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Journal articles on the topic 'Artère pulmonaire'

Author: Grafiati

Published: 4 June 2021

Last updated: 6 February 2022

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1

Mailly, N., P. Gandia, S. Pesenti-Duterque, S. Arnaud, F. Blain, A. Camezind, M. Kany, et al. "Artère pulmonaire gauche rétrotrachéale chez un adulte." Journal de Radiologie 88, no. 9 (September 2007): 1197–99. http://dx.doi.org/10.1016/s0221-0363(07)89934-3.

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2

Ukkola-Pons, E., G. Weber-Donat, J. Potet, F. Minvielle, J. Baccialone, G. Bonardel, and C. Teriitehau. "Agénésie unilatérale d’une artère pulmonaire, à propos d’un cas." Journal de Radiologie 91, no. 6 (June 2010): 723–25. http://dx.doi.org/10.1016/s0221-0363(10)70105-0.

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3

Carfagna, L., O. Bouali, P. Galinier, P. Vaysse, J. Moscovici, J. Guitard, and P. Chaynes. "Artère pulmonaire gauche rétrotrachéale : mise au point embryologique et anatomique." Morphologie 93, no. 300 (March 2009): 30–34. http://dx.doi.org/10.1016/j.morpho.2008.12.001.

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4

Mahdhaoui, A., H. Bouraoui, J. Souissi, K. H. Mabrouk, F. Bahri, H. Amara, S. Ernez-Hajri, G. Jeridi, and H. Ammar. "Échinococcose cardiaque à propos d’une double localisation : artère pulmonaire–ventricule gauche." La Revue de Médecine Interne 25, no. 1 (January 2004): 94–96. http://dx.doi.org/10.1016/j.revmed.2003.09.011.

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5

Carfagna, L., R. Lopez, O. Bouali, F. Lauwers, P. Chaynes, Ph Vaysse, J. Guitard, and J. Moscovici. "Artère pulmonaire gauche rétro trachéale: mise au point embryologique et anatomique à propos d’un cas." Morphologie 91, no. 293 (July 2007): 103. http://dx.doi.org/10.1016/j.morpho.2007.09.052.

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6

Milhe, F., J. Schranz, S. Boniface, D. Vervloet, and A. Magnan. "Un faux asthme lié à une compression bronchique par une artère pulmonaire gauche à trajet aberrant chez une enfant de 7 ans." Revue des Maladies Respiratoires 21, no. 2 (April 2004): 402–6. http://dx.doi.org/10.1016/s0761-8425(04)71303-0.

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7

Radford, James Michael. "What to Do When They’re Eating for Two? A Case of Catheter-Directed Thrombolysis for Submassive Pulmonary Embolism in Pregnancy." Canadian Journal of General Internal Medicine 16, no. 1 (March 26, 2021): 24–30. http://dx.doi.org/10.22374/cjgim.v16i1.440.

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ABSTRACTA 33-year-old G7P0 female at 8 weeks gestation presented to the emergency department (ED) following a syncopal episode. She complained of chest pain and dyspnea, and had hemodynamic instability, which responded to intravenous fluids. Continued fluid resuscitation, supplemental oxygen, as well as therapeutic dose low molecular weight heparin (LMWH) were administered in the ED. Computed tomography (CT) pulmonary angiogram confirmed saddle pulmonary embolism (PE). After 12 h of continued chest pain and high oxygen requirements, a decision was made to use catheter-directed thrombolysis (CDT) involving alteplase with manual thrombus maceration in bilateral pulmonary arteries. There were no immediate hemorrhagic complications and follow-up fetal ultrasound demonstrated a normal viable intrauterine pregnancy. She clinically improved and was discharged on LMWH. Cesarean section was scheduled, and the patient delivered a healthy term infant at 37 weeks gestation without complications. Our case demonstrates that CDT may be a safe and effective treatment for submassive PE in pregnancy. RÉSUMÉUne femme âgée de 33 ans et enceinte de huit semaines (G7P0) se présente aux urgences à la suite d’un épisode syncopal. Elle se plaint de douleurs thoraciques et de dyspnée et présente une instabilité hémodynamique qui répond aux solutés intraveineux. Une réanimation liquidienne continue, une oxygénothérapie ainsi qu’une dose thérapeutique d’héparine de faible poids moléculaire sont administrées aux urgences. L’angiographie pulmonaire par tomodensitométrie confirme une embolie pulmonaire en selle. Après 12 heures de douleurs thoraciques continues et de besoins élevés en oxygène, on décide d’utiliser la thrombolyse dirigée par cathéter (TDC) à l’aide de l’altéplase avec macération manuelle du thrombus dans les artères pulmonaires bilatérales. Il n’y a pas eu de complications hémorragiques immédiates et le suivi échographique du fœtus a démontré une grossesse intra utérine normale et viable. Son état clinique s’est amélioré et elle a obtenu son congé sous héparine de faible poids moléculaire. Une césarienne a été planifiée et la patiente a accouché d’un enfant à terme et en bonne santé à 37 semaines de grossesse, sans complications. Notre cas démontre que la TDC peut être un traitement sûr et efficace de l’embolie pulmonaire submassive en cours de grossesse.

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8

Hicks, J., and S. Comeau. "VAGAL REGULATION OF INTRACARDIAC SHUNTING IN THE TURTLE PSEUDEMYS SCRIPTA." Journal of Experimental Biology 186, no. 1 (January 1, 1994): 109–26. http://dx.doi.org/10.1242/jeb.186.1.109.

