Relevant bibliographies by topics / Anti-IL-23

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Anti-IL-23'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Anti-IL-23"

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Schwartz, David. "Anti-IL-12/23." Inflammatory Bowel Diseases 15, no. 12 (2009): 1927–28. http://dx.doi.org/10.1002/ibd.20990.

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Gras, J. "Guselkumab. Anti-IL-23 antibody, Antipsoriatic." Drugs of the Future 42, no. 2 (2017): 81. http://dx.doi.org/10.1358/dof.2017.042.02.2564098.

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Held, Katherine, William Glass, Yevgeniya Orlovsky, et al. "Generation of a protective T cell response following viral infection of the CNS is independent of either IL-12 or IL-23 (B141)." Journal of Immunology 178, no. 1_Supplement (2007): LB29. http://dx.doi.org/10.4049/jimmunol.178.supp.b141.

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Abstract The functional role of IL-12 and IL-23 in generating effector T cells following viral infection of the central nervous system (CNS) was determined. Instillation of mouse hepatitis virus (MHV) into the CNS of C57BL/6 mice results in an acute encephalitis followed by a chronic demyelinating disease. MHV-infected mice express mRNA transcripts specific for IL-12p35 and IL-23p19 subunits within the brains. Antibody-mediated blocking of IL-23 (anti-IL-23p19) or IL-12 and IL-23 (anti-IL-12/23p40) did not reduce numbers of virus-specific T cells and treated mice cleared virus from the CNS dem
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Brod, Staley A. "Ingested (oral) anti-IL-12/23 inhibits EAE." Journal of the Neurological Sciences 361 (February 2016): 19–25. http://dx.doi.org/10.1016/j.jns.2015.12.011.

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Wendling, Daniel, Clément Prati, Mickael Chouk, and Frank Verhoeven. "Effects of anti-IL-23 and anti-IL-17: The hidden side of spondyloarthritis polymorphism?" Joint Bone Spine 87, no. 1 (2020): 5–7. http://dx.doi.org/10.1016/j.jbspin.2019.06.012.

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Cao, Hao, Qin Lan, Qian Shi, et al. "Anti-IL-23 antibody blockade of IL-23/IL-17 pathway attenuates airway obliteration in rat orthotopic tracheal transplantation." International Immunopharmacology 11, no. 5 (2011): 569–75. http://dx.doi.org/10.1016/j.intimp.2010.11.007.

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Hamada, Kaoru, Yu Sawada, Ryosuke Hino, and Motonobu Nakamura. "HTLV-1 carrier psoriasis patients treated by anti-IL-23/IL-17." Australasian Journal of Dermatology 59, no. 2 (2017): e154-e154. http://dx.doi.org/10.1111/ajd.12701.

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Kyttaris, Vasileios C., Ourania Kampagianni, and George C. Tsokos. "Treatment with Anti-Interleukin 23 Antibody Ameliorates Disease in Lupus-Prone Mice." BioMed Research International 2013 (2013): 1–5. http://dx.doi.org/10.1155/2013/861028.

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Interleukin 23 receptor expressing IL-17 producing T cells have been shown to be important in the development of murine lupus. The usefulness of IL-23 inhibition in ameliorating lupus nephritis is unknown. We hypothesized that inhibition of IL-23 will ameliorate nephritis in lupus-prone mice. To this end, we treated MRL/lprlupus-prone mice for 6 weeks with a rat anti-IL-23p19 antibody, which resulted in delaying the onset of nephritis without affecting the production of anti-dsDNA antibodies. The effect of the treatment was hampered by the production of murine anti-rat IgG antibodies. The amel
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Alhasani, Salam, and Nasser Yousif. "Critical role of IL-23 signaling in prostatic cancer." American Journal of BioMedicine 1, no. 1 (2013): 4–6. http://dx.doi.org/10.18081/ajbm/2333-5106-013-11/4-6.

