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Journal articles on the topic 'Arginine alpha-ketoglutarate'

Author: Grafiati

Published: 4 June 2025

Last updated: 1 August 2025

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1

Le Boucher, Jacques, Biol Eng, Colette Coudray-Lucas, et al. "Enteral administration of ornithine alpha-ketoglutarate or arginine alpha-ketoglutarate." Critical Care Medicine 25, no. 2 (1997): 293–98. http://dx.doi.org/10.1097/00003246-199702000-00017.

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2

Tell, Grethe S., Livar Frøyland, Margaretha Haugen, et al. "Risk Assessment of "Other Substances" –L-arginine and Arginine Alpha-ketoglutarate." European Journal of Nutrition & Food Safety 9, no. 1 (2018): 33–35. http://dx.doi.org/10.9734/ejnfs/2019/45632.

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3

Tseh, Wayland, Brian W. Cioci, and Don W. Morgan. "The Effects of Arginine Alpha-Ketoglutarate Supplementation on Endurance-Trained Females." Medicine & Science in Sports & Exercise 40, Supplement (2008): S401. http://dx.doi.org/10.1249/01.mss.0000322710.71572.ef.

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4

Wax, B., H. E. Webb, M. C. Harris, et al. "Effect of L-Arginine Alpha-Ketoglutarate Ingestion on Muscular Strength and Endurance." Journal of Strength and Conditioning Research 25 (March 2011): S111. http://dx.doi.org/10.1097/01.jsc.0000395756.12955.a0.

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5

Gospodaryov, Dmytro, Vitaliy Balatskiy, and Maria Bayliak. "Bioenergetic basis for the effects of arginine and alpha-ketoglutarate on lifespan." Biochimica et Biophysica Acta (BBA) - Bioenergetics 1859 (September 2018): e59. http://dx.doi.org/10.1016/j.bbabio.2018.09.177.

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6

Forbes, Scott C., Darren G. Candow, Jonathon Little, and Philip D. Chilibeck. "Effects of a Nutritional Supplement Containing Creatine Monohydrate and Arginine Alpha-Ketoglutarate in Young Males." Medicine & Science in Sports & Exercise 39, Supplement (2007): S364. http://dx.doi.org/10.1249/01.mss.0000274427.90982.a8.

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7

Horbal, Liliya, Marc Stierhof, Anja Palusczak, Nikolas Eckert, Josef Zapp, and Andriy Luzhetskyy. "Cyclofaulknamycin with the Rare Amino Acid D-capreomycidine Isolated from a Well-Characterized Streptomyces albus Strain." Microorganisms 9, no. 8 (2021): 1609. http://dx.doi.org/10.3390/microorganisms9081609.

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Targeted genome mining is an efficient method of biosynthetic gene cluster prioritization within constantly growing genome databases. Using two capreomycidine biosynthesis genes, alpha-ketoglutarate-dependent arginine beta-hydroxylase and pyridoxal-phosphate-dependent aminotransferase, we identified two types of clusters: one type containing both genes involved in the biosynthesis of the abovementioned moiety, and other clusters including only arginine hydroxylase. Detailed analysis of one of the clusters, the flk cluster from Streptomyces albus, led to the identification of a cyclic peptide t

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8

Willoughby, Darryn S., Tony Boucher, Jeremy Reid, Garson Skelton, and Mandy Clark. "Effects of 7 Days of Arginine-Alpha-Ketoglutarate Supplementation on Blood Flow, Plasma L-Arginine, Nitric Oxide Metabolites, and Asymmetric Dimethyl Arginine After Resistance Exercise." International Journal of Sport Nutrition and Exercise Metabolism 21, no. 4 (2011): 291–99. http://dx.doi.org/10.1123/ijsnem.21.4.291.

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Background:Arginine-alpha-ketoglutarate (AAKG) supplements are alleged to increase nitric oxide production, thereby resulting in vasodilation during resistance exercise. This study sought to determine the effects of AAKG supplementation on hemodynamics and brachial-artery blood flow and the circulating levels of L-arginine, nitric oxide metabolites (NOx; nitrate/nitrite), asymmetric dimethyl arginine (ADMA), and L-arginine:ADMA ratio after resistance exercise.Methods:Twenty-four physically active men underwent 7 days of AAKG supplementation with 12 g/day of either NO2 Platinum or placebo (PLC)

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9

Wax, Benjamin, Jerry J. Mayo, Laura A. Hilton, et al. "Acute Ingestion Of L-Arginine Alpha-Ketoglutarate Fails To Improve Muscular Strength And Endurance In ROTC Cadets." International Journal of Exercise Science 6, no. 2 (2013): 91–97. http://dx.doi.org/10.70252/ssjj5515.

