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Journal articles on the topic 'Arterial media'

Author: Grafiati

Published: 4 June 2021

Last updated: 8 February 2022

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1

Boonen, H. C., P. M. Schiffers, G. E. Fazzi, G. M. Janssen, M. J. Daemen, and J. G. De Mey. "DNA synthesis in isolated arteries. Kinetics and structural consequences." American Journal of Physiology-Heart and Circulatory Physiology 260, no. 1 (1991): H210—H217. http://dx.doi.org/10.1152/ajpheart.1991.260.1.h210.

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We evaluated changes in DNA synthesis, structure, and mechanical activity in isolated arteries during exposure to growth factors. Renal arteries were isolated from rats, sympathectomized, denuded of endothelium, and maintained in tissue culture. Up to 4 days of culture did not affect maximal contractile responses to depolarization. From the results of nuclear incorporation of the thymidine analogue 5-bromo-2-deoxyuridine (BrdUrd), culture stimulated DNA synthesis. In the media, incorporation of BrdUrd was maximal after 3 days but fell precipitously thereafter. Culture of arterial segments did

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2

Gać, Paweł, Małgorzata Poręba, Lidia Januszewska, et al. "The Total Antioxidant Status, Serum Selenium Concentrations and the Ultrasound Assessment Carotid Intima Media Thickness in Patients with Arterial Hypertension." Antioxidants 10, no. 1 (2021): 63. http://dx.doi.org/10.3390/antiox10010063.

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The objective of the study was to establish the correlation between serum selenium concentrations, total antioxidant status, and the carotid intima media thickness in ultrasound assessment in patients with arterial hypertension. A group of 76 people suffering from arterial hypertension was qualified to participate in the study. The mean age of the respondents was 53.48 ± 12.78. Serum selenium concentrations (Se-S) and total antioxidant status (TAS) were determined in all respondents. Se-S were determined by hydride generation atomic absorption spectroscopy (HGAAS). The antioxidant status was a

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Van den Bergh, Geoffrey, Britt Opdebeeck, Patrick C. D’Haese, and Anja Verhulst. "The Vicious Cycle of Arterial Stiffness and Arterial Media Calcification." Trends in Molecular Medicine 25, no. 12 (2019): 1133–46. http://dx.doi.org/10.1016/j.molmed.2019.08.006.

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4

Werber, A. H., and D. D. Heistad. "Diffusional support of arteries." American Journal of Physiology-Heart and Circulatory Physiology 248, no. 6 (1985): H901—H906. http://dx.doi.org/10.1152/ajpheart.1985.248.6.h901.

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The relative role of the arterial lumen, adventitial vasa vasorum, and medial vasa vasorum in nutritional support of arteries is unclear. We have used a newly developed autoradiographic method to study diffusion of metabolically inert [14C]antipyrine into arteries to determine the relative importance of different pathways in nutritional support of arteries. [14C]antipyrine was homogenously distributed across the media of small arteries within 15 s, which indicates that diffusion into the central media was rapid. In the thoracic and abdominal aorta, levels of antipyrine were higher in the inner

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Grassl, E. D., T. R. Oegema, and R. T. Tranquillo. "A fibrin-based arterial media equivalent." Journal of Biomedical Materials Research 66A, no. 3 (2003): 550–61. http://dx.doi.org/10.1002/jbm.a.10589.

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Vasconcelos, Rosemeri Oliveira, Severo Sales de Barros, Denise Russowski, Silvia Maria Grando, and Luiz Francisco Irigoyen. "Arterial diffuse intimal thickening associated with enzootic calcinosis of sheep." Pesquisa Veterinária Brasileira 18, no. 1 (1998): 9–15. http://dx.doi.org/10.1590/s0100-736x1998000100002.

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Morphometric, immunohistochemical and ultrastructural studies were carried out on the diffuse intimal thickening (DIT) in arteries of 7 sheep with clinical signs of naturally occurring enzootic calcinosis due to ingestion of the plant Nierembergia veitchii. Arterial lesions consisted of medial deposition of calcium salts and DIT. Calcification of the intima was rare, mild and located near the elastic lamina. By immunohistochemistry <FONT FACE="Symbol">a</FONT>-actin was detected in cells of the media and in cells forming the intimal thickening. Receptors for 1,25(OH)2 vitamin D3 we

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Žaloudíková, Marie, Adam Eckhardt, Richard Vytášek, et al. "Decreased collagen VI in the tunica media of pulmonary vessels during exposure to hypoxia: a novel step in pulmonary arterial remodeling." Pulmonary Circulation 9, no. 3 (2019): 204589401986074. http://dx.doi.org/10.1177/2045894019860747.