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Interleukin 23 (IL-23) belongs to interleukin 6 super-family [1]. IL-12/23 p40 is the common subunit for them, which is covalently linked to either a p19 subunit to form IL-23 or a p35 subunit to form IL-12 [2]. Both cytokines are mainly expressed by activating dendrite cells or macrophages under the stimulation of pathogens. IL-23 was also reported to be secreted by tumor associated macrophages in tumor microenvironment [3]. Interestingly, IL-23 spur different immune pathways [4]. IL-12 induces IFN-γ-producing Th1 cell development and enhances cytotoxic, anti-microbial and anti-tumor response
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Whitley, Sarah K., Mushi Li, Tracy Tabib, et al. "IL-23 maintains tissue resident memory Th17 cells in murine and psoriatic skin." Journal of Immunology 206, no. 1_Supplement (2021): 98.50. http://dx.doi.org/10.4049/jimmunol.206.supp.98.50.

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Abstract Tissue resident memory Th17 cells (TRM17) are the key cell type driving the chronic skin inflammation of psoriasis. Although IL-23 is strongly associated with autoimmunity and chronic inflammatory disorders including psoriasis, and anti-IL-23 biologic agents have remarkable efficacy in the treatment of psoriasis, the precise role of IL-23 in supporting IL-17-mediated skin inflammation remains unclear. In mice, we found that circulating memory T cells were dispensable for anamnestic protection from C. albicans skin infection, and TRM17 mediated protection from C. albicans reinfection r
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Dissertations / Theses on the topic "Anti-IL-23"

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Milanez, Fernanda Manente. "O uso de medicações anti-TNF não influencia o eixo IL-23/IL-17 em pacientes com espondilite anquilosante." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/5/5164/tde-23082017-111419/.

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Introdução: Apesar dos recentes avanços no entendimento da fisiopatologia e no tratamento da espondilite anquilosante (EA), pouco se sabe acerca da influência das medicações anti-fator de necrose tumoral (anti-TNF) sobre as novas vias inflamatórias descritas na patogênese das espondiloartrites. Objetivo: Dessa forma, o objetivo desse estudo é investigar e descrever a influência a longo prazo das medicações anti-TNF sobre o eixo da IL-23/IL-17 em pacientes com EA e sua possível correlação com o tratamento, parâmetros clínicos, laboratoriais e radiológicos. Métodos: Oitenta e seis pacientes com
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SILVA, Fabiana Leticia da. "Efeitos da amamentação em camundongos esquistossomóticos na imunidade anti-ovalbumina de descendentes adultos deficientes na produção das citocinas IL-12/IL-23." Universidade Federal de Pernambuco, 2014. https://repositorio.ufpe.br/handle/123456789/17158.

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Submitted by Fabio Sobreira Campos da Costa (fabio.sobreira@ufpe.br) on 2016-06-28T14:37:59Z No. of bitstreams: 2 license_rdf: 1232 bytes, checksum: 66e71c371cc565284e70f40736c94386 (MD5) TESE_FABIANA_LETICIA_DIGITAL.pdf: 2539644 bytes, checksum: ffb2ea48912cab654c0a8e8bdc397ac2 (MD5)<br>Made available in DSpace on 2016-06-28T14:37:59Z (GMT). No. of bitstreams: 2 license_rdf: 1232 bytes, checksum: 66e71c371cc565284e70f40736c94386 (MD5) TESE_FABIANA_LETICIA_DIGITAL.pdf: 2539644 bytes, checksum: ffb2ea48912cab654c0a8e8bdc397ac2 (MD5) Previous issue date: 2014-08-27<br>FACEPE<br>O contato prévi
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Konarski, Yulia. "SHP-1/ Src Complex is a Master Regulator of the IL-12/IL-23 pro- and IL-10/IL-27 Anti-inflammatory Axis in TLR4-activated Signaling Pathways in Human Monocytes and Macrophages." Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/25999.

