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Relevant bibliographies by topics / Arterial stenoses

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Academic literature on the topic 'Arterial stenoses'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Arterial stenoses"

1

Kleinekofort, W., M. Kraemer, C. Rode, and V. Wizemann. "Extracorporeal Pressure Monitoring and the Detection of Vascular Access Stenosis." International Journal of Artificial Organs 25, no. 1 (2002): 45–50. http://dx.doi.org/10.1177/039139880202500108.

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Prospective monitoring of static venous pressure is an established tool to detect outflow stenoses in a vascular access. However, with this method it is not possible to identify vascular stenoses which are localized between the arterial and venous dialysis needle. We describe a new approach based on both static arterial and venous extracorporeal pressures. Pressure data of 9 dialysis patients with normal vascular access function and 9 patients with stenotic access were analyzed. Extracorporeal pressure was found to depend on the position of the heart relative to the extracorporeal blood circui

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2

Planken, R. N., T. Leiner, R. J. Nijenhuis, et al. "Contrast-Enhanced Magnetic Resonance Angiography Findings Prior to Hemodialysis Vascular access Creation: A Prospective Analysis." Journal of Vascular Access 9, no. 4 (2008): 269–77. http://dx.doi.org/10.1177/112972980800900408.

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Purpose To determine prospectively the clinical value of contrast-enhanced magnetic resonance angiography (CE-MRA) for assessment of the arterial inflow and venous outflow prior to vascular access (VA) creation. Methods Seventy-three patients underwent duplex ultrasonography (DUS) and CE-MRA prior to VA creation for detection of stenoses and occlusions. Two observers read the CE-MRA images for determination of inter-observer agreement. A VA was considered functional if it could be used for successful two-needle hemodialysis therapy within 2 months after creation. Results CE-MRA detected 6 sten

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Heinen, Stefan GH, Wouter Huberts, Daniel AF van den Heuvel, Frans N. van de Vosse, Jean-Paul PM de Vries, and Tammo Delhaas. "A comparative study of geometry-based methods and intra-arterial pressure measurements to assess the hemodynamic significance of equivocal iliac artery stenoses." Vascular 27, no. 2 (2018): 119–27. http://dx.doi.org/10.1177/1708538118805659.

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Objectives To date, the ultimate decision to treat iliac artery stenoses in patients suffering from symptomatic peripheral arterial disease is based on the patient’s symptoms and on visual inspection of angiographical images. The primary aim of this study was to investigate the accuracy of geometry-based methods (i.e. visual inspection and quantitative vascular analysis (Viewforum version R7.2v1 Advanced vessel analysis, Philips Healthcare, Best, The Netherlands) of 3D rotational angiography) to identify the severity of equivocal iliac artery stenosis in peripheral arterial disease patients wi

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MUSTAPHA, NORZIEHA, SANTABRATA CHAKRAVARTY, PRASHANTA K. MANDAL, and NORSARAHAIDA AMIN. "UNSTEADY RESPONSE OF BLOOD FLOW THROUGH A COUPLE OF IRREGULAR ARTERIAL CONSTRICTIONS TO BODY ACCELERATION." Journal of Mechanics in Medicine and Biology 08, no. 03 (2008): 395–420. http://dx.doi.org/10.1142/s0219519408002723.

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A two-dimensional (2D) nonlinear mathematical model to study the response of the pulsatile flow of blood through a couple of irregular stenoses influenced by externally imposed periodic body acceleration is developed. The model is 2D and axisymmetric with an outline of the stenosis obtained from the three-dimensional (3D) casting of a mildly stenosed artery. The combined influence of an asymmetric shape and surface irregularities of the constrictions is explored in a computational study of blood flow through arterial stenoses with 48% areal occlusion. The arterial wall is treated as an elastic

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Li, Xiaoyun, Ling Wang, Chi Zhang, et al. "Why Is ABI Effective in Detecting Vascular Stenosis? Investigation Based on Multibranch Hemodynamic Model." Scientific World Journal 2013 (2013): 1–10. http://dx.doi.org/10.1155/2013/185691.

