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Relevant bibliographies by topics / Anti ischemic

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Academic literature on the topic 'Anti ischemic'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Anti ischemic"

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Sanz, E., D. Garcia Dorado, J. Oliveras, et al. "Dissociation between anti-infarct effect and anti-edema effect of ischemic preconditioning." American Journal of Physiology-Heart and Circulatory Physiology 268, no. 1 (1995): H233—H241. http://dx.doi.org/10.1152/ajpheart.1995.268.1.h233.

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This study tested the hypothesis that preconditioning, by reducing catabolite accumulation during ischemia, reduces osmotic swelling and myocardial necrosis during subsequent reperfusion. Farm pigs were randomly allocated to one of three groups of treatment: a control group undergoing a 48-min coronary occlusion (CO) of the middle left anterior descending artery, a preconditioned group (2 cycles of 5-min CO and 5-min reperfusion before the 48-min CO), or an intracoronary perfusion group receiving a substrate-free anoxic buffer perfusion into the area at risk between minutes 5 and 10 of the pro

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Zeng, Xian-Si, Wen-Shuo Geng, Lei Chen, and Jin-Jing Jia. "Thioredoxin as a Therapeutic Target in Cerebral Ischemia." Current Pharmaceutical Design 24, no. 25 (2018): 2986–92. http://dx.doi.org/10.2174/1381612824666180820143853.

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Background: Cerebral ischemia is a common cause of disability and death. Ischemic brain injury results from complex pathological processes, including oxidative stress, inflammation, and apoptosis. Thioredoxin( Trx) is an important multifunctional protein, which regulates cellular redox status. Increasing studies have demonstrated that Trx provides a neuroprotective role against cerebral ischemia-induced injury. Methods: A systematic search of PMC and the PubMed Database was conducted to summarize the protective effects of Trx against cerebral ischemia. Results: This article reviews the underst

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Bertelmann, Thomas, Hans Ulrich Frank, Hendrik Ansgar Fuchs, and Nicolas Feltgen. "Branch Retinal Vein Occlusion, Macular Ischemia, and Intravitreal Anti-VEGF Therapy." Case Reports in Ophthalmology 8, no. 1 (2017): 271–78. http://dx.doi.org/10.1159/000475520.

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Purpose: To report a case with ischemic macular edema (ME) due to an acute branch retinal vein occlusion (BRVO) which was treated with repeated intravitreal anti-VEGF injections. Methods: Retrospective case presentation. Results: A 66-year-old female patient was treated with repeated intravitreal anti-VEGF injections due to ischemic ME following an acute BRVO. Over a period of 2.5 years best corrected visual acuity increased from 0.06 to 0.6 (decimal notation) accompanied by a reduction in central retinal thickness from 546 to 292 µm. Overall 17 anti-VEGF injections were administered to treat

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Pluta, Ryszard, Sławomir Januszewski, and Stanisław J. Czuczwar. "Myricetin as a Promising Molecule for the Treatment of Post-Ischemic Brain Neurodegeneration." Nutrients 13, no. 2 (2021): 342. http://dx.doi.org/10.3390/nu13020342.

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The available drug therapy for post-ischemic neurodegeneration of the brain is symptomatic. This review provides an evaluation of possible dietary therapy for post-ischemic neurodegeneration with myricetin. The purpose of this review was to provide a comprehensive overview of what scientists have done regarding the benefits of myricetin in post-ischemic neurodegeneration. The data in this article contribute to a better understanding of the potential benefits of myricetin in the treatment of post-ischemic brain neurodegeneration, and inform physicians, scientists and patients, as well as their

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Pluta, Ryszard, Barbara Miziak, and Stanisław J. Czuczwar. "Apitherapy in Post-Ischemic Brain Neurodegeneration of Alzheimer’s Disease Proteinopathy: Focus on Honey and Its Flavonoids and Phenolic Acids." Molecules 28, no. 15 (2023): 5624. http://dx.doi.org/10.3390/molecules28155624.

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Neurodegeneration of the brain after ischemia is a major cause of severe, long-term disability, dementia, and mortality, which is a global problem. These phenomena are attributed to excitotoxicity, changes in the blood–brain barrier, neuroinflammation, oxidative stress, vasoconstriction, cerebral amyloid angiopathy, amyloid plaques, neurofibrillary tangles, and ultimately neuronal death. In addition, genetic factors such as post-ischemic changes in genetic programming in the expression of amyloid protein precursor, β-secretase, presenilin-1 and -2, and tau protein play an important role in the

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Khabibulina, M., D. Elistratov, and M. Shamilov. "Anti-ischemic and cardiometabolic therapy in cardiovascular pathology." Vrach 35, no. 4 (2024): 47–50. http://dx.doi.org/10.29296/25877305-2024-04-09.

