Relevant bibliographies by topics / Aphasia Cerebrovascular disease

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Academic literature on the topic 'Aphasia Cerebrovascular disease'

Author: Grafiati
Published: 4 June 2021
Last updated: 16 February 2022

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Journal articles on the topic "Aphasia Cerebrovascular disease"

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Hinckley, Jacqueline. "Clinical Decision-Making for Stroke and Aphasia in the Older Adult." Perspectives on Gerontology 14, no. 1 (2009): 4–11. http://dx.doi.org/10.1044/gero14.1.4.

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Abstract A patient with aphasia that is uncomplicated by other cognitive abilities will usually show a primary impairment of language. The frequency of additional cognitive impairments associated with cerebrovascular disease, multiple (silent or diagnosed) infarcts, or dementia increases with age and can complicate a single focal lesion that produces aphasia. The typical cognitive profiles of vascular dementia or dementia due to cerebrovascular disease may differ from the cognitive profile of patients with Alzheimer's dementia. In order to complete effective treatment selection, clinicians must know the cognitive profile of the patient and choose treatments accordingly. When attention, memory, and executive function are relatively preserved, strategy-based and conversation-based interventions provide the best choices to target personally relevant communication abilities. Examples of treatments in this category include PACE and Response Elaboration Training. When patients with aphasia have co-occurring episodic memory or executive function impairments, treatments that rely less on these abilities should be selected. Examples of treatments that fit these selection criteria include spaced retrieval and errorless learning. Finally, training caregivers in the use of supportive communication strategies is helpful to patients with aphasia, with or without additional cognitive complications.
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Koh, Ji-yoo, Ah-hyu Son, and Hyeon-su Shin. "Review of Clinical Research about the Treatment of Aphasia after Cerebrovascular Disease." Journal of Internal Korean Medicine 39, no. 6 (2018): 1105–15. http://dx.doi.org/10.22246/jikm.2018.39.6.1105.

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Ramasubbu, Rajamannar, and Sidney H. Kennedy. "Factors Complicating the Diagnosis of Depression in Cerebrovascular Disease, Part Ii — Neurological Deficits and Various Assessment Methods*." Canadian Journal of Psychiatry 39, no. 10 (1994): 601–7. http://dx.doi.org/10.1177/070674379403901004.

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Neurological deficits associated with cerebrovascular disease such as aphasia, dementia, anosognosia and aprosodia may impair the ability to express or experience depressive symptoms. Identification of depression in the absence of verbal report on subjective mood state is a difficult task. The value of various diagnostic methods including depressive rating scales, standard psychiatric interviews and biological variables in the diagnosis of depression in cerebrovascular disease is considered. This review concludes by focusing on the deficiencies of existing approaches in the diagnostic assessment of depression in patients with severe communication and comprehension deficits and emphasizes the importance of devising a standard diagnostic method with less reliance on verbal responses.
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Spanou, Ioanna, Maria-Eleftheria Evangelopoulos, Georgios Velonakis, et al. "Moyamoya Disease May Mimic Multiple Sclerosis?" Case Reports in Neurological Medicine 2019 (May 2, 2019): 1–4. http://dx.doi.org/10.1155/2019/1276950.

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Introduction. A wide range of medical conditions may mimic multiple sclerosis. Among them, cerebrovascular diseases, including moyamoya disease, need to be excluded since they share common clinical features and radiographic findings with multiple sclerosis. Case Report. A 44-year-old woman experienced transient numbness of her right sided face and arm and was referred to our unit due to small brain lesions in magnetic resonance imaging, with a possible diagnosis of multiple sclerosis. Neurological examination was unremarkable except for plantar reflexes and jerky deep tendon reflexes. Brain magnetic resonance angiography revealed findings typically seen in moyamoya disease, confirmed with digital subtraction angiography. Antiplatelet therapy started, but few days later, she developed suddenly global aphasia and right hemiparesis (National Institutes of Health Stroke Scale/NIHSS 6). Brain magnetic resonance imaging revealed acute infarct in the distribution of the left middle cerebral artery. At her discharge, she was significantly improved (NIHSS 3). Conclusion. Diagnosis of multiple sclerosis is often challenging. In particular, in young patients with transient neurological symptoms and atypical white matter lesions in magnetic resonance imaging, cerebrovascular disorders such as moyamoya disease should be considered in the differential diagnosis. Detailed clinical and neuroimaging evaluation are mandatory for the correct diagnosis.
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VITTURI, Bruno Kusznir, and Rubens José GAGLIARDI. "Post-stroke aphasia in famous writers: when Neurology left geniuses speechless." Arquivos de Neuro-Psiquiatria 79, no. 3 (2021): 251–53. http://dx.doi.org/10.1590/0004-282x-anp-2020-0282.

