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Journal articles on the topic 'Arteriopathy'

Author: Grafiati

Published: 7 July 2024

Last updated: 31 July 2025

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1

Chen, Alan M., Kunal B. Karani, J. Michael Taylor, et al. "Cervicocerebral quantitative arterial tortuosity: a biomarker of arteriopathy in children with intracranial aneurysms." Journal of Neurosurgery: Pediatrics 24, no. 4 (2019): 389–96. http://dx.doi.org/10.3171/2019.5.peds1982.

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OBJECTIVEAlthough intracranial arterial aneurysms (IAAs) of childhood are usually idiopathic, it is possible that underlying arteriopathy escapes detection when using conventional diagnostic tools. Quantitative arterial tortuosity (QAT) has been studied as a biomarker of arteriopathy. The authors analyzed cervicocerebral QAT in children with idiopathic IAAs to assess the possibility of arteriopathy.METHODSCases were identified by text-string searches of imaging reports spanning the period January 1993 through June 2017. QAT of cervicocerebral arterial segments was measured from cross-sectional

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2

Rafay, Mubeen F., Kevin A. Shapiro, Ann-Marie Surmava, et al. "Spectrum of cerebral arteriopathies in children with arterial ischemic stroke." Neurology 94, no. 23 (2020): e2479-e2490. http://dx.doi.org/10.1212/wnl.0000000000009557.

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ObjectiveTo determine that children with arterial ischemic stroke (AIS) due to an identifiable arteriopathy are distinct from those without arteriopathy and that each arteriopathy subtype has unique and recognizable clinical features.MethodsWe report a large, observational, multicenter cohort of children with AIS, age 1 month to 18 years, enrolled in the International Pediatric Stroke Study from 2003 to 2014. Clinical and demographic differences were compared by use of the Fisher exact test, with linear step-up permutation min-p adjustment for multiple comparisons. Exploratory analyses were co

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3

Kothur, Kavitha, Christopher Troedson, Richard Webster, et al. "Elevation of cerebrospinal fluid cytokine/chemokines involved in innate, T cell, and granulocyte inflammation in pediatric focal cerebral arteriopathy." International Journal of Stroke 14, no. 2 (2018): 154–58. http://dx.doi.org/10.1177/1747493018799975.

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Aim To determine the role of inflammation in pediatric transient focal cerebral arteriopathy using cerebrospinal fluid cytokine/chemokines as biomarkers. Methods We measured 32 cytokine/chemokines in acute cerebrospinal fluid collected from children with stroke due to focal cerebral arteriopathy (n = 5) using multiplex immunoassay and compared with two patients with arterial ischemic stroke due to other causes (non-focal cerebral arteriopathy group, vertebral dissection, n = 1; cryptogenic, n = 1), pediatric encephalitis (n = 43), and non-inflammatory neurological disease controls (n = 20). Re

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4

Joyal, France, and Pascal Margaroli. "Calcific Arteriopathy." New England Journal of Medicine 352, no. 26 (2005): e24. http://dx.doi.org/10.1056/nejmicm040777.

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5

Wagenvoort, C. A. "Plexogenic arteriopathy." Thorax 49, Suppl (1994): S39—S45. http://dx.doi.org/10.1136/thx.49.suppl.s39.

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6

Mitchell, Richard N. "Allograft arteriopathy." Cardiovascular Pathology 13, no. 1 (2004): 33–40. http://dx.doi.org/10.1016/s1054-8807(03)00108-x.

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7

Gupta, R. K. "Transplant Arteriopathy." Transplantation Proceedings 39, no. 3 (2007): 763–65. http://dx.doi.org/10.1016/j.transproceed.2007.01.068.

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8

Smith, Edward R. "Moyamoya Arteriopathy." Current Treatment Options in Neurology 14, no. 6 (2012): 549–56. http://dx.doi.org/10.1007/s11940-012-0195-4.

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9

Cuoco, Joshua A., Christopher M. Busch, Brendan J. Klein, et al. "ACTA2 Cerebral Arteriopathy: Not Just a Puff of Smoke." Cerebrovascular Diseases 46, no. 3-4 (2018): 159–69. http://dx.doi.org/10.1159/000493863.

