Relevant bibliographies by topics / Arsenite de sodium

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Academic literature on the topic 'Arsenite de sodium'

Author: Grafiati
Published: 26 April 2025

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Journal articles on the topic "Arsenite de sodium"

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Muhammad, Aliyu, Oyeronke A. Odunola, Michael A. Gbadegesin, Abdullahi B. Sallau, Uche S. Ndidi, and Mohammed A. Ibrahim. "Inhibitory Effects of Sodium Arsenite and Acacia Honey on Acetylcholinesterase in Rats." International Journal of Alzheimer's Disease 2015 (2015): 1–7. http://dx.doi.org/10.1155/2015/903603.

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This study was conducted to investigate the effect of sodium arsenite and Acacia honey on acetylcholinesterase (AChE) activity and electrolytes in the brain and serum of Wistar rats. Male Wistar albino rats in four groups of five rats each were treated with distilled water, sodium arsenite (5 mg/kg body weight), Acacia honey (20% v/v), and sodium arsenite and Acacia honey, daily for one week. The sodium arsenite and Acacia honey significantlyP<0.05decreased AChE activity in the brain with the combined treatment being more potent. Furthermore, sodium arsenite and Acacia honey significantlyP<0.05decreased AChE activity in the serum. Strong correlation was observed between the sodium and calcium ion levels with acetylcholinesterase activity in the brain and serum. The gas chromatography mass spectrometry analysis of Acacia honey revealed the presence of a number of bioactive compounds such as phenolics, sugar derivatives, and fatty acids. These findings suggest that sodium arsenite and/or Acacia honey modulates acetylcholinesterase activities which may be explored in the management of Alzheimer’s diseases but this might be counteracted by the hepatotoxicity induced by arsenics.
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Shakya, Sangita, and B. Pradhan. "Isolation and Characterization of Arsenic Resistant Pseudomonas Stutzeri ASP3 for its Potential in Arsenic Resistance and Removal." Kathmandu University Journal of Science, Engineering and Technology 9, no. 1 (July 31, 2013): 48–59. http://dx.doi.org/10.3126/kuset.v9i1.63842.

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Inorganic arsenic both arsenite As (III) and arsenate As (V) constitute the highest toxicological risk associated with arsenic in drinking water. Novel methods are in demand for its removal from water, especially in rural areas. For this purpose, the potential of different microbes in arsenic resistance and removal from water has gained interests worldwide. This study investigates the arsenic resistance and removal capacity of a bacterial strain isolated from arsenic enriched water of Rautahat district in Nepal. The concentration of arsenic was by hydride generation atomic absorption spectrophotometer. The isolated bacterium showed high resistance to sodium arsenate up to 4,680.15 mg/l and sodium arsenite up to 649.55 mg/l. The bacterium also conferred multiple heavy metal resistance to zinc chloride (136.28 mg/l), copper sulphate (249.68 mg/l), mercuric chloride (5.43 mg/l) and silver nitrate (3.39 mg/l). The growth rate calculated in the presence of 129.01 mg/l of sodium arsenite was 0.35 h-1 with doubling time of 1.96 h. The strain showed growth in the range of 25–45 °C (optimum 30-35 °C), pH 6 – 9 (optimum 7.5-8.5) and tolerated up to 10% of NaCl. The PCR-based 16S rDNA sequence analysis revealed that the isolated As resistant bacterium is a close relative to P. stutzeri (99%) with 30 hits. The bacterium removed 82.97 % of As (V) and 49.4% of As (III) from culture medium amended with 200 mg/l sodium arsenate and 74.92 mg/l of sodium arsenite respectively.
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Divine Obichukwu Anakor and Kelechi Light Ekeke. "Effects of combined leaf extracts of Vernonia amygdalina and Ocimum gratissimum on biochemical parameters of sodium arsenite induced toxicity in albino Wister rat." World Journal of Advanced Research and Reviews 19, no. 2 (August 30, 2023): 669–74. http://dx.doi.org/10.30574/wjarr.2023.19.2.1624.

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This study was carried out to examine the effect of different leaf extract treatment on Sodium Arsenite toxicity in albino Wister rat model. Twenty-five (25) rats weighing between 120-270 g were used in this experiment and randomly divided into five (5) groups containing five rats each. Group 1 animals served as control and was administered placebo, while group 2 was induced with only sodium arsenite, 3 and 4 were both induced with sodium arsenite sand treated with Ocimum gratissimum ethanoic extract and Vernonia amygalina ethanoic extract respectively. Group 5 received concomitant administration of both extract after induction for 14 days. All drugs and extract administration were dose dependent on kilogram body weight using a cannula attached to a syringe. The result showed a significant elevation of sodium arsenite in group 2 serving as a biochemical marker defining sodium arsenide toxicity to living rat model tissue when compared to group 1. Group 5 showed no significant (p<0.05) difference when compared to group 1. Thus, showing an overall improvement in the effect of combined administration of the extract in the management of sodium arsenite level in sodium asenite induced toxicity when compared to groups 3 and 4 which may be associated with the phytochemicals present in both herbs.
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Bhatti, Zulfiqar Ali. "Removal of Arsenite and Arsenate by Indigenous Iron Ores of Pakistan." Pakistan Journal of Analytical & Environmental Chemistry 21, no. 2 (December 24, 2020): 293–302. http://dx.doi.org/10.21743/pjaec/2020.12.31.

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This study is focusing on the comparative study of arsenite and arsenate adsorption from the water via indigenous iron ores. The Sindh and Punjab provinces of Pakistan are badly affected by Arsenic (As) toxicity as the people are consuming arsenic contaminated groundwater. The aim of this study is to investigate the effect of anions on adsorption of arsenite As(III) and arsenate As(V). Impact of pH, contact time, adsorbent dose and shaking speed on adsorption of arsenite and arsenate is studied with the two selected iron ores from Hoshi and Shikarap from Balochistan. Hoshi and Shikarap ores exhibited higher As(III) and As (V) adsorption, respectively thus selected for further removal studies. Hoshi iron ore without sodium carbonate yields higher adsorption as compared to the samples with 100 mg/L and 1000 mg/L sodium carbonate in both As(III) and As(V). Hoshi ore exhibited the highest adsorption of 85% for As (V) without sodium phosphate dibasic and 83% for As(III). Shikarap ore for As(V) adsorbs 75% without sodium phosphate dibasic and 67% adsorption for As(III) without sodium phosphate dibasic. Shikarap ore with sodium silicate at 100 mg/L adsorbs 62% As(III) and at 1000 mg/L adsorb 52% As(III). Shikarap ore As(V) adsorption decreases from 75% without sodium silicate to 70% at 100 mg/L and even lower adsorption of 65% at a higher concentration of 1000 mg/L.
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Poudel, Pramod, Ashish Nepal, Rashmi Roka Magar, Pratibha Rauniyar, and Lil Buda Magar. "Screening of Potent Arsenic Resistant and Plant Growth Promoting Bacillus species from the Soil of Terai Region of Nepal." Tribhuvan University Journal of Microbiology 6 (December 6, 2019): 1–9. http://dx.doi.org/10.3126/tujm.v6i0.26572.

