Relevant bibliographies by topics / Anti-inflammatory agents Therapeutic use

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Anti-inflammatory agents Therapeutic use'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Anti-inflammatory agents Therapeutic use"

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La Manna, Sara, Concetta Di Natale, Daniele Florio, and Daniela Marasco. "Peptides as Therapeutic Agents for Inflammatory-Related Diseases." International Journal of Molecular Sciences 19, no. 9 (2018): 2714. http://dx.doi.org/10.3390/ijms19092714.

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Inflammation is a physiological mechanism used by organisms to defend themselves against infection, restoring homeostasis in damaged tissues. It represents the starting point of several chronic diseases such as asthma, skin disorders, cancer, cardiovascular syndrome, arthritis, and neurological diseases. An increasing number of studies highlight the over-expression of inflammatory molecules such as oxidants, cytokines, chemokines, matrix metalloproteinases, and transcription factors into damaged tissues. The treatment of inflammatory disorders is usually linked to the use of unspecific small m
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Grbovic, Leposava, and Miroslav Radenkovic. "Therapeutic use of glucocorticoids and immunosuppressive agents." Srpski arhiv za celokupno lekarstvo 133, Suppl. 1 (2005): 67–73. http://dx.doi.org/10.2298/sarh05s1067g.

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Pharmacotherapy of autoimmune thyroid disease (AITD) is complex. Apart from the replacement hormone therapy, antithyroid agents, beta adrenoceptor blockers and other drugs, in regard to the present symptoms, it also includes the administration of glucocorticoids and immunosuppressive agents. Physiological actions of glucocorticoids are significant in number, well known and described in details. The most prominent pharmacological properties of glucocorticoids, that are important for their clinical use, are antiinflammatory and immunosuppressive actions. In this article, the most notable clinica
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Sparrow, Miles P., Konstantinos Papamichael, Mark G. Ward, et al. "Therapeutic Drug Monitoring of Biologics During Induction to Prevent Primary Non-Response." Journal of Crohn's and Colitis 14, no. 4 (2019): 542–56. http://dx.doi.org/10.1093/ecco-jcc/jjz162.

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Abstract Biologic therapies have revolutionized the management of inflammatory bowel disease [IBD], but primary and secondary non-responses occur in a significant proportion of patients. Therapeutic drug monitoring [TDM] now has an established role in the treatment algorithm for managing secondary loss of response to anti-tumour necrosis factor [anti-TNF] agents during maintenance therapy. Data to support the use of TDM in the management of secondary loss of response to vedolizumab and ustekinumab are emerging. The potential to prevent primary non-response to biologic agents during induction i
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Shafiee, Nor Hamizah, Zahara Abdul Manaf, Norfilza M. Mokhtar, and Raja Affendi Raja Ali. "Anti-inflammatory diet and inflammatory bowel disease: what clinicians and patients should know?" Intestinal Research 19, no. 2 (2021): 171–85. http://dx.doi.org/10.5217/ir.2020.00035.

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Current treatment for inflammatory bowel disease (IBD) includes the application of anti-inflammatory agents for the induction and remission of IBD. However, prolonged use of anti-inflammatory agents can exert adverse effects on patients. Recently, formulated dietary approach in treating IBD patients is utilized to improve clinical activity scores. An alteration of gastrointestinal microbiota through dietary therapy was found to reduce IBD and is recognized as a promising therapeutic strategy for IBD. One of the recommended formulated diets is an anti-inflammatory diet (AID) that restricts the
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Aardoom, Martine A., Gigi Veereman, and Lissy de Ridder. "A Review on the Use of Anti-TNF in Children and Adolescents with Inflammatory Bowel Disease." International Journal of Molecular Sciences 20, no. 10 (2019): 2529. http://dx.doi.org/10.3390/ijms20102529.