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The purpose of this study was to study the effect of Kardioton therapy on myocardial ischemia in patients with arterial hypertension. The drug Kardioton is a natural cardioprotector; strengthens the heart muscle by improving its oxygen supply, stops inflammatory processes in the myocardium; has anti-ischemic, cardiometabolic, hypotensive effect. It is recommended for disorders of the cardiovascular system, with increased physical activity. Based on the conducted research, it can be concluded that Kardioton is well tolerated by patients and is an effective treatment: cardiometabolic and anti-is

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Lin, Ming-Cheng, Chien-Chi Liu, Yu-Chen Lin, and Chin-Sheng Liao. "Resveratrol Protects against Cerebral Ischemic Injury via Restraining Lipid Peroxidation, Transition Elements, and Toxic Metal Levels, but Enhancing Anti-Oxidant Activity." Antioxidants 10, no. 10 (2021): 1515. http://dx.doi.org/10.3390/antiox10101515.

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Cerebral ischemia is related to increased oxidative stress. Resveratrol displays anti-oxidant and anti-inflammatory properties. The transition elements iron (Fe) and copper (Cu) are indispensable for the brain but overload is deleterious to brain function. Aluminum (Al) and arsenic (As) are toxic metals that seriously threaten brain health. This study was conducted to elucidate the correlation of the neuroprotective mechanism of resveratrol to protect cerebral ischemic damage with modulation of the levels of lipid peroxidation, anti-oxidants, transition elements, and toxic metals. Experimental

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Baranova, Ksenia, Natalia Nalivaeva, and Elena Rybnikova. "Neuroadaptive Biochemical Mechanisms of Remote Ischemic Conditioning." International Journal of Molecular Sciences 24, no. 23 (2023): 17032. http://dx.doi.org/10.3390/ijms242317032.

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This review summarizes the currently known biochemical neuroadaptive mechanisms of remote ischemic conditioning. In particular, it focuses on the significance of the pro-adaptive effects of remote ischemic conditioning which allow for the prevention of the neurological and cognitive impairments associated with hippocampal dysregulation after brain damage. The neuroimmunohumoral pathway transmitting a conditioning stimulus, as well as the molecular basis of the early and delayed phases of neuroprotection, including anti-apoptotic, anti-oxidant, and anti-inflammatory components, are also outline

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Pluta, Ryszard, Marzena Ułamek-Kozioł, and Stanisław Czuczwar. "Neuroprotective and Neurological/Cognitive Enhancement Effects of Curcumin after Brain Ischemia Injury with Alzheimer’s Disease Phenotype." International Journal of Molecular Sciences 19, no. 12 (2018): 4002. http://dx.doi.org/10.3390/ijms19124002.

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In recent years, ongoing interest in ischemic brain injury research has provided data showing that ischemic episodes are involved in the development of Alzheimer’s disease-like neuropathology. Brain ischemia is the second naturally occurring neuropathology, such as Alzheimer’s disease, which causes the death of neurons in the CA1 region of the hippocampus. In addition, brain ischemia was considered the most effective predictor of the development of full-blown dementia of Alzheimer’s disease phenotype with a debilitating effect on the patient. Recent knowledge on the activation of Alzheimer’s d

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LAWSON, CLIVE S., and DAVID J. HEARSE. "Ischemic Preconditioning against Arrhythmias: An Anti-Arrhythmic or an Anti-Ischemic Phenomenon?" Annals of the New York Academy of Sciences 723, no. 1 (1994): 138–57. http://dx.doi.org/10.1111/j.1749-6632.1994.tb36723.x.

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Dissertations / Theses on the topic "Anti ischemic"

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Chunder, Rittika [Verfasser]. "Anti-neuronal autoantibodies in ischemic stroke and in anti-MAG IgM Gammopathy / Rittika Chunder." Lübeck : Zentrale Hochschulbibliothek Lübeck, 2018. http://d-nb.info/1153437724/34.

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Yusof, Mozow. "Antecedent hydrogen sulfide elicits an anti-inflammatory phenotype in postischemic murine small intestine." Diss., Columbia, Mo. : University of Missouri-Columbia, 2007. http://hdl.handle.net/10355/4779.