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ABSTRACT Aphasia is a frequent and devastating stroke complication that does not spare even great writers. In these cases, not only one of the highest cognitive functions is suddenly lost but also the act of bringing beauty into the world. Herein, we discuss the case of three writers who had to abandon their art compulsorily due to a cerebrovascular disease: Charles Baudelaire, Ralph Waldo Emerson, and Stendhal. They were magnificent writers, united by excellence in literature and an inevitable destiny that restricted their art to just a few words. They are also examples of the proximity of Neurology to Art, History, and Literature.
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Schmugge, Markus, Hannes Frischknecht, Yasuhiro Yonekawa, Ralf W. Baumgartner, Eugen Boltshauser, and James Humbert. "Stroke in hemoglobin (SD) sickle cell disease with moyamoya: successful hydroxyurea treatment after cerebrovascular bypass surgery." Blood 97, no. 7 (2001): 2165–67. http://dx.doi.org/10.1182/blood.v97.7.2165.

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Abstract An 11-year-old boy with hemoglobin sickle disease (HbSD), bilateral stenosis of the intracranial carotid arteries, and moyamoya syndrome had recurrent ischemic strokes with aphasia and right hemiparesis. His parents (Jehovah's Witnesses) refused blood transfusions. After bilateral extracranial–intracranial (EC-IC) bypass surgery, hydroxyurea treatment increased hemoglobin F (HbF) levels to more than 30%. During a follow-up of 28 months, flow velocities in the basal cerebral arteries remained stable, neurologic sequelae regressed, and ischemic events did not recur. This is the first report of successful hydroxyurea treatment after bypass surgery for intracranial cerebral artery obstruction with moyamoya syndrome in sickle cell disease. The patient's religious background contributed to an ethically challenging therapeutic task.
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Villain, Nicolas, and Bruno Dubois. "Alzheimer's Disease Including Focal Presentations." Seminars in Neurology 39, no. 02 (2019): 213–26. http://dx.doi.org/10.1055/s-0039-1681041.

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AbstractAlzheimer's disease (AD) is the commonest neurodegenerative disease and the most frequent cause of dementia. It affects 30 million people worldwide. Current research criteria focus on biomarkers' status for amyloid and tau using positron emission tomography and cerebrospinal fluid analysis, independent of clinical status. Current epidemiological data, which mostly rely on biomarker-undetermined AD cases, have highlighted ApoE4 and age as the main risk factors. Rare autosomal dominant mutations also account for a small fraction of early-onset AD. The main clinical phenotype at presentation is the amnestic phenotype targeting episodic memory. This is followed by rarer phenotypes such as posterior cortical atrophy, logopenic variant of primary progressive aphasia, frontal variant AD, corticobasal syndrome, and other even rarer presentations mimicking language variants of frontotemporal dementia. Main differential diagnoses include hippocampal sclerosis with TDP-43, primary age-related tauopathy, argyrophilic grain disease, frontotemporal lobar degeneration, Lewy body disease, chronic traumatic encephalopathy as well as nondegenerative disorders such as cerebrovascular disease, chronic alcohol consumption, limbic encephalitis, medial temporal lobe epilepsy, and others. Co-occurrence of AD pathology with other neurodegenerative and vascular diseases is common and increases with age. This presents a challenge in current clinical practice due to a lack of reliable biomarkers for non-AD neurodegenerative diseases.
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Bhavsar, Minal Dhairya, and Vinal Charpot. "Takayasu Arteritis - A Case Study." International Journal of Health Sciences and Research 11, no. 6 (2021): 362–66. http://dx.doi.org/10.52403/ijhsr.20210654.