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The ankle-brachial index (ABI), defined as the ratio of systolic pressure in the ankle arteries and that in the brachial artery, was a useful noninvasive method to detect arterial stenoses. There had been a lot of researches about clinical regularities of ABI; however, mechanism studies were less addressed. For the purpose of a better understanding of the correlation between vascular stenoses and ABI, a computational model for simulating blood pressure and flow propagation in various arterial stenosis circumstances was developed with a detailed compartmental description of the heart and main a

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Hansen, Peter, Kristoffer Hansen, Mads Pedersen, et al. "Atherosclerotic Lesions in the Superficial Femoral Artery (SFA) Characterized with Velocity Ratios using Vector Velocity Ultrasound." Ultrasound International Open 04, no. 03 (2018): E79—E84. http://dx.doi.org/10.1055/a-0637-2437.

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Abstract Purpose Atherosclerotic arteries are challenging to evaluate quantitatively using spectral Doppler ultrasound because of the turbulent flow conditions that occur in relation to the atherosclerotic stenoses. Vector velocity ultrasound is angle independent and provides flow information, which could potentially improve the diagnosis of arterial stenoses. The purpose of the study is to distinguish significant stenoses in the superficial femoral artery (> 50% diameter reduction) from non-significant stenoses based on velocity ratios derived from the commercially available vector velocit

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Kleinekofort, W. "Improved Measurement of Vascular Access Pressure." Journal of Vascular Access 3, no. 2 (2002): 58–63. http://dx.doi.org/10.1177/112972980200300203.

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Vascular access stenosis and thrombosis is one of the key problems for hemodialysis patients. Prospective monitoring of static venous dialysis pressures can be applied to detect outflow stenoses in a vascular access. However, the location of stenoses within the access may influence the diagnostic value of venous pressure measurements. Whereas a decrease in access flow occurs with all types of stenosis, strictures within the arterial anastomosis or between arterial and venous dialysis needle cannot be detected with venous pressure measurements alone. A new approach is discussed, which bases on

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8

Santamore, W. P., and A. A. Bove. "A theoretical model of a compliant arterial stenosis." American Journal of Physiology-Heart and Circulatory Physiology 248, no. 2 (1985): H274—H285. http://dx.doi.org/10.1152/ajpheart.1985.248.2.h274.

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Recent clinical and experimental evidence indicates that coronary artery stenoses may rapidly change their size and shape in response to alterations in vasomotor tone and intraluminal pressure. This theoretical study models a partially compliant arterial stenosis to examine the hemodynamic impact of these alterations. In rigid vessels, a 98% reduction in luminal area would predictably produce subendocardial ischemia in the resting state. In contrast, stenoses, with part of the arterial wall normal by the underlying plaque, responded to vasoconstriction and to changes in intraluminal pressure.

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Su, C. M., D. Lee, R. Tran-Son-Tay, and W. Shyy. "Fluid Flow Structure in Arterial Bypass Anastomosis." Journal of Biomechanical Engineering 127, no. 4 (2005): 611–18. http://dx.doi.org/10.1115/1.1934056.

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The fluid flow through a stenosed artery and its bypass graft in an anastomosis can substantially influence the outcome of bypass surgery. To help improve our understanding of this and related issues, the steady Navier-Stokes flows are computed in an idealized arterial bypass system with partially occluded host artery. Both the residual flow issued from the stenosis—which is potentially important at an earlier stage after grafting—and the complex flow structure induced by the bypass graft are investigated. Seven geometric models, including symmetric and asymmetric stenoses in the host artery,

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Siegel, John M., Christos P. Markou, David N. Ku, and S. R. Hanson. "A Scaling Law for Wall Shear Rate Through an Arterial Stenosis." Journal of Biomechanical Engineering 116, no. 4 (1994): 446–51. http://dx.doi.org/10.1115/1.2895795.

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Atherosclerosis of the human arterial system produces major clinical symptoms when the plaque advances to create a high-grade stenosis. The hemodynamic shear rates produced in high-grade stenoses are important in the understanding of atheromatous plaque rupture and thrombosis. This study was designed to quantify the physiologic stress levels experienced by endothelial cells and platelets in the region of vascular stenoses. The steady hemodynamic flow field was solved for stenoses with percent area reductions of 50, 75, and 90 percent over a range of physiologic Reynolds numbers (100–400). The

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Dissertations / Theses on the topic "Arterial stenoses"

1

Tait, W. F. "The use of continuous wave ultrasound to diagnose stenoses in the carotid arteries." Thesis, University of Glasgow, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.381464.

