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Relevant bibliographies by topics / Aortic wall thickness and media as well as advential layers

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Academic literature on the topic 'Aortic wall thickness and media as well as advential layers'

Author: Grafiati

Published: 7 June 2025

Last updated: 2 August 2025

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Journal articles on the topic "Aortic wall thickness and media as well as advential layers"

1

Mahaparea, APK., G. Srinivas, D.V. Ramanjaneyulu, et al. "An Essential Aspects of Ruprure of Aortic Aneurysm Include Etiology, Epidemiology, Path Physiology, Histo Pathology, Diagnosis and Treatment." Journal of Research and Reviews in Yoga & Ayurvedic Sciences 2, no. 1 (2025): 18–24. https://doi.org/10.5281/zenodo.15086982.

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<em>An abdominal aortic aneurysm rupture is a life-threatening emergency that carries a high risk of morbidity and death if left untreated, with an untreated mortality rate of nearly 100%. Most of them rupture into the retro</em><em> </em><em>peritoneal cavity, </em><em>making</em><em> symptoms </em><em>such as</em><em> pain, lightheadedness and a pulsating mass in the abdomen. Along with ruptured abdominal aortic aneurysms, this activity explains the etiology, clinical features, evaluation, and management of patients. Optimal outcomes rely on the coordinated effort of an interprofessional tea

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2

Stern, Christian, Bernhard Scharinger, Adrian Tuerkcan, et al. "Strong Signs for a Weak Wall in Tricuspid Aortic Valve Associated Aneurysms and a Role for Osteopontin in Bicuspid Aortic Valve Associated Aneurysms." International Journal of Molecular Sciences 20, no. 19 (2019): 4782. http://dx.doi.org/10.3390/ijms20194782.

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Central processes in the pathogenesis of TAV- (tricuspid aortic valve) and BAV- (bicuspid aortic valve) associated ascending thoracic aortic aneurysm (ATAA) development are still unknown. To gain new insights, we have collected aortic tissue and isolated smooth muscle cells of aneurysmal tissue and subjected them to in situ and in vitro analyses. We analyzed aortic tissue from 78 patients (31 controls, 28 TAV-ATAAs, and 19 BAV-ATAAs) and established 30 primary smooth muscle cell cultures. Analyses included histochemistry, immuno-, auto-fluorescence-based image analyses, and cellular analyses i

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3

Matsumoto, Takeo, and Kozaburo Hayashi. "Stress and Strain Distribution in Hypertensive and Normotensive Rat Aorta Considering Residual Strain." Journal of Biomechanical Engineering 118, no. 1 (1996): 62–73. http://dx.doi.org/10.1115/1.2795947.

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The effects of hypertension on the stress and strain distributions through the wall thickness were studied in the rat thoracic aorta. Goldblatt hypertension was induced by constricting the left renal artery for 8 weeks. Static pressure-diameter-axial force relations were determined on excised tubular segments. The segments were then sliced into thin ring specimens. Circumferential strain distributions were determined from the cross-sectional shape of the ring specimens observed before and after releasing residual stresses by radial cutting. Stress distributions were calculated using a logarith

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Dabagh, Mahsa, Payman Jalali, and John M. Tarbell. "The transport of LDL across the deformable arterial wall: the effect of endothelial cell turnover and intimal deformation under hypertension." American Journal of Physiology-Heart and Circulatory Physiology 297, no. 3 (2009): H983—H996. http://dx.doi.org/10.1152/ajpheart.00324.2009.

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A multilayered model of the aortic wall is introduced to investigate the transport of low-density lipoprotein (LDL) under hypertension, taking into account the influences of increased endothelial cell turnover and deformation of the intima at higher pressure. Meanwhile, the thickness and properties of the endothelium, intima, internal elastic lamina (IEL), and media are affected by the transmural pressure. The LDL macromolecules enter the intima through leaky junctions over the endothelium, which are created by dying or dividing cells. Water molecules enter the intima via the paracellular path

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5

Sokolis, Dimitrios P., Nausicaa Gouskou, Stavroula A. Papadodima, and Stavros K. Kourkoulis. "Layer-Specific Residual Deformations and Their Variation Along the Human Aorta." Journal of Biomechanical Engineering 143, no. 9 (2021). http://dx.doi.org/10.1115/1.4050913.

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Abstract This study described the regional distribution of layer-specific residual deformations in fifteen human aortas collected during autopsy. Circumferentially and axially cut strips of standardized dimensions from the anterior quadrant of nine consecutive aortic levels were photographed to obtain the zero-stress state for the intact wall. The strips were then dissected into layers that were also photographed to obtain their zero-stress state. Changes in layer-specific opening angle, residual stretches, and thickness at each aortic level and direction were determined via image analysis. Th

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Böck, Marcelo José, Thiago José Fernandez, Vitor Pires Pereira, Marcelo Leite da Veiga, Claudia de Mello Bertoncheli dos Santos, and Luciano de Morais‐Pinto. "Morphological and morphometric characterization of the aorta of Brazilian horses (Equus ferus caballus, Linnaeus—1758): Comparative analysis of the aortic bulb and ascending aorta." Anatomical Record, July 31, 2025. https://doi.org/10.1002/ar.70026.

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AbstractWe examined 52 horse aortas to characterize the morphological aspects of the aortic bulb wall and the ascending aorta. The morphometric data were analyzed using ImageJ®‐Fiji 1.5 software. The Tunica intima was composed of endotheliocytes with scarce cytoplasm and ellipsoid nuclei, supported by a collagenous subendothelial stratum. The Tunica media was composed entirely of fascicles of vascular smooth muscle cells interspersed with bundles of collagen fibers and elastic lamellae, characterizing the aortic medial lamellar unit. The Tunica adventitia consisted of two distinct sub‐layers:

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7

Cao, Jiumei, Liang Geng, Qihong Wu, Lin Lu, Ying Chen, and Weifeng Shen. "Abstract 97: Spatiotemporal Expression Of Matrix Metalloproteinases (mmps) Is Regulated By Ca2+-signal Transducer S100a4 In The Thoracic Aortic Aneurysm Pathogenesis." Arteriosclerosis, Thrombosis, and Vascular Biology 32, suppl_1 (2012). http://dx.doi.org/10.1161/atvb.32.suppl_1.a97.

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Purpose: Ca 2+ signaling pathways play critical roles in a wide range of physiological functions including control and regulation of smooth muscle cell performance and vascular tone. Previous studies have indicated that Ca 2+ signaling transducer S100A4 could increase the secretion of MMPs from endothelial cells and fibroblasts and is greatly expressed in smooth muscle cells in atherosclerotic lesions. This study investigated whether S100A4 has a potential role in the formation of TAA. Methods: Thoracic aorta of male Sprague-Dawley rats was exposed to 0.5 M CaCl 2 or normal saline (NaCl). Anim

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