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L-Arginine Alpha-Ketoglutarate (AAKG) is purported to stimulate the release of nitric oxide, and is suggested to facilitate muscular performance by increasing blood flow and increase oxygen and nutrient delivery to the working muscle. However, the ergogenic benefit of AAKG during resistance exercise has not been established. Therefore the purpose of this study was to investigate the effects of acute AAKG ingestion in active ROTC Cadets on measures of one-repetition maximal strength (1RM) and muscular endurance. Nineteen apparently healthy males ingested either AAKG (3 g) or a placebo 45 minute

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10

Gumina, S., D. Passaretti, M. D. Gurzì, and V. Candela. "Arginine L-alpha-ketoglutarate, methylsulfonylmethane, hydrolyzed type I collagen and bromelain in rotator cuff tear repair: a prospective randomized study." Current Medical Research and Opinion 28, no. 11 (2012): 1767–74. http://dx.doi.org/10.1185/03007995.2012.737772.

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11

Chang, C. J., and R. C. Donaldson. "Nutritional requirements of Xylella fastidiosa, which causes Pierce's disease in grapes." Canadian Journal of Microbiology 46, no. 3 (2000): 291–93. http://dx.doi.org/10.1139/w99-141.

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A defined medium (XF-26) containing 3 inorganic salts, 2 tricarboxylic acids, 17 amino acids, potato starch, phenol red, and agar was used as the starting point for the study. Deletions of one or more ingredients were performed to prepare various media. A medium was considered able to support growth of Xylella fastidiosa strains responsible for Pierce's disease in grapes, only after 10 serial passages had been completed. Of 3 inorganic salts, K2HPO4 and MgSO4·7H2O were essential, and (NH4)2HPO4 was nonessential for growth. Of the Krebs cycle intermediates, all (citrate, alpha-ketoglutarate, su

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12

Mirabile, Aurora, Licia Rivoltini, Elena Daveri, et al. "Metabolism and Immune Modulation in Patients with Solid Tumors: Systematic Review of Preclinical and Clinical Evidence." Cancers 12, no. 5 (2020): 1153. http://dx.doi.org/10.3390/cancers12051153.

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Several immunotherapy agents are the standard of care of many solid malignancies. Nevertheless, the majority of patients do not benefit from the currently available immunotherapies. It is therefore of paramount importance to identify the prognostic and predictive factors of tumor response/resistance and to design effective therapeutic strategies to overcome primary resistance and improve the efficacy of immunotherapy. The aim of this review is to underline the influence of the tumor and host metabolism on the antitumor immune response and to discuss possible strategies to improve the efficacy

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13

Kruszewski, Marek, Maciej Merchelski, Artur Kruszewski, Rafał Tabęcki, Maksim Olegovich Aksenov, and Łukasz Pągowski. "Effects of Multi-Ingredient Pre-Workout Supplement and Caffeine on Bench Press Performance: A Single-Blind Cross-Over Study." Nutrients 14, no. 9 (2022): 1750. http://dx.doi.org/10.3390/nu14091750.

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The problem addressed in this study is the appropriateness of using different pre-training supplementation strategies and their ability to improve training performance and psychological measures. The aim of the study is the evaluation of the effectiveness of a multi-ingredient pre-workout supplement (MIPS) containing beta-alanine, L-citrulline malate, arginine alpha-ketoglutarate, L-taurine, L-tyrosine and caffeine compared to an exact dosage of anhydrous caffeine in bench press strength endurance, feeling scale (FS), felt arousal scale (FAS) and session rating of perceived exertion (sRPE). A

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14

Božić, Dorotea, Ante Tonkić, Katarina Vukojevic, and Maja Radman. "A Case Report: Idiopathic or Drug-Induced Autoimmune Hepatitis—Can We Draw a Line?" Clinics and Practice 13, no. 6 (2023): 1393–99. http://dx.doi.org/10.3390/clinpract13060125.

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Idiosyncratic drug-induced liver injury (DILI) is an unpredictable reaction of individuals exposed to a certain drug, and drug-induced autoimmune hepatitis (DIAIH) presents a DILI phenotype that mimics idiopathic autoimmune hepatitis (AIH) when considering the clinical, biochemical, serological and histological parameters. We present a case report of a 48-year-old male who was hospitalized due to severe hepatocellular liver injury two months after self-treatment with a muscle-building dietary supplement based on arginine-alpha-ketoglutarate, L-citrulline, L tyrosine, creatine malate and beet e

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15

Mu, Ruina, Songze Li, Yunxi Zhang, et al. "Microbiota and Metabolite Profiles in the Feces of Juvenile Sika Deer (Cervus nippon) from Birth to Weaning." Animals 14, no. 3 (2024): 432. http://dx.doi.org/10.3390/ani14030432.

