Academic literature on the topic 'Antigliadin antibodies'

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Journal articles on the topic "Antigliadin antibodies"

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Wong, D., M. Dwinnel, M. Schulzer, M. Nimmo, B. R. Leavitt, and S. D. Spacey. "Ataxia and the Role of Antigliadin Antibodies." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 34, no. 2 (2007): 193–96. http://dx.doi.org/10.1017/s031716710000603x.

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Background:Although it is acknowledged that patients with celiac disease can develop neurological complications such as ataxia, the association of antigliadin antibodies in the etiology of sporadic ataxia and the usefulness of this testing in diagnosis of ataxia is controversial.Methods:We investigated this association by testing for the presence of IgG and IgA antigliadin antibodies in 56 ataxic patients and 59 controls. The ataxia patients were subsequently classified into three groups: sporadic, hereditary and MSA.Results:Of the total ataxic patients, 6/56 (11%) were positive for either IgG
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TÜRKOĞLU, Recai, Nuriye ÇÖMEZ, Mehmet GENCER, Yılmaz ÇETİNKAYA, Züleyha AKKAN ÇETİNKAYA, and Hülya TİRELİ. "Antigliadin Antibodies and Multiple Sclerosis." Turkiye Klinikleri Journal of Medical Sciences 30, no. 2 (2010): 577–83. http://dx.doi.org/10.5336/medsci.2008-9050.

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Sultan, Sheikh Javeed, Qazi Masood Ahmad, and Sheikh Tariq Sultan. "Antigliadin antibodies in psoriasis." Australasian Journal of Dermatology 51, no. 4 (2010): 238–42. http://dx.doi.org/10.1111/j.1440-0960.2010.00648.x.

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Zachor, Ditza A., Elizabeth Mroczek‐Musulman, and Pamela Brown. "Prevalence of Celiac Disease in Down Syndrome in the United States." Journal of Pediatric Gastroenterology and Nutrition 31, no. 3 (2000): 275–79. http://dx.doi.org/10.1002/j.1536-4801.2000.tb07116.x.

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ABSTRACTBackgroundNumerous studies in Europe have documented a high prevalence of celiac disease in Down syndrome. This study was undertaken to estimate the prevalence of celiac disease in Down syndrome in the southeastern United States.MethodsSeventy‐five patients with Down syndrome were screened using immunoglobulin (Ig)A‐anti antiendomysium antibodies, IgA‐antigliadin antibodies, and total IgA level. When either antiendomysium or antigliadin antibodies produced positive findings, patients were referred to a pediatric gastroenterologist for consideration of a duodenal biopsy.ResultsThirteen
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Bushara, K. O., M. Nance, and C. M. Gomez. "Antigliadin antibodies in Huntington's disease." Neurology 62, no. 1 (2004): 132–33. http://dx.doi.org/10.1212/wnl.62.1.132.

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Barta, Z., G. Mekkel, M. Zeher, and K. O. Bushara. "Antigliadin antibodies in Huntington's disease." Neurology 63, no. 4 (2004): 762. http://dx.doi.org/10.1212/wnl.63.4.762-a.

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Kalayciyan, A., and A. Kotogyan. "Psoriasis, enteropathy and antigliadin antibodies." British Journal of Dermatology 154, no. 4 (2006): 778–79. http://dx.doi.org/10.1111/j.1365-2133.2006.07132.x.

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Schlesinger, I., and R. Hering. "Antigliadin Antibodies in Migraine Patients." Cephalalgia 17, no. 6 (1997): 712. http://dx.doi.org/10.1177/033310249701700616.

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El-Alameey, Inas R., Hanaa H. Ahmed, Sawsan M. Tawfik, Fawzia Hassaballa, Ayman M. Abdel Gawad, and Eman Eltahlawy. "Gluten Sensitivity among Egyptian Infants with Congenital Heart Disease." Open Access Macedonian Journal of Medical Sciences 5, no. 1 (2017): 58–63. http://dx.doi.org/10.3889/oamjms.2017.026.