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Background: Missense mutations in the gene that codes for smooth muscle actin, ACTA2, cause diffuse smooth muscle dysfunction and a distinct cerebral arteriopathy collectively known as multisystemic smooth muscle dysfunction syndrome (MSMDS). Until recently, ACTA2 cerebral arteriopathy was considered to be a variant of moyamoya disease. However, recent basic science and clinical data have demonstrated that the cerebral arteriopathy caused by mutant ACTA2 exhibits genetic loci, histopathology, neurological sequelae, and radiographic findings unique from moyamoya disease. We conducted a literatu

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10

Park, S. J., R. Pifarre, H. Sullivan, A. Montoya, and S. Y. Kim. "Natural History of Allograft Coronary Arteriopathy: A Retrospective Study of 54 Patients over a 81/2-Year Period." Cardiovascular Surgery 4, no. 1 (1996): 37–41. http://dx.doi.org/10.1177/096721099600400107.

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A total of 288 patients underwent heart transplantation at Loyola University Medical Center over a 81/2-year period starting in March, 1984. Of these patients. 54 were identified as having allograft coronary arteriopathy. Diagnosis was made on the basis of abnormal findings on coronary angiography in 44 patients; either an autopsy or explanted heart revealed the diagnosis in the remainder. A total of 279 abnormal lesions were identified by coronary angiography, and over 70% of these stenoses were discrete and located in large epicardial vessels. The left ventricular ed-diastolic pressure demon

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11

Olivero, Juan Jose, Juan Jorge Olivero, Peter Nguyen, and Anna Kagan. "Nephrogenic Calcific Arteriopathy." Methodist DeBakey Cardiovascular Journal 7, no. 4 (2011): 33. http://dx.doi.org/10.14797/mdcvj.304.

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12

Veinot, John P., and Virginia M. Walley. "Cardiac Transplant Arteriopathy." American Journal of Surgical Pathology 19, no. 6 (1995): 727. http://dx.doi.org/10.1097/00000478-199506000-00014.

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13

Edwards, W. D. "Plexogenic pulmonary arteriopathy." Histopathology 17, no. 2 (1990): 188a—189. http://dx.doi.org/10.1111/j.1365-2559.1990.tb00704.x.

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14

Heath, D., and A. W. Caslin. "Plexogenic pulmonary arteriopathy." Histopathology 17, no. 2 (1990): 189. http://dx.doi.org/10.1111/j.1365-2559.1990.tb00705.x.

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15

Wu, Mei-Han, Shang-Feng Yang, and Yao-Ping Lin. "Calcific Uremic Arteriopathy." American Journal of the Medical Sciences 347, no. 2 (2014): 146. http://dx.doi.org/10.1097/maj.0b013e31826b2d2f.

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16

Olivero, Juan Jose, Juan Jorge Olivero, Peter Nguyen, and Anna Kagan. "Nephrogenic Calcific Arteriopathy." Methodist DeBakey Cardiovascular Journal 7, no. 4 (2011): 33–36. http://dx.doi.org/10.14797/mdcj-7-4-33.

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17

Finsterer, J., and S. Zarrouk Mahjoub. "Primary mitochondrial arteriopathy." Nutrition, Metabolism and Cardiovascular Diseases 22, no. 5 (2012): 393–99. http://dx.doi.org/10.1016/j.numecd.2012.01.002.

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18

Steinlin, Maja, Sandra Bigi, Belinda Stojanovski, et al. "Focal Cerebral Arteriopathy." Stroke 48, no. 9 (2017): 2375–82. http://dx.doi.org/10.1161/strokeaha.117.016818.

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19

Dlamini, Nomazulu, Dawn E. Saunders, Michael Bynevelt, et al. "Nocturnal oxyhemoglobin desaturation and arteriopathy in a pediatric sickle cell disease cohort." Neurology 89, no. 24 (2017): 2406–12. http://dx.doi.org/10.1212/wnl.0000000000004728.