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Thesis (Ph. D.)--University of Missouri-Columbia, 2007.<br>The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Vita. Includes bibliographical references.

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Raghavan, Aparna. "Neuroprotective Potential of Withania Somnifera in Cerebral Ischemia." University of Toledo Health Science Campus / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=mco1416570371.

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Parikh, Suchi Vipin. "Ependymin peptide mimetics that assuage ischemic damage increase gene expression of the anti-oxidative enzyme SOD." Link to electronic thesis, 2003. http://www.wpi.edu/Pubs/ETD/Available/etd-0429103-132144.

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Li, Xiaofei. "Improving Stem Cell Survival and Differentiation in Ischemic and Inflammatory Tissues." The Ohio State University, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=osu1469174124.

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Inagaki, Koichi. "Anti-Ischemic Effect of a Novel Cardioprotective Agent JTV519 Is Mediated Through Specific Activation of δ-Isoform of Protein Kinase C in Rat Ventricular Myocardium". Kyoto University, 2000. http://hdl.handle.net/2433/180830.

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Richard, Sébastien. "Explorations des fonctions plaquettaires exposées à l'aspirine au décours de l'accident vasculaire cérébral ischémique." Thesis, Nancy 1, 2011. http://www.theses.fr/2011NAN10132.

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L'aspirine est l'anti-plaquettaire le plus largement prescrit à la phase aiguë de l'accident vasculaire cérébral (AVC) ischémique. Cependant, la survenue de récidives, malgré cette prescription, est fréquente. La description de l'effet de l'aspirine sur l'activité plaquettaire durant cette phase n'a jamais été réalisée. Elle pourrait mettre en évidence une moindre réponse plaquettaire et aider à établir de nouvelles stratégies thérapeutiques. Cinquante patients, ont reçu par voie orale 300 mg d'aspirine, suite à un AVC ischémique. Ensuite, des prélèvements sanguins ont été réalisés : entre 2 e

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Hoxha, M. "THE POTENTIAL THERAPEUTIC ROLE OF MONTELUKAST AND NEW HYBRID AGENTS, TXA2 ANTAGONIST-COX-2 INHIBITORS IN CARDIOVASCULAR EVENTS." Doctoral thesis, Università degli Studi di Milano, 2016. http://hdl.handle.net/2434/347148.

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The main target of my PhD research project was to explore new alternatives to reduce the cardiovascular (CV) risk targeting the arachidonic acid metabolites. Hence, I have been part of two different projects, one studying multitarget compounds with balanced COXIB and TP receptor antagonist properties, and the other evaluating the potential role of a leukotriene (LT) antagonist drug such as montelukast in improving the CV outcome. In reference to the first project, new multitarget compounds were synthesized at the University of Turin, by substituting the carboxylic function of Lumiracoxib.

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Huang, Zhigao. "Implication of anti-apoptotic genes in neuronal death following focal cerebral ischemia in rats." Thesis, University of Ottawa (Canada), 2002. http://hdl.handle.net/10393/6270.

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Accumulating biochemical and morphological evidence suggests that apoptosis contributes to neuronal cell death following cerebral ischemia. Recent research which has examined changes in expression of proapoptotic proteins has further strengthened the important role of apoptosis in ischemic cell death. In this thesis I first addressed the role of apoptosis in ischemic death by examining p53, which is itself a complex multifunctional tumor suppresser gene, following transient focal ischemia. Of particular note were the alterations of the anti-apoptosic gene, naip, that were observed under stress

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Bouma, Maarten Godfried. "The anti-inflammatory potential of adenosine an experimental study with emphasis on ischemia-reperfusion injury /." Maastricht : Maastricht : UPM, Universitaire Pers Maastricht ; University Library, Maastricht University [Host], 1997. http://arno.unimaas.nl/show.cgi?fid=5945.

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Books on the topic "Anti ischemic"

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1946-, Frishman William H., and Weiner Donald A. 1944-, eds. Cardiovascular pharmacotherapy I: Anti-ischemic drugs. W.B. Saunders, 1988.

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Zoppo, Gregory J. Del. Innate inflammation and stroke. Edited by New York Academy of Sciences. Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2010.

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1946-, Feuerstein Giora Z., ed. Inflammation and stroke. Birkhäuser, 2001.

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M, Evans J., ed. Potassium channels and their modulators: From synthesis to clinical experience. Taylor & Francis, 1996.