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Two principal hypotheses account for intracardiac shunting in reptiles. The ‘pressure shunting’ hypothesis proposes that there is no fuctional separation between the ventricular cava during systole. The ‘washout shunting’ hypothesis suggests that the cavum pulmonale is functionally separated from the rest of the ventricle during systole. The purpose of this study was to test the two principal hypotheses in a turtle, Pseudemys scripta, after inducing a right-to-left shunt by electrical stimulation of the vagus nerve. Animals were anaesthetized with sodium pentobarbital (30–40 mg kg-1), tracheotomized and mechanically ventilated. Two experimental groups were used. Both groups had the right and left cervical vagi exposed and sectioned and silver bipolar electrodes were attached for electrical stimulation. In addition, cardiac function was evaluated by determining the pulmonary blood flow, pulmonary arterial pressure, peak systolic pressure in the cavum pulmonale, central arterial pressure, pulmonary vascular resistance and heart rate. In group I, hydrogen electrodes were inserted into the right aorta, the left aorta and the pulmonary artery. Hydrogen, dissolved in saline, was infused into the left atrium, jugular vein and cavum pulmonale. Blood flow from these sites was deduced from detection of a H2 signal in the right and left aortae and the pulmonary artery. In group II, catheters were inserted in the left and right atria and aortae for the measurement of blood gases. For both groups, the protocol consisted of control periods and periods of electrical stimulation of the efferent and afferent ends of the vagus nerve. During the control periods, infusion of a H2 solution into either the left atrium or the jugular vein resulted in the detection of H2 in the right and left aortae and the pulmonary artery. This suggested that both right-to-left and left-to-right intracardiac shunts were present. H2 infused into the cavum pulmonale was always detected in the pulmonary artery but never in the left or right aortae. During stimulation of the right vagal efferents, a bradycardia developed (heart rate declined by 65 %), pulmonary blood flow was reduced by 73 % and pulmonary vascular resistance increased by 158 %. This was accompanied by a reduction in the PO2 of both the right and left aortae, although the PO2 of the left and right atria remained constant. Under these conditions, H2 infused into the jugular vein and the left atrium was detected in the right and left aortae and the pulmonary artery of all animals studied. Infusion of H2 into the cavum pulmonale was detected in the right and left aortae in only two animals. The results supported the washout mechanism for right-to-left intracardiac shunting.

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9

Başaran, Murat. "Pulmonary artery aneurysm leading to coronary ischemia." Turkish Journal of Thoracic and Cardiovascular Surgery 22, no. 1 (January 21, 2014): 149–51. http://dx.doi.org/10.5606/tgkdc.dergisi.2014.7711.

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10

Venkatachala, Rao Preethi, C. N. Sheela, Shobha G., Uma Devaraj, Arpana Kedlaya, and Kiron Varghese. "A successful obstetric outcome in pregnancy complicated with cor pulmonale, pulmonary hypertension secondary to pulmonary tuberculosis: a case report." International Journal of Reproduction, Contraception, Obstetrics and Gynecology 7, no. 7 (June 27, 2018): 2975. http://dx.doi.org/10.18203/2320-1770.ijrcog20182921.

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Pregnancy in patients with pulmonary hypertension and cor pulmonale is associated with high morbidity and mortality. The timing of termination of pregnancy, mode of delivery, labour analgesia and anaesthesia and management during puerperium are challenging issues which need to be resolved. Authors report a case of a 30-year-old primigravida with cor pulmonale and severe pulmonary artery hypertension secondary to past pulmonary tuberculosis, who delivered a healthy baby vaginally, due to its rarity and good outcome. Early diagnosis, effective management of aggravating factors like anaemia and hypertension, informed choice by the couple regarding route of delivery and multidisciplinary approach are the keys to successful outcomes in patients with cor pulmonale.

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11

Adedeji, Moses O., Julio Cespedes, Kay Allen, Charu Subramony, and Michael D. Hughson. "Pulmonary Thrombotic Arteriopathy in Patients With Sickle Cell Disease." Archives of Pathology & Laboratory Medicine 125, no. 11 (November 1, 2001): 1436–41. http://dx.doi.org/10.5858/2001-125-1436-ptaipw.

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Abstract Background.—Shortened life expectancy due to pulmonary hypertension (PH) is seen in 5% to 10% of patients with sickle cell disease. The principal factors suspected of causing PH are pulmonary thromboemboli (PE) and in situ arterial thrombosis. Objective.—To investigate the possible role that PE or in situ arterial thrombosis play in the development of PH in sickle cell disease. Methods.—Autopsies of 12 patients with sickle cell disease were correlated with clinical data from medical records. Results.—Right ventricular hypertrophy was present in 9 of 12 patients. Six patients with right ventricular hypertrophy had thrombi in large elastic pulmonary arteries. All patients with elastic artery thrombi had fresh or organized thrombi in small muscular pulmonary arteries. Hypertensive small arterial changes were present in 5 of these 6 patients. Six patients showed no thrombi in elastic arteries. Among these 6 patients, 3 had right ventricular hypertrophy and recent and organized thrombi, as well as hypertensive changes in small arteries. One of these 3 patients demonstrated plexiform-like lesions and fibrinoid necrosis of small arteries. Three patients without right ventricular hypertrophy had pneumonia or pulmonary edema with no identifiable pulmonary artery pathology. Conclusions.—Arterial thrombosis with PH and cor pulmonale was regarded as the cause of death among most of these patients. Elastic artery thrombi are pulmonary thromboemboli, but pulmonary thromboemboli are always associated with widespread thrombosis of small arteries. Widespread thrombosis of small arteries alone was associated with PH in some cases. This finding suggests that pulmonary thromboemboli may be a late complication of PH and cor pulmonale and that an in situ thrombotic arteriopathy underlies the development of PH in most patients with sickle cell disease.

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12

P. Thakrar, Monil, Sapna Gandhi, Alpana Jain, and Arpita Jindal. "Anomalous Origin of Left Coronary Artery from Pulmonary Trunk." Indian Journal of Forensic Medicine and Pathology 9, no. 3 (2016): 185–87. http://dx.doi.org/10.21088/ijfmp.0974.3383.9316.17.