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Although the etiology surrounding many autoimmune diseases remains unknown, the underlying characteristic of many of these diseases is a disruption in the balance of pro- and anti- inflammatory cytokines It is well established that the dysregulation of the IL-12 family of cytokines, an increase in IL-12/IL-23 and a decrease in IL-27 production has been implicated in these conditions. We used ELISA, RT-PCR, Immunofluorescence and Western immunoblotting in conjunction with pharmalogical inhibitors and siRNA to demonstrate the role of SHP-1/Src in the regulation of IL-12, IL-23, IL-27 and IL-10 i
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Giudice, Elena <1996&gt. "La resistenza nel Sichuan: Chongqing capitale ed il giornalismo anti giapponese." Master's Degree Thesis, Università Ca' Foscari Venezia, 2020. http://hdl.handle.net/10579/18206.

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Dal 1937 al 1945, anni della Guerra di resistenza Sino-Giapponese, il Partito Nazionalista Cinese – allora alla guida del paese – spostò provvisoriamente la capitale a Chongqing. Questa scelta fu dettata fondamentalmente dalla sua posizione strategica che la rendeva facilmente difendibile da aggressioni straniere via terra (che invece stavano devastando il nord e l’est della Cina). È in questi anni che Chongqing, da cittadina sottosviluppata e periferica, diventa municipalità e, soprattutto, simbolo della resistenza cinese ai raid giapponesi. Sulla base della letteratura secondaria prodotta, i
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Books on the topic "Anti-IL-23"

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Pitzalis, Costantino, Frances Humby, and Michael P. Seed. Synovial pathology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0052.

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Synovial pathology is seen in a variety of disease states, including rheumatoid arthritis (RA), osteoarthritis (OA), psoriatic arthritis, and systemic lupus erythmatosus (SLE). This chapter highlights recent advances that characterize the cellular composition of these tissues according to surface markers and chemokine and cytokine expression, and describes synovial functional status and response to therapeutics. In RA, after initiation, pannus migrates over and under cartilage, and into subchondral bone, in a destructive process. Cartilage-pannus junction (CPJ) is characterized as invasive or
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Gaston, J. S. Hill. Reactive arthritis and enteropathic arthropathy. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0115.

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Reactive arthritis (ReA), and enteropathic arthritis secondary to inflammatory bowel disease, are forms of spondyloarthritis, all of which share an association with HLA B27 and can involve both axial and peripheral joints. Genetic studies strongly implicate the cytokines IL-17 and IL-23 in their pathogenesis, and evidence for autoimmunity is lacking. ReA is triggered by particular bacteria, mainly affecting the gut and genitourinary tract, though infections are sometimes asymptomatic. Classically an acute oligo- or monoarthritis with enthesitis occurs, often with inflammatory back pain, though
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Gaston, J. S. Hill. Reactive arthritis and enteropathic arthropathy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0115_update_002.

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Reactive arthritis (ReA), and enteropathic arthritis secondary to inflammatory bowel disease, are forms of spondyloarthritis, all of which share an association with HLA B27 and can involve both axial and peripheral joints. Genetic studies strongly implicate the cytokines IL-17 and IL-23 in their pathogenesis, and evidence for autoimmunity is lacking. ReA is triggered by particular bacteria, mainly affecting the gut and genitourinary tract, though infections are sometimes asymptomatic. Classically an acute oligo- or monoarthritis with enthesitis occurs, often with inflammatory back pain, though
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Book chapters on the topic "Anti-IL-23"

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Salazar González, Fernando, Júlia Pardo Pastor, Nuria Meca Casasnovas, et al. "Estudio en vida real de efectividad y calidad de vida en pacientes con psoriasis moderada/grave y anti IL-12/23, IL-17, IL-23." In Libro de comunicaciones 69 Congreso SEFH. Sociedad Española de Farmacia Hospitalaria, 2024. http://dx.doi.org/10.62917/24.69.0391.

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Pitzalis, Costantino, Frances Humby, and Michael P. Seed. "Synovial pathology." In Oxford Textbook of Rheumatology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0052_update_001.

