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Relevant bibliographies by topics / Anti-inflammatory signaling

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Academic literature on the topic 'Anti-inflammatory signaling'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Anti-inflammatory signaling"

1

Meyer, Urs. "Anti-inflammatory signaling in schizophrenia." Brain, Behavior, and Immunity 25, no. 8 (2011): 1507–18. http://dx.doi.org/10.1016/j.bbi.2011.05.014.

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Siegemund, Sabine, and Karsten Sauer. "Balancing pro- and anti-inflammatory TLR4 signaling." Nature Immunology 13, no. 11 (2012): 1031–33. http://dx.doi.org/10.1038/ni.2452.

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Yeung, Yiu To, Faisal Aziz, Angelica Guerrero-Castilla, and Sandro Arguelles. "Signaling Pathways in Inflammation and Anti-inflammatory Therapies." Current Pharmaceutical Design 24, no. 14 (2018): 1449–84. http://dx.doi.org/10.2174/1381612824666180327165604.

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During the past decade, an abundance of new evidence highlighted the importance of inflammation in the development of chronic pathologies such as neurodegeneration, cancer, diabetes, cardiovascular disease and inflammatory bowel disease. However, most of the current therapies do not address the underlying problem and better therapies are urgently needed. A growing number of researchers have discovered various signaling pathways that are associated with the initiation and progression of inflammation. Among different pathways, we will focus on three classical inflammatory pathways: p38 MAPK, IL-

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Cui, Taixing, Francisco J. Schopfer, Jifeng Zhang, et al. "Nitrated Fatty Acids: Endogenous Anti-inflammatory Signaling Mediators." Journal of Biological Chemistry 281, no. 47 (2006): 35686–98. http://dx.doi.org/10.1074/jbc.m603357200.

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Yoon, Sonia S., and Jun Sun. "Probiotics, Nuclear Receptor Signaling, and Anti-Inflammatory Pathways." Gastroenterology Research and Practice 2011 (2011): 1–16. http://dx.doi.org/10.1155/2011/971938.

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There is increased investigation of the human microbiome as it relates to health and disease. Dysbiosis is implicated in various clinical conditions including inflammatory bowel disease (IBD). Probiotics have been explored as a potential treatment for IBD and other diseases. The mechanism of action for probiotics has yet to be fully elucidated. This paper discusses novel mechanisms of action for probiotics involving anti-inflammatory signaling pathways. We highlight recent progress in probiotics and nuclear receptor signaling, such as peroxisome-proliferator-activated receptor gamma (PPARγ) an

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Groeger, A. L., and B. A. Freeman. "Signaling Actions of Electrophiles: Anti-inflammatory Therapeutic Candidates." Molecular Interventions 10, no. 1 (2010): 39–50. http://dx.doi.org/10.1124/mi.10.1.7.

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Ge, Yun-jun, Qi-wen Liao, Ye-chun Xu, Qiang Zhao, Bei-li Wu, and Richard D. Ye. "Anti-inflammatory signaling through G protein-coupled receptors." Acta Pharmacologica Sinica 41, no. 12 (2020): 1531–38. http://dx.doi.org/10.1038/s41401-020-00523-1.

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Landis, R. Clive, Pandelis Philippidis, Jan Domin, Joseph J. Boyle, and Dorian O. Haskard. "Haptoglobin Genotype-Dependent Anti-Inflammatory Signaling in CD163+Macrophages." International Journal of Inflammation 2013 (2013): 1–7. http://dx.doi.org/10.1155/2013/980327.

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Intraplaque hemorrhage causes adaptive remodelling of macrophages towards a protective phenotype specialized towards handling iron and lipid overload, denoted Mhem. The Mhem phenotype expresses elevated levels of hemoglobin (Hb) scavenger receptor, CD163, capable of endocytosing pro-oxidant free Hb complexed to acute phase protein haptoglobin (Hp). It is notable that individuals homozygous for the Hp 2 allele (a poorer antioxidant) are at increased risk of cardiovascular disease compared to the Hp 1 allele. In this study, we examined whether scavenging of polymorphic Hp:Hb complexes differenti

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Surh, Young-Joon. "Redox modulation of pro-inflammatory and anti-inflammatory signaling by chemopreventive phytochemicals." Free Radical Biology and Medicine 53 (September 2012): S7—S8. http://dx.doi.org/10.1016/j.freeradbiomed.2012.08.083.

