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Relevant bibliographies by topics / Arginine vasopressine [AVP]

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Academic literature on the topic 'Arginine vasopressine [AVP]'

Author: Grafiati

Published: 4 June 2021

Last updated: 3 February 2022

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Journal articles on the topic "Arginine vasopressine [AVP]"

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Pósa, Anikó, Krisztina Kupai, Rudolf Ménesi, et al. "Sexual Dimorphism of Cardiovascular Ischemia Susceptibility Is Mediated by Heme Oxygenase." Oxidative Medicine and Cellular Longevity 2013 (2013): 1–11. http://dx.doi.org/10.1155/2013/521563.

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We investigated the gender differences in heme-oxygenase (HO) enzyme, which produces endogenous vascular protective carbon monoxide (CO). We studied (1) the activity and expression of HO enzymes in the left ventricle (LV) and aorta, (2) basal increase in basal blood pressure provoked by arginine vasopressine (AVP)in vivo, (3) the heart perfusion induced by AVP, (4) the ST segment depression provoked by adrenaline and 30 seconds later phentolamine, and (5) the aorta ring contraction induced by AVP in female and male Wistar rats. We found that HO activity and the expression of HO-1 and HO-2 were

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Lindheimer, Marshall D., and John M. Davison. "Osmoregulation, the secretion of arginine vasopressin and its metabolism during pregnancy." European Journal of Endocrinology 132, no. 2 (1995): 133–43. http://dx.doi.org/10.1530/eje.0.1320133.

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Lindheimer MD, Davison JM. Osmoregulation, the secretion of arginine vasopressin and its metabolism during pregnancy. Eur J Endocrinol 1995;132:133–43. ISSN 0804–4643 This review stresses changes in osmoregulation as well as the secretion and metabolism of aŕginine vasopressin during pregnancy, focusing on human gestation. Pregnant women experience a decrease in body tonicity, plasma osmolality decreasing immediately after conception to a nadir ~ 10 mosmol/kg below non-pregnant levels early in pregnancy, after which a new steady state is maintained until term. Data from both human and rodent g

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Van der Bent, V., D. J. Church, M. B. Vallotton, et al. "[Ca2+]i and protein kinase C in vasopressin-induced prostacyclin and ANP release in rat cardiomyocytes." American Journal of Physiology-Heart and Circulatory Physiology 266, no. 2 (1994): H597—H605. http://dx.doi.org/10.1152/ajpheart.1994.266.2.h597.

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Exposure of cultured, spontaneously beating rat cardiomyocytes to arginine vasopressin (AVP) led to marked increases in the release of prostacyclin (PGI2) and atrial natriuretic peptide (ANP). These responses were accompanied by a rapid, transient rise of cytosolic free Ca2+ concentration ([Ca2+]i) and of membranous protein kinase C (PKC) activity. Ca2+ influx and PKC activity appeared to play important but distinct roles in AVP-induced cellular responses, insofar as only AVP-induced ANP secretion was abolished by the Ca2+ channel antagonist nifedipine, whereas both AVP-induced PGI2 production

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Bankowski, Krzysztof, Bernard Lammek, Marian Kruszynski, Maurice Manning, Janny Seto, and Wilbur H. Sawyer. "2-O-methyl and 2-O-ethyl-tyrosine derivatives of [8-arginine]vasopressin analogs: Highly specific antidiuretic agonists." Collection of Czechoslovak Chemical Communications 53, no. 11 (1988): 2617–26. http://dx.doi.org/10.1135/cccc19882617.

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The solid phase synthesis of seven 2-O-methyl- and 2-O-ethyl-tyrosine substituted analogs of [8-arginine]vasopressin (AVP) with enhanced antidiuretic agonistic specificity is reported. These peptides are: [2-O-ethyltyrosine, 8-arginine]vasopressin (I), [2-O-methyltyrosine, 8-D-arginine]vasopressin (II), [2-O-ethyltyrosine, 8-D-arginine]vasopressin (III), [2-O-methyltyrosine, 4-valine, 8-arginine]vasopressin (IV), [2-O-ethyltyrosine, 4-valine, 8-arginine]vasopressin (V), [2-O-methyltyrosine, 4-valine, 8-D-arginine]vasopressin (VI), [2-O-ethyltyrosine, 4-valine, 8-D-arginine]vasopressin (VII). A

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Smith, C. P., and R. J. Balment. "Arginine vasopressin-induced natriuresis in the anaesthetized rat: involvement of V1 and V2 receptors." Journal of Endocrinology 136, no. 2 (1993): 283–88. http://dx.doi.org/10.1677/joe.0.1360283.

