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Journal articles on the topic 'Aortic override'

Author: Grafiati

Published: 5 June 2025

Last updated: 2 August 2025

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1

Anderson, Robert H., Diane E. Spicer, G. William Henry, Cynthia Rigsby, Anthony M. Hlavacek, and Timothy J. Mohun. "What is aortic overriding?" Cardiology in the Young 25, no. 4 (2014): 612–25. http://dx.doi.org/10.1017/s1047951114001139.

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AbstractBackground: Disagreement currently exists regarding the definition of aortic dextroposition. It is suggested that the term be used interchangeably with aortic overriding, along with suggestions that the aortic valve overrides in the normal heart. The dextroposed aorta, however, does not always override the crest of the muscular ventricular septum. It is incorrect to argue that the normal aortic valve overrides. It is the cavity of the right aortic valvar sinus, rather than the valvar orifice, that sits above the muscular septum when the septum itself is intact. Therefore, to circumvent

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2

Khan, Saad M., Nigel E. Drury, John Stickley, et al. "Tetralogy of Fallot: morphological variations and implications for surgical repair." European Journal of Cardio-Thoracic Surgery 56, no. 1 (2019): 101–9. http://dx.doi.org/10.1093/ejcts/ezy474.

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AbstractOBJECTIVESTetralogy of Fallot is characterized by anterocephalad deviation of the outlet septum, along with abnormal septoparietal trabeculations, which lead to subpulmonary infundibular stenosis. Archives of retained hearts are an important resource for improving our understanding of congenital heart defects and their morphological variability. This study aims to define variations in aortic override, coronary arterial patterns and ventricular septal defects in tetralogy of Fallot as observed in a morphological archive, highlighting implications for surgical management.METHODSThe Birmi

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3

Elsaka, O., M. A. Noureldean, Gamil M. Adel, M. T. Ghazali, Al-Razik A. H. Abd, and D. Hisham. "Tetralogy of Fallot: Diagnosis and Management." Asian Journal of Research in Medicine and Medical Science 4, no. 1 (2022): 18–31. https://doi.org/10.5281/zenodo.7636348.

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Tetralogy of Fallot is a complex heart defect characterized by the presence of a multilevel right ventricular outflow pathway, resulting in right ventricular hypertrophy and misalignment of the ventricular septal defect with aortic overlap. The pulmonary valve is usually small and dysplastic, as is the pulmonary artery. In Tetralogy of Fallot with pulmonary atresia, it represents the most advanced form of Tetralogy of Fallot, the pulmonary stress valve. Tetralogy of Fallot with the absence of a pulmonary valve is a rare form of the disease, in which only inactive remains of the pulmonary valve

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4

YEN, Chia-Hung, та Ying-Tung LAU. "17β-Oestradiol enhances aortic endothelium function and smooth muscle contraction in male spontaneously hypertensive rats". Clinical Science 106, № 5 (2004): 541–46. http://dx.doi.org/10.1042/cs20030334.

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17β-Oestradiol (E2; oestrogen) is known to increase endothelium-dependent vasodilatation and NO (nitric oxide) production. It is also known to decrease the response of VSMCs (vascular smooth muscle cells) to vasoconstrictors in vitro. E2 induces a decrease in age-related BP (blood pressure) development in MSHR (male spontaneously hypertensive rats). Whether this decrease is due to an effect on the endothelium or smooth muscle is unknown. To determine this effect, we examined the role of E2 on vascular endothelium and smooth muscle separately. We treated MSHR with E2 (2 mg·kg-1 of body weight·w

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5

Silverman, Norman H. "The essential echocardiographic features of tetralogy of Fallot." Cardiology in the Young 23, no. 6 (2013): 871–82. http://dx.doi.org/10.1017/s1047951113001704.

