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Relevant bibliographies by topics / Arterial calcification

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Academic literature on the topic 'Arterial calcification'

Author: Grafiati

Published: 4 June 2021

Last updated: 3 February 2022

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Journal articles on the topic "Arterial calcification"

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Bartstra, Jonas W., Wilko Spiering, Jody M. W. van den Ouweland, Willem P. T. M. Mali, Rob Janssen, and Pim A. de Jong. "Increased Elastin Degradation in Pseudoxanthoma Elasticum Is Associated with Peripheral Arterial Disease Independent of Calcification." Journal of Clinical Medicine 9, no. 9 (2020): 2771. http://dx.doi.org/10.3390/jcm9092771.

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Pseudoxanthoma elasticum (PXE) results in extensive fragmentation and calcification of elastin fibers in the peripheral arteries, which results in peripheral arterial disease (PAD). Current research focuses on the role of calcifications in the pathogenesis of PXE. Elastin degradation and calcification are shown to interact and may amplify each other. This study aims to compare plasma desmosines, a measure of elastin degradation, between PXE patients and controls and to investigate the association between desmosines and (1) arterial calcification, (2) PAD, and (3) PAD independent of arterial ca

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Schurgers, Leon J., Hermann Aebert, Cees Vermeer, Burkhard Bültmann, and Jan Janzen. "Oral anticoagulant treatment: friend or foe in cardiovascular disease?" Blood 104, no. 10 (2004): 3231–32. http://dx.doi.org/10.1182/blood-2004-04-1277.

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Abstract Calcification is a common complication in cardiovascular disease and may affect both arteries and heart valves. Matrix γ-carboxyglutamic acid (Gla) protein (MGP) is a potent inhibitor of vascular calcification, the activity of which is regulated by vitamin K. In animal models, vitamin K antagonists (oral anticoagulants [OACs]) were shown to induce arterial calcification. To investigate whether long-term OAC treatment may induce calcification in humans also, we have measured the grade of aortic valve calcification in patients with and without preoperative OAC treatment. OAC-treated sub

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Kei, Olivia C. Y., Walter J. Duncan, and Derek G. Human. "Pulmonary arterial and intracranial calcification in the recipient of a twin–twin transfusion." Cardiology in the Young 12, no. 5 (2002): 488–90. http://dx.doi.org/10.1017/s1047951102000859.

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Pulmonary arterial and intracranial calcifications are rarely found in children. A female infant, the recipient of a twin–twin transfusion syndrome was found, by ultrasound and computed tomography, to have both pulmonary arterial and intracerebral calcification. A rare condition, termed idiopathic arterial calcification of infancy, is the likely cause. This condition carries a poor prognosis and is usually fatal.

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Devriese, Magali, Anne Legrand, Marie-Cécile Courtois, Xavier Jeunemaitre, and Juliette Albuisson. "Pseudoxanthoma elasticum with prominent arterial calcifications evoking CD73 deficiency." Vascular Medicine 24, no. 5 (2019): 461–64. http://dx.doi.org/10.1177/1358863x19853360.

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Pseudoxanthoma elasticum (PXE) is a rare disorder characterized by skin, eye, and cardiovascular lesions due to ectopic mineralization and fragmentation of elastic fibers of connective tissues. We present an atypical case of PXE with diffuse vascular calcification and negligible skin and eye lesions. The patient was a 37-year-old man suffering from severe bilateral arterial calcifications in superficial femoral and posterior tibial arteries. Eye fundoscopy and skin examination were first considered normal. This phenotype suggested first the diagnosis of Arterial Calcification due to Deficiency

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Seely, Stephen. "On arterial calcification." International Journal of Cardiology 61, no. 2 (1997): 105–8. http://dx.doi.org/10.1016/s0167-5273(97)00131-9.

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Ho, Chin Yee, and Catherine M. Shanahan. "Medial Arterial Calcification." Arteriosclerosis, Thrombosis, and Vascular Biology 36, no. 8 (2016): 1475–82. http://dx.doi.org/10.1161/atvbaha.116.306717.

