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Journal articles on the topic 'Antituberculous'

Author: Grafiati

Published: 5 June 2025

Last updated: 1 August 2025

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1

Unni, Jeeson C. "Antituberculous Drugs." Pediatric Infectious Disease 1, no. 1 (2019): 37–38. http://dx.doi.org/10.5005/jp-journals-10081-1109.

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2

VAN SCOY, ROBERT E., and CONRAD J. WILKOWSKE. "Antituberculous Agents." Mayo Clinic Proceedings 67, no. 2 (1992): 179–87. http://dx.doi.org/10.1016/s0025-6196(12)61320-2.

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3

VAN SCOY, ROBERT E., and CONRAD J. WILKOWSKE. "Antituberculous Agents." Mayo Clinic Proceedings 62, no. 12 (1987): 1129–36. http://dx.doi.org/10.1016/s0025-6196(12)62507-5.

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4

Aleta, Lara Theresa. "Antituberculous drug rechallenge." World Allergy Organization Journal &NA; (November 2007): S46. http://dx.doi.org/10.1097/01.wox.0000301213.64918.aa.

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5

Dyachenko, P. A. "CANDIDATE OF MEDICINE, SENIOR RESEARCHER OF THE DEPARTMENT OF NEUROINFECTIONS OF L. HROMASHEVSKYI INSTITUTE OF EPIDEMIOLOGY AND INFECTION DISEASES OF THE NATIONAL ACADEMY OF MEDICAL SCIENCES OF UKRAINE." Інфекційні хвороби, no. 3 (October 11, 2018): 60–64. http://dx.doi.org/10.11603/1681-2727.2018.3.8569.

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Background. Central nervous system tuberculosis is one of the most severe forms of extra-pulmonary tuberculosis. Tuberculous meningoencephalitis (TBM) is highly prevalent globally in resource-limited countries and in patients with immunosuppression. We present here a case of meningoencephalitis with proved acute Herpes simplex virus infection. However, our patient responded to antituberculosis therapy. This raises the possibility that some cases of “idiopathic” (cryptogenic) meningoencephalitis may represent occult tuberculosis disease. Result. A 22-year-old woman was admitted to our hos

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6

Tsenova, Liana, Bande Mangaliso, George Muller, et al. "Use of IMiD3, a Thalidomide Analog, as an Adjunct to Therapy for Experimental Tuberculous Meningitis." Antimicrobial Agents and Chemotherapy 46, no. 6 (2002): 1887–95. http://dx.doi.org/10.1128/aac.46.6.1887-1895.2002.

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ABSTRACT Tuberculous meningitis (TBM), the most severe form of Mycobacterium tuberculosis infection in humans, is associated with significant morbidity and mortality despite successful treatment with antituberculous drugs. This is due to the irreversible brain damage subsequent to the local inflammatory response of the host to M. tuberculosis. Corticosteroids have been used in conjunction with antituberculous therapy in an attempt to modulate the inflammatory response, but this strategy has been of limited success. Therefore, we examined whether combining antituberculous drugs with the immunom

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7

Muhammed Ameen, S., and M. Drancourt. "Ivermectin lacks antituberculous activity." Journal of Antimicrobial Chemotherapy 68, no. 8 (2013): 1936–37. http://dx.doi.org/10.1093/jac/dkt089.

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8

COWARD, R. "CYCLOSPORIN AND ANTITUBERCULOUS THERAPY." Lancet 325, no. 8441 (1985): 1342–43. http://dx.doi.org/10.1016/s0140-6736(85)92845-4.

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9

Seth, V. "Antituberculous therapy in children." Indian Journal of Pediatrics 53, no. 3 (1986): 428. http://dx.doi.org/10.1007/bf02760435.

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10

Seth, Vimlesh. "Antituberculous therapy in children." Indian Journal of Pediatrics 53, no. 2 (1986): 179–98. http://dx.doi.org/10.1007/bf02748507.

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11

Das, Anirban, Sibes Kumar Das, Abhijit Mandal, and Arup Kumar Halder. "Cerebral tuberculoma as a manifestation of paradoxical reaction in patients with pulmonary and extrapulmonary tuberculosis." Journal of Neurosciences in Rural Practice 03, no. 03 (2012): 350–54. http://dx.doi.org/10.4103/0976-3147.102622.