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AbstractThis presentation will demonstrate the essential features of tetralogy of Fallot in the infant and child before surgery, as well as some noteworthy features in the foetus. The four features, namely, subpulmonary stenosis, ventricular septal defect, aortic override, and right ventricular hypertrophy, can all be easily demonstrated by echocardiography. In addition, morphology of the pulmonary valve and the main and branch pulmonary arteries can be seen. The position of the coronary arteries and the major variants of proximal coronary anatomy can be defined. The arch anatomy and the prese

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Pump, Bettina, Tsutomu Kamo, Anders Gabrielsen, Peter Bie, Niels Juel Christensen, and Peter Norsk. "Central volume expansion is pivotal for sustained decrease in heart rate during seated to supine posture change." American Journal of Physiology-Heart and Circulatory Physiology 281, no. 3 (2001): H1274—H1279. http://dx.doi.org/10.1152/ajpheart.2001.281.3.h1274.

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During prolonged, static carotid baroreceptor stimulation by neck suction (NS) in seated humans, heart rate (HR) decreases acutely and thereafter gradually increases. This increase has been explained by carotid baroreceptor adaptation and/or buffering by aortic reflexes. During a posture change from seated to supine (Sup) with similar carotid stimulation, however, the decrease in HR is sustained. To investigate whether this discrepancy is caused by changes in central blood volume, we compared ( n = 10 subjects) the effects of 10 min of seated NS (adjusted to simulate carotid stimulation of a p

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Pitra, Dian Ayu Hamama, and Lydia Susanti. "Ischemic Stroke In infant With Tetralogy of Fallot." Health & Medical Journal 1, no. 1 (2019): 64–70. http://dx.doi.org/10.33854/heme.v1i1.221.

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Objectives: Although stroke is often viewed as occurring primarily in the elderly, it also strikes infants, children, young adults. Stroke in children are rare but increasingly important conditions due to the severity of their complications and diverse diagnostic differentials. It is not yet clearly understood and with multifactorial etiologies. When it comes to stroke, children are not just little adults. Stroke risk factors, symptoms, prevention efforts, and treatment are often different in children than in adults. Up to 80 % of children with ischemic stroke have cardiovascular disease. Case

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Hinson, Jeremiah S., Matthew D. Medlin, Kashelle Lockman, Joan M. Taylor, and Christopher P. Mack. "Smooth muscle cell-specific transcription is regulated by nuclear localization of the myocardin-related transcription factors." American Journal of Physiology-Heart and Circulatory Physiology 292, no. 2 (2007): H1170—H1180. http://dx.doi.org/10.1152/ajpheart.00864.2006.

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On the basis of our previous studies on RhoA signaling in smooth muscle cells (SMC), we hypothesized that RhoA-mediated nuclear translocalization of the myocardin-related transcription factors (MRTFs) was important for regulating SMC phenotype. MRTF-A protein and MRTF-B message were detected in aortic SMC and in many adult mouse organs that contain a large SMC component. Both MRTFs upregulated SMC-specific promoter activity as well as endogenous SM22α expression in multipotential 10T1/2 cells, although to a lesser extent than myocardin. We used enhanced green fluorescent protein (EGFP) fusion

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9

Schmit, Bradley M., Pu Yang, Chunhua Fu, Kenneth DeSart, Scott A. Berceli, and Zhihua Jiang. "Hypertension overrides the protective effect of female hormones on the development of aortic aneurysm secondary to Alk5 deficiency via ERK activation." American Journal of Physiology-Heart and Circulatory Physiology 308, no. 2 (2015): H115—H125. http://dx.doi.org/10.1152/ajpheart.00521.2014.

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The prevalence of aortic aneurysm is five times higher in men than women among the general population. Similar sexual dimorphism also exists in syndromic aortic aneurysms triggered by TGF-β signaling disorders. To understand the responsible mechanisms, we developed an animal model where inducible deletion of the type I TGF-β receptor, Alk5, specifically in smooth muscle cells ( Alk5 iko) causes spontaneous aortic aneurysm formation. This model recapitulated an extreme scenario of the dimorphism in aortic aneurysm development between genders. In a comparative experiment, all Alk5 iko males ( n

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Bowler, Meghan A., Matthew R. Bersi, Larisa M. Ryzhova, Rachel J. Jerrell, Aron Parekh, and W. David Merryman. "Cadherin-11 as a regulator of valve myofibroblast mechanobiology." American Journal of Physiology-Heart and Circulatory Physiology 315, no. 6 (2018): H1614—H1626. http://dx.doi.org/10.1152/ajpheart.00277.2018.