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Synovial pathology is seen in a variety of disease states, including rheumatoid arthritis (RA), osteoarthritis (OA), psoriatic arthritis, and systemic lupus erythmatosus (SLE). This chapter highlights recent advances that characterize the cellular composition of these tissues according to surface markers and chemokine and cytokine expression, and describes synovial functional status and response to therapeutics. In RA, after initiation, pannus migrates over and under cartilage, and into subchondral bone, in a destructive process. Cartilage-pannus junction (CPJ) is characterized as invasive or
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3

Gaston, J. S. Hill. "Reactive arthritis and enteropathic arthropathy." In Oxford Textbook of Rheumatology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0115_update_003.

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Reactive arthritis (ReA), and enteropathic arthritis secondary to inflammatory bowel disease, are forms of spondyloarthritis, all of which share an association with HLA B27 and can involve both axial and peripheral joints. Genetic studies strongly implicate the cytokines IL-17 and IL-23 in their pathogenesis, and evidence for autoimmunity is lacking. ReA is triggered by particular bacteria, mainly affecting the gut and genitourinary tract, though infections are sometimes asymptomatic. Classically an acute oligo- or monoarthritis with enthesitis occurs, often with inflammatory back pain, though
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4

Gaston, J. S. Hill. "Reactive arthritis and enteropathic arthropathy." In Oxford Textbook of Rheumatology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0115_update_005.

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Abstract:
Reactive arthritis (ReA), and enteropathic arthritis secondary to inflammatory bowel disease, are forms of spondyloarthritis, all of which share an association with HLA B27 and can involve both axial and peripheral joints. Genetic studies strongly implicate the cytokines IL-17 and IL-23 in their pathogenesis, and evidence for autoimmunity is lacking. ReA is triggered by particular bacteria, mainly affecting the gut and genitourinary tract, though infections are sometimes asymptomatic. Classically an acute oligo- or monoarthritis with enthesitis occurs, often with inflammatory back pain, though
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Poddubnyy, Denis, and Joachim Sieper. "Axial spondyloarthritis." In Oxford Textbook of Rheumatology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0113_update_005.

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Axial spondyloarthritis (axSpA) is a chronic inflammatory disease predominantly of the sacroiliac joint (SIJ) and the spine. It starts normally in the second decade of life and has a slight male predominance. The prevalence is between 0.2% and 0.8% and is strongly dependent on the prevalence of HLA-B27 in a given population. AxSpA can be split in patients with radiographic axSpA (also termed ankylosing spondylitis (AS)) and in patients with non-radiographic axSpA (nr-axSpA). For the diagnosis of AS, the presence of radiographic sacroiliitis is mandatory. However, radiographs do not detect acti
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Spector, Céline. "Le moment européen." In Le moment européen. Hermann, 2024. http://dx.doi.org/10.3917/herm.crign.2024.01.0061.

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Cette contribution entend revenir sur les arguments du discours conservateur et eurosceptique tenu par Viktor Orbán dans son allocution du 23 juillet 2023. Peut-on surmonter l’objection selon laquelle la démocratie trouve à s’exprimer de manière privilégiée — voire unique — au sein des États-nations ? Que répondre aux politiques qui soutiennent que la nation est la condition sine qua non de la démocratie, le seul lieu d’exercice possible des droits politiques, le terreau de la liberté, de l’égalité et de la fraternité ? N’a-t-on plus le choix qu’entre le Charybde de la mondialisation et le Scy
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Conference papers on the topic "Anti-IL-23"

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Pang, Dan-Dan, LI Cai, and Sheng-Ming Dai. "AB0152 IL-23 INDUCES OSTEOCLASTS TO EXPRESS PRO-OSTEOGENIC FACTORS RATHER THAN ANTI-OSTEOGENIC FACTORS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.764.

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Frederick, Josh P., Ailin Bai, Susannah Hewitt та ін. "Abstract 1607: Durable efficacy and anti-cancer immunity following intratumoral administration of messenger RNAs encoding IL-36γ, IL-23 and OX40L". У Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1607.

