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Journal articles on the topic 'ARRHYTHMOGENIC'

Author: Grafiati

Published: 1 April 2023

Last updated: 25 July 2025

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1

Podgoršek, Blaž, Gregor Poglajen, Andraž Cerar, Matjaž Šinkovec, and Bojan Vrtovec. "Arrhythmogenic Cardiomyopathy." Slovenian Medical Journal 87, no. 11-12 (2019): 599–618. http://dx.doi.org/10.6016/zdravvestn.2723.

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Arrhythmogenic cardiomyopathy (AC) is a genetic disease of the myocardium characterized by fibro-fatty replacement of the apoptotic myocardium. It primarily affects the right ventricle, however in advanced stages of the disease the left ventricle can also be significantly affected. AC is a challenging diagnosis, especially in the early stages of the disease, and should be considered in all patients presenting with palpitations, syncope or sudden cardiac death when other, more common causes of these symptoms/signs are excluded. In patients with suspected AC, evaluation according to the cu

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2

Priori, Silvia G., and Demetrio J. Santiago. "Arrhythmogenic Cardiomyopathy." Circulation Research 121, no. 12 (2017): 1296–98. http://dx.doi.org/10.1161/circresaha.117.312211.

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3

Abraham, Terri. "Arrhythmogenic Mechanisms." AACN Advanced Critical Care 3, no. 1 (1992): 157–65. http://dx.doi.org/10.4037/15597768-1992-1020.

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The arrhythmogenic mechanisms are the basis for the genesis of a wide variety of complex dysrhythmias that can arise in both pacemaker and nonpacemaker cells. Automaticity, or the ability to rhythmically and spontaneously depolarize cardiac cells, is normally the domain of the sinus node. Altered automaticity takes place when conduction is enhanced or abnormal. A second mechanism, reentry, refers to a phenomenon that occurs when an impulse is delayed within a pathway of slow conduction and then reenters surrounding tissue and produces another impulse. One-way conduction is necessary to produce

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4

Corrado, Domenico, Cristina Basso, and Daniel P. Judge. "Arrhythmogenic Cardiomyopathy." Circulation Research 121, no. 7 (2017): 784–802. http://dx.doi.org/10.1161/circresaha.117.309345.

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5

Beffagna, Giorgia, Alessandro Zorzi, Kalliopi Pilichou, et al. "Arrhythmogenic Cardiomyopathy." European Heart Journal 41, no. 47 (2020): 4457–62. http://dx.doi.org/10.1093/eurheartj/ehaa719.

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6

Mestroni, Luisa, and Orfeo Sbaizero. "Arrhythmogenic Cardiomyopathy." Circulation 137, no. 15 (2018): 1611–13. http://dx.doi.org/10.1161/circulationaha.118.033558.

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7

Pilichou, Kalliopi, Connie R. Bezzina, Gaetano Thiene, and Cristina Basso. "Arrhythmogenic Cardiomyopathy." Circulation: Cardiovascular Genetics 4, no. 3 (2011): 318–26. http://dx.doi.org/10.1161/circgenetics.110.959031.

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8

Jallon, P. "Arrhythmogenic Seizures." Epilepsia 38 (November 1997): S43—S47. http://dx.doi.org/10.1111/j.1528-1157.1997.tb06127.x.

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9

Olivetti, Natalia, Luciana Sacilotto, and Marcelo Luiz Campos Vieira. "Arrhythmogenic Cardiomyopathy." JACC: Asia 5, no. 7 (2025): 924–26. https://doi.org/10.1016/j.jacasi.2025.05.010.

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10

Sadredini, Mani, Marie Haugsten Hansen, Michael Frisk, et al. "CaMKII inhibition has dual effects on spontaneous Ca2+ release and Ca2+ alternans in ventricular cardiomyocytes from mice with a gain-of-function RyR2 mutation." American Journal of Physiology-Heart and Circulatory Physiology 321, no. 2 (2021): H446—H460. http://dx.doi.org/10.1152/ajpheart.00011.2021.

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Genetically increased RyR2 activity promotes arrhythmogenic Ca2+ release. Inhibition of CaMKII suppresses RyR2 activity and arrhythmogenic Ca2+ release. Suppression of RyR2 activity prolongs refractoriness of Ca2+ release. Prolonged refractoriness of Ca2+ release leads to arrhythmogenic Ca2+ alternans. CaMKII inhibition promotes Ca2+ alternans by prolonging Ca2+ release refractoriness.