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The development of hypoxic pulmonary hypertension is characterized by the structural remodeling of pulmonary arteries. However, the relationship between changes of arterial cells and the extracellular matrix remains unclear. We focused on the evaluation of the non-fibrillar collagen changes in tunica media induced by a four-day exposure to hypoxia and the correlation of these changes with the pulmonary arterial wall structure modifications. We used 20 adult male Wistar rats. The amount and localization of collagen VI, collagen IV, matrix metalloproteinase (MMP) 2, and MMP9 were tested in pulmo

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HUGHES, Jennifer M., and Stuart J. BUND. "Influence of experimental reduction of media/lumen ratio on arterial myogenic properties of spontaneously hypertensive and Wistar–Kyoto rats." Clinical Science 106, no. 2 (2004): 163–71. http://dx.doi.org/10.1042/cs20030225.

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In the present study, myogenic properties of femoral arteries from control hindlimbs and those distal to external iliac artery partial ligation of spontaneously hypertensive rats (SHRs) and normotensive Wistar–Kyoto (WKY) rats were assessed. Arterial pressure was reduced distal to the ligature in both strains. Media thickness/lumen diameter (M/L) ratios of control (unligatured) SHRs were greater than in unligatured WKY rats and were reduced in arteries distal to the ligature (ligatured) within each strain. In none of the comparisons was a greater M/L ratio associated with greater maximal myoge

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Bois, Melanie C., Adam M. May, Robert Vassallo, Sarah M. Jenkins, Eunhee S. Yi, and Anja C. Roden. "Morphometric Study of Pulmonary Arterial Changes in Pulmonary Langerhans Cell Histiocytosis." Archives of Pathology & Laboratory Medicine 142, no. 8 (2018): 929–37. http://dx.doi.org/10.5858/arpa.2017-0463-oa.

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Context.— Pulmonary hypertension (PHT) is a complication of pulmonary Langerhans cell histiocytosis (PLCH); however, the pathogenesis remains largely unknown. Few studies have evaluated histopathologic changes in pulmonary arteries (PAs) of patients with PLCH; systematic quantification of arterial remodeling has yet to be undertaken. Objective.— To quantify the extent of arterial remodeling among patients with PLCH through morphometry and to correlate these results with pertinent clinical parameters. Design.— Patients with PLCH were identified from institutional files (1995–2015) along with ag

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10

Pollanen, M. S., J. H. N. Deck, L. Boutilier, and G. Davidson. "Lesions of the Tunica Media in Traumatic Rupture of Vertebral Arteries: Histologic and Biochemical Studies." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 19, no. 1 (1992): 53–56. http://dx.doi.org/10.1017/s0317167100042530.

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ABSTRACT:Discontinuous non-circumferential lesions of tunica media were observed in four cases of traumatic rupture of the vertebral artery. We hypothesize that these lesions were due to mechanical disruption of smooth muscle cells and the liberation of catabolic enzymes with subsequent degradation of the arterial media. To test this hypothesis, healthy vertebral arteries were incubated with crude extracts of bovine smooth muscle cytosol in attempt to reproduce the histological changes of the arterial media in traumatized vertebral arteries. We observed cytosol-induced degradation of tunica me

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Eufrásio-da-Silva, Tatiane, Eduardo Ruiz-Hernandez, Joanne O’Dwyer, Dolores Picazo-Frutos, Garry P. Duffy, and Bruce P. Murphy. "Enhancing medial layer recellularization of tissue-engineered blood vessels using radial microchannels." Regenerative Medicine 14, no. 11 (2019): 1013–28. http://dx.doi.org/10.2217/rme-2019-0011.

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Aim: Cell repopulation of tissue-engineered vascular grafts (TEVGs) from decellularized arterial scaffolds is limited by dense concentric tunica media layers which impede cells migrating radially between the layers. We aimed to develop and validate a new microneedle device to modify decellularized carotid arteries with radial microchannels to enhance medial layer repopulation. Material & methods: Modified decellularized porcine arteries were seeded with rat mesenchymal stem cells using either standard longitudinal injection, or a dual vacuum-perfusion bioreactor. Mechanical tests were used

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Mozafari, Hozhabr, Changchun Zhou, and Linxia Gu. "Mechanical contribution of vascular smooth muscle cells in the tunica media of artery." Nanotechnology Reviews 8, no. 1 (2019): 50–60. http://dx.doi.org/10.1515/ntrev-2019-0005.

