Relevant bibliographies by topics / Aplastic anemia

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Aplastic anemia'

Author: Grafiati
Published: 4 June 2021
Last updated: 25 July 2025

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Journal articles on the topic "Aplastic anemia"

1

Luchkin, A. V., E. A. Mikhailova, I. V. Galtseva, et al. "Telomere length of various blood and bone marrow cells in patients with aplastic anemia." Oncohematology 18, no. 3 (2023): 57–64. http://dx.doi.org/10.17650/1818-8346-2023-18-3-57-64.

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Background. Aplastic anemia proceeds with bone marrow failure and is associated with immunological suppression of normal blood stem cells’ proliferation, which lead to bone marrow aplasia. autoimmune aggression and internal defects of blood stem cell that cause abnormal hematopoiesis are being actively studied. An important role in the pathogenesis of the aplastic anemia is played by instability of telomere length (TL). determination of the initial TL makes it possible to clearly differentiate between the aplastic anemia and dyskeratosis congenita. also, it helps to identify the group of patie
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Ritz, Caitlin, Wenzhao Meng, Natasha L. Stanley, et al. "Postvaccination graft dysfunction/aplastic anemia relapse with massive clonal expansion of autologous CD8+ lymphocytes." Blood Advances 4, no. 7 (2020): 1378–82. http://dx.doi.org/10.1182/bloodadvances.2019000853.

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Key Points Acquired aplastic anemia is a T-cell–mediated autoimmune bone marrow aplasia, without a known etiologic trigger. Clonal expansion of CD8+ effector T lymphocytes can occur following vaccination and accompany graft dysfunction or aplastic anemia relapse.
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Drelichman, Guillermo I., Nora F. Basack, Alejandra Maro, et al. "Inherited Bone Marrow Failure Syndromes in Children: 32 Years Experience in a Single Institution." Blood 106, no. 11 (2005): 3751. http://dx.doi.org/10.1182/blood.v106.11.3751.3751.

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Abstract Inherited bone marrow failure syndromes (IBMFS) present chronic bone marrow failures, a familial incidence and high risk of malignancy. Objective: we describe our experience with patients with IBMFS. Since 02/73 to 02/05, 60 patients (pts) with IBMSF were diagnosed. Fanconi’s Anemia Blackfand-Diamond TAR-Shwachman-Diamond-Dyskeratosis Congenita Amegakaryocytic Thrombocytopenia.- Kostmann’s S. Familial AA-Pearson’s S N Pts 26 17 6 – 2 – 2 2 – 2 2 – 1 Median age at diagnosis 7.1 y 3 m 3m – 4 m – 6.4 y 2 m – 1.8 m 4 y – 3 y First hematologic sign Pancytopenia Anemia Thrombocytop. – Neutr
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Lefaan, Yannie Febby Martina, Wahyu Hidayat, and Nanan Nur'aeny. "Tatalaksana infeksi herpes simpleks virus tipe-1 dan necrotizing ulcerative stomatitis pada anak dengan anemia aplastik<p></p>Management of type-1 herpes simplex virus infection and necrotising ulcerative stomatitis in children with aplastic anaemia<p>." Jurnal Kedokteran Gigi Universitas Padjadjaran 33, no. 3 (2022): 1. http://dx.doi.org/10.24198/jkg.v33i3.30642.

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ABSTRAKPendahuluan: Necrotizing ulcerative stomatitis (NUS) merupakan penyakit inflamasi pada rongga mulut, ditandai oleh ulser nekrosis akut, berkembang cepat serta menyebabkan kerusakan masif pada jaringan. NUS dapat berkembang dari kondisi necrotizing ulcerative gingivitis (NUG) dan necrotizing ulcerative periodontitis (NUP). NUS dapat disertai infeksi lain, salah satunya infeksi herpes simplex tipe 1 (HSV-1). Infeksi HSV-1 dan NUS pada anak dengan anemia aplastik jarang ditemukan. Anemia aplastik merupakan kelainan hematologi, ditandai dengan hipoplastik/aplasia sumsum tulang dan pansitope
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Čermák, Jaroslav. "Aplastic anemia." Vnitřní lékařství 64, no. 5 (2018): 501–7. http://dx.doi.org/10.36290/vnl.2018.070.