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The gut microbiota establishment in young ruminants has a profound impact on their adult production performance. However, the critical phase for the succession of the gut microbial composition and metabolic profiles of juvenile sika deer still needs to be further investigated. Here, we analyzed the fecal microbiota and metabolites of juvenile sika deer during the birth (D1), transition (D42), and rumination (D70) periods based on 16S rRNA sequencing and gas chromatography–time–of–flight mass spectrometry (GC–TOF–MS). The results showed that the fecal bacteria and metabolites composition were s

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16

Lasho, Terra, Alexander Tischer, Guadalupe Belen Antelo, et al. "Functional Interrogation of Variants of Undetermined Significance of the Isocitrate Dehydrogenase 1 and 2 Genes in Myeloid Neoplasms." Blood 134, Supplement_1 (2019): 1697. http://dx.doi.org/10.1182/blood-2019-128067.

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Introduction: Isocitrate dehydrogenase 1 and 2 (IDH1/2) are metabolic enzymes in the citric acid cycle, producing alpha-ketoglutarate (αKG). Mutations in specific regions of these genes have been characterized in gliomas, AML and chronic myeloid neoplasms such as MDS and MPN. These mutations produce an oncometabolite 2-hydroxyglutarate (2HG) that disrupts the epigenetic phenotype of myeloid cells promoting oncogenesis. While this process has been characterized for arginine hot spot (R132/R140) mutations, there exist variants of unclear significance (IDHVUS), with potential pathogenic predictio

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17

Thomas, Daniel, Subarna Sinha, Steven M. Chan, et al. "Isocitrate Dehydrogenase 1 Mutant Cancers Are Metabolically Vulnerable to Inhibition of Acetyl CoA Carboxylase Via a 2-Hydroxyglutarate Independent Mechanism." Blood 128, no. 22 (2016): 1054. http://dx.doi.org/10.1182/blood.v128.22.1054.1054.

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Abstract Introduction: Mutations substituting arginine 132 of isocitrate dehydrogenase 1 (IDH1) are recurrent in acute myeloid leukemia (AML) and several other cancers, resulting in the aberrant production of the onco-metabolite, R-2-hydroxyglutarate (2-HG), as well as an inability to convert cytoplasmic alpha-ketoglutarate to isocitrate via reductive carboxylation. Currently, small molecules that effectively inhibit the neomorphic enzyme and abrogate the production of 2-HG, such as AG-120, are in clinical trials with promising results. However, these inhibitors have not proven to be curative

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18

Chandhok, Namrata S., Wei Wei, Stephanie Halene, and Thomas Prebet. "Ivo-Nivo: A Phase II Study of the IDH1 Inhibitor Ivosidenib (AG-120) in Combination with the Checkpoint Blockade Inhibitor Nivolumab for Patients with IDH1 Mutated Relapsed/Refractory AML and High Risk MDS." Blood 134, Supplement_1 (2019): 1374. http://dx.doi.org/10.1182/blood-2019-130033.

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Background: The normal function of isocitrate dehydrogenase 1 (IDH1) is to catalyze the conversion of cytoplasmic isocitrate to α-ketoglutarate (aKG) in the Krebs cycle. In hematologic malignancies a hotspot mutation at arginine 132 within the conserved active site of IDH1 leads to loss of the expected Krebs Cycle reaction as well as production of the oncometabolite 2- hydroxyglutarate (2-HG). In acute myeloid leukemia (AML) increased cellular 2-HG levels has been shown to impact the tumor microenvironment and promotes tumorigenesis via paracrine stimulation as well repression of the tumor ass

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19

Udumula, Mary Priyanka, Harshit Singh, Faraz Rashid, et al. "Abstract 527: Targeting citric acid cycle anaplerosis reverses the immunosuppressive phenotype of CD11b+Gr1+ myeloid cells in ovarian cancer." Cancer Research 83, no. 7_Supplement (2023): 527. http://dx.doi.org/10.1158/1538-7445.am2023-527.