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Abstract The stiffness of arterial wall in response to cardiovascular diseases has been associated with the changes in extracellular matrix (ECM) proteins, i.e., collagen and elastin. Vascular smooth muscle cells (VSMCs) helped to regulate the ECM reorganizations and thus contributed to arterial stiffness. This article reviewed experimental and computational studies for quantifying the roles of ECM proteins and VSMCs in mechanical properties of arteries, including nanostructure and mechanical properties of VSMCs and ECMs, cell-ECM interaction, and biomimetic gels/scaffolds induced contractile

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Inkeri, Jussi, Anniina Tynjälä, Carol Forsblom, et al. "Carotid intima-media thickness and arterial stiffness in relation to cerebral small vessel disease in neurologically asymptomatic individuals with type 1 diabetes." Acta Diabetologica 58, no. 7 (2021): 929–37. http://dx.doi.org/10.1007/s00592-021-01678-x.

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Abstract Aims To determine if arterial functional and structural changes are associated with underlying cerebral small vessel disease in neurologically asymptomatic individuals with type 1 diabetes. Methods We enrolled 186 individuals (47.8% men; median age 40.0, IQR 33.0—45.0 years) with type 1 diabetes (median diabetes duration of 21.6, IQR 18.2—30.3 years), and 30 age- and sex-matched healthy controls, as part of the Finnish Diabetic Nephropathy (FinnDiane) Study. All individuals underwent a biochemical work-up, brain magnetic resonance imaging (MRI), ultrasound of the common carotid arteri

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Gajnitdinova, V. V., A. B. Bakirov, E. Kh Akhmetzyanova, N. F. Berdikaeva, and V. B. Zakirova. "Arterial stiffness of peripheral vasculature in patients with chronic obstructive pulmonary disease and its association with arterial hypertension." Kazan medical journal 94, no. 6 (2013): 808–12. http://dx.doi.org/10.17816/kmj1795.

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Aim. To study the structural and functional state of vascular wall, arterial stiffness of large peripheral arteries (common carotid arteries, renal arteries) in patients with severe chronic obstructive pulmonary disease and its association with arterial hypertension. Methods. The study included 67 patients of working age, mainly males, having chronic obstructive pulmonary disease. Among them, 52 patients had severe chronic obstructive pulmonary disease (defined by GOLD III, 2011), 15 had concomitant arterial hypertension of I and II stage. Structural and functional status of common carotid art

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15

Janzen, Jan, Lanzer, Rothenberger-Janzen, and Vuong. "Die Übergangszone in der Tunica media der Nierenarterie mit einer maximalen Länge von 10 Millimetern." Vasa 29, no. 3 (2000): 168–72. http://dx.doi.org/10.1024/0301-1526.29.3.168.

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Background: The goal of our study was to demonstrate and to determine the length of the transitional zone in the tunica media in renal arteries. The majority of renal artery atherosclerotic stenotic lesions occurs in this segment. Patients and methods: Anatomical and histological studies were performed on 26 renal arteries from 13 adults at autopsy (mean age 61.6 years, range 33 to 87 years). Results: In the macroscopical examination the right renal arteries (RRA) were longer with a median 53.8 mm (range 38 to 65 mm) than the left renal arteries (LRA) with a median 47.6 mm (range 35 to 63 mm),

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16

Hughes, Jennifer M., and Stuart J. Bund. "Influence of experimental reduction of arterial media." Journal of Hypertension 22, no. 3 (2004): 519–27. http://dx.doi.org/10.1097/00004872-200403000-00014.

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17

Titov, Vladimir N., T. A. Rozhkova, and V. A. Amelyushkina. "Atheromatosis of arterial intima." Clinical Medicine (Russian Journal) 94, no. 8 (2016): 582–90. http://dx.doi.org/10.18821/0023-2149-2016-94-8-582-590.

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Phylogenetically late arterial intima of the elastic type contains no proteins for the transfer of ligandless oxidized low density lipoproteins (LDLP) for sedentary macrophages adsorbed on the matrix. Phylogenetically early cells realize the extracellular digestive reaction by releasing proteolytic enzymes (metalloproteinases) into intimal matrix that hydrolize matrix proteoglycans, adsorbed ligandless LDLP, detritus, and complete lysosomal hydrolysis of the most hydrophobic polyenic cholesterol esters (poly-ECS). Smooth muscle cells migrate from the middle muscular layer of the arterial wall,

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Zaletel, Lorna Zadravec, Matjaz Popit, and Marjan Zaletel. "Is carotid stiffness a possible surrogate for stroke in long-term survivors of childhood cancer after neck radiotherapy?" Radiology and Oncology 52, no. 2 (2018): 136–42. http://dx.doi.org/10.2478/raon-2018-0006.

