Relevant bibliographies by topics / Antigen tolerance

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Antigen tolerance'

Author: Grafiati
Published: 4 June 2021
Last updated: 11 February 2022

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Journal articles on the topic "Antigen tolerance"

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Oehen, S., L. Feng, Y. Xia, C. D. Surh, and S. M. Hedrick. "Antigen compartmentation and T helper cell tolerance induction." Journal of Experimental Medicine 183, no. 6 (1996): 2617–26. http://dx.doi.org/10.1084/jem.183.6.2617.

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The process of antigen recognition depends in part on the amount of peptide antigen available and the affinity of the T cell receptor for a particular peptide-major histocompatibility complex (MHC) molecule complex. The availability of self antigen is limited by antigen processing, which is compartmentalized such that peptide antigens presented by MHC class I molecules originate in the cytoplasm, whereas peptide antigens presented by MHC class II molecules are acquired from the endocytic pathway. This segregation of the antigen-processing pathways may limit the diversity of antigens that influ
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Carmona, Priscila, Yordanka Medina-Armenteros, Amanda Cabral, et al. "Regulatory/inflammatory cellular response discrimination in operational tolerance." Nephrology Dialysis Transplantation 34, no. 12 (2019): 2143–54. http://dx.doi.org/10.1093/ndt/gfz114.

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Abstract Background Antigen-specific cellular response is essential in immune tolerance. We tested whether antigen-specific cellular response is differentially modulated in operational tolerance (OT) in renal transplantation with respect to critical antigenic challenges in allotransplantation—donor antigens, pathogenic antigens and self-antigens. Methods We analysed the profile of immunoregulatory (REG) and pro-inflammatory (INFLAMMA) cytokines for the antigen-specific response directed to these three antigen groups, by Luminex. Results We showed that, in contrast to chronic rejection and heal
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Taylor, Justin J., Ryan J. Martinez, Philip J. Titcombe, et al. "Deletion and anergy of polyclonal B cells specific for ubiquitous membrane-bound self-antigen." Journal of Experimental Medicine 209, no. 11 (2012): 2065–77. http://dx.doi.org/10.1084/jem.20112272.

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B cell tolerance to self-antigen is critical to preventing antibody-mediated autoimmunity. Previous work using B cell antigen receptor transgenic animals suggested that self-antigen–specific B cells are either deleted from the repertoire, enter a state of diminished function termed anergy, or are ignorant to the presence of self-antigen. These mechanisms have not been assessed in a normal polyclonal repertoire because of an inability to detect rare antigen-specific B cells. Using a novel detection and enrichment strategy to assess polyclonal self-antigen–specific B cells, we find no evidence o
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Chung, Yeonseok, Jae-Hoon Chang, Mi-Na Kweon, Paul D. Rennert та Chang-Yuil Kang. "CD8α–11b+ dendritic cells but not CD8α+ dendritic cells mediate cross-tolerance toward intestinal antigens". Blood 106, № 1 (2005): 201–6. http://dx.doi.org/10.1182/blood-2004-11-4240.

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Cross-presentation is a critical process by which antigen is displayed to CD8 T cells to induce tolerance. It is believed that CD8α+ dendritic cells (DCs) are responsible for cross-presentation, suggesting that the CD8α+ DC population is capable of inducing both cross-priming and cross-tolerance to antigen. We found that cross-tolerance against intestinal soluble antigen was abrogated in C57BL/6 mice lacking mesenteric lymph nodes (MLNs) and Peyer patches (PPs), whereas mice lacking PPs alone were capable of developing CD8 T-cell tolerance. CD8α–CD11b+ DCs but not CD8α+ DCs in the MLNs present
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Duong, Bao Hoa, Hua Tian, Takayuki Ota, et al. "Decoration of T-independent antigen with ligands for CD22 and Siglec-G can suppress immunity and induce B cell tolerance in vivo." Journal of Experimental Medicine 207, no. 1 (2009): 173–87. http://dx.doi.org/10.1084/jem.20091873.

