Academic literature on the topic 'ARN activateur'
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Journal articles on the topic "ARN activateur":
Antoniewski, C. "Le co-activateur du récepteur nucléaire était... un ARN !" médecine/sciences 15, no. 10 (1999): 1153. http://dx.doi.org/10.4267/10608/1230.
Martella, Christophe, Laetitia Waast, and Claudine Pique. "Tax, marionnettiste de la transcription du HTLV-1." médecine/sciences 38, no. 4 (April 2022): 359–65. http://dx.doi.org/10.1051/medsci/2022039.
Blake, Timo, Anne Barnard, Stephen J. W. Busby, and Jeffrey Green. "Transcription Activation by FNR: Evidence for a Functional Activating Region 2." Journal of Bacteriology 184, no. 21 (November 1, 2002): 5855–61. http://dx.doi.org/10.1128/jb.184.21.5855-5861.2002.
Liu, Xinhuai, and Allan Herbison. "Kisspeptin Regulation of Arcuate Neuron Excitability in Kisspeptin Receptor Knockout Mice." Endocrinology 156, no. 5 (March 10, 2015): 1815–27. http://dx.doi.org/10.1210/en.2014-1845.
Xu, Bo, Hong Zheng, Xuefei Liu, and Kaushik P. Patel. "Activation of afferent renal nerves modulates RVLM-projecting PVN neurons." American Journal of Physiology-Heart and Circulatory Physiology 308, no. 9 (May 1, 2015): H1103—H1111. http://dx.doi.org/10.1152/ajpheart.00862.2014.
de Croft, Simon, Ulrich Boehm, and Allan E. Herbison. "Neurokinin B Activates Arcuate Kisspeptin Neurons Through Multiple Tachykinin Receptors in the Male Mouse." Endocrinology 154, no. 8 (August 1, 2013): 2750–60. http://dx.doi.org/10.1210/en.2013-1231.
Derrien, Thomas, and Roderic Guigó. "De longs ARN non codants activateurs de la transcription des gènes." médecine/sciences 27, no. 4 (April 2011): 359–61. http://dx.doi.org/10.1051/medsci/2011274009.
Qi, Guan-Ming, Li-Xin Jia, Yu-Lin Li, Hui-Hua Li, and Jie Du. "Adiponectin Suppresses Angiotensin II-Induced Inflammation and Cardiac Fibrosis through Activation of Macrophage Autophagy." Endocrinology 155, no. 6 (June 1, 2014): 2254–65. http://dx.doi.org/10.1210/en.2013-2011.
Caverson, M. M., and J. Ciriello. "Contribution of paraventricular nucleus to afferent renal nerve pressor response." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 254, no. 3 (March 1, 1988): R531—R543. http://dx.doi.org/10.1152/ajpregu.1988.254.3.r531.
Li, Yuan, and Zong Jie Cui. "Photodynamic Activation of Cholecystokinin 1 Receptor with Different Genetically Encoded Protein Photosensitizers and from Varied Subcellular Sites." Biomolecules 10, no. 10 (October 8, 2020): 1423. http://dx.doi.org/10.3390/biom10101423.
Dissertations / Theses on the topic "ARN activateur":
Sharov, Grigory. "Etude structurale du co-activateur transcriptionnel SAGA et de son module d'acétylation des histones." Thesis, Strasbourg, 2015. http://www.theses.fr/2015STRAJ040/document.