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ABSTRACT The present study was undertaken to determine the involvement of the two established vasopressin receptor subtypes (V1 and V2) in arginine vasopressin (AVP)-induced natriuresis and also to determine whether changes in mean arterial pressure (MAP) and/or the renally active hormones atrial natriuretic peptide (ANP), angiotensin II (AII) and aldosterone are a prerequisite for the expression of AVP-induced natriuresis. In Sprague–Dawley rats which were anaesthetized with Inactin (5-ethyl-5-(1′-methylpropyl)-2-thiobarbiturate) and infused with 0·077 mol NaCl/l, infusion of 63 fmol AVP/min

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Jochberger, Stefan, Nils G. Morgenthaler, Viktoria D. Mayr, et al. "Copeptin and Arginine Vasopressin Concentrations in Critically Ill Patients." Journal of Clinical Endocrinology & Metabolism 91, no. 11 (2006): 4381–86. http://dx.doi.org/10.1210/jc.2005-2830.

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Abstract Context: Determination of arginine vasopressin (AVP) concentrations may be helpful to guide therapy in critically ill patients. A new assay analyzing copeptin, a stable peptide derived from the AVP precursor, has been introduced. Objective: Our objective was to determine plasma copeptin concentrations. Design: We conducted a post hoc analysis of plasma samples and data from a prospective study. Setting: The setting was a 12-bed general and surgical intensive care unit (ICU) in a tertiary university teaching hospital. Patients: Our subjects were 70 healthy volunteers and 157 ICU patien

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Pittman, Quentin J., and Marshall F. Wilkinson. "Central arginine vasopressin and endogenous antipyresis." Canadian Journal of Physiology and Pharmacology 70, no. 5 (1992): 786–90. http://dx.doi.org/10.1139/y92-104.

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Arginine vasopressin (AVP) is a centrally synthesized nonapeptide that exerts classical endocrine effects as well as a host of centrally mediated actions. A strong case can be argued in support of a neurotransmitter–neuromodulator role for AVP. Acting within the central nervous system (CNS), AVP has been demonstrated to be involved in the modulation of febrile body temperature. Because AVP acts to reduce pyrogen-induced fevers, but not normal body temperature, its actions are deemed to be antipyretic. However, to demonstrate an endogenous antipyretic function, AVP must be shown to be active du

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Rouse, D., M. Leite, and W. N. Suki. "ATP inhibits the hydrosmotic effect of AVP in rabbit CCT: evidence for a nucleotide P2u receptor." American Journal of Physiology-Renal Physiology 267, no. 2 (1994): F289—F295. http://dx.doi.org/10.1152/ajprenal.1994.267.2.f289.

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In rabbit renal cortical collecting tubule (CCT), perfused in vitro at 38 degrees C, ATP in concentrations of 10(-7) M and greater inhibits arginine vasopressin (AVP)-stimulated osmotic water permeability (Pf). The P1-purinergic receptor antagonist 8-phenyltheophylline did not attenuate the inhibitory action of ATP, and the poorly hydrolyzable ATP analogue, 5'-adenylylimidodiphosphate (AMP-PNP), mimicked the effect of ATP, arguing against an effect of ATP on a P1 receptor or the "P site." Purinergic receptor agonists inhibited AVP-stimulated Pf with the following rank order efficacy: ATP = ADP

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Penney, M. D., M. J. Levell, and R. P. Hullin. "Arginine vasopressin in manic-depressive psychosis." Psychological Medicine 17, no. 4 (1987): 861–67. http://dx.doi.org/10.1017/s0033291700000659.

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SynopsisUrinary excretions of arginine vasopressin (AVP), sodium, potassium, osmoles and creatinine were measured in three in-patients with bipolar manic-depressive psychosis on at least eight 24-hour periods in each affective phase. Mood and body weight were recorded twice daily. The excretion by each patient of sodium, water and osmoles was greater in mania than during depression. Comparison of electrolytes and osmoles suggested that the increase was due to increased intake of salt and water rather than of total diet. There was a fall of mean AVP excretion during mania, the magnitude of the

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Ervin, M. G., M. G. Ross, R. D. Leake, and D. A. Fisher. "Fetal recirculation of amniotic fluid arginine vasopressin." American Journal of Physiology-Endocrinology and Metabolism 250, no. 3 (1986): E253—E258. http://dx.doi.org/10.1152/ajpendo.1986.250.3.e253.

