Relevant bibliographies by topics / Arrhythmie

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Arrhythmie'

Author: Grafiati
Published: 4 June 2021
Last updated: 12 February 2022

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Journal articles on the topic "Arrhythmie"

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Brunckhorst, C. B., J. Holzmeister, C. Scharf, C. Binggeli, and F. Duru. "Stress, Depression und kardiale Arrhythmien." Therapeutische Umschau 60, no. 11 (November 1, 2003): 673–81. http://dx.doi.org/10.1024/0040-5930.60.11.673.

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Stress und Depressionen verändern die elektrophysiologischen Eigenschaften des Myokards insbesondere durch den Einfluss des autonomen Nervensystems und können somit Herzrhythmusstörungen auslösen. Durch die asymmetrische autonome Innervation des Herzens wirkt sich die Interaktion von Sympathikus und Parasympathikus auf die verschiedenen Lokalisationen des Reizleitungssystems unterschiedlich aus. Bei einer Arrhythmie handelt es sich um ein komplexes Zusammenspiel aus einem elektrophysiologischen Substrat und einem auslösenden Trigger, der auf einer autonomen Stimulation oder anderen Einflussfaktoren beruht, was einen spezifischen Arrhythmie-Mechanismus auslösen kann. Die Korrelation zwischen dem Auftreten von Arrhythmien und einer gestörten autonomen Balance im Sinne eines reduzierten parasympathischen und gesteigerten sympathischen Tonus ist in der Literatur gut dokumentiert. Die Patienten zeichnen sich klinisch durch eine reduzierte Herzfrequenzvariabilität, verstärkte QT-Dispersion und verminderte Barorezeptorsensitivität aus.Kasuistiken bestätigen, dass autonome Innervationszustände unter bestimmten Umständen Rhythmusstörungen bis hin zu lebensbedrohlichen ventrikulären Arrhythmien triggern können.
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Bischoff, Angelika. "Notfall akute Arrhythmie." MMW - Fortschritte der Medizin 152, no. 18 (May 2010): 20–21. http://dx.doi.org/10.1007/bf03366494.

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Höltgen, Reinhard, and Bernd Lemke. "Modernes Arrhythmie-Management." Herzschrittmachertherapie + Elektrophysiologie 31, no. 1 (February 27, 2020): 1–2. http://dx.doi.org/10.1007/s00399-020-00663-1.

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sti. "Arrhythmie-Mechanismus bestimmt Ablationsstrategie." CardioVasc 16, no. 5 (November 2016): 31. http://dx.doi.org/10.1007/s15027-016-0994-8.

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Müller, Thomas. "Sportsucht: Trainieren bis zur Arrhythmie." InFo Neurologie & Psychiatrie 17, no. 12 (December 2015): 55–56. http://dx.doi.org/10.1007/s15005-015-1580-9.

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Molitor, Nadine, and Corinna B. Brunckhorst. "CME-EKG 67: Arrhythmie bei Belastung." Praxis 109, no. 16 (December 2020): 1231–37. http://dx.doi.org/10.1024/1661-8157/a003607.

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Zusammenfassung. Ventrikuläre Tachykardien sind potenziell lebensbedrohliche Herzrhythmusstörungen mit einer Herzfrequenz >100 Schläge/min und einem Ursprungsort der Arrhythmie unterhalb des Hisbündels im spezifischen Reizleitungssystem oder im ventrikulären Myokard. Die Morphologie im Oberflächen-EKG kann bereits Hinweise auf den zugrundeliegenden Mechanismus und die damit assoziierte Grunderkrankung liefern. Der jeweilige Pathomechanismus ist entscheidend für die weiterführende Therapie. Dieser Artikel soll einen Einblick in die verschiedenen Ursachen und Therapieoptionen sowie die Differenzialdiagnose der ventrikulären Tachykardien geben.
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Leers, Hans. "Der Lochkartenfall: Kardialer Hochdruck mit Arrhythmie." Zeitschrift für Klassische Homöopathie 25, no. 02 (April 2, 2007): 89. http://dx.doi.org/10.1055/s-2006-937959.

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Ortiz, Miriam, Felix Saha, Volker Schmiedel, and Klaus Trinczek. "4 Fachleute – 4 Behandlungsstrategien bei Arrhythmie." Zeitschrift für Komplementärmedizin 07, no. 01 (February 10, 2015): 36–37. http://dx.doi.org/10.1055/s-0035-1545371.

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Reißig, Angelika, Rudolf Bartunek, Ulf Hengst, and Claus Kroegel. "Absolute Arrhythmie und Pneumatosis cystoides intestinalis." Medizinische Klinik 97, no. 5 (May 1, 2002): 308. http://dx.doi.org/10.1007/s00063-002-1159-3.

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Epifanio, Hindalis Ballesteros, Marcelo Katz, Melania Aparecida Borges, Alessandra da Graça Corrêa, Fátima Dumas Cintra, Rodrigo Leandro Grinberg, Ana Cristina Pinotti Pedro Ludovice, Bruno Pereira Valdigem, Nilton José Carneiro da Silva, and Guilherme Fenelon. "The use of external event monitoring (web-loop) in the elucidation of symptoms associated with arrhythmias in a general population." Einstein (São Paulo) 12, no. 3 (September 2014): 295–99. http://dx.doi.org/10.1590/s1679-45082014ao2939.

