Academic literature on the topic 'Apoptosis. Schwein'

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Journal articles on the topic "Apoptosis. Schwein"

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Glatzle, Megan. "Einfluss eines Selenmangels auf Milzzustand und Immunabwehr beim Schwein." Tierärztliche Praxis Ausgabe G: Großtiere / Nutztiere 49, no. 04 (2021): 288. http://dx.doi.org/10.1055/a-1543-4432.

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Li S, Sun W, Zhang K et al. Selenium deficiency induces spleen pathological changes in pigs by decreasing selenoprotein expression, evoking oxidative stress, and activating inflammation and apoptosis. J Anim Sci Biotechnol 2021; 12 (1): 65 Selen (Se) ist ein essenzielles Spurenelement mit wichtigen Aufgaben im antioxidativen System, Schilddrüsenhormonstoffwechsel und Immunsystem. Diätetisch bedingter Selenmangel kann Auswirkungen auf die Funktion der Milz, einem wichtigen Immunorgan, haben. Da Untersuchungen hierzu limitiert sind, war es Ziel dieser Studie, in einem Schweinemodell die Pathogen
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Oliveira, Rosane B., Maria T. Ochoa, Peter A. Sieling, et al. "Expression of Toll-Like Receptor 2 on Human Schwann Cells: a Mechanism of Nerve Damage in Leprosy." Infection and Immunity 71, no. 3 (2003): 1427–33. http://dx.doi.org/10.1128/iai.71.3.1427-1433.2003.

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ABSTRACT Nerve damage is a clinical hallmark of leprosy and a major source of patient morbidity. We investigated the possibility that human Schwann cells are susceptible to cell death through the activation of Toll-like receptor 2 (TLR2), a pattern recognition receptor of the innate immune system. TLR2 was detected on the surface of human Schwann cell line ST88-14 and on cultured primary human Schwann cells. Activation of the human Schwann cell line and primary human Schwann cell cultures with a TLR2 agonist, a synthetic lipopeptide comprising the N-terminal portion of the putative Mycobacteri
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Xu, Shiqing, Weijie Bao, Xiuli Men та ін. "Interleukin-10 Protects Schwann Cells against Advanced Glycation End Products-Induced Apoptosis via NF-κB Suppression". Experimental and Clinical Endocrinology & Diabetes 128, № 02 (2019): 89–96. http://dx.doi.org/10.1055/a-0826-4374.

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AbstractDemyelination resulting from Schwann cell injury is a main pathological feature of diabetic neuropathy, and a key contributor to this process may be inflammation due to advanced glycation end products (AGEs). Therefore, protection by anti-inflammation agents is anticipated. In this study, we showed that interleukin-10 (IL-10), an anti-inflammatory cytokine, inhibits apoptosis of Schwann cells induced by AGEs in vitro. We isolated and cultured Schwann cells from rat sciatic nerves. As detected by flow cytometry, apoptosis of Schwann cells markedly increased following incubation with AGE
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Tiong, Yee Lian, Khuen Yen Ng, Rhun Yian Koh, Gnanajothy Ponnudurai та Soi Moi Chye. "Melatonin Prevents Oxidative Stress-Induced Mitochondrial Dysfunction and Apoptosis in High Glucose-Treated Schwann Cells via Upregulation of Bcl2, NF-κB, mTOR, Wnt Signalling Pathways". Antioxidants 8, № 7 (2019): 198. http://dx.doi.org/10.3390/antiox8070198.

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Neuropathy is a complication that affects more than 50% of long-standing diabetic patients. One of the causes of diabetes neuropathy (DN) is the apoptosis of Schwann cells due to prolonged exposure to high glucose and build-up of oxidative stress. Melatonin is a hormone that has a known antioxidant property. In this study, we investigated the protective effect of melatonin on high glucose-induced Schwann cells’ apoptosis. Our results revealed that high glucose promoted apoptosis via mitochondrial-related oxidative stress and downregulated Bcl-2 family proteins in Schwann cells. In this signall
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Zorick, T. S., D. E. Syroid, A. Brown, T. Gridley, and G. Lemke. "Krox-20 controls SCIP expression, cell cycle exit and susceptibility to apoptosis in developing myelinating Schwann cells." Development 126, no. 7 (1999): 1397–406. http://dx.doi.org/10.1242/dev.126.7.1397.

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The transcription factors Krox-20 and SCIP each play important roles in the differentiation of Schwann cells. However, the genes encoding these two proteins exhibit distinct time courses of expression and yield distinct cellular phenotypes upon mutation. SCIP is expressed prior to the initial appearance of Krox-20, and is transient in both the myelinating and non-myelinating Schwann cell lineages; while in contrast, Krox-20 appears approximately 24 hours after SCIP and then only within the myelinating lineage, where its expression is stably maintained into adulthood. Similarly, differentiation
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CONTI, G., E. SCARPINI, A. ROSTAMI, et al. "Schwann Cell Apoptosis during Cell-Mediated Demyelination." Annals of the New York Academy of Sciences 883, no. 1 (1999): 518–19. http://dx.doi.org/10.1111/j.1749-6632.1999.tb08625.x.