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13

Ying, Xiaonan, Wenqin Li, Yan Wang, and Biaoru Li. "In silico Modeling Personalized Therapy of Pulmonary Artery Hypertension." SSR Institute of International Journal of Life Sciences 5, no. 4 (July 4, 2019): 2341–48. http://dx.doi.org/10.21276/ssr-iijls.2019.5.4.2.

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14

Huang, Chun-Yao, Cheng-Hsiung Huang, An-Huang Yang, Mei-Han Wu, Yu-An Ding, and Wen-Chung Yu. "Solitary pulmonary artery myxoma manifesting as pulmonary embolism and subacute cor pulmonale." American Journal of Medicine 115, no. 8 (December 2003): 680–81. http://dx.doi.org/10.1016/j.amjmed.2003.06.001.

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15

Huang, Shao-Sung, Cheng-Hsiung Huang, An-Hang Yang, and Wen-Chung Yu. "Solitary Pulmonary Artery Intima Sarcoma Manifesting as Pulmonary Embolism and Subacute Cor Pulmonale." Circulation 120, no. 22 (December 2009): 2269–70. http://dx.doi.org/10.1161/circulationaha.109.899724.

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16

Navarro-Guillamón, Laura, Javier Vasserot-Vargas, Dolores Purificación Cárdenas-Cruz, Sergio Martínez-Escobar, Sofía García-Ordoñez, and Antonio Cárdenas-Cruz. "Traumatic pulmonary artery perforation." ACTUALIDAD MEDICA 104, no. 808 (December 31, 2019): 182–83. http://dx.doi.org/10.15568/am.2019.808.cc01.

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17

Avci, Sema, Gokhan Perincek, and Muammer Karakayali. "Prediction of Mortality Associated with Cardiac and Radiological Findings in Patients with Pulmonary Embolism." Journal Of Cardiovascular Emergencies 6, no. 4 (December 1, 2020): 84–90. http://dx.doi.org/10.2478/jce-2020-0020.

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AbstractBackground: In this study, we aimed to compare echocardiography, electrocardiography (ECG) abnormalities, Doppler ultrasonography (USG), and computed tomography pulmonary angiography (CTPA) results in predicting 3-month mortality in patients with acute pulmonary embolism (PE).Methods: This retrospective cohort study included 124 patients (72 females, 52 males) with acute PE. Demographics, symptoms, clinical signs, comorbidities, history of surgery, arterial blood gas, liver-renal functions, complete blood count, echocardiography, ECG, Doppler USG, and CTPA results, as well as 3-month mortality were recorded.Results: pH (z = –2.623; p <0.01), hemoglobin (z = –3.112; p <0.01), and oxygen saturation (z = –2.165; p <0. 01) were significantly higher in survivors. White blood cell (z = –2.703; p <0.01), blood urea nitrogen (z = –3.840; p <0.01), creatinine (z = –3.200; p <0.01), respiratory rate (z = –2.759; p <0.01), and heart rate (z = –2.313; p <0.01) were significantly higher in non-survivors. Nonspecific ST changes (AUC 0.52, 95% CI 0.43–0.61), p pulmonale (AUC 0.52, 95% CI 0.43–0.61), normal axis (AUC 0.61), right axis deviation (AUC 0.56), right ventricle strain pattern (AUC 0.59), and right pulmonary artery embolism (AUC 0.54) on CTPA showed the highest mortality prediction.Conclusions: Nonspecific ST changes, p pulmonale, normal axis and right axis deviation in ECG, RV strain in echocardiography, and right pulmonary artery embolism on CTPA are associated with a higher mortality in patients with PE.

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18

Vernhet, H., G. Dogas, C. Bousquet, O. Serre-Cousiné, and J. P. Sénac. "Imagerie des artères pulmonaires." EMC - Cardiologie-Angéiologie 1, no. 1 (February 2004): 23–37. http://dx.doi.org/10.1016/j.emcaa.2003.12.004.

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19

Waghmare, Vivekanand N. "Correlation of the Spirometric and Arterial Blood Gas Variables with Pulmonary Artery Systolic Pressure in Predicting Pulmonary Hypertension in Chronic Obstructive Pulmonary Disease Patients." Journal of Cardiovascular Medicine and Surgery 4, no. 2 (2018): 170–75. http://dx.doi.org/10.21088/jcms.2454.7123.4218.17.

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20

Parikh, Nirav, Jeevraj Rajawat, Deepal Prajapati, Ramesh Patel, and Himani Pandya. "Comparative Study of Pulmonary Artery Catheter vs Central Venous Catheter in Coronary Artery Bypass Grafting Surgery Patients." Indian Journal of Anesthesia and Analgesia 6, no. 5 (P-2) (2019): 1770–78. http://dx.doi.org/10.21088/ijaa.2349.8471.6519.40.

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21

Avci, Sema, Gokhan Perincek, and Muammer Karakayali. "Prediction of Mortality Associated with Cardiac and Radiological Findings in Patients with Pulmonary Embolism." Journal Of Cardiovascular Emergencies 6, no. 4 (December 1, 2020): 84–90. http://dx.doi.org/10.2478/jce-2020-0020.