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Kojima, Seiji, Norbert Frickhofen, H. Joachim Deeg, et al. "Aplastic Anemia." International Journal of Hematology 82, no. 5 (2005): 408–11. http://dx.doi.org/10.1532/ijh97.05138.

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Kook, Hoon. "Aplastic Anemia." Journal of the Korean Medical Association 49, no. 10 (2006): 885. http://dx.doi.org/10.5124/jkma.2006.49.10.885.

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Marchand, Tony, and Michaël Loschi. "Aplastic anemia." Hématologie 20, no. 6 (2014): 329–41. http://dx.doi.org/10.1684/hma.2014.0977.

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Young, Neal S. "Aplastic Anemia." New England Journal of Medicine 379, no. 17 (2018): 1643–56. http://dx.doi.org/10.1056/nejmra1413485.

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Young, Neal S., Phillip Scheinberg, and Rodrigo T. Calado. "Aplastic anemia." Current Opinion in Internal Medicine 7, no. 4 (2008): 338–44. http://dx.doi.org/10.1097/mci.0b013e32830c6d12.

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Dissertations / Theses on the topic "Aplastic anemia"

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Kojima, Seiji. "Use of granulocyte colony-stimulating factor for treatment of aplastic anemia." Nagoya University School of Medicine, 1999. http://hdl.handle.net/2237/5344.

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Hamzic, Edita. "Characterisation of mesenchymal stroma and discovery of biomarkers in aplastic anemia." Thesis, Kingston University, 2012. http://eprints.kingston.ac.uk/26276/.

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Haemopoietic activity in Aplastic anemia (AA) is significantly reduced and is generally attributed to failure of Haemopoietic stem cells (HSC) within the bone marrow. The extent to which Mesenchymal stromal cells (MSC) are involved in the functional restriction of HSC is largely unknown. To understand this, the physical and functional properties of AA MSC were studied in vitro. MSC were characterised by their phenotype and ability to form adherent stromal layers. The functional properties of AA MSC were assessed through proliferative, clonogenic and cross¬over culture assays. Results show seve
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Teo, Steve Keng Ong. "METABOLISM OF AMINO-CHLORAMPHENICOL: POSSIBLE ROLE IN CHLORAMPHENICOL-INDUCED APLASTIC ANEMIA AND LEUKEMIA." Thesis, The University of Arizona, 1985. http://hdl.handle.net/10150/291285.

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Wellman, Maxey Lee. "Studies on the pathogenesis of feline leukemia virus-induced erythroid aplasia /." The Ohio State University, 1986. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487268021746533.

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Arruda, Daisy Maria Meireles. "Aloimmunity against HLA class I antigens in patients with myelodysplastic syndrome and aplastic anemia." Universidade Federal do CearÃ, 2005. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=38.

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CoordenaÃÃo de AperfeiÃoamento de Pessoal de NÃvel Superior<br>Myelodysplastic syndrome (MDS) and aplastic anemia (AA) are two of the hematological disorders which present peripheral cytopenias, with extensive clinical manifestations that vary from slight anemia to severe pancytopenia; the latter requiring continuous transfusional reposition of red cell (RC) and platelet concentrates (PC), which can induce aloimunization in patients. Such patients can develop a post-transfusional refractory state, rendering further transfusions unviable. The objective of the present study was to investigate th
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Novitzky, Nicolas. "Interactions between the haematopoietic stem cell and the myeloid microenvironment in aplastic anaemia." Doctoral thesis, University of Cape Town, 1993. http://hdl.handle.net/11427/24943.