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Inflammatory bowel disease (IBD) presents with disabling symptoms and may lead to insufficient growth and late pubertal development in cases of disease onset during childhood or adolescence. During the last decade, the role of anti-tumor necrosis factor (TNF) in the treatment of paediatric-onset IBD has gained more ground. The number of biologicals presently available for children and adolescents with IBD has increased, biosimilars have become available, and practices in adult gastroenterology with regards to anti-TNF have changed. The aim of this study is to review the current evidence on the
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Gorabi, Kiaie, Reiner, Carbone, Montecucco, and Sahebkar. "The Therapeutic Potential of Nanoparticles to Reduce Inflammation in Atherosclerosis." Biomolecules 9, no. 9 (2019): 416. http://dx.doi.org/10.3390/biom9090416.

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Chronic inflammation is one of the main determinants of atherogenesis. The traditional medications for treatment of atherosclerosis are not very efficient in targeting atherosclerotic inflammation. Most of these drugs are non-selective, anti-inflammatory and immunosuppressive agents that have adverse effects and very limited anti-atherosclerotic effects, which limits their systemic administration. New approaches using nanoparticles have been investigated to specifically deliver therapeutic agents directly on atherosclerotic lesions. The use of drug delivery systems, such as polymeric nanoparti
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Winston, Kevin, Hasan Maulahela, Lusiani Lusiani, Raditya Dewangga, and Lazuardi G. Ilhami. "Current Role of Anti-Integrin Therapy in Inflammatory Bowel Disease." Indonesian Journal of Gastroenterology, Hepatology, and Digestive Endoscopy 21, no. 2 (2020): 137–45. http://dx.doi.org/10.24871/2122020137-145.

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Inflammatory bowel disease (IBD) is a chronic inflammatory intestinal disorder with multifactorial etiology. Management of IBD is divided into conventional treatment and new treatment with biologic agents. The first biologic agents used for IBD was tumor necrosis factor (TNF)-inhibitor. However, TNF-inhibitor as a biologic agent has several limitations such as low rate of clinical response and systemic immunosuppressive side effects. Anti-integrin is a recently developed biologic agent which selectively inhibits leukocyte trafficking towards site of inflammation. The inhibition is caused by bl
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Veerasamy, Ravichandran, Anitha Roy, Rohini Karunakaran, and Harish Rajak. "Structure–Activity Relationship Analysis of Benzimidazoles as Emerging Anti-Inflammatory Agents: An Overview." Pharmaceuticals 14, no. 7 (2021): 663. http://dx.doi.org/10.3390/ph14070663.

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A significant number of the anti-inflammatory drugs currently in use are becoming obsolete. These are exceptionally hazardous for long-term use because of their possible unfavourable impacts. Subsequently, in the ebb-and-flow decade, analysts and researchers are engaged in developing new anti-inflammatory drugs, and many such agents are in the later phases of clinical trials. Molecules with heterocyclic nuclei are similar to various natural antecedents, thus acquiring immense consideration from scientific experts and researchers. The arguably most adaptable heterocyclic cores are benzimidazole
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Parikh, Chinar R., Jaya K. Ponnampalam, George Seligmann, et al. "Impact of immunogenicity on clinical efficacy and toxicity profile of biologic agents used for treatment of inflammatory arthritis in children compared to adults." Therapeutic Advances in Musculoskeletal Disease 13 (January 2021): 1759720X2110026. http://dx.doi.org/10.1177/1759720x211002685.

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The treatment of inflammatory arthritis has been revolutionised by the introduction of biologic treatments. Many biologic agents are currently licensed for use in both paediatric and adult patients with inflammatory arthritis and contribute to improved disease outcomes compared with the pre-biologic era. However, immunogenicity to biologic agents, characterised by an immune reaction leading to the production of anti-drug antibodies (ADAs), can negatively impact the therapeutic efficacy of biologic drugs and induce side effects to treatment. This review explores for the first time the impact of
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Blebea, Nicoleta Mirela, Laura Adriana Bucur, and Simona Negreș. "The cannabinoids – important therapeutic approach in the field of oncology." Romania Journal of Pharmaceutical Practice 57, no. 2 (2021): 63–67. http://dx.doi.org/10.37897/rjphp.2021.2.1.