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Abstract Background: In this study, we aimed to compare echocardiography, electrocardiography (ECG) abnormalities, Doppler ultrasonography (USG), and computed tomography pulmonary angiography (CTPA) results in predicting 3-month mortality in patients with acute pulmonary embolism (PE). Methods: This retrospective cohort study included 124 patients (72 females, 52 males) with acute PE. Demographics, symptoms, clinical signs, comorbidities, history of surgery, arterial blood gas, liver-renal functions, complete blood count, echocardiography, ECG, Doppler USG, and CTPA results, as well as 3-month mortality were recorded. Results: pH (z = –2.623; p <0.01), hemoglobin (z = –3.112; p <0.01), and oxygen saturation (z = –2.165; p <0. 01) were significantly higher in survivors. White blood cell (z = –2.703; p <0.01), blood urea nitrogen (z = –3.840; p <0.01), creatinine (z = –3.200; p <0.01), respiratory rate (z = –2.759; p <0.01), and heart rate (z = –2.313; p <0.01) were significantly higher in non-survivors. Nonspecific ST changes (AUC 0.52, 95% CI 0.43–0.61), p pulmonale (AUC 0.52, 95% CI 0.43–0.61), normal axis (AUC 0.61), right axis deviation (AUC 0.56), right ventricle strain pattern (AUC 0.59), and right pulmonary artery embolism (AUC 0.54) on CTPA showed the highest mortality prediction. Conclusions: Nonspecific ST changes, p pulmonale, normal axis and right axis deviation in ECG, RV strain in echocardiography, and right pulmonary artery embolism on CTPA are associated with a higher mortality in patients with PE.

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22

Anikin, S. V., V. V. Yanovoy, and A. A. Brajnikova. "THE SOFTWARE SCORING OF THE PULMONARY ARTERY THROMBOEMBOLISM RISK IN ONCOLOGY." Amur Medical Journal, no. 3 (2017): 129. http://dx.doi.org/10.22448/amj.2017.3.129-129.

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23

Haskal, Z. J., M. C. Soulen, E. A. Huettl, H. I. Palevsky, and C. Cope. "Life-threatening pulmonary emboli and cor pulmonale: treatment with percutaneous pulmonary artery stent placement." Radiology 191, no. 2 (May 1994): 473–75. http://dx.doi.org/10.1148/radiology.191.2.8153324.

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24

Leonova, E. I., E. I. Shmelev, and R. B. Amansakhedov. "Intracardiac hemodynamic abnormalities in patients with sarcoidosis." Russian Pulmonology 28, no. 5 (December 24, 2018): 567–75. http://dx.doi.org/10.18093/0869-0189-2018-28-5-567-575.

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The aim of this study was to evaluate intracardiac hemodynamic abnormalities in patients with pulmonary sarcoidosis and to investigate relevant factors. Methods. The study involved 42 patients with chronic pulmonary sarcoidosis. Chest computed tomography (CT), echocardiography, body plethysmography, spirometry, 6-minute walking test, and measurements of lung diffusing capacity (DLCO), blood gases and blood C-reactive protein (CRP) were performed in all patients. Results. Chronic cor pulmonale was detected in 26.2% of patients. The right heart diastolic dysfunction was found in 14.3% of patients, the left heart diastolic dysfunction was found in 23.8% of patients, the mean pulmonary artery pressure (mPAP) was increased in 19% of patients. The right ventricular diameter was found to be related to the inspiratory bronchial resistance (Rin) (R = 0.480; p = 0.02), the intrathoracic gas volume (ITGV) (R = –0.670; p = 0.001), DLCO (R = 0.438; p = 0.013), and pulmonary fibrosis. The right heart systolic function (TAPSE) was related to DLCO (R = 0.518; p = 0.006), the total lung capacity (TLC) (R = 0.639; p = 0.001) and pulmonary fibrosis. The increased mPAP was related to the extension of disseminated lung lesions (R = 0.716; p = 0.018), blood oxygen partial pressure (R = 0.486; p = 0.017) and CRP level. The 6-min distance was related to Rin, the right ventricular diameter, parameters of the right heart diastolic function (tricuspid E / A ratio) (R = 0.486; p = 0.01), and ITGV (R = 0.494; p = 0.006). Conclusion. The development of cor pulmonale in patients with pulmonary sarcoidosis was related to DLCO, Rin and pulmonary fibrosis. The extension of disseminated lung lesions, hypoxemia and increased CRP were related to pulmonary hypertension. Different factors associated with cor pulmonale and pulmonary hypertension in sarcoidosis could prompt further investigation of different phenotypes of this disease with the intrinsic central hemodynamic abnormalities.

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25

Sperling, Brita L., Donald W. Cockcroft, and Rajni Chibbar. "Microscopic Pulmonary Tumour Embolism: An Unusual Presentation of Thymic Carcinoma." Canadian Respiratory Journal 9, no. 5 (2002): 347–50. http://dx.doi.org/10.1155/2002/439459.

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The present report describes the first reported case of microscopic pulmonary tumour embolism (MPTE) from thymic carcinoma. The carcinoma was discovered during an autopsy in a 55-year-old man who had undergone surgery for a pilonidal sinus two weeks before presentation. Pulmonary thromboembolism was suspected. This case was unusual because MPTE has never before been associated with thymic carcinoma, MPTE was the first clinical indication of an occult malignancy, and the clinical presentation was that of sudden onset of dyspnea associated with acute cor pulmonale. The cause of death was determined to be hypoxia secondary to extrinsic compression of the right pulmonary artery and extensive tumour emboli in the small arteries, arterioles and venules of the pulmonary parenchyma. A review of the clinical presentation and diagnosis of MPTE is included.

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26

CORAZZA, IVAN, CHIARA PINARDI, LUIGI MANCO, DAVID BIANCHINI, LAURA CERCENELLI, EMANUELA MARCELLI, and ROMANO ZANNOLI. "BIOMECHANICAL APPROACH TO THE CLINICAL TREATMENT OF PULMONARY ARTERIAL HYPERTENSION." Journal of Mechanics in Medicine and Biology 13, no. 05 (October 2013): 1340005. http://dx.doi.org/10.1142/s0219519413400058.