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Polonsky, Tamar S., and Philip Greenland. "Breast Arterial Calcification." Circulation 135, no. 6 (2017): 499–501. http://dx.doi.org/10.1161/circulationaha.116.025277.

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Lanzer, Peter, Fadil M. Hannan, Jan D. Lanzer, et al. "Medial Arterial Calcification." Journal of the American College of Cardiology 78, no. 11 (2021): 1145–65. http://dx.doi.org/10.1016/j.jacc.2021.06.049.

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Bäck, Magnus, and Jean-Baptiste Michel. "From organic and inorganic phosphates to valvular and vascular calcifications." Cardiovascular Research 117, no. 9 (2021): 2016–29. http://dx.doi.org/10.1093/cvr/cvab038.

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Abstract Calcification of the arterial wall and valves is an important part of the pathophysiological process of peripheral and coronary atherosclerosis, aortic stenosis, ageing, diabetes, and chronic kidney disease. This review aims to better understand how extracellular phosphates and their ability to be retained as calcium phosphates on the extracellular matrix initiate the mineralization process of arteries and valves. In this context, the physiological process of bone mineralization remains a human model for pathological soft tissue mineralization. Soluble (ionized) calcium precipitation

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Rennenberg, Roger J. M. W., Bernard J. van Varik, Leon J. Schurgers, et al. "Chronic coumarin treatment is associated with increased extracoronary arterial calcification in humans." Blood 115, no. 24 (2010): 5121–23. http://dx.doi.org/10.1182/blood-2010-01-264598.

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Abstract Vascular calcification is a marker of increased cardiovascular risk. Vitamin K–dependent matrix Gla protein (MGP) is important in inhibiting calcification. Because MGP activation is vitamin K dependent, we performed a cross-sectional study investigating the relationship between the use of vitamin K antagonists and extracoronary vascular calcification. From the Dutch thrombosis services we selected 19 patients younger than 55 years who had no other cardiovascular risk factors and who had used coumarins for more than 10 years, and compared these to 18 matched healthy controls. MGP was m

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Dissertations / Theses on the topic "Arterial calcification"

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Young, Melissa Denton. "Arterial Calcification and the Clinical Implications on Stent Function." Case Western Reserve University School of Graduate Studies / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=case1364999090.

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Curinga, Gabrielle Mercedes. "The role of runt-related transcription factor 2 in arterial smooth muscle cell mineralization /." Thesis, Connect to this title online; UW restricted, 2003. http://hdl.handle.net/1773/6353.

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Ford, Martin. "The role of calcification regulatory proteins in the arterial stiffening of chronic kidney disease stages 3 & 4." Thesis, University of Brighton, 2012. https://research.brighton.ac.uk/en/studentTheses/61dae01b-dd29-4dd8-acfd-36f6021d4140.

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Background: Chronic Kidney Disease (CKD) is common and is associated with an increased cardiovascular morbidity and mortality which is not completely explained by traditional risk factors. Non-traditional risk factors include arterial stiffening and calcification. Several calcification regulatory proteins (CaRP) are implicated in arterial stiffening. Fetuin-A may be important in inhibiting mineralisation via physicochemical interaction with calcium and phosphate, and can form circulating CalciProtein Particles (CPP) in pro-calcific states. Osteoprotegerin (OPG) & Receptor Activator of Nuclear

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Fish, R. S. "A study of the P2X7 purinoceptor and vascular ATP metabolic pathways in chronic kidney disease-associated arterial calcification." Thesis, University College London (University of London), 2014. http://discovery.ucl.ac.uk/1456574/.

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The risk of cardiovascular-related death is several-fold higher in patients with chronic kidney disease (CKD) compared with the general population. Arterial calcification (AC) is extremely common in patients with CKD and strongly associates with cardiovascular-related mortality, however, there are currently no specific treatments to prevent its development and/or progression. Abundant evidence now suggests that AC is cell-mediated and actively-regulated, involving mechanisms linked to bone homeostasis, production of calcification inhibitors and vascular smooth muscle cell (VSMC) function. The

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Martins, Christine Men [UNESP]. "Influência da hipertensão arterial no padrão fenotípico da lesão periapical, na diferenciação dos osteoclastos, na resposta teciduale na capacidade de mineralização do cimento reparador MTA." Universidade Estadual Paulista (UNESP), 2016. http://hdl.handle.net/11449/138048.