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Ene, Corina-Daniela, Mircea Penescu, Ilinca Nicolae, and Emanoil Ceauşu. "Anti-inflammatory effects of nicotine." Romanian Journal of Infectious Diseases 18, no. 1 (2015): 5–12. http://dx.doi.org/10.37897/rjid.2015.1.1.

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A number of epidemiological and experimental data have shown that nicotine stimulates anti-inflammatory response in extraneuronal tissues, response mediated by homopentameric nicotinic receptors, 7nAChR- alpha. The receptors are located mainly on immune and inflammatory cells. These pharmacological mechanisms, through which inflammatory process could be adjusted is the use of selective nicotinic agonists and antagonists, called anti-inflammatory. Treatment with nicotine initiates a series of intracellular events associated with stimulation of several signaling pathways: NFkB, JAK/STAT, MyD88/T

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Dissertations / Theses on the topic "Anti-inflammatory signaling"

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Vale, Duarte Pereira Claudia Patricia. "Inflammatory Wnt5A signaling in macrophages as novel target for anti-inflammatory action of activated protein C /." [S.l.] : [s.n.], 2009. http://opac.nebis.ch/cgi-bin/showAbstract.pl?sys=000278496.

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Parthasarathy, Upasana. "Identifying epitopes of anti-FcaRI monoclonal antibodies on FcaRI ectodomain that trigger the anti-inflammatory ITAMi signaling pathway." University of Cincinnati / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1418910350.

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Rossato, Elisabetta. "Anti-inflammatory role of monomeric IgA through the inhibitory FcαRI-ITAMi signaling in rheumatoid arthritis". Paris 7, 2014. http://www.theses.fr/2014PA077056.

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De par leur hétérogénéité, les IgA possèdent des propriétés pro- ou anti-inflammatoires et contribuent à l'homéostasie immunitaire. Ces activités dépendent de l'interaction des IgA avec le récepteur RFcal contenant le motif ITAM (immunoreceptor tyrosine-based motif). Les IgA sous forme de complexes entraînent une activation pro-inflammatoire, tandis qu'en l'absence d'antigène, celles-ci (ou le fragment Fab d'un anticorps anti-RFcat) engendrent un signal anti-inflammatoire (nommé ITAMi ou ITAM inhibiteur) capable d'inhiber l'activation des récepteurs hétérologues. L'ITAMi nécessite, au niveau d

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Gatbonton-Schwager, Tonibelle N. "Biological and Chemical Analysis of Small Molecule Activators of Anti-inflammatory and Antioxidant Nrf2-Keap1 Signaling." Case Western Reserve University School of Graduate Studies / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=case1390560628.

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Davarya, Shekar Ligia. "Inflammatory and Thrombotic Responses to Microbial Products in Fetal Vessels Are Mediated through Divergent Toll-Like Receptor Signaling Pathways: Implications in Fetal Inflammatory Response Syndrome." Yale University, 2008. http://ymtdl.med.yale.edu/theses/available/etd-08072007-132134/.

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Placental vessels and the umbilical circulatory network function to carry oxygen and nutrients to the fetus. It is at this level that placental lesions such as villitis, obliterative vasculopathy, and thrombotic vasculopathy have been observed in association with fetal inflammatory response syndrome (FIRS) and cerebral palsy. We used human umbilical vein endothelial cells (HUVECs) as a model to study the regulation of inflammation and thrombosis in fetal vessels by microbial products. In this thesis we measured interleukin-8 (IL-8) and tissue factor (TF) expression by HUVECs treated with lipop

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Ma, Loretta T. K. "PKA Signaling in ABCA1 Function: A Role in Modulation of Cholesterol Efflux and Macrophage Inflammation." Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/26272.

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Formation of lipid-laden macrophage foam cells and inflammation are the central components in the initiation and progression of atherosclerosis. ABCA1 is well established as an anti-atherogenic factor that facilitates cellular cholesterol and phospholipid efflux, promotes reverse cholesterol transport, and suppresses pro-inflammatory cytokine secretion. Through these functions, ABCA1 is capable of reducing the lipid burden in atherosclerotic plaque. PKA signaling is an integral factor in promoting many anti-atherogenic functions of ABCA1; however, mechanistic aspects of PKA signaling associate

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Loveridge, Carolyn J. "The effects of nonsteroidal anti-inflammatory drugs on nuclear factor-kappaB and Beta-catenin signaling in colorectal cancer cells." Thesis, University of Edinburgh, 2007. http://hdl.handle.net/1842/29229.