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ABSTRACTExpansion of cerebral tuberculomas or their new appearance as a manifestation of paradoxical reaction in patients under antituberculous chemotherapy is well documented. Distinguishing paradoxical reaction from disease progression or treatment failure is an important issue in tuberculosis management. Five cases of cerebral tuberculomas are reported here as manifestations of paradoxical reaction in patients with pulmonary and extrapulmonary tuberculosis on antituberculous treatment. Case 1 and 2 had tuberculous meningitis, Case 3 had miliary tuberculosis, Case 4 had miliary tuberculosis

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12

Xiao, Gang. "Association of Genetic Polymorphism of GSTM1 and GSTT1 with the Susceptibility to Antituberculosis Drug-induced Hepatotoxicity in Chinese Population." Journal of Advances in Medicine Science 1, no. 3 (2018): 80. http://dx.doi.org/10.30564/jams.v1i3.56.

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Objective To investigate the relationship between the polymorphism of glutathione S transferase M1, T1(GSTM1, GSTT1) gene and the susceptibility to antituberculosis drug induced hepatotoxicity (ATDH) in patients with tuberculosis. Methods GSTM1 and GSTT1 gene polymorphisms in patients with or without liver toxicity after antituberculous treatment were analyzed using multiple PCR method. Results In ATDH group and control group, the proportion of GSTM1 gene deletion was 58.0% and 50.7%respectively, and the difference was not statistically signifcant (OR=1.322, 95%CI=0.921~1.878), the frequencies

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13

Kim, Sang Cheol, Jae Joong Baik, Tae Hoon Lee, and Yeon Tae Chung. "Joint symptoms during antituberculous chemotherapy." Tuberculosis and Respiratory Diseases 49, no. 2 (2000): 162. http://dx.doi.org/10.4046/trd.2000.49.2.162.

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14

DE MARIA, A. "Side effects of antituberculous treatment." Thorax 56, no. 12 (2001): 983. http://dx.doi.org/10.1136/thorax.56.12.983.

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15

Kreytsberg, G. N., I. E. Gracheva, B. S. Kibrik, and I. V. Golikov. "Antituberculous effect of silver nanoparticles." Journal of Physics: Conference Series 291 (April 1, 2011): 012030. http://dx.doi.org/10.1088/1742-6596/291/1/012030.

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16

Inoue, Ken-ichiro, Yutaka Kawahito, Hajime Sano, and Toshikazu Yoshikawa. "Antituberculous Drugs for Wegener’s Granulomatosis." Chest 120, no. 6 (2001): 2112. http://dx.doi.org/10.1378/chest.120.6.2112.

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17

Huycke, Mark M., Jerry D. Leu, M. Alex Jacocks, Ralph T. Guild, and Robert Whang. "Colitis Due to Antituberculous Chemotherapy." Southern Medical Journal 84, no. 2 (1991): 285. http://dx.doi.org/10.1097/00007611-199102000-00043.

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18

Ibraheem, Tamer. "Paradoxical response to antituberculous therapy." Egyptian Journal of Chest Diseases and Tuberculosis 63, no. 1 (2014): 1–2. http://dx.doi.org/10.1016/j.ejcdt.2013.09.004.

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19

Toyoshima, Mikio, Kingo Chida, Takafumi Suda, Shiro Imokawa, and Hirotoshi Nakamura. "Antituberculous Drugs for Wegener’s Granulomatosis." Chest 120, no. 6 (2001): 2112–13. http://dx.doi.org/10.1016/s0012-3692(15)38651-7.

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20

ERTEM, EKIN, KEMAL YÜCE, G. ÜNEY KARAKARTAL, ORHAN ÜNAL, and GÜL YÜCE. "The antituberculous effect of bleomycin." Journal of Antimicrobial Chemotherapy 26, no. 6 (1990): 862–63. http://dx.doi.org/10.1093/jac/26.6.862.

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21

Lalloo, Umesh G., and Anish Ambaram. "New Antituberculous Drugs in Development." Current HIV/AIDS Reports 7, no. 3 (2010): 143–51. http://dx.doi.org/10.1007/s11904-010-0054-4.

