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Books on the topic 'Aromatic amines'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 June 2025

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1

Schröder, Stefan. Selektiv O-acylierte Arylhydroxylamine: Herstellung und Reaktionen mit Aminen als Modelle für Schlüsselreaktionen der Carcinogenese aromatischer Amine. A.S. Intemann und C.C. Intemann, 1989.

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2

Organization, World Health, and International Agency for Research on Cancer, eds. Some aromatic amines, organic dyes, and related exposures. IARC Press, 2010.

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3

Maslennikova, I. S. Regulirovanie aminokompleksnymi soedinenii͡a︡mi vlazhnosti dispersnykh ali͡u︡mosilikatnykh sistem. Izd-vo Leningradskogo universiteta, 1985.

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4

Daszkiewicz, Zdzisław. Aromatyczne nitraminy: Synteza, struktura, właściwości, przegrupowanie. Wydaw. UO, 2004.

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5

Association, Chemical Industries, ed. Safe handling of potentially carcinogenic aromatic amines and nitro compounds. Chemical Industries Association, 1992.

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6

Sin, Han-sŭng. Sikpʻum chung heterocyclic amines monitʻŏring mit wihae pʻyŏngka =: Monitoring and risk analysis for heterocyclic amines in foods. Sikpʻum Ŭiyakpʻum Anjŏnchʻŏng, 2007.

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7

S, Lee Linda, and National Exposure Research Laboratory (U.S.). Ecosystems Research Division., eds. Modeling soil-water distribution of aromatic amines in water saturated soil systems. U.S. Environmental Protection Agency, Ecosystems Research Division, National Exposure Research Laboratory, 2000.

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8

S, Lee Linda, and National Exposure Research Laboratory (U.S.). Ecosystems Research Division, eds. Modeling soil-water distribution of aromatic amines in water saturated soil systems. U.S. Environmental Protection Agency, Ecosystems Research Division, National Exposure Research Laboratory, 2000.

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9

S, Lee Linda, and National Exposure Research Laboratory (U.S.). Ecosystems Research Division, eds. Modeling soil-water distribution of aromatic amines in water saturated soil systems. U.S. Environmental Protection Agency, Ecosystems Research Division, National Exposure Research Laboratory, 2000.

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10

S, Lee Linda, and National Exposure Research Laboratory (U.S.). Ecosystems Research Division., eds. Modeling soil-water distribution of aromatic amines in water saturated soil systems. U.S. Environmental Protection Agency, Ecosystems Research Division, National Exposure Research Laboratory, 2000.

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11

Humans, IARC Working Group on the Evaluation of Carcinogenic Risks to. Some naturally occurring substances: Food items and constituents, heterocyclic aromatic amines and mycotoxins. Distributed for the International Agency for Research on Cancer by the Secretariat of the World Health Organization, 1993.

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12

National Institute for Occupational Safety and Health, ed. Morton International Chemical Company, Patterson [i.e. Paterson], New Jersey. U.S. Dept. of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, 1994.

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13

Hestermann, Eli V. Mechanisms of action for aryl hydrocarbon receptor ligands in the PLHC-1 cell line. Massachusetts Institute of Technology, 2000.

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14

Minako, Nago, and Sugimura Takashi, eds. Food borne carcinogens: Heterocyclic amines. Wiley, 2000.

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15

M, Castegnaro, and International Agency for Research on Cancer., eds. Laboratory decontamination and destruction of carcinogens in laboratory wastes: Some aromatic amines and 4-nitrobiphenyl. International Agency for Research on Cancer, 1985.

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16

Dolara, Piero, ed. TOX: lezioni di tossicologia. Firenze University Press, 2006. http://dx.doi.org/10.36253/88-8453-412-7.

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TOX is a manual aimed primarily at the Toxicology courses of the Faculty of Pharmacy and Science, that can also be profitably utilised by students following different degree courses in other Faculties (Medicine, Agriculture, Engineering)and by anyone else looking for a thorough but succinct overview of toxicological questions. TOX covers the principal sectors of general toxicology (acute and chronic toxicity, mutagenesis, teratogenesis, carcinogenesis, reproductive toxicity, oxidative damage, epidemiological methods), specialist toxicology(dietary toxicity, tobacco smoke, pesticides, N-nitroso compounds, heterocyclic amines and aromatic amines toxicity) and environmental toxicology (environmental estrogens, PAH, heavy metals, dioxins and polychlorinated di-benzo-furans, water and air pollution).

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17

International Conference on Carcinogenic and Mutagenic N-Substituted Aryl Compounds (3rd 1987 Dearborn, Mich.). Carcinogenic and mutagenic responses to aromatic amines and nitroarenes: Proceedings of the Third International Conference on Carcinogenic and Mutagenic N-Substituted Aryl Compounds, held April 25-28, 1987, in Dearborn Michigan. Edited by King Charles M. 1932-, Romano Louis James 1950-, and Schuetzle Dennis 1942-. Elsevier, 1988.