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Autoreactive B lymphocytes first encountering self-antigens in peripheral tissues are normally regulated by induction of anergy or apoptosis. According to the “two-signal” model, antigen recognition alone should render B cells tolerant unless T cell help or inflammatory signals such as lipopolysaccharide are provided. However, no such signals seem necessary for responses to T-independent type 2 (TI-2) antigens, which are multimeric antigens lacking T cell epitopes and Toll-like receptor ligands. How then do mature B cells avoid making a TI-2–like response to multimeric self-antigens? We presen
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Jung, Kyeong Cheon, Chung-Gyu Park, Yoon Kyung Jeon, et al. "In situ induction of dendritic cell–based T cell tolerance in humanized mice and nonhuman primates." Journal of Experimental Medicine 208, no. 12 (2011): 2477–88. http://dx.doi.org/10.1084/jem.20111242.

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Induction of antigen-specific T cell tolerance would aid treatment of diverse immunological disorders and help prevent allograft rejection and graft versus host disease. In this study, we establish a method of inducing antigen-specific T cell tolerance in situ in diabetic humanized mice and Rhesus monkeys receiving porcine islet xenografts. Antigen-specific T cell tolerance is induced by administration of an antibody ligating a particular epitope on ICAM-1 (intercellular adhesion molecule 1). Antibody-mediated ligation of ICAM-1 on dendritic cells (DCs) led to the arrest of DCs in a semimature
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Feng, Xiangru, Jiaxue Liu, Weiguo Xu, Gao Li, and Jianxun Ding. "Tackling autoimmunity with nanomedicines." Nanomedicine 15, no. 16 (2020): 1585–97. http://dx.doi.org/10.2217/nnm-2020-0102.

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Tolerogenic immunotherapy aims to blunt pathogenic inflammation without affecting systemic immunity. However, the anti-inflammatory drugs and immunosuppressive biologics that are used in the clinic usually result in nonspecific immune cell suppression and off-target toxicity. For this reason, strategies have been developed to induce antigen-specific immune tolerance through the delivery of disease-relevant antigens by nanocarriers as a benefit of their preferential internalization by antigen-presenting cells. Herein, we discuss the recent advances in the nanotechnology-based antigen-specific t
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Mapara, Markus Y., and Megan Sykes. "Tolerance and Cancer: Mechanisms of Tumor Evasion and Strategies for Breaking Tolerance." Journal of Clinical Oncology 22, no. 6 (2004): 1136–51. http://dx.doi.org/10.1200/jco.2004.10.041.

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The development of malignant disease might be seen as a failure of immune surveillance. However, not all tumors are naturally immunogenic, and even among those that are immunogenic, the uncontrolled rapid growth of a tumor may sometimes out-run a robust immune response. Nevertheless, recent evidence suggests that mechanisms of tolerance that normally exist to prevent autoimmune disease may also preclude the development of an adequate antitumor response and that tumors themselves have the ability to thwart the development of effective immune responses against their antigens. A major challenge h
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Horst, Andrea Kristina, Kingsley Gideon Kumashie, Katrin Neumann, Linda Diehl, and Gisa Tiegs. "Antigen presentation, autoantibody production, and therapeutic targets in autoimmune liver disease." Cellular & Molecular Immunology 18, no. 1 (2020): 92–111. http://dx.doi.org/10.1038/s41423-020-00568-6.

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AbstractThe liver is an important immunological organ that controls systemic tolerance. The liver harbors professional and unconventional antigen-presenting cells that are crucial for tolerance induction and maintenance. Orchestrating the immune response in homeostasis depends on a healthy and well-toned immunological liver microenvironment, which is maintained by the crosstalk of liver-resident antigen-presenting cells and intrahepatic and liver-infiltrating leukocytes. In response to pathogens or autoantigens, tolerance is disrupted by unknown mechanisms. Intrahepatic parenchymal and nonpare
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Parks, Christopher A., Kalli R. Henning, Kevin D. Pavelko, et al. "Breaking tolerance with engineered class I antigen-presenting molecules." Proceedings of the National Academy of Sciences 116, no. 8 (2019): 3136–45. http://dx.doi.org/10.1073/pnas.1807465116.

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Successful efforts to activate T cells capable of recognizing weak cancer-associated self-antigens have employed altered peptide antigens to activate T cell responses capable of cross-reacting on native tumor-associated self. A limitation of this approach is the requirement for detailed knowledge about the altered self-peptide ligands used in these vaccines. In the current study we considered allorecognition as an approach for activating CTL capable of recognizing weak or self-antigens in the context of self-MHC. Nonself antigen-presenting molecules typically contain polymorphisms that influen
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Dissertations / Theses on the topic "Antigen tolerance"

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Divekar, Rohit Dilip Zaghouani Habib. "Two aspects of peripheral immune tolerance systemic and mucosal tolerance mechanisms /." Diss., Columbia, Mo. : University of Missouri-Columbia, 2008. http://hdl.handle.net/10355/6868.