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Objectives: To isolate arsenic resistant Bacillus spp. and to determine plant growth promoting activities. Methods: Eighteen soil samples were collected from the agricultural soil of Terai region of Nepal. Selective isolation of Bacillus species was done by heating the soil at 80 ºC for 15 minutes before the isolation. Nutrient agar was used as an isolation medium. Screening of arsenic resistant Bacillus species was done using nutrient agar supplemented with 100 ppm sodium arsenate and sodium arsenite. For plant growth promoting activity; IAA production was detected taking 0.1% tryptophane and measuring absorbance at 540 nm, NH3 production was tested by Nessler’s reagent and phosphate solubilization activity was detected by growing colonies on Pikovskaya’s agar. Sugar assimilation test was performed to identify the isolates. Most potent arsenic resistant isolate was identified by 16S rRNA gene sequencing. Results: Among 54 randomly selected isolates, 42 were found to be Gram-positive rod-shaped, spore-forming while 12 isolates were Gram-negative bacteria. The isolates IN12a, M12a and BG34a showed growth on 100 ppm sodium arsenite containing NA. Only isolate M12a tolerated up to 1000 ppm and 15000 ppm of sodium arsenite and sodium arsenate respectively, while other isolates could not grow above 400 ppm sodium arsenite. The isolates IN12a and M12a were able to produce IAA and solubilize phosphate while BG34a could not. Both the isolates IN12a and M12a were able to utilize the sugars glucose, fructose, lactose, sucrose, galactose, mannose, mannitol, maltose and xylose. Based on the 16S rRNA gene sequencing, isolate M12a was identified to be Bacillus flexus with highest similarity of 99.2%. Conclusion: Arsenic resistant and plant growth promoting Bacillus spp. was isolated from the agricultural soil of Terai region of Nepal
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Chanda, Dipanjan, Sung-Jin Kim, In-Kyu Lee, Minho Shong, and Hueng-Sik Choi. "Sodium arsenite induces orphan nuclear receptor SHP gene expression via AMP-activated protein kinase to inhibit gluconeogenic enzyme gene expression." American Journal of Physiology-Endocrinology and Metabolism 295, no. 2 (August 2008): E368—E379. http://dx.doi.org/10.1152/ajpendo.00800.2007.

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Sodium arsenite has been demonstrated to alter the expression of genes associated with glucose homeostasis in tissues involved in the pathogenesis of type 2 diabetes; however, the underlying molecular mechanism has not been fully elucidated yet. In this study, we report that the sodium arsenite-induced gene expression of the small heterodimer partner (SHP; NR0B2), an atypical orphan nuclear receptor, regulates the expression of hepatic gluconeogenic genes. Sodium arsenite augments hepatic SHP mRNA levels in an AMP-activated protein kinase (AMPK)-dependent manner. Sodium arsenite activated AMPK and was shown to perturb cellular ATP levels. The arsenite-induced SHP mRNA level was blocked by adenoviral overexpression of dominant negative AMPK (Ad-dnAMPKα) or by the AMPK inhibitor compound C in hepatic cell lines. We demonstrated the dose-dependent induction of SHP mRNA levels by sodium arsenite and repressed the forskolin/dexamethasone-induced gene expression of the key hepatic gluconeogenic genes phosphoenolpyruvate carboxykinase (PEPCK) and glucose-6-phosphatase (G6Pase). Ad-dnAMPKα blocked the repressive effects of arsenite-induced SHP on PEPCK and G6Pase. Sodium arsenite inhibited the promoter activity of PEPCK and G6Pase, and this repression was abolished by small interfering (si)RNA SHP treatments. The knockdown of SHP expression by oligonucleotide siRNA SHP or adenoviral siRNA SHP released the sodium arsenite-mediated repression of forskolin/dexamethasone-stimulated PEPCK and G6Pase gene expression in a variety of hepatic cell lines. Results from our study suggest that sodium arsenite induces SHP via AMPK to inhibit the expression of hepatic gluconeogenic genes and also provide us with a novel molecular mechanism of arsenite-mediated regulation of hepatic glucose homeostasis.
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Ajit Kumar, Keshav Singh, Anand Pratap Singh, Sonal Singh, Prem Sagar, Shalini Yadav, Shekhar Biswas, and Sandeep Kumar. "A Critical Review on Hepatotoxicity in Albino Rats after Assessment of Sodium Arsenite." Journal of Science Innovations and Nature of Earth 4, no. 4 (December 30, 2024): 85–91. https://doi.org/10.59436/jsiane.306.2583-2093.

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In toxicological research, hepatotoxicity is a major worry, especially when looking at environmental contaminants like sodium arsenite. The health of people and animals is seriously endangered by sodium arsenite, a very poisonous substance that results from both natural and industrial processes and is found in air, water, and the soil. The hepatotoxic effects of sodium arsenite in Albino rats, a commonly utilized model organism for liver toxicity research, are extensively examined in this paper. With a focus on the consequences for the environment and public health, the paper summarizes previous research findings to clarify the impact of sodium arsenite on hepatic tissue in terms of biochemical, histological, and antioxidant indices. A detailed review of research indicates that sodium arsenite causes notable changes in indicators of liver function. Furthermore, exposure to sodium arsenite has been demonstrated to alter the liver histological architecture, resulting in inflammatory cell infiltration, sinusoidal dilatation, and hepatocyte destruction. The significance of dosage, exposure time, and delivery method in assessing the degree of hepatotoxic effects is also emphasized in this review. The administration methods, oral, intraperitoneal, or inhaled, have a major impact on sodium arsenite distribution and bioavailability, which in turn affects how hazardous it is. In conclusion, a great deal of research in albino rat models has shown that sodium arsenite is a serious hazard to liver health. We can more effectively handle the problems caused by this environmental toxin and protect the health of people and animals by improving our knowledge of sodium arsenite-induced hepatotoxicity.
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Alaniz-Andrade, Azucena Lucero, Consuelo Letechipía de León, Rosa María Ramírez-Santoyo, Jesús Guzmán-Moreno, and Luz Elena Vidales-Rodríguez. "Arsenic tolerance in bacterial cultures isolated from metal contaminated soil." Acta Universitaria 27, no. 3 (August 2, 2017): 9–18. http://dx.doi.org/10.15174/au.2017.1189.

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Several centuries of uninterrupted mining activities in Zacatecas, Mexico becomes a problem of soil pollution with toxic metals and metalloids as the arsenic. In this study, the arsenic-tolerance of ten bacterial isolates from a metal contaminated site were analyzed and high tolerance was observed in both solid (40 - 300 mM of sodium arseniate and 4 - 25 mM of sodium arsenite) and liquid media (7.2 and 11.3 mM arsenite). The arsenic tolerant isolates were identified by biochemical and 16S rRNA-encoding gene amplification analysis as members of the Bacillus, Micrococcus and Acinetobacter genus. A study of resistance to antibiotics revealed a high prevalence of resistance to beta-lactams and moderate prevalence to nitrofurantoin, vancomycin and ceftriaxone suggesting that antibiotic multiresistance of this isolates is probably related to arsenic tolerance throughout a plasmid or chromosomally encoded resistance mechanism.
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Parajuli, Pravin, Kuppan Gokulan, and Sangeeta Khare. "Preclinical In Vitro Model to Assess the Changes in Permeability and Cytotoxicity of Polarized Intestinal Epithelial Cells during Exposure Mimicking Oral or Intravenous Routes: An Example of Arsenite Exposure." International Journal of Molecular Sciences 23, no. 9 (April 27, 2022): 4851. http://dx.doi.org/10.3390/ijms23094851.