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Submitted by CHRISTINE MEN MARTINS null (christinemen@hotmail.com) on 2016-04-20T16:44:53Z No. of bitstreams: 1 Tese Ciência Odontológica - Christine Men Martins.pdf: 3676381 bytes, checksum: c35e89b5716dcdfe110457cff21f6d5c (MD5)<br>Approved for entry into archive by Juliano Benedito Ferreira (julianoferreira@reitoria.unesp.br) on 2016-04-25T17:40:01Z (GMT) No. of bitstreams: 1 martins_cm_dr_araca.pdf: 3676381 bytes, checksum: c35e89b5716dcdfe110457cff21f6d5c (MD5)<br>Made available in DSpace on 2016-04-25T17:40:01Z (GMT). No. of bitstreams: 1 martins_cm_dr_araca.pdf: 3676381 bytes, check

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Rodríguez, María Jaime. "Modelling of the mechanobiological adaptation to vascular occlusion in the arterial tree." Thesis, Linköpings universitet, Mekanik och hållfasthetslära, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-129339.

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It is known that there are many cardiovascular diseases caused by the alterations in the blood vessels, that affect most of the world population. The knowledge of the mechanobiological behavior of blood vessels is used for understanding how cardiovascular diseases could affect the human body. So, by studying the growth and remodeling (G&R) of the arterial tree, it is possible to predict how these diseases will develop and consequently, how they can be treated or even prevented. The human body naturally tries to find the optimum steady-state by changing either the production of the constitu

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Craiem, Damian. "Développement et évaluation de nouveaux outils d'analyse géométrique 3D pour la prévention et le traitement des maladies aortiques." Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCB058/document.

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Les nouvelles technologies d'imagerie basées sur la tomodensitométrie en coupe permettent l'évaluation de très haute qualité de la structure 3D de l'aorte thoracique. La reconstruction virtuelle et les modèles géométriques de l'aorte sont indispensables à l'exploitation des images dont le temps de traitement manuel reste cependant considérable et les outils numériques insuffisants ou inadaptés pour mesurer correctement sa morphologie. L'aorte n'est pas un simple tube de conduction du sang mais un organe de régulation de la pulsatilité des ondes de pression provoquées par l'éjection cardiaque.

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Cancela, Ana Ludimila Espada. "Avaliação da relação entre metabolismo mineral e doença arterial coronariana em pacientes com função renal preservada." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-30112011-174239/.

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INTRODUÇÃO: Os níveis séricos de fósforo (P) têm sido associados a doenças cardiovasculares e mortalidade em pacientes com doença renal crônica e na população geral. Estudos in vitro demonstram que altas concentrações de fósforo extracellular são capazes de induzir calcificação vascular e disfunção endotelial. O Fibroblast Growth Factor 23 (FGF-23) é um hormônio fosfatúrico e foi relacionado à presença de aterosclerose em pacientes idosos. OBJETIVO: O objetivo deste estudo foi investigar as relações entre P, FGF-23 e outros atores do metabolismo mineral e a ocorrência de doença arterial corona

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Bruscato, Neide Maria. "Calcificação coronária e sua associação com fatores de risco cardiovascular e hábitos dietéticos em homens assintomáticos vivendo em comunidade." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2016. http://hdl.handle.net/10183/157678.

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Introdução: As doenças cardiovasculares (DCV) são a principal causa de morte no mundo, sendo a doença arterial coronária (DAC) a mais comum das DCV, juntamente com acidente vascular cerebral. O cálcio das artérias coronárias é um marcador da DAC subclínica (assintomática) e é preditivo de eventos coronários futuros. Vários fatores de risco cardiovascular contribuem para o desenvolvimento da calcificação da artéria coronária (CAC). Adicionalmente, os fatores dietéticos podem influenciar no processo de aterosclerose e CAC. Objetivo: Avaliar a CAC e sua associação com fatores de risco cardiovascu

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Ferreira, Juliana Cunha. "Efeito do carbonato de cálcio e do carbonato de sevelamer na remodelação óssea e na calcificação arterial em um modelo experimental de uremia com doença óssea adinâmica." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-09082013-131710/.