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Objective To correlate arrhythmic symptoms with the presence of significant arrhythmias through the external event monitoring (web-loop). Methods Between January and December 2011, the web-loop was connected to 112 patients (46% of them were women, mean age 52±21 years old). Specific arrhythmic symptoms were defined as palpitations, pre-syncope and syncope observed during the monitoring. Supraventricular tachycardia, atrial flutter or fibrillation, ventricular tachycardia, pauses greater than 2 seconds or advanced atrioventricular block were classified as significant arrhythmia. The association between symptoms and significant arrhythmias were analyzed. Results The web-loop recorded arrhythmic symptoms in 74 (66%) patients. Of these, in only 14 (19%) patients the association between symptoms and significant cardiac arrhythmia was detected. Moreover, significant arrhythmia was found in 11 (9.8%) asymptomatic patients. There was no association between presence of major symptoms and significant cardiac arrhythmia (OR=0.57, CI95%: 0.21-1.57; p=0.23). Conclusion We found no association between major symptoms and significant cardiac arrhythmia in patients submitted to event recorder monitoring. Event loop recorder was useful to elucidate cases of palpitations and syncope in symptomatic patients.
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Dissertations / Theses on the topic "Arrhythmie"

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Seebohm, Guiscard Friedrich Aldous. "Untersuchung der spannungsabhängigen Kaliumkanäle KCNQ1 und IKs unter dem Einfluss des Chromanols 293B und die Bedeutung des IKs bei Arrhythmien." [S.l.] : [s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=963926837.

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Hofe, Valérie. "Murine Desmoglein-2-Mutanten als Tiermodell zur Untersuchung der arrhythmogenen rechtsventrikulären Kardiomyopathie." Giessen : VVB Laufersweiler, 2009. http://d-nb.info/994761694/04.

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Wolf, Susanne. "Einfluss blutdrucksenkender Pharmaka auf die Ausbildung einer ARVC bei heterozygot plakoglobindefizienten Mäusen." Giessen VVB Laufersweiler, 2009. http://d-nb.info/996005285/04.

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Voigt, Henning. "Charakterisierung der lichtinduzierten Arrhythmie des circadianen Systems beim Dsungarischen Zwerghamster (Phodopus sungorus) auf physiologischer und molekularer Ebene." [S.l. : s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=968536530.

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Seidel, Thomas. "Eine computermodellgestützte Analyse der elektrophysiologischen Effekte von Gap-Junction-Lateralisierung und zellulärer Hypertrophie in kardialem Gewebe." Doctoral thesis, Universitätsbibliothek Leipzig, 2011. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-79038.

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Die vorliegende Dissertation befasst sich mit Entstehungsmechanismen kardialer Arrhythmien auf der Grundlage pathologisch veränderten Myokards. Es wurde eine systematische Analyse der elektrophysiologischen Veränderungen, die als Folge von Gap-Junction- Lateralisierung und zellulärer Hypertrophie auftreten, durchgeführt. Die Analyse beruht auf einem mathematischen Computermodell, das zur Simulation der Aktionspotentialausbreitung innerhalb einer Einzelzellschicht humaner ventrikulärer Kardiomyozyten entwickelt wurde. Ausgehend von bestehenden Einzelzellmodellen wurde ein räumlich und zeitlich hoch aufgelöstes Multizellmodell generiert und in der Programmiersprache Object Pascal implementiert. Nach Validierung des Modells wurde es zur gezielten, an experimentellen Daten orientierten Manipulation geometrischer Eigenschaften der Zellen (Länge, Durchmesser) und des Zellverbandes (Anordnung der Zellen untereinander) sowie der Gap-Junction-Verteilung genutzt. Die Analyse der elektrophysiologischen Effekte im Vergleich zur Kontrolle fand sowohl unter Normalbedingungen als auch unter Bedingungen, die pathologischen Veränderungen entsprechen (Entkopplung der Gap-Junctions, verringerte Aktivität des schnellen Natriumkanals, erhöhte Inhomogenität), statt. Es zeigte sich, dass ein größerer Zelldurchmesser bzw. erhöhte laterale Gap-Junction-Leitfähigkeit (Simulation von kardialer Hypertrophie bzw. Connexin- Lateralisierung) die Entstehungswahrscheinlichkeit eines unidirektionalen Leitungsblocks erhöhte. Die Erregungsausbreitungsgeschwindigkeit in hypertrophierten Zellen war zudem weniger stabil als in normalen Zellen. Beide Effekte gehören zu den Hauptursachen der Entstehung und Aufrechterhaltung ventrikulärer Arrhythmien. Die Ergebnisse der Arbeit erklären somit Ursachen des erhöhten Arrhythmierisikos in pathologisch veränderten und hypertrophierten Herzen und liefern eine theoretische Grundlage für zukünftige Studien.
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Grothoff, Matthias. "Ermittlung von Einflußfaktoren auf die Arrhythmiegenese bei Patienten mit korrigierter Fallotscher Tetralogie mittels der Magnetresonanztomographie (MRT)." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2004. http://dx.doi.org/10.18452/14994.