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Negative clinical trend of severe pulmonary arterial hypertension (PAH) is due to right ventricular chamber dilatation and consequent dysfunction. On a biomechanical basis the clinical evolution is associated to a progressive mismatching of the artero-ventricular junction and a possible mechanical intervention may be foreseen. This paper demonstrates how the progressive right ventricular dysfunction, in terms of biochemical–mechanical efficiency, is related to the stress–strain curve of the pulmonary artery, simulated by superimposing different elastance tubes in a cardiovascular mock. Applying the experimental signals to an "ideal" normal and dilated ventricle, it was demonstrated that the very high pulmonary elastance at high pressure is the prime cause of a sharp efficiency drop, in comparison with an aortic similar condition. This evidence supports the proposal of mechanical interventions to reduce pulmonary arterial elastance to improve the artero-ventricular matching, increase the reduced ventricular efficiency and change the negative trend of the illness.

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27

Bigot, J., M. Rémy-Jardin, J. B. Faivre, V. Deken, A. Duhamel, N. Tacelli, and J. Rémy. "Embolie pulmonaire non massive : impact de la distensibilite des arteres pulmonaires sur le score d’obstruction." Journal de Radiologie 88, no. 10 (October 2007): 1498. http://dx.doi.org/10.1016/s0221-0363(07)81573-3.

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28

RICHARD, C., and J. TEBOUL. "Cathétérisme artériel pulmonaire : pourquoi un tel mépris ?Pulmonary artery catheter: why such a derision?" Réanimation 12, no. 2 (March 2003): 127–33. http://dx.doi.org/10.1016/s1624-0693(03)00008-2.

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29

Doğan, Orhan Veli, Murat Günday, Ozgür Koral, and Ali İhsan Parlar. "Left Coronary Artery-Pulmonary Artery Fistula in Coronary Artery Disease." Kosuyolu Heart Journal 15, no. 2 (August 15, 2012): 84–86. http://dx.doi.org/10.5578/kkd.3426.

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30

Rubin, Lewis J. "Pulmonary Artery Denervation for Pulmonary Artery Hypertension." JACC: Cardiovascular Interventions 8, no. 15 (December 2015): 2024–25. http://dx.doi.org/10.1016/j.jcin.2015.10.022.

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31

Picchi, Chiara, Marisa Barone, Chiara Beltrametti, Stefano Barco, Diana Irina Iosub, Montanari Laura, and Franco Piovella. "Pregnancy and Successful Delivery in a Patient with Chronic Thromboembolic Pulmonary Hypertension (CTEPH) Previously Submitted to Pulmonary Endarterectomy (PEA)." Blood 118, no. 21 (November 18, 2011): 4329. http://dx.doi.org/10.1182/blood.v118.21.4329.4329.

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Abstract Abstract 4329 Chronic thromboembolic pulmonary hypertension (CTEPH) results from obstruction of the major pulmonary arteries by incompletely resolved or organized pulmonary emboli which have become incorporated into the pulmonary artery wall, eventually causing an increase in pulmonary vascular resistances. CTEPH is a condition that is recognised in an increased percentage of patients. Pulmonary endarterectomy (PEA) is recognized as being the only curative option for a subgroup of those patients, but anaesthesiologists and intensivists face many challenges in how they manage these patients perioperatively. Ultimately, it is the combination of skills in a multidisciplinary team that leads to a successful procedure and dramatically improves patient's quality of life and life expectancy. Careful pre- and post-operative management is therefore essential for such a successful outcome following PEA. In 1994 in Pavia was started a program in which members of a multidisciplinary team work in close interaction with the aim of increase experience in the challenging problems these patients present in the evaluative, surgical, and post-operative phases of their care. Pregnancy in women with pulmonary hypertension (PH) is reported to carry a maternal mortality rate of 30–56%. No report is available on the management of pregnancy and delivery in patients with CTPH. We report our experience of the management of a pregnancy in a patient previously submitted to PEA in whom pressures decreased significantly but remained higher than normal due to partial intervention. EFS, 29 yrs, had first hemoptysis in 2001. Since then, she suffered effort dyspnea. A second hemoptysis occurred in 2004. She was admitted to hospital as having “Multiple foci pneumonia, with pulmonary hypertension of unclear origin”. In 2005 patient was admitted to a different hospital. A CT scan showed: “Congenital right pulmonary artery agenesia associated with bilateral multiple artero-venous malformations. NYHA Class III”. Patients was transferred to our hospital. Thrombophilic workout resulted negative. PaO2: 79.9 mmHg, pulmonary artery pressures (PAP): 130/60/13 mmHg, pulmonary vascular resistances (PVR): 1.083 dynes/sec/min−5. CT angio-scan: Severe dilation of the common pulmonary artery (34 mm). Right pulmonary artery visible only at proximal level. Bronchial artery dilation, bilaterally. V/Q scan: Absent visualization of the right pulmonary artery. Perfusion absent. Arteriography: Clearcut thrombosis of the right pulmonary artery (initial tract). Multiple typical CTPH lesions of the left pulmonary artery. Lower limb compression ultrasound (CUS): No sign of deep or superficial vein thrombosis. On 11 april, 2005 PEA was performed on the left side, with an attempt on the right side. No agenesia of the right pulmonary artery was found. Probable occlusion in early age, with evolution in fibrosis. Post surgery, PaO2: 94.3 mmHg, PAP: 53/32/15 mmHg, PVR: 453 dynes/sec/min−5. On September 16, 2005 she was pregnant, seventh week. After careful multidisciplinary counseling, patient decided to continue pregnancy. Anticoagulant treatment was switched from warfarin to low molecular weight heparin (LMWH), therapeutic dosage. Two days before elective delivery, LMWH was reduced to prophylactic dosages. On march 2, 2006 after an uncomplicated Caesarean section under general anesthesia, she delivered a healthy baby girl. During the following months, PAPs and functional parameters normalized. Patient is today in relatively good health. Is under oral vitamin K antagonists treatment. In 2007, being pregnant again, opted for therapeutic abortion. Maternal mortality in parturients with PAH or CTPH remains prohibitively high, despite lower death rates than previous decades. Early advice on pregnancy risks, including contraception, remains paramount. Women with PAH or CTPH who become pregnant warrant a multidisciplinary approach with consideration of appropriate therapies. Disclosures: No relevant conflicts of interest to declare.