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INTRODUÇÃO: Há poucos modelos experimentais de doença óssea adinâmica (DOA) e os mecanismos fisiopatológicos dessa doença não são completamente compreendidos. Além disso, os efeitos dos quelantes de fósforo (P) na DOA têm sido pouco estudados. Este estudo objetivou estabelecer um modelo de DOA e avaliar os efeitos da terapia com carbonato de cálcio (Ca) e carbonato de sevelamer (sevelamer) sobre os distúrbios do metabolismo mineral e ósseo da doença renal crônica (DMO-DRC), no modelo estabelecido. MÉTODOS: Experimento 1: A DOA e a DRC foram induzidas por nefrectomia 5/6 (Nx) e paratireoidectom

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Books on the topic "Arterial calcification"

1

Mulhall, Douglas. The calcium bomb: The nanobacteria link to heart disease & cancer. Writers' Collective, 2005.

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Covic, Adrian, Mugurel Apetrii, Luminita Voroneanu, and David J. Goldsmith. Vascular calcification. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0120_update_001.

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Vascular calcification (VC) is a common feature of patients with advanced CKD and it could be, at least in part, the cause of increased cardiovascular mortality in these patients. From a morphologic point of view, there are at least two types of pathologic calcium phosphate deposition in the arterial wall—namely, intima calcification (mostly associated with atherosclerotic plaques) and media calcification (associated with stiffening of the vasculature, resulting in significantly adverse cardiovascular outcomes). Although VC was viewed initially as a passive phenomenon, it appears to be a cell-

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Aikawa, Elena. Cardiovascular Imaging: Arterial and Aortic Valve Inflammation and Calcification. Springer, 2014.

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Aikawa, Elena. Cardiovascular Imaging: Arterial and Aortic Valve Inflammation and Calcification. Springer, 2016.

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Kahn, S. Lowell. Techniques for Traversing Difficult Aortic Bifurcations and Aortobifemoral Grafts. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0023.

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Contralateral extremity interventions are a mainstay of endovascular therapy for the lower extremity. The retrograde common femoral arterial access for contralateral infrainguinal interventions remains one of the most common access sites. Traversing the aortic bifurcation for an up-and-over approach is readily accomplished with little difficulty, but sharp angulation, severe calcification, iliac occlusive disease, kissing common iliac stents, and surgical or endovascular bifurcations can make this challenging or impossible. This chapter describes multiple techniques to transverse the difficult

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Madassery, Sreekumar. Obtaining Hemostasis at Puncture Sites. Edited by S. Lowell Kahn, Bulent Arslan, and Abdulrahman Masrani. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199986071.003.0046.

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This chapter pertains to femoral artery access. Attaining an “ideal” access is essential for maximizing the chances of maintaining effective hemostasis. Deciding whether to perform arterial access closure using the gold standard of manual compression versus a vascular closure device (VCD) requires the operator to evaluate many characteristics of the patient. The location of the arteriotomy, vessel size, degree of calcification, use of anticoagulants, and sheath size during the procedure are the primary factors that need to be considered. The ISAR-CLOSURE study reported that use of VCDs was not

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Holbrook, Anna I. Vascular Calcifications. Edited by Christoph I. Lee, Constance D. Lehman, and Lawrence W. Bassett. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190270261.003.0034.

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Vascular calcifications lie within the artery wall, and appear to be linear, usually in association with blood vessels. The parallel or “tram-track” appearance of the calcifications in opposite walls of the artery is pathognomonic. They are more prevalent with age. They are also associated with diabetes, renal disease, hyperparathyroidism, parity, and history of lactation, and possibly, cardiovascular disease. Vascular calcifications in the breast are within the arterial wall media, where they are known as Mönckeberg medial calcific sclerosis. This chapter reviews the key imaging features, ima

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Levy, David. Macrovascular complications, hypertension, and lipids. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198766452.003.0008.

