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Relevant bibliographies by topics / Arterial calcification / Dissertations / Theses

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Dissertations / Theses on the topic 'Arterial calcification'

Author: Grafiati

Published: 4 June 2021

Last updated: 3 February 2022

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1

Young, Melissa Denton. "Arterial Calcification and the Clinical Implications on Stent Function." Case Western Reserve University School of Graduate Studies / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=case1364999090.

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2

Curinga, Gabrielle Mercedes. "The role of runt-related transcription factor 2 in arterial smooth muscle cell mineralization /." Thesis, Connect to this title online; UW restricted, 2003. http://hdl.handle.net/1773/6353.

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3

Ford, Martin. "The role of calcification regulatory proteins in the arterial stiffening of chronic kidney disease stages 3 & 4." Thesis, University of Brighton, 2012. https://research.brighton.ac.uk/en/studentTheses/61dae01b-dd29-4dd8-acfd-36f6021d4140.

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Background: Chronic Kidney Disease (CKD) is common and is associated with an increased cardiovascular morbidity and mortality which is not completely explained by traditional risk factors. Non-traditional risk factors include arterial stiffening and calcification. Several calcification regulatory proteins (CaRP) are implicated in arterial stiffening. Fetuin-A may be important in inhibiting mineralisation via physicochemical interaction with calcium and phosphate, and can form circulating CalciProtein Particles (CPP) in pro-calcific states. Osteoprotegerin (OPG) & Receptor Activator of Nuclear

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4

Fish, R. S. "A study of the P2X7 purinoceptor and vascular ATP metabolic pathways in chronic kidney disease-associated arterial calcification." Thesis, University College London (University of London), 2014. http://discovery.ucl.ac.uk/1456574/.

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The risk of cardiovascular-related death is several-fold higher in patients with chronic kidney disease (CKD) compared with the general population. Arterial calcification (AC) is extremely common in patients with CKD and strongly associates with cardiovascular-related mortality, however, there are currently no specific treatments to prevent its development and/or progression. Abundant evidence now suggests that AC is cell-mediated and actively-regulated, involving mechanisms linked to bone homeostasis, production of calcification inhibitors and vascular smooth muscle cell (VSMC) function. The

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5

Martins, Christine Men [UNESP]. "Influência da hipertensão arterial no padrão fenotípico da lesão periapical, na diferenciação dos osteoclastos, na resposta teciduale na capacidade de mineralização do cimento reparador MTA." Universidade Estadual Paulista (UNESP), 2016. http://hdl.handle.net/11449/138048.

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Submitted by CHRISTINE MEN MARTINS null (christinemen@hotmail.com) on 2016-04-20T16:44:53Z No. of bitstreams: 1 Tese Ciência Odontológica - Christine Men Martins.pdf: 3676381 bytes, checksum: c35e89b5716dcdfe110457cff21f6d5c (MD5)<br>Approved for entry into archive by Juliano Benedito Ferreira (julianoferreira@reitoria.unesp.br) on 2016-04-25T17:40:01Z (GMT) No. of bitstreams: 1 martins_cm_dr_araca.pdf: 3676381 bytes, checksum: c35e89b5716dcdfe110457cff21f6d5c (MD5)<br>Made available in DSpace on 2016-04-25T17:40:01Z (GMT). No. of bitstreams: 1 martins_cm_dr_araca.pdf: 3676381 bytes, check

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6

Rodríguez, María Jaime. "Modelling of the mechanobiological adaptation to vascular occlusion in the arterial tree." Thesis, Linköpings universitet, Mekanik och hållfasthetslära, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-129339.

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It is known that there are many cardiovascular diseases caused by the alterations in the blood vessels, that affect most of the world population. The knowledge of the mechanobiological behavior of blood vessels is used for understanding how cardiovascular diseases could affect the human body. So, by studying the growth and remodeling (G&R) of the arterial tree, it is possible to predict how these diseases will develop and consequently, how they can be treated or even prevented. The human body naturally tries to find the optimum steady-state by changing either the production of the constitu

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7

Craiem, Damian. "Développement et évaluation de nouveaux outils d'analyse géométrique 3D pour la prévention et le traitement des maladies aortiques." Thesis, Sorbonne Paris Cité, 2016. http://www.theses.fr/2016USPCB058/document.