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Cadherin-11 (CDH11) is upregulated in a variety of fibrotic diseases, including arthritis and calcific aortic valve disease. Our recent work has identified CDH11 as a potential therapeutic target and shown that treatment with a CDH11 functional blocking antibody can prevent hallmarks of calcific aortic valve disease in mice. The present study investigated the role of CDH11 in regulating the mechanobiological behavior of valvular interstitial cells believed to cause calcification. Aortic valve interstitial cells were harvested from Cdh11+/+, Cdh11+/−, and Cdh11−/− immortomice. Cells were subjec

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Caron, Alexis, Patrick Menu, Beatrice Faivre-Fiorina, Pierre Labrude, Abdu I. Alayash, and Claude Vigneron. "Cardiovascular and hemorheological effects of three modified human hemoglobin solutions in hemodiluted rabbits." Journal of Applied Physiology 86, no. 2 (1999): 541–48. http://dx.doi.org/10.1152/jappl.1999.86.2.541.

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The cardiovascular effects of human albumin (Alb) and three human hemoglobin (Hb) solutions, dextran-benzene-tetracarboxylate Hb, αα-crosslinked Hb, and o-raffinose-polymerized Hb were compared in anesthetized rabbits undergoing acute isovolemic hemodilution with Hct reduction from 41.4 ± 2.7 to 28.8 ± 1.6%. The impact of the vasoconstricting properties of Hb was examined by measuring heart rate (HR), mean arterial pressure (MAP), abdominal aortic, and femoral arterial blood flow, vascular resistance (VR), and aortic distension during the first 3 h after hemodilution. The impact of the hemorhe

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Zhang, Z. Q., C. Julien, M. P. Gustin, C. Cerutti, and C. Barres. "Hemodynamic analysis of arterial pressure lability in sympathectomized rat." American Journal of Physiology-Heart and Circulatory Physiology 267, no. 1 (1994): H48—H56. http://dx.doi.org/10.1152/ajpheart.1994.267.1.h48.

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Male Sprague-Dawley rats, treated with either saline (n = 14) or guanethidine (n = 12) from 1 to 13 wk of age, were instrumented for the measurement of mean arterial pressure (MAP) and indexes of regional blood flows (pulsed Doppler flow probes) in the subdiaphragmatic aorta, superior mesenteric artery, and distal aorta (hindquarters). Hemodynamic parameters were simultaneously recorded in the conscious rats on a beat-to-beat basis by a computer. Chronic sympathectomy did not change the MAP level but nearly doubled its spontaneous variability, mainly due to large decreases in MAP associated wi

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Rolland, Pierre-Henri, Pascal de Lagausie, Euletheris Stathopoulos, et al. "Phasic hemodynamics and reverse blood flows in the aortic isthmus and pulmonary arteries of preterm lambs with pulmonary vascular dysfunction." American Journal of Physiology-Heart and Circulatory Physiology 295, no. 6 (2008): H2231—H2241. http://dx.doi.org/10.1152/ajpheart.00410.2008.

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Time-domain representations of the fetal aortopulmonary circulation were carried out in lamb fetuses to study hemodynamic consequences of congenital diaphragmatic hernia (CDH) and the effects of endothelin-receptor antagonist tezosentan (3 mg/45 min). From the isthmic aortic and left pulmonary artery (PA) flows (Q) and isthmic aortic, PA, and left auricle pressures (P) on day 135 in 10 controls and 7 CDH fetuses (28 ewes), discrete-triggered P and Q waveforms were modelized as P t and Q t functions to obtain basic hemodynamic profiles, pulsatile waves [P, Q, and entry impedance ( Ze)], and P a

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Elsaka, O., M. A. Noureldean, M. A. Gamil, M. T. Ghazali, A. H. A. Al-Razik, and D. Hisham. "Truncus Arteriosus: Pathophysiology, Investigations, and Treatment." Asian Basic and Applied Research Journal 4, no. 2 (2021): 220–36. https://doi.org/10.5281/zenodo.7632101.