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Objective:The purpose of this study of sickle cell disease (SCD) was to determine whether arteriopathy, measurable as intracranial vessel signal loss on magnetic resonance angiography (MRA), was associated with low nocturnal hemoglobin oxygen saturation (SpO2) or hemolytic rate, measurable as reticulocytosis or unconjugated hyperbilirubinemia.Methods:Ninety-five East London children with SCD without prior stroke had overnight pulse oximetry, of whom 47 (26 boys, 39 hemoglobin SS; mean age 9.1 ± 3.1 years) also had MRA, transcranial Doppler (TCD), steady-state hemoglobin, and reticulocytes with

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20

Rutledge, W. Caleb, Omar Choudhri, Brian P. Walcott, et al. "Indirect and direct revascularization of ACTA2 cerebral arteriopathy: feasibility of the superficial temporal artery to anterior cerebral artery bypass with posterior auricular artery interposition graft: case report." Journal of Neurosurgery: Pediatrics 18, no. 3 (2016): 339–43. http://dx.doi.org/10.3171/2016.3.peds15694.

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Mutations in the smooth muscle–specific isoform of alpha actin (ACTA2) cause smooth muscle dysfunction in arteries. This rare loss-of-function mutation may cause a diffuse occlusive cerebral arteriopathy, resulting in stroke. While ACTA2 arteriopathy is often described as moyamoya-like, it has a distinct phenotype characterized by dilation of the proximal internal carotid artery (ICA) and occlusion of the terminal ICA and proximal middle cerebral artery. Intracranial arteries have an abnormally straight course, often with small aneurysms. There is limited experience with revascularization proc

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21

Nordstrom, Matthew, Erin Felton, Katherine Sear, et al. "Large Vessel Arteriopathy After Cranial Radiation Therapy in Pediatric Brain Tumor Survivors." Journal of Child Neurology 33, no. 5 (2018): 359–66. http://dx.doi.org/10.1177/0883073818756729.

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Among childhood cancer survivors, increased stroke risk after cranial radiation therapy may be caused by radiation-induced arteriopathy, but limited data exist to support this hypothesis. Herein, we assess the timing and presence of cerebral arteriopathy identified by magnetic resonance angiography (MRA) after cranial radiation therapy in childhood brain tumor survivors. In a cohort of 115 pediatric brain tumor survivors, we performed chart abstraction and prospective annual follow-up to assess the presence of large vessel cerebral arteriopathy by MRA. We identified 10 patients with cerebral a

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22

Oka, Masahiko, Ivan F. McMurtry, and Kaori Oshima. "How does pulmonary endarterectomy cure CTEPH: a clue to cure PAH?" American Journal of Physiology-Lung Cellular and Molecular Physiology 311, no. 4 (2016): L766—L769. http://dx.doi.org/10.1152/ajplung.00288.2016.

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Chronic thromboembolic pulmonary hypertension (CTEPH) is a hot topic in the field of pulmonary hypertension because many CTEPH patients are now curable by surgical pulmonary endarterectomy. However, there are still uncertainties regarding the pathogenesis of CTEPH, specifically how and where the small vessel arteriopathy that is indistinguishable from that in pulmonary arterial hypertension (plexogenic arteriopathy) develops and how pulmonary endarterectomy improves hemodynamics and possibly cures CTEPH. Based on our recent experimental finding that hemodynamic stress is fundamental for the de

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23

Wang, Hanlin L. "Carcinomatous Arteriopathy as an Unusual Feature of Pulmonary Spread of Cholangiocarcinoma Arising in Caroli Disease." Archives of Pathology & Laboratory Medicine 126, no. 6 (2002): 717–20. http://dx.doi.org/10.5858/2002-126-0717-caaauf.

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Abstract Carcinomatous arteriopathy is a rare and distinct form of pulmonary spread of solid malignant tumors characterized by fibrointimal proliferation of small pulmonary arteries and arterioles initiated by tumor emboli. Although it has been reported in many types of adenocarcinomas, no case of carcinomatous arteriopathy induced by cholangiocarcinoma has been described in the literature. We report a unique case of cholangiocarcinoma that arose in Caroli disease. Postmortem examination revealed extensive pulmonary vascular spread in the form of carcinomatous arteriopathy.

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24

Savitr Sastri, B. V., Bhagvatula Indira Devi, Jagathlal Gangadharan, and Dhaval Shukla. "Cerebral Aneurysmal Childhood Arteriopathy." Pediatric Neurosurgery 47, no. 3 (2011): 233–34. http://dx.doi.org/10.1159/000331571.

