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1

Hammerschick, Tim, Tim Wagner, and Walter Vetter. "Countercurrent chromatographic fractionation followed by gas chromatography/mass spectrometry identification of alkylresorcinols in rye." Analytical and Bioanalytical Chemistry 412, no. 30 (2020): 8417–30. http://dx.doi.org/10.1007/s00216-020-02980-3.

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AbstractAlkylresorcinols (5-alkyl-1,3-dihydroxybenzenes, ARs) are bioactive phenolic lipid compounds which are particularly abundant in rye and partly other cereals. In this study on ARs, whole rye grain extracts were gained with cyclohexane/ethyl acetate (46/54, w/w). Silylated extracts were used to develop a gas chromatography with mass spectrometry method in the selected ion monitoring mode (GC/MS-SIM) for the sensitive detection of conventional ARs along with keto-substituted (oxo-AR) and ring-methylated ARs (mAR) with 5-alkyl chain lengths of 14 to 27 carbon atoms and 0 to 4 double bonds in one run. Analysis was performed by countercurrent chromatographic (CCC) fractionation using the solvent system n-hexane/ethyl acetate/methanol/water (9/1/9/1, v/v/v/v). Subsequent GC/MS-(SIM) analysis of 80 silylated CCC fractions enabled the detection of 74 ARs in the sample. The CCC elution of the ARs followed the equivalent chain length (ECL) rule in which one double bond compensated the effect of two (additional) carbon atoms. Novel or rarely reported ARs were detected in virtually all classes, i.e. saturated AR (AR14:0), even-numbered monounsaturated AR isomers (AR16:1-AR26:1), triunsaturated ARs (AR25:3), oxo-ARs (AR17:0 oxo, AR19:1 oxo, AR21:2 oxo, AR23:2 oxo) and odd-numbered methyl-ARs (mAR15:0-mAR23:0). Positions of the double bonds of monounsaturated ARs and oxo-ARs were determined with the help of dimethyl disulfide (DMDS) derivatives.
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2

Kidd, Bruce L., Richard M. Langford, and Theresa Wodehouse. "Arthritis and pain. Current approaches in the treatment of arthritic pain." Arthritis Research & Therapy 9, no. 3 (2007): 214. http://dx.doi.org/10.1186/ar2147.

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3

Fu, Yun-Chang, Hongshen Jiang, and Paul Bishop. "An Inhibition Study of the Effect of Azo Dyes on Bioactivity of Biofilms." Water Science and Technology 29, no. 7 (1994): 365–72. http://dx.doi.org/10.2166/wst.1994.0363.

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An inhibition study showed that toxic compounds caused two responses when present at low concentration. One is stimulation of the biomass by simply serving as an energy source; this caused an increase in the total respiration rate. The other is inhibition of the reaction. AR14 was more toxic than AO7 for biofilm from reactors fed with a primary substrate. However, AO7 demonstrated inhibition for biofilm from reactors fed with AR14 and primary substrate, and AR14 could serve as a carbon source for the same film.
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4

Sheppard, Ryan L., Espen E. Spangenburg, Eva R. Chin, and Stephen M. Roth. "Androgen receptor polyglutamine repeat length affects receptor activity and C2C12 cell development." Physiological Genomics 43, no. 20 (2011): 1135–43. http://dx.doi.org/10.1152/physiolgenomics.00049.2011.

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Testosterone (T) has an anabolic effect on skeletal muscle and is believed to exert its local effects via the androgen receptor (AR). The AR harbors a polymorphic stretch of glutamine repeats demonstrated to inversely affect receptor transcriptional activity in prostate and kidney cells. The effects of AR glutamine repeat length on skeletal muscle are unknown. In this study we examined the effect of AR CAG repeat length on AR function in C2C12 cells. AR expression vectors harboring 14, 24, and 33 CAG repeats were used to assess AR transcriptional activity. C2C12 cell proliferation, differentiation, gene expression, myotube formation, and myonuclear fusion index were assessed. Transcriptional activity increased with increasing repeat length and in response to testosterone (AR14 = 3.91 ± 0.26, AR24 = 25.21 ± 1.72, AR33 = 36.08 ± 3.22 relative light units; P < 0.001). Ligand activation was increased for AR33 (2.10 ± 0.04) compared with AR14 (1.54 ± 0.09) and AR24 (1.57 ± 0.05, P < 0.001). AR mRNA expression was elevated in each stably transfected line. AR33 cell proliferation (20,512.3 ± 1,024.0) was decreased vs. AR14 (27,604.17 ± 1,425.3; P < 0.001) after 72 h. Decreased CK activity in AR14 cells (54.9 ± 2.9 units/μg protein) in comparison to AR33 (70.8 ± 8.1) ( P < 0.05) was noted. The myonuclear fusion index was lower for AR14 (15.21 ± 3.24%) and AR33 (9.97 ± 3.14%) in comparison to WT (35.07 ± 5.60%, P < 0.001). AR14 and AR33 cells also displayed atypical myotube morphology. RT-PCR revealed genotype differences in myostatin and myogenin expression. We conclude that AR polyglutamine repeat length is directly associated with transcriptional activity and alters the growth and development of C2C12 cells. This polymorphism may contribute to the heritability of muscle mass in humans.
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5