Transcription initiation in eukaryotes requires the recruitment of RNA polymerase II (Pol II) and general transcription factors to the promoters of protein coding genes in order to form a PreInitiation Complex (PIC). Sequence specific activators bind up stream of the promoter, stimulating chromatin opening and PIC formation via recruitment of coactivator complexes. SAGA is such a coactivator, conserved in all eukaryotes, known to modify the histones on all expressed genes in yeast and human and involved in Pol II transcription. In this work I have analyzed SAGA’s molecular organization mostly by electron microscopy. I have (i) studied the architecture and sub unit interactions of SAGA histone acetylation (HAT) module and localized it in the full SAGA complex; (ii) obtained the first cryo-EM map of yeast SAGA and analyzed its flexibility; (iii) defined the interaction site of SAGA with TBP protein and shown that the complex under goes a large conformational change upon TBP binding
Yang, Qi. "Regulation of the yifK locus by multi-target small RNA GcvB in Salmonella." Phd thesis, Université Paris Sud - Paris XI, 2013. http://tel.archives-ouvertes.fr/tel-00954416.
Hache, Antoine. "Molecular basis of transcriptional dysregulations in the spinocerebellar ataxia type 7, a neurodegenerative polyglutamine disorder." Thesis, Strasbourg, 2020. http://www.theses.fr/2020STRAJ083.
SCA7 is a genetic disorder whose one of its main symptoms is a progressive loss of visual acuity which can ultimately lead to blindness. The mutation responsible for this disease is an unstable CAG expansion within ATXN7, a gene encoding a subunit of the SAGA complex, a co-activator of the RNA polymerase II. Previous studies performed on transgenic mouse models highlighted a neuronal identity loss of the photoreceptors at the morphological, functional and molecular levels. During my PhD a characterization of a new SCA7 knock-in mouse model was performed. This model, which expresses the mutated genes at endogenous level recapitulates the retinal impairments observed in transgenic models and in patients. A transcriptomic (RNA-seq) and epigenomic (ChIP-seq) analyses were performed on this model and highlight global acetylation defects on lysine 9 and 27 of histone H3 (H3K9ac and H3K27ac). Moreover, investigations on non-coding RNAs identified the presence of enhancer RNAs (eRNAs) on photoreceptor specific genes such as Rho. These eRNAs, which were never described before, undergo a downregulation in symptomatic SCA7 mice
Laustriat, Delphine. "Les cellules souches pluripotentes en modélisation pathologique pour l'identification de nouvelles cibles thérapeutiques : application à la Dystrophie Myotonique de type 1." Thesis, Evry-Val d'Essonne, 2010. http://www.theses.fr/2010EVRY0017.
Myotonic Dystrophy type 1 (DM1), or Steinert disease, is one of the most common neuromuscular disorders in adults for whom no therapeutic solution other than symptomatic is available at present. The mutation, which consists in an unstable CTG expansion located in the 3' transcribed but untranslated region of the DMPK gene, leads in particular to the deregulation of several RNA-binding proteins. This engenders various splicing defects that are associated to the multisystemic symptoms. The availability of a preimplantation genetic diagnosis enabled the derivation of human embryonic stem cell lines (hESC) from embryos carrying the causative mutation. These cell lines, and now the induced pluripotent stem cells established from patients, constitute promising models for genetic disease’s exploration. Besides the possibility to investigate the pathology in a natural context, these cells open the way to every cell phenotypes without any quantitative restriction and thus appear as a valuable tool for large scale screening, and particularly for functional genomics approaches that systematically explore the involvement of genes in a given phenotype. The aim of my work was to explore the potential of such an approach by developing a loss of function RNA interference screen in order to identify new therapeutic targets by using a hESC line expressing the DM1 mutation. To that purpose we first identified a disease relevant cell population, i.e. hESC derived-mesenchymal progenitors expressing two DM1 biomarkers – foci and INSR splicing defects, suitable for large scale siRNA transfection. Then we developed, miniaturized and automated the transfection’s and phenotype detection’s steps. Finally, once all the conditions determined, we conducted a screening of a siRNA library targeting RNA-binding proteins that led to the identification of several candidate genes, including ELAVL1 which appears as a new DM1 therapeutic target. Indeed, its knockdown resulted in the improvement of several pathological splicing defects and normalized the glucose uptake impairment. We also showed that the enrichment of its nuclear fraction through the use of AMPK activators, some of which being widely prescribed anti-diabetic drug, was able to mimic this corrective effect. My work thus underlies the interest of coupling RNAi screening to pathological pluripotent stem cells for the identification of new therapeutic targets with the view to accelerate drug discovery, and particularly in the case of rare diseases
Saguy, Matthieu. "Activateurs précoces du processus de trans-traduction : rôle de la protéine ribosomique S1 et recherche d’une activité ribonucléase liée au ribosome." Rennes 1, 2008. http://www.theses.fr/2008REN1S001.