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Guliyeva, Aynur, Erkan Cakir, Hakan Yazan, Lina AlShadfan, Rasul Sharifov, Hafize Otcu Temur, Utku Karaarslan, et al. "Assessment of Right Heart Functions in Children with Mild Cystic Fibrosis." Klinische Pädiatrie 233, no. 05 (February 18, 2021): 231–36. http://dx.doi.org/10.1055/a-1341-1698.

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ABSTRACT Background Cystic fibrosis (CF) is a multisystemic disease that prevalently involves the lungs. Hypoxemia occurs due to the existing of progressive damage to the pulmonary parenchyma and pulmonary vessels. The condition may cause systolic and diastolic dysfunction to the right ventricle due to the effects of high pulmonary artery systolic pressure (PASP). The study aimed to determine echocardiographic alterations in PASP, right ventricle (RV) anatomy, and functions in mild CF children. Materials and Methods RV anatomy, systolic, and diastolic functions were evaluated with conventional echocardiographic measurements. Estimated PASP was used measured with new echocardiographic modalities, including pulmonary artery acceleration time (PAAT), right ventricular ejection time (RVET), and their ratio (PAAT/RVET). The obtained echocardiographic data were statistically compared between the patient group and the control group. Results The study consisted of 30 pediatric patients with mild CF and 30 healthy children with similar demographics. In patient group, conventional parameters disclosed differences in RV anatomy, both systolic and diastolic functions of RV compared with the healthy group. We did not compare the patient group with published standard data because of the wide range variability. However, new echocardiographic parameters showed notable increase in pulmonary artery pressure compared with values of control group and published standard data (p<0.001). Conclusion Elevated PASP, RV failure, and Cor pulmonale usually begin early in children with mild CF. In addition to routine echocardiographic measurements to evaluate RV, we recommend the use of new echocardiographic modalities for routine examinations and in the follow up of children with mild CF.

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Sengupta, A., S. P. Dubey, D. Chaudhuri, A. K. Sinha, and P. Chakravarti. "Ortner's syndrome revisited." Journal of Laryngology & Otology 112, no. 4 (April 1998): 377–79. http://dx.doi.org/10.1017/s0022215100140514.

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AbstractHoarseness of voice due to paralysis of the left recurrent laryngeal nerve caused by a dilated left atrium in mitral stenosis as discussed by Ortner, is a subject of controversy. Different authors have cited different mechanisms as explanation. A variety of cardiac problems such as primary pulmonary hypertension, ischaemic heart disease, various congenital heart disorders can all lead to paralysis of the left recurrent laryngeal nerve. Most authors believe that pressure in the pulmonary artery causes the nerve compression. In Papua New Guinea cor pulmonale and rheumatic heart disease are the commonest cardiac disorders seen. Ortner's syndrome is a rarity and has never been reported from here before. Here three different case reports are presented with mitral stenosis, primary pulmonary hypertension and combined mitral stenosis and regurgitation and the pathogenesis of hoarseness is discussed.

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34

Kaushik, Atul. "Pulmonary Artery Pseudsoaneurysm due to Mycobacterium Tuberculosis Managed Successfully by Transcatheter Coil Embolization." Clinical Cardiology and Cardiovascular Interventions 4, no. 4 (February 22, 2021): 01–04. http://dx.doi.org/10.31579/2641-0419/130.

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Pulmonary artery pseudoaneurysm due to mycobacterium tuberculosis is a rare clinical entity. A 29 years old female presented to us with hemoptysis. She was a known case of pulmonary tuberculosis and was on antitubercular treatment. Emergency transcatheter coil embolization of the neck of the sac of pseudoaneurysm arising from the right descending pulmonary artery was done. At 6 months of follow up, the patient was asymptomatic. We are reporting this case due to a rare presentation of pulmonary artery pseudoaneurysm due to mycobacterium tuberculosis and pulmonary artery as an unusual source of hemoptysis instead of the bronchial artery. Learning objectives Pulmonary artery pseudoaneurysm due to Mycobacterium tuberculosis although rare but it's a potential life threatening situation, however early detection and prompt management may prevent fatal complications. Thus, the treating physician should keep this as a differential diagnosis whenever a patients presents with a massive hemoptysis.

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35

Grothues, Frank, Tobias Welte, Christof Huth, and Helmut U. Klein. "Pulmonary Artery." Circulation 108, no. 2 (July 15, 2003): 245–46. http://dx.doi.org/10.1161/01.cir.0000074787.22845.89.

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36

Agematsu, Kota, Toru Okamura, Yoji Takiguchi, Fumiya Yoneyama, and Yorikazu Harada. "Rapid growth of pulmonary artery after intrapulmonary artery septation." Asian Cardiovascular and Thoracic Annals 26, no. 6 (June 7, 2018): 479–81. http://dx.doi.org/10.1177/0218492318782821.

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Intrapulmonary artery septation is employed in patients with unbalanced pulmonary artery growth. A baby girl received a systemic pulmonary shunt in the neonatal period and bilateral Glenn shunts in early infancy. Once the Glenn shunts were established, the right pulmonary artery became hypoplastic. We performed intrapulmonary artery septation involving the Glenn shunts to the left pulmonary artery and the systemic pulmonary shunt to the right pulmonary artery. As early as 3 months after intrapulmonary artery septation, right pulmonary artery growth was observed. Eventually, Fontan circulation was achieved with fenestration.

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A., et al, Diasmada. "Pulmonary Artery Dissection Following Severe Pulmonary Artery Hypertension." ACI (Acta Cardiologia Indonesiana) 3, no. 2(S) (September 4, 2017): 21. http://dx.doi.org/10.22146/aci.27983.