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The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Title from PDF of title page (University of Missouri--Columbia, viewed on April 1, 2010). Vita. Thesis advisor: Habib Zaghouani. "May 2008" Includes bibliographical references.
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Chen, Tse-Ching. "Dominant tolerance to a minor histocompatibility antigen." Thesis, University of Oxford, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.400052.

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Whitley, Nathaniel T. "Mechanisms in antigen-specific tolerance induction therapy." Thesis, University of Bristol, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.411069.

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Matriano, James Abcede. "Peripheral tolerance to an organ-specific antigen." Case Western Reserve University School of Graduate Studies / OhioLINK, 1991. http://rave.ohiolink.edu/etdc/view?acc_num=case1059484721.

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Ferry, Helen. "B cell tolerance to systemic, intracellular self-antigen." Thesis, University of Oxford, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.442947.

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Marshall, Naomi Jane. "Antigen presentation in autoimmune disease." Thesis, University of Edinburgh, 2009. http://hdl.handle.net/1842/4212.

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The aim of my project was to examine the extent to which endogenous expression of a largely renal-specific antigen influences the repertoire in adulthood of autoreactive T cells specific to that antigen. The renal-specific antigen, human α3(IV)NC1, is the target of autoimmune attack in Goodpasture’s disease. This protein was expressed and purified in recombinant (using bacterial and mammalian cell expression systems) and purified in native (extracted from human tissue) forms. Transgenic mice were generated that express HLA-DR15 (associated with Goodpasture’s disease) as their sole MHC class II
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Wheeler, Paul Richard. "Characterisation of T cell anergy in allo-antigen specific CD4⁺ cells." Thesis, University of Oxford, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.288516.

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Konkel, Joanne Elizabeth. "Signals required for the induction of antigen-based therapeutic tolerance." Thesis, University of Edinburgh, 2009. http://hdl.handle.net/1842/3942.

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Despite the actions of central tolerance during thymic selection, it is clear that the peripheral T cell repertoire contains significant numbers of self-reactive T cells. The immune system needs to curtail the risk of autoimmune disease by controlling the activity of these self-reactive T cells. Various mechanisms are in place to achieve this control (peripheral tolerance). Activation of CD4+ T cells requires two signals; engagement of the T cell receptor (TCR) with an appropriate peptide:MHC complex (signal 1), and the aggregate effect of multiple signals generated following ligation of costi
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Yuschenkoff, Victoria Nicole. "Tolerance Induction to a Foreign Protein Antigen: Analysing the Role of B Cells in Establishing Peripheral Tolerance." eScholarship@UMMS, 1995. http://escholarship.umassmed.edu/gsbs_diss/298.

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Tolerance to self proteins is largely dependent upon the deletion of immature, self-specific T and B cells in the thymus and bone marrow. Although highly efficient, the elimination of these self-reactive lymphocytes is dependent on the expression of their target antigen in these primary lymphoid organs. Many proteins, however, such as hormones, are developmentally regulated and expressed at different stages of life, while other proteins are expressed outside the thymus and marrow. To ensure self-tolerance, other mechanisms must exist to inactivate or prevent the activation of mature, potential
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Sefia, Eseberuo. "Mechanism of immune tolerance induction in antigen-specific human autoimmune disease." Thesis, Queen Mary, University of London, 2014. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8982.

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Multiple sclerosis (MS) is an inflammatory disease that affects the central nervous system and is considered to be a T-cell mediated autoimmune disease. The “ideal” method in treating MS would be an antigen-specific therapy that does not require generalized immunosuppression. To date there are no definitive treatments for MS but there are several licensed therapies such as -interferon. Unfortunately the effect of interferon (IFN) is reduced by the development of neutralizing antibodies (NAbs) in up to 35% of MS patients within two years of starting treatment. An immunization schedule was d
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Books on the topic "Antigen tolerance"

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Teng, Yen-Tung Andy. Analysis of the mechanism(s) of immunological tolerance to a physiological soluble antigen in transgenic mice. National Library of Canada = Bibliothèque nationale du Canada, 1997.