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Amniotic fluid volume reflects a balance between fetal lung fluid and fetal urine production and fluid reabsorption via fetal swallowing. Arginine vasopressin (AVP) infusion decreases both fetal lung fluid and urine production and increases amniotic fluid osmolality and AVP concentration. In the present study we assessed the effect of amniotic fluid AVP injection on plasma AVP (n = 6) and renal function (n = 4) in chronically catheterized fetal lambs (X gestation = 130 days). Thirty minutes after addition of 25 micrograms of synthetic AVP into the amniotic cavity, mean +/- SE fetal plasma AVP

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Dissertations / Theses on the topic "Arginine vasopressine [AVP]"

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Boulkroun, Sheerazed. "Caractérisation de protéines régulées précocement par l'aldostérone et la vasopressine dans le tubule collecteur rénal." Paris 7, 2003. http://www.theses.fr/2003PA077138.

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Deux composés benzéniques poly-substitués par des groupes pyrroliques, le 1,3,5-tripyrrolylbenzène et l'hexapyrrolylbenzène, ont étés utilisés comme réactifs de synthèse de semi-conducteurs organique p-conjugué. Le couplage des groupes pyrroliques du 1,3,5-tripyrrolylbenzène est intermoléculaire et conduit à la formation d'un polymère organique p-conjugué tandis qu'il est pour l'hexapyrrolylbenzène intramoléculaire de type radical-cation substrat et conduit à la formation de molécules discoi͏̈des qui s'assemblent en matériau organique conducteur p-empilé. Notre approche a permis de dégager des

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Paulmyer-Lacroix, Odile. "Double marquage en hybridation in situ des arnm de la corticotropin-releasing hormone (crh) et de l'arginine vasopressine (avp) dans le noyau paraventriculaire du rat : effet du stress et des hormones steroidiennes gonadiques et corticosurrenaliennes (doctorat : interactions cellulaires en endocrinologie)." Aix-Marseille 2, 1998. http://www.theses.fr/1998AIX2661U.

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Kirchner, Vincent. "The elderly, arginine vasopressin & selective serotonin reuptake inhibitors." Master's thesis, University of Cape Town, 1999. http://hdl.handle.net/11427/26271.

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The association between selective serotonin reuptake inhibitors (SSRis) and hyponatraemia has been well documented, the elderly appearing to be at greatest risk. An analysis of data of hyponatraemia in the elderly using SSRis from all published cases and from the Committee on Safety of Medicines found that the mean time to detection was about 3 weeks after commencing SSRis. A wide range of time to detection (1-253 days) and non-specific symptoms suggest hyponatraemia is detected by chance rather than being specifically looked for. This is probably a sporadic, idiosyncratic phenomenon that is n

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Ashton, N. "The role of arginine-vasopressin in the New Zealand genetically hypertensive rat." Thesis, University of Manchester, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.382753.

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Wayte, Judith. "Investigations into the role of nitric oxide in the control of vasopressin release from the rat hypothalamus in vitro." Thesis, Imperial College London, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.294845.

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Hedrich, Christian Michael, Agnieszka Zachurzok-Buczynska, Aneta Gawlik, et al. "Autosomal Dominant Neurohypophyseal Diabetes Insipidus in Two Families." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-134493.

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Background: Autosomal dominant familial neurohypophyseal diabetes insipidus (adFNDI) is a rare disease with symptoms of polydipsia, polyuria and dehydration caused by arginine vasopressin deficiency. Disease onset is within infancy or adolescence. A variety of disease-causing mutations of the arginine vasopressin neurophysin II gene (AVP) on chromosome 20p13 have been described. Methods: Two Polish families with adFNDI were screened for mutations. Processing of wild-type (WT) and mutant AVP was monitored using immunocytochemical methods in stably transfected Neuro2A cells. AVP secretion into t

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Hedrich, Christian Michael, Agnieszka Zachurzok-Buczynska, Aneta Gawlik, et al. "Autosomal Dominant Neurohypophyseal Diabetes Insipidus in Two Families: Molecular Analysis of the Vasopressin-Neurophysin II Gene and Functional Studies of Three Missense Mutations." Karger, 2009. https://tud.qucosa.de/id/qucosa%3A27572.