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The first research strand in this thesis was to determine whether non-aspirin NSAIDs induce apoptosis of colorectal cancer cells via modulation of the NF-κB pathway and to elucidate the upstream mechanisms involved. Experimental evidence presented here shows that sulindac, sulindac sulfone and indomethacin cause NF-κB pathway activation. It was found that non-aspirin NSAIDs induce nucleolar translocation of the NF-κB component, RelA, repression of NF-κB driven transcriptional activity and apoptosis. Furthermore, in a similar manner to aspirin, nucleolar translocation of Re1A was demonstrated t

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Pangburn, Heather Ann. "Effects of the non-steroidal anti-inflammatory drug (NSAID) sulindac on epidermal growth factor receptor (EGFR) expression and signaling in colorectal cancer /." Connect to full text via ProQuest. Limited to UCD Anschutz Medical Campus, 2007.

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Thesis (Ph.D. in Toxicology) -- University of Colorado Denver, 2007.<br>Typescript. Includes bibliographical references (leaves 156-176). Free to UCD affiliates. Online version available via ProQuest Digital Dissertations;

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Konarski, Yulia. "SHP-1/ Src Complex is a Master Regulator of the IL-12/IL-23 pro- and IL-10/IL-27 Anti-inflammatory Axis in TLR4-activated Signaling Pathways in Human Monocytes and Macrophages." Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/25999.

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Although the etiology surrounding many autoimmune diseases remains unknown, the underlying characteristic of many of these diseases is a disruption in the balance of pro- and anti- inflammatory cytokines It is well established that the dysregulation of the IL-12 family of cytokines, an increase in IL-12/IL-23 and a decrease in IL-27 production has been implicated in these conditions. We used ELISA, RT-PCR, Immunofluorescence and Western immunoblotting in conjunction with pharmalogical inhibitors and siRNA to demonstrate the role of SHP-1/Src in the regulation of IL-12, IL-23, IL-27 and IL-10 i

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Kaci, Ghalia. "Caractérisation des propriétés anti-inflammatoires de souches commensales de Streptococcus salivarius." Phd thesis, Université Paris Sud - Paris XI, 2012. http://tel.archives-ouvertes.fr/tel-01069898.

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Les bactéries commensales digestives jouent un rôle primordial dans l'homéostasie épithéliale et la santé de l'hôte, avec notamment un rôle modulateur du système immunitaire. Des effets bénéfiques dans le traitement des pathologies inflammatoires intestinales ont été caractérisés chez certaines souches de bactéries commensales. La compréhension de ces effets dans le maintien de l'homéostasie intestinale repose sur la connaissance des interactions entre les bactéries, l'épithélium intestinal et le système immunitaire muqueux. Streptococcus salivarius est l'un des premiers colonisateurs de la ca

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Books on the topic "Anti-inflammatory signaling"

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Ferran, Christiane. The Multiple Therapeutic Targets of A20. Springer, 2014.

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Ferran, Christiane. The Multiple Therapeutic Targets of A20. Springer, 2016.

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Fleischmann, Roy. Signalling pathway inhibitors. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0081.

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Oral, small-molecule signalling pathway inhibitors, including ones that inhibit the JAK and SyK pathways, are currently in development for the treatment of rheumatoid arthritis (RA). Tofacitinib is an orally administered small-molecule inhibitor that targets the intracellular Janus kinase 3 and 1 (JAK1/3) molecules to a greater extent than JAK2 while baricitinib (formerly INCB028050) predominantly inhibits JAK1/2. Many of the proinflammatory cytokines implicated in the pathogenesis of RA utilize cell signalling that involves the JAK-STAT pathways and therefore inhibition of JAK-STAT signalling

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Siebert, Stefan, Sengupta Raj, and Alexander Tsoukas. Drug treatment for axial spondyloarthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198755296.003.0015.

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The pharmacological therapy for patients with axial spondyloarthritis (axSpA), especially those with severe disease, has been transformed by the introduction of the biologic therapies, and anti-TNF therapy in particular. Until approximately 2005, treatment options for ankylosing spondylitis (AS) were limited to exercise therapy and non-steroidal anti-inflammatory drugs (NSAIDs). The TNF inhibitors appear to have a good safety profile in axSpA, with no new safety signals. The high cost of innovator biologics remains an issue and it will be interesting to observe the effect of the introduction o

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Endlich, Karlhans, and Rodger Loutzenhiser. Regulation of vasomotor tone in the afferent and efferent arterioles. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0208.