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Morteau, Olivier. Oral tolerance: The response of the intestinal mucosa to dietary antigens. Landes Bioscience/Eurekah.com, 2004.

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Morteau, Olivier. Oral tolerance: The response of the intestinal mucosa to dietary antigens. Landes Bioscience/Eurekah.com, 2004.

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Morteau, Olivier. Oral tolerance: The response of the intestinal mucosa to dietary antigens. Landes Bioscience, 2001.

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Razzaghi, Hamid. Establishment of transgenic mice carrying mutated human insulin gene: A model system for studying the immunological self-tolerance to a soluble antigen. National Library of Canada, 1993.

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EMBO, Workshop on Tolerance (1986 Basel Switzerland). The tolerance workshop: Proceedings of the EMBO Workshop on Tolerance held at the Basel Institute for Immunology, 20-26 October 1986. Editiones Roche, 1987.

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I, Johnston Paul, ed. Anthology of the theological writings of J. Michael Reu. E. Mellen Press, 1997.

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Morteau, Olivier. Oral Tolerance: The Response of the Intestinal Mucosa to Dietary Antigens. Springer, 2010.

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Morteau, Olivier. Oral Tolerance: Cellular and Molecular Basis, Clinical Aspects, and Therapeutic Potential (Medical Intelligence Unit). Springer, 2004.

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(Editor), B. Kyewski, and Elisabeth Suri-Payer (Editor), eds. CD4+CD25+ Regulatory T Cells: Origin, Function and Therapeutic Potential (Current Topics in Microbiology and Immunology). Springer, 2005.

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Book chapters on the topic "Antigen tolerance"

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Stoy, Nicholas S. "Macrophages - Balancing Tolerance and Immunity." In Antigen Presenting Cells. Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/3527607021.ch10.

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McGeachy, Mandy J., Richard O’Connor, Leigh A. Stephens, and Stephen M. Anderton. "Antigen-Based Therapy and Immune-Regulation in Experimental Autoimmune Encephalomyelitis." In Immunological Tolerance. Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-395-0_18.

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de Heusch, Magali, Guillaume Oldenhove, and Muriel Moser. "Dendritic Cells (DCs) in Immunity and Maintenance of Tolerance." In Antigen Presenting Cells. Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/3527607021.ch14.

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McBride, J. S. "From Serology of the Chicken MHC to Polymorphism of Malaria Antigen Genes." In Realm of Tolerance. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74712-0_18.

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Bruce, M. G., and A. Ferguson. "Oral Tolerance Induced by Gut-Processed Antigen." In Recent Advances in Mucosal Immunology. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5344-7_84.

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Kretschmer, Karsten, Irina Apostolou, Panos Verginis, and Harald von Boehmer. "Regulatory T Cells and Antigen-Specific Tolerance." In Chemical Immunology and Allergy. KARGER, 2008. http://dx.doi.org/10.1159/000154846.

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Zheng, Lixin, Stefen A. Boehme, Jeffrey M. Critchfield, Juan Carlos Zuniga-Pflucker, Matthew Freedman, and Michael J. Lenardo. "Immunological Tolerance by Antigen-Induced Apoptosis of Mature T Lymphocytes." In Advances in Experimental Medicine and Biology. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4899-0987-9_9.

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Woods, David M., Andressa Laino, Alejandro Villagra, and Eduardo M. Sotomayor. "Molecular Pathways in Antigen-Presenting Cells Involved in the Induction of Antigen-specific T-cell Tolerance." In Tumor-Induced Immune Suppression. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4899-8056-4_15.

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Izon, David J., John D. Nieland, Lori A. Jones, and Ada M. Kruisbeek. "T Cell Tolerance and Antigen Presenting Cell Function in the Thymus." In Advances in Experimental Medicine and Biology. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2930-9_27.

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Anderton, S. M., C. Burkhart, G. Y. Liu, B. Metzler, and D. C. Wraith. "Antigen-Specific Tolerance Induction and the Immunotherapy of Experimental Autoimmune Disease." In Novartis Foundation Symposia. John Wiley & Sons, Ltd., 2007. http://dx.doi.org/10.1002/9780470515525.ch9.

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Conference papers on the topic "Antigen tolerance"

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Ruhland, Megan K., Edward W. Roberts, and Matthew F. Krummel. "Abstract B184: Modulating antigen flow to control immune tolerance." In Abstracts: Fourth CRI-CIMT-EATI-AACR International Cancer Immunotherapy Conference: Translating Science into Survival; September 30 - October 3, 2018; New York, NY. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr18-b184.