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AlJaroudi, Wael. Risk Assessment in Acute Coronary Syndromes. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0013.

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Acute coronary syndromes (ACS) include unstable angina pectoris (UAP), non-ST elevation (NSTEMI), and ST elevation acute myocardial infarction (STEMI). Each year, more than 2 million people are hospitalized with ACS in the United States. The initial treatment has evolved over the last few decades from conservative management to early reperfusion therapy. Medical therapy has also significantly changed with the use of newer more potent antiplatelet agents, beta-blockers, angiotensin converting enzyme inhibitors, statins, and anti-anginal drugs, which have resulted in improvement of patient care

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Ahuja, Christopher S., and Michael Fehlings. Neuroprotection for Spinal Cord Injury. Edited by David L. Reich, Stephan Mayer, and Suzan Uysal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190280253.003.0015.

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Traumatic spinal cord injuries (SCI) often have a devastating impact on quality of life for patients and their families. Neuroprotection for spinal cord injury is aimed at improving functional outcomes by limiting secondary injury processes that occur within the first minutes, hours, and days following the primary injury. The primary mechanical trauma initiates a secondary injury cascade where ischemia, inflammatory cell infiltration, and cytotoxic changes in the microenvironment cause further cell death and loss of function. Time-sensitive neuroprotective measures targeting these secondary in

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Book chapters on the topic "Anti ischemic"

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Lopez-Sendón, Jose, and Esteban López de Sá. "Anti-ischemic Therapy." In Pharmacological Treatment of Acute Coronary Syndromes. Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-5424-2_1.

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Kate, Mahesh P., Rajsrinivas Parthasarathy, and Ashfaq Shuaib. "Intravenous Thrombolysis and Anti-thrombotics." In Ischemic Stroke Therapeutics. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-17750-2_3.

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Deeg, P., K. H. Weiss, and H. Schmitz. "Anti-ischemic therapy with nisoldipine." In Predisposing Conditions for Acute Ischemic Syndromes. Steinkopff, 1989. http://dx.doi.org/10.1007/978-3-662-09434-1_20.

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Tzivoni, Dan, A. Keren, A. Gavish, J. Benhorin, and S. Stern. "Guiding anti-ischemic therapy by Holter monitoring." In Silent Ischemia. Steinkopff, 1987. http://dx.doi.org/10.1007/978-3-662-12997-5_23.

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Schmid-Schönbein, G. W., and G. J. del Zoppo. "Anti-PMN Leukocyte Strategies and Their Application to Focal Cerebral Ischemia." In Thrombolytic Therapy in Acute Ischemic Stroke II. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-78061-5_35.

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Simon, Rüdiger, Gunhild Herrmann, Ivo Amende, Gert H. Reil, and Paul R. Lichtlen. "Influence of anti-ischemic drug treatment on the ischemic response to acute coronary occlusion in man." In Developments in Cardiovascular Medicine. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-0453-8_21.

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Paulson, Dennis J., and Austin L. Shug. "Experimental evidence of the anti-ischemic effect of L-carnitine." In The Carnitine System. Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0275-9_13.

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Komarov, Pavel G., Alexander A. Morgunov, Mariana V. Bilenko, and Renat I. Zhdanov. "Anti-Ischemic Effect of 1-Hydroxy Derivatives of Nitroxyl Bioantioxidants." In Bioactive Spin Labels. Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-48724-8_20.

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Deeg, P., K. H. Weiss, and H. Schmitz. "Anti-Ischemic Effect of Nisoldipine in Patients with Stable Angina Pectoris." In Nisoldipine 1987. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-73010-8_36.

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Boisseau, M. R. "The role of anti-myeloid series treatment in ischemic vascular disorders." In Anticoagulation. Springer New York, 1994. http://dx.doi.org/10.1007/978-1-4612-2668-0_9.

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Conference papers on the topic "Anti ischemic"

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Guerci, A. D., and R. Beyar. "Intermittent coronary sinus occlusion: hemodynamic and anti-ischemic effects." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1988. http://dx.doi.org/10.1109/iembs.1988.94400.

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Rossillon, O., B. Bayet, M. Huymans, et al. "THE EFFECT OF PIRACETAM IN ISCHEMIC FLAP VASCULARISATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643425.