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Biz, Sophie. "Flow in collapsible stenoses : an experimental study." Thesis, Georgia Institute of Technology, 1993. http://hdl.handle.net/1853/18244.

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Etebari, Ali. "Wall shear measurements in arterial flows." Diss., Virginia Tech, 2006. http://hdl.handle.net/10919/27326.

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Cardiovascular disease is responsible for the majority of morbidity and mortality in the United States. Physiologically healthy flow rarely displays turbulent behavior, thereby maintaining normal shear levels. The presence of vortical flow structures, however, alters the hemodynamical characteristics within the system, which has significant effect upon shear stress (SS) and wall shear stress (WSS) levels, as well as particle residence times. The relationship between these hemodynamic parameters and vascular injury response is of great relevance to understanding the cardiovascular disease proc

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Konala, Bhaskar Chandra. "Effect of Arterial Wall-Stenosis Compliance on Coronary Diagnostic Parameters." University of Cincinnati / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1291146768.

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Flannery, Conor James. "Thrombus Formation under High Shear in Arterial Stenotic Flow." Thesis, Georgia Institute of Technology, 2005. http://hdl.handle.net/1853/6943.

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Acute thrombotic and thromboembolic occlusion of atherosclerotic vessels are events that precipitate most heart attacks and strokes. In arterial stenotic flow, thrombus formation is shear dependent and may or may not lead to complete occlusion of the vessel. Platelets in whole blood adhere to collagen-coated surfaces and as they accumulate the resistance of the stenosis increases because of the decreasing passageway of the occluded stenosis. As a model of blood clotting in stenoses, porcine blood is heparinized and perfused over tubular glass test sections that are coated with collagen type

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Simons, Dianne Margaret. "The hydrodynamics of idiopathic hypertrophic subaortic stenosis." Thesis, Georgia Institute of Technology, 1987. http://hdl.handle.net/1853/10257.

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Siegel, John Mather Jr. "Wall shear stress through an arterial stenosis and its implications to thrombosis." Thesis, Georgia Institute of Technology, 1992. http://hdl.handle.net/1853/17674.

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Molla, Md Mamun. "LES of pulsatile flow in the models of arterial stenosis and aneurysm." Thesis, University of Glasgow, 2009. http://theses.gla.ac.uk/905/.

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The Large Eddy Simulation (LES) technique is used to simulate the different types of Newtonian and non-Newtonian pulsatile blood flow in a constricted as well as in a dilated channel to gain insight of the transition-to-turbulent blood flow due to the arterial stenosis and aneurysm. In the stenosed model, a cosine shape stenosis is placed at the upper wall of a 3D channel which reduces the cross-sectional area, whereas the aneurysm which is also placed at the upper wall dilates the channel cross-sectional area. In LES, a top-hat spatial grid-filter is applied to the Navier-Stokes equations of

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Hye, Md Abdul. "Simulation of transient blood flow in models of arterial stenosis and aneurysm." Thesis, University of Glasgow, 2012. http://theses.gla.ac.uk/3836/.

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The Large Eddy Simulation (LES) technique with the Smagorinsky-Lilly dynamic subgrid model and two-equation Standard k-ω Transitional turbulence model are applied to investigate non-spiral and spiral blood flow through three dimensional models of arterial stenosis and aneurysm. A spiral pattern of blood flow is thought to have many beneficial effects on hemodynamics. Previous computational studies on spiral blood flow involve only steady spiral flow in a straight stenosed pipe without considering an upstream curved section of the artery. But a spiral pattern in the blood flow may exist due to

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Lara-Montalvo, Ruben Angel. "Ultrasound determination of absolute backscatter from arterial wall structures." Link to electronic thesis, 2002. http://www.wpi.edu/Pubs/ETD/Available/etd-1203102-165958.