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The gastrointestinal tract (GIT) is exposed to xenobiotics, including drugs, through both: local (oral) and systemic routes. Despite the advances in drug discovery and in vitro pre-clinical models, there is a lack of appropriate translational models to distinguish the impact of these routes of exposure. Changes in intestinal permeability has been observed in different gastrointestinal and systemic diseases. This study utilized one such xenobiotic, arsenic, to which more than 200 million people around the globe are exposed via their food, drinking water, work environment, soil, and air. The purpose of this study was to establish an in vitro model to mimic gastrointestinal tract exposure to xenobiotics via oral or intravenous routes. To achieve this, we compared the route (mimicking oral and intravenous exposure to GIT and the dose response (using threshold approach) of trivalent and pentavalent inorganic arsenic species on the permeability of in vitro cultured polarized T84 cells, an example of intestinal epithelial cells. Arsenic treatment to polarized T84 cells via the apical and basolateral compartment of the trans-well system reflected oral or intravenous routes of exposure in vivo, respectively. Sodium arsenite, sodium arsenate, dimethyl arsenic acid sodium salt (DMAV), and disodium methyl arsonate hydrate (MMAV) were assessed for their effects on intestinal permeability by measuring the change in trans-epithelial electrical resistance (TEER) of T-84 cells. Polarized T-84 cells exposed to 12.8 µM of sodium arsenite from the basolateral side showed a marked reduction in TEER. Cytotoxicity of sodium arsenite, as measured by release of lactate dehydrogenase (LDH), was increased when cells were exposed via the basolateral side. The mRNA expression of genes related to cell junctions in T-84 cells was analyzed after exposure with sodium arsenite for 72 h. Changes in TEER correlated with mRNA expression of focal-adhesion-, tight-junction- and gap-junction-related genes (upregulation of Jam2, Itgb3 and Notch4 genes and downregulation of Cldn2, Cldn3, Gjb1, and Gjb2). Overall, exposure to sodium arsenite from the basolateral side was found to have a differential effect on monolayer permeability and on cell-junction-related genes as compared to apical exposure. Most importantly, this study established a preclinical human-relevant in vitro translational model to assess the changes in permeability and cytotoxicity during exposure, mimicking oral or intravenous routes.
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Bruez, Emilie, Philippe Larignon, Christophe Bertsch, Guillaume Robert-Siegwald, Marc-Henri Lebrun, Patrice Rey, and Florence Fontaine. "Impacts of Sodium Arsenite on Wood Microbiota of Esca-Diseased Grapevines." Journal of Fungi 7, no. 7 (June 22, 2021): 498. http://dx.doi.org/10.3390/jof7070498.

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Although sodium arsenite was widely used in Europe until its ban in 2003, its effects on microorganisms is not clearly understood. To improve our understanding of sodium arsenite curative effect on GTDs, grapevines displaying esca-foliar symptoms from different French regions (Alsace, Champagne, Languedoc) were treated or not with sodium arsenite, and analyzed for their wood microbiota. Using metabarcoding, we identified the fungal and bacterial taxa composition of microbiota colonizing woody trunk tissues. Large differences in fungal microbiota composition between treated and untreated grapevines were observed while no major impacts were observed on bacteria microbiota. The main fungal species detected in untreated necrotic woody tissues was Fomitiporia mediterranea (63–94%), a fungal pathogen associated with esca. The relative abundance of this fungal species significantly decreased after sodium arsenite treatment in the three vineyards, in particular in white-rot necrotic tissues and their borders (−90%). F. mediterranea was the most sensitive to sodium arsenite among fungi from grapevine woody tissues. These results strongly suggest that the effect of sodium arsenite on GTDs is due to its ability to efficiently and almost specifically eliminate F. mediterranea from white-rot necrotic tissues, allowing saprobic fungi to colonize the tissues previously occupied by this pathogenic fungus.
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Dissertations / Theses on the topic "Arsenite de sodium"

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Pereira, Flavia Elias. "Role of Sodium Arsenite in Atherogenesis." The University of Montana, 2007. http://etd.lib.umt.edu/theses/available/etd-12282007-163850/.

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Epidemiological studies as well as controlled animal studies have associated exposure to arsenic through drinking water with the development of atherosclerosis. In this study, we have shown for the first time that low and environmentally relevant concentrations of arsenic accelerate atherogenesis. The objective of this study was to elucidate the mechanisms of arsenic-induced atherosclerosis by (1) characterizing the time- and concentration-dependent effects of sodium arsenite [As(III)] on the development of atherosclerosis in ApoE-/- /LDLr-/- mice, (2) determining whether As(III)-induced peroxynitrite activates protein kinase C (PKC) isotypes, α and β, in human aortic endothelial cells (HAECs) and (3) determining the effects of activation of PKC isotypes, α and β, on the endothelial barrier. Accordingly, exposure of ApoE-/- /LDLr-/- mice to As(III) in drinking water showed an increasing trend in atherosclerotic plaque formation in as early as 5 weeks within the innominate arteries. Most remarkable was the evidence that environmentally relevant concentrations of As(III) resulted in significant increase in plaque formation. Initiation of atherosclerosis results from activation/dysfunction of the vascular endothelium that maintains a semipermeable barrier between the blood and vessel wall. To elucidate the mechanism of arsenic-induced atherosclerosis, we analyzed the effect of As(III) on the endothelial monolayer integrity. Endothelial barrier is maintained by proteins of the adherens junction (AJ) such as vascular endothelial cadherin (VE-cadherin) and β-catenin, and their association with the actin cytoskeleton. Treatment of HAECs with As(III) resulted in reorganization of actin filaments into stress fibers and non-uniform VE-cadherin and β-catenin staining at cell-cell junctions. Intercellular gaps were observed with a measured increase in endothelial permeability. In addition, an increase in tyrosine phosphorylation (PY) of β-catenin was observed. These effects were mediated through As(III)-induced activation of PKCα without peroxynitrite formation. No change in PKCβ levels was detected with As(III) treatment. Inhibition of PKCα restored VE-cadherin and β-catenin staining at cell-cell junctions and abolished the formation of intercellular gaps and stress fibers. Endothelial permeability and PY of β-catenin were also reduced to basal levels. These results demonstrate that As(III) induced activation of PKCα causes PY of β-catenin and formation of stress fibers. PY of β-catenin causes weakening of the AJ and this in association with the contractile force generated by stress fibers results in gap formation and increased endothelial permeability. This could potentially accelerate the development of atherosclerosis by increasing the accumulation of oxidized low density lipoproteins and monocytes into the neo-intima of the blood vessel. The findings in this study demonstrate that arsenic disrupts the endothelial monolayer by activation of PKC signaling. Damage to the endothelium plausibly accelerates the process of atherosclerosis at an early stage as evidenced by the increase in atherosclerotic plaques in the ApoE-/- /LDLr-/- mouse model.
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Cox, David Paul. "Disruption of 8-hydroxy-2'-deoxyguanosine DNA Glycosylase (OGG1) Antioxidant Response Capacity by Sodium Arsenite." The University of Montana, 2008. http://etd.lib.umt.edu/theses/available/etd-05272008-134949/.

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8-hydroxy-2'-deoxyguanosine DNA glycosylase is the first step and rate-limiting enzyme involved in the removal of 8-hydroxy-2'-deoxyguanosine via the base excision repair pathway. Transcriptional regulation of human Ogg1 is sensitive to redox changes via modulation of intracellular glutathione. In response to changes in glutathione, changes in hOgg1 transcription occur similar to genes regulated by the transcription factor Nrf2. It was determined that positions - 47 to - 44 in the hOgg1 promoter are necessary for basal transcription of Ogg1 determined by site-directed deletion. This region is capable of interacting with nuclear protein determined by binding assays. Furthermore, transcription factor Nrf2 is identified as binding to this region determined by parallel, and competition EMSA binding assays. Exposure to arsenic has also been associated with oxidative stress and damage to DNA, specifically oxo8dG. This study identified significant increases in the cellular antioxidant glutathione, and alterations in superoxide dismutase activities subsequent to arsenite exposure in actively dividing and NGF treated PC12 cells. Assessment of Ogg1 activity and mRNA levels demonstrated a significant decrease for both measures subsequent to arsenite exposure. The effect seen was due in large part to alterations in gene transcription since direct testing revealed no effect by arsenite on Ogg1 activity. Levels of oxo8dG did not significantly change subsequent to arsenite exposure, however increased trends were evident. Characterization of Sp1 binding revealed that treatment with sodium arsenite could decrease Sp1 binding at two unique Sp1 sites in the human Ogg1 promoter. In summary, transcription factor Nrf2 is an important factor in the inducible regulation of Ogg1. Transcriptional changes in Ogg1 are further dependent on the redox status of the cell. Despite the role of Nrf2 in response to oxidative stress, sodium arsenite disrupted both the transcription and activity of Ogg1 in PC12 cells. This disruption occurred despite the induction of cellular stress response via increases in GSH and Mn SOD activity. This suggests that arsenite is acting through other mechanisms potentially through disruption of the Sp1 transcription factor.
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Mesguida, Ouiza. "Biocontrôle d’un champignon pathogène, Fomitiporia mediterranea, impliqué dans l'Esca, une maladie dévastatrice du bois de la vigne, en utilisant des bactéries isolées de Vitis vinifera." Electronic Thesis or Diss., Pau, 2024. http://www.theses.fr/2024PAUU3054.