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38

KUMAR, R., P. ROUGHNEEN, and M. DELEVAL. "Mycotic pulmonary artery aneurysm following pulmonary artery banding." European Journal of Cardio-Thoracic Surgery 8, no. 12 (1994): 665–66. http://dx.doi.org/10.1016/s1010-7940(05)80178-2.

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Robida, A., and B. Venkatraman. "Anomalous left pulmonary artery without pulmonary artery sling." Heart 79, no. 5 (May 1, 1998): 521–22. http://dx.doi.org/10.1136/hrt.79.5.521.

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40

Terra, Ricardo M., Angelo Fernandez, Ricardo H. Bammann, Jader J. M. Junqueira, and Vera L. Capelozzi. "Pulmonary Artery Sarcoma Mimicking a Pulmonary Artery Aneurysm." Annals of Thoracic Surgery 86, no. 4 (October 2008): 1354–55. http://dx.doi.org/10.1016/j.athoracsur.2008.04.005.

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41

Robertson, Murray A., Patricia A. Penkoske, and Neil F. Duncan. "Right pulmonary artery obstruction after pulmonary artery banding." Annals of Thoracic Surgery 51, no. 1 (January 1991): 73–75. http://dx.doi.org/10.1016/0003-4975(91)90453-w.

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42

Mainwaring, Richard D., R. Thomas Collins, Kirstie L. MacMillen, Michal Palmon, and Frank L. Hanley. "Pulmonary Artery Reconstruction After Failed Pulmonary Artery Stents." Annals of Thoracic Surgery 110, no. 3 (September 2020): 949–55. http://dx.doi.org/10.1016/j.athoracsur.2020.01.007.

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43

Vernhet-Kovacsik, H., and J. P. Sénac. "Exploration des artères pulmonaires en imagerie." EMC - Radiologie 1, no. 6 (December 2004): 621–31. http://dx.doi.org/10.1016/j.emcrad.2004.09.003.

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44

Huang, C. H., H. H. Huang, T. L. Chen, and M. J. Wang. "Perioperative Changes of Plasma Endothelin-1 Concentrations in Patients Undergoing Cardiac Valve Surgery." Anaesthesia and Intensive Care 24, no. 3 (June 1996): 342–47. http://dx.doi.org/10.1177/0310057x9602400307.

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Twenty-one patients who underwent cardiac valvular replacement procedures were studied to determine the peri-operative changes of plasma endothelin-1 (ET-1) concentration and disposition of ET-1 in the pulmonary and systemic vasculature between patients with elevated (over 20 mmHg) mean pulmonary artery pressure and patients with normal pulmonary artery pressure. The overall profile alterations of plasma ET- 1 concentration did not differ between the two groups of patients. The plasma ET- 1 levels in the pulmonary artery and right atrium were significantly higher in patients with high pulmonary artery pressure than in patients with normal pulmonary artery pressure. Before cardiopulmonary bypass, significant pulmonary extraction of the plasma ET- 1 existed in patients with high pulmonary artery pressure but the pulmonary extraction was not seen after bypass. There was no transpulmonary difference of the plasma ET- 1 concentration in patients with normal pulmonary artery pressure either before or after bypass. The high levels of ET- 1 in the pulmonary circuit and the pulmonary extraction of the ET- 1 in patients with high pulmonary artery pressure might be a protective mechanism for rheumatic valvular patients with elevated pulmonary artery pressure.

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45

Ko, Seien, Jin Komuro, Yoshinori Katsumata, Yasuyuki Shiraishi, Takashi Kawakami, Yoshitake Yamada, Shinsuke Yuasa, Takashi Kohno, Kenjiro Kosaki, and Keiichi Fukuda. "Peripheral pulmonary stenosis with Noonan syndrome treated by balloon pulmonary angioplasty." Pulmonary Circulation 10, no. 4 (October 2020): 204589402095431. http://dx.doi.org/10.1177/2045894020954310.

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Noonan syndrome is known to have various cardiovascular defects, which include pulmonary artery stenosis. Pulmonary artery stenosis is characterized by obstruction of pulmonary artery blood flow that can cause elevated pulmonary artery pressure and ventilation-perfusion inequality, which can cause dyspnea on exertion and eventually, heart failure. Although the etiology of pulmonary artery stenosis related to congenital diseases is still unknown, balloon pulmonary angioplasty has being reported to be effective to selected patients with Alagille and Williams syndromes, but not from Noonan syndrome despite of modest prevalence of pulmonary artery stenosis. Here, we report the first Noonan syndrome patient with pulmonary artery stenosis who underwent successful balloon pulmonary angioplasty. The strategy used in balloon pulmonary angioplasty was planned with careful morphologic evaluation by computed tomographic angiography, and performed with scoring balloons in a graded approach with multiple sessions. After balloon pulmonary angioplasty, we confirmed maintained dilation of lesions and symptom alleviation, suggesting that balloon pulmonary angioplasty can be performed safely on pulmonary artery stenosis in a Noonan syndrome patient.

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Yakubov, Nodirbek Ilkhomjon Ogli, Dilfuzahon Zakirjanovna Mamarasulova, Nataliya Gamletovna Dadamyants, and Anvar Arabbayevich Dalimov. "Risk Of Pulmonary Artery Thrombosis In Type 2 DM Patients Hospitalized With Covid-19." American Journal of Medical Sciences and Pharmaceutical Research 03, no. 08 (August 31, 2021): 37–43. http://dx.doi.org/10.37547/tajmspr/volume03issue08-07.