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Books on the topic "Arterial stenoses"

1

National Institute of Diabetes and Digestive and Kidney Diseases (U.S.), ed. Renal artery stenosis. U.S. Dept. of Health and Human Services, National Institutes of Health, National Institute of Diabetes and Digestive and Kidney Diseases, 2007.

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2

Radczewski, Irina. Chirurgische Behandlung von Stenosen an der Arteria carotis: Operationsindikation - Früh- und Spätergebnisse. [s.n.], 1989.

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Gould, K. Lance. Coronary artery stenosis and reversing atherosclerosis. Chapman & Hall, 1997.

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Minnesota. Health Technology Advisory Committee., ed. Intracoronary brachytherapy. Health Technology Advisory Committee, 2001.

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Stephen, Balter, Chan Rosanna C, Shope Thomas B, and American Association of Physicists in Medicine., eds. Intravascular brachytherapy fluoroscopically guided interventions: American Association of Physicists in Medicine, 2002 summer school proceedings, McGill University, Montreal, Quebec, July 18-21, 2002. Published for the American Association of Physicists in Medicine by Medical Physics Pub., 2002.

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6

1951-, Schwartz Robert S., ed. Coronary restenosis. Blackwell Scientific Publishers, 1992.

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7

E, Rabbani LeRoy, ed. Applications of antisense therapies to restenosis. Kluwer Academic Publishers, 1999.

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W, Serruys P., Strauss Bradley H. 1957-, and King Spencer B. 1937-, eds. Restenosis after intervention with new mechanical devices. Kluwer Academic Publishers, 1992.

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9

Daemen, Joost. "The caveats of drug-eluting stents": A critical appraisal of the safety concerns. s.n.], 2008.

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J, White Christopher, ed. Drug-eluting stents. Taylor & Francis, 2005.

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Book chapters on the topic "Arterial stenoses"

1

Cowling, M. G. "Management of Renal and Visceral Arterial Stenoses." In Medical Radiology. Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/174_2011_516.

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2

Ventre, Jeanne, Francesca Raimondi, Nathalie Boddaert, José Maria Fullana, and Pierre-Yves Lagrée. "Reduced-Order Models for Blood Pressure Drop Across Arterial Stenoses." In Lecture Notes in Computational Vision and Biomechanics. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-43195-2_1.

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3

Saffitz, Jeffrey E., Charles L. McIntosh, and William C. Roberts. "Massive Right Ventricular Outflow Tract Aneurysm After Ventriculotomy for Subvalvular Pulmonic Stenosis Associated With Peripheral Pulmonary Arterial Stenoses." In Case Reports in Cardiology. CRC Press, 2023. http://dx.doi.org/10.1201/9781003409342-31.

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Block, Peter C. "The Mechanism of Transluminal Angioplasty Pathology of the Arterial Stenoses that Are Most Amenable to PTCA." In Practice of Coronary Angioplasty. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-70815-2_2.

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Westerhof, Nicolaas, Nikolaos Stergiopulos, and Mark I. M. Noble. "Arterial Stenosis." In Snapshots of Hemodynamics. Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-6363-5_5.

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Westerhof, Nicolaas, Nikolaos Stergiopulos, Mark I. M. Noble, and Berend E. Westerhof. "Arterial Stenosis." In Snapshots of Hemodynamics. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91932-4_5.

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Ingraham, C., G. Johnson, S. Padia, and Sandeep Vaidya. "Arterial Inflow Stenosis." In Hemodialysis Access. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-40061-7_29.

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Mitsos, Aristotelis P. "Intracranial Arterial Stenosis." In Endovascular Neurosurgery Through Clinical Cases. Springer Milan, 2014. http://dx.doi.org/10.1007/978-88-470-5687-9_8.

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Order, Stanley E., and Sarah S. Donaldson. "Arterial Stenosis and Restenosis." In Radiation Therapy of Benign Diseases. Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-58719-1_17.

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Spencer, M. P. "Hemodynamics of arterial stenosis." In Developments in Cardiovascular Medicine. Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-4305-6_9.