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BACKGROUND: Gastrointestinal symptoms are a common feature in infants with congenital heart disease.AIM: This study was designed to evaluate age-dependent serum levels of antigliadin antibodies among malnourished Egyptian infants with congenital heart disease (CHD) and gastrointestinal symptoms.SUBJECTS AND METHODS: This case-control study conducted on 60 infants with established congenital heart disease. They were subdivided into cyanotic and acyanotic groups, and each group includes 30 patients compared with thirty apparently healthy infants of matched age, sex, and social class. Serum antig
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Juto, Per, Bo Fredrikzon, and Olle Hernell. "Gliadin‐Specific Serum Immunoglobulins A, E, G, and M in Childhood." Journal of Pediatric Gastroenterology and Nutrition 4, no. 5 (1985): 723–29. http://dx.doi.org/10.1002/j.1536-4801.1985.tb08945.x.

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Summary:An enzyme‐linked immunosorbent assay technique was developed to determine serum antigliadin antibodies of the IgA, IgE, IgG, and IgM classes. The antibody level of each serum specimen was expressed as an index value, i.e., optical density of test serum/optical density of cutoff, where cutoff was calculated for each immunoglobulin class as the mean + 3 SD for six healthy controls. Indices for each immunoglobulin class were determined in 69 children who were admitted for their first small intestinal mucosal biopsy due to either symptoms of malabsorption compatible with celiac disease, or
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Book chapters on the topic "Antigliadin antibodies"

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Bizzaro, Nicola, Elio Tonutti, and Danilo Villalta. "Antigliadin and Antideamidated Gliadin Peptide Antibodies." In Autoantibodies. Elsevier, 2014. http://dx.doi.org/10.1016/b978-0-444-56378-1.00055-1.

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Hekkens, Wim Th J. M., and Marja van Twist - de Graaf. "Relation of Antigliadin Antibodies to Gluten-Sensitive Enteropathy." In Serologic Diagnosis of Celiac Disease. CRC Press, 2020. http://dx.doi.org/10.1201/9780429281839-2a.

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Bürgin-Wolff, Annemarie, and M. J. Lentze. "Relation of Antigliadin Antibodies to Gluten-Sensitive Enteropathy." In Serologic Diagnosis of Celiac Disease. CRC Press, 2020. http://dx.doi.org/10.1201/9780429281839-2b.

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Vainio, Eeva, Markku Viander, Olavi Hällström, and Timo Reunala. "Relation of Antigliadin Antibodies to Gluten-Sensitive Enteropathy." In Serologic Diagnosis of Celiac Disease. CRC Press, 2020. http://dx.doi.org/10.1201/9780429281839-2c.

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Miller, Aaron E., Tracy M. DeAngelis, Michelle Fabian, and Ilana Katz Sand. "A Case of Cognitive Impairment and Ataxia." In Neuroimmunology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190693190.003.0012.

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Celiac disease is an autoimmune enteropathy characterized by the presence of antigliadin antibodies and small intestinal mucosal injury, mediated by the adaptive immune system in response to gluten. Gastrointestinal symptoms typically include diarrhea, malabsorption/weight loss, and bloating/abdominal pain. Extra-intestinal symptoms often occur. The most commonly reported neurological syndromes are peripheral neuropathy, headache, and “gluten ataxia.” Gluten ataxia may occur in the setting of celiac disease or with non-celiac gluten sensitivity. Consistent with this being a cerebellar phenomen
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Niederhofer, Helmut, and Klaus Pittschieler. "Behavioral and Cognitive Manifestations of Celiac Disease." In Cognitive and Behavioral Abnormalities of Pediatric Diseases. Oxford University Press, 2010. http://dx.doi.org/10.1093/oso/9780195342680.003.0020.

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Celiac disease (CD) is an immune-mediated chronic inflammatory disorder characterized by permanent gluten intolerance in genetically susceptible individuals. Exposure to gluten perpetuates an enteropathy leading to malabsorption with chronic diarrhea, weight loss, and abdominal distension. The small intestine mucosa is abnormal, and jejunal biopsy demonstrates various degrees of villous atrophy, absence of surface mucosa, and crypt hyperplasia. The diagnosis is based on the demonstration of a more or less pronounced villus atrophy in a jejunal biopsy. The villous atrophy improves after withdra
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Conference papers on the topic "Antigliadin antibodies"

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Corres, J. M., I. R. Matias, J. Goicoechea, et al. "Experimental results of antigliadin antibodies detection using long period fiber grating." In 19th International Conference on Optical Fibre Sensors, edited by David D. Sampson. SPIE, 2008. http://dx.doi.org/10.1117/12.786076.

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