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<strong>Background:</strong> The failure of the primitive arterial trunk to divide into the aorta and the pulmonary artery causes persistent truncus arteriosus, an uncommon heart abnormality. As a result, the heart has only one outflow, which is an arterial vessel that supplies the systemic, pulmonary, and coronary circulations. The truncal valve serves both ventricles and overrides the ventricular septal defect at the output. Truncal valve dysplasia is common, resulting in stenosis and/or regurgitation in variable degrees. Three forms of truncus arteriosus can be recognized based on the

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15

King, Jackson M., Morgan T. Black, Ruyun Jin, et al. "Clinical Factors and Outcomes When Real-World Heart Teams Overruled STS Risk Scores in TAVR Cases." Journal of Interventional Cardiology 2022 (June 25, 2022): 1–9. http://dx.doi.org/10.1155/2022/9926423.

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Objectives. This study was conducted to determine why heart teams recommended transcatheter aortic valve replacement (TAVR) versus surgical AVR (SAVR) for patients at low predicted risk of mortality (PROM) and describe outcomes of these cases. Background. Historically, referral to TAVR was based predominately on the Society of Thoracic Surgeons (STS) risk model’s PROM >3%. In selected cases, heart teams had latitude to overrule these scores. The clinical reasons and outcomes for these cases are unclear. Methods. Retrospective data were gathered for all TAVR and SAVR cases conducted by 9 hos

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16

Garcia-Arango, Mariana, Shravya Vinnakota, and Hector R. Villarraga. "Abstract 17189: Radiation-induced Accelerated Multi-valve Disease In A Patient With Subaortic Membrane." Circulation 144, Suppl_2 (2021). http://dx.doi.org/10.1161/circ.144.suppl_2.17189.

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A 69-year-old woman with history of right breast cancer status post lumpectomy and radiation (4.2 cGy) 18 months prior, presented with progressive NYHA class III dyspnea. TTE obtained prior to her cancer diagnosis was notable for normal LV size and wall thickness, a subaortic membrane with systolic mean Doppler gradient of 21 mmHg and a mildly thickened mitral valve without significant stenosis or regurgitation. On presentation, TTE was notable for severely increased concentric LV wall thickness (LV mass index 140 g/m 2 ), preserved ejection fraction, increased RV wall thickness, subaortic ste

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Bates, Oliver, Thomas Semple, Sylvia Krupickova, and Carles Bautista-Rodriguez. "Case report of a Gore-Tex TCPC conduit dissection causing severe stenosis." European Heart Journal - Case Reports 5, no. 11 (2021). http://dx.doi.org/10.1093/ehjcr/ytab377.

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Abstract Background The patient is a 15-year-old male with situs inversus, dextrocardia, bilateral superior caval veins, atrioventricular discordance with a single outlet, large perimembranous ventricular septal defect, aortic override, pulmonary atresia, and right aortic arch. The complex anatomy with a Ventricular Septal Defect (VSD) distant from the aorta (unsuitable for baffling to the aorta) meant he was unsuitable for biventricular repair and proceeded down a univentricular palliation pathway. Case summary Post-total cavopulmonary connection his clinical course was uneventful until the a

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Thomas, Julia, Sophie Marshall, Karin Gormley, Georgia Conway, and Kieran Borgeat. "Does medical or surgical treatment for aortic stenosis improve outcome in dogs?" Veterinary Evidence 6, no. 2 (2021). http://dx.doi.org/10.18849/ve.v6i2.368.

