Journal articles on the topic 'APP-CTFs'
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Vaillant-Beuchot, Loan, Arnaud Mary, Raphaëlle Pardossi-Piquard, Alexandre Bourgeois, Inger Lauritzen, Fanny Eysert, Paula Fernanda Kinoshita, et al. "Accumulation of amyloid precursor protein C-terminal fragments triggers mitochondrial structure, function, and mitophagy defects in Alzheimer’s disease models and human brains." Acta Neuropathologica 141, no. 1 (October 20, 2020): 39–65. http://dx.doi.org/10.1007/s00401-020-02234-7.
Marttinen, Mikael, Catarina B. Ferreira, Kaisa M. A. Paldanius, Mari Takalo, Teemu Natunen, Petra Mäkinen, Luukas Leppänen, et al. "Presynaptic Vesicle Protein SEPTIN5 Regulates the Degradation of APP C-Terminal Fragments and the Levels of Aβ." Cells 9, no. 11 (November 15, 2020): 2482. http://dx.doi.org/10.3390/cells9112482.
Hao, Candy Yan, Michael S. Perkinton, William Wai-Lun Chan, Ho Yin Edwin Chan, Christopher C. J. Miller, and Kwok-Fai Lau. "GULP1 is a novel APP-interacting protein that alters APP processing." Biochemical Journal 436, no. 3 (May 27, 2011): 631–39. http://dx.doi.org/10.1042/bj20110145.
Sivanesan, Senthilkumar, Ravi Mundugaru, and Jayakumar Rajadas. "Possible Clues for Brain Energy Translation via Endolysosomal Trafficking of APP-CTFs in Alzheimer’s Disease." Oxidative Medicine and Cellular Longevity 2018 (October 21, 2018): 1–11. http://dx.doi.org/10.1155/2018/2764831.
Lauritzen, I., R. Pardossi-Piquard, A. Bourgeois, A. Bécot, and F. Checler. "Does Intraneuronal Accumulation of Carboxyl-terminal Fragments of the Amyloid Precursor Protein Trigger Early Neurotoxicity in Alzheimer’s Disease?" Current Alzheimer Research 16, no. 5 (May 21, 2019): 453–57. http://dx.doi.org/10.2174/1567205016666190325092841.
Wang, Bo-Jeng, Guor Mour Her, Ming-Kuan Hu, Yun-Wen Chen, Ying-Tsen Tung, Pei-Yi Wu, Wen-Ming Hsu, et al. "ErbB2 regulates autophagic flux to modulate the proteostasis of APP-CTFs in Alzheimer’s disease." Proceedings of the National Academy of Sciences 114, no. 15 (March 28, 2017): E3129—E3138. http://dx.doi.org/10.1073/pnas.1618804114.
Shinohara, Mitsuru, Naoyuki Sato, Hitomi Kurinami, Daisuke Takeuchi, Shuko Takeda, Motoko Noma, Hiromi Rakugi, and Ryuichi Morishita. "Fluvastatin reduces Aβ levels in brain by upregulating APP-CTFs degradation and Aβ clearance." Neuroscience Research 65 (January 2009): S64. http://dx.doi.org/10.1016/j.neures.2009.09.194.
Xie, Zhongcong, Yuanlin Dong, Uta Maeda, Weiming Xia, and Rudolph E. Tanzi. "RNA Interference Silencing of the Adaptor Molecules ShcC and Fe65 Differentially Affect Amyloid Precursor Protein Processing and Aβ Generation." Journal of Biological Chemistry 282, no. 7 (December 14, 2006): 4318–25. http://dx.doi.org/10.1074/jbc.m609293200.
Yang, Chuanbin, Cui-Zan Cai, Ju-Xian Song, Jie-Qiong Tan, Siva Sundara Kumar Durairajan, Ashok Iyaswamy, Ming-Yue Wu, et al. "NRBF2 is involved in the autophagic degradation process of APP-CTFs in Alzheimer disease models." Autophagy 13, no. 12 (December 2, 2017): 2028–40. http://dx.doi.org/10.1080/15548627.2017.1379633.
Matsushima, Takahide, Tadashi Nakaya, and Toshiharu Suzuki. "P1-155: Exclusion of phosporylated APP CTFs from membrane region rich in active γ-secretase." Alzheimer's & Dementia 5, no. 4S_Part_7 (July 2009): P223. http://dx.doi.org/10.1016/j.jalz.2009.04.161.
K. Lakshmana, Madepalli, Subhojit Roy, Kaihong Mi, and David E. Kang. "Amyloidogenic Processing of APP in Lipid Rafts." Open Biology Journal 3, no. 1 (March 19, 2010): 21–31. http://dx.doi.org/10.2174/18741967010030100021.