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Conference papers on the topic "Arterial stenoses"

1

Rotman, Oren, Uri Zaretsky, Avraham Shitzer, and Shmuel Einav. "Effect of Arterial Distensibility and Stenoses on Pressure Drop in Pulsatile Flow." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80058.

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The most accepted standard tool of cardiologists for assessing stenoses severity in the coronary tree is coronary angiography. Information from coronary angiography is limited to geometrical data on the coronary lumen, and provides limited functional data on the severity of stenoses. In addition, histopathological studies have demonstrated that angiographic evidence of stenosis is usually detected when the cross-sectional area of a plaque approaches 40% to 50% of the total cross-sectional area of the vessel [1]. Hence, managing the intermediate coronary lesions (40% to 70% diameter stenosis) a

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Wootton, David M., Christos P. Markou, Stephen R. Hanson, and David N. Ku. "Mechanistic Model of Arterial Thrombosis in Collagen-Coated Stenoses." In ASME 1998 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 1998. http://dx.doi.org/10.1115/imece1998-0057.

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Abstract A mechanistic model of arterial thrombosis has been developed, based on platelet transport by convection and shear-enhanced diffusion, and platelet adhesion at a lumped kinetic rate. The model is compared to platelet deposition on collagen-coated stenoses in an ex vivo thrombosis experiment that uses non-anticoagulated blood. In the converging regions of the stenosis, the model reproduces the spatial platelet deposition pattern, and matches deposition rates reasonably well. The model suggests that platelet thrombosis on type I collagen in stenotic arterial flow is transport-dominated.

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Konala, Bhaskar Chandra, Ashish Das, Mohamed Effat, Arif Imran, and Rupak K. Banerjee. "Misinterpretation of the Functional Severity of Coronary Stenosis Due To Variability in Arterial Wall Compliance." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19564.

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Effect of arterial wall compliance on the invasive coronary diagnostic parameters for various severities of coronary stenoses was assessed. The Mooney-Rivlin model was used to define the non-linear properties of the arterial wall and the plaque regions. The non-Newtonian viscosity of blood was modeled using the Carreau model. A finite element method was employed to solve the pulsatile fluid (blood)-structure (arterial wall) interaction (FSI) equations. Variability in the diagnostic parameter values can occur near the cut-off value due to change in compliance of stenotic arteries between the ra

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Karri, Satyaprakash Babu, Etebari Ali, and Pavlos Vlachos. "Time Resolved DPIV Analysis of Pulsatile Flow in Symmetric Stenotic Arteries: A Comparison Between Arterial and Coronary Flow." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176533.

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The presence of stenoses in the vasculature is responsible for significant alterations in the blood flow patterns. The flow within the stenosis is characterized by regions of high shear rates and flow separation, and downstream of stenosis recirculation region is formed, with low wall shear stresses (WSS) and large residence time[1,2. Flow in a symmetric stenosis is characterized by a central jet region with axisymmetrically separated flow surrounding it. A shear layer is formed between the jet and the recirculation region, which may be responsible for platelet activation [3,4] Wall shear stre

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Goswami, Ishan, Srikara V. Peelukhana, Marwan Al-Rjoub, Lloyd H. Back, and Rupak K. Banerjee. "Influence of Variable Native Arterial Diameter on Fractional Flow Reserve: An In-Vitro Study." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80881.

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Fractional flow reserve (FFR), the ratio of the pressures distal (Pd) and proximal (Pa) to a stenosis, and coronary flow reserve (CFR), the ratio of flows at maximal vasodilation to the resting condition, are widely used for determining the functional severity of a coronary artery stenosis. However, the diameter of the native artery might influence the FFR values. Therefore, using an in-vitro experimental study, we tested the variation of FFR for two arterial diameters, 2.5 mm (N1) and 3 mm (N2). We hypothesize that FFR is not influenced by native arterial diameter. For both N1 and N2, vasodil

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Chen, Kuangxu, Bin Zhang, and Chunlei Liang. "A 3D Parallel High-Order Solver With Curved Local Mesh Refinement for Predicting Arterial Flow Through Varied Degrees of Stenoses." In ASME 2020 Pressure Vessels & Piping Conference. American Society of Mechanical Engineers, 2020. http://dx.doi.org/10.1115/pvp2020-21667.