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Oncological diseases are the most common cause of death worldwide, it is estimated that 25% of the population will face the diagnosis of cancer during life. Phytochemicals such as cannabinoids (CBs) have been used in various branches of medicine for their properties, and the discovery of the anti-tumor, anti-emetic and anti-inflammatory effects of some of these substances has encouraged their use in oncology. Phytocannabinoids, cannabidiol (CBD) and Δ-9-tetra-hydrocannabinol (THC), have numerous anti-emetic, analgesic, orexigenic, anti-inflammatory / immunosuppressive pharmacodynamic effects.
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Dissertations / Theses on the topic "Anti-inflammatory agents Therapeutic use"

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Dairam, Amichand. "An investigation into the neuroprotective properties of the non-steroidal anti-inflammatory agents tolmetin, sulindac and turmeric." Thesis, Rhodes University, 2006. http://hdl.handle.net/10962/d1003230.

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Accumulating evidence suggests that anti-inflammatory agents and antioxidants have neuroprotective properties and may be beneficial in the treatment of neurodegenerative disorders. In the present study, the possible neuroprotective properties of tolmetin, sulindac and turmeric were investigated. The antioxidant effects of tolmetin and sulindac were determined by inducing free radical generation with quinolinic acid (QA), cyanide or iron (II) in rat brain homogenates or primary hippocampal neurons. Tolmetin and sulindac significantly reduce lipid peroxidation and scavenge the superoxide anion.
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Chew, Angela Christine. "The anti-proliferative effects of thiazolidinediones and non-steriodal anti-inflammatory drugs on androgen-independent prostate cancer." University of Western Australia. School of Medicine and Pharmacology, 2009. http://theses.library.uwa.edu.au/adt-WU2009.0200.

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[Truncated abstract] In recent years a better understanding of the biology of PPAR , a nuclear transcription factor, has emerged, leading to a resurgence in targeting PPAR for chemotherapy. The family of synthetic PPAR agonists, the thiazolidinediones (TZDs), and non-steroidal anti-inflammatory drugs (NSAIDs) have been implicated in the inhibition of cell proliferation, apoptosis and cell cycle arrest of androgen-sensitive (LNCaP) and androgen-independent (PC-3 and DU145) prostate cancer cells generating much interest in their use for potential curative cancer therapies. In light of the pote
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Keet, Lana. "Development of in vitro models to investigate the anti-inflammatory properties of Cyclopia Maculata and other South African herbal teas : a comparative study." Thesis, Stellenbosch : Stellenbosch University, 2015. http://hdl.handle.net/10019.1/97030.

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Thesis (MSc)--Stellenbosch University, 2015.<br>ENGLISH ABSTRACT: Chronic inflammation is suggested to contribute to cancer development and therefore a potential target for chemoprevention. In the skin, keratinocytes and macrophages play an integral part in acute and chronic inflammation, with interleukin 1-α (IL-1α) and tumor necrosis factor α (TNF-α) as key cytokines governing this process. Green tea (Camellia sinensis) and the South African herbal teas, rooibos (Aspalathus linearis) and honeybush (Cyclopia spp.) displayed antiinflammatory effects in mouse and human skin. To further in
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Wong, Lai-Ming Ella, and 黃禮明. "Iron and ruthenium complexes with nitrogen and oxygen donor ligands for anti-cancer and anti-viral studies." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B3587742X.

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Garrett, Ian Ross. "Studies of the effect of metal containing drugs on acute and chronic inflammation /." Title page, table of contents and summary only, 1986. http://web4.library.adelaide.edu.au/theses/09PH/09phg2386.pdf.

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Sagar, Mohamed. "The importance of the CYP2C19 polymorphism for disposition and effects of omeprazole treatment /." Stockholm, 1999. http://diss.kib.ki.se/1999/91-628-3624-2/.

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Demasi, Maryanne. "The effects of hypoxia on cyclooxygenase-2 expression and eicosanoid synthesis /." Title page, table of contents and summary only, 2004. http://web4.library.adelaide.edu.au/theses/09PH/09phd3729.pdf.