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Piracetam. a drug traditionally used for stimulating the telencephalon has a strong anti-aggregant, anti spasmodic and rheological action, due to its antisludge effect and the increase of the red cell deformability capacity. The authors show the interest of this substance in reconstructive surgery : Piracetam significantly increases the circulation in the flaps, reduces the zones prone to necrosis and allows for a faster skin expansion. The first three experiments have been performed on rats - on an epigastric flap with thoracic pedicle-. In the first one. we operated on 150 rats divided into

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Rajendran, Rasvini, and Zaitul Marlizawati Zainuddin. "The Determining Factors in Prescribing Anti-Hypertensive Drugs to First-Ever Ischemic Stroke Patients." In 2016 International Conference on Industrial Engineering, Management Science and Application (ICIMSA). IEEE, 2016. http://dx.doi.org/10.1109/icimsa.2016.7504041.

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Pawar, Meghna, Surekha Dabla, and B. D. Sharma. "Anti-NMDAR Encephalitis in a Young Girl Presenting as a Left MCA Ischemic Stroke." In ECON 2023. Thieme Medical and Scientific Publishers Pvt. Ltd., 2023. http://dx.doi.org/10.1055/s-0044-1791436.

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Ramos, Gabriel Bortoli, Rebecca Ranzani Martins, Júlia Carvalhinho Carlos de Souza, Cesar Castello Branco Lopes, and Guilherme Diogo Silva. "Spinal cord lesion and ischemic stroke after chiropractic: a case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.337.

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Context: Chiropractic is a form of spinal manipulation used to treat cervical pain. This therapy is considered safer than chronic use of anti-inflammatory drugs, opioids or spine surgery. However, chiropractic may cause severe complications such as myelopathy and ischemic stroke. Case report: A 48-year-old woman was admitted to our hospital due to acute tetraparesis. During a chiropractic session for chronic neck pain, patient referred lancinating cervical pain, weakness in the upper and lower limbs, and numbness below the level of the neck. Neurologic examination showed grade two tetraparesis

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Ben-Shabat, N., A. Krasun, L. Fisher, et al. "POS1252 ANTI-RO AND ANTI-LA SEROPOSITIVITY IS ASSOCIATED WITH INCREASED RATES OF ISCHEMIC HEART DISEASE IN ADULTS- RESULTS FROM A LARGE POPULATION-BASED STUDY." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.901.

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Hongfei, Su, Zeng Peng, Fang Yingyan, and Wang Qiyi. "A Network Pharmacology Approach to Uncover the Key Ingredients in Chuanxiong Rhizoma and Their Anti-Ischemic Stroke Mechanism." In 2021 International Conference on Public Health and Data Science (ICPHDS). IEEE, 2021. http://dx.doi.org/10.1109/icphds53608.2021.00041.

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Saad, Yasmine, Mariem Mhiri, Rihab Ben Dhia, et al. "Dual anti-platelet aggregation Versus Aspirin alone in acute minor ischemic stroke and high-risk TIA: A Tunisian Study (S20.006)." In 2023 Annual Meeting Abstracts. Lippincott Williams & Wilkins, 2023. http://dx.doi.org/10.1212/wnl.0000000000204113.

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Ahsan, Taby, Adele M. Doyle, Garry P. Duffy, Frank Barry, and Robert M. Nerem. "Stem Cells and Vascular Regenerative Medicine." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193591.

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Vascular applications in regenerative medicine include blood vessel substitutes and vasculogenesis in ischemic or engineered tissues. For these repair processes to be successful, there is a need for a stable supply of endothelial and smooth muscle cells. For blood vessel substitutes, the immediate goal is to enable blood flow, but vasoactivity is necessary for long term success. In engineered vessels, it is thought that endothelial cells will serve as an anti-thrombogenic lumenal layer, while smooth muscle cells contribute to vessel contractility. In other clinical applications, what is needed

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Li Jiansheng, Hu Jinliang, Liu Jingxia, Shen Jianjing, and Wang Jiehua. "AHP-based comprehensive evaluation of the effects of combination of thrombolytic and Chinese medicines on anti- cerebral ischemic injury in rats." In 2009 Chinese Control and Decision Conference (CCDC). IEEE, 2009. http://dx.doi.org/10.1109/ccdc.2009.5192592.

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Reports on the topic "Anti ischemic"

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He, Miao, Sen Wang, Jing You, Jing Shi, Yanbing Yin, and Weirong Li. Neuroprotective effects of resveratrol against cerebral ischemia/reperfusion injury through anti-oxidant and anti-inflammatory mechanisms：A Systematic Review and Meta-Analysis in Rodents. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.8.0059.

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