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Abstract A 3D parallel high-order spectral difference (SD) solver with curved local mesh refinement is developed in this research to simulate flow through stenoses of varied degrees (50%, 60%, 65%, 70% and 75%) of radius constriction at inlet Reynolds number of 500. This solver employs high-order curved mesh in the vicinity of arterial wall and the local mesh refinement technique reduces the overall computational cost by distributing more elements in critical regions. In simulation of flow through stenosis of 50% radius constriction, velocity profiles predicted from the SD solver agree well wi

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Geven, Maartje C. F., Arjen Van Der Horst, Marcel C. M. Rutten, Wilbert Aarnoudse, Nico H. J. Pijls, and Frans N. van de Vosse. "In-Vitro Evaluation of Hot-Film Anemometry Using a Sensor-Tipped Coronary Guide-wire." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176124.

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During coronary catheterization, the epicardial coronary arteries are visually assessed for stenoses on the coronary angiogram. However, the functional significance of disease in the coronary arterial tree, the increased resistance to blood flow, may easily be over- or underestimated by using a 2D projection.

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Holzapfel, Gerhard A., and Thomas Ch Gasser. "A Constitutive Framework for the Inelastic Mechanical Behavior of Arteries." In ASME 2001 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2001. http://dx.doi.org/10.1115/imece2001/bed-23117.

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Abstract A reliable constitutive model of arterial walls is an essential prerequisite to (i) understand the functions of diseased and non-diseased arteries, to (ii) optimize the design of arterial prostheses, and to (iii) improve diagnostics and therapeutical procedures that are based on mechanical treatments, such as percutaneous transluminal angioplasty (PTA) [1] or bypass surgery. PTA is the most frequent therapeutical intervention worldwide [2] to reduce the severity of atherosclerotic stenoses. It is of great and steadily growing medical, economical and scientific interest [3].

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D’Souza, Gavin A., Srikara V. Peelukhana, and Rupak K. Banerjee. "Misinterpretation of Stenosis Severity in the Presence of Serial Coronary Stenoses." In ASME 2013 Conference on Frontiers in Medical Devices: Applications of Computer Modeling and Simulation. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/fmd2013-16180.

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Diagnosis of the functional severity of an epicardial coronary stenosis using parameters like Fractional Flow Reserve, FFR (ratio of distal to proximal pressure of a stenotic region), might be affected in the presence of an additional downstream stenosis. In order to assess this effect, we have performed an in-vitro experiment which is used to validate a computational study. Three combinations of serial stenoses were tested: 80%-64%, 80%-80% and 80%-90% area stenosis (AS). The physiological mean hyperemic flow (flow at maximal arterial dilatation) values were obtained using an in-vitro experim

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Valdez-Jasso, Daniela, Mansoor A. Haider, Stephen L. Campbell, et al. "Modeling Viscoelastic Wall Properties of Ovine Arteries." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-205640.

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Generation of a complete map of arterial wall mechanical properties can improve treatment of cardiovascular diseases via contributions to design of patient specific vascular substitutes used to alleviate atherosclerosis and stenoses, which are predominant in arterial pathways (i.e., abdominal aorta, carotids, or femoral arteries). Clinically useful estimation of arterial properties from patient data requires both efficient algorithms and models that are both complex enough to capture clinically important properties and simple enough to allow rapid computation. In this study, we used mechanical

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Reports on the topic "Arterial stenoses"

1

Zhang, Ruizhe, and Qingya Xie. A meta-analysis of cholesteryl ester transfer protein(CETP) gene rs708272(G>A) polymorphism in association with cornoary heart disease risk. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.6.0021.

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Review question / Objective: To seek the association of the CETP rs708272 polymorphism with CHD.To figure out if the carriers of allele rs708272-A reduce or increase the risk of CHD in comparison with carriers of allele rs708272-G under allele model, dominant model and recessive model. Condition being studied: The inclusion criteria of CHD:(1)the presence of stenosis≥50% in a minimum of one main segment of coronary arteries (the right coronary artery, left circumfex, or left anterior descending arteries) by coronary angiography.(2) symptoms representing angina pectoris, electrocardiographic ch

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