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25

Garcia, Julio H., and Khang-Loon Ho. "Pathology of Hypertensive Arteriopathy." Neurosurgery Clinics of North America 3, no. 3 (1992): 497–507. http://dx.doi.org/10.1016/s1042-3680(18)30641-7.

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26

Lou, Min, and Louis R. Caplan. "Vertebrobasilar dilatative arteriopathy (dolichoectasia)." Annals of the New York Academy of Sciences 1184, no. 1 (2009): 121–33. http://dx.doi.org/10.1111/j.1749-6632.2009.05114.x.

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27

Bucci, F., F. Robert, L. Fiengo, and P. Plagnol. "Radiotherapy-related axillary arteriopathy." Interactive CardioVascular and Thoracic Surgery 15, no. 1 (2012): 176–77. http://dx.doi.org/10.1093/icvts/ivr147.

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28

Jack, Andrew S., Michael M. Chow, Loretta Fiorillo, Thea Chibuk, Jerome Y. Yager, and Vivek Mehta. "Bilateral pial synangiosis in a child with PHACE syndrome." Journal of Neurosurgery: Pediatrics 17, no. 1 (2016): 70–75. http://dx.doi.org/10.3171/2015.5.peds1578.

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The acronym PHACE has been used to denote a constellation of abnormalities: posterior fossa anomalies, facial hemangiomas, arterial anomalies, cardiac anomalies, and eye abnormalities. Approximately 30% of patients with large facial hemangiomas have PHACE syndrome, with the vast majority having intracranial arteriopathy. Few reports characterize neurological deterioration from this intracranial arteriopathy, and even fewer report successful treatment thereof. The authors report on a case of a child with PHACE syndrome who presented with an ischemic stroke from a progressive intracranial arteri

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29

Chabrier, Stéphane, Guillaume Sébire, and Joel Fluss. "Transient Cerebral Arteriopathy, Postvaricella Arteriopathy, and Focal Cerebral Arteriopathy or the Unique Susceptibility of the M1 Segment in Children With Stroke." Stroke 47, no. 10 (2016): 2439–41. http://dx.doi.org/10.1161/strokeaha.116.014606.

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30

Perez-Atayde, A. R., D. I. Kearney, J. T. Bricker, et al. "Cardiac, Aortic, and Pulmonary Arteriopathy in HIV-Infected Children: The Prospective P2C2 HIV Multicenter Study." Pediatric and Developmental Pathology 7, no. 1 (2004): 61–70. http://dx.doi.org/10.1007/s10024-003-1001-9.

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Arteriopathy in human immunodeficiency virus (HIV)-infected patients is being increasingly recognized, especially in children. However, few studies have histologically evaluated the coronary arteries in HIV-infected children, and none have systematically assessed the aorta and pulmonary arteries. The coronary arteries, thoracic aorta, and the main and branch pulmonary arteries from the postmortem hearts of 14 HIV-infected children were systematically reviewed for vasculopathic lesions and compared with 14 age-matched controls. Findings from the HIV-infected children were compared with clinical

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31

Wang, Zhen, Jingxuan Guo, Xian Wang, Yiming Zhao, and Lingfei Hou. "A Study of the Relationships between Serum Calcitonin Gene-Related Peptide, Sex Hormone, Homocysteine and Coronary Heart Disease in Postmenopausal Women." International Journal of Biomedical Science 2, no. 1 (2006): 42–48. http://dx.doi.org/10.59566/ijbs.2006.2042.

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To investigate the relationships among serum calcitonin gene-related peptide (CGRP), homocysteine (Hcy), female hormone, and coronary arteriopathy in postmenopausal women. Method. In a cross-sectional study, serum CGRP, estrodiol (E2), progesterone（P）and Hcy levels of 144 postmenopausal women with coronary heart disease (CHD) and non-coronary heart disease (NCHD) were measured. Results. The mean serum CGRP level was significantly lower in CHD patients than in NCHD subjects. The mean serum E2 and P level were significantly lower in CHD patients than in NCHD subjects. The mean serum Hcy level wa

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32

Oesch, Gabriela, Francisco A. Perez, Mark S. Wainwright, Dennis W. W. Shaw, and Catherine Amlie-Lefond. "Focal Cerebral Arteriopathy of Childhood." Stroke 52, no. 7 (2021): 2258–65. http://dx.doi.org/10.1161/strokeaha.120.031880.