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PICO question In adult dogs with aortic stenosis does treatment with beta blockers compared with surgical intervention show a longer survival time with improved clinical parameters?  Clinical bottom line Category of research question Treatment The number and type of study designs reviewed Eight studies were reviewed. One was a randomised controlled study, three were cohort studies, one was a case series and three were case reports Strength of evidence Moderate to weak Outcomes reported Intervention appeared to improve survival t

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Chowdhury, Ujjwal Kumar, Robert H. Anderson, Diane E. Spicer, et al. "The Surgical Significance of Phenotypic Variability in the Setting of Tetralogy of Fallot." World Journal for Pediatric and Congenital Heart Surgery, September 26, 2024. http://dx.doi.org/10.1177/21501351241274731.

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The phenotypic feature of tetralogy of Fallot is anterocephalad deviation of the muscular outlet septum, or its fibrous remnant, relative to the septoparietal trabeculation, coupled with hypertrophy of septoparietal trabeculations. Although this feature permits recognition of the entity, no two cases are identical. Once diagnosed, treatment is surgical. The results of surgical treatment have improved remarkably over recent decades. The results are now sufficiently excellent, including those in the developing world, that attention is now directed toward avoidance of morbidity, while still seeki

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20

Sabarigirivasan, V., R. S. Sivera, A. C. Crucean, and A. C. Cook. "The ventricular septal defect in tetralogy of Fallot: A multi-centre morphological and statistical shape analysis study." European Heart Journal 45, Supplement_1 (2024). http://dx.doi.org/10.1093/eurheartj/ehae666.2110.

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Abstract Background Ventricular septal defects (VSDs) in Tetralogy of Fallot (TOF) are often thought to be ‘large and unrestricted’ but experience suggests they have a more heterogenous morphology. Understanding the morphological variations and dimensions is the first step in modifying surgical closure or optimising potential device design. Purpose The objective of this study was to perform a comprehensive multi-center morphological analysis of the characteristics of the VSD in TOF. Additionally, we sought to perform a preliminary 2D shape analysis and principal component analysis (PCA) on sta

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Vico, T., M. Taglinger, V. Haacke, et al. "Recruited macrophages incorporate into the resident cardiac macrophage pool and converge into a common phenotype in the healing infarct, but not in the failing heart exposed to pressure overload." European Heart Journal 44, Supplement_2 (2023). http://dx.doi.org/10.1093/eurheartj/ehad655.3083.

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Abstract Introduction Resident macrophages account for 5% of the cells in the healthy heart. In response to cardiac injury, monocytes infiltrate and differentiate into recruited macrophages which function distinctly from the resident subset of primarily embryonic origin. In this work, we aim to identify the extent to which macrophage origin, tissue location and type of cardiac injury determine macrophage phenotypes in ischemic and non-ischemic cardiac injuries over time. Results and methods We use a tamoxifen inducible CX3CR1 Yfp CreER/+ :R26tdT/+ mouse line to visualize and quantify fluxes of

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Vico, T., G. Witte, M. Taglinger, et al. "Macrophage transcriptome changes in response to cardiac injuries are determined by the type of lesion rather than their ontogenetic nature." Cardiovascular Research 120, Supplement_1 (2024). http://dx.doi.org/10.1093/cvr/cvae088.136.

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Abstract Funding Acknowledgements Type of funding sources: Public grant(s) – National budget only. Main funding source(s): CRC1425 - DFG Introduction Resident macrophages account for 5% of the cells in the healthy heart. In response to cardiac injury, monocytes infiltrate and differentiate into recruited macrophages complementing the original resident macrophage population, and together orchestrate cardiac remodeling. We aim to identify the extent to which macrophage origin, tissue location and type of cardiac injury determine macrophage phenotypes in ischemic and non-ischemic cardiac injuries

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23

PV, Anu, Shubham Haribhau Mehatre, Catherine M. Verfaillie, Mohammad Tauqeer Alam, and Satish Khurana. "Glycolytic state of aortic endothelium favors hematopoietic transition during the emergence of definitive hematopoiesis." Science Advances 10, no. 7 (2024). http://dx.doi.org/10.1126/sciadv.adh8478.