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Ziel: Ermittlung von rechts- und linksventrikulären Herzparametern sowie von Zeitparametern, welche die größte Beziehung zum Auftreten von ventrikulären Arrhythmien bei Patienten nach chirurgisch korrigierter Fallotscher Tetralogie haben. Methoden: 67 Patienten, davon 22 weiblich und 45 männlich, mit einem mittleren Alter von 20 a (Spanne von 3,5 bis 54 a) wurden in einem MRT Gyroscan ACS-NT (Philips, Best, The Netherlands) untersucht. Das mittlere Alter zum Zeitpunkt der Korrekturoperation war 6,8 a (mit einer Spanne von 1 Monat bis 40 a). Bei 39 Patienten wurde als Operationsmethode ein transannulärer oder subvalvulärer Patch gewählt, bei 28 Patienten die Anlage eines Conduits. Bei 31 Patienten erfolgten eine oder zwei Reoperationen. Die Volumetrie der Ventrikel erfolgte mit einer SSFP-Sequenz in Multislice-Multiphasen-Technik entweder in Atemstop oder mit Hilfe der Navigatortechnik. Es wurden links- und rechtsventrikuläre Volumina, Ejektionsfraktionen (RV-EF und LV-EF) und Muskelmassen erhoben. Zur Ermittlung der pulmonalen Regurgitationsfraktion (RF) wurde eine MR-Flußmessung in Phasenkontrast-Technik im Pulmonalarterienhauptstamm senkrecht zum Gefäßverlauf durchgeführt. Alle erhobenen Daten wurden sowohl in einer univariaten Regressionsanalyse zueinander, als auch in einer multiplen Regressionsanalyse zur Dauer des QRS-Komplexes in Beziehung gesetzt. Ergebnisse: 58 Patienten hatten einen RSB. Die mittlere Dauer des QRS-Komplexes betrug 132 ms (+/- 29 ms). Die Flußmessung wurde bei 58 Patienten durchgeführt und zeigte 42 milde bis moderate sowie 12 schwere Pulmonalinsuffizienzen. In der univariaten Regressionsanalyse fand sich eine Korrelation zwischen RF und QRS-Dauer (r=0,49; p
Objective - to evaluate those right and left ventricular parameters which have the greatest effect on genesis of cardiac arrhythmias in patients after surgical repair of tetralogy of fallot (TOF). Methods - 67 patients, 22 female and 45 male, with a mean age of 20.0 years (range 3 ½ to 54 years) were studied using a Gyroscan ACS-NT (Philips, Best, The Netherlands). Mean age at surgical repair was 6,8 years (range 1 month to 40 years). Valveless repair with a transannular or subvalvular patch was performed in 39 patients whereas in 28 patients a conduit was used. 31 patients underwent two or three surgical interventions. Measurements of the ventricles were performed with a multislice multiphase gradient echo sequence either with a breathhold technique or with respiratory gating. We obtained left and right ventricular volumes, ejection fractions (EF) and myocardial masses. To quantify the regurgitation fraction (RF) of the pulmonary insuffiency (PI) we performed a velocity encoded gradient echo measurement in the main pulmonary artery in a transverse section. Acquired data was evaluated in an univariate logistic regression analysis on all factors assessed and in a multi variate data analysis with backward selection. Results - 58 patients had a right bundle branch block. Mean QRS duration was 132 ms (+/-29 ms). Flow velocity mapping could be performed in 58 patients and showed 42 mild to moderate and 12 severe cases of PI. In the univariate analysis a significant correlation was found between RF and QRS duration (r=0,49; p
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Dietze, Anna. "Die Wirkung von Desipramin an kardialen Gap Junctions unter ischämischen Bedingungen." Doctoral thesis, Universitätsbibliothek Leipzig, 2016. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-216543.

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Kardiovaskuläre Erkrankungen in Deutschland führen die Todesursachenstatistik an (19,1 % 2013) und verursachen die höchsten Krankheitskosten (14,5 % 2008) (Statistisches Bundesamt, 2015a,b). Im Rahmen von ischämischen Ereignissen am Herzen kann es zu Rhythmusstörungen kommen. In der Therapie dieser Störungen werden traditionell klassische Antiarrhythmika mit Wirkort Ionenkanal eingesetzt, welche jedoch stets ein proarrhythmisches Potenzial aufweisen. Im Fokus der Forschung der letzten Jahre stehen deswegen Peptide wie AAP10 (Antiarrhythmisches Peptid 10), welche direkt an den Gap Junctions ansetzen. In Radioligandenbindungsstudien konnte gezeigt werden, dass Desipramin AAP10 von seinem Rezeptor verdrängen kann. In der vorliegenden Arbeit wurde der Einfluss von Desipramin auf die Gap Junction-Leitfähigkeit in adulten humanen atrialen Kardiomyozyten bestimmt (Jozwiak 2012). Die Bestimmung der Leitfähigkeit erfolgte durch die Technik des Double-Cell-Voltage-Clamp. Es konnte gezeigt werden, dass Desipramin die elektrische Kopplung in humanen Kardiomyozyten, welche vorab durch CO2-induzierte Azidose partiell entkoppelt wurden, erhöht. Weiterhin wurde in der Mapping-Analyse mit dem Langendorff-System gezeigt, dass Desipramin in ischämischen Gebieten am ganzen Kaninchenherzen eine Reduktion der Homogenität und eine Steigerung der Dispersion verhindern kann. In anschließend hergestellten Western Blots aus Gewebeproben derselben Kaninchenherzen ließ sich eine verminderte Dephosphorylierung von Connexin 43 in ischämischen Gebieten unter Desipramin nachweisen. Ebenso vermag Desipramin eine Lateralisierung des Connexin 43 entlang der Zellmembran zu verhindern. Die Ergebnisse zeigen, dass Desipramin die Wahrscheinlichkeit für das Auftreten von Herzrhythmusstörungen unter ischämischen Bedingungen signifikant verringern und damit möglicherweise zur Senkung der Morbidität und Mortalität von Herzkreislauferkrankungen beitragen kann.
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Wadé, Diana [Verfasser], Heidi L. [Akademischer Betreuer] Estner, Isabel V. [Akademischer Betreuer] Deisenhofer, and Steffen [Akademischer Betreuer] Massberg. "Kurz - und Langzeitergebnisse bei Patienten mit AV-Knoten-Reentrytachykardie nach Ablation der langsamen Leitungsbahn : eine Analyse der prädiktiven Faktoren für ein Rezidiv der Arrhythmie / Diana Wadé. Gutachter: Isabel V. Deisenhofer ; Steffen Massberg. Betreuer: Heidi L. Estner." München : Universitätsbibliothek der TU München, 2011. http://d-nb.info/1020706619/34.