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Les nouvelles technologies d'imagerie basées sur la tomodensitométrie en coupe permettent l'évaluation de très haute qualité de la structure 3D de l'aorte thoracique. La reconstruction virtuelle et les modèles géométriques de l'aorte sont indispensables à l'exploitation des images dont le temps de traitement manuel reste cependant considérable et les outils numériques insuffisants ou inadaptés pour mesurer correctement sa morphologie. L'aorte n'est pas un simple tube de conduction du sang mais un organe de régulation de la pulsatilité des ondes de pression provoquées par l'éjection cardiaque.

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8

Cancela, Ana Ludimila Espada. "Avaliação da relação entre metabolismo mineral e doença arterial coronariana em pacientes com função renal preservada." Universidade de São Paulo, 2011. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-30112011-174239/.

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INTRODUÇÃO: Os níveis séricos de fósforo (P) têm sido associados a doenças cardiovasculares e mortalidade em pacientes com doença renal crônica e na população geral. Estudos in vitro demonstram que altas concentrações de fósforo extracellular são capazes de induzir calcificação vascular e disfunção endotelial. O Fibroblast Growth Factor 23 (FGF-23) é um hormônio fosfatúrico e foi relacionado à presença de aterosclerose em pacientes idosos. OBJETIVO: O objetivo deste estudo foi investigar as relações entre P, FGF-23 e outros atores do metabolismo mineral e a ocorrência de doença arterial corona

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9

Bruscato, Neide Maria. "Calcificação coronária e sua associação com fatores de risco cardiovascular e hábitos dietéticos em homens assintomáticos vivendo em comunidade." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2016. http://hdl.handle.net/10183/157678.

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Introdução: As doenças cardiovasculares (DCV) são a principal causa de morte no mundo, sendo a doença arterial coronária (DAC) a mais comum das DCV, juntamente com acidente vascular cerebral. O cálcio das artérias coronárias é um marcador da DAC subclínica (assintomática) e é preditivo de eventos coronários futuros. Vários fatores de risco cardiovascular contribuem para o desenvolvimento da calcificação da artéria coronária (CAC). Adicionalmente, os fatores dietéticos podem influenciar no processo de aterosclerose e CAC. Objetivo: Avaliar a CAC e sua associação com fatores de risco cardiovascu

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10

Ferreira, Juliana Cunha. "Efeito do carbonato de cálcio e do carbonato de sevelamer na remodelação óssea e na calcificação arterial em um modelo experimental de uremia com doença óssea adinâmica." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-09082013-131710/.

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INTRODUÇÃO: Há poucos modelos experimentais de doença óssea adinâmica (DOA) e os mecanismos fisiopatológicos dessa doença não são completamente compreendidos. Além disso, os efeitos dos quelantes de fósforo (P) na DOA têm sido pouco estudados. Este estudo objetivou estabelecer um modelo de DOA e avaliar os efeitos da terapia com carbonato de cálcio (Ca) e carbonato de sevelamer (sevelamer) sobre os distúrbios do metabolismo mineral e ósseo da doença renal crônica (DMO-DRC), no modelo estabelecido. MÉTODOS: Experimento 1: A DOA e a DRC foram induzidas por nefrectomia 5/6 (Nx) e paratireoidectom

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11

Fung, Po-yan Eliza, and 馮寶恩. "Breast arterial calcifications in mammograms and their association with cardiovascular risk factors." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B42994706.

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12

Fung, Po-yan Eliza. "Breast arterial calcifications in mammograms and their association with cardiovascular risk factors." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B42994706.

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13

Fahel, Mateus Guimarães. "Integração do estudo anatômico coronariano através da angiotomografia/escore de cálcio ao estudo funcional de perfusão miocárdica pelo PET-CT utilizando rubídio na investigação da doença arterial coronariana." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/5/5151/tde-12012018-094159/.