Behnajady, Mohammad A., and Mahsa Hajiahmadi. "Intensification of Azo Dye Removal Rate in the Presence of Immobilized Nanoparticles and Inorganic Anions under UV-C Irradiation: Optimization by Response Surface Methodology." International Journal of Photoenergy 2013 (2013): 1–11. http://dx.doi.org/10.1155/2013/289290.

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Wastewaters contain inorganic anions that affect the removal rate of organic pollutants. The present study aims to optimize the effects of inorganic anions such as , Cl−, , and on the removal rate of an organic pollutant in the presence of immobilized TiO2nanoparticles using response surface methodology (RSM). C.I. Acid Red 17 (AR17) was used as a model organic pollutant. Thirty experiments were required to study the effects of anions in various concentrations. The results indicate that the addition of and ions intensifies the removal rate of AR17. The results of the analysis of variance (ANOVA) showed a high coefficient of determination value ( and ). The results indicate that RSM is a suitable method for modeling and optimizing the process. The results prove that in the presence of and and ions especially in the combination situation the removal rate of AR17 is enhanced considerably. An important synergy effect was observed in the combination of and ions, so that AR17 removal percent under the optimized RSM conditions was considerably more than the sum of removal percent when these ions are used individually.
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6

Shojaat, Rahim, Afzal Karimi, Naghi Saadatjoo, and Soheil Aber. "Dye removal from artificial wastewater using heterogeneous bio-fenton system." Chemical Industry and Chemical Engineering Quarterly 23, no. 4 (2017): 447–56. http://dx.doi.org/10.2298/ciceq160621058s.

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In the present study, GOx/MnFe2O4/calcium alginate nano-composite was prepared by the trapping enzyme/nanoparticles in calcium alginate. The prepared absorbent was applied for decolorization of artificial dye wastewater of acid red 14 (AR14) by heterogeneous bio-Fenton system. Kinetic and isotherm studies were carried out. The decolorization of acid red 14 followed the Michaelis- Menten, pseudo-first order and pseudo-second order kinetic models. Good correlation coefficients were obtained by fitting the experimental data to Michaelis- Menten and pseudo-second order kinetic models. The adsorption isotherms were described by Langmuir, Freundlich and Temkin isotherms. Among the three isotherm models, the Freundlich model was fitted with the equilibrium data obtained from adsorption of AR14 onto MnFe2O4/calcium alginate; while Temkin isotherm gave the best correlation for adsorption on MnFe2O4 nanoparticles. The effect of various parameters such as initial pH of solution, initial dye concentration, and contact time on the adsorption of AR14 on MnFe2O4 and MnFe2O4/ /calcium alginate as well as dye enzymatic decomposition was studied. The decolorization of AR14 with initial concentration of 10 mg.L?1 by using GOx/ /MnFe2O4/calcium alginate was 60.17%.
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7

Yang, Z. H., X. M. Chen, Y. P. Zhang, et al. "X-Ray Spectra of Hydrogen-Like and Helium-Like Argon Ions on Solid Surfaces." Solid State Phenomena 121-123 (March 2007): 995–98. http://dx.doi.org/10.4028/www.scientific.net/ssp.121-123.995.