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In patients with aplastic anaemia that respond to immunosuppressive therapy, quantitative, morphological and functional haematologic derangement have been reported. To explain these findings, abnormalities in the marrow stroma or the stem cell have been postulated. To define the relative contribution of each of the latter, the integrity of the bone marrow from sixteen patients that responded to anti-lymphocyte globulin and high dose methyl prednisolone was compared to normal individuals. Bone marrow mononuclear cells were divided into two fractions. From the first, stroma was cultured in aMEM
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Tellechea, Maria Florencia. "Geração de células-tronco pluripotentes induzidas (iPSCs) a partir de células de pacientes com anemia aplástica adquirida." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/17/17135/tde-05122016-120214/.

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A anemia aplástica (AA) é uma doença hematológica rara caracterizada pela hipocelularidade da medula óssea, o que provoca pancitopenia. Esta pode ser de origem genética (associada a encurtamento telomérico) ou adquirida (não-associada a desgaste excessivo dos telômeros). Na forma adquirida, a ativação anormal de linfócitos T provoca a destruição das células hematopoéticas. O mecanismo que leva a essa destruição ainda não foi elucidado. Um dos tratamentos mais eficazes para repovoar a medula óssea hipocelular é o transplante com célulastronco hematopoéticas (CTHs). Porém, uma grande porcentagem
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MICHELOZZI, ILARIA MARINA. "The role of the bone marrow microenvironment in aplastic anaemia and acute myeloid leukaemia: from pathogenesis to chemoresistance." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2019. http://hdl.handle.net/10281/241079.

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La nicchia staminale ematopoietica è formata da molteplici tipi cellulari che contribuiscono alla regolazione dell’ematopoiesi all’interno del midollo osseo. In alcuni disordini ematopoietici il microambiente midollare risulta alterato, sebbene il suo contributo alla patogenesi non sia ancora chiaro, fornisce supporto alle cellule staminali leucemiche (LSC), a spese delle normali cellule staminali ematopoietiche, e protezione contro i chemioterapici. Dunque, per eradicare le LSC, le strategie terapeutiche devono considerare l’effetto protettivo contro i chemioterapici esercitato dal microambie
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Padilha, Pedro Henrique. "Variantes do gene THPO em pacientes com anemia aplástica adquirida." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/17/17154/tde-25042018-150914/.

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Introdução: A anemia aplástica (AA) adquirida é uma doença grave, caracterizada por pancitopenia e medula óssea hipocelular sem que haja associação com aumento de reticulina ou infiltração anormal na medula. Embora o mecanismo fisiopatológico não esteja totalmente elucidado, atribui-se a uma resposta imunomediada dos linfócitos T no ambiente medular. A trombopoetina (codificada pelo gene THPO) é um hormônio glicoproteico produzido pelo fígado e responsável pelo estímulo de crescimento de megacariócitos, desenvolvimento plaquetário e de demais linhagens e, quando disfuncional, contribui para o
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Rodrigues, Fernanda Gutierrez. "Identificação de moduladores genéticos em pacientes com anemia aplástica por sequenciamento de nova geração." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/17/17154/tde-25072018-110641/.

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A fisiopatologia das síndromes de falência da medula óssea (FMO) está relacionada a mecanismos adquiridos de destruição das células-tronco hematopoeiticas na medula ou a defeitos constitucionais em genes fundamentais para o reparo do DNA e manutenção dos telômeros. A anemia aplástica (AA), o protótipo das doenças de FMO, pode ter etiologia adquirida ou constitucional. A avaliação genética de pacientes com AA adquirida tem como objetivo a detecção de mutações somáticas que possam ser usadas como marcadores de resposta ao tratamento imunossupressor. Diferentemente, em pacientes com AA constituci
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Books on the topic "Aplastic anemia"

1

P, Alter Blanche, ed. Aplastic anemia, acquired and inherited. Saunders, 1994.

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2

A, Bacigalupo, and Schrezenmeier H, eds. Aplastic anemia: Pathophysiology and treatment. Cambridge University Press, 1999.

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3

1950-, Coulombel Laure, and Gluckman Eliane, eds. Ontogeny of hematopoiesis: Aplastic anemia = Ontogénie de l'hématopoïèse : aplasie médullaire. Editions J. Libbey Eurotext, 1995.