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Abstract Background The risk for cerebrovascular late effects among childhood cancer survivors is considerable. According to recent studies it is not clear which marker could be reliable for the screening of cerebrovascular diseases among the long-term survivors of childhood cancer. The purpose of this study is to analyse arterial stiffness and intima-media thickness as possible early markers of later occurring stroke in long-term survivors of childhood cancer after neck radiotherapy. Patients and methods Twenty-three patients, treated for Hodgkin disease (HD) in childhood, were included. They

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Opdebeeck, Britt, Patrick C. D’Haese, and Anja Verhulst. "Molecular and Cellular Mechanisms that Induce Arterial Calcification by Indoxyl Sulfate and P-Cresyl Sulfate." Toxins 12, no. 1 (2020): 58. http://dx.doi.org/10.3390/toxins12010058.

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The protein-bound uremic toxins, indoxyl sulfate (IS) and p-cresyl sulfate (PCS), are considered to be harmful vascular toxins. Arterial media calcification, or the deposition of calcium phosphate crystals in the arteries, contributes significantly to cardiovascular complications, including left ventricular hypertrophy, hypertension, and impaired coronary perfusion in the elderly and patients with chronic kidney disease (CKD) and diabetes. Recently, we reported that both IS and PCS trigger moderate to severe calcification in the aorta and peripheral vessels of CKD rats. This review describes t

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Vera, Lisse, Sofie Muylle, Glenn Van Steenkiste, et al. "Histological and biomechanical properties of systemic arteries in young and old Warmblood horses." PLOS ONE 16, no. 7 (2021): e0253730. http://dx.doi.org/10.1371/journal.pone.0253730.

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Arterial rupture is a well-recognized cause of sudden death in horses, which mainly affects older horses. The arterial wall is known to stiffen with age, although the underlying age-related histological and biomechanical changes remain unclear. The purpose of this study was to investigate the effect of aging by histological analysis of the arterial wall and examination of the arterial wall biomechanical properties using an inflation-extension test. Entire circular samples of the proximal and distal aorta, cranial and caudal common carotid, external iliac, femoral and median artery were collect

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Provost, Patrick, and Yahye Merhi. "Endothelium-derived Nitric Oxide Decreases Polymorphonuclear Leukocyte Interaction with the Deeply Injured Arterial Wall under Intermediate and High Shear Conditions." Thrombosis and Haemostasis 78, no. 02 (1997): 939–46. http://dx.doi.org/10.1055/s-0038-1657656.

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SummaryPrevious studies have shown that nitric oxide (NO) inhibits specific agonist-induced polymorphonuclear leukocyte (neutrophil) and platelet aggregation in vitro. However, the inhibitory effects of NO on neutrophil interaction with the deeply injured arterial wall under conditions of flow is unknown. Therefore, we investigated the influence of NO derived from the endothelium on neutrophil and platelet interactions with the downstream arterial media under controlled flow conditions. Porcine aortic media, simulating deep arterial wall injury, was exposed to flowing porcine non-anticoagulate

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Vargas Moranth, Rusvelt F., Adalgisa E. Alcocer Olaciregui, Paola Sanjuan Gómez, et al. "Presión arterial media: ¿importa el uso de escaleras?" Biociencias 14, no. 1 (2019): 65–77. http://dx.doi.org/10.18041/2390-0512/biociencias.1.5332.

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Objetivo: Determinar si el uso diario de escaleras se relaciona con valores de presión arterial media, en un grupo de habitantes de un conjunto residencial con edificios de 4 pisos. Materiales y métodos: Estudio descriptivo, transversal. Se tomó la presión arterial a 125 sujetos sanos (50,4% mujeres) residentes en los 4 pisos de cada uno de los 4 edificios de un conjunto residencial. Se compararon los valores de presión arterial media según el piso de residencia mediante ANOVA de un factor y prueba de Bonferroni, y se estratificó el análisis según edad, sexo, obesidad, fumar, beber alcohol con

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Jiménez-Ballén, Valentina, and Pedro Vargas-Pinto. "Comparación de valores de presión arterial indirecta medida en cinco puntos anatómicos en caninos despiertos." Revista de Investigaciones Veterinarias del Perú 29, no. 4 (2018): 1156. http://dx.doi.org/10.15381/rivep.v29i4.15194.

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En el presente estudio se buscó determinar si existe una variación significativa en perros despiertos entre la presión arterial entre los cuatro miembros, incluyendo la cola. Las presiones arteriales diastólica, sistólica y media fueron tomadas, utilizando el equipo SunTech Medical®, mediante la técnica de oscilometría, en cada uno de los miembros y la cola de 17 caninos pacientes de una clínica de pequeños animales en Bogotá, Colombia. No se encontró diferencias significativas entre los valores de la presión arterial diastólica, sistólica y media entre cada uno de los miembros y la cola.