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18

IARC Working Group on the Evaluation of Carcinogenic Risks to Humans (1992 Lyon, France). Occupational exposures of hairdressers and barbers and personal use of hair colourants: Some hair dyes, cosmetic colourants, industrial dyestuffs and aromatic amines. IARC, 1993.

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19

Goddijn, Oscar Johannes Maria. Regulation of terpenoid indole alkaloid biosynthesis in Catharanthus roseus: The tryptophan decarboxylase gene. Offsetdrukkerij Haveka BV, 1992.

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20

1949-, Sayler Gary S., and Blackburn James W. 1950-, eds. Microbiological decomposition of chlorinated aromatic compounds. M. Dekker, 1987.

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21

Lednicer, Daniel. The organic chemistry of drug synthesis. Wiley, 1995.

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22

Lednicer, Daniel. The organic chemistry of drug synthesis. Wiley, 1990.

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23

Marczylo, Timothy Hywel. Bioactivation of aromatic amines. 1996.

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24

Metabolism of Aromatic Amino Acids and Amines. Elsevier, 1987. http://dx.doi.org/10.1016/s0076-6879(00)x0055-9.

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25

Kaufman, Seymour, Nathan P. Colowick, and Nathan P. Kaplan. Metabolism of Aromatic Amino Acids and Amines. Elsevier Science & Technology Books, 1987.

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26

Bolden, Michael E. Investigation of the tris(2,2'-bipyridine)ruthenium(III) chemiluminescence reaction for the determination of aromatic amines and aldehydes by flow injection or liquid chromatography. 2001.

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27

Toth, Krisztina. Reactivity and selectivity of the nitrenium ions derived from food carcinogens Glu-P-1 and Glu-P-2. 2001.

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28

Nguyen, Thach-Mien Duong. Decomposition kinetics and azide trapping of ester derivative of A C, N-acetyl-N-acetoxy-2-amino- -carboline. 2002.

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29

Nguyen, Thach-Mien Duong. Decomposition kinetics and azide trapping of ester derivative of A C, N-acetyl-N-acetoxy-2-amino- -carboline. 2002.

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30

Metabolism of Aromatic Amino Acids and Amines, Volume 142 (Methods in Enzymology). Academic Press, 1987.

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31

(Editor), Nathan P. Colowick, Nathan P. Kaplan (Editor), and Seymour Kaufman (Editor), eds. Metabolism of Aromatic Amino Acids and Amines, Volume 142 (Methods in Enzymology). Academic Press, 1987.

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32

Jayasinghe, Dudley Shelton. The distribution of organic bases in reverse phase liquid chromatography: A study of mechanisms. 1989.

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33

(Editor), C. M. King, L. J. Romano (Editor), and D. Schuetzle (Editor), eds. Carcinogenic and Mutagenic Responses to Aromatic Amines and Nitroarenes. Elsevier, 1987.

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34

Modeling soil-water distribution of aromatic amines in water saturated soil systems. U.S. Environmental Protection Agency, Ecosystems Research Division, National Exposure Research Laboratory, 2000.

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35

Modeling soil-water distribution of aromatic amines in water saturated soil systems. U.S. Environmental Protection Agency, Ecosystems Research Division, National Exposure Research Laboratory, 2000.

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36

Takashi, Sugimura, and Minako Nagao. Food Borne Carcinogens: Heterocyclic Amines. Wiley, 2000.

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37

Dunton, Leila Elisabeth. On Colorimetric Methods for the Determination of Chlorine in Water, with Special Reference to Aromatic Amines. Creative Media Partners, LLC, 2018.

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38

On Colorimetric Methods for the Determination of Chlorine in Water, with Special Reference to Aromatic Amines. Creative Media Partners, LLC, 2023.

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39

Aromatic amines in air and on surfaces: Laboratory method using pumped acid-coated filter, desorption and liquid chromatography. HMSO, 1993.

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40

Request for assistance in preventing bladder cancer from exposure to o-toluidine and aniline (NIOSH alert). For sale by the Supt. of Docs., U.S. G.P.O, 1990.

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41

Castegnaro, M. Laboratory Decontamination and Destruction of Carcinogens in Laboratory Wastes: Some Aromatic Amines and 4-Nitrobiphenyl (I a R C Scientific Publication). Oxford University Press, USA, 1985.

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42

(Contributor), WHO, ed. Some Naturally Occurring Substances: Food Items and Constituents, Heterocyclic Aromatic Amines and Mycotoxins (IARC Monographs on the Evaluation of Carcinogenic Risks to H). World Health Organisation, 1993.

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43

Kogevinas, Manolis, Jonine Figueroa, Montserrat Garcia-Closas, and Lorelei Mucci. Urinary Bladder Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0022.