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22

Lipunova, G. N., É. V. Nosova, M. A. Kravchenko, et al. "Fluorinated quinolones possessing antituberculous activity." Pharmaceutical Chemistry Journal 38, no. 11 (2004): 597–601. http://dx.doi.org/10.1007/s11094-005-0037-8.

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23

Devarbhavi, Harshad. "Antituberculous drug-induced liver injury." Tropical Gastroenterology 32, no. 3 (2011): 167–74. https://doi.org/10.4103/trog_20113203_167.

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Drug-induced liver injury (DILI) is a minor but significant cause of liver injury across all regions. Antituberculosis drug-induced liver injury (TB DILI) is a leading cause of DILI and drug-induced acute liver failure (DIALF) in India and much of the developing world. Single center registries of DILI continue to highlight the high incidence of DILI and DIALF, much of it due to diagnostic errors and inappropriate prescriptions. The clinical spectrum includes asymptomatic elevation in liver tests to acute hepatitis and acute liver failure. TB DILI can occur across all age groups including child

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24

Liu, Barbara A., Sandra R. Knowles, Lawrence B. Cohen, Marsha R. Werb, and Neil H. Shear. "Pancreatic Insufficiency Due to Antituberculous Therapy." Annals of Pharmacotherapy 31, no. 6 (1997): 724–26. http://dx.doi.org/10.1177/106002809703100610.

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OBJECTIVE: To describe a case of chronic pancreatic insufficiency related to antituberculous therapy. CASE SUMMARY: A 57-year-old man developed rash, fever, and hepatitis (aspartate aminotransferase 369 IU/L, alanine aminotransferase 506 IU/L), 6 weeks after starting isoniazid, rifampin, ethambutol, and pyrazinamide. He also developed severe metabolic acidosis secondary to diabetic ketoacidosis and lactic acidosis (serum bicarbonate 7 mEq/L, glucose 1778 mg/dL, and lactate 4.0 mEq/L). Acute pancreatitis was diagnosed on the basis of a mildly elevated amylase concentration (392 U/L) and radiolo

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25

Mandour, Cherkaoui, Jawad Laaguili, Miloudi Gazzaz, and El Mostarchid Brahim. "Paradoxical Tuberculomas Complicating Tuberculous Meningitis: About Two Cases." Indian Journal of Neurosurgery 07, no. 01 (2017): 051–53. http://dx.doi.org/10.1055/s-0036-1585426.

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AbstractParadoxical tuberculomas in patients with tuberculous meningitis during antituberculous chemotherapy is an unusual phenomenon. This reaction is due to the immune response of the host to antituberculous drugs. It is commonly seen in the intensive phase of chemotherapy. We report the cases of two patients with tuberculous meningitis who had developed tuberculomas during adequate and appropriate antitubercular therapy. Tuberculous meningitis requires the regular clinical and radiological follow-up to detect paradoxical response.

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26

Riasensii, Dmitri Sergeevich, N. A. Grishkina, and A. V. Aseev. "IMPACT OF TUBERCULOSIS AND ANTITUBERCULOUS CHEMOTHERAPY ON LIPID COMPOSITION OF THE BLOOD PLASMA." Epidemiology and Infectious Diseases (Russian Journal) 23, no. 5 (2018): 220–24. http://dx.doi.org/10.18821/1560-9529-2018-23-5-220-224.

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Tuberculosis is an infectious disease caused by tuberculosis mycobacteria of human or bovine types and is characterized by multiple organs failure and chronic recurrent course. The blood plasma lipid spectrum state is one of the antituberculous chemotherapy toxic effect markers. The important role of the ratio of various fractions of general and blood phospholipids for the evaluation of the state of the organism in infectious pathology is proved. The purpose of this work is to study the features of the lipid spectrum of blood plasma in patients with pulmonary tuberculosis prior to treatment an

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27

Riasensii, Dmitri Sergeevich, N. A. Grishkina, and A. V. Aseev. "IMPACT OF TUBERCULOSIS AND ANTITUBERCULOUS CHEMOTHERAPY ON LIPID COMPOSITION OF THE BLOOD PLASMA." Epidemiology and Infectious Diseases (Russian Journal) 23, no. 5 (2018): 220–24. http://dx.doi.org/10.18821/1560-9529-2019-23-5-220-224.