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Premature vascular disease is common in Type 1 diabetes, especially in women and those with long duration. Many studies have identified early vascular involvement, using carotid Doppler and coronary artery calcification. Symptoms of coronary heart disease are often absent or muted, and the best methods for identifying occult coronary heart disease in Type 1 patients are not known. The concept of ideal cardiovascular health is valuable in planning preventive lifestyle and medical interventions. ‘Essential’ hypertension in young Type 1 patients is common, and reflects increased arterial stiffnes

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Appel, Annette Marie. Dietary intake and coronary artery calcification. 1994.

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Mulhall, Douglas, and Katja Hansen. Has Heart Disease Been Cured? Writers' Collective, 2003.

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Book chapters on the topic "Arterial calcification"

1

Procaccini, Luca, Marzia Olivieri, Francesco Lorenzo Serafini, Cesare Mantini, Erica Maffei, and Filippo Cademartiri. "Imaging Peripheral Arterial Calcifications." In Cardiovascular Calcification. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-81515-8_10.

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Moawad, Magdy R. "Management of Peripheral Arterial Calcification." In Cardiovascular Calcification. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-81515-8_11.

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Jashari, Fisnik, Per Wester, and Michael Henein. "Arterial Calcification and Cerebral Disease: Stroke and Dementia." In Cardiovascular Calcification. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-81515-8_12.

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Chen, Harold. "Generalized Arterial Calcification of Infancy." In Atlas of Genetic Diagnosis and Counseling. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-2401-1_104.

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Chen, Harold. "Generalized Arterial Calcification of Infancy." In Atlas of Genetic Diagnosis and Counseling. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6430-3_104-2.

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Rogers, Maximillian A., and Elena Aikawa. "Differential Mechanisms of Arterial and Valvular Calcification." In Contemporary Cardiology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-46725-8_4.

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Vervloet, Marc G. "Reversing Arterial Stiffening and Calcification: A Pipe Dream?" In Cardio-Renal Clinical Challenges. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09162-4_13.

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Cheng, Jie-Zhi, Elodia B. Cole, Etta D. Pisano, and Dinggang Shen. "Detection of Arterial Calcification in Mammograms by Random Walks." In Lecture Notes in Computer Science. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-642-02498-6_59.

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Mazidi, Najim, Carl Roobottom, and Giovanni Masala. "Automatic Quantification of Breast Arterial Calcification on Mammographic Images." In Innovation in Medicine and Healthcare Systems, and Multimedia. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-8566-7_28.

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Sakamoto, Atsushi, Yu Sato, Aloke V. Finn, and Renu Virmani. "Basic Pathology of Arterial and Valvular Calcification in Humans." In Contemporary Cardiology. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-46725-8_2.

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Conference papers on the topic "Arterial calcification"

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Holm, Mikayle, and Paul A. Iaizzo. "Distributions of Arterial Calcification Along Transcatheter Delivery System Pathway." In 2019 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2019. http://dx.doi.org/10.1115/dmd2019-3259.

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Medical device designers must develop catheters that avoid harming the vascular pathway during therapy delivery. To better characterize the necessary design features to minimize damage to central and peripheral vessels, the vascular pathway used for femoral delivery of a cardiac device is studied through high resolution, computed tomography images of fresh, human cadavers. Comparisons of the amount and location of vessel calcification are made between specified anatomical sections. Though the potential complications may be less fatal, the descending aorta and iliac regions were found to be mos

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Canton, Gador, Dalin Tang, Daniel S. Hippe, and Chun Yuan. "Distensibility of the Atherosclerotic Carotid Artery: Relationship With Plaque Burden and Composition?" In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80845.

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Arterial distensibility is a marker that can measure vessel wall functional and structural changes resulting from atherosclerosis [5] with applications including estimation of mechanical properties of the wall for biomechanical models. Although arterial segments affected by atherosclerosis are characterized by marked stiffening [2], little is known about the relationship between local specific atherosclerotic plaque features and wall stiffness. In particular, calcification has been shown to be associated with greater wall stiffness, however, this relationship is not consistent in different art

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Wang, Kaier, Nabeel Khan, and Ralph Highnam. "Automated Segmentation of Breast Arterial Calcifications from Digital Mammography." In 2019 International Conference on Image and Vision Computing New Zealand (IVCNZ). IEEE, 2019. http://dx.doi.org/10.1109/ivcnz48456.2019.8960956.