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11

Bonet, Fernando, Oscar Campuzano, José Córdoba-Caballero, et al. "Role of miRNA–mRNA Interactome in Pathophysiology of Arrhythmogenic Cardiomyopathy." Biomedicines 12, no. 8 (2024): 1807. http://dx.doi.org/10.3390/biomedicines12081807.

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Arrhythmogenic cardiomyopathy is an inherited entity characterized by irregular cell–cell adhesion, cardiomyocyte death and fibro-fatty replacement of ventricular myocytes, leading to malignant ventricular arrythmias, contractile dysfunction and sudden cardiac death. Pathogenic variants in genes that encode desmosome are the predominant cause of arrhythmogenic cardiomyopathy. Moreover, signalling pathways such as Wnt/ß-catenin and transforming growth factor-β have been involved in the disease progression. However, still little is known about the molecular pathophysiological mechanisms that und

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12

Mirmomen, Seyedeh Mojdeh, Andrew Jay Bradley, Andrew Ernest Arai, and Arlene Sirajuddin. "Arrhythmogenic left ventricular cardiomyopathy." BJR|case reports 6, no. 1 (2020): 20190079. http://dx.doi.org/10.1259/bjrcr.20190079.

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Arrhythmogenic ventricular cardiomyopathy (AVC) is a heritable heart muscle disorder characterized by fibrofatty infiltration of the myocardium. Intramyocardial fat deposition is considered arrhythmogenic and predisposes patients to life-threatening arrhythmias and sudden cardiac death. The classic subtype of AVC is characterized by fibrofatty replacement of the right ventricular myocardium (i.e. arrhythmogenic right ventricular cardiomyopathy). In advanced cases of arrhythmogenic right ventricular cardiomyopathy, the left ventricle may be involved as well. Predominantly left ventricular invol

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13

Rawal, Aranyak S., Tara VanCleave, Neeraja Yedlapati, Jeffery E. Saffitz, William James Craigen, and John L. Jefferies. "Arrhythmogenic Ventricular Cardiomyopathy." JACC: Case Reports 3, no. 3 (2021): 438–42. http://dx.doi.org/10.1016/j.jaccas.2020.12.012.

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14

Priori, Silvia G. "Inherited Arrhythmogenic Diseases." Circulation Research 94, no. 2 (2004): 140–45. http://dx.doi.org/10.1161/01.res.0000115750.12807.7e.

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15

Topaz, On. "Arrhythmogenic RV dysplasia." American Heart Journal 112, no. 6 (1986): 1343. http://dx.doi.org/10.1016/0002-8703(86)90380-7.

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16

Millane, Teri A., David E. Ward, and A. John Camm. "Is hypomagnesemia arrhythmogenic?" Clinical Cardiology 15, no. 2 (1992): 103–8. http://dx.doi.org/10.1002/clc.4960150210.

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17

Corrado, Domenico, Alessandro Zorzi, Alberto Cipriani, et al. "Scarring/arrhythmogenic cardiomyopathy." European Heart Journal Supplements 25, Supplement_C (2023): C144—C154. http://dx.doi.org/10.1093/eurheartjsupp/suad017.

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Abstract The designation of ‘arrhythmogenic cardiomyopathy’ reflects the evolving concept of a heart muscle disease affecting not only the right ventricle (ARVC) but also the left ventricle (LV), with phenotypic variants characterized by a biventricular (BIV) or predominant LV involvement (ALVC). Herein, we use the term ‘scarring/arrhythmogenic cardiomyopathy (S/ACM)’ to emphasize that the disease phenotype is distinctively characterized by loss of ventricular myocardium due to myocyte death with subsequent fibrous or fibro-fatty scar tissue replacement. The myocardial scarring predisposes to

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18

Sarquella-Brugada, Georgia, and Oscar Campuzano. "Inherited Arrhythmogenic Syndromes." Cardiogenetics 13, no. 4 (2023): 173–74. http://dx.doi.org/10.3390/cardiogenetics13040016.

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19

Mathavan, Akshay, Urszula Krekora, Miguel Belaunzaran Dominguez, and Akash Mathavan. "Heterozygous desmoplakin (DSP) variants presenting with early onset cardiomyopathy and refractory ventricular tachycardia." BMJ Case Reports 17, no. 2 (2024): e259308. http://dx.doi.org/10.1136/bcr-2023-259308.