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Les maladies du bois de la vigne (MDBs), notamment l'Esca, constituent un enjeu majeur pour la viticulture mondiale. Ces MDBs réduisent la durée de vie des vignobles, impactent la qualité des raisins et donc du vin, entraînant des pertes de revenus estimées à un milliard d'euros par an en France. Aucun traitement curatif n'est disponible depuis l'interdiction de l'arsénite de sodium au début des années 2000, le dernier pesticide chimique homologué pour contrôler les MDBs en Europe. Le développement de méthodes alternatives pour gérer les MDBs, telles que le biocontrôle, est par conséquent de la plus haute importance. Notre objectif a été de sélectionner des bactéries de biocontrôle capables d’inhiber Fomitiporia mediterranea (Fmed), l'un des principaux champignons pathogènes de l'Esca. Ce champignon dégrade le bois de la vigne, conduisant à la formation de la pourriture blanche dans le bois, l’amadou, un symptôme clé de l'Esca, où s'accumulait l'arsénite de sodium. Nos objectifs ont été d’identifier les mécanismes d'action des bactéries sélectionnées, puis d'évaluer leur efficacité au vignoble. Un criblage de 58 souches bactériennes isolées de la vigne a été réalisé in vitro afin d’évaluer leur capacité à inhiber la croissance de six souches de Fmed, via la production de métabolites volatils et diffusibles. Un second criblage a été effectué en microcosme, constitué de sciure de bois de vigne provenant de sept cépages. In vitro, les composés volatils de 51 des 58 souches ont montré une efficacité élevée pour inhiber la croissance de Fmed (>50%). Concernant les métabolites diffusibles, huit souches avaient un niveau d’efficacité similaire. En microcosme, les souches Pseudomonas lactis SV9, Pseudomonas paracarnis S45 et Paenibacillus polymyxa SV13 ont également montré une forte efficacité à inhiber Fmed, elle était cependant dépendante du cépage. Les génomes des trois souches ont été analysés, ainsi que leurs métabolomes par LC-MS/MS pour les composés diffusibles, et par SPME GC-MS pour les volatils. Paenibacillus polymyxa SV13 a inhibé la croissance de Fmed via la production de métabolites diffusibles, tandis que les deux souches de Pseudomonas ont agi principalement via leurs métabolites volatils. Parmi les composés diffusibles produits par P. polymyxa SV13 en présence de Fmed, des molécules de type fusaricidine ayant une activité antifongique ont été identifiés. Pour les métabolites volatils bactériens, les deux souches de Pseudomonas ont produit du disulfure de diméthyle, également connu comme étant un composé antifongique. Les analyses génomiques des trois souches bactériennes ont révélé des groupes de gènes responsables de la régulation des mécanismes d’antagonisme directs et indirects. Pour comprendre le mode d'action de l'arsénite de sodium sur Fmed, le transcriptome du champignon a été examiné lors de son interaction avec ce produit, ouvrant la voie à de futures analyses transcriptomiques lors de l'interaction entre Fmed et les bactéries. Le potentiel de biocontrôle de P. paracarnis S45 et P. polymyxa SV13 a été évalué lors d'un essai au vignoble (cépage Sauvignon Blanc) où ces bactéries ont été injectées dans les troncs de ceps de vigne symptomatiques. Une réduction de la sévérité de l'Esca suite à ces injections a été observée. L'efficacité du traitement variait selon le type de bois injecté, la plus grande efficacité étant observée lors d’injection dans l’amadou. Des analyses de la physiologie de la vigne, de la qualité des baies et du microbiome du bois ont également été réalisées, suite à l’injection de deux bactéries dans le tronc des ceps. Nos résultats soulignent l’importance d’études combinant la sélection d’agents de biocontrôle in vitro, la détermination de leurs modes d’action et l’évaluation de leur efficacité au champ. De futures études pourraient examiner différents modes d’application, et tester différentes formulations d’agents de biocontrôle afin d’améliorer l’efficacité de la protection au vignoble
Grapevine trunk diseases (GTDs), particularly Esca, are a major challenge for viticulture worldwide. GTDs decrease the profitable lifespan of vineyards and affect berry and wine quality, leading to an estimated one billion euros in lost revenues annually in France. No curative control treatments are available since the ban of sodium arsenate in the early 2000s, the last chemical pesticide registered to control Esca in Europe. As a consequence, development of alternative methods to manage Esca, such as biological control, has become essential. Our aim was to select bacterial biological control agents (BCAs) effective against one of the main pathogenic fungi of Esca, Fomitiporia mediterranea (Fmed). This fungus degrades grapevine wood acting as a white rot pathogen. White rot is a key symptom of Esca in grapevine wood, where sodium arsenate accumulated after grapevine treatment. Our objectives were to explore the mechanisms of action of selected bacterial strains against Fmed, and subsequently assess their effectiveness in field conditions. A stepwise screening of 58 bacterial strains isolated from grapevine was carried out in vitro for their ability to inhibit the growth of six Fmed strains, through the production of volatile and agar-diffusible metabolites. A second screening was performed on microcosm, made of wood sawdust from seven grapevine cultivars. Fifty-one bacterial strains out of 58 strains tested had high efficacy in inhibiting the growth of Fmed (>50%) through volatile compounds, while eight bacterial strains exhibited strong antifungal efficacy through the production of agar-diffusible metabolites. The strains Pseudomonas lactis SV9, Pseudomonas paracarnis S45, and Paenibacillus polymyxa SV13 demonstrated strong efficacy in inhibiting Fmed in microcosms, in a cultivar-dependent manner. The whole genomes of the three strains were analyzed along with their metabolomes (LC-MS/MS for diffusible compounds and SPME GC-MS for volatile compounds). Paenibacillus polymyxa SV13 inhibited Fmed growth mainly via the production of diffusible metabolites, while the two Pseudomonas strains acted mainly via their volatile metabolites. Among the bacterial diffusible compounds, P. polymyxa SV13 produced mainly fusaricidin-type compounds in the presence of Fmed, compounds known for their antifungal activity. Regarding bacterial volatiles, both Pseudomonas strains produced dimethyl disulfide, which is also known as an antifungal molecule. Genome analyses of the three bacterial strains revealed gene clusters responsible for regulating both direct and indirect mechanisms of action in BCAs. To understand the mode of action of sodium arsenite on Fmed, the transcriptome of the fungus was examined while interacting with this chemical compound, hence paving the way for future transcriptomic analyses during the pathogen interaction with the bacterial BCAs. The biocontrol potential of the two strains P. paracarnis S45 and P. polymyxa SV13 was evaluated in a vineyard trial, by injecting them into the trunks of Esca-symptomatic grapevines of the cultivar Sauvignon Blanc. This study demonstrated a reduction in Esca severity after trunk injections of P. paracarnis S45 or P. polymyxa SV13. Treatment efficacy varied depending on the type of wood tissue injected, with the greatest efficacy observed when injected into white-rot. Analyses of grapevine physiology, berry quality and wood microbiome were also carried out, following the injection of the two bacteria into the trunk of the grapevines. Our results highlight the importance of studies combining the selection of biological control agents in vitro, deciphering their modes of action and the evaluation of their efficacy in the field. Future research could focus on optimizing application methods and refining biocontrol formulations to further enhance their effectiveness
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Aquino, Ariana Musa. "Efeito da exposição pré-puberal ao arsênio sobre parâmetros morfofuncionais na próstata ventral de ratos pubescentes." Botucatu, 2019. http://hdl.handle.net/11449/181437.