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Tuberculosis is an infectious disease caused by tuberculosis mycobacteria of human or bovine types and is characterized by multiple organs failure and chronic recurrent course. The blood plasma lipid spectrum state is one of the antituberculous chemotherapy toxic effect markers. The important role of the ratio of various fractions of general and blood phospholipids for the evaluation of the state of the organism in infectious pathology is proved. The purpose of this work is to study the features of the lipid spectrum of blood plasma in patients with pulmonary tuberculosis prior to treatment an

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28

Turner, Mark O., and R. Kevin Elwood. "Severe Hepatic Complications of Antituberculous Therapy." Canadian Journal of Infectious Diseases 10, no. 2 (1999): 167–69. http://dx.doi.org/10.1155/1999/342613.

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Hepatotoxicity from antituberculous therapy is well described, but fortunately severe complications are rare. The optimal methods of monitoring for significant hepatotoxicity while on treatment are uncertain. Some authorities recommend measuring liver enzymes only if symptoms develop, whereas others recommend regular liver enzyme monitoring throughout the course of therapy. In British Columbia, from 1990 to 1997, 2624 active and approximately 8000 chemoprophylaxis cases have been treated, but only two severe complications directly related to antituberculous therapy have occurred. A 33-year-old

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29

Ozarmagan, G., EZ Ozgüroglu, MU Maden, and S. Oztürk. "Refractory ulcerative lupus vulgaris associated with CD4 lymphocytopenia, inversion of chromosome 14, primary amenorrhoea and mental retardation." Acta Dermato-Venereologica 77, no. 4 (1997): 309–10. http://dx.doi.org/10.2340/0001555577309310.

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A case of ulcerative lupus vulgaris, confirmed by polymerase chain reaction (PCR) is reported. The initial lesion of our case was a papule on the nose, which progressed during antituberculous treatment and caused cartilage destruction and ectropion. Immunological analysis revealed CD4 lymphocytopenia, and the possibility of idiopathic CD4 lymphocyte deficiency was considered. In addition, the patient had primary amenorrhoea, mental retardation and inversion of chromosome 14. CD4 lymphocytopenia and chromosomal abnormality are the possible causes of antituberculous treatment failure.

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30

Igarashi, Masayuki, Yoshimasa Ishizaki, and Yoshiaki Takahashi. "New antituberculous drugs derived from natural products: current perspectives and issues in antituberculous drug development." Journal of Antibiotics 71, no. 1 (2017): 15–25. http://dx.doi.org/10.1038/ja.2017.126.

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31

Idilman, Ramazan, Sadik Ersoz, Sahin Coban, Ozlem Kumbasar, and Hakan Bozkaya. "Antituberculous therapy–induced fulminant hepatic failure: Successful treatment with liver transplantation and nonstandard antituberculous therapy." Liver Transplantation 12, no. 9 (2006): 1427–30. http://dx.doi.org/10.1002/lt.20839.

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32

Jiang, Yu-Gang, Jing Chen, and Yong Peng. "TUBERCULOUS BRAINSTEM ABSCESS." Neurosurgery 65, no. 6 (2009): E1206—E1207. http://dx.doi.org/10.1227/01.neu.0000356986.74982.2d.

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Abstract OBJECTIVE Tuberculous brainstem abscess is a clinically rare condition with potentially high mortality and morbidity. We present this report to draw attention to the importance of early recognition and adequate treatment of tuberculous brainstem abscess. CLINICAL PRESENTATION A 24-year-old man complained of longstanding fever, headache, and weakness followed by development of progressive slurred speech and hemiparesis of the right extremities. Magnetic resonance imaging revealed a large thick-walled cystic lesion lying within the brainstem. INTERVENTION The patient demonstrated a rema

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33

Dicks, Kristen V., David P. Holland, Myra G. Allen, et al. "Impact of radiology reports on timely tuberculosis diagnosis." Postgraduate Medical Journal 94, no. 1115 (2018): 495–98. http://dx.doi.org/10.1136/postgradmedj-2018-135984.