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Introdução: A doença arterial coronariana (DAC) persiste com alta morbimortalidade. Várias modalidades diagnósticas não-invasivas estão disponíveis para sua avaliação, incluindo escore de cálcio coronariano (EC), angiotomografia coronariana (AngioTC) e tomografia por emissão de pósitrons com rubídio (PET-CT82Rb), com ótimas sensibilidade e especificidade. A integração destes métodos em um exame híbrido permite delinear extensão anatômica e funcional da aterosclerose, possibilitando diagnósticos mais corretos. Objetivo: Avaliar anatomia coronariana e perfusão miocárdica de pacientes suspeitos o

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14

Miranda, Érique José Peixoto de. "Associação entre doença tireoidiana subclínica, aterosclerose coronariana, índice de espessura de médio-íntima carotídea e rigidez arterial aórtica em análise transversal do Estudo Longitudinal de Saúde do Adulto (ELSA-Brasil)." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/5/5169/tde-19062017-154141/.

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Introdução: Doenças tireoidianas subclínicas incluem hipotireoidismo e hipertireoidismo subclínicos. A associação entre doença tireoidiana subclínica e morbimortalidade cardiovascular é controversa e os dados sobre a relação entre essas condições clínicas e aterosclerose subclínica são escassos. Objetivos: Este estudo objetiva avaliar a associação entre doença tireoidiana subclínica, calcificação arterial coronariana (CAC), doença arterial coronariana (DAC), índice de espessura de médio-íntima carotídea média (IMT) e velocidade de onda de pulso carotídeo-femoral (cf-VOP) no Estudo Longitudinal

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15

Sadekova, Nataliya. "La rigidité artérielle, induite par une calcification des carotides, altère l’homéostasie cérébrale chez la souris." Thèse, 2013. http://hdl.handle.net/1866/10290.

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La rigidité artérielle est considérée comme un facteur de risque important pour le développement du déclin cognitif. Toutefois, les effets précis de la rigidité artérielle sur le cerveau sont peu connus et, à ce jour, aucun modèle animal ne permet d’étudier l’effet isolé de ce facteur sur l’homéostasie cérébrale. Dans cette étude, nous avons développé un nouveau modèle de rigidité artérielle qui se base sur la calcification de l’artère carotide chez la souris. Au niveau artériel, ce modèle présente une fragmentation de l’élastine, une augmentation de la distribution du collagène et de l’épaiss

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16

Doyon, Marielle. "L'accélération de la rigidité vasculaire associée au diabète de type 1 : implication de la protéine Gla de la matrice." Thèse, 2013. http://hdl.handle.net/1866/12802.

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L'hypertension systolique isolée (HSI), amenée par une augmentation de la rigidité vasculaire, est la forme d'hypertension la plus fréquente chez les personnes âgées de plus de 60 ans. L'augmentation de la rigidité vasculaire, causée en partie par la calcification aortique médiale, est accélérée de 15 ans chez les diabétiques. Il est suggéré que la calcification aortique serait responsable de la résistance aux agents antihypertenseurs chez les patients souffrant d'HSI, d'où la nécessité de développer de nouvelles stratégies thérapeutiques ciblant la calcification artérielle. La protéine Gla

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17

Moreau, Simon. "Modifications de la matrice extracellulaire dans la rigidité artérielle." Thèse, 2010. http://hdl.handle.net/1866/5103.

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La paroi vasculaire est composée de cellules endothéliales, de cellules musculaires lisses vasculaires et de fibroblastes qui sont entourés d’un réseau structuré et complexe de protéines, la matrice extracellulaire. Les interactions réciproques entre la matrice et les cellules sont nécessaires à la croissance, au développement et au remodelage. Or, différents contextes pathologiques entraînent la perturbation de ces interactions et sont la cause de différentes maladies. Au cours du vieillissement, la matrice extracellulaire des grosses artères élastiques est modifiée. Ainsi, les lamelles élast

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18

Bloch, Sherri. "Exploration of the Cerebral Dysfunctions Induced by Arterial Rigidity and/or the Overexpression of TGFβ in a Mouse Model". Thèse, 2017. http://hdl.handle.net/1866/20528.

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19

Moodley, Karanigie. "An assessment of coronary artery calcification, using the calcium scoring technique, in an asymptomatic Indian population in Durban, KwaZulu-Natal." Thesis, 2008. http://hdl.handle.net/10321/359.

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Thesis (M.Tech.: Radiography)-Dept. of Radiography, Durban University of Technology, 2008. xxi, 146 leaves, Appendices A-S<br>The main aim of this study, was to assess the prevalence of coronary artery calcification in asymptomatic risk and non risk individuals in the South African Indian population, within the age group of 20-70 years.

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