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Background: Autosomal dominant familial neurohypophyseal diabetes insipidus (adFNDI) is a rare disease with symptoms of polydipsia, polyuria and dehydration caused by arginine vasopressin deficiency. Disease onset is within infancy or adolescence. A variety of disease-causing mutations of the arginine vasopressin neurophysin II gene (AVP) on chromosome 20p13 have been described. Methods: Two Polish families with adFNDI were screened for mutations. Processing of wild-type (WT) and mutant AVP was monitored using immunocytochemical methods in stably transfected Neuro2A cells. AVP secretion into t

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Ho, Shuang-Bao. "Corticotropin-releasing factor-41 (CRF-41), arginine vasopressin (AVP) and oxytocin regulation in hypothalamic cells in culture." Thesis, Brunel University, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.306850.

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Zimmermann, Ulrich, Konstanze Spring, Hans-Ulrich Wittchen, et al. "Arginine vasopressin and adrenocorticotropin secretion in response to psychosocial stress is attenuated by ethanol in sons of alcohol-dependent fathers." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2013. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-110031.

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Familial risk and environmental stress promote the development of alcohol dependence. We investigated whether a positive family history of alcoholism affects the neuroendocrine response to a standardized laboratory stress test in healthy subjects without alcohol use disorders. Twenty-four high-risk subjects with a paternal history of alcoholism (PHA) and 16 family history negative (FHN) controls were evaluated. Psychosocial stress was induced by having subjects deliver a 5-min speech and mental arithmetics in front of an audience on separate days, after drinking either placebo or ethanol (0.6

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Zimmermann, Ulrich, Konstanze Spring, Hans-Ulrich Wittchen, et al. "Arginine vasopressin and adrenocorticotropin secretion in response to psychosocial stress is attenuated by ethanol in sons of alcohol-dependent fathers." Technische Universität Dresden, 2004. https://tud.qucosa.de/id/qucosa%3A26808.

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Familial risk and environmental stress promote the development of alcohol dependence. We investigated whether a positive family history of alcoholism affects the neuroendocrine response to a standardized laboratory stress test in healthy subjects without alcohol use disorders. Twenty-four high-risk subjects with a paternal history of alcoholism (PHA) and 16 family history negative (FHN) controls were evaluated. Psychosocial stress was induced by having subjects deliver a 5-min speech and mental arithmetics in front of an audience on separate days, after drinking either placebo or ethanol (0.6

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Books on the topic "Arginine vasopressine [AVP]"

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Hu, Shuang-Bao. Corticothropin-releasing factor-41 (CRF-41), arginne vasopressin (AVP), and oxytocin regulation in hypothalamic cellsin culture. Brunel University, 1992.

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Carter, C. Sue, and Suma Jacob. Oxytocin and Vasopressin: Mechanisms for Potential Sex Differences Observed in Autism Spectrum Disorders. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199744312.003.0018.

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The effects of oxytocin and vasopressin on the brain and behavior can be sexually dimorphic, especially during the course of development (Bales, Kim, et al., 2004; Bales, Pfeifer, et al., 2004; Bales, Plotsky, et al., 2007; Bielsky et al., 2005a; Carter, 2003; Thompson et al., 2006; Yamamoto et al., 2005; Yamamoto et al., 2004). Given the sexual discrepancy observed in autism spectrum disorders (ASDs), these two neuropeptides, oxytocin (OT) and arginine vasopressin (AVP), have received attention for their potential role in ASDs (Green and Hollander, 2010; Insel et al., 1999; Leckman & Herm

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Book chapters on the topic "Arginine vasopressine [AVP]"

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Champion, Howard R., Nova L. Panebianco, Jan J. De Waele, et al. "Arginine Vasopressin (AVP)." In Encyclopedia of Intensive Care Medicine. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-00418-6_1148.

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"Arginin-Vasopressin (AVP)." In Springer Reference Medizin. Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_300203.

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Luck, Martin. "4. Water, salt, and blood pressure." In Hormones: A Very Short Introduction. Oxford University Press, 2014. http://dx.doi.org/10.1093/actrade/9780199672875.003.0004.