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Orientador: Wellerson Rodrigo Scarano
Resumo: O arsênio é um metaloide associado ao desenvolvimento de algumas patologias, como doenças cardiovasculares, lesões dérmicas e diferentes tipos de câncer. Pouco se sabe sobre a ação do arsênio ou compostos de arsênio na próstata durante o período pré-puberal e puberdade, estágios essenciais para a morfogênese tardia da próstata. Nesse sentido, este estudo teve como objetivo estabelecer se a exposição ao arsenito de sódio (NaAsO2) interfere na morfofisiologia da próstata ventral de ratos púberes. Para isso, 30 ratos machos da linhagem Wistar, no dia pós-natal 23 (DPN23), foram distribuídos, aleatoriamente, em 3 grupos experimentais (n =10/grupo). O grupo controle (Ctrl) recebeu água filtrada (veículo); o grupo As1 recebeu 0.01 mg/L de NaAsO2; e o grupo As2 recebeu 10.0 mg/L de NaAsO2. Todas as soluções foram diluídas na água do bebedouro e estiveram disponíveis aos animais do DPN23 ao DPN53. Os hábitos alimentares e a evolução do peso corpóreo dos animais foram acompanhados durante todo o período experimental. Ao final deste período, os animais foram pesados e, em seguida, eutanasiados (DPN53). Coletou-se o sangue para mensurar os níveis de testosterona. O fígado, os rins e a próstata ventral (PV) foram coletados e pesados. Apenas a PV foi dissecada e destinada às análises histológicas (hemilobo esquerdo) e moleculares (hemilobo direito). Os resultados dos parâmetros analisados durante o período experimental revelaram que o NaAsO2 não foi capaz de causar toxicidade sistêmica em... (Resumo completo, clicar acesso eletrônico abaixo)
Abstract: Arsenic is an endocrine disruptor associated with the development of some pathologies such as cardiovascular diseases, dermal lesions and different types of cancer. Little is known about the action of arsenic or arsenic compounds in the prostate during the prepubertal and puberty period, an essential stage for late morphogenesis of the prostate. Therefore, this study aimed to establish whether exposure to sodium arsenite (NaAsO2) interferes in the morphophysiology of the ventral prostate of pubertal rats. In this study, thirty male Wistar rats, on the postnatal day 23 (PND23), were randomly distributed to 3 experimental groups (n = 10/group). The control group (Ctrl) received only saline solution; the second group (As1) received 0.01 mg/L of NaAsO2; and the third group (As2) received 10.0 mg/L of NaAsO2. All solutions were diluted in drinking water and were available to the animals from DPN23 to DPN53. The eating habits and the evolution of the body weight of the animals were evaluated throughout the experimental period. At the end of this period, the animals were weighed and then euthanized (DPN53). Blood was collected to measure testosterone levels. The liver, kidneys and ventral prostate (VP) were collected and weighed. Only VP was dissected for histological analysis (left hemilobo) and molecular (right hemilobo). The results of the parameters analyzed during the experimental period revealed that NaAsO2 was not able to cause systemic toxicity in both exposed groups nor cha... (Complete abstract click electronic access below)
Mestre
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Steffens, Amanda Ann. "Low-dose of sodium arsenite causes delayed differentiation in C2C12 mouse myoblast cells through the repression of the transcription factor myogenin." Connect to this title online, 2009. http://etd.lib.clemson.edu/documents/1263410069/.

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Winski, Shannon Lee 1967. "Metabolism and toxicity of sodium arsenate in human erythrocytes." Diss., The University of Arizona, 1997. http://hdl.handle.net/10150/282320.

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Toxicity of arsenic species is dependent on chemical oxidation state. Inorganic arsenic in the trivalent state, arsenite or As(III), is more biologically active than pentavalent arsenic, arsenate or As(V), and is more toxic by most measures. As(V), however, is more stable and prevalent in the environment. One consequence of environmental exposure is peripheral vascular disease, which is primarily due to vascular changes, but toxicity to the erythrocyte has not been evaluated. To understand toxicity and the implications of arsenic oxidation state, human erythrocytes were utilized to model the uptake, biotransformation (metabolism) and toxicity of sodium arsenate, As(V). It was first established that biotransformation, both in vivo and in vitro, would not be limited by uptake of As(V) into the cell. Evidence suggested that reduction was accomplished by at least two separate pathways. All reductive metabolism was dependent on the presence of reduced thiols including both non-protein thiols (glutathione; GSH) and protein thiols (ProSH). These pathways are: (1) chemically mediated reduction by GSH and (2) protein mediated reduction. It was established that the protein-dependent pathway required a reduced protein thiol and also required the presence of GSH. This points to reduction through a redox coupling to form a protein mixed disulfide (ProSSG). Toxicity to the erythrocyte was evaluated by determining total cell death, morphologic changes and effects on the energy cofactor adenosine triphosphate (ATP). Based on these three parameters, the erythrocyte was more susceptible to As(V) and not As(III) as other tissues are. The morphologic effects on the cell were also consistent with ATP depletion. These changes were characterized by formation of morphologically altered cells that are unable to deform in circulation effectively and occlude the microcirculation. This could contribute to vascular tissue damage associated with arsenic-induced circulatory disorders. In summary, the erythrocyte is able to take in As(V) which is detrimental to the ability of the cell to perform its intended function. Biotransformation to As(III) would therefore be a detoxifying event, and understanding the factors involved in biotransformation will help to understand human susceptibility to arsenic-induced vascular disease.
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Li, Wen. "Synthesis and solubility of arsenic tri-sulfide and sodium arsenic oxy-sulfide complexes in alkaline sulfide solutions." Thesis, University of British Columbia, 2013. http://hdl.handle.net/2429/44546.

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Alkaline sulfide leaching (ASL) at approximately 100 ºC has been used to selectively extract arsenic and antimony from enargite and tetrahedrite concentrates. Sodium thio-arsenate has been postulated to crystallize from alkaline sulfide leaching solutions upon cooling. However, literature data on the solubility of sodium thio-arsenate as well as proof of its crystallization from ASL solutions is scant. In this thesis, the solubility of leach-produced and synthetic sodium thio-arsenate is studied. To determine arsenic solubility in ASL solutions, sodium thio-arsenate and sodium arsenic oxide sulfide complexes are synthesized by various means and characterized by EDX, QXRD, and ICP. The synthesis of amorphous As₂S₃, sodium arsenic oxy-sulfide complexes, and sodium thio-arsenate is first presented. For amorphous As₂S₃ synthesis, the effect of concentration of sodium sulfide (0.1 M) and hydrochloric acid (1 M), temperature (40 ~ 60 ºC), and aging time (48 hours) was optimized. The solubility of synthetic sodium arsenic oxy-sulfide complexes and sodium thio-arsenate in ASL solutions increases significantly as temperature is increased to 95 ºC. More importantly, the solubility of sodium thio-arsenate at certain temperatures is significantly affected by the concentration of sodium hydroxide and sulfide in solution. Due to the common ion effect, if NaOH and HS- concentrations are very high, the solubility of sodium thio-arsenate decreases. Enargite leaching tests were done to characterize the precipitate that occurred upon cooling and to verify the arsenic saturation point, which should be between 38.5 ~ 58 g/L (0.51 M ~ 0.78 M) As depending on the NaOH and HS- concentration. Comparison with solubility experiments of pure sodium thio-arsenate shows that arsenic solubility in ASL solutions is supersaturated. However, direct comparison of saturation in ASL solutions and the solubility as obtained by the synthetic solutions/crystallites prepared here is not possible given the complex nature of the ASL crystallites that appear not to contain the often discussed “sodium thio-arsenate”.
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OLIVEIRA, GLEN A. M. de. "Estudo comparativo do efeito analgésico do laser em baixa intensidade de emissão infravermelha e da pasta de fluoreto de sódio a 33 porcento no tratamento da hipersensibilidade dentinária." reponame:Repositório Institucional do IPEN, 2003. http://repositorio.ipen.br:8080/xmlui/handle/123456789/11335.