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Takayasu arteritis (TA) is a chronic inflammatory disease of unknown etiology. The mechanism of this disease is not exactly defined. The inflammatory process is generally (but not exclusively) initiated in the second or third decade of life through the actions of non-specific inflammatory cells. As the disease progresses, fibrotic stenosis occurs in aorta and its main branches. The consequence of this inflammatory process can be stenosis, thrombosis, dilatation or aneurysm formation in aorta and/ or its branches. In Asia, its incidence (2.69 in a million per year) has been reported to be 100 times higher than in Europe and North America. Because of the delay in diagnosing the disease, patients often experience claudication, absence of pulses, hypertension, myocardial infarction (MI), and cerebrovascular accidents (CVAs). Accurate and early diagnosis of TA can reduce the economic, social, and psychological burdens. We present clinical, laboratory and imaging findings of a 15 years old girl with TA, who initially presented with clinical manifestations of stroke in form of weakness in Lt Side upper and lower limb and aphasia. The rarity of the disease and especially such a presentation can cause considerable delay in the diagnosis and treatment. Key words: Takayasu arteritis, Cerebrovascular accident, Infarction, physical therapy.
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Santos, Jolina Pamela, Zaher Hamadeh, and Naheed Ansari. "Cerebrovascular Accident Secondary to Paradoxical Embolism Following Arteriovenous Graft Thrombectomy." Case Reports in Nephrology 2012 (2012): 1–3. http://dx.doi.org/10.1155/2012/183730.

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Thrombectomy is a common procedure performed to declot thrombosed dialysis arteriovenous fistula (AVF) or arteriovenous graft (AVG). Complications associated with access thrombectomy like pulmonary embolism have been reported, but paradoxical embolism is extremely rare. We report a case of a 74-year-old black man with past medical history significant for end-stage renal disease (ESRD), atrial fibrillation on anticoagulation with warfarin, who presented to our hospital with lethargy, aphasia, and right-sided hemiparesis following thrombectomy of a clotted AVG. Computed tomography (CT) scan of brain showed a hypodensity within the left posterior parietal lobe. INR was 2.0 on admission. Echocardiogram revealed a normal sized left atrium with no intracardiac thrombus, and bubble study showed the presence of right-to-left shunting. These findings suggest that the stroke occurred as a result of an embolus originating from the AVG. Paradoxical cerebral embolism is uncommon but can occur after thrombectomy of clotted vascular access in ESRD patients. Clinicians and patients should be aware of this serious and potentially fatal complication of vascular access procedure.
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Kulesh, A. A., N. L. Starikova, V. E. Drobakha, L. Yu Brokhin, and A. S. Chubarov. "The syndrome of transient headache and neurologic deficits with cerebrospinal fluid lymphocytosis (HaNDL): a description of three patients." Neurology, Neuropsychiatry, Psychosomatics 12, no. 4 (2020): 65–72. http://dx.doi.org/10.14412/2074-2711-2020-4-65-72.

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Three clinical cases of the syndrome of transient headache and neurologic deficits with cerebrospinal fluid lymphocytosis (HaNDL) are first described in Russian literature. The patients were young (aged 30–35 years), had symptoms characteristic of the prodromal period of infections. In all the patients, the disease started with intense headache, followed by focal neurological symptoms: aphasia and hemihypesthesia in all cases and confusion with psychomotor agitation in two cases. All the three patients showed spontaneous recovery within 2–3 days. Perfusion computed tomography, magnetic resonance imaging, and electroencephalography are compared in one of the cases. The differential diagnosis of HaNDL with acute cerebrovascular accident, herpetic encephalitis, epilepsy, and migraine is discussed.
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Dissertations / Theses on the topic "Aphasia Cerebrovascular disease"

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狄荻. "针灸治疗中风后失语症的临床文献评估". HKBU Institutional Repository, 2015. https://repository.hkbu.edu.hk/etd_oa/138.