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‘Water, salt, and blood pressure’ describes how the balance of water in the body is controlled by several hormones, including vasopressin (arginine vasopressin (AVP)). AVP reduces urine production by the kidneys and also causes small blood vessels to contract, raising blood pressure. Blood volume and pressure are also adjusted by changing the amount of sodium ions reclaimed by kidney nephrons. The renin–Ang-II–aldosterone hormone system balances blood volume and circulatory space to keep pressure stable when the volume and dilution of the blood change. But what happens if the concentration of salt (sodium and other ions) in the blood starts to rise? Is there a direct way to get rid of excess salt? A hormone secreted by the heart, called ANP, does exactly this.

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Samson, Willis K. "The Posterior Pituitary and Water Metabolism." In Textbook of Endocrine Physiology. Oxford University Press, 2011. http://dx.doi.org/10.1093/oso/9780199744121.003.0009.

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The neurohypophysis, also called the posterior pituitary or neural lobe , is the ventral extension of hypothalamic tissue derived from a developmental down growth of the neuroectoderm forming the floor of the third cerebroventricle. It weighs approximately 0.10–0.15 g in humans and is well developed at birth, having been present since the fifth month of intrauterine life. In addition to containing glial elements called pituicytes, the posterior pituitary is composed of unmyelinated nerve fibers and axon terminals of neurons whose cell bodies reside primarily in the supraoptic and paraventricular hypothalamic nuclei. These hypothalamo-neurohypophyseal fibers deliver the two primary posterior pituitary hormones, oxytocin (OT) and arginine vasopressin (AVP), to the neural lobe in association with specific proteins, the neurophysins, once thought to be carrier proteins but now known to be portions of the OT and AVP precursor molecules. The neurons produce either OT or AVP, and under some circumstances both, and recent studies indicate that in addition to one of these two hormones, other neuropeptides, such as corticotropin-releasing hormone (CRH) and nesfatin-1, and neurotransmitters are also produced in OT- or AVP-containing cells. The phenomenon of colocalization of neuromodulatory agents has aroused a great deal of clinical interest in the role of neuropeptides such as OT and AVP in brain function. Both OT- and AVP-containing nerve fibers, originating in the supraoptic and paraventricular nuclei, project to a variety of other brain structures that are thought to be the sites of their observed central nervous system actions, and to the vicinity of the hypophyseal portal vessels in the median eminence. Release from these fibers of both OT and AVP explains the high levels of these hormones in portal blood and provides the framework for the actions of OT and AVP as modulators of anterior pituitary function. The arterial blood supply of the posterior pituitary is via the inferior (and to some degree the superior) hypophyseal arteries, which originate from the cavernous and postclinoid portions of the internal carotid artery.

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Borgers, Anke J., Eric Fliers, Jacqueline E. Siljee-Wong, Dick F. Swaab, Peter H. Bisschop, and Anneke Alkemade. "Arginine Vasopressin Immunoreactivity in the Suprachiasmatic Nucleus Is Decreased in Patients Treated for a Suprasellar Tumor Leading to Visual Field Defects." In BASIC/TRANSLATIONAL - Reproduction & Neuroendocrinology. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part1.p12.p1-277.

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Mott, Natasha N., and Toni R. Pak. "Ligand-Independent Transcriptional Actions of Human ERβ Splice Variants on ERE, AP-1 and Human Arginine Vasopressin (hAVP) Promoters in Neuronal Cells." In BASIC/TRANSLATIONAL - Molecular & Physiological Actions of the Sex Steroid Receptors. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part3.p17.p3-24.

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Conference papers on the topic "Arginine vasopressine [AVP]"

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BRINK, K., F. DERKX, E. BROMMER, J. STIBBE, H. KOLSTEE, and M. SCHALEKAMP. "THE FIBRINOLYTIC, FACTOR VIII:C, VON WILLEBRAND FACTOR AND HEMODYNAMIC RESPONSES TO DDAVP IN PATIENTS WITH HEREDITARY NEPHROGENIC DIABETES INSIPIDUS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644709.

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The pressor response of vasopressin (AVP) is mediated by a calcium-dependent mechanism (VI-receptor), whereas its antidiuretic effect depends on c-ANP (V2-receptor). DDAVP (1-desamino-8-D-arginine vasopressin) is a synthetic V2 analog of AVP. AVP and DDAVP also increase FVIII:C vWF:Ag and tissue-type plasminogen activator (t-PA) in plasma. The mechanism by which AVP and DDAVP elevate these factors is unclear. Patients with X-linked nephrogenic diabetes insipidus (NDI) are resistant to the V2-mediated antidiuretic action of AVP and DDAVP. We therefore have studied the effect of DDAVP (0.4 ug/kg

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