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Dissertacao (Mestrado Profissionalizante em Lasers em Odontologia)
IPEN/D-MPLO
Intituto de Pesquisas Energeticas e Nucleares, IPEN/CNEN-SP; Faculdade de Odontologia, Universidade de Sao Paulo
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Pickering, Edmund Ian Marcus. "Mechanical characterisation of nanowires through resonance techniques." Thesis, Queensland University of Technology, 2018. https://eprints.qut.edu.au/123008/1/Edmund_Pickering_Thesis.pdf.

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This thesis uses the mechanical resonance technique to investigate and characterise the mechanical behaviour of nanowires. While previous work has mainly focused on simple, uniform nanowires, this thesis extends the resonance technique to incorporate more complex morphologies. Specifically, tapered nanowires with surface effects and curved nanowires with irregular cross-sections were investigated through experiments and modelling. This works will aid in advancing the pace of nanowire development by extending the resonance technique to describe such nanowire morphologies.
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AVEGLIANO, ROSEANE P. "Estudo de dieta total no estado de Sao Paulo: estimativa de ingestao dietetica de elementos toxicos (arsenico e cadmio) e essenciais (calcio, cromo, ferro, selenio, sodio, potassio e zinco)." reponame:Repositório Institucional do IPEN, 2009. http://repositorio.ipen.br:8080/xmlui/handle/123456789/11523.

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Tese (Doutoramento)
IPEN/T
Instituto de Pesquisas Energeticas e Nucleares - IPEN-CNEN/SP
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Books on the topic "Arsenite de sodium"

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Malaysia. Poisons Act 1952: Poisons regulations 1952 ; Poisons (Sodium Arsenite) Ordinance and regulations 1949 ; Poisons (Sodium Hydroxide) regulations 1962 ; Poisons (Psychotropic substances) regulations 1989, with index : all amendments up to May 2004 : Act 366. 8th ed. Kuala Lumpur: MDC Publishers, 2004.

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Malaysia. Poisons Act 1952: Poisons regulations 1952 ; Poisons (Sodium Arsenite) Ordinance and regulations 1949 ; Poisons (Sodium Hydroxide) regulations 1962 ; Poisons (Psychotropic substances) regulations 1989, with index : all amendments up to August 1998 : Act 366. 4th ed. Kuala Lumpur: Published & printed by MDC Publishers Printers, 1998.

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Nelson, Morales, and United States. National Aeronautics and Space Administration., eds. Solar-electrochemical power system for a Mars mission. [Washington, DC]: National Aeronautics and Space Administration, 1994.

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Nelson, Morales, and United States. National Aeronautics and Space Administration., eds. Solar-electrochemical power system for a Mars mission. [Washington, DC]: National Aeronautics and Space Administration, 1994.

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Nelson, Morales, and United States. National Aeronautics and Space Administration., eds. Solar-electrochemical power system for a Mars mission. [Washington, DC]: National Aeronautics and Space Administration, 1994.

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Tice, Raymond, Glenda Moser, and Thomas Goldsworthy. Sodium Arsenite Studies in P53+/-Mice. Amer Water Works Assn, 2000.

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C. S. (Charles S. ) Walters, K. R. (Kenneth R. ) Peterson, and W. L. (Wayne Lewis) Meek. Barking Black Oak and Jack Pine Fence Posts with Sodium Arsenite. Creative Media Partners, LLC, 2021.

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Solar-electrochemical power system for a Mars mission. [Washington, DC]: National Aeronautics and Space Administration, 1994.

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Book chapters on the topic "Arsenite de sodium"

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Katsifis, Spiros P., and Patrick L. Kinney. "Antagonistic Interaction of Sodium Arsenite and Lead Sulfate with UV Light on Sister Chromatid Exchanges in Human Peripheral Lymphocytes." In Toxicity Assessment Alternatives, 53–61. Totowa, NJ: Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-718-5_5.

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Predel, B. "As-Na (Arsenic - Sodium)." In Ac-Ag ... Au-Zr, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2006. http://dx.doi.org/10.1007/10793176_185.

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Sokolowski, M., and E. Bilger. "Destruction of Adamsite by Sodium." In Arsenic and Old Mustard: Chemical Problems in the Destruction of Old Arsenical and ‘Mustard’ Munitions, 157–58. Dordrecht: Springer Netherlands, 1998. http://dx.doi.org/10.1007/978-94-015-9115-7_13.

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"Sodium Arsenite." In Encyclopedia of Metalloproteins, 2077. New York, NY: Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-1533-6_101177.

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Hightower, L. E., J. A. Ryan, and C. E. Norris. "Fish HSP70 proteins." In Guidebook to Molecular Chaperones and Protein-Folding Catalysts, 47–48. Oxford University PressOxford, 1997. http://dx.doi.org/10.1093/oso/9780198599494.003.0017.

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Abstract Fish Hsp70s were originally identified as proteins induced in cultured fish cells by exposure to heat, heavy metal ions or sodium arsenite. Initial biochemical characterizations were done using denaturing polyacrylamide gel electrophoresis (Heikkila et al., 1982; Kothary, Candido, 1982).
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Ramírez, P., L. Del Razo, and M. Gonsebatt. "Mouse liver cytokeratin 18 (CK18) modulation by sodium arsenite." In Arsenic in the Environment, 467–72. CRC Press, 2008. http://dx.doi.org/10.1201/b11334-60.

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Palmieri, M., and B. Molinari. "Effect of sodium arsenite in a model of experimental carcinogenesis in mouse skin." In Arsenic in the Environment - Proceedings, 634–36. CRC Press, 2014. http://dx.doi.org/10.1201/b16767-235.

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Okoji, Russell S., Joel Leininger, and John R. Froines. "Subchronic Toxicity Study of Sodium Arsenite in Methyl-Deficient Male C57BL/6 Mice." In Arsenic Exposure and Health Effects III, 225–32. Elsevier, 1999. http://dx.doi.org/10.1016/b978-008043648-7/50026-1.

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"- PARAMETERS OF MOBILITY FOR POLLUTION BY SODIUM ARSENITE FOR SOIL OF KAMBARKA DISTRICT." In Multifunctional Materials and Modeling, 234–41. Apple Academic Press, 2015. http://dx.doi.org/10.1201/b18552-30.

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Huang, H., S. H. Tyan, and S. G. Lin. "THE EFFECT OF SODIUM ARSENITE ON THE DNA REPAIR IN GAMMA-IRRADIATED MAMMALIAN CELLS." In Radiation Research: A Twentieth-century Perspective, 359. Elsevier, 1991. http://dx.doi.org/10.1016/b978-0-12-168561-4.51191-4.

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Conference papers on the topic "Arsenite de sodium"

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Perveen, Hasina, Vertika Rai, Dipankar Das, Debayan Chakraborty, Suchismita Mukherjee, Subhayan Das, Aishwarya Saha, Sangram Bhattacharyya, and Rupkatha Baidya. "Ameliorative effect of Curcumin on sodium arsenite-induced uterine-ovarian toxicity in rats: An in vivo dose dependent manner." In 2024 15th International Conference on Computing Communication and Networking Technologies (ICCCNT), 1–6. IEEE, 2024. http://dx.doi.org/10.1109/icccnt61001.2024.10724565.