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Abdulkadyrov, K. M. Aplasticheskai͡a anemii͡a. Nauka, 1995.

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Food, Ontario Ministry of Agriculture and. Inclusion Body Hepatitis in Chickens (Aplastic Anemia). s.n, 1987.

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Shahidi, Nasrollah T., ed. Aplastic Anemia and Other Bone Marrow Failure Syndromes. Springer New York, 1990. http://dx.doi.org/10.1007/978-1-4612-3254-4.

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W, Kaufman David, ed. The Drug etiology of agranulocytosis and aplastic anemia. Oxford University Press, 1991.

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T, Shahidi Nasrollah, ed. Aplastic anemia and other bone marrow failure syndromes. Springer-Verlag, 1990.

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9

N, Frickhofen, Raghavachar A, and Schrezenmeier H, eds. Aplastic anemia: Current perspectives on the pathogenesis and treatment. Blackwell-MZV, 1993.

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Ibrāhīm, Saʻdīyah. Chlorination of chloramphenicol amine by myeloperoxidase: A possible mechanism for chloramphenicol-induced oplastic anemia. [s.n., 1990.

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Book chapters on the topic "Aplastic anemia"

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Menter, Thomas, and Alexandar Tzankov. "Aplastic Anemia." In Encyclopedia of Pathology. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-95309-0_3878.

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Menter, Thomas, and Alexandar Tzankov. "Aplastic Anemia." In Encyclopedia of Pathology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-28845-1_3878-1.

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Schwabkey, Zaker, Yehuda Deutsch, and David Sallman. "Aplastic Anemia." In Non-Neoplastic Hematologic Disorders. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-62373-8_21.

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Gupta, Arun. "Aplastic Anemia." In Decision Making Through Problem Based Learning in Hematology. Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-99-8933-1_16.

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Dror, Yigal, and Michaela Cada. "Acquired Aplastic Anemia." In Pediatric Oncology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-61421-2_2.

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Lior, Baraf, and Levy Yair. "Idiopathic Aplastic Anemia." In Diagnostic Criteria in Autoimmune Diseases. Humana Press, 2008. http://dx.doi.org/10.1007/978-1-60327-285-8_95.

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Visconte, Valeria, and Ramon V. Tiu. "Acquired Aplastic Anemia." In Cancer Consult: Expertise for Clinical Practice. John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118589199.ch19.

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Alter, Blanche P. "Constitutional Aplastic Anemia." In Aplastic Anemia and Other Bone Marrow Failure Syndromes. Springer New York, 1990. http://dx.doi.org/10.1007/978-1-4612-3254-4_3.

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Yagasaki, Hiroshi. "Childhood Aplastic Anemia." In Hematological Disorders in Children. Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-3886-0_6.

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Lior, Baraf, Levy Yair, Yehuda Shoenfeld, Gabriela Roque González, and Abihai Lucas Hernández. "Idiopathic Aplastic Anemia." In Autoimmune Disease Diagnosis. Springer Nature Switzerland, 2024. https://doi.org/10.1007/978-3-031-69895-8_105.

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Conference papers on the topic "Aplastic anemia"

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Sahu, N., D. Rosenblum, T. Fech, and M. Turki. "HSV-induced Aplastic Anemia." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a3592.

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Xiang-yuan, Zeng, and Zhang Ying. "EXPERIMENTAL RESEARCHES OF EFFECTS OF POLYACTIN A ON HEMOPOIETIC STEM CELL OF MARROW." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643210.

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Polyactin A is a new immunological enhancement agent which was developped in China for the first time1 It is a polysaccharide extracted from cultured a-hemolytic streptococcus No.33 the mouth.Clinical observation suggests that the drug has marked inhibiting effects on some tumors and can increase the number of leucocytes, enhance immunity of the organism.Polyactin. A has especially good effect on aplastic anemia and it is a new drug in treatment of aplastic anemia.However, it mechanism isn’t understand.Effects of Polyactin A on hemopoietic stem cells of marrow are studied. The hemopoietic stem
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Bakoš, Matija, Vanja Ille, Matea Kovačić, et al. "233 Transitory aplastic anemia following acute autoimmune hepatitis in a young boy." In 10th Europaediatrics Congress, Zagreb, Croatia, 7–9 October 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-europaediatrics.233.