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For the first time on the Fergana Valley model, prognostic markers of the risk of thrombotic complications in COVID-19 patients with type 2 diabetes were studied. It was found that the level of glycemia, the concentration of proinflammatory cytokines IL-6, ferritin and CRP, and the marker of thrombus formation - d dimer, have a prognostic significance, with the maximum prognostic significance of the concentration of CRP. The prognostic significance of ultrasound in the aspect of thrombosis of the branches of the PA in patients with COVID-19 against the background of type 2 diabetes was established. Predictor markers are mean PA pressure, LV myocardial mass index, signs of increased intraventricular pressure in the RV, and evidence of peripheral venous thrombosis.

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Kato, Daiki, Shohei Mori, Eriko Harada, Rintaro Shigemori, Takamasa Shibazaki, Hideki Matsudaira, Jun Hirano, et al. "Pulmonary Artery Intimal Sarcoma Diagnosed Preoperatively by Endovascular Biopsy and Treated via Right Pneumonectomy and Pulmonary Arterioplasty." Case Reports in Pulmonology 2021 (June 27, 2021): 1–4. http://dx.doi.org/10.1155/2021/5573869.

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Introduction. Intimal sarcoma is a very rare tumor arising within the intima of the pulmonary artery. Preoperative diagnosis of pulmonary artery sarcoma is difficult, and the tumor is sometimes misdiagnosed as pulmonary thromboembolism. We report a case of pulmonary artery intimal sarcoma successfully diagnosed by preoperative endovascular biopsy and treated via right pneumonectomy and pulmonary arterioplasty. Presentation of a Case. A 72-year-old woman was referred to our hospital with a low-attenuation defect in the lumen of the right main pulmonary artery by computed tomography. Pulmonary artery thromboembolism was suspected, and anticoagulation therapy was administered. However, the defect in the pulmonary artery did not improve. Endovascular catheter aspiration biopsy was performed. Histological examination revealed pulmonary artery sarcoma. The patient was treated with right pneumonectomy and arterioplasty with the use of cardiopulmonary bypass. Discussion. Preoperative biopsy by endovascular catheter is worth considering for a patient with a tumor in the pulmonary artery and can help in planning treatment strategies.

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Sohns, Jan M., Jan Menke, Leonard Bergau, Bernhard G. Weiss, Hannah Kröhn, Desiree Weiberg, Thorsten Derlin, and Sebastian Schmuck. "Screening of extravascular findings in pulmonary embolism computer tomography: 397 patients with 1950 non-pulmonary artery findings." Vascular 26, no. 1 (August 18, 2017): 99–110. http://dx.doi.org/10.1177/1708538117724628.

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Objectives The aim of this study was to investigate the possible benefits from computed tomography scans of patients with a suspected pulmonary artery embolism with a focus on relevant extravascular findings. Methods A total of 400 consecutive computed tomography pulmonary angiographies were evaluated. Computed tomography scans were analyzed in detail for the presence of pulmonary artery embolisms, as well as any other findings. Extra-artery discoveries were classified into none-relevant (Group A), intermediate (Group B), or relevant (Group C) findings. Results Aggregated computed tomography pulmonary angiographies detected other diagnosis than pulmonary artery embolism in 236 patients (59%). There were 1950 non-pulmonary artery embolism findings (4.9 per patient; n = 397). In the pulmonary artery embolism group, there were 447 extra-pulmonary artery embolism findings (5.2 per patient; n = 86) and in the non-pulmonary artery embolism group, 1503 findings (4.8 per patient; n = 311). Patients with pulmonary artery embolism had a significantly higher rate of pro-coagulate risk factors ( p < 0.001). Conclusions Computed tomography pulmonary angiographies may help to identify further diagnoses. This study represents a retrospective review of a single center experience for incidental computed tomography findings during pulmonary artery embolism work-up and emphasizes the importance of analyzing the whole field-of-view.

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Durmaz, Selim, Tünay Kurtoğlu, Hasan Güngör, Muhammet Hüseyin Erkan, and Muharrem İsmail Badak. "Retrieval of the Atrial Septal Defect Closure Device Embolized to Pulmonary Artery." Kosuyolu Heart Journal 20, no. 2 (August 1, 2017): 153–56. http://dx.doi.org/10.5578/khj.9941.

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50

Wani, Abdul Haseeb, Yassar Shiekh, and Najeeb Tallal Ahangar. "Main pulmonary artery diameter assessment in a large sample of Indian population: a MDCT based study." International Journal of Scientific Reports 5, no. 6 (May 28, 2019): 145. http://dx.doi.org/10.18203/issn.2454-2156.intjscirep20192064.

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<p class="abstract"><strong>Background:</strong> The gold standard for pulmonary artery pressure measurement is right heart catheterization but its invasive nature precludes its routine use. Main pulmonary arterial trunk calibre increase is a strong indicator of underlying pulmonary arterial hypertension. MDCT can accurately measure the diameter of main pulmonary artery. The objective of the study was to establish the normative values of main pulmonary artery caliber using contrast enhanced CT and try to ascertain any significant difference in main pulmonary artery calibers between two genders and correlation of age and main pulmonary artery diameter.</p><p class="abstract"><strong>Methods:</strong> Contrast enhanced CT images of 462 subjects were analysed on a PACS workstation monitor and widest diameter perpendicular to long axis of the main pulmonary artery as seen on reformatted axial image was measured with electronic caliper tool at the level of the main pulmonary artery bifurcation. </p><p class="abstract"><strong>Results:</strong> The mean main pulmonary artery diameter in females was 22.54±2.19 mm and 23.34±3.06 mm in males. The mean pulmonary artery diameter in males was larger than females with statistically significant difference seen (p<0.05). The correlation coefficient between age of whole sample and their mean main pulmonary artery was found to be 0.1006 with no statistically significant difference.</p><p class="abstract"><strong>Conclusions:</strong> There is a statistically significant difference in the mean main pulmonary artery calibre between males and females with no strong correlation between the age and mean main pulmonary artery calibre. Further studies are warranted to find the complex interaction between main pulmonary artery diameter and sex, age and body mass index.</p>

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