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Martins, Jose L. M. G. "A Rapid Access Arrhythmia Clinic for the Diagnosis and Management of Incident Atrial Fibrillation and Other Cardiac Arrhythmias : The Imperial College New Atrial Fibrillation Study." Thesis, Imperial College London, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.520897.

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Walfridsson, Ulla. "Assessing Symptom Burden and Health-Related Quality of Life in patients living with arrhythmia and ASTA : Arrhythmia-Specific questionnaire in Tachycardia and Arrhythmia." Doctoral thesis, Linköpings universitet, Omvårdnad, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-71873.

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Background: Health-Related Quality of Life (HRQOL) can be negatively affected in patients living with arrhythmias and many patients experience a pronounced symptom burden. The arrhythmia can cause both uncertainty and limitations, including interference with work, reluctance to perform and plan for leisure activities and leading to self-imposed restrictions in daily life situations. There are patients striving to find strategies to manage the arrhythmia and for some this can become the focus in their lives. Treatment options are often a choice between pharmaceuticals and radiofrequency ablation (RFA) where RFA is an option for many arrhythmia-patients to be cured. In the care of arrhythmia-patients it is of great importance to combine objective examinations with patient-reported outcomes (PROs) to achieve patient’s own experiences of treatment efficacy and arrhythmias interference in daily life situations. Aims: The overall aims of this thesis were to assess symptom burden and HRQOL in patients with arrhythmias and to develop and validate an arrhythmia-specific questionnaire, suitable for most arrhythmia-patients. Design and Methods: Studies I and II were single-centre studies including patients referred for RFA, with two different arrhythmia diagnoses. Assessments of patient-reported outcomes (PROs) concerning HRQOL were performed using two questionnaires, SF-36 and EQ-5D (I-II). Further, patients were asked some disease-specific questions (I). Study I describes assessments before the RFA treatment and Study II the follow-up assessments at three and twelve months after RFA. Patients’ scoring of HRQOL was compared to age and gender matched reference groups before and after RFA (I-II). Studies III and IV describe the development and validation of a disease-specific questionnaire ASTA (Arrhythmia-Specific questionnaire in Tachycardia and Arrhythmia) assessing symptom burden and HRQOL. Studies III and IV were multicentre studies. Patients planned for DC-conversion, AF patients seeking emergency care and those with different forms of arrhythmias referred for RFA were included. Results: Patients scored significantly lower HRQOL in seven of SF-36’s eight scales compared to the age and gender matched reference groups before RFA treatment. Frequent arrhythmia attacks had a great negative impact on HRQOL, and female gender and older age were factors contributing to worse HRQOL (I). Treatment with RFA restored the patients’ HRQOL. Most positive effects were seen at three months follow-up. One year after treatment patients and the matched reference group scored their HRQOL to a similar level, assessed with SF-36 and EQ-5D index (II). The validated ASTA questionnaire was found to have good psychometric properties. Construct validity was confirmed with sufficient levels of item-total correlations in the ASTA symptom burden scale and HRQOL scales. The dimensionality of the ASTA HRQOL scale was established with confirmatory factor analysis, supporting a physical and a mental subscale. The internal consistency, demonstrated with Cronbach’s alpha (α), was satisfactory for the ASTA symptom burden scale and the ASTA HRQOL scales, varying from α 0.79 to α 0.91 (III-IV). Conclusions and clinical implications: The studies in this thesis confirmed how negatively affected the arrhythmia-patients can be with a pronounced symptom burden and impaired HRQOL. Treatment with RFA was demonstrated to restore the patients HRQOL to an equal level of that of the matched reference group. PROs are important to take into consideration in the care of arrhythmia-patients, to achieve the patients’ subjective experiences of their daily life situation. To the best of our knowledge ASTA is the first arrhythmia-specific questionnaire assessing symptom burden and HRQOL, suitable for most arrhythmia forms. The newly validated ASTA questionnaire can be an important contribution to assessment of PROs in arrhythmia-patients.
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Books on the topic "Arrhythmie"

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Sesto, Fred, ed. Arrhythmie-Kompendium IV. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-93378-3.

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Sesto, Fred, ed. Arrhythmie-Kompendium III. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70758-2.

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Sesto, Fred, ed. Arrhythmie-Kompendium I. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71840-3.

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Arrhythmias essentials. Sudbury, Mass: Jones & Bartlett Learning, 2012.

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1931-, Conover Mary Boudreau, ed. Advanced concepts in arrhythmias. 3rd ed. St. Louis: Mosby, 1998.

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1931-, Conover Mary Boudreau, ed. Advanced concepts in arrhythmias. 2nd ed. St. Louis: Mosby, 1989.

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P, Zipes Douglas, ed. Electrocardiography of arrhythmias: A comprehensive review. Philadelphia, PA: Elsevier/Saunders, 2012.

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Arrhythmia. Edmonton: NeWest Press, 2011.