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Thesis (Ph.D.)--University of Adelaide, Dept. of Medicine and Royal Adelaide Hospital, Rheumatology Unit, 2004.<br>Includes list of publications arising from this thesis. Erratum attached to inside back cover. "25/03/2004." Includes bibliographical references (leaves 185-257).
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Lee, Yee Man. "Anti-tumor mechanisms of 2-methoxyestradiol in nasopharyngeal carcinoma." HKBU Institutional Repository, 2007. http://repository.hkbu.edu.hk/etd_ra/866.

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Dong, Hang. "Study of the anti-cancer effect and mechanism of compound 9 : a novel derivative of the PPD-type ginsenoside." HKBU Institutional Repository, 2012. https://repository.hkbu.edu.hk/etd_ra/1446.

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Wu, Jun, and 吴隽. "Drug delivery devices fabricated by microfluidic method and their applications in long-term antimicrobial therapy." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/198816.

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Controlled drug delivery devices provide numerous advantages such as reduced side effects, higher therapeutic efficiency and improved patient compliance. Biodegradable polymer has become the most important material for controlled drug delivery device because of the excellent biocompatibility and tunable physicochemical properties. Biodegradable polymeric drug delivery devices are usually processed into various types of micro-particles due to the ease of fabrication and administration. However, controlling the drug release kinetics of these microparticles is still a challenge. One important rea
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Books on the topic "Anti-inflammatory agents Therapeutic use"

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Pelt, Annemarie C. Glucocorticoids: Effects, action mechanisms, and therapeutic uses. Nova Science, 2011.

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Inflammation Research Association. International Conference. Therapeutic approaches to inflammatory diseases: Proceedings of the Fourth International Conference of the Inflammation Research Association, held October 23-27, 1988 in White Haven, Pennsylvania, U.S.A. Edited by Lewis Alan 1945-, Doherty Niall S, and Ackerman Neil. Elsevier, 1989.

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New York Academy of Sciences, ed. Antimicrobial therapeutics reviews. Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2010.

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Jacob, Stanley W. MSM - the definitive guide: A comprehensive review of the science and therapeutics of methylsulfonylmethane. Freedom Press, 2003.

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Antacids and anti-reflux agents. CRC Press, 1991.

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Fabio, Anthony Di. The art of getting well!: Rheumatoid arthritis cured at last!! Rheumatoid Disease Foundation, 1988.

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Gratieri, Tais. Pharmacological treatment of ocular inflammatory diseases. Nova Science Publishers, 2010.

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Zänker, Kurt S., and Srini V. Kaveri. Mistletoe: From mythology to evidence-based medicine. Karger, 2015.

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Neil, Ackerman, Bonney R. J. 1942-, and Welton A. F. 1947-, eds. Progress in inflammation research and therapy: Based on the Fifth International Conference of the Inflammation Research Association, September 23-27, 1990, White Haven, PA, USA. Birkhaüser Verlag, 1991.

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NATO Advanced Study Institute on Prostanoids and Drugs (1988 Erice, Italy). Prostanoids and drugs. Plenum Press, 1989.

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Book chapters on the topic "Anti-inflammatory agents Therapeutic use"

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Mathur, Rajani, and Renu Agarwal. "Steroidal and Nonsteroidal Anti-inflammatory Agents for Ocular Use." In Pharmacology of Ocular Therapeutics. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-25498-2_8.

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Finberg, Robert W., and Roy Guharoy. "Vancomycin Dosing: New Area-Under-the-Curve-Based Therapeutic Monitoring." In Clinical Use of Anti-infective Agents. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-67459-5_9.

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Ogawa, Rei. "Long-Pulsed 1064 nm Nd:YAG Laser Treatment for Keloids and Hypertrophic Scars." In Textbook on Scar Management. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-44766-3_32.

