulalongkorn University, 2009. https://doi.org/10.58837/chula.res.2009.30.

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Systemic Lupus Erythematosus (SLE) is an autoimmune disorder which affects various systems. Currently, the etiology of this disease has not been fully elucidated. One of potential causes which may play an important role is the defects in cytokine network and the functions of T lymphocytes. Previously, it was reported that SLE patients showed elevated elevated level of various cytokines such as IL-1β IL-6 IL-23. The aim of this study was to investigate the level of cytokine IL-17 which could be produced by various cell types including T lymphocytes CD4+ T lymphocytes mainly producing IL-17 form
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ตั้งกิจวานิชย์, พิสิฐ, อัญชลี อวิหิงสานนท์, วรพจน์ ทรัพย์ศิริสวัสดิ์ та ін. การศึกษาธรรมชาติการดำเนินโรคของการติดเชื้อไวรัสตับอักเสบซี ความสำคัญทางคลินิคปัจจัยที่เกี่ยวข้องในการรักษาการตอบสนองต่อการรักษา และผลข้างเคียงในผู้ติดเชื้อเอชไอวีที่มีการติดเชื้อไวรัสตับอักเสบซี. จุฬาลงกรณ์มหาวิทยาลัย, 2015. https://doi.org/10.58837/chula.res.2015.24.

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การศึกษานี้เป็นแบบไปข้างหน้าในผู้ป่วยที่ติดเชื้อเอชไอวีร่วมกับการติดเชื้อไวรัสตับอักเสบซีในพื้นที่กรุงเทพมหานคร ประเทศไทย โดยมีการตรวจสายพันธุ์ของไวรัสตับอักเสบซี ปริมาณไวรัสตับอักเสบซีในเลือด ยีน IL-288 และการเกิดพังผืดในตับด้วยเครื่องไฟโบรสแกน และได้แบ่งระยะของพังผืดในตับได้ดังนี้ ระยะแรก (Metavir F0-F1) เป็นระยะที่มีค่า stiffness น้อยกว่าหรือเท่ากับ 7.1 kPa ระยะที่ 2 หรือระยะปานกลาง (F2) เป็นระยะที่มีค่า stiffness ระหว่าง 7.2-9.4 kPa ระยะที่ 3 หรือระยะรุนแรง (F3) เป็นระยะที่มีค่า stiffness ระหว่าง 9.5-14 kPa และระยะสุดท้าย (F4) หรือ โรคตับแข็งที่มีค่า stiffness มากกว่า 14 kPa การตรวจประเมิน
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