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Šrámek, A., J. H. C. Reiber, F. R. Rosendaal, and P. Bucciarelli. "Arterial Intima-Media Thickness and its Relationship with Cardiovascular Disease and Atherosclerosis: A Possible Contribution of Medium-Sized Arteries." Thrombosis and Haemostasis 88, no. 12 (2002): 961–66. http://dx.doi.org/10.1055/s-0037-1613341.

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SummaryThe intima-media thickness (IMT) of the arterial wall, measured by B-mode ultrasonography, has been related to cardiovascular disease and atherosclerosis in many studies, most of which have considered carotid and common femoral arteries. No study has related the IMT of medium-sized arteries (e.g. brachial) to the atherosclerotic process. The aim of this study was to evaluate whether more peripheral arteries (e.g. brachial, superficial femoral and popliteal) represent a good model in studies of arterial wall IMT, as well as carotid artery. Twenty-six male patients with demonstrated coron

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Pasa, Márcio Bastiani, Adamastor Humberto Pereira, and Cyro Castro Júnior. "Morphometric analysis of intimal thickening secondary to stent placement in pig carotid arteries." Acta Cirurgica Brasileira 23, no. 2 (2008): 165–72. http://dx.doi.org/10.1590/s0102-86502008000200009.

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OBJECTIVE: To evaluate intimal thickening secondary to stent placement or to insertion of the delivery system without stent placement in pig carotid arteries. METHODS: Stents were placed in the right common carotid arteries of 7 pigs, and 7 other control pigs underwent only insertion of the delivery system without stent placement. Uninjured contralateral common carotid arteries of the two groups were also used as controls. Samples of arterial tissue, obtained from the area adjacent to the distal segment of the stent four weeks after placement, underwent morphometric analysis. Morphometric data

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Bastrikov, O. Yu, E. A. Grigoricheva, V. V. Belov, and V. V. Milegov. "Association of arterial stiffness and cardiovascular system remodeling in patients with hypertension." Kazan medical journal 95, no. 4 (2014): 485–90. http://dx.doi.org/10.17816/kmj1827.

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Aim. To study the indices of arterial wall local stiffness and it association with structural and functional parameters of cardiovascular system remodeling in hypertensive patients. Methods. Cross-sectional population study included 213 industrial workers, among them 106 patients with stage I-II hypertension and 107 healthy individuals. The stage of arterial hypertension and additional cardiovascular risk category was performed in accordance with national guidelines. All patients underwent echocardiography, carotid arteries ultrasonography, including arterial stiffness evaluation. Results. In

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Kanda, Keiichi, and Takehisa Matsuda. "In Vitro Reconstruction of Hybrid Arterial Media with Molecular and Cellular Orientations." Cell Transplantation 3, no. 6 (1994): 537–45. http://dx.doi.org/10.1177/096368979400300611.

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A hybrid medial tissue composed of a type I collagen gel, into which smooth muscle cells (SMCs) derived from bovine aortic media were 3-dimensionally (3D) embedded, was constructed around an elastomeric silicone tube (outer diameter: 8 mm). Subsequently, hybrid tissues thus prepared were subjected to three modes of mechanical stimulation in the medium: one was subjected to flotation with no disturbance (isotonic control), the second was kept isometrically (static stress) and the third was subjected to continuous periodic stretch by inflation of the embedded silicone tube which simulated arteri

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Hoffmann, Julia, Leigh M. Marsh, Mario Pieper, et al. "Compartment-specific expression of collagens and their processing enzymes in intrapulmonary arteries of IPAH patients." American Journal of Physiology-Lung Cellular and Molecular Physiology 308, no. 10 (2015): L1002—L1013. http://dx.doi.org/10.1152/ajplung.00383.2014.

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Alterations in extracellular matrix (ECM) have been implicated in the pathophysiology of pulmonary hypertension. Here, we have undertaken a compartment-specific study to elucidate the expression profile of collagens and their processing enzymes in donor and idiopathic pulmonary arterial hypertension (IPAH) pulmonary arteries. Predominant intimal, but also medial and perivascular, remodeling and reduced lumen diameter were detected in IPAH pulmonary arteries. Two-photon microscopy demonstrated accumulation of collagen fibers. Quantification of collagen in pulmonary arteries revealed collagen ac

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Chang, Zhuo, Paolo Paoletti, Maria Lyck Hansen, et al. "AFM Characterization of the Internal Mammary Artery as a Novel Target for Arterial Stiffening." Scanning 2018 (November 5, 2018): 1–10. http://dx.doi.org/10.1155/2018/6340425.