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Abstract Immunosuppressive myeloid cells (CD11b+Gr1+) are a major constituent of the epithelial ovarian cancer (EOC) microenvironment that confer immunosuppression and promote tumor growth. We recently showed that the EOC microenvironment can induce metabolic reprogramming inCD11b+Gr1+ cells by increasing oxidative phosphorylation via TCA cycle, resulting in increased immunosuppressive ability. Increased TCA cycle activity was due to glutamine anaplerosis, which was facilitated by the upregulation of DLST (dihydrolipoamide succinyl transferase), the E2transferase subunit of α-KGDC (alpha-ketog

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20

Norwegian, Scientific Committee for Food Safety (VKM). "Risk assessment of "other substances" – L-arginine and arginine alpha-ketoglutarate. Opinion of the Panel on Nutrition, dietetic products, Novel Food an Allergy of the Norwegian Scientific Committee for Food Safety." March 1, 2016. https://doi.org/10.5281/zenodo.4024172.

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The Norwegian Scientific Committee for Food Safety (VKM) has, at the request of the Norwegian Food Safety Authority, assessed the risk of specified doses of L-arginine and arginine alpha-ketoglutarate in food supplements. VKM concludes that: In adults (≥18 years), the specified doses of 3000, 3500, 4000, 4500, 5000, 5500 and 6000 mg/day of arginine in food supplements are considered unlikely to cause adverse health effects. The dose 6800 mg/day may represent a risk of adverse health effects. In adolescents (14 to <18 years), the specified doses 3000, 3500, 4000, 4500, 5000, 5500 mg/day L

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21

Karlstaedt, Anja, Heidi Vitrac, Koen Raedschelders, et al. "Abstract 16708: Inhibition of Alpha-Ketoglutarate Dehydrogenase Activates Autophagy." Circulation 138, Suppl_1 (2018). http://dx.doi.org/10.1161/circ.138.suppl_1.16708.

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Introduction: Metabolic reprogramming is a hallmark of cancer and heart failure. In isocitrate dehydrogenase 1 and 2 mutant tumors, increased plasma levels of the oncometabolite D-2-hydroxyglutarate (D2-HG) inhibit α-ketoglutarate dehydrogenase (α-KGDH) in the heart. This inhibition is associated with muscle atrophy and reduced ATP and NADH provision. The connection between metabolic changes and proteome remodeling are still poorly understood. Hypothesis: Inhibition of α-KGDH promotes autophagy activation through the sirtuin family of NAD + -dependent deacetylases. Methods: We treated both mal

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22

Thor, Peter, Fanny Vermandele, Allison Bailey, et al. "Ocean acidification causes fundamental changes in the cellular metabolism of the Arctic copepod Calanus glacialis as detected by metabolomic analysis." Scientific Reports 12, no. 1 (2022). http://dx.doi.org/10.1038/s41598-022-26480-9.

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AbstractUsing a targeted metabolomic approach we investigated the effects of low seawater pH on energy metabolism in two late copepodite stages (CIV and CV) of the keystone Arctic copepod species Calanus glacialis. Exposure to decreasing seawater pH (from 8.0 to 7.0) caused increased ATP, ADP and NAD+ and decreased AMP concentrations in stage CIV, and increased ATP and phospho-L-arginine and decreased AMP concentrations in stage CV. Metabolic pathway enrichment analysis showed enrichment of the TCA cycle and a range of amino acid metabolic pathways in both stages. Concentrations of lactate, ma

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23

Moosakhani, Alireza, Siroos Choobineh, and Rahman Soori. "Effect of L-arginine alpha ketoglutarate supplementation on hormonal response during recovery in male endurance swimmers." Gazzetta Medica Italiana Archivio per le Scienze Mediche 177, no. 3 (2018). http://dx.doi.org/10.23736/s0393-3660.17.03497-0.

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24

Wax, Benjamin, Andreas N. Kavazis, Heather E. Webb, and Stanley P. Brown. "Acute L-arginine alpha ketoglutarate supplementation fails to improve muscular performance in resistance trained and untrained men." Journal of the International Society of Sports Nutrition 9, no. 1 (2012). http://dx.doi.org/10.1186/1550-2783-9-17.

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25

Druseikis, Marina E., and Shay Covo. "Synthetic lethality between toxic amino acids, RTG‐target genes and chaperones in Saccharomyces cerevisiae." Yeast, July 30, 2024. http://dx.doi.org/10.1002/yea.3975.