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Arrhythmogenic cardiomyopathy is a non-ischaemic cardiomyopathy characterised by the presence of myocardial dysfunction and inherited conduction disease that predisposes patients to malignant ventricular arrhythmias and sudden cardiac death. There is a growing awareness of the diverse phenotypic presentation of arrhythmogenic cardiomyopathy, which may demonstrate preferential involvement of the left, right or both ventricles. A subset of arrhythmogenic cardiomyopathy may be due to mutations of desmosomes, intercellular junctions of the myocardium that promote structural and electrical integrit

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20

Tapoi, Laura, Alexandra Clement, Rodica Radu, and Radu Sascau. "Multimodality imaging in arrhythmogenic cardiomyopathy." Romanian Journal of Cardiology 31, no. 1 (2021): 10–16. http://dx.doi.org/10.47803/rjc.2021.31.1.10.

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Arrhythmogenic cardiomyopathy, as it has been recently redefi ned, is characterized by progressive myocyte loss with fibrosis and fat infiltration of the myocardium, which finally leads to a broad clinical spectrum ranging from heart failure symptoms to sudden cardiac death. The diagnosis of arrhythmogenic cardiomyopathy is challenging particularly because of its heterogeneity in presentation, which varies from focal right ventricular involvement to biventricular or prominent left ventricular phenotype. In the past decades, the development of new electrocardiographic and imaging diagnostic cri

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21

Murakawa, Y., K. Sezaki, T. Yamashita, Y. Kanese, and M. Omata. "Three-dimensional activation sequence of cesium-induced ventricular arrhythmias." American Journal of Physiology-Heart and Circulatory Physiology 273, no. 3 (1997): H1377—H1385. http://dx.doi.org/10.1152/ajpheart.1997.273.3.h1377.

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To investigate the electrophysiological and electrocardiographic characteristics of ventricular arrhythmia due to abnormal repolarization, we studied the three-dimensional activation sequence of cesium-induced ventricular tachycardia (VT) in 10 anesthetized dogs using a 384-channel recording system. Seventeen monomorphic VT (mVT) and eight polymorphic VT (pVT) episodes induced by cesium chloride (2 or 3 mM/kg) were analyzed. Only a single arrhythmogenic focus was detected in most beats of VT, whereas two competing foci were temporarily observed in two episodes of pVT. The site of arrhythmogeni

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22

Lala, Ioan Radu, and Adina Pop-Moldovan. "Inflammation—A Possible Link between Myocarditis and Arrhythmogenic Cardiomyopathy." Diagnostics 14, no. 3 (2024): 248. http://dx.doi.org/10.3390/diagnostics14030248.

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Arrhythmogenic cardiomyopathy is a primary genetic disease caused by mutations in the desmosome genes. Ever since the introduction of new imaging techniques, like cardiovascular magnetic resonance, the diagnosis of arrhythmogenic cardiomyopathy has become more challenging as left ventricular or biventricular variants may have resemblance to other cardiomyopathies or myocarditis. Not only this but they may also share an acute phase, which might cause even more confusion and misdiagnoses and influence the prognosis and outcome. In this case report, we present a 31-year-old patient with multiple

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23

López-Flores, Luis, Rocio Cózar-León, and Ernesto Díaz-Infante. "Epsilon Wave in Arrhythmogenic Cardiomyopathy." Revista de Medicina Clínica 8, no. 1 (2024): e22042408004. https://doi.org/10.5281/zenodo.14170391.

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24

Bednarski, Richard M., Lynda J. Majors, and John L. Atlee. "Epinephrine-induced ventricular arrhythmias in dogs anesthetized with halothane: Potentiation by thiamylal and thiopental." American Journal of Veterinary Research 46, no. 9 (1985): 1829–31. https://doi.org/10.2460/ajvr.1985.46.09.1829.

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SUMMARY Epinephrine-induced ventricular arrhythmias were studied in 8 dogs anesthetized at weekly intervals with halothane (1.09% end-tidal concentration) preceded by thiamylal or thiopental (20 mg/kg of body weight). Lead II, bundle of His and high right atrial electrograms, and femoral artery and airway pressures were recorded. Epinephrine was infused in logarithmically spaced increasing rates (initial rate = 0.25 μg/kg/min) for a maximum of 2.5 minutes. The maximal (≥ 4 ventricular premature depolarizations within 15 s of each other) and minimal (all other ventricular or junctional rhythms)

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25

Balwierz, Magdalena. "Arrhythmogenic right ventricular cardiomyopathy - What Is New since 2023 European Society of Cardiology Guidelines? Current Knowledge, Literature Review." Quality in Sport 38 (February 7, 2025): 58173. https://doi.org/10.12775/qs.2025.38.58173.