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Using the atomic force microscopy- (AFM-) PeakForce quantitative nanomechanical mapping (QNM) technique, we have previously shown that the adventitia of the human internal mammary artery (IMA), tested under dehydrated conditions, is altered in patients with a high degree of arterial stiffening. In this study, we explored the nanoscale elastic modulus of the tunica media of the IMA in hydrated and dehydrated conditions from the patients with low and high arterial stiffening, as assessed in vivo by carotid-femoral pulse wave velocity (PWV). In both hydrated and dehydrated conditions, the medial

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Bobryshev, Yuri V., Reginald S. A. Lord, and Dinh Tran. "Chlamydia pneumoniaein foci of “early” calcification of the tunica media in arteriosclerotic arteries: an incidental presence?" American Journal of Physiology-Heart and Circulatory Physiology 290, no. 4 (2006): H1510—H1519. http://dx.doi.org/10.1152/ajpheart.01055.2005.

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Only a few previous works investigated the involvement of Chlamydia pneumoniae ( Chlamydophila pneumoniae) in arterial calcification. The present study investigated a possible association between C. pneumoniae and medial calcification. Carotid artery segments obtained by endarterectomy from 60 patients were examined by PCR and immunohistochemistry to identify the presence of C. pneumoniae. Arterial specimens showing double-positive ( n = 17), double-negative ( n = 22), and single-positive results ( n = 21) were further analyzed by a combination of histology, immunohistochemistry, and electron

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Brekke, Johan Fredrik, Natalia I. Gokina, and George Osol. "Vascular smooth muscle cell stress as a determinant of cerebral artery myogenic tone." American Journal of Physiology-Heart and Circulatory Physiology 283, no. 6 (2002): H2210—H2216. http://dx.doi.org/10.1152/ajpheart.00633.2002.

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Although the level of myogenic tone (MT) varies considerably from vessel to vessel, the regulatory mechanisms through which the actual diameter set point is determined are not known. We hypothesized that a unifying principle may be the equalization of active force at the contractile filament level, which would be reflected in a normalization of wall stress or, more specifically, media stress. Branched segments of rat cerebral arteries ranging from <50 μm to >200 μm in diameter were cannulated and held at 60 mmHg with the objectives of: 1) evaluating the relationship between arterial diam

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Marinelli, A., P. Luciano, A. Buono, et al. "Early Detection of Arterial Media Calcifications with Ultrasonography." Ultrasound in Medicine & Biology 37, no. 8 (2011): S156. http://dx.doi.org/10.1016/j.ultrasmedbio.2011.05.701.

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Tellides, George, and Jordan S. Pober. "Inflammatory and Immune Responses in the Arterial Media." Circulation Research 116, no. 2 (2015): 312–22. http://dx.doi.org/10.1161/circresaha.116.301312.

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Quayle, Shaun M., Peter Koonce, Darryl Depencier, and Darcy M. Bullock. "Arterial Performance Measures with Media Access Control Readers." Transportation Research Record: Journal of the Transportation Research Board 2192, no. 1 (2010): 185–93. http://dx.doi.org/10.3141/2192-18.

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Simons, Petra C. G., Ale Algra, Michiel L. Bots, Diederick E. Grobbee, and Yolanda van der Graaf. "Common Carotid Intima-Media Thickness and Arterial Stiffness." Circulation 100, no. 9 (1999): 951–57. http://dx.doi.org/10.1161/01.cir.100.9.951.

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GRATTON, R. "Conditioned media from hypoxic trophoblast alters arterial function." Journal of the Society for Gynecologic Investigation 3, no. 2 (1996): 61A. http://dx.doi.org/10.1016/1071-5576(96)82452-x.

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Oestreicher-Kedem, Yael, Liat Ben-Sira, Galia Grissaru, and Ari DeRowe. "Arterial brain infarction in complicated acute otitis media." International Journal of Pediatric Otorhinolaryngology 71, no. 4 (2007): 659–63. http://dx.doi.org/10.1016/j.ijporl.2006.12.008.

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Ferrara, A., and A. Pandolfi. "A numerical study of arterial media dissection processes." International Journal of Fracture 166, no. 1-2 (2010): 21–33. http://dx.doi.org/10.1007/s10704-010-9480-y.

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Fujii, Satoshi, and Kazuhiko Fujitsu. "Experimental vasospasm in cultured arterial smooth-muscle cells." Journal of Neurosurgery 69, no. 1 (1988): 92–97. http://dx.doi.org/10.3171/jns.1988.69.1.0092.