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AbstractThe toxicity of non‐proteinogenic amino acids has been known for decades. Numerous reports describe their antimicrobial/anticancer potential. However, these molecules are often toxic to the host as well; thus, a synthetic lethality approach that reduces the dose of these toxins while maintaining toxicity can be beneficial. Here we investigate synthetic lethality between toxic amino acids, the retrograde pathway, and molecular chaperones. In Saccharomyces cerevisiae, mitochondrial retrograde (RTG) pathway activation induces transcription of RTG‐target genes to replenish alpha‐ketoglutar

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26

"Acetyl tyrosine/alpha ketoglutarate/arginine/ascorbic acid/beta alanine/caffeine/cobalamin/creatine/folic acid/mucuna pruiens/niacin/pyridoxine/theacrine overdose." Reactions Weekly 1907, no. 1 (2022): 16. http://dx.doi.org/10.1007/s40278-022-15275-0.

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27

Reid, Jeremy, Garson Skelton, Mandy Clark, Anthony Boucher, and Darryn S. Willoughby. "Effects of 7 days of arginine-alpha-ketoglutarate supplementation using NO2 Platinum on brachial artery blood flow and the levels of plasma L-arginine, nitric oxide, and eNOS after resistance exercise." Journal of the International Society of Sports Nutrition 7, S1 (2010). http://dx.doi.org/10.1186/1550-2783-7-s1-p22.

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28

Mazzetti. "Influence of Xpand Nitric Oxide Reactor, L-Arginine Alpha-Ketoglutarate, and Caffeine Supplementation on Calf Muscle Re-Oxygenation During and after Acute Resistance Exercise." Journal of Nutritional Therapeutics, 2012. http://dx.doi.org/10.6000/1929-5634.2012.01.02.5.

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29

Burton, Ian, and Aisling McCormack. "Nutritional Supplements in the Clinical Management of Tendinopathy: A Scoping Review." Journal of Sport Rehabilitation, 2023, 1–12. http://dx.doi.org/10.1123/jsr.2022-0244.

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Introduction: Tendinopathy has a high prevalence and incidence in the general population and among athletes, with a lack of consensus among medical practitioners on optimal management strategies. The objective of this scoping review was to evaluate current research on the use of nutritional supplements for treating tendinopathies, including what supplements have been used and what outcomes, outcome measures, and intervention parameters have been reported. Methods: Databases searched included Embase, SPORTDiscus, the Cochrane Library, MEDLINE, CINAHL, and AMED. This scoping review considered pr

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30

Gonzalez Izundegui, Daniel, Patricia E. Miller, Ravi V. Shah, et al. "Response of circulating metabolites to an oral glucose challenge and risk of cardiovascular disease and mortality in the community." Cardiovascular Diabetology 21, no. 1 (2022). http://dx.doi.org/10.1186/s12933-022-01647-w.

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Abstract Background New biomarkers to identify cardiovascular disease (CVD) risk earlier in its course are needed to enable targeted approaches for primordial prevention. We evaluated whether intraindividual changes in blood metabolites in response to an oral glucose tolerance test (OGTT) may provide incremental information regarding the risk of future CVD and mortality in the community. Methods An OGTT (75 g glucose) was administered to a subsample of Framingham Heart Study participants free from diabetes (n = 361). Profiling of 211 plasma metabolites was performed from blood samples drawn be

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31

Jewell, Dennis Edward, Jodi C. Vondran, and Melissa A. Vanchina. "Cat's choose 30% of their calories as protein which is associated with both body composition and circulating metabolites." FASEB Journal 30, S1 (2016). http://dx.doi.org/10.1096/fasebj.30.1_supplement.915.42.

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As in other mammals, domestic cats have specific taste preferences that can be influenced by macronutrient composition and physical characteristics of food as well as the presence or absence of specific taste imparting compounds (palatability enhancers). This study investigated the relationship of body composition and food choice and that choice on subsequent metabolomic shifts. Experimental foods were prepared with similar palatability for a population (as evaluated by the ratio of the test food to the total daily food consumed [grams test/(grams test+grams control)] through manipulations of

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32

Semenikhina, Marharyta, Courtney J. Christopher, Mykhailo Fedoriuk, et al. "Sex-Specific Mitochondrial Metabolic and Transcriptomic Signatures in Type 2 Diabetic Kidney Disease." Physiology 40, S1 (2025). https://doi.org/10.1152/physiol.2025.40.s1.0202.

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Background: Type 2 diabetic kidney disease (T2DKD) is a leading cause of end-stage renal disease (ESRD), characterized by metabolic dysregulation and progressive mitochondrial pathology. Emerging evidence highlights sex differences in the pathophysiology of T2DKD, yet the underlying mechanisms remain poorly understood. Men exhibit accelerated development of kidney damage with high oxidative stress and fast development of fibrosis and podocyte dysfunction; however, women have a higher risk of ESRD development and poorer cardiovascular outcomes. We hypothesized that mitochondria-specific metabol

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