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Background and Purpose: Focal cerebral arteriopathy (FCA) of childhood with unilateral stenosis of the anterior circulation is reported to account for up to one-quarter of childhood arterial ischemic stroke, with stroke recurrence in 25% of cases. Limited knowledge regarding pathophysiology and outcome results in inconsistent treatment of FCA. Methods: Children with arterial ischemic stroke due to FCA between January 1, 2009, and January 1, 2019, were retrospectively identified at our institution which serves the US Pacific Northwest region. Electronic health record data, including neuroimagin

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33

Kløw, N. E., K. Levorstad, S. Simonsen, and O. Geiran. "Coronary Arteriopathy after Heart Transplantation." Acta Radiologica 39, no. 6 (1998): 656–62. http://dx.doi.org/10.3109/02841859809175492.

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34

Millichap, J. Gordon. "Intracranial Arteriopathy and Ischemic Stroke." Pediatric Neurology Briefs 23, no. 3 (2009): 18. http://dx.doi.org/10.15844/pedneurbriefs-23-3-3.

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35

Tanabe, Shigeru, Young-Sin Han, Tatsuya Nakatani, et al. "OBLITERATIVE ARTERIOPATHY IN RENAL TRANSPLANTATION." Japanese Journal of Urology 84, no. 10 (1993): 1828–34. http://dx.doi.org/10.5980/jpnjurol1989.84.1828.

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36

&NA;. "Pathologic markers of allograft arteriopathy." Current Opinion in Cardiology 16, no. 3 (2001): 218. http://dx.doi.org/10.1097/00001573-200105000-00010.

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37

Sébire, Guillaume. "Transient cerebral arteriopathy in childhood." Lancet 368, no. 9529 (2006): 8–10. http://dx.doi.org/10.1016/s0140-6736(06)68944-7.

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38

Evans, D. J., S. J. Cashman, and M. Walport. "PROGRESSIVE SYSTEMIC SCLEROSIS: AUTOIMMUNE ARTERIOPATHY." Lancet 329, no. 8531 (1987): 480–82. http://dx.doi.org/10.1016/s0140-6736(87)92091-5.

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39

Voelkel, Norbert F., and Harm Jan Bogaard. "Adding complexity to plexogenic arteriopathy." European Respiratory Journal 48, no. 6 (2016): 1553–55. http://dx.doi.org/10.1183/13993003.01867-2016.

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40

Fullerton, Heather J., Nicholas Stence, Nancy K. Hills, et al. "Focal Cerebral Arteriopathy of Childhood." Stroke 49, no. 11 (2018): 2590–96. http://dx.doi.org/10.1161/strokeaha.118.021556.

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41

Liu, Fang, Wei Wei, Gaoyi Yang, et al. "Therapeutic Effects and Finite Element Analysis of a Combined Treatment Using Laser Needle-Knife with Supine Repositioning Massage on Patients with Cervical Spondylotic Vertebral Arteriopathy." International Journal of Pattern Recognition and Artificial Intelligence 31, no. 11 (2017): 1757008. http://dx.doi.org/10.1142/s0218001417570087.

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Objective: To investigate the effectiveness of combined laser needle-knife and massage on cervical spondylotic vertebral arteriopathy patients. Summary of background data: With the recent rise of electronic businesses, the incidence of cervical spondylosis has also risen rapidly. Methods: Cervical spondylotic vertebral arteriopathy patients were treated using laser needle-knife with massage, and compared to patients who only received the massage. A 3D anatomical and hemodynamic model was developed. Results: The symptomatic and functional overall scores were reduced by 71.43% after the combined

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42

B Kale, Gautam, Prajnya Ranganath, and Jagarlapudi M K Murthy. "Cerebral autosomal recessive arteriopathy with subcortical infarcts and leukoencephalopathy (CARASIL): A rare cause of ischemic stroke in young." Neurology Asia 29, no. 3 (2024): 839–42. http://dx.doi.org/10.54029/2024muu.