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Galiatsatos, Panagis, and Kenneth Remy. "Granuloycte Therapy As Treatment For Candida Parapsilosis Sepsis In An Aplastic Anemia Patient." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4600.

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Babushok, Daria V., Nieves Perdigones, Juan C. Perin, et al. "Abstract 2977: Most patients with acquired aplastic anemia develop clonal hematopoiesis early in disease." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-2977.

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Tyshkevich, V. S., and I. V. Puhteeva. "ANALYSIS OF THE INCIDENCE OF ACQUIRED APLASTIC ANEMIA AMONG CHILDREN IN THE REPUBLIC OF BELARUS." In SAKHAROV READINGS 2021: ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. International Sakharov Environmental Institute, 2021. http://dx.doi.org/10.46646/sakh-2021-1-341-345.

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The treatment methods of acquired aplastic anemia are considered in the work; some difficulties arising in the process of stem cell transplantation and therapy are described. Also, an analysis of statistical data was carried out, on the basis of which graphs and charts were designed. In the process of data analysis, it was found that boys get sick 1.8 times more often than girls. Two peaks were found when the disease arose more often: at 11 and 16 years old. A rough intense indicator of the incidence of AAA among children 0-17 years old from 2000 till 2018 was 0.26 cases per 100 thousand child
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Stevens, G., D. Egbe, and R. G. Patel. "A Case Report of Aplastic Anemia in a Patient with SLE and COVID-19 Infection." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a2627.

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T. Abdulrazzaq, Ghanem, and Huda F. Hasan. "Evaluation the Pharmacodynamics Interaction of Cyclosporine and Azacitidine On the Treatment of Induced Aplastic Anemia in Rat." In IX. International Scientific Congress of Pure, Applied and Technological Sciences. Rimar Academy, 2023. http://dx.doi.org/10.47832/minarcongress9-24.

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The study was done to assess the effective dose and type of relative combination between cyclosporin and azacitidine on the treatment of induced aplastic anemia in rats. Eighty-five rats were divided into five groups: The 1st group: Five female rats were non induced and treated with distilled. The 2nd group: Five female rats were induced and treated with distilled water. The 3rd and 4th groups included twenty-five rats for each one were induced and treated with cyclosporine and azacitidine respectively and each group divided into five equal subgroups with doses of 2, 3, 4, 5, and 6 mg/kg/. The
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Pal, Subrata, and R. Sheba N. David. "Effect of Cryoprocessing on Umbilical Cord Blood for Hematological Disorders." In ASME 2001 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2001. http://dx.doi.org/10.1115/imece2001/bed-23152.

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Abstract Cellular therapies promise to become major therapeutic modalities of this new century. The hematopoietic restoration with umbilical cord blood transplant can be a useful source of hematopoietic stem cells for routine bone marrow reconstitution. Thus it can be used as an effective source of stem cells for the treatment of various hematological disorders like leukemia, thalessaemia, aplastic anemia and it also can be used to replace the damaged tissue in various pathological conditions like Parkinson’s disease, Alzheimer’s, osteoarthritis etc. The advantage of the umbilical cord blood t
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Gupta, S., M. Parmar, M. Nadeem, and F. Chaudary. "Severe Pneumonia and Bacteremia by Rhodococcus Species in a Patient with Refractory Aplastic Anemia on Alemtuzumab Therapy." In American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a2892.

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Reports on the topic "Aplastic anemia"

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Wang, Yan, Zhenzhen Wang, Jie Xu, et al. Efficacy and safety of haploidentical hematopoietic stem cell transplantation on severe aplastic anemia using busulfan-based myeloablative regimenA protocol for a Bayesian network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.1.0116.

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