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✓ Smooth-muscle cells were cultured from rat aortic media, then oxyhemoglobin and other agents including serotonin, norepinephrine, and angiotensin II were added separately to the medium. Contractile and ultrastructural changes of the cells were examined with electron microscopy during the first 2 weeks of incubation. Oxyhemoglobin not only produced progressive contraction of the arterial smooth-muscle cells, but it also caused ultrastructural changes that resembled myonecrosis. In contrast, there was no evidence of progressive contraction or ultrastructural changes either in control cultures

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Chyatte, Douglas, Jeffrey Reilly, and David M. Tilson. "Morphometric Analysis of Reticular and Elastin Fibers in the Cerebral Arteries of Patients with Intracranial Aneurysms." Neurosurgery 26, no. 6 (1990): 939–43. http://dx.doi.org/10.1227/00006123-199006000-00003.

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Abstract Elastin and reticular fibers were identified using standard histological stains in middle cerebral arteries taken from patients who had died from aneurysmal subarachnoid hemorrhage and control patients who did not have cerebral aneurysms, Examination of cerebral arteries from normal individuals revealed a dense network of fine reticular fibers in the arterial media that were uniformly distributed. Computerized morphometric analysis indicated that reticular fibers in the arterial media of cerebral arteries were significantly decreased in patients with aneurysms. In addition, these fibe

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Petersen, Karin K., Arne Hørlyck, Kristine H. Østergaard, et al. "Protection against high intravascular pressure in giraffe legs." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 305, no. 9 (2013): R1021—R1030. http://dx.doi.org/10.1152/ajpregu.00025.2013.

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The high blood pressure in giraffe leg arteries renders giraffes vulnerable to edema. We investigated in 11 giraffes whether large and small arteries in the legs and the tight fascia protect leg capillaries. Ultrasound imaging of foreleg arteries in anesthetized giraffes and ex vivo examination revealed abrupt thickening of the arterial wall and a reduction of its internal diameter just below the elbow. At and distal to this narrowing, the artery constricted spontaneously and in response to norepinephrine and intravascular pressure recordings revealed a dynamic, viscous pressure drop along the

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Macdonald, R. Loch, M. Christopher Wallace, Walter J. Montanera, and Jennifer A. Glen. "Pathological effects of angioplasty on vasospastic carotid arteries in a rabbit model." Journal of Neurosurgery 83, no. 1 (1995): 111–17. http://dx.doi.org/10.3171/jns.1995.83.1.0111.

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✓ To define the pathological effects of angioplasty on vasospastic arteries, 36 rabbits underwent angiography and induction of vasospasm by placement of blood-filled (vasospasm groups) or empty (control group) silastic sheaths around the cervical carotid arteries. Two (Day 2) or 7 days (Day 7) later, angiography was repeated and one carotid artery in each animal was dilated by balloon angioplasty. The rabbits were sacrificed 1 day, 7 days, or 3 to 4 weeks after angioplasty. Significant vasospasm developed after placement of silastic sheaths with blood (mean reductions in diameter 39% ± 6% at D

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Timmins, Lucas H., Qiaofeng Wu, Alvin T. Yeh, James E. Moore, and Stephen E. Greenwald. "Structural inhomogeneity and fiber orientation in the inner arterial media." American Journal of Physiology-Heart and Circulatory Physiology 298, no. 5 (2010): H1537—H1545. http://dx.doi.org/10.1152/ajpheart.00891.2009.

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The microstructural orientation of vascular wall constituents is of interest to scientists and clinicians because alterations in their native states are associated with various cardiovascular diseases. In the arterial media, the orientation of these constituents is often described as circumferential. However, it has been noted that, just below the endothelial surface, the vascular wall constituents are oriented axially. To further study this reported change in orientation, and to resolve previous observations (which were made under conditions of no load), we used nonlinear optical microscopy t

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Su-angka, N., A. Khositseth, S. Vilaiyuk, K. Tangnararatchakit, and W. Prangwatanagul. "Carotid intima-media thickness and arterial stiffness in pediatric systemic lupus erythematosus." Lupus 26, no. 9 (2017): 989–95. http://dx.doi.org/10.1177/0961203317692434.

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Objectives The carotid intima-media thickness (CIMT) and carotid arterial stiffness index (CASI) act as the surrogate markers of atherosclerosis. We aim to assess CIMT and CASI in pediatric systemic lupus erythematosus (SLE). Methods Patients ≤ 20 years old fulfilling diagnostic criteria for SLE were enrolled. Patients with active smoking, coronary heart disease, cerebrovascular disease, arterial thrombosis, family history of hypercholesterolemia, chronic liver disease, or other chronic severe diseases were excluded. The patients were categorized into four groups: active SLE, age- and sex-matc

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Silva, Gonçalo Teixeira de Almeida, Bruce B. Guest, Diego E. Gomez, et al. "Development of a technique for determination of pulmonary artery pulse wave velocity in horses." Journal of Applied Physiology 122, no. 5 (2017): 1088–94. http://dx.doi.org/10.1152/japplphysiol.00962.2016.