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J, Wilber David, ed. Cryoablation of cardiac arrhythmias. Philadelphia, PA: Elsevier/Saunders, 2011.

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Cardiac arrhythmia management: A practical guide for nurses and allied professionals. Chichester, West Sussex, UK: Wiley-Blackwell, 2011.

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Book chapters on the topic "Arrhythmie"

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Sesto, Fred. "Welche Merkmale können in der präklinischen Diagnostik das Spektrum möglicher Rhythmusstörungen einengen?" In Arrhythmie-Kompendium IV, 12–13. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-93378-3_6.

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Sesto, Fred. "Was sind die Ursachen des Vorhofflimmerns?" In Arrhythmie-Kompendium IV, 29–30. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-93378-3_18.

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Sesto, Fred. "Welche Fasertypen des Myokards können aufgrund ihrer elektrophysiologischen Eigenschaften differenziert werden?" In Arrhythmie-Kompendium III, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70758-2_1.

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Sesto, Fred. "Was versteht man unter einer verborgenen Leitung?" In Arrhythmie-Kompendium III, 11–12. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70758-2_10.

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Sesto, Fred. "Welche Erregungsprozesse führen am häufigsten zum plötzlichen Herztod?" In Arrhythmie-Kompendium III, 13–14. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70758-2_11.

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Sesto, Fred. "Neben der spontanen diastolischen Reizschwelle spricht man auch von der Flimmerschwelle. Was ist darunter zu verstehen, und welche Bedeutung hat die Flimmerschwelle?" In Arrhythmie-Kompendium III, 15. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70758-2_12.

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Sesto, Fred. "Wird Kammerflimmern vorwiegend durch abnorm gesteigerte ektopische Automatie oder durch kreisende Erregungen ausgelöst?" In Arrhythmie-Kompendium III, 16. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70758-2_13.

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Sesto, Fred. "Liegen z.Z. neuere, aussagekräftige Studien über die Art von Rhythmusstörungen vor, die kurz vor dem Auftreten von Kammerflimmern registriert werden konnten?" In Arrhythmie-Kompendium III, 17. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70758-2_14.

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Sesto, Fred. "Welche Herzerkrankungen, die zum plötzlichen Tod führten, konnten in der vorausgehend aufgeführten Studie (Frage 14) am häufigsten festgestellt werden?" In Arrhythmie-Kompendium III, 18. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70758-2_15.

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Sesto, Fred. "Was versteht man unter schnellen und langsamen Erregungsformen, und welche klinische Bedeutung fällt ihnen zu?" In Arrhythmie-Kompendium III, 19–20. Berlin, Heidelberg: Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70758-2_16.

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Conference papers on the topic "Arrhythmie"

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Asbach, JC, A. Moussa, T. Bürger, and E. Stegemann. "Arrhythmie und akute Beinischämie – Diagnosefindung mittels FKDS." In Interdisziplinärer Kongress | Ultraschall 2018 – 42. Dreiländertreffen SGUM | DEGUM | ÖGUM. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1670369.

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Boyer, Richard B., Pramode Chiruvolu, Arun Jose, Joshua Liu, Adam Sifuentes, Allison Connolly, Britni Crocker, and Peter Stempriewica. "Enhancing SVT Discrimination in Implantable Cardioverter Defibrillators Using MEMS Accelerometers." In ASME 2007 2nd Frontiers in Biomedical Devices Conference. ASMEDC, 2007. http://dx.doi.org/10.1115/biomed2007-38056.

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Sudden cardiac death (SCD) accounts for over 325,000 deaths in the United States per year. Implantable cardioverter defibrillators (ICDs), about 100,000 of which are implanted each year, are used to diagnose and treat cardiac arrhythmias in patients that are at risk for sudden cardiac death due to ventricular fibrillation. Upon detection of an arrhythmia, the ICD has several treatment options, all of which deliver varied amounts of electric current to the myocardium. Detection of ventricular tachycardia (VT) or ventricular fibrillation (VF) prompts the ICD to administer high-energy defibrillation shocks, which can exceed 30J. The current method for sensing arrhythmias is the use of electrodes implanted in the myocardium which are capable of detecting electric potentials. The extensively studied algorithms that analyze electrogram sensor data have allowed ICD’s to achieve a 0% false negative rate for detection of fibrillation. The drawback, however, is the high false positive rate of over 22%. False positives result in inappropriate shocks which have detrimental effects on patient health and quality of life [1].
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Yazawa, Toru, Albert M. Hutapea, Tomoo Katsuyama, and Yukio Shimoda. "Detrended Fluctuation Analysis of Arrhythmia: Scaling Exponent as an Index of Heart Wellness." In ASME 2011 International Mechanical Engineering Congress and Exposition. ASMEDC, 2011. http://dx.doi.org/10.1115/imece2011-62184.