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14-GHz electron cyclotron resonance ion source at the Heavy Ion Research Facility National Laboratory in Lanzhou has been used to investigate x-rays from the interaction of slow highly charged Ar17+and Ar16+ions for different energies with Be, Al, Ni, Mo and Au surfaces. Interaction of Ar17+ and Ar16+ ions with Metallic Surfaces for a wide range of energies has been studied by measuring the resulting x-ray emission. The characteristic features of the x-ray spectra have been explained.
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8

Freund, Monique, Jean-Pierre Cazenave, Michael Courtney, et al. "Inhibition by Recombinant Hirudins of Experimental Venous Thrombosis and Disseminated Intravascular Coagulation Induced by Tissue Factor in Rats." Thrombosis and Haemostasis 63, no. 02 (1990): 187–92. http://dx.doi.org/10.1055/s-0038-1645193.

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SummaryAntithrombotic potency of recombinant hirudins rHV2, rHV2–Lys47 and rHV2–Arg47 was studied in a model of experimental thrombosis induced by tissue factor in the rat. Venous thrombosis was induced by i.v. injection of 25 mg/kg tissue factor followed by stasis of the inferior vena cava. In this model natural recombinant hirudins, rHV2 and rHV2–Lys47 injected 5 min before thromboplastin totally inhibited thrombosis in the same μg range as heparin or natural hirudin extracted from leeches. However, the mutant variant rHV2–Arg47 gave a maximal 60% inhibition of thrombosis. Variants rHV2–Lys47 (30 μg/kg) and rHV2–Arg47 (157 μg/kg) injected 5 min before thromboplastin prevented by 90 to 100% the drop in platelet count observed during the disseminated intravascular coagulation induced by thromboplastin injection. Recombinant hirudins were less anticoagulant than heparin as measured by an APTT on rat plasma. After rat tail transection, rHV2–Lys47 caused a 2–fold smaller prolongation of the bleeding time than an equivalent antithrombotic dose of heparin. Plasmatic elimination of rHV2–Lys47 from rat plasma after i.v. injection had a fast distribution phase with a half-life of 3 min during which 90% of injected rHV2–Lys47 was lost and was followed by a slower elimination phase. Thus recombinant hirudin rHV2–Lys47 appears as a promising potent antithrombotic agent for the prevention of thrombin-dependent venous thrombosis and disseminated intravascular coagulation
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9

Ma, Wei, Gloria Fuentes, Xiaohe Shi, Chandra Verma, George K. Radda, and Weiping Han. "FoxO1 negatively regulates leptin-induced POMC transcription through its direct interaction with STAT3." Biochemical Journal 466, no. 2 (2015): 291–98. http://dx.doi.org/10.1042/bj20141109.

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The amino acid region Gly140–Leu160 (key residues Gln145, Arg147, Lys148, Arg153 and Arg154) of FoxO1 is identified as a STAT3 binding site, which is critical for FoxO1 to inhibit STAT3-mediated leptin-induced POMC activation.
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10

Pfoh, Roland, Emil F. Pai, and Vivian Saridakis. "Nicotinamide mononucleotide adenylyltransferase displays alternate binding modes for nicotinamide nucleotides." Acta Crystallographica Section D Biological Crystallography 71, no. 10 (2015): 2032–39. http://dx.doi.org/10.1107/s1399004715015497.

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Nicotinamide mononucleotide adenylyltransferase (NMNAT) catalyzes the biosynthesis of NAD+and NaAD+. The crystal structure of NMNAT fromMethanobacterium thermoautotrophicumcomplexed with NAD+and SO42−revealed the active-site residues involved in binding and catalysis. Site-directed mutagenesis was used to further characterize the roles played by several of these residues. Arg11 and Arg136 were implicated in binding the phosphate groups of the ATP substrate. Both of these residues were mutated to lysine individually. Arg47 does not interact with either NMN or ATP substrates directly, but was deemed to play a role in binding as it is proximal to Arg11 and Arg136. Arg47 was mutated to lysine and glutamic acid. Surprisingly, when expressed inEscherichia coliall of these NMNAT mutants trapped a molecule of NADP+in their active sites. This NADP+was bound in a conformation that was quite different from that displayed by NAD+in the native enzyme complex. When NADP+was co-crystallized with wild-type NMNAT, the same structural arrangement was observed. These studies revealed a different conformation of NADP+in the active site of NMNAT, indicating plasticity of the active site.
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11

Zhang, Zheng-rong, Xin-lu Cheng, Hui-fang Li, Hong Zhang, and Zi-jiang Liu. "Inner-shell filling and X-ray emission of Ar17+ ion in grazing incidence on an Al (111) surface." Canadian Journal of Physics 91, no. 1 (2013): 48–53. http://dx.doi.org/10.1139/cjp-2012-0344.