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Bladder cancer is the ninth most common cancer worldwide, resulting in 430,000 new cases in 2012, and its incidence is substantially higher in men than women. Urothelial cell carcinoma, also known as transitional cell carcinoma, is the predominant histopathologic type. Bladder cancer occupies an important place in occupational epidemiology, in which associations with occupations exposed to aromatic amines were first identified in the 1950s. It is also among the first cancers for which an infectious etiology was identified, through parasitic infection with Schistosoma haematobium, which occurs in Africa and the eastern Mediterranean. Smokers have a two- to threefold increased risk of bladder cancer, and a fivefold higher risk for heavy smokers. Specific medical conditions, including urinary stones and diabetes, are positively associated with risk. Finally, bladder cancer is one of the few examples with consistent evidence of interactions between environmental exposures and genetic polymorphisms in cancer epidemiology.

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44

Aromatic Amines in Air and on Surfaces: Laboratory Method Using Pumped Acid-coated Filter Desorption and Liquid Chromatography (MDHS) (Methods for the Determination of Hazardous Substances). Health and Safety Executive (HSE), 1993.

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45

Columb, Malachy O. Local anaesthetic agents. Edited by Michel M. R. F. Struys. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0017.

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Local anaesthetic agents cause a pharmacologically induced reversible neuropathy characterized by axonal conduction blockade. They act by blocking the sodium ionophore and exhibit membrane stabilizing activity by inhibiting initiation and propagation of action potentials. They are weak bases consisting of three components: a lipophilic aromatic ring, a link, and a hydrophilic amine. The chemical link classifies them as esters or amides. Local anaesthetics diffuse through the axolemma as unionized free-base and block the ionophore in the quaternary ammonium ionized form. The speed of onset of block is therefore dependent on the pKa of the agent and the ambient tissue pH. Esters undergo hydrolysis by plasma esterases and amides are metabolized by hepatic microsomal mixed-function oxidases. Local anaesthetics are bound in the blood to α‎1-acid glycoproteins. Pharmacological potency is dependent on the lipid solubility of the drug as is the potential for systemic toxicity. The blood concentrations required to cause cardiovascular system (CVS) collapse and early central nervous system (CNS) toxicity are used to quantify the CVS:CNS toxicity ratio. Local anaesthetics also have the potential to induce direct neuronal damage. Intravenous lipid emulsion is used for the treatment of systemic toxicity but the scientific evidence is inconsistent. With regard to the pipecoloxylidine local anaesthetics, early evidence indicated that the S- was less toxic than the R-enantiomer. However, clinical research using minimum local analgesic concentration designs suggests that reduced systemic toxicity and motor block sparing is mainly explained by potency rather than enantiomerism.

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46

Baek, Jae-Kyeong. Behavioral studies of dopa-decarboxylase mutant Drosophila lacking serotonin and dopamine in central nervous system. 1987.

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47

Berry, Alan. The physiology and regulation of aromatic amino acid biosynthesis in Pseudomonas aeruginosa. 1985.

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48

Pearl, Phillip L., and William P. Welch. Pediatric Neurotransmitter Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0059.

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The pediatric neurotransmitter disorders represent an enlarging group of neurological syndromes characterized by inherited abnormalities of neurotransmitter synthesis, metabolism, and transport. Disorders involving monoamine synthesis include guanosine triphosphate cyclohydrolase deficiency (Segawa disease or classical Dopa-responsive dystonia as the heterozygous form), aromatic amino acid decarboxylase deficiency, tyrosine hydrolase deficiency, sepiapterin reductase deficiency, and disorders of tetrahydrobiopterin synthesis. These disorders can be classified according to whether they feature elevated serum levels of phenylalanine. Disorders of γ-amino butyric acid (GABA) metabolism include succinic semialdehyde dehydrogenase deficiency and GABA-transaminase deficiency. Glycine encephalopathy is typically manifested by refractory neonatal seizures due to a defect in the glycine degradative pathway. Pyridoxine-responsive seizures have now been associated with deficiency of α-aminoadipic semialdehyde dehydrogenase as well as a variants requiring therapy with pyridoxal-5-phosphate and folinic acid.

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49

D'Amato, Thomas Andrew. Gene-enzyme relationships in Nicotina silvestris: Subcellular localization of genes and enzymes for aromatic amino acid biosynthesis. 1986.

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50

Heales, Simon, Simon Pope, Viruna Neergheen, and Manju Kurian. Abnormalities of CSF Neurotransmitters/Folates. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0082.

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The term Neurotansmitter disorder, in the area of metabolic disease, focuses particularly on inborn errors affecting monoamine (dopamine & serotonin), pyridoxal phosphate (B6) and folate metabolism. Whilst there has been considerable focus on these disorders with regards to the paediatric population, it is clear that an increasing number of adult patients are being identified. Adult neurologists need to be aware of the clinical presentation of such patients and the appropriate tests that need to be requested to ensure a correct diagnosis is achieved. CSF profiling, by a specialist laboratory, is often required. This has the ability to very often identify the nature of a primary defect with regards to implementation of appropriate treatment. For some of these disorders, treatment can be effective. This may be in the form of monoamine/vitamin replacement. However there are exceptions, e.g. aromatic amino acid decarboxylase and dopamine transporter deficiencies. There also needs also to be an awareness of the growing list of secondary factors that can cause impaired dopamine and serotonin metabolism.

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