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Xenos, Michalis, Suraj Rambhia, Yared Alemu, et al. "A Patient Based Approach for Fluid Structure Interaction in Ruptured Abdominal Aortic Aneurysms." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206490.

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Fluid structure interaction (FSI) simulations were conducted to assess the risk of rupture in reconstructed AAA from patients who had contained ruptured AAAs. The goal was to test to ability of our FSI methodology to predict the location of rupture, by correlating the high wall stress regions with the actual rupture location. We also present a parametric study in which the relationship of iliac bifurcation angle and the role of embedded calcifications were studied in respect to the aneurismal wall stress. The patient specific AAA FSI simulations were carried out with advanced constitutive mate

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Berrahmoune, Nousra, Mamdouh S. Al-Enezi, Abdelillah Douhi, et al. "Coupling of CT and PET-18F-FDG Imaging in Arteries with Calcification." In 2020 IEEE Nuclear Science Symposium and Medical Imaging Conference (NSS/MIC). IEEE, 2020. http://dx.doi.org/10.1109/nss/mic42677.2020.9507834.

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Xenos, Michalis, Suraj Rambhia, Yared Alemu, et al. "A Fluid Structure Interaction Approach for Patient Based Abdominal Aortic Aneurysm Rupture Risk Prediction." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19298.

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Fluid structure interaction (FSI) simulations of patient-specific fusiform non-ruptured and contained ruptured Abdominal Aortic Aneurysm (AAA) geometries were conducted. The goals were: (1) to test the ability of our FSI methodology to predict the location of rupture, by correlating the high wall stress regions with the rupture location, (2) estimate the state of the pathological condition by calculating the ruptured potential index (RPI) of the AAA and (3) predict the disease progression by comparing healthy and pathological aortas. The patient specific AAA FSI simulations were carried out wi

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Boekhoven, Renate W., Richard G. P. Lopata, Marcel C. M. Rutten, Marc R. H. M. van Sambeek, and Frans N. van de Vosse. "Novel Strategy of the Determination of Mechanical Properties of Human Carotid Atherosclerotic Plaques." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80669.

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Carotid endarterectomy is the procedure of choice in patients with a recent symptomatic stenosis of 70–99%. Currently, the selection of candidates eligible for carotid endarterectomy is based on stenosis size only. However, the treatment is only beneficial for patients with unstable plaques, which comprises only 16% of the patient population [1]. Hence, identifying plaque stability at an early stage would permit timely intervention, while substantially reducing overtreatment of stable plaques. The objective of this study is to distinguish between stable and unstable carotid atherosclerotic pla

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Biswas, Dipankar, Francis Loth, Susan McCormick, and Hisham Bassiouny. "Three Dimensional Reconstruction of the Components of the Carotid Plaque From Standard CT Medical Images." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53750.

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The present study compares plaque and calcium volumes between sixteen diseased human carotid arteries. Half were from symptomatic patients, and half were from asymptomatic patients. In-house software was developed for the 3-D reconstruction of the plaque components from computerized tomography (CT) images. Results revealed higher mean total volume (TV) in the symptomatic group compared to the asymptomatic group. In contrast, the mean ratio of calcium volume (CV) to TV was lower for the symptomatic group compared to the asymptomatic group. This shows that symptomatic patients have a significant

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Kapnisis, Konstantinos, Dina Halwani, Brigitta Brott, Jack Lemons, Peter Anderson, and Andreas Anayiotos. "Stent Overlapping and Geometric Curvature Influence the Structural Integrity and Surface Characteristics of Coronary Stents." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80231.

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Abstract:

A common clinical practice in interventional procedures, is the deployment of two or more overlapping stents, especially in areas of branches and bifurcations where the stenotic area is diffuse and cannot be covered by a single stent or in the cases of treating long or recurrent lesions. However, complex in vivo conditions such as vessel tortuousity, high curvature, vascular wall stresses as well as blood flow wall shear stresses and diffuse calcification, may cause additional interactions within overlapping stents resulting in enhanced surface damage and fracture of stents. Preliminary studie

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