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Pre-glomerular vessels are regulated by membrane potential alterations affecting the activity of L-type voltage-activated Ca2+ channels; whereas voltage-independent mechanisms regulate the efferent arteriole, notably influenced by angiotensin II and therefore by angiotensin converting enzyme inhibitors, and by non-steroidal anti-inflammatory drugs.These properties underlie the physiologic control of glomerular capillary pressure, for example, by prostaglandin E2, during conditions of reduced renal perfusion and the stabilization of glomerular capillary pressure when the kidney is exposed to pr

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Book chapters on the topic "Anti-inflammatory signaling"

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Basu, Manju, Subhash Basu, and Michael Pugia. "Uristatin Anti-inflammatory Cellular Signaling." In Inflammatory Pathways in Diabetes. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-21927-1_10.

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Rego, Stephen L., Tian McCann, and Didier Dréau. "Pro- and Anti-Inflammatory Cytokine Signaling within 3D Tissue Models." In Engineering 3D Tissue Test Systems. CRC Press, 2017. http://dx.doi.org/10.1201/9781315118260-12.

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Rutherford, Claire, and Timothy M. Palmer. "Molecular Basis of Protective Anti-Inflammatory Signalling by Cyclic AMP in the Vascular Endothelium." In Systems Biology for Signaling Networks. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-5797-9_23.

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Gachanja, Naomi N., David A. Dorward, Adriano G. Rossi, and Christopher D. Lucas. "Assays of Eosinophil Apoptosis and Phagocytic Uptake." In Methods in Molecular Biology. Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1095-4_10.

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AbstractEosinophil apoptosis (programmed cell death) plays an important role in several inflammatory and allergic conditions. Apoptosis triggers various mechanisms including activation of cysteine-aspartic proteases (caspases) and is characterized by morphological and biochemical changes. These include cellular condensation, nuclear fragmentation, increased mitochondrial permeability with loss of membrane potential, and exposure of phosphatidylserine on the cell membrane. A greater understanding of apoptotic mechanisms, subsequent phagocytosis (efferocytosis), and regulation of these processes is critical to understanding disease pathogenesis and development of potential novel therapeutic agents. Release of soluble factors and alterations to surface marker expression by eosinophils undergoing apoptosis aid them in signaling their presence to the immediate environment, and their subsequent recognition by phagocytic cells such as macrophages. Uptake of apoptotic cells usually suppresses inflammation by restricting proinflammatory responses and promoting anti-inflammatory and tissue repair responses. This, in turn, promotes resolution of inflammation. Defects in the apoptotic or efferocytosis mechanisms perpetuate inflammation, resulting in chronic inflammation and enhanced disease severity. This can be due to increased eosinophil life span or cell necrosis characterized by loss of cell membrane integrity and release of toxic intracellular mediators. In this chapter, we detail some of the key assays that are used to assess eosinophil apoptosis, as well as the intracellular signaling pathways involved and phagocytic clearance of these cells.

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Bazan, Nicolas G. "Neurotrophins Induce Neuroprotective Signaling in the Retinal Pigment Epithelial Cell by Activating the Synthesis nl of the Anti-inflammatory and Anti-apoptotic Neuroprotectin D1." In Advances in Experimental Medicine and Biology. Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-74904-4_3.

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Skaper, Stephen D. "Mast Cells and Glia as Targets for the Anandamide Congener Palmitoylethanolamide: an Anti-inflammatory and Neuroprotective Lipid Signaling Molecule." In Endocannabinoids and Lipid Mediators in Brain Functions. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-57371-7_12.

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Na, Hye-Kyung, and Young-Joon Surh. "Nutrigenomic Perspectives on Cancer Chemoprevention with Anti-Inflammatory and Antioxidant Phytochemicals: NF-κB and Nrf2 Signaling Pathways as Potential Targets." In Genomics, Proteomics, and Metabolomics in Nutraceuticals and Functional Foods. Wiley-Blackwell, 2010. http://dx.doi.org/10.1002/9780813821474.ch12.

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Shin, Woen-Bin, Xin Dong, Yon-Suk Kim, et al. "Anti-inflammatory Effects of Batillaria multiformis Water Extracts via NF-кB and MAPK Signaling Pathways in LPS-Induced RAW 264.7 Cells." In Advances in Experimental Medicine and Biology. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-8023-5_83.

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Paik, Jihye, Joo Y. Lee, and Daniel Hwang. "Signaling Pathways for Tnfa-Induced Cox-2 Expression: Mediation Through Map Kinases And Nfkb, And Inhibition By Certain Nonsteroidal Anti-Inflammatory Drugs." In Advances in Experimental Medicine and Biology. Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-0193-0_77.

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Silveira, Vivian Thaise da, Eduardo Candelario-Jalil, and Antonio Carlos Pinheiro de Oliveira. "Immunoneuropharmacology." In Immunopsychiatry. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190884468.003.0002.