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Well-established technologies to analyze biological signals including rhythmic heartbeat are available and accessible to scholars. However, stronger empirical evidence is required to justify the use of these technologies as practical tools in the field of biomedicine. Here we conducted analyses of heartbeat interval time series using an analytical technology developed across three decades—detrended fluctuation analysis (DFA)—to verify the power-law/scaling characteristics of signals that fluctuate in a regular, irregular, or erratic manner. We believe that DFA is a useful tool because it can quantify the heart condition by a scaling exponent, with a value of one (1) set as the default for a healthy state. This baseline value can be compared to a clinical thermometer, where the baseline is 37 °C for a physiologically healthy condition. Our study aimed to ascertain and confirm the utility of DFA in evaluating heart wellness, specifically in the context of studying arrhythmic heartbeat. We present case studies to confirm that DFA is a beneficial tool that quantifies the scaling exponent of a heart’s condition as “nonstationarily” beating and dynamically controlled. From an engineering perspective, we show that the heart condition can be classified into two typical categories: a healthy rhythm with a scaling exponent of one (1.0), and arrhythmia with a lower scaling exponent (0.7 or less).
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Silva, Anderson P. N., Gibeon S. Aquino-Júnior, João C. Xavier-Júnior, and Cephas A. S. Barreto. "A cardiac arrhythmia monitoring platform based on feature selection and classification methods." In X Simpósio Brasileiro de Computação Ubíqua e Pervasiva. Sociedade Brasileira de Computação - SBC, 2018. http://dx.doi.org/10.5753/sbcup.2018.3292.

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Heart arrhythmia, also known as irregular heartbeat, affects millions of people around the world. One of the ways to detect this cardiac dysrhythmia is by performing an electrocardiogram (ECG) exam which records the electrical activity of the heart. However, this type of exam is always interpreted by a doctor. In order to provide an alternative in heart arrhythmia diagnosis, this paper aims at developing a platform based on Internet of Things infrastructure capable of automatically monitoring and identifying cardiac arrhythmia based on feature selection and classification methods.
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Iles, Tinen L., Timothy G. Laske, David L. Garshelis, Lars Mattison, Brian Lee, Val Eisele, Erik Gaasedelen, and Paul A. Iaizzo. "Medtronic Reveal LINQ™ Devices Provide Better Understanding of Hibernation Physiology in the American Black Bear (Ursus Americanus)." In 2017 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dmd2017-3498.

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The American black bear (Ursus americanus) has been called a metabolic marvel6. In northern Minnesota, where we have conducted long-term physiological and ecological studies of this species, bears may remain in their winter dens for 6 months or more without eating, drinking, urinating or defecating and yet lose very little muscle mass2. We also found that hibernating black bears elicit asystolic events of over 30 seconds and experience an exaggerated respiratory sinus arrhythmia2. In this previous work we employed Medtronic Reveal® XT devices that required us to visit the den and temporarily extract the bear (under anesthesia) to download the stored data.4 Here we describe Medtronic’s latest generation of Insertable Cardiac Monitor (ICM), the Reveal LINQ™, which enables continuous transmission of data via a relay station from the den site3. Black bear hibernation physiology remains of high interest because of the multiple potential applications to human medicine. ICMs have been used for nearly two decades by clinicians as a critical diagnostic tool to assess the nature of cardiac arrhythmias in humans. Such devices are primarily implanted subcutaneously to record electrocardiograms. The device size, battery life and transmission capabilities have evolved in recent years. The first devices were relatively large and a programmer was needed to retrieve information during each clinical (or in our case, den visit). These devices were programmed to capture cardiac incidents such as asystolic events, arrhythmias and tachycardias and apply algorithms that ensure proper data collection: e.g. ectopy rejection and p-wave presence algorithms. The new generation Reveal LINQ was made to telemetrically transmit heart data from human patients, but we needed to develop a system to enable transmission from bear dens, which are remote (cannot easily be checked and adjusted) and are subject to extreme winter weather conditions. Besides the advantage of these devices transmitting data automatically, they are considerably smaller and thus less prone to rejection by the extraordinary immune system of the hibernating bear1.
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Milazzotto, F., M. Carelli, C. Citone, G. Di Macro Tullio, G. C. Gambelli, P. Giampaolo, U. Malinconico, C. Polizzi, and U. Cornelli. "EFFECTIVENESS OF DEFIBROTIDE IN THE TREATMENT OF ACUTE MYOCARDIAL INFARCTION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643146.

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Defibrotide (D) is an extractive polydesoxyribonucleotide. In preclinical studies the product was shown to be active as a pro -fibrinolytic, antithrombotic and thrombolytic agent while comply tely devoid of anticoagulant activity. In animal models, D was found to afford striking protection from the effects of acute lethal and non lethal myocardial ischemia as well as from myo -cardial injury following reperfusion. In this open single - blim trial, D was administered to patients with acute myocardial infaj ction (AMI) for the prevention of complicating arrhythmias; throy bus formation, pericarditis, etc.Sixty patients with AMI were divided randomly into two groups of 30 patients each. One group was treated with D by 6-hour drip infusion for 3 consecutive days (2.8 g on the first day, then 2.4 g daily). The other group was treated with equal volumes of physiological salt solution. All patients received conventional trea_t ment for AMI. The two trial groups were sufficiently homogeneous in terms of AMI type, age and sex distribution, PCT, Forrester index, Holter, and Peel index. D treatment proved effective in reducing the incidence of severe arrhythmia (p < 0.05), thrombus formation (p < 0.05), and pericarditis (p < 0.01). CPK, TT and PTT readings were not modified by the treatment; the incidence of post-AMI angina and the number of deaths (4 in each group) were similar in the two groups. The results of this pilot study are encouraging; further clinical trials are currently in progress to assess D activity in larger groups of patients treated with the product at higher dosages.
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Rege, Sanket, Tyler Barkey, and Mariano Lowenstern. "Heart arrhythmia detection." In 2015 IEEE Virtual Conference on Applications of Commercial Sensors (VCACS). IEEE, 2015. http://dx.doi.org/10.1109/vcacs.2015.7439569.

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Wu, Meng-Hsi, and Edward Y. Chang. "DeepQ Arrhythmia Database." In MM '17: ACM Multimedia Conference. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3132635.3132647.