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PurposeAs tuberculosis becomes less common in higher income countries, clinician familiarity with the disease is declining. Little is known about how chest radiograph interpretations affect tuberculosis care. We sought to determine how tuberculosis-related terminology in an initial chest radiograph reading impacted patient care.Study designWe examined a retrospective cohort of patients with pulmonary tuberculosis in North Carolina from 1 January 2011 to 31 December 2014. Tuberculosis-related terminology was categorised into four mutually exclusive categories. The primary outcomes of interest w

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34

Thomas, Merlin, and Mushtak AlGherbawe. "Acute Myeloid Leukemia Presenting with Pulmonary Tuberculosis." Case Reports in Infectious Diseases 2014 (2014): 1–4. http://dx.doi.org/10.1155/2014/865909.

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We report the case of a 58-year-old immunocompetent man presenting with fever, cough, anorexia, weight loss, and cervical lymphadenopathy. Blood investigations revealed severe neutropenia with monocytosis. Chest imaging showed bilateral reticular infiltrates with mediastinal widening. Bronchoalveolar lavage culture and molecular test were positive forMycobacterium tuberculosisand treatment with isoniazid, rifampicin, pyrazinamide, and ethambutol was started. Although pulmonary tuberculosis could explain this clinical presentation we suspected associated blood dyscrasias in view of significant

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35

Sanzhakov, M. A., O. M. Ipatova, T. I. Torkhovskaya, et al. "NANOPARTICLES AS DRUG DELIVERY SYSTEM FOR ANTITUBERCULOUS DRUGS." Annals of the Russian academy of medical sciences 68, no. 8 (2013): 37–44. http://dx.doi.org/10.15690/vramn.v68i8.722.

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The increase of tuberculosis incidence in last decade stimulated elaboration of both new antituberculous drugs and also searches of optimizing delivery systems for existing drugs. It is determined by their side effects and low bioavailability of effective first line drug rifampicin. Various nanosystems for transport of antituberculous drugs are considered on the basis of various polymers, liposomes, lipid nanoparticles, nanoemulsios, nanosuspensions, dendrimers, cyclodextrines. Influence of drug incorporation into nanoparticles, most often for rifampicin, on pharmacokinetics and efficiency in

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36

Iqbal, Javed, and Muzamil Nazir. "TUBERCULOSIS; RELAPSE IN INTERRUPTED ANTITUBERCULOUS THERAPY." PROFESSIONAL MEDICAL JOURNAL 24, no. 07 (2017): 942–46. http://dx.doi.org/10.17957/tpmj/17.3810.

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37

Han, Sang Hoon, Geun Young Cha, Young Mok Lee, et al. "Study of Antituberculous Medications in Anthracofibrosis." Tuberculosis and Respiratory Diseases 51, no. 3 (2001): 224. http://dx.doi.org/10.4046/trd.2001.51.3.224.

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38

NAGARE, Hitoshi, Yuko KITANO, and Shin KAWAHARA. "In Vitro Antituberculous Activity of Levofloxacin." Journal of the Japanese Association for Infectious Diseases 68, no. 6 (1994): 794–95. http://dx.doi.org/10.11150/kansenshogakuzasshi1970.68.794.

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39

Scalcini, Marcella, George Carré, Michel Jean-Baptiste, et al. "Antituberculous Drug Resistance in Central Haiti." American Review of Respiratory Disease 142, no. 3 (1990): 508–11. http://dx.doi.org/10.1164/ajrccm/142.3.508.

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40

Ulubelen, Ayhan, Gülaçtì Topcu, and Candan Bozok Johansson. "Norditerpenoids and Diterpenoids fromSalviamulticauliswith Antituberculous Activity." Journal of Natural Products 60, no. 12 (1997): 1275–80. http://dx.doi.org/10.1021/np9700681.

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41

Durand, F., D. Pessayre, M. Fournier, et al. "Antituberculous therapy and acute liver failure." Lancet 345, no. 8958 (1995): 1170–72. http://dx.doi.org/10.1016/s0140-6736(95)91000-x.