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Domínguez Senín, L., S. Camacho Parreño, E. Sánchez Gómez, M. Rodriguez Jorge, and MD Santos-Rubio. "5PSQ-116 Persistence of inhibitors of interleukin-23 (anti-IL-23) for the treatment of moderate-to-severe psoriasis (msPs) in the routine clinical practice conditions." In 27th EAHP Congress, Lisbon, Portugal, 22-23-24 March 2023. British Medical Journal Publishing Group, 2023. http://dx.doi.org/10.1136/ejhpharm-2023-eahp.314.

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Gonçalves, Ana Isabel de Almeida, ANA LÍGIA TIEMI SOARES NISHIMURA, BRUNO AUGUSTO DA SILVA CORDEIRO, GUILHERME ALVES SILVA, and MONIELLI MURIBECA SILVA. "Mecanismos patológicos da doença de Crohn." In II Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conbrai/6701.

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Introdução: A doença de Crohn (DC) é uma doença inflamatória crônica gastrointestinal, multifatorial de etiologia inconclusiva, resultando da interação entre genética, ambiente e microbiota intestinal alterada, desregulando a resposta imunológica. Essa doença afeta pessoas de várias idades, sexo e raça, com maior incidência em regiões urbanas. Objetivo: Analisar os aspectos imunológicos e fisiopatológicos ligados à DC. Métodos: Trata-se de um estudo transversal, com revisão bibliográfica, com uma busca por periódicos publicados entre 2016 e 2022, nos bancos de dados: PUBMED, SCIELO e LILACS, c
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Campbell-Harding, Gemma, Hannah Sawkins, Nicole Bedke, Stephen T. Holgate, Donna E. Davies, and Allison-Lynn Andrews. "The Innate Anti-Viral Response Upregulates IL-13R±2 In Bronchial Fibroblasts." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5388.

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Counihan, Ian P., Sarah L. O'Beirne, Robert V. Lumsden, et al. "The Anti Fibrotic Effects Of Mesenchymal Stem Cells Are Mediated In Part By IL-13R±2." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6532.

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McClintock, Dana, Jonathan Corren, Nicola A. Hanania, et al. "Lebrikizumab, An Anti-IL-13 Monoclonal Antibody, Reduces Severe Asthma Exacerbations Over 32 Weeks In Adults With Inadequately Controlled Asthma." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3959.

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Piper, Edward, Dewei She, and Nestor A. Molfino. "Subgroup Analysis Of A Phase 2a Randomized, Double-Blind, Placebo-Controlled Study Of Tralokinumab, An Anti-IL-13 Monoclonal Antibody, In Moderate To Severe Asthma." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2759.

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Taylor, VM, T. Patrick, AP Hearn, et al. "P210 The impact of introducing anti-IL-5/5R biologic therapies on weight and BMI in prednisolone-dependent patients with severe asthma." In British Thoracic Society Winter Meeting 2022, QEII Centre, Broad Sanctuary, Westminster, London SW1P 3EE, 23 to 25 November 2022, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2022. http://dx.doi.org/10.1136/thorax-2022-btsabstracts.343.

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Oh, Chad K., Kimmie K. McLaurin, Keunpyo Kim, Micki Hultquist, and Nestor A. Molfino. "A Phase 2b, Randomized Study To Evaluate The Clinical Activity And Safety Profile Of Subcutaneous MEDI-528, An Anti-IL-9 Monoclonal Antibody, In Adults With Uncontrolled Asthma." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2760.

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Reports on the topic "Anti-IL-23"

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Palaga, Tanapat, Nattiya Hirankarn, and Hathaipat Phuwapirom. Level of IL-17 in Thai SLE patients. Chulalongkorn University, 2009. https://doi.org/10.58837/chula.res.2009.30.

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Systemic Lupus Erythematosus (SLE) is an autoimmune disorder which affects various systems. Currently, the etiology of this disease has not been fully elucidated. One of potential causes which may play an important role is the defects in cytokine network and the functions of T lymphocytes. Previously, it was reported that SLE patients showed elevated elevated level of various cytokines such as IL-1β IL-6 IL-23. The aim of this study was to investigate the level of cytokine IL-17 which could be produced by various cell types including T lymphocytes CD4+ T lymphocytes mainly producing IL-17 form
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