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Based on “length and energy scale matching” theory, we put forward a model for the Ar17+ ion in grazing incidence on an Al surface. Using the model and a more detailed side feeding rate, the inner-shell filling and X-ray emission of the incident Ar17+ ion are minutely studied. The results show that the final charged state of the ion gradually increases with increasing incident velocity, which is in accordance with experimental results. The central energies of K X-ray lines (Kα and Kβ) are mainly influenced by the vertical energy of the ion, that is, for a given incident angle the X-ray spectrum from the ion with bigger incident energy is dominated by the decay of the ion with fewer L- and M-shell vacancies. However, the intensity of the lines depends not only on the vertical velocity, but also the horizontal velocity of the incident ion. In addition, both of the K X-ray lines have large widths, which shows that they are emitted from an ion with a broad spectrum of L- and M-shell populations.
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12

Tang, Dan, Jingwen Sheng, Liangting Xu, et al. "Cryo-EM structure of C9ORF72–SMCR8–WDR41 reveals the role as a GAP for Rab8a and Rab11a." Proceedings of the National Academy of Sciences 117, no. 18 (2020): 9876–83. http://dx.doi.org/10.1073/pnas.2002110117.

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A massive intronic hexanucleotide repeat (GGGGCC) expansion in C9ORF72 is a genetic origin of amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD). Recently, C9ORF72, together with SMCR8 and WDR41, has been shown to regulate autophagy and function as Rab GEF. However, the precise function of C9ORF72 remains unclear. Here, we report the cryogenic electron microscopy (cryo-EM) structure of the human C9ORF72–SMCR8–WDR41 complex at a resolution of 3.2 Å. The structure reveals the dimeric assembly of a heterotrimer of C9ORF72–SMCR8–WDR41. Notably, the C-terminal tail of C9ORF72 and the DENN domain of SMCR8 play critical roles in the dimerization of the two protomers of the C9ORF72–SMCR8–WDR41 complex. In the protomer, C9ORF72 and WDR41 are joined by SMCR8 without direct interaction. WDR41 binds to the DENN domain of SMCR8 by the C-terminal helix. Interestingly, the prominent structural feature of C9ORF72–SMCR8 resembles that of the FLNC–FNIP2 complex, the GTPase activating protein (GAP) of RagC/D. Structural comparison and sequence alignment revealed that Arg147 of SMCR8 is conserved and corresponds to the arginine finger of FLCN, and biochemical analysis indicated that the Arg147 of SMCR8 is critical to the stimulatory effect of the C9ORF72–SMCR8 complex on Rab8a and Rab11a. Our study not only illustrates the basis of C9ORF72–SMCR8–WDR41 complex assembly but also reveals the GAP activity of the C9ORF72–SMCR8 complex.
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13

Kang, Le, Fulin Li, and Yan Li. "Identifying Factors that Influence International Student Mobility in Chinese Higher Education Institutions." Science Insights Education Frontiers 4, no. 1 (2019): 339–54. http://dx.doi.org/10.15354/sief.19.ar107.

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14

Bostock, Christopher J., Dmitry V. Fursa та I. Bray. "Polarization of the Lyman-α1 X-ray line emitted by hydrogen-like Ti21+, Ar17+, and Fe25+ ions excited by electron impact1This review is part of a Special Issue on the 10th International Colloquium on Atomic Spectra and Oscillator Strengths for Astrophysical and Laboratory Plasmas." Canadian Journal of Physics 89, № 5 (2011): 503–7. http://dx.doi.org/10.1139/p11-012.

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We present a review of the relativistic convergent close-coupling (RCCC) method and describe how it has been used to resolve the discrepancy between theory and experiment for the polarization of the Lyman-α1 X-ray line emitted by hydrogen-like Ti21+, Ar17+, and Fe25+ ions excited by electron impact. We find that taking account of Breit relativistic corrections is important to resolve the discrepancy between experiment and theoretical calculations.
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15

Arocas, Véronique, Susan C. Bock, Steven T. Olson, and Ingemar Björk. "The Role of Arg46 and Arg47 of Antithrombin in Heparin Binding†." Biochemistry 38, no. 31 (1999): 10196–204. http://dx.doi.org/10.1021/bi990686b.