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42

Toyoshima, Mikio, Kingo Chida, Takafumi Suda, Shiro Imokawa, and Hirotoshi Nakamura. "Wegener's Granulomatosis Responding to Antituberculous Drugs." Chest 119, no. 2 (2001): 643–45. http://dx.doi.org/10.1378/chest.119.2.643.

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43

Noble, Ann, S. L. Janes, and J. Behrens. "Antituberculous therapy and acute liver failure." Lancet 345, no. 8953 (1995): 867. http://dx.doi.org/10.1016/s0140-6736(95)93007-8.

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44

Richards, E. M., J. M. Shneerson, and T. P. Baglin. "Thrombocytopenia responding to empirical antituberculous therapy." Clinical & Laboratory Haematology 16, no. 1 (2008): 89–90. http://dx.doi.org/10.1111/j.1365-2257.1994.tb00392.x.

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45

Zaslavsky, Denis V., S. S. Yakushenko, M. A. Koroleva, I. N. Chuprov, and A. A. Sidikov. "ERYTHRODERMA AND ANTITUBERCULOUS THERAPY: CLINICAL CASE." Russian Journal of Skin and Venereal Diseases 20, no. 6 (2017): 342–46. http://dx.doi.org/10.18821/1560-9588-2017-20-6-342-346.

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Erythroderma represents one of the most complex problems of dermatology. First of all, it is explained by variety of reasons, a pathogenesis ambiguity, monotonous clinical picture, tendency of many forms of an erythroderma to a long and heavy current and resistance to therapy. The erythroderma doesn’t represent a certain disease, it is, most likely, clinical implication of set of various diseases. The majority of erythroderma cases described in literature have been induced by different types of medicines and dietary supplements. In English-speaking literature more than ten cases of erythroderm

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46

NAYYAR, V., B. RAMAKRISHNA, G. MATHEW, R. R. WILLIAMS, and P. KHANDURI. "Response to antituberculous chemotherapy after splenectomy." Journal of Internal Medicine 233, no. 1 (1993): 81–83. http://dx.doi.org/10.1111/j.1365-2796.1993.tb00653.x.

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47

Seaman, John M., Marian Goble, Lorie Madsen, and James C. Steigerwald. "Fasciitis and polyarthritis during antituberculous therapy." Arthritis & Rheumatism 28, no. 10 (1985): 1179–84. http://dx.doi.org/10.1002/art.1780281017.

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48

Kulchavenya, E. "P303 Specific complication of antituberculous chemotherapy." International Journal of Antimicrobial Agents 42 (June 2013): S137—S138. http://dx.doi.org/10.1016/s0924-8579(13)70544-2.

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49

Mavlyanov, Ibragim Kamilovich. "NEUTROTOXIC SIDE EFFECTS OF ANTITUBERCULOUS DRUGS." INTERNATIONAL BULLETIN OF MEDICAL SCIENCES AND CLINICAL RESEARCH 3, no. 5 (2023): 97–99. https://doi.org/10.5281/zenodo.7931820.

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Data from 36 literature sources on the neurotoxic properties of anti-tuberculosis drugs, their clinical manifestations and mechanisms of neurotoxic action were analyzed. Predisposing factors to the development of neurotoxicity and risk groups have been identified. The necessity of early detection of neurotoxicity of chemotherapy regimens for timely correction and full treatment of patients is substantiated.

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50

Thomsen, Vibeke Østergaard, Axel Kok-Jensen, Mauro Buser, Sabine Philippi-Schulz, and H. J. Burkardt. "Monitoring Treatment of Patients with Pulmonary Tuberculosis: Can PCR Be Applied?" Journal of Clinical Microbiology 37, no. 11 (1999): 3601–7. http://dx.doi.org/10.1128/jcm.37.11.3601-3607.1999.

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To assess whether PCR is applicable for monitoring the efficacy of antituberculous treatment, respiratory specimens obtained during treatment and follow-up from sputum smear-positive tuberculosis (TB) patients were examined. First, results of smear, culture, and PCR forMycobacterium tuberculosis complex (MTB) and an internal inhibition control (MCC) were correlated retrospectively on 1,601 respiratory specimens from patients with no previous cultures of MTB. MTB optical density (OD) values increased to a maximum level of 3.5 to 4.0, with both increasing numbers of acid-fast bacilli and CFU. MT

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