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AbstractThere are many therapeutic options for keloids and hypertrophic scars, including surgery, radiation, corticosteroids, 5-fluorouracil, cryotherapy, laser therapy, anti-allergy agents, anti-inflammatory agents, bleaching creams, and make-up therapies. In terms of laser therapy, we have used long-pulsed 1064 nm Nd:YAG laser to treat keloids and hypertrophic scars. This laser was developed for the treatment of vascular diseases, including inflammatory scars that exhibit neovascularization. The depth that is reached is determined by the spot size, the laser power, and the fluence: the larger the spot size, power, or fluence, the deeper the laser beam penetrates. The laser should generally be applied to the skin surface with the following standard treatment settings: a spot diameter of 5 mm, an energy density of 75 J/cm2, an exposure time per pulse of 25 ms, and a repetition rate of 2 Hz.
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Rossi, John J., and Nava Sarver. "Catalytic Antisense RNA (Ribozymes): Their Potential and Use as ANTI-HIV-1 Therapeutic Agents." In Advances in Experimental Medicine and Biology. Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3462-4_9.

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Gachanja, Naomi N., David A. Dorward, Adriano G. Rossi, and Christopher D. Lucas. "Assays of Eosinophil Apoptosis and Phagocytic Uptake." In Methods in Molecular Biology. Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1095-4_10.

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AbstractEosinophil apoptosis (programmed cell death) plays an important role in several inflammatory and allergic conditions. Apoptosis triggers various mechanisms including activation of cysteine-aspartic proteases (caspases) and is characterized by morphological and biochemical changes. These include cellular condensation, nuclear fragmentation, increased mitochondrial permeability with loss of membrane potential, and exposure of phosphatidylserine on the cell membrane. A greater understanding of apoptotic mechanisms, subsequent phagocytosis (efferocytosis), and regulation of these processes is critical to understanding disease pathogenesis and development of potential novel therapeutic agents. Release of soluble factors and alterations to surface marker expression by eosinophils undergoing apoptosis aid them in signaling their presence to the immediate environment, and their subsequent recognition by phagocytic cells such as macrophages. Uptake of apoptotic cells usually suppresses inflammation by restricting proinflammatory responses and promoting anti-inflammatory and tissue repair responses. This, in turn, promotes resolution of inflammation. Defects in the apoptotic or efferocytosis mechanisms perpetuate inflammation, resulting in chronic inflammation and enhanced disease severity. This can be due to increased eosinophil life span or cell necrosis characterized by loss of cell membrane integrity and release of toxic intracellular mediators. In this chapter, we detail some of the key assays that are used to assess eosinophil apoptosis, as well as the intracellular signaling pathways involved and phagocytic clearance of these cells.
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Read, Daniel, James Skinner, Daniel Lock, and Aaron CT Smith. "Therapeutic use exemptions." In WADA, the World Anti-Doping Agency. Routledge, 2021. http://dx.doi.org/10.4324/9781003084297-6-6.

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De Pauw, Ines, Carolien Boeckx, and An Wouters. "Mechanisms of Cetuximab Resistance and How to Overcome It." In Critical Issues in Head and Neck Oncology. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-63234-2_3.

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AbstractDeregulated or increased signalling of the epidermal growth factor receptor (EGFR) plays an integral role in the development of various cancer types, including head and neck squamous cell carcinoma (HNSCC), making it a compelling drug target. However, after initially promising results of EGFR-targeted therapies, such as the monoclonal antibody cetuximab, it became clear that both intrinsic and acquired therapeutic resistance are major roadblocks in the field of personalised cancer treatments.In order to unravel and overcome resistance to cetuximab, at least two strategies can be adopted.Firstly, therapeutic resistance to anti-EGFR therapy may arise from mechanisms that can compensate for reduced EGFR signalling and/or mechanisms that can modulate EGFR-dependent signalling. In this chapter, we discuss which mechanisms of cetuximab resistance are already known and which ones deserve further investigation. This enhanced knowledge will guide us to rationally design and test novel combination therapies that overcome resistance to EGFR-targeting agents in cancer treatment.Secondly, an urgent need remains to develop novel targeted treatments for single-agent or combined therapy use. In this view, due to the particular mode of activation of the EGFR receptor, involving ligand-induced homo- and heterodimerization of the four HER receptors, an increased inhibition scope of HER receptors most likely results in a more potent blockade of the HER network, preventing premature emergence of resistance and leading to a more pronounced therapeutic benefit. We discuss two multitargeted compounds, being MEHD7945A (duligotuzumab) and afatinib, in this chapter.Despite the huge efforts to unravel the molecular landscape of HNSCC, the main clinically validated target remains EGFR. However, immune checkpoints, like programmed cell death protein 1 (PD-1), are gaining clinical approvals as well. We underscore the importance of adopting rational drug combinations to enhance the therapeutic effect of the EGFR-inhibitor cetuximab and highlight the ongoing search for predictive biomarkers, with the ultimate goal of delivering individualized cancer therapy to HNSCC patients.
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Monteillier, Aymeric, and Muriel Cuendet. "The Use of Anti-Inflammatory Agents for Cancer Chemoprevention." In Natural Products for Cancer Chemoprevention. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-39855-2_17.