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Wang, Dapeng, Liming Zhang, Jian Li, Jian Liu, Xing Liu, Huanyu Jin, Chunyan Ji, Chunling Fu, and Yan An. "Disposition of arsenic in saliva, blood and urine of Sprague-Dawley rats following repeated oral exposure to sodium arsenite." In 2011 International Conference on Human Health and Biomedical Engineering (HHBE). IEEE, 2011. http://dx.doi.org/10.1109/hhbe.2011.6029031.

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Adegboyega, Adenike M. "Abstract 3083: Ameliorative effect of ethanolic extract ofAnnona muricataagainst sodium arsenite-induced hepatotoxicty in Wistar rats." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-3083.

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Owumi, Solomon E., Oyeronke A. Odunola, Michael A. Gbadegesin, Kathleen L. Nulah, Aliyu Mohammed, Cinzia O. Oloye, Abiodun C. Oloidi, Moses O. Obadare, and Oludare O. Oladeji. "Abstract B61: Chemoprotective properties of some African spices on sodium arsenite-induced toxicities in albino Wistar rats." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Nov 7-10, 2010; Philadelphia, PA. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1940-6207.prev-10-b61.

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Akinwumi, Kazeem Akinyinka, Olabode Olatunbosun Osifeso, Olajumoke Jumoke Aboyewa, and Oyeronke Aduni Odunola. "Abstract B21: Potassium dichromate and sodium arsenite toxicities: Modification by methanol extract of Rauvolfia vomitoria in albino mice." In Abstracts: AACR International Conference: New Frontiers in Cancer Research; January 18-22, 2017; Cape Town, South Africa. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.newfront17-b21.

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Min, Young Joo, Hee-Soon Lee, Wha Ja Cho, Ho Yong Lee, Jong Cheol Lee, Sungchan Park, Hee-jung Cha, et al. "Abstract 2435: Assessment of vascular disrupting properties of sodium meta-arsenite treatment using Dynamic contrast-enhanced MRI in colon cancer allograft model." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2435.

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Dararatana, Naruphorn, Matina Thammachart, Suchada Punpruk, Kornrawee Srisawat, Nattawut Yotapan, Pimpa Limthongkul, Amnuaysak Chianpairot, and Korakot Sombatmankhong. "Arsenic Decontamination: Exploring the Efficacy of Sodium Hydroxide in Altering Arsenic Solubility and Ensuring System Compatibility." In APOGCE 2024. SPE, 2024. http://dx.doi.org/10.2118/221119-ms.

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Abstract Oil and natural gas reservoirs in the Gulf of Thailand are contaminated with mercury and arsenic. The arsenic contamination not only raises concerns related to health and safety but also significantly impacts the overall quality and market value of the crude product. The conventional filtration effectively removes only insoluble arsenic contaminants in the condensate, but not soluble arsenic species. Thus, posing the challenge of soluble arsenic removal by alternative approaches. This study aims to investigate the feasibility of employing sodium hydroxide (NaOH) as an active reagent in chemical treatment process particularly for removing soluble arsenic contaminants from condensate. The experimental setup was designed to simulate the real operating conditions in the 1st and 2nd stage production separators in a simplified manner. This study involved selecting 100% and 50% condensate-to-produced water ratios and adjusting the reaction time for each condition to mimic real-life scenarios. A wide range of NaOH concentrations from 0-550 ppm was introduced in the experimentation process. The efficiency of arsenic removal was assessed by comparing the arsenic levels in samples before and after treated with NaOH. A comprehensive crude specification was conducted for quality evaluation. Furthermore, this study encompassed a compatibility test evaluating the interaction of NaOH with demulsifiers discern inhibitory effects on the chemicals employed. An immersion corrosion test was conducted on carbon steel and corrosion-resistant alloys (CRA) to verify the corrosion compatibility of NaOH with the materials constituting the system. The predominant arsenic species in field A comprise triphenylarsine and triethylarsine which are soluble in condensate. The concentration screening test for suitable NaOH dosage demonstrated that higher NaOH dosage promoted higher arsenic removal efficiency. With the optimal NaOH dosage at 550 ppm, the arsenic removal efficiency ranged from 50% to 65% were obtained for both 100% and 50% condensate-in-produced water. The addition of NaOH increased transformed of arsenic to water-soluble forms. This led to a decrease in arsenic concentration detected in the condensate phase while increasing in the water phase. The adsorbent flow testing with NaOH-treated condensate indicated that the selected NaOH dosage did not adversely affect demulsifier activities. Additionally, crude specification confirmed a slight decrease in total chloride ion, while other crude properties exhibited no observable changes. Corrosion test results showed that the average corrosion rate of carbon steel in the condensate treated with NaOH was lower than the non-treated system after immersion for 14 days. No corrosion product was observed on CRA after 45 days of immersion testing in both NaOH-treated and non-treated conditions. This study elucidates the arsenic decontamination process by altering its solubility in condensate using an alkaline chemical. The investigation provides a comprehensive feasibility analysis addressing a practical application concern encompassing the compatibility, integrity, and safety of the NaOH solution within the operational framework of the system.
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Spayd, Steve, Yelena Stroiteleva, John Dooley, Brady L. Lubenow, Megan Rockafellow-Baldoni, Rachel Filo, Nicholas A. Procopio, Greg Herman, and Jessie A. Gleason. "ELEVATED BORON ASSOCIATED WITH LITHIUM, SODIUM, SULFATE, AND ARSENIC IN NEWARK BASIN PRIVATE WELLS." In 54th Annual GSA Northeastern Section Meeting - 2019. Geological Society of America, 2019. http://dx.doi.org/10.1130/abs/2019ne-328703.

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Krishnamohan, M., A. A. Seawright, M. R. Moore, and J. C. Ng. "Carcinogenesis in female C57Bl/6J mice chronically exposed to sodium arsenate (Asv) in drinking water for 2 years." In ENVIRONMENTAL TOXICOLOGY 2010. Southampton, UK: WIT Press, 2010. http://dx.doi.org/10.2495/etox100021.

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Mahmud, Marike, Fitryane Lihawa, Aryati Alitu, Rawiyah Husnan, and Barry Labdul. "An Analysis of Water Quality of Lake Perintis and Lake Limboto as Irrigation Water Sources in Gorontalo Province." In Unima International Conference on Science and Technology 2022. Switzerland: Trans Tech Publications Ltd, 2023. http://dx.doi.org/10.4028/p-l3rp7i.

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This study aims to analyze the water quality of Lake Perintis and Lake Limboto as irrigation water sources in Gorontalo Province. This study was carried out by considering the importance of water from these two lakes for irrigating the rice fields. 2 lakes became the locations for sampling in this study, namely Lake Limboto and Lake Perintis. Because this study was an initial survey, the researchers only applied one-site and one-time sampling for each lake. The observed parameters were temperature, TDS, COD, turbidity, phosphate, pH, nitrate, iron, manganese, cyanide, arsenic, fluoride, chlorine, manganese, magnesium, calcium, sodium, potassium, total coliform, and E. coli. The analysis was carried out at the Water Quality Laboratory, Health Office of Gorontalo and Integrated Research & Testing Laboratory of Gadjah Mada University. The sampling for total coliform was carried out aseptically, while measurements in the laboratory applied the MPN method. The way of taking chemical and physical samples followed SNI 8995-2021. Furthermore, the collected data were analyzed following Government Regulation No. 22/2021 Appendix VI regarding Lake Water Quality Standards. In analyzing water for irrigation purposes, the researchers applied the Sodium Adsorption Ratio (SAR) classification. Results showed that the water quality of Lake Perintis and Lake Limboto did not meet the requirements stipulated in Government Regulation No. 22/2021 because the chlorine, COD, and phosphate parameters were above the required quality standard. In addition, we also found that the pollution level in Lake Limboto was higher than that of Lake Perintis. Moreover, the results of the SAR analysis indicated a score of 7.975 for Lake Perintis (classified in the low sodium criteria) and a score of 11.23 for Lake Limboto (classified in the medium sodium criteria).
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Reports on the topic "Arsenite de sodium"

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Sadka, Avi, Mikeal L. Roose, and Yair Erner. Molecular Genetic Analysis of Citric Acid Accumulation in Citrus Fruit. United States Department of Agriculture, March 2001. http://dx.doi.org/10.32747/2001.7573071.bard.