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16

Takács, E., Z. Ónodi-Szűcs, L. P. Ratliff, J. D. Gillaspy, and J. Pálinkás. "X-ray emission for 3–137 keV Ar17+ impacting SiO2." Nuclear Instruments and Methods in Physics Research Section B: Beam Interactions with Materials and Atoms 124, no. 2-3 (1997): 431–34. http://dx.doi.org/10.1016/s0168-583x(96)01054-3.

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17

Rice, J. E., M. L. Reinke, J. M. A. Ashbourn та ін. "The Ar17 +Lyα2/Lyα1ratio in Alcator C-Mod tokamak plasmas". Journal of Physics B: Atomic, Molecular and Optical Physics 44, № 16 (2011): 165702. http://dx.doi.org/10.1088/0953-4075/44/16/165702.

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18

Hamasha, Safeia M., and Yasmeen Abu-Nassar. "Theoretical spectral analysis of Ar ions from Ar9+ to Ar14+." Journal of Quantitative Spectroscopy and Radiative Transfer 266 (May 2021): 107567. http://dx.doi.org/10.1016/j.jqsrt.2021.107567.

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19

Wallace, T. L. "Acute, Multiple-Dose, and Genetic Toxicology of AR177, an Anti-HIV Oligonucleotide." Toxicological Sciences 53, no. 1 (2000): 63–70. http://dx.doi.org/10.1093/toxsci/53.1.63.

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20

Douady, J., E. Jacquet, E. Giglio, D. Zanuttini, and B. Gervais. "Non-adiabatic molecular dynamics of excited Na2+ solvated in Ar17 clusters." Chemical Physics Letters 476, no. 4-6 (2009): 163–67. http://dx.doi.org/10.1016/j.cplett.2009.06.032.

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21

Nakano, Y., A. A. Sokolik, A. V. Stysin, et al. "Metastable Ar17+(2 s) production by Stark-assisted resonant coherent excitation." Journal of Physics B: Atomic, Molecular and Optical Physics 48, no. 14 (2015): 144026. http://dx.doi.org/10.1088/0953-4075/48/14/144026.

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22

Este, J. A., D. Schols, M. Witvrouw, et al. "Human immunodeficiency virus gp120 as the primary target of action of AR177 (Zintevir)." Antiviral Research 34, no. 2 (1997): A57. http://dx.doi.org/10.1016/s0166-3542(97)83191-9.

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23

Boldinova, Elizaveta O., Аnna А. Manukyan, and Аlena V. Makarova. "The DNA ligands Arg47 and Arg76 are crucial for catalysis by human PrimPol." DNA Repair 100 (April 2021): 103048. http://dx.doi.org/10.1016/j.dnarep.2021.103048.

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24

Azuma, T., T. Ito, Y. Yamazaki, et al. "Resonant coherent excitation of relativistic Ar17+ ions channeled in a Si crystal." Nuclear Instruments and Methods in Physics Research Section B: Beam Interactions with Materials and Atoms 135, no. 1-4 (1998): 61–65. http://dx.doi.org/10.1016/s0168-583x(97)00563-6.

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25

Yang Zhi-Hu, Song Zhang-Yong, Cui Ying, et al. "X-ray spectra produced by interaction of Ar16+ and Ar17+ with Zr." Acta Physica Sinica 57, no. 2 (2008): 803. http://dx.doi.org/10.7498/aps.57.803.

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26

GOU, BINGCONG, YIDONG LIU, and FENG WANG. "ELECTRON CORRELATION EFFECTS FOR DOUBLY-EXCITED STATES OF Be-LIKE Ar14+ ION." International Journal of Modern Physics B 18, no. 17n19 (2004): 2590–94. http://dx.doi.org/10.1142/s0217979204025725.