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Sherman, Larry S., Tilat A. Rizvi, Saikumar Karyala, and Nancy Ratner. "Cd44 Enhances Neuregulin Signaling by Schwann Cells." Journal of Cell Biology 150, no. 5 (2000): 1071–84. http://dx.doi.org/10.1083/jcb.150.5.1071.

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We describe a key role for the CD44 transmembrane glycoprotein in Schwann cell–neuron interactions. CD44 proteins have been implicated in cell adhesion and in the presentation of growth factors to high affinity receptors. We observed high CD44 expression in early rat neonatal nerves at times when Schwann cells proliferate but low expression in adult nerves, where CD44 was found in some nonmyelinating Schwann cells and to varying extents in some myelinating fibers. CD44 constitutively associated with erbB2 and erbB3, receptor tyrosine kinases that heterodimerize and signal in Schwann cells in r
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Liu, Y., Y. Huang, S. Sun, W. Deng, and T. W. LI. "POS1135 MONOSODIUM URATE CRYSTALS REDUCE SCHWANN CELLS VIABILITY." Annals of the Rheumatic Diseases 80, Suppl 1 (2021): 847.1–847. http://dx.doi.org/10.1136/annrheumdis-2021-eular.1989.

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Background:The prevalence of peripheral neuropathy in patients with gout almostly reaches 25%[1]. Demyelination caused by Schwann cell (SCs) injury and apoptosis is the major pathological feature of peripheral[2]. None of study has focused on the effects of monosodium urate (MSU) crystals on SCs.Objectives:To assess the effect of MSU crystals on SCs.Methods:Mouse-derived Schwann cells (RSC96) are stimulated with different concentrations of MSU crystals (0mg/ml,0.25mg/ml,0.5mg/ml) and time (24h,48h,72h). The migration ability of Schwann cells is evaluated by acratch assay, the proliferation lev
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Wu, Sheng-Hua, Shu-Hung Huang, Kuang-I. Cheng, et al. "Third-Degree Hindpaw Burn Injury Induced Apoptosis of Lumbar Spinal Cord Ventral Horn Motor Neurons and Sciatic Nerve and Muscle Atrophy in Rats." BioMed Research International 2015 (2015): 1–9. http://dx.doi.org/10.1155/2015/372819.

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Background. Severe burns result in hypercatabolic state and concomitant muscle atrophy that persists for several months, thereby limiting patient recovery. However, the effects of burns on the corresponding spinal dermatome remain unknown. This study aimed to investigate whether burns induce apoptosis of spinal cord ventral horn motor neurons (VHMNs) and consequently cause skeletal muscle wasting.Methods. Third-degree hindpaw burn injury with 1% total body surface area (TBSA) rats were euthanized 4 and 8 weeks after burn injury. The apoptosis profiles in the ventral horns of the lumbar spinal
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Jirsova, Katerina, Vaclav Mandys, Willem Hendrik Gispen, and Peter Rudolf Bär. "Cisplatin-induced apoptosis in cultures of human Schwann cells." Neuroscience Letters 392, no. 1-2 (2006): 22–26. http://dx.doi.org/10.1016/j.neulet.2005.08.068.

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Dissertations / Theses on the topic "Apoptosis. Schwein"

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Mentschel, Joachim. "Erhöhung der Butyratbildung durch Fütterung von Resistenter Stärke beim Schwein Konsequenzen für die Mitose und Apoptoseregulation der Colonmucosa /." [S.l. : s.n.], 2004. http://www.bsz-bw.de/cgi-bin/xvms.cgi?SWB11406734.

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Reiter, Katja. "Einfluss des Probiotikums Enterococcus faecium SF 68 (NCIMB 10415) auf die Morphologie der Darmschleimhaut des Schweines." [S.l.] : [s.n.], 2005. http://www.diss.fu-berlin.de/2006/215/index.html.

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Gösele, Michael. "Effekte einer frühen Beatmung mit reinem Sauerstoff auf histomorphologische Parameter von Lunge und Leber im Langzeitmodell des septischen Schocks beim Schwein." [S.l. : s.n.], 2008. http://nbn-resolving.de/urn:nbn:de:bsz:289-vts-65831.

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Rideout, Hardy Joseph. "Schwann cell apoptosis in an in vitro model of diabetic neuropathy, mitochondrial dysfunction and the permeability transition pore." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0016/NQ49286.pdf.