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The acid content of the juice sac cells is a major determinant of maturity and fruit quality in citrus. Many citrus varieties accumulate acid in concentrations that exceed market desires, reducing grower income and consumer satisfaction. Pulp acidity is thought to be dependent on two mechanisms: the accumulation of citric acid in the vacuoles of the juice sac cells, and acidification of the vacuole. The major aim of the project was to direct effort toward understanding the mechanism of citric acid accumulation in the fruit. The following objectives were suggested: Measure the activity of enzymes likely to be involved in acid accumulation and follow their pattern of expression in developing fruit (Sadka, Erner). Identify and clone genes which are associated with high and low acid phenotypes and with elevated acid level (Roose, Sadka, Erner). Convert RAPD markers that map near a gene that causes low acid phenotype to specific co dominant markers (Roose). Use genetic co segregation to test whether specific gene products are responsible for low acid phenotype (Roose and Sadka). Objective 1 was fully achieved. Most of the enzymes of organic acid metabolism were cloned from lemon pulp. Their expression was studied during fruit development in low and high acid varieties. The activity and expression of citrate synthase, aconitase and NADP-isocitrate dehydrogenase (IDH) were studied in detail. The role that each enzyme plays in acid accumulation and decline was evaluated. As a result, a better understanding of the metabolic changes that contribute to acid accumulation was achieved. It was found that the activity of the mitochondrial aconitase is greatly reduced early in high-acid fruits, but not in acidless ones, suggesting that this enzyme plays an important role in acid accumulation. In addition, it was demonstrated that increases in the cytosolic forms of aconitase and NADP-IDH towards fruit maturation play probably a major role in acid decline. Our studies also demonstrated that the two mechanisms that contribute to fruit acidity, vacuolar acidification and citric acid accumulation, are independent, although they are tightly co-regulated. Additional, we demonstrated that sodium arsenite, which reduce fruit acidity, causes a transient inhibition in the activity of citrate synthase, but an induction in the gene expression. This part of the work has resulted in 4 papers. Objective 3 was also fully achieved. Using bulked segregant analysis, three random amplified polymorphic DNA (RAPD) markers were identified as linked to acitric, a gene controlling the acidless phenotype of pummelo 2240. One of them, which mapped 1.2 cM from acitric was converted into sequence characterized amplified region (SCAR marker, and into co dominant restriction length polymorphism (RFLP) marker. These markers were highly polymorphic among 59 citrus accessions, and therefore, they should be useful for selecting seedling progeny heterozygous for acitric in nearly all crosses between pummelo 2240 and other citrus genotypes. This part of the project resulted in one paper. Objective 4 was also fully achieved. Clones isolated by the Israeli group were sent to the American laboratory for co segregation analysis. However, none of them seemed to co segregate with the low acid phenotype. Both laboratories invested much effort in achieving the goals of Objective 2, namely the isolation of genes that are elevated in expression in low and high acid phenotypes, and in tissue cultures treated with arsenite (a treatment which reduces fruit acidity). However, conventional differential display and restriction fragment differential display analyses could not identify any differentially expressed genes. The isolation of such genes was the major aim of a continuation project, which was recently submitted.
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Das, Tanmoy, Alexander V. Balatsky, Chenglin Zhang, Haifeng Li, Yiki Su, Tucker Nethertom, Caleb Redding, et al. Distinguishing S-plus-minus and S-plus-plus electron pairing symmetries by neutron spin resonances in superconducting Sodium-Iron-Cobalt-Arsenic (transitional temperature = 18 Kelvin). Office of Scientific and Technical Information (OSTI), June 2012. http://dx.doi.org/10.2172/1043014.

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3

Kirby, Stefan M., J. Lucy Jordan, Janae Wallace, Nathan Payne, and Christian Hardwick. Hydrogeology and Water Budget for Goshen Valley, Utah County, Utah. Utah Geological Survey, November 2022. http://dx.doi.org/10.34191/ss-171.

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Goshen Valley contains extensive areas of agriculture, significant wetlands, and several small municipalities, all of which rely on both groundwater and surface water. The objective of this study is to characterize the hydrogeology and groundwater conditions in Goshen Valley and calculate a water budget for the groundwater system. Based on the geologic and hydrologic data presented in this paper, we delineate three conceptual groundwater zones. Zones are delineated based on areas of shared hydrogeologic, geochemical, and potentiometric characteristics within the larger Goshen Valley. Groundwater in Goshen Valley resides primarily in the upper basin fill aquifer unit (UBFAU) and lower carbonate aquifer unit (LCAU) hydrostratigraphic units. Most wells in Goshen Valley are completed in the UBFAU, which covers much of the valley floor. The UBFAU is the upper part of the basin fill, which is generally less than 1500 feet thick in Goshen Valley. Important spring discharge at Goshen Warm Springs issues from the LCAU. Relatively impermeable volcanic rocks (VU) occur along much of the upland parts of the southern part of Goshen Valley. Large sections of the southwest part of the Goshen Valley basin boundary have limited potential for interbasin flow. Interbasin groundwater flow is likely at several locations including the Mosida Hills and northern parts of Long Ridge and Goshen Gap in areas underlain by LCAU. Depth to groundwater in Goshen Valley ranges from at or just below the land surface to greater than 400 feet. Groundwater is within 30 feet of the land surface near and north of Goshen, in areas of irrigated pastures and wetlands that extend east toward Long Ridge and Goshen Warm Springs, and to the north towards Genola. Groundwater movement is from upland parts of the study area toward the valley floor and Utah Lake. Long-term water-level change is evident across much of Goshen Valley, with the most significant decline present in conceptual zone 2 and the southern part of conceptual zone 1. The area of maximum groundwater-level decline—over 50 feet—is centered a few miles south of Elberta in conceptual zone 2. Groundwater in Goshen Valley spans a range of chemistries that include locally high total dissolved solids and elevated nitrate and arsenic concentrations and varies from calcium-bicarbonate to sodium-chloride-type waters. Overlap in chemistry exists in surface water samples from Currant Creek, the Highline Canal, and groundwater. Stable isotopes indicate that groundwater recharges from various locations that may include local recharge, from the East Tintic Mountains, or far-traveled groundwater recharged either in Cedar Valley or east of the study area along the Wasatch Range. Dissolved gas recharge temperatures support localized recharge outside of Goshen. Most groundwater samples in Goshen Valley are old, with limited evidence of recent groundwater recharge. An annual water budget based on components of recharge and discharge yields total recharge of 32,805 acre-ft/yr and total discharge of 35,750 acre-ft/yr. Most recharge is likely from interbasin flow and lesser amounts from precipitation and infiltration of surface water. Most discharge is from well water withdrawal with minor spring discharge and groundwater evapotranspiration. Water-budget components show discharge is greater than recharge by less than 3000 acreft/yr. This deficit or change in storage is manifested as longterm water-level decline in conceptual zone 2, and to a lesser degree, in conceptual zone 1. The primary driver of discharge in conceptual zone 2 is well withdrawal. Conceptual zone 3 is broadly in balance across the various sources of recharge and discharge, and up to 1830 acre-ft/yr of water may discharge from conceptual zone 3 into Utah Lake. Minimal groundwater likely flows to Utah Lake from zones 1 or 2.
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