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The energies, radiative and Auger rates of the doubly excited states of Be-like Ar 14+ ion are studied by using the multi-configuration-interaction method and model potential method. The doubly excited states of Be-like Ar 14+ are labeled by the quantum numbers K, T and A to show the systematic regularity. The results show that the spectroscopy of Be-like ions is different from that of He-like ions because of the polarization and core penetration effects from the 1s2 core electrons.
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27

Dong, C. Z., C. Z. Dong, S. Fritzsche, B. Fricke, and W. D. Sepp. "Ab-initio Calculations for Forbidden M1 Transitions in Ar13+ and Ar14+ Ions." Physica Scripta T92, no. 1 (2001): 294–96. http://dx.doi.org/10.1238/physica.topical.092a00294.

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28

Ishikawa, Yasuyuki, Hideo Sekino, and Robert C. Binning. "Relativistic many-body perturbation theory calculations on Be, Ne6+, Ar14+ AND Ne." Chemical Physics Letters 160, no. 2 (1989): 206–11. http://dx.doi.org/10.1016/0009-2614(89)87583-9.

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29

Ma, Kun, Zhan-Bin Chen, Lu-You Xie, et al. "State-selective quantum interference studied in the photo-recombination of Ar17+ ion." Journal of Electron Spectroscopy and Related Phenomena 232 (April 2019): 29–34. http://dx.doi.org/10.1016/j.elspec.2019.01.004.

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30

Salvi, F., F. Pastorelli, R. Plasmati, et al. "Early onset aggressive hereditary amyloidosis: report of an Italian family with TTR Arg47 mutation." Neurological Sciences 26, no. 2 (2005): 140–42. http://dx.doi.org/10.1007/s10072-005-0449-y.

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31

Marchuk, O., Yu Ralchenko, R. K. Janev, G. Bertschinger, and W. Biel. "Kinetics of highly excited states in Ar17+charge exchange recombination fusion plasma spectroscopy." Journal of Physics B: Atomic, Molecular and Optical Physics 42, no. 16 (2009): 165701. http://dx.doi.org/10.1088/0953-4075/42/16/165701.

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32

Marchuk, O., Yu Ralchenko, R. K. Janev, G. Bertschinger, and W. Biel. "Kinetics of highly excited states in Ar17+charge exchange recombination fusion plasma spectroscopy." Journal of Physics B: Atomic, Molecular and Optical Physics 42, no. 18 (2009): 189801. http://dx.doi.org/10.1088/0953-4075/42/18/189801.

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33

Natarajan, L., and Y. G. Mulye. "Multi-configuration Dirac-Fock and configuration-interaction calculations of Ar7+to Ar17+ions." Journal of Physics B: Atomic, Molecular and Optical Physics 34, no. 9 (2001): 1839–54. http://dx.doi.org/10.1088/0953-4075/34/9/318.

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34

Salehzadeh, Arash, and Tom Kirchner. "Strong multiple-capture effect in slow Ar17+–Ar collisions: a quantum mechanical analysis." Journal of Physics B: Atomic, Molecular and Optical Physics 46, no. 2 (2013): 025201. http://dx.doi.org/10.1088/0953-4075/46/2/025201.

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35

Lamour, Emily, Christophe Prigent, Benjamin Eberhardt, Jean Pierre Rozet, and Dominique Vernhet. "2E1 Ar17+ decay and conventional radioactive sources to determine efficiency of semiconductor detectors." Review of Scientific Instruments 80, no. 2 (2009): 023103. http://dx.doi.org/10.1063/1.3077284.

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36

Este, J. A., P. Proost, S. Struyf, et al. "Interactions of the HIV fusion inhibitor AR177 (ZINTEVIR) with the HIV type I second receptor." Antiviral Research 34, no. 2 (1997): A41. http://dx.doi.org/10.1016/s0166-3542(97)83141-5.

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37

Esté, José A., Cecilia Cabrera, Dominique Schols, et al. "Human Immunodeficiency Virus Glycoprotein gp120 as the Primary Target for the Antiviral Action of AR177 (Zintevir)." Molecular Pharmacology 53, no. 2 (1998): 340–45. http://dx.doi.org/10.1124/mol.53.2.340.

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38

Caso, R., D. A. Lane, E. Thompson, et al. "Antithrombin padua I: Impaired heparin binding caused by an ARG47 to his (CGT to CAT) substitution." Thrombosis Research 58, no. 2 (1990): 185–90. http://dx.doi.org/10.1016/0049-3848(90)90175-c.