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Baia, Alexandre Farias, and Adriana Rosa Garcez Castro. "A Competitive Structure of Convolutional Autoencoder Networks for Electrocardiogram Signals Classification." In XV Encontro Nacional de Inteligência Artificial e Computacional. Sociedade Brasileira de Computação - SBC, 2018. http://dx.doi.org/10.5753/eniac.2018.4446.

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This paper presents the proposal of an electrocardiogram (ECG) signals classification system through a competitive structure of Convolutional Autoencoders (CAE). Two Convolutional Autoencoders were trained to reconstruct ECG signals for the cases of patients with arrhythmia and patients with signals considered normals. After the training, the two networks were arranged in a competitive parallel structure to classify these signals. For the development and testing of the system, the MIT-BIH Arrhythmia Database of ECG signals was used. An accuracy of 88,9% was achieved considering the database used for system testing.
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Gallenberger, Danny, Min Xiong, Tony Z. Zhuang, Kai Sun, Elena G. Tolkacheva, and Xiaopeng Zhao. "A Cellular Automata Model for Dynamics and Control of Cardiac Arrhythmias." In ASME 2020 Dynamic Systems and Control Conference. American Society of Mechanical Engineers, 2020. http://dx.doi.org/10.1115/dscc2020-3129.

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Abstract As a leading cause of death in 325,000 adults per year in the United States, a significant proportion of sudden cardiac arrest (SCA) result from arrhythmias. To better understand the onset of arrhythmias and its potential treatment with more rapid and effective control approaches, a two-dimensional 50 × 50 cellular automata (CA) model is used in this study to illustrate the propagation of electrical waves across its tissue, and a constant diastolic interval (DI) control mechanism is adopted to help stabilize and prevent cardiac arrhythmias. Simulations of various scenarios including normal conduction and spiral waves in the presence of scar, normal conduction and alternans under control conditions are shown. The results validate that the CA model and constant DI control method are very efficient and effective in the study of dynamics and control of cardiac arrhythmias.
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Reports on the topic "Arrhythmie"

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Ye, Yanping. Designing New Drugs to Treat Cardiac Arrhythmia. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.638.

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Jinming, Song, Tang Yao, Gao Chao, and Xu Zongpei. Efficiency between Guan-fu base A versus propafenone in the treatment of arrhythmia: a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2021. http://dx.doi.org/10.37766/inplasy2021.3.0077.

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Qiu, Mei, Liang-Liang Ding, and Hai-Rong Zhou. Effects of SGLT2 inhibitors and GLP1 receptor agonists on risk of various arrhythmias: a meta-analysis of large randomized trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, January 2021. http://dx.doi.org/10.37766/inplasy2021.1.0102.

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Dong, Zhenyu, Muyassar Yusup, Yanmei Lu, and Baopeng Tang. The effectiveness of angiotensin receptor-neprilysin inhibitor in ventricular arrhythmia in patients with Heart Failure with Reduced Ejection Fraction: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2021. http://dx.doi.org/10.37766/inplasy2021.9.0012.

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Pshezhetskiy, Dmitry, Tanveer Alam, and Heba Alshaker. Unsynchronised Cardioversion as a Cause of Ventricular Tachycardia in a Patient with Atrial Fibrillation. Nature Library, November 2020. http://dx.doi.org/10.47496/nl.ccr.2020.01.02.

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Background: Synchronised cardioversion (SC) is used to terminate tachycardic arrhythmia by applying electric current to the thorax. SC is synchronised to the R wave of the cardiac cycle and ventricular tachycardia (VT) or ventricular fibrillation (VF) can occur if an electrical shock is provided in a nonsynchronised way. Case Presentation: Here we present a case of a 66-year-old man who had elective cardioversion for atrial fibrillation worsened by severe left ventricular impairment. A manual defibrillator was used for the cardioversion, which, after the first synchronised shock, reverted to defibrillator mode. An unsynchronised shock was administered and induced VT, which was reverted to sinus rhythm with a defibrillation shock. Conclusion: When using manual defibrillator for SC, the machine needs to be set to a synchronised mode. The synchronisation to the R wave needs to be checked before every shock.
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Treadwell, Jonathan R., James T. Reston, Benjamin Rouse, Joann Fontanarosa, Neha Patel, and Nikhil K. Mull. Automated-Entry Patient-Generated Health Data for Chronic Conditions: The Evidence on Health Outcomes. Agency for Healthcare Research and Quality (AHRQ), March 2021. http://dx.doi.org/10.23970/ahrqepctb38.