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Introduction: Arrhythmogenic right ventricular cardiomyopathy (ARVC) is a genetically conditioned disease, characterized by gradual fibro-fatty replacement of myocardium and tendency to ventricular arrhythmias and heart failure. ARVC is included in a group of inherited similar clinical entities characterized by similar remodeling changes, named in general arrhythmogenic cardiomyopathies (ACM). The new guidelines of European Society of Cardiology (ESC) for the management of cardiomyopathies (2023) redefine arrhythmogenic left ventricular cardiomyopathy (ALVC), left dominant ARVC, or arrhythmoge

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26

Wichter, Thomas, Peter Milberg, Henry D. Wichter, and Dirk G. Dechering. "Pregnancy in arrhythmogenic cardiomyopathy." Herzschrittmachertherapie + Elektrophysiologie 32, no. 2 (2021): 186–98. http://dx.doi.org/10.1007/s00399-021-00770-7.

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AbstractArrhythmogenic cardiomyopathy (AC) is a rare heart muscle disease with a genetic background and autosomal dominant mode of transmission. The clinical manifestation is characterized by ventricular arrhythmias (VA), heart failure (HF) and the risk of sudden cardiac death (SCD). Pregnancy in young female patients with AC represents a challenging condition for the life and family planning of young affected women. In addition to genetic mechanisms that influence the complex pathophysiology of AC, experimental and clinical data have confirmed the pathogenetic role of strenuous exercise and c

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27

Bhardwaj, Parveen, Minoo Sharma, and Neeraj Ganju. "Arrhythmogenic right ventricular cardiomyopathy." Nigerian Journal of Cardiology 12, no. 2 (2015): 142. http://dx.doi.org/10.4103/0189-7969.152035.

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28

Yu, Chao, Tian-Gang Zhu, Wen-Ling Liu, and Wen-Feng Huang. "Left-dominant Arrhythmogenic Cardiomyopathy." Chinese Medical Journal 129, no. 14 (2016): 1763–64. http://dx.doi.org/10.4103/0366-6999.185874.

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29

Bockeria, O. L., and T. G. Le. "Arrhythmogenic right ventricular dysplasia." Annaly aritmologii 12, no. 2 (2015): 89–99. http://dx.doi.org/10.15275/annaritmol.2015.2.4.

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30

Chaulin, A. M., and D. V. Duplyakov. "Arrhythmogenic effects of doxorubicin." Complex Issues of Cardiovascular Diseases 9, no. 3 (2020): 69–80. http://dx.doi.org/10.17802/2306-1278-2020-9-3-69-80.

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The article discusses the adverse arrhythmogenic effects of an antitumor drug – doxorubicin.Doxorubicin has a significant effect on the action potentials and ion currents of cardiomyocytes, the dynamics of intracellular calcium concentration.Oncological diseases are the leading causes of death and disability of the population, causing extremely high socio-economic damage. Among the many currently available drugs for the treatment of cancer, an important place is taken by the anthracycline antibiotic – doxorubicin. However, adverse concomitant effects on several organs and systems of the human

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31

McCullough, Jocelyn, and Rohan G. Perera. "Arrhythmogenic Right Ventricular Cardiomyopathy." New England Journal of Medicine 387, no. 10 (2022): e20. http://dx.doi.org/10.1056/nejmicm2119255.

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32

Jug, Juraj, and Martina Lovrić Benčić. "Arrhythmogenic right ventricular cardiomyopathy." Cardiologia Croatica 14, no. 1-2 (2019): 12–20. http://dx.doi.org/10.15836/ccar2019.12.

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33

Tseluyko, V. Y., and O. O. Butko. "Right ventricular arrhythmogenic cardiomyopathy." Medicine of Ukraine, no. 3(249) (May 7, 2021): 38–42. http://dx.doi.org/10.37987/1997-9894.2021.3(249).238044.

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The article describes the questions of prevalence, etiology and pathogenesis, clinical presentation, instrumental diagnostics of arrhythmogenic right ventricular cardiomyopathy (ARVC). Diagnostic criteria, sudden cardiac death risk stratification in patients with ARVC and basic approaches in the treatment of this disease are proposed.

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34

Grishina, N. V. "Arrhythmogenic right ventricular cardiomyopathy." PULMONOLOGIYA 32, no. 2 (2022): 47–53. http://dx.doi.org/10.18093/0869-0189-2022-32-2s-47-53.