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Mahmoudian, Mahmoud. "Development of Bioprocesses for the Generation of Anti-Inflammatory, Anti-Viral and Anti-Leukaemic Agents." In Novel Frontiers in the Production of Compounds for Biomedical Use. Springer Netherlands, 2001. http://dx.doi.org/10.1007/0-306-46885-9_15.

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Rainsford, K. D. "Mechanisms of gastrointestinal ulceration from non-steroidal anti-inflammatory drugs: a basis for use and development of protective agents." In Side-Effects of Anti-Inflammatory Drugs 3. Springer Netherlands, 1992. http://dx.doi.org/10.1007/978-94-011-2982-4_14.

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Conference papers on the topic "Anti-inflammatory agents Therapeutic use"

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Porta, R., R. Pescador, R. Niada, M. Mantovani, and G. Prino. "FIBRINOLYTIC POTENCY OF NON ANTICOAGULANT, OXI-EEDUCED SLOW AND FAST MOVING HEPARINS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644180.

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It is well known that heparin is able to induce an increase of fibrinolytic activity when i.v. administered in man and in several animal species. Nevertheless, its anticoagulant properties can cause serious problems of bleeding and this restricts the therapeutic use of heparin. An oxi-reductive process applied to heparin leads to a conpound with reduced anticoagulant activity. Moreover, heparin can be separated into slow moving (SM) and fast moving (FM) components on the basis of electrcphoretic properties. The SM and FM components display quantitatively different biological activities. The ai
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Chen, Kok Hao, and Jong Hyun Choi. "Nanoparticle-Aptamer: An Effective Growth Inhibitor for Human Cancer Cells." In ASME 2009 International Mechanical Engineering Congress and Exposition. ASMEDC, 2009. http://dx.doi.org/10.1115/imece2009-11966.

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Semiconductor nanocrystals have unique optical properties due to quantum confinement effects, and a variety of promising approaches have been devised to interface the nanomaterials with biomolecules for bioimaging and therapeutic applications. Such bio-interface can be facilitated via a DNA template for nanoparticles as oligonucleotides can mediate the aqueous-phase nucleation and capping of semiconductor nanocrystals.[1,2] Here, we report a novel scheme of synthesizing fluorescent nanocrystal quantum dots (NQDs) using DNA aptamers and the use of this biotic/abiotic nanoparticle system for gro
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Mahendran, Rhamiya, Paul Bassin, Mike Cook, Sharon Rossiter, Abigail Martin, and Victoria Hutter. "Late Breaking Abstract - Anti-inflammatory activity of novel transtilbene sulfonamide analogues as potential novel therapeutic agents for inflammatory lung disease." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa2471.

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Arora, Rahul D. "Definition, etiopathogenesis, management and role of flouroquinolone prophylaxis in prevention of spontaneous bacterial peritonitis complicating malignant ascites." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685345.

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Background: Malignancy related ascites encompasses multiple etiologies which include peritoneal carcinomatosis, hepatic synthetic dysfunction due to parenchymal involvement by the tumour, transcoeloemic metastasis and chylous ascites due to lymphatic obstruction. Primary Cancer type, liver metastasis and serum albumin have been listed as independent prognostic markers in malignant ascites. Spontaneous Bacterial Peritonitis is usually seen as a complication of decompensated chronic liver disease due to translocation of bacteria or haematogenous dissemination from a distant focus of infection. T
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Sandri, Monica, Michele Iafisco, Silvia Panseri, Elisa Savini, and Anna Tampieri. "Fully Biodegradable Magnetic Micro-Nanoparticles: A New Platform for Tissue Regeneration and Theranostic." In ASME 2013 2nd Global Congress on NanoEngineering for Medicine and Biology. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/nemb2013-93223.