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Klement, Johannes Maximilian [Verfasser], and Helmut [Akademischer Betreuer] Schweikl. "Die Bedeutung der MAP-Kinasen in der monomer-induzierten Apoptose / Johannes Maximilian Klement. Betreuer: Helmut Schweikl." Regensburg : Universitätsbibliothek Regensburg, 2016. http://d-nb.info/1085550079/34.

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Krause, Sylvia [Verfasser], Gernot [Akademischer Betreuer] Marx, Felicitas [Akademischer Betreuer] Eckoldt, and Kerstin [Akademischer Betreuer] Amann. "Einfluss unterschiedlicher Volumenersatzlösungen auf Apoptose und Inflammation der Niere : eine immunhistologische und molekularbiologische Untersuchung am septischen Schwein / Sylvia Krause. Gutachter: Gernot Marx ; Felicitas Eckoldt ; Kerstin Amann." Jena : Thüringer Universitäts- und Landesbibliothek Jena, 2011. http://d-nb.info/1016481268/34.

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Oliveira, Rosane Barbosa de. "Expressão e funçãode receptores Toll-Like em linhagem de células Schwann: uma contribuição a patogênese de lesão neural na hanseníase." reponame:Repositório Institucional da FIOCRUZ, 2003. https://www.arca.fiocruz.br/handle/icict/4139.

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Submitted by Tatiana Oliveira (tsilva@icict.fiocruz.br) on 2012-06-06T12:07:05Z No. of bitstreams: 1 rosane_b_oliveira_ioc_bp_0007_2003.pdf: 28903576 bytes, checksum: 4167a22455a36ea7147bd41b4dc7b566 (MD5)<br>Made available in DSpace on 2012-06-06T12:07:05Z (GMT). No. of bitstreams: 1 rosane_b_oliveira_ioc_bp_0007_2003.pdf: 28903576 bytes, checksum: 4167a22455a36ea7147bd41b4dc7b566 (MD5) Previous issue date: 2003<br>Fundação Oswaldo Cruz.Instituto Oswaldo Cruz. Rio de janeiro, RJ, Brasil<br>A principal complicação na hanseníase é o desenvolvimento de deformidades ao longo do curso crônico da
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Santos, Stéfanie Vanessa. "Classificação morfológica, imunoistoquímica e prognóstica dos hemangiopericitomas caninos." Universidade de São Paulo, 2005. http://www.teses.usp.br/teses/disponiveis/10/10133/tde-26092006-155648/.

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Hemangiopericitomas (CHP) assim como schwanomas são neoplasias cutâneas de origem mesenquimal, sendo os hemangiopericitomas freqüentemente relatados em cães, ao contrário dos neurofibromas que são mais raros nos mesmos. Relata-se que o CHP origina-se de pericitos, ou células que se localizam ao redor de vasos sanguíneos. São observadas mais freqüentemente nos membros, como massas, bem circunscritas, firmes e grandes. Os hemangiopericitomas têm características histológicas comuns aos schwanomas (neurofibromas), sugerindo uma possível semelhante histogênese. Na realidade, na experiência deste Se
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Beuermann, Christian. "Molekulargenetische und physiologische Untersuchungen zur Vererbung des Erbdefektes Hernia inguinalis/scrotalis beim Schwein." Doctoral thesis, 2009. http://hdl.handle.net/11858/00-1735-0000-0006-B03F-D.

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Zwiehoff, Julia Marilena. "Einfluss der extrakorporalen Zirkulation und systemischen Hypothermie auf die Lebermorphologie neugeborener Schweine." Doctoral thesis, 2013. http://hdl.handle.net/11858/00-1735-0000-0019-89D6-2.

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Abdominelle Komplikationen, zu denen auch das akute Leberversagen nach Einsatz der Herz-Lungen-Maschine und systemischer Hypothermie nach herzchirurgischen Korrekturoperationen neugeborener Patienten zählt, sind seltene, aber dennoch erst zu nehmende unerwünschte Folgen. Die Untersuchungen erfolgten an neugeborenen Schweinen, die in 4 Gruppen eingeteilt wurden: Eine Gruppe wurde mit extrakorporaler Zirkulation in moderater Hypothermie (32°C) operiert (CPB), die zweite Gruppe wurde in tiefer Hypothermie (18°C) und totalem Kreislaufstillstand operiert (DHCA). In der dritten Gruppe (Sham) erfolgt
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Book chapters on the topic "Apoptosis. Schwein"

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Li, Kaixin, Inam-u-llah, Xiaoxia Shi, et al. "Anti-apoptotic Effect of Taurine on Schwann Cells Exposed to High Glucose In Vitro." In Advances in Experimental Medicine and Biology. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-8023-5_68.

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