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中风后失语，现代医学称“急性交流障碍” ，它是急性脑血管病的常见症状之一，古代医籍中称失语为“喑痹” 、“风懿” 、“风喑” 、“风癔” 、“难言” 、“不语”等。〈素问－脉解〉云：“所谓入中为喑者’阳盛己衰，故为喑也。内夺而厥，则为喑痹。”《素问－刺禁论〉云：“……刺足少阴脉’重虚出血’为舌难以言。”《金匮要略·中风历节病脉证治〉：“邪入于腑，即不识人；邪入于脏’舌即难言’口吐涎”等。 言语障碍是脑血管疾病的常见症状，在卒中急性期，合并有失语的患者比例高达21%-38% ，据国内报道，脑血管疾病患者约25%伴有言语障碍。另外，卒中患者中除了明显失语之外，仍有60%患者在交流上存在问题。由于中风失语不仅给家庭、社会和国家带来沉重的经济负担，同时也给中风患者的身心带来了巨大伤害，故探索有效的中风失语治疗方案、提高其临床疗效、改善失语症患者的生存质量是当今亟待解决的问题。 本文对于中风后失语症的论述主要分为两大部分： 第一部分从中、西医角度认识中风后失语症。中风的病因是以正气不足、肝肾阴亏、肝阳上扰、肝风内动、血脉不通、风邪留而不去为致病之本，以风、火、痰、湿、气、血为致病之标。由此而并发的失语症主要是舌体瘫痪强直、记忆力下降健忘或认知障碍思维不清所导致，其病在心（脑），肝涉及脾、肾等脏腑。众多研究者进行了对于中风后失语症的各种治疗方法的多方面研究， 笔者收集了2010年1月至2014年12月的相失临床文献并进行了整理分析。 第二部分从针灸临床角度，对所收集到关于针灸治疗中风后失语的文献进行综合分析。运用计量学评价方法，从期刊分布、年份分布、诊断标准、随机对照、言法、脱落病例、样本量、治疗方法等多个方面， 对针灸治疗中风后失语症的研究状况做一个全面系统的分析及，并在此基础上对临床研究质量的总体水平作出评价，讨论中风后失语症的针灸临床规范研究方案，筛选有效的针灸处方，提高针灸临床科研的质量。 關键词：中风后失语症 针灸治疗
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Young, Mary Cherilyn. "Anterior aphasia as a natural category of acquired cognitive-communicative impairment implications for cognitive neurolinguistic theory, experimental methods, and clinical practice /." 2002. http://wwwlib.umi.com/cr/utexas/fullcit?p3089491.

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Cyr, Regan. "Resilience in aphasia perspectives of stroke survivors and their families /." Master's thesis, 2010. http://hdl.handle.net/10048/917.

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Thesis (M.Sc.)--University of Alberta, 2010.<br>A thesis submitted to the Faculty of Graduate Studies and Research in partial fulfillment of the requirements for the degree of Master of Science in Speech Language Pathology, Department of Speech Pathology and Audiology. Title from pdf file main screen (viewed on January 24, 2010). Includes bibliographical references.
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Books on the topic "Aphasia Cerebrovascular disease"

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Tonkonogiĭ, Iosif M. Vascular aphasia. MIT Press, 1986.

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Vascular aphasia. MIT Press, 1986.

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Portrait of aphasia. Wayne State University Press, 1985.

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1955-, Pfalzgraf Beth, ed. Pathways: Moving beyond stroke and aphasia. Wayne State University Press, 1990.

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Parr, Susie. Living with severe aphasia: The experience of communication impairment after stroke. Pavilion Publishing, 2004.

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Underwood, Helen. I need help: A stroke patient's plea : understanding simplified medical terminology and anatomy with emphasis on stroke (cerebrovascular accident of CVA) : including symptoms and concrete suggestions for patients and caregivers. Blue Dolphin Pub., 1991.

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The man who lost his language: A case of aphasia. Jessica Kingsley Publishers, 2007.

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1956-, Byng Sally, Gilpin Sue, and Ireland Chris, eds. Talking about aphasia: Living with loss of language after stroke. Open University Press, 1997.

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S, Halper Anita, ed. Speech/language treatment of the aphasias: An integrated clinical approach. Aspen Publishers, 1988.

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Stroke!: The ordeal & the rainbow. Patrice Press, 1986.

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Book chapters on the topic "Aphasia Cerebrovascular disease"

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Espert, Raúl, and Marien Gadea. "Neuropsychology of Moyamoya Disease." In Moyamoya Disease [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96558.

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Moyamoya disease (MMD) is an occlusive cerebrovascular disease characterized by progressive stenosis or occlusion in the terminal portion of the bilateral internal carotid arteries, and affect both children and adults. In this pathology, which presents itself through ischemia or cerebral hemorrhage, an unusual compensatory vascular network (moyamoya vessels) develops at the brain´s base in the form of collateral channels. MMD can present clinically as hemiparesis, dysarthria, aphasia, headache, seizures, visual deficits, syncope, or personality changes. Neuropsychologically, and even in the absence of obvious stroke, patients often present impaired attention, memory, behavior, and executive functions. This book chapter reviews the current literature regarding the neuropsychological deficits of MMD both in children and adults.
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