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Nowadays, magnetic materials are receiving special attention due to their potential applications in different fields and in particular in medicine. Magnetic micro-nano-particles have been progressively employed as support materials for enzyme immobilization, and have been used as drug-delivery vehicles, contrast agents for magnetic resonance imaging as well as heat mediators for hyperthermia-based anti-cancer treatments and many other exciting biomedical applications. Magnetic materials have also attracted a big interest in the field of bone tissue regeneration because it has been demonstrated
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Smith, Katisha D., and Liang Zhu. "Theoretical Evaluation of a Simple Cooling Pad Inducing Hypothermia in the Spinal Cord Following Traumatic Injury." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206190.

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Although significant damage is caused by the mechanics of the traumatic spinal cord injury (SCI), secondary injury that follows is often times even more dangerous. It occurs within the first 12–24 hours following the injury and can last up to 5–10 days, depending on the severity of the injury [1]. Secondary injury causes physiological disturbances that disrupt the body’s homeostasis like initiating a cellular inflammatory response at the injury site and increasing the release of free radicals. An overabundance of free radicals contributes to tissue ischemia, cerebral edema, and disruption of t
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Saeednia, L., and R. Asmatulu. "Methotrexate Loaded Magnetic Nanoparticles as a Targeted Drug Delivery Device." In ASME 2015 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/imece2015-51193.

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Targeted drug delivery systems have been shown to be promising alternative for the conventional drug delivery methods. Among numerous nanocarriers developed for therapeutic applications, iron oxide magnetic nanoparticles have attracted considerable attention. Fe3O4 (magnetite) is one of the most commonly used iron oxide in biomedical applications due to its biocompatibility and can be easily produced in research and industrial laboratories. The core/shell structure of magnetic nanoparticles allows the surface coating to avoid their agglomeration. Moreover, coating of Fe3O4 nanoparticles provid
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Kurup, Sonali, Pavlina Liskova, Paiboon Jungsuwadee, Prashant Sakharkar, and Soniya Tambe. "Abstract 589: A systematic review of randomized clinical trials for therapeutic outcomes of anti-EGFR agents used in combination with other targeted therapies." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-589.

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Al-Ansari, Dana E., Nura A. Mohamed, Isra Marei, Huseyin Yalcin, and Haissam Abou-Saleh. "Assessment of Metal Organic Framework as Potential Drug Carriers in Cardiovascular Diseases." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0127.

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Abstract:
Background: Cardiovascular diseases (CVDs) are considered the major cause of death worldwide. Therapeutic delivery to the cardiovascular system may play an important role in the successful treatment of a variety of CVDs, including atherosclerosis, ischemic-reperfusion injury, and microvascular diseases. Despite their clinical benefits, current therapeutic drugs are hindered by their short half-life and systemic side effects. This limitation could be overcome using controlled drug release with the potential for targeted drug delivery using a nanomedicine approach. In the current study, we have
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10

Danesh, B. J. Z., A. R. Saniabadi, R. I. Russell, G. D. O. Lowe, and C. D. Forbes. "COMPARISON OF THE EFFECT OF ASPIRIN (ASA) AND CHOLINE MAGNESIUM TRISALICYLATE (CMT) ON PLATELET AGGREGATION IN WHOLE BLOOD EX-VIVO." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644866.

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Abstract:
Suppression of platelet aggregation by ASA limits the therapeutic use of this drug as an analgesic in patients with bleeding tendencies. CMT is a non-acetylated salicylate derivative with analgesic and anti-inflammatory effect similar to that of ASA. We compared platelet aggregation in human whole blood ex-vivo, three hours after ingestion of ASA and. CMT. Using a whole blood platelet counter, platelet aggregation was quantified by measuring the fall in the number of single platelets at peak aggregation in response to collagen (lμg/ml) arachidonic acid (AA, 0.5 mM) as well as spontaneous aggre
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