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The first definitive hematopoietic progenitors emerge through the process of endothelial-to-hematopoietic transition in vertebrate embryos. With molecular regulators for this process worked out, the role of metabolic pathways used remains unclear. Here, we performed nano–LC-MS/MS–based proteomic analysis and predicted a metabolic switch from a glycolytic to oxidative state upon hematopoietic transition. Mitochondrial activity, glucose uptake, and glycolytic flux analysis supported this hypothesis. Systemic inhibition of lactate dehydrogenase A (LDHA) increased oxygen consumption rate in the he

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"Pulmonary valve replacement for treatment of severe pulmonary regurgitation in patients with previous tetralogy of Fallot repair surgery." Multimedia Manual of Cardio-Thoracic Surgery, April 12, 2023. http://dx.doi.org/10.1510/mmcts.2023.017.

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Tetralogy of Fallot was classically described as the combination of pulmonary stenosis, right ventricle hypertrophy, perimembranous ventricular septal defect and an aortic root that overrides the ventricular septal defect. Full surgical repair, which is usually indicated in toddlers, comprises closure of the ventricular septal defect and alleviation of the pulmonary stenosis. Because there is a wide anatomical spectrum, the cause and severity of the pulmonary stenosis are variable. However, when the pulmonary valve is stenotic and not well-developed, it has to be removed, and one has to accept

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Kothari, Vishal, Baohai Shao, Yi He, et al. "Abstract 118: Cholesteryl Ester Transfer Protein Overrides The Protective Effect Of APOA1 On Arterial APOB Lipoprotein Trapping And Atherosclerosis Progression In A Mouse Model Of Type 1 Diabetes." Arteriosclerosis, Thrombosis, and Vascular Biology 42, Suppl_1 (2022). http://dx.doi.org/10.1161/atvb.42.suppl_1.118.

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Despite the benefits of LDL-cholesterol-reducing medications, patients with type 1 diabetes (T1D) continue to have an increased risk of incident CVD. Preclinical and clinical observations indicate that triglyceride-rich lipoproteins (TRLs) and their remnants may contribute significantly to CVD risk in T1D. Recently, we have shown that diabetes increases levels of atherogenic TRL remnants and that improvement of remnant clearance halts atherosclerosis in a mouse model of T1D. Cholesteryl ester transfer protein (CETP), which is normally absent in mice, links metabolism of TRLs to that of HDL by

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Laakkonen, Eija K., Jari E. Karppinen, Satu Lehti, et al. "Associations of Sex Hormones and Hormonal Status With Arterial Stiffness in a Female Sample From Reproductive Years to Menopause." Frontiers in Endocrinology 12 (November 30, 2021). http://dx.doi.org/10.3389/fendo.2021.765916.

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ObjectiveLoss of sex hormones has been suggested to underlie menopause-associated increment in cardiovascular risk. We investigated associations of sex hormones with arterial stiffness in 19–58-years-old women. We also studied associations of specific hormonal stages, including natural menstrual cycle, cycle with combined oral contraceptives (COC) and menopausal status with or without hormone therapy (HT), with arterial stiffness.MethodsThis study includes repeated measurements of 65 healthy women representing reproductive (n=16 natural, n=10 COC-users) and menopause (n=5 perimenopausal, n=26

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Panico, Carolina, Earl H. Rudolph, Roland C. Blantz, Giovambattista Capasso, Christopher S. Wilcox, and William J. Welch. "Abstract 224: Differential Actions of Angiotensin II on Proximal Tubule Transport." Hypertension 60, suppl_1 (2012). http://dx.doi.org/10.1161/hyp.60.suppl_1.a224.

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Studies with the proximal tubule-specific angiotensin type 1 receptor (AT 1 R) knockout mouse have shown that the proximal tubule (PT) contributes to Ang II-induced hypertension. Ang II levels in the PT fluid are 100- to 1000-fold higher than in plasma, yet the function of the AT 1 -Rs on the luminal side of the PT is unclear. Moreover, hemodynamic actions of Ang II that raise the filtration fraction and the peritubular fluid uptake forces also stimulate PT fluid uptake. We tested the effects of Ang II and AT 1 -Rs on PT fluid uptake in rats. Ang II (200 ng/kg/min) delivered for 2 weeks increa

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