When bacterial translation stalls on a truncated messenger RNA (mRNA), ribosomes are released by process called trans-translation. This system requires a particular ribonucleic acid: transfer-messenger RNA (tmRNA), acting in concert with several protein partners including ribosomal protein S1. In vitro studies showed that S1 is essential to the resume of translation on the internal open reading frame of tmRNA. Accordingly, any variation of the expression level of S1 in vivo is harmful to the process. Trans-translation occurs sometimes on non truncated mRNAs, which are subsequently cleaved into the ribosomal A site to trigger the process. The factor responsible of the cleavage is still unknown. In order to find out the way these mRNA are cleaved, an in vivo system allowing purification of stalled ribosomes on non truncated mRNAs was set up. No endoribonuclease could be identified, nevertheless an exonucleotytic activity was observed. A new model of trans-translation intervention was established
Kieffer, Kyong-Rim. "Stimulation de la réparation de l'ADN par des activateurs de la transcription." Université Louis Pasteur (Strasbourg) (1971-2008), 2002. http://www.theses.fr/2002STR13085.
Eukaryotic genes are contained within a higher order complex of DNA and histones called chromatin. Although packaging of DNA into chromatin provides the means for compaction of the entire genome to fit in the nucleus, it restricts the access of the many regulatory proteins required for essential biological processes such as DNA replication, transcription, and recombination. The chromatin, however, is not a static structure, but rather a dynamic assembly that condenses and decondenses (remodeling) in response to specific signals during cell life. Chromatin remodeling requires a specific set of enzymes that modify the nucleosome, the building block of chromatin. These enzymes fall into two classes: the first includes ATP-dependent chromatin remodeling activities that use energy derived from ATP hydrolysis to alter nucleosomal structure and/or arrangement, whereas the second class includes enzymes that add acetyl groups to the histone N termini. This thesis has described that DNA repair is also hindered by chromatin structure and requires a subset of chromatin remodeling enzymes from each class to optimally occur. In the promoter region, chromatin remodeling enzymes are dictated by sequence-specific activators, resulting in facilitated damage removal in the proximity of transcription initiation site. The mechanism of this preferential repair is independent of transcription machinery and transcription per se, although two events pass on the same template. Furthermore, transcriptionally inactive activator accomplishes the stimulation of repair by binding to its cognate sequences. It is likely that the function of activators is dual : (i) they help to derepress chromatin, a step common to DNA processes, (ii) in parallel or subsequently, and possibly in a cooperative manner according to activities demanded by the surrounding DNA, they recruit specific factors involved in transcription, DNA repair or replication
Diouf, Sarah. "Le co-activateur transcriptionnel CREB-binding protein (CBP) : un nouvel acteur de la fonction mitochondriale." Thesis, Toulouse 3, 2018. http://www.theses.fr/2018TOU30050.