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Calcification of the tunica media of the axial pulmonary arteries (PA) has been reported in a large proportion of racehorses. In humans, medial calcification is a significant cause of arterial stiffening and is implicated in the pathogenesis of cardiac, cerebral, and renal microvascular diseases. Pulse wave velocity (PWV) provides a measure of arterial stiffness. This study aimed to develop a technique to determine PA-PWV in horses and, secondarily, to investigate a potential association between PA-PWV and arterial fibro-calcification. A dual-pressure sensor catheter (PSC) was placed in the ma

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Jovelic, Stojan, Zoran Hajdukovic, Aleksandra Jovelic, and Slavica Radjen. "Cigarette smoking in military pilots and intima-media thickness of the carotid arteries." Vojnosanitetski pregled 62, no. 5 (2005): 365–70. http://dx.doi.org/10.2298/vsp0505365j.

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Background. It is well known that smoking is associated with an increase in arterial wall thickness. However, most studies of this problem have been undertaken in age and sex heterogeneous groups, as well as in patients with already present other conventional risk factors. The aim of this study was to assess the effect of cigarette smoking on arterial wall thickness of the common carotid artery in asymptomatic pilots. Methods. The imaging of intima?media thickness of the posterior wall of the distal 1 cm of both common carotid arteries was performed using a B mode ultrasound device, in 39 pilo

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Kankilic, Nazım, Aydın Aslan, Oguz Karahan, Sinan Demirtas, Ahmet Caliskan, and Celal Yavuz. "Investigation of the arterial intima-media thickness in Behcet’s disease patients without vascular complaints." Vascular 26, no. 4 (2017): 356–61. http://dx.doi.org/10.1177/1708538117742827.

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Objectives Behcet’s disease is a multisystemic, inflammatory disease. Various factors have been implicated in the disease, including genetics, infections, immunoglobulins, immune complexes, antibodies, and oxidative stress. However, the underlying etiopathogenesis remains unclear. Behcet’s disease can occur with or without vascular involvement. This prospective study investigated the relationship between the intima-media thickness of the major arteries, in addition to other factors affecting the disease process, in Behcet’s disease without vascular involvement. Methods Twenty-four patients (av

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Villamonte, Wilfredo, Carolina Saavedra, María Jerí, and Janet Fernández. "Glucosa y lactato en sangre de arteria umbilical en neonatos a término en altura." Revista Peruana de Ginecología y Obstetricia 60, no. 3 (2014): 221–26. http://dx.doi.org/10.31403/rpgo.v60i139.

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Objetivos: Determinar los valores de glucosa y lactato en sangre arterial de cordón umbilical en recién nacidos a término a 3 400 m de altitud. Diseño: Estudio prospectivo de corte transversal. Institución: Hospital Nacional Adolfo Guevara Velazco, EsSalud, Cusco, Perú. Participantes: Neonatos a término, a 3400 m sobre el nivel. Métodos: Se obtuvo sangre de la arteria umbilical de 240 neonatos cuyo parto ocurrió en el Hospital entre agosto 2010 y diciembre 2011. Se determinó la media y los percentiles 5 y 95 de glucosa y lactato de sangre arterial del cordón umbilical de los neonatos. Principa

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Toda, N., K. Ayajiki, M. Enokibori, and T. Okamura. "Monkey cerebral arterial relaxation caused by hypercapnic acidosis and hypertonic bicarbonate." American Journal of Physiology-Heart and Circulatory Physiology 265, no. 3 (1993): H929—H933. http://dx.doi.org/10.1152/ajpheart.1993.265.3.h929.

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In helical strips of Japanese monkey cerebral arteries contracted with vasoconstrictors, applications of high CO2 (15% compared with 5% CO2 in control media) and hypertonic NaHCO3 (50 mM) produced relaxations. Similar relaxations were also obtained in human cerebral arterial strips. Hypercapnia increased PCO2 and resulted in acidosis in the bathing media, and the addition of NaHCO3 restored the pH to normal with high PCO2 and increased the osmotic pressure. The relaxant responses were not influenced by endothelium denudation and treatment with indomethacin. The hypercapnia-induced relaxation w

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Pushina, O. S. "Impact of Social Media on Prevention of Arterial Hypertension." HYPERTENSION, no. 3.47 (August 4, 2016): 97. http://dx.doi.org/10.22141/2224-1485.3.47.2016.76375.

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