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This chapter presents an overview of the current literature on the pathophysiological mechanisms of inflammatory mediators in neuroimmune modulation, as well as pharmacological strategies in inflammatory signaling pathways in the central nervous system (CNS). Increasing attention has been paid to the importance of brain–immune interaction for the maintenance of brain homeostasis. If immune activation persists, inflammatory mediators and their signaling pathways can influence neurons/neuronal circuits, leading to mood, cognitive, and behavioral impairment. Therefore, immune dysregulation can play a role in the pathophysiology of psychiatric disorders through its direct and indirect ability to alter the synthesis, reuptake, and release of multiple neurotransmitters, and to induce oxidative stress, mitochondrial dysfunction, cytoskeletal disruption, and cytotoxic lipid peroxidation, among other effects. In this sense, inflammatory mediators and their signaling pathway are currently regarded as attractive targets for promising therapeutic and preventative therapeutic strategies in clinical psychiatry, especially for patients who are resistant to treatment and exhibit inflammatory phenotypes. The efficacy and precision of anti-inflammatory treatments can be further tuned, and they depend on the methodologically rigorous design of future clinical trials.

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Conference papers on the topic "Anti-inflammatory signaling"

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Titova, Olga, Natalya Kuzubova, and Elena Lebedeva. "Anti-inflammatory effect of hypoxic signaling suppression in the severe COPD model." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa4269.

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Chen, C. T., S. Park, M. Bhargava, and P. A. Torzilli. "Inhibitory Effect of Mechanical Load on IL-1 Induced Cartilage Degradation Is Mediated by Interferon-Gamma and IL-1 Receptor 1." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193230.

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Matrix remodeling in articular cartilage is regulated by the elevation and activation of aggrecanases (ADAMTS-4 and ADAMTS-5) and matrix metalloproteinases (MMPs) [2–4, 7–9, 10]. Several recent studies from our and other groups have shown that mechanical loading can counteract interleukin 1 (IL-1) induced pro-inflammatory and catabolic events by down-regulating aggrecanases, MMPs, and pro-inflammatory genes [1, 3, 5, 6], but the molecular mechanism is not clear. Many previous studies have shown that the regulation of pro-inflammatory effect of IL-1 come from several aspects: anti-inflammatory

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Surh, Young-Joon. "Abstract CN07-01: Modulation of proinflammatory and anti-inflammatory signaling for cancer chemoprevention." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Oct 22-25, 2011; Boston, MA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1940-6207.prev-11-cn07-01.

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Carneiro de Oliveira Jr, Manoel, Alana dos Santos Dias, Nicole Cristine Rigonato Oliveira, et al. "The anti-inflammatory effects of aerobic exercise in asthma is mediated by purinergic signaling." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.pa698.

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Agarwal, Chapla, Komal Raina та Rajesh Agarwal. "Abstract 5692: Anti-inflammatory effects of silibinin in colorectal cancer: Targeting the activation of NF-κB signaling". У Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-5692.

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Gapeyev, A. B. "The role of some signaling and effector systems in realization of anti-inflammatory action of extremely high-frequency electromagnetic radiation." In 2010 20th International Crimean Conference "Microwave & Telecommunication Technology" (CriMiCo 2010). IEEE, 2010. http://dx.doi.org/10.1109/crmico.2010.5632439.

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Fuchs, CD, V. Mlitz, M. Tardelli, et al. "Absence of BSEP (ABCB11) protects MDR2 (ABCB4) KO mice from cholestatic liver and bile duct injury through anti-inflammatory bile acid composition and signaling." In 52. Jahrestagung & 30. Fortbildungskurs der Österreichischen Gesellschaft für Gastroenterologie & Hepatologie (ÖGGH). Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1691922.

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Kuo, Chieh-Ti, Kristen Stoner, Yi-Wen Huang, et al. "Abstract 816: Anti-inflammatory effects of black raspberries in ulcerative colitis are associated with demethylation of genes in the Wnt signaling and protective modulation of toll-like receptor pathway." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-816.

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O'Malley, Grace, Kevin Lynch, Serika Naicker, et al. "Abstract 2693: Inflammatory signalling in the colon tumour microenvironment enhances stromal cell mediated suppression of anti-tumour immune responses." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-2693.

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Wang, PS, JC Chou, S. Hu, and SW Wang. "PO-320 Anti-inflammatory effects of gossypol on human lymphocytic jurkat cells via regulation of MAPK signalling and cell cycle." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.833.

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