CBP (CREB Binding Protein also called KAT3A) is a nuclear co-activator with an acetyltransferase activity. It interacts with a large number of transcription factors and thus regulates the transcription of many genes thereby being involved in a wide range of processes (proliferation, differentiation, apoptosis ...). CBP expression is also deregulated in several pathologies such as Rubinstein-Taybi Syndrome or cancers. For instance, CBP has been shown to regulate myogenesis, i.e. the formation of muscles during embryogenesis and during regeneration in adults. In this work, we studied the role of CBP in this process using human primary myoblasts as a model. The analysis of CBP expression and sub-cellular localization in these cells showed that a fraction of the protein is found inside mitochondria. This finding suggests for the first time a role of this well-known nuclear co-activator outside of the nucleus. Hence, my Ph.D. project focused on deciphering the functions of CBP in this organelle. Mitochondria have their own genome and one of their main functions is to produce the energy required for the cell functions. We have shown that the cellular metabolic state controls the import of CBP into mitochondria. We also found that CBP and its acetyltransferase activity regulate the mitochondrial DNA replication and transcription. Furthermore, our results indicate that CBP is involved in the regulation of mitochondrial oxidative phosphorylation and therefore in the energy production. Finally, we show that CBP is also in mitochondria in other cell types and in other species, strongly suggesting that CBP functions in mitochondria are important and conserved
Jomrich, Gerd, Florian Maroske, Jasmin Stieger, Matthias Preusser, Aysegül Ilhan-Mutlu, Daniel Winkler, Ivan Kristo, Matthias Paireder, and Sebastian Friedrich Schoppmann. "MK2 and ETV1 Are Prognostic Factors in Esophageal Adenocarcinomas." IVyspring International Publisher, 2018. http://dx.doi.org/10.7150/jca.22310.
Mikaelian, Ivan. "Identification des domaines fonctionnels de la protéine EB1, activateur des gènes précoces du virus d'Epstein-Barr." Lyon 1, 1992. http://www.theses.fr/1992LYO1T178.
Gruffat, Henri. "Caractérisation fonctionnelle des éléments géniques transmettant l'effet activateur du facteur de transcription R codé par le virus d'Epstein-Barr." Lyon 1, 1991. http://www.theses.fr/1991LYO1T128.
Books on the topic "ARN activateur":
Martorell, Francisco. How do earnings change when reservists are activated?: A reconciliation of estimates derived from survey and administrative data. Santa Monica, CA: RAND Corp., 2008.
Trambakoulos, Dimitra Mimi. Cardiovascular effects of activated coagulation FXII, "new pressor protein" (NPP), are associated with potent adrenal medullary catecholamine release. Ottawa: National Library of Canada, 1999.
Bashin, Yuriy, Gennadiy Grinev, and Yuliya Dremova. Economics of the information society. ru: INFRA-M Academic Publishing LLC., 2020. http://dx.doi.org/10.12737/1039916.
Beninger, Richard J. Dopamine as the dependent variable. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.003.0005.
Sullivan, Mark D. Advancing from Activated Patient to Autonomous Patient. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780195386585.003.0008.
Beninger, Richard J. Dopamine and the elements of incentive learning. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.003.0003.
De Souza, Jonathan. Sounding Actions. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780190271114.003.0003.
Kleihues, Paul, Elisabeth Rushing, and Hiroko Ohgaki. The 2016 revision of the WHO classification of tumours of the central nervous system. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0001.
Grom, Alexei A., and Athimalaipet V. Ramanan. Macrophage activation syndrome. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0168.
Beninger, Richard J. Life's rewards. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.001.0001.
Book chapters on the topic "ARN activateur":
Klapars, Artis. "Glucokinase Activator." In The Art of Process Chemistry, 223–39. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2010. http://dx.doi.org/10.1002/9783527633562.ch8.
Duesberg, P. H., M. Nunn, Nancy Kan, D. Watson, P. H. Seeburg, and T. Papas. "Are Activated Proto-onc Genes Cancer Genes?" In Modern Trends in Human Leukemia VI New Results in Clinical and Biological Research Including Pediatric Oncology, 9–27. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70385-0_4.
Duesberg, Peter H., Michael Nunn, Nancy Kan, Dennis Watson, Peter H. Seeburg, and Takis Papas. "Are Activated Proto-ONC Genes Cancer Genes?" In Cell Transformation, 21–63. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-5009-5_2.