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Nilsson, I. M., E. Berntorp, and O. Zettervall. "TOLERANCE INDUCTION IN HIGH-RESPONDING HEMOPHILIACS WITH F VIII ANTIBODIES BY MEANS OF COMBINED TREATMENT WITH IgG, CYCLOPHOSPHAMIDE AND F VIII." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644717.

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Of 10 patients with hemophilia A and antibodies, 7 have been rendered tolerant by means of combined treatment with high-dose IgG i.v., cyclophosphamide and F VIII. When the initial antibody concentration exceeded 10 Bethesda inhibitor units per ml, the treatment was preceded by antibody adsorption to protein A. Six of the tolerant patients were originally classified as high-responders. After one week of the combined treatment, VI11:C dropped and the inhibitor reappeared in low titer. The F VIII infusions being continued alone, the inhibitor disappeared in the following week and VIII:C increase
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Khan, Muhammad Tahir, Toar Imanuel, Yelnil Gabo, and C. W. de Silva. "Robust Multi-Robot Cooperation Using an Idiotypic Model of Artificial Immune Systems." In ASME 2010 International Mechanical Engineering Congress and Exposition. ASMEDC, 2010. http://dx.doi.org/10.1115/imece2010-37966.

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The human immune system is a network of cells, tissues, and other organs that defend the body against foreign invaders called antigens. Jerne’s Idiotypic network theory concerns how an antibody in the immune system stimulates or suppresses another antibody and recognizes an antigen. Based on the principles of the human immune system and Jerne’s idiotypic network theory this paper presents a method for cooperation among robots in a multi-robot system. The developed cooperative multi-robot system is fully autonomous and distributed. In the present paper, cooperation is not assumed a priori. If a
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Nordfang, O., M. Ezban, and J. B. Knudsen. "IMMUNOASSAY FOR FACTOR VIII-HEAVY CHAIN. AN INDICATOR FOR IMMUNE COMPLEXES DURING HIGH DOSE FVIII INHIBITOR TREATMENT." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644718.

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Specificity studies have shown that most hemophilia A inhibitor antibodies are directed towards the light chain of coagulation factor VIII (FVIII). Thus, conventional immunoassays for FVIII-antigen (FVIII:Ag) presumably have reactivity for FVIII-Light Chain (FVIII-LC) . Our sandwich FVIII :Ag assay has been shewn to be specific for only FVIII-LC. We have now developed a specific immunoassay for FVIII-Heavy Chain (FVIII-HC) . This has made it possible to investigate the FVIII-HC content in hemophilia A plasma, and to study the expression of FVIII-HC in culture medium frcm transfected cell lines
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Ryabov, Vladimir, Peter Pushko, Irina Tretyakova, Rikka Saito, Richard B. Alexander, and Elena N. Klyushnenkova. "Abstract 2880: Novel alphavirus-based vaccine targets dendritic cells and efficiently breaks immunological tolerance to “self” tumor-associated antigen (PSA) in an HLA-DR mouse model of prostate cancer." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-2880.

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Jachetti, Elena, Alice Rigoni, Lucia Bongiovanni, Claudio Tripodo, and Mario P. Colombo. "Abstract 4054: Mast cells contribute to T cell tolerance against prostate cancer- associated antigens favoring tumor growth." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-4054.

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Grasso, C., JS Hähnlein, R. Nadafi, et al. "P113/O17 Human lymph node stromal cells express self-antigens targeted by anti-citrullinated protein antybodies: role for tolerance induction in rheumathoid arthritis." In 39th European Workshop for Rheumatology Research, 28 February–2 March 2019, Lyon, France. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2018-ewrr2019.101.

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Bryson, Paul D., Xiaolu Han, Norman Truong та Pin Wang. "Abstract 2888: Dendritic cell-targeted lentiviral vector vaccines overcome tolerance to generate a protective T-cell immune response to breast cancer antigens ERBB2 and α-lactalbumin". У Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-2888.

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Pere, Helene, Yves Montier, Jagadeesh Bayry, et al. "Abstract 752: A CCR4 antagonist combined with protein-or DNA-based vaccines efficiently breaks tolerance and elicits CD8+T cells directed against self and viral associated tumor antigens." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-752.

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