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39

Prigent, C., E. Lamour, J. Mérot, et al. "X-ray spectroscopy characterization of Ar17+produced by an ECRIS in the afterglow mode." Journal of Physics: Conference Series 163 (April 1, 2009): 012111. http://dx.doi.org/10.1088/1742-6596/163/1/012111.

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40

Nakano, Y., A. Hatakeyama, Y. Nakai, et al. "Effects of fine-structure depolarization studied by resonant coherent excitation of H-like Ar17+." Physica Scripta T156 (September 1, 2013): 014061. http://dx.doi.org/10.1088/0031-8949/2013/t156/014061.

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41

Bhat, Ashwin, and Durgadas P. Kasbekar. "Escape From Repeat-Induced Point Mutation of a Gene-Sized Duplication in Neurospora crassa Crosses That Are Heterozygous for a Larger Chromosome Segment Duplication." Genetics 157, no. 4 (2001): 1581–90. http://dx.doi.org/10.1093/genetics/157.4.1581.

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Abstract In Neurospora crassa the ability of an ectopic gene-sized duplication to induce repeat-induced point mutation (RIP) in its target gene was suppressed in crosses that were heterozygous for another larger chromosome segment duplication. Specifically, the frequency of RIP in the erg-3 gene due to a 1.3-kb duplication was reduced if the chromosome segment duplications Dp(IIIR > [I;II]) AR17, Dp(VIR > IIIR) OY329, or Dp(IVR > VII) S1229 were present in either the same or the other parental nucleus of the premeiotic dikaryon. We suggest that the larger duplications act as sinks to titrate the RIP machinery away from the smaller duplication. In contrast, RIP efficiency was relatively unaffected in comparably unproductive interspecies crosses with N. intermedia and N. tetrasperma. These findings offer a novel explanation for the observed persistence of the transposable element Tad in only a subset of Neurospora strains.
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42

Guerin, M. "Familial lecithin:cholesterol acyltransferase deficiency: molecular analysis of a compound heterozygote: LCAT (Arg147→Trp) and LCAT (Tyr171→Stop)." Atherosclerosis 131, no. 1 (1997): 85–95. http://dx.doi.org/10.1016/s0021-9150(97)06079-6.

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43

Wan, Jianjie, and Chenzhong Dong. "The effects of quantum interference between direct and resonant photorecombination of H-like Ar17+ions." Physica Scripta 84, no. 4 (2011): 045301. http://dx.doi.org/10.1088/0031-8949/84/04/045301.

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44

Babaev, A. A., and Yu L. Pivovarov. "Resonant coherent excitation of Ar17+ ions taking into account a fine structure of energy levels." Journal of Surface Investigation. X-ray, Synchrotron and Neutron Techniques 2, no. 2 (2008): 241–44. http://dx.doi.org/10.1134/s1027451008020158.

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45

Takabayashi, Y., T. Ito, T. Azuma, et al. "Convoy Electron Production and Ionization in 390 MeV/u Ar17+ Ion Collisions with Thin Foils." Physica Scripta T80, B (1999): 249. http://dx.doi.org/10.1238/physica.topical.080a00249.

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Takács, E., Z. Berényi, J. D. Gillaspy, et al. "Separation of inner-shell vacancy transfer mechanisms in collisions of slow Ar17+ions with SiO2." Journal of Physics B: Atomic, Molecular and Optical Physics 34, no. 7 (2001): 1277–87. http://dx.doi.org/10.1088/0953-4075/34/7/310.

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47

Brouwer, Rick, Ger JM Pruijn, and Walther J. van Venrooij. "The human exosome: an autoantigenic complex of exoribonucleases in myositis and scleroderma." Arthritis Research & Therapy 3, no. 2 (2000). http://dx.doi.org/10.1186/ar147.

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48

Zigon, P., A. Hocevar, S. Cucnik, et al. Arthritis Research & Therapy 6, Suppl 1 (2004): 5. http://dx.doi.org/10.1186/ar1047.

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49

Rosen, Oliver, Andreas Thiel, Gero Massenkeil, et al. Arthritis Research 2, no. 4 (2000): 327. http://dx.doi.org/10.1186/ar107.

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50

Cavazzana, I., F. Franceschini, C. Vassalini, M. Quinzanini, L. Andreoli, and R. Cattaneo. Arthritis Research & Therapy 6, Suppl 1 (2004): 105. http://dx.doi.org/10.1186/ar1147.

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