Waller, Michael, and Ulrich Kohnert. "Reteplase, a recombinant plasminogen activator." In Biopharmaceuticals, an Industrial Perspective, 185–216. Dordrecht: Springer Netherlands, 1999. http://dx.doi.org/10.1007/978-94-017-0926-2_8.
Dirican, Enver Kerem. "Magnetic-Activated Cell Sorting of Human Spermatozoa." In Gamete Assessment, Selection and Micromanipulation in ART, 131–44. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-8360-1_9.
Duesberg, Peter H., Michael Nunn, Nancy Kan, Dennis Watson, Peter H. Seeburg, and Takis Papas. "Which Cancers are Caused by Activated Proto-Onc Genes?" In RNA Tumor Viruses, Oncogenes, Human Cancer and AIDS: On the Frontiers of Understanding, 168–90. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2583-3_14.
Newton, Peter W., Peter W. G. Newman, Stephen Glackin, and Giles Thomson. "The Global Greyfields Transition: Why Urban Redevelopment in Low-Density, Car-Based Middle Suburbs Needs a New Model." In Greening the Greyfields, 1–48. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-6238-6_1.
Cooper, Jonathan A., Nathaniel S. Allen, and Libing Feng. "Protein Kinases and Signaling Pathways that Are Activated by Reelin." In Reelin Glycoprotein, 193–216. New York, NY: Springer New York, 2008. http://dx.doi.org/10.1007/978-0-387-76761-1_13.
Ilg, U. J., and P. Thier. "MST Neurons are Activated by Smooth Pursuit of Imaginary Targets." In Parietal Lobe Contributions to Orientation in 3D Space, 173–84. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60661-8_10.
Ilg, U. J., J. A. Rommel, and P. Thier. "Parietal Neurons are Activated by Smooth Pursuit of Imaginary Targets." In Current Oculomotor Research, 37–44. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4757-3054-8_5.
Conference papers on the topic "ARN activateur":
Govers-Riemslag, J. W. P., M. H. J. Knapen, G. Tans, R. F. A. Zwaal, and J. Rosing. "STRUCTURAL AND FUNCTIONAL PROPERTIES OF A PROTHROMBIN ACTIVATOR FROM THE VENOM OF BOTHROPS NEUWIDI." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644321.
Hariman, H., J. R. Hughes, P. J. Grant, and J. A. Davies. "THE EFFECTS OF PHYSIOLOGICAL CONCENTRATIONS OF VASOPRESSIN ON COMPONENTS OF THE FIBRINOLYTIC PATHWAY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643121.
Rose, Jr., C. E., Marie D. Burdick, Brett A. Strieter, Ling Liu, and Robert M. Strieter. "Increased Circulating Activated Fibrocytes Are Associated With Severe Asthma." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a3722.
Taylor-Clark, Tom, Lika Nesuashvili, Stephen Hadley, and Parmvir Bahia. "Airway Sensory Nerves Are Activated By Acute Mitochondrial Modulation." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a2158.
Roberts, NJ, and S. McNarry. "P107 Non completers in pulmonary rehabilitation – are they activated?" In British Thoracic Society Winter Meeting 2018, QEII Centre, Broad Sanctuary, Westminster, London SW1P 3EE, 5 to 7 December 2018, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2018. http://dx.doi.org/10.1136/thorax-2018-212555.265.
Lambers, J. W. J., M. Cammenga, B. Konig, H. Pannekoek, and J. A. van Mourik. "ACTIVATION OF HUMAN ENDOTHELIAL TYPE PLASMINOGEN ACTIVATOR INHIBITOR (PAI-1) BY NEGATIVELY CHARGED PHOSPHOLIPIDS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642807.
Comp, P. C., and C. T. Esmon. "Defects in the protein C pathway." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643715.
Hiraga, F., N. Nakamura, and I. Miki. "Remote Dismantling of Activated Reactor Internals with Plasma Arc Torch." In 6th International Symposium on Automation and Robotics in Construction. International Association for Automation and Robotics in Construction (IAARC), 1989. http://dx.doi.org/10.22260/isarc1989/0037.