Kwart, Dylan, Andrew Gregg, Claudia Scheckel, Elisabeth A. Murphy, Dominik Paquet, Michael Duffield, John Fak, Olav Olsen, Robert B. Darnell, and Marc Tessier-Lavigne. "A Large Panel of Isogenic APP and PSEN1 Mutant Human iPSC Neurons Reveals Shared Endosomal Abnormalities Mediated by APP β-CTFs, Not Aβ." Neuron 104, no. 2 (October 2019): 256–70. http://dx.doi.org/10.1016/j.neuron.2019.07.010.
Kwart, Dylan, Andrew Gregg, Claudia Scheckel, Elisabeth A. Murphy, Dominik Paquet, Michael Duffield, John Fak, Olav Olsen, Robert B. Darnell, and Marc Tessier-Lavigne. "A Large Panel of Isogenic APP and PSEN1 Mutant Human iPSC Neurons Reveals Shared Endosomal Abnormalities Mediated by APP β-CTFs, Not Aβ." Neuron 104, no. 5 (December 2019): 1022. http://dx.doi.org/10.1016/j.neuron.2019.11.010.
Repetto, Emanuela, Claudio Russo, Valentina Venezia, Mario Nizzari, and Gennaro Schettini. "P4-179 BACE1 enhances APP cleavage, CTFS formation and their interaction with SHCA and GRB2 adaptor proteins." Neurobiology of Aging 25 (July 2004): S526. http://dx.doi.org/10.1016/s0197-4580(04)81737-8.
Sergeant, Nicolas, Jean-Philippe David, Danie Champain, Antoine Ghestem, Annick Wattez, and André Delacourte. "Progressive decrease of amyloid precursor protein carboxy terminal fragments (APP-CTFs), associated with tau pathology stages, in Alzheimer's disease." Journal of Neurochemistry 81, no. 4 (May 7, 2002): 663–72. http://dx.doi.org/10.1046/j.1471-4159.2002.00901.x.
Li, Fengling, Guosheng Han, and Kexiang Wu. "Tanshinone IIA Alleviates the AD Phenotypes in APP and PS1 Transgenic Mice." BioMed Research International 2016 (2016): 1–8. http://dx.doi.org/10.1155/2016/7631801.
Shinohara, Mitsuru, Naoyuki Sato, Hitomi Kurinami, Daisuke Takeuchi, Shuko Takeda, Munehisa Shimamura, Toshihide Yamashita, Yasuo Uchiyama, Hiromi Rakugi, and Ryuichi Morishita. "Reduction of Brain β-Amyloid (Aβ) by Fluvastatin, a Hydroxymethylglutaryl-CoA Reductase Inhibitor, through Increase in Degradation of Amyloid Precursor Protein C-terminal Fragments (APP-CTFs) and Aβ Clearance." Journal of Biological Chemistry 285, no. 29 (May 14, 2010): 22091–102. http://dx.doi.org/10.1074/jbc.m110.102277.
Lauritzen, Inger, Anaïs Bécot, Alexandre Bourgeois, Raphaëlle Pardossi-Piquard, Maria-Grazia Biferi, Martine Barkats, and Fréderic Checler. "Targeting γ-secretase triggers the selective enrichment of oligomeric APP-CTFs in brain extracellular vesicles from Alzheimer cell and mouse models." Translational Neurodegeneration 8, no. 1 (December 2019). http://dx.doi.org/10.1186/s40035-019-0176-6.
Lee, Seung-Eun, Daekee Kwon, Nari Shin, Dasom Kong, Nam Gyo Kim, Hee-Yeong Kim, Min-Ji Kim, Soon Won Choi, and Kyung-Sun Kang. "Accumulation of APP-CTF induces mitophagy dysfunction in the iNSCs model of Alzheimer’s disease." Cell Death Discovery 8, no. 1 (January 10, 2022). http://dx.doi.org/10.1038/s41420-021-00796-3.
Shen, Ruinan, and Chengbiao Wu. "RIN3 Binds to BIN1 and CD2AP to increase APP‐CTFS in early endosomes." Alzheimer's & Dementia 16, S3 (December 2020). http://dx.doi.org/10.1002/alz.047161.
Wu, Qi, Leonardo Cortez, Razieh Kamali-Jamil, Valerie Sim, Holger Wille, and Satyabrata Kar. "Implication of exosomes derived from cholesterol-accumulated astrocytes in Alzheimer's disease pathology." Disease Models & Mechanisms, September 15, 2021. http://dx.doi.org/10.1242/dmm.048929.
Cai, Cui-Zan, Chuanbin Yang, Xu-Xu Zhuang, Ning-Ning Yuan, Ming-Yue Wu, Jie-Qiong Tan, Ju-Xian Song, et al. "NRBF2 is a RAB7 effector required for autophagosome maturation and mediates the association of APP-CTFs with active form of RAB7 for degradation." Autophagy, June 16, 2020, 1–19. http://dx.doi.org/10.1080/15548627.2020.1760623.