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The article provides current information about the clinical manifestations and diagnosis of arrhythmogenic right ventricular cardiomyopathy, highlights the genetic aspects of the disease, and analyzes the recent scientific research. A clinical case of diagnosis of this rare disease is given.

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35

Krahn, Andrew D., Arthur A. M. Wilde, Hugh Calkins, et al. "Arrhythmogenic Right Ventricular Cardiomyopathy." JACC: Clinical Electrophysiology 8, no. 4 (2022): 533–53. http://dx.doi.org/10.1016/j.jacep.2021.12.002.

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36

Nogami, Akihiko. "Arrhythmogenic Right Ventricular Cardiomyopathy." Japanese Journal of Electrocardiology 34, no. 3 (2014): 245–63. http://dx.doi.org/10.5105/jse.34.245.

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37

Gallo, Cristina, Alessandro Blandino, Carla Giustetto, et al. "Arrhythmogenic right ventricular cardiomyopathy." Journal of Cardiovascular Medicine 17, no. 6 (2016): 418–24. http://dx.doi.org/10.2459/jcm.0000000000000354.

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38

Poloni, Giulia, Marzia De Bortoli, Martina Calore, Alessandra Rampazzo, and Alessandra Lorenzon. "Arrhythmogenic right-ventricular cardiomyopathy." Journal of Cardiovascular Medicine 17, no. 6 (2016): 399–407. http://dx.doi.org/10.2459/jcm.0000000000000385.

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39

Graziosi, Maddalena, and Claudio Rapezzi. "Right ventricular arrhythmogenic cardiomyopathy." Journal of Cardiovascular Medicine 18 (January 2017): e157-e160. http://dx.doi.org/10.2459/jcm.0000000000000470.

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40

Basso, Cristina, Domenico Corrado, Barbara Bauce, and Gaetano Thiene. "Arrhythmogenic Right Ventricular Cardiomyopathy." Circulation: Arrhythmia and Electrophysiology 5, no. 6 (2012): 1233–46. http://dx.doi.org/10.1161/circep.111.962035.

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41

Stoughton, Samuel Joseph. "Arrhythmogenic Right Ventricular Cardiomyopathy." Journal of Diagnostic Medical Sonography 25, no. 5 (2009): 263–66. http://dx.doi.org/10.1177/8756479309344098.

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42

Corrado, Domenico, Mark S. Link, and Hugh Calkins. "Arrhythmogenic Right Ventricular Cardiomyopathy." New England Journal of Medicine 376, no. 1 (2017): 61–72. http://dx.doi.org/10.1056/nejmra1509267.

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43

Dalal, Darshan, Khurram Nasir, Chandra Bomma, et al. "Arrhythmogenic Right Ventricular Dysplasia." Circulation 112, no. 25 (2005): 3823–32. http://dx.doi.org/10.1161/circulationaha.105.542266.

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44

HAMILTON, ROBERT M. "Arrhythmogenic Right Ventricular Cardiomyopathy." Pacing and Clinical Electrophysiology 32 (July 2009): S44—S51. http://dx.doi.org/10.1111/j.1540-8159.2009.02384.x.

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45

Johansson, Bengt, Christer Backman, Erik Tossavainen, and Michael Henein. "Unusual arrhythmogenic myocardial disease." International Cardiovascular Forum Journal 1, no. 4 (2015): 195. http://dx.doi.org/10.17987/icfj.v1i4.52.

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46

Krejčí, Jan. "Arrhythmogenic right ventricular cardiomyopathy." Cor et Vasa 52, no. 7-8 (2010): 405–8. http://dx.doi.org/10.33678/cor.2010.110.

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47

Widimský, Petr, and Rostislav Polášek. "Arrhythmogenic left ventricular dysplasia." Cor et Vasa 52, Suppl. (2010): 12–14. http://dx.doi.org/10.33678/cor.2010.197.

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48

Ferrari, Victor A., and Craig H. Scott. "Arrhythmogenic Right Ventricular Cardiomyopathy:." Journal of Cardiovascular Electrophysiology 14, no. 5 (2003): 483–84. http://dx.doi.org/10.1046/j.1540-8167.2003.03130.x.

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49

Huynh, Karina. "Ketogenesis in arrhythmogenic cardiomyopathy." Nature Reviews Cardiology 17, no. 5 (2020): 266. http://dx.doi.org/10.1038/s41569-020-0356-1.

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50

Fontaine, Guy. "Arrhythmogenic right ventricular dysplasia." Current Opinion in Cardiology 10, no. 1 (1995): 16–20. http://dx.doi.org/10.1097/00001573-199501000-00004.

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