Ganguly, Sourik S., Leann S. Fiore, Jonathan T. Sims, J. Woodrow Friend, DivyaMani Srinivasan, Matthew Thacker, Michael L. Cibull, Wang Chi, and Rina Plattner. "Abstract C51: c-Abl and Arg are activated in human primary melanomas, promote melanoma cell invasion, proliferation, survival, and drive metastatic progression." In Abstracts: AACR Special Conference on Tumor Invasion and Metastasis - January 20-23, 2013; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.tim2013-c51.
Matsuyama, H., T. Isshiki, A. Chiba, T. Yamaguchi, G. Murayama, Y. Akasaka, Y. Eishi, S. Miyake, and S. Homma. "Mucosal-Associated Invariant T Cells Are Activated in Lungs of Sarcoidosis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a2427.
Reports on the topic "ARN activateur":
Trowbridge, L. Isotopic selectivity of surface diffusion: An activated diffusion model. Office of Scientific and Technical Information (OSTI), November 1989. http://dx.doi.org/10.2172/5462238.
Prusky, Dov, Nancy P. Keller, and Amir Sherman. global regulation of mycotoxin accumulation during pathogenicity of Penicillium expansum in postharvest fruits. United States Department of Agriculture, January 2014. http://dx.doi.org/10.32747/2014.7600012.bard.
Wall, M. A., T. W. Jr Barbee, and T. P. Weihs. An in situ electron microscopy technique for the study of thermally activated reactions in multilayered materials. Office of Scientific and Technical Information (OSTI), April 1995. http://dx.doi.org/10.2172/132651.
Daniel J. Stepan, Thomas A. Moe, Melanie D. Hetland, and Margaret L. Laumb. POWDERED ACTIVATED CARBON FROM NORTH DAKOTA LIGNITE: AN OPTION FOR DISINFECTION BY-PRODUCT CONTROL IN WATER TREATMENT PLANTS. Office of Scientific and Technical Information (OSTI), June 2001. http://dx.doi.org/10.2172/825585.
Sessa, Guido, and Gregory Martin. MAP kinase cascades activated by SlMAPKKKε and their involvement in tomato resistance to bacterial pathogens. United States Department of Agriculture, January 2012. http://dx.doi.org/10.32747/2012.7699834.bard.
Moran, Nava, Richard Crain, and Wolf-Dieter Reiter. Regulation by Light of Plant Potassium Uptake through K Channels: Biochemical, Physiological and Biophysical Study. United States Department of Agriculture, September 1995. http://dx.doi.org/10.32747/1995.7571356.bard.
Gu, B., and K. E. Dowlen. An investigation of groundwater organics, soil minerals, and activated carbon on the complexation, adsorption, and separation of technetium-99. Office of Scientific and Technical Information (OSTI), January 1996. http://dx.doi.org/10.2172/219315.
Prusky, Dov, Noel T. Keen, and Stanley Freeman. Elicitation of Preformed Antifungal Compounds by Non-Pathogenic Fungus Mutants and their Use for the Prevention of Postharvest Decay in Avocado Fruits. United States Department of Agriculture, January 1996. http://dx.doi.org/10.32747/1996.7570573.bard.
Splitter, Gary, Zeev Trainin, and Yacov Brenner. Lymphocyte Response to Genetically Engineered Bovine Leukemia Virus Proteins in Persistently Lymphocytic Cattle from Israel and the U.S. United States Department of Agriculture, July 1995. http://dx.doi.org/10.32747/1995.7570556.bard.
Rafaeli, Ada, and Russell Jurenka. Molecular Characterization of PBAN G-protein Coupled Receptors in Moth Pest Species: Design of Antagonists. United States Department of Agriculture, December 2012. http://dx.doi.org/10.32747/2012.7593390.bard.