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Background. Automated-entry consumer devices that collect and transmit patient-generated health data (PGHD) are being evaluated as potential tools to aid in the management of chronic diseases. The need exists to evaluate the evidence regarding consumer PGHD technologies, particularly for devices that have not gone through Food and Drug Administration evaluation. Purpose. To summarize the research related to automated-entry consumer health technologies that provide PGHD for the prevention or management of 11 chronic diseases. Methods. The project scope was determined through discussions with Key Informants. We searched MEDLINE and EMBASE (via EMBASE.com), In-Process MEDLINE and PubMed unique content (via PubMed.gov), and the Cochrane Database of Systematic Reviews for systematic reviews or controlled trials. We also searched ClinicalTrials.gov for ongoing studies. We assessed risk of bias and extracted data on health outcomes, surrogate outcomes, usability, sustainability, cost-effectiveness outcomes (quantifying the tradeoffs between health effects and cost), process outcomes, and other characteristics related to PGHD technologies. For isolated effects on health outcomes, we classified the results in one of four categories: (1) likely no effect, (2) unclear, (3) possible positive effect, or (4) likely positive effect. When we categorized the data as “unclear” based solely on health outcomes, we then examined and classified surrogate outcomes for that particular clinical condition. Findings. We identified 114 unique studies that met inclusion criteria. The largest number of studies addressed patients with hypertension (51 studies) and obesity (43 studies). Eighty-four trials used a single PGHD device, 23 used 2 PGHD devices, and the other 7 used 3 or more PGHD devices. Pedometers, blood pressure (BP) monitors, and scales were commonly used in the same studies. Overall, we found a “possible positive effect” of PGHD interventions on health outcomes for coronary artery disease, heart failure, and asthma. For obesity, we rated the health outcomes as unclear, and the surrogate outcomes (body mass index/weight) as likely no effect. For hypertension, we rated the health outcomes as unclear, and the surrogate outcomes (systolic BP/diastolic BP) as possible positive effect. For cardiac arrhythmias or conduction abnormalities we rated the health outcomes as unclear and the surrogate outcome (time to arrhythmia detection) as likely positive effect. The findings were “unclear” regarding PGHD interventions for diabetes prevention, sleep apnea, stroke, Parkinson’s disease, and chronic obstructive pulmonary disease. Most studies did not report harms related to PGHD interventions; the relatively few harms reported were minor and transient, with event rates usually comparable to harms in the control groups. Few studies reported cost-effectiveness analyses, and only for PGHD interventions for hypertension, coronary artery disease, and chronic obstructive pulmonary disease; the findings were variable across different chronic conditions and devices. Patient adherence to PGHD interventions was highly variable across studies, but patient acceptance/satisfaction and usability was generally fair to good. However, device engineers independently evaluated consumer wearable and handheld BP monitors and considered the user experience to be poor, while their assessment of smartphone-based electrocardiogram monitors found the user experience to be good. Student volunteers involved in device usability testing of the Weight Watchers Online app found it well-designed and relatively easy to use. Implications. Multiple randomized controlled trials (RCTs) have evaluated some PGHD technologies (e.g., pedometers, scales, BP monitors), particularly for obesity and hypertension, but health outcomes were generally underreported. We found evidence suggesting a possible positive effect of PGHD interventions on health outcomes for four chronic conditions. Lack of reporting of health outcomes and insufficient statistical power to assess these outcomes were the main reasons for “unclear” ratings. The majority of studies on PGHD technologies still focus on non-health-related outcomes. Future RCTs should focus on measurement of health outcomes. Furthermore, future RCTs should be designed to isolate the effect of the PGHD intervention from other components in a multicomponent intervention.
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Schwieger, Alexandra, Kaelee Shrewsbury, and Paul Shaver. Dexmedetomidine vs Fentanyl in Attenuating the Sympathetic Surge During Endotracheal Intubation: A Scoping Review. University of Tennessee Health Science Center, July 2021. http://dx.doi.org/10.21007/con.dnp.2021.0007.

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Purpose/Background Direct laryngoscopy and endotracheal intubation after induction of anesthesia can cause a reflex sympathetic surge of catecholamines caused by airway stimulation. This may cause hypertension, tachycardia, and arrhythmias. This reflex can be detrimental in patients with poor cardiac reserve and can be poorly tolerated and lead to adverse events such as myocardial ischemia. Fentanyl, a potent opioid, with a rapid onset and short duration of action is given during induction to block the sympathetic response. With a rise in the opioid crisis and finding ways to change the practice in medicine to use less opioids, dexmedetomidine, an alpha 2 adrenergic agonist, can decrease the release of norepinephrine, has analgesic properties, and can lower the heart rate. Methods In this scoping review, studies published between 2009 and 2021 that compared fentanyl and dexmedetomidine during general anesthesia induction and endotracheal intubation of surgical patients over the age of 18 were included. Full text, peer-reviewed studies in English were included with no limit on country of study. The outcomes included post-operative reviews of decrease in pain medication usage and hemodynamic stability. Studies that were included focused on hemodynamic variables such as systolic blood pressure, diastolic blood pressure, mean arterial pressure, heart rate, and use of opioids post-surgery. Result Of 2,114 results from our search, 10 articles were selected based on multiple eligibility criteria of age greater than 18, patients undergoing endotracheal intubation after induction of general anesthesia, and required either a dose of dexmedetomidine or fentanyl to be given prior to intubation. Dexmedetomidine was shown to effectively attenuate the sympathetic surge during intubation over fentanyl. Dexmedetomidine showed a greater reduction in heart rate, systolic blood pressure, diastolic blood pressure, mean arterial pressure than fentanyl, causing better hemodynamic stability in patients undergoing elective surgery.Implications for Nursing Practice Findings during this scoping review indicate that dexmedetomidine is a safe and effective alternative to fentanyl during induction of general anesthesia and endotracheal intubation in attenuating the hemodynamic response. It is also a safe choice for opioid-free anesthesia.
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Fire fighter suffers fatal heart arrhythmia at structure fire - Illinois. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, July 2003. http://dx.doi.org/10.26616/nioshfffacef200309.

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Fire fighter suffers heart arrhythmia and dies at wildland fire - Washington. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, August 2002. http://dx.doi.org/10.26616/nioshfffacef200137.

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Fire fighter suffers cardiac arrhythmia during grass fire operations and dies 10 days later - North Carolina. U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National Institute for Occupational Safety and Health, March 2010. http://dx.doi.org/10.26616/nioshfffacef200928.

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