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Cerebral autosomal recessive arteriopathy with subcortical infarcts and leukoencephalopathy (CARASIL) is the second known genetic form of cerebral small vessel disease after cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL). It is a very rare single gene disorder affecting cerebral small blood vessels. Diagnosis of CARASIL should strongly be suspected in a young non-hypertensive patient with lacunar stroke in the basal ganglia and brainstem and alopecia limited to scalp.

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43

Low, Audrey, Elijah Mak, Maura Malpetti, et al. "In vivo neuroinflammation and cerebral small vessel disease in mild cognitive impairment and Alzheimer’s disease." Journal of Neurology, Neurosurgery & Psychiatry 92, no. 1 (2020): 45–52. http://dx.doi.org/10.1136/jnnp-2020-323894.

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IntroductionAssociations between cerebral small vessel disease (SVD) and inflammation have been largely examined using peripheral blood markers of inflammation, with few studies measuring inflammation within the brain. We investigated the cross-sectional relationship between SVD and in vivo neuroinflammation using [11C]PK11195 positron emission tomography (PET) imaging.MethodsForty-two participants were recruited (according to NIA-AA guidelines, 14 healthy controls, 14 mild Alzheimer’s disease, 14 amyloid-positive mild cognitive impairment). Neuroinflammation was assessed using [11C]PK11195 PE

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44

Cospite, M., F. Ferrara, G. Milio, V. Scrivano, and F. Meli. "Ticlopidine in the Treatment of Multiple Atherosclerotic Arteriopathy: a Strain Gauge Plethysmography and Döppler Spectrum Analysis Evaluation." Journal of International Medical Research 15, no. 5 (1987): 303–11. http://dx.doi.org/10.1177/030006058701500506.

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The effect of ticlopidine was compared with flunarizine in patients with iliac–femoral and / or femoral–popliteal arteriosclerotic arteriopathy accompanied by lesions of the cervical arteries of no haemodynamic significance. In the lower limbs, plethysmography (strain gauge measurements) and Döppler ultrasonography integrated by spectral analysis of the cervical arteries showed qualitative and quantitative improvements of the regional haematic flow. Side-effects were negligible which suggests that ticlopidine is useful in the treatment of multiple arteriosclerotic arteriopathy.

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45

Furukawa, Yutaka, Peter Libby, Jennifer L. Stinn, Gerold Becker, and Richard N. Mitchell. "Cold Ischemia Induces Isograft Arteriopathy, but Does Not Augment Allograft Arteriopathy in Non-Immunosuppressed Hosts." American Journal of Pathology 160, no. 3 (2002): 1077–87. http://dx.doi.org/10.1016/s0002-9440(10)64928-0.

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46

Bulder, M., K. Braun, S. Chabrier, F. Kirkham, M. Tardieu, and G. Sébire. "SO01 Transient Cerebral Arteriopathy; follow up vascular imaging in 79 children with unilateral intracranial arteriopathy." European Journal of Paediatric Neurology 11 (September 2007): 22–23. http://dx.doi.org/10.1016/s1090-3798(08)70325-8.

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47

Alhadid, K., M. Kirby-Allen, G. DeVeber, W. Logan, and N. Dlamini. "P.054 Bone marrow transplant restores Cerebrovascular Reactivity (CVR) in Sickle Cell Disease (SCD): a case presentation." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 44, S2 (2017): S27. http://dx.doi.org/10.1017/cjn.2017.139.

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Background: A diagnosis of SCD in childhood confers a 200-fold increase in the risk of arterial ischemic stroke. Blood flow velocity measures provide better identification of ischemic risk compared to angiography. This indicates that steno-occlusive arteriopathy is not the singular causative factor. Cerebrovascular reactivity allows for augmentation of cerebral blood flow when needed. Kosinski et al in 2016 demonstrated a direct correlation between CVR and hematocrit levels in SCD. We report a case where CVR persistently normalized in an SCD patient following bone marrow transplant therapy (BM

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48

Hong, Ying, Annette Keylock, Barbara Jensen, et al. "Cerebral arteriopathy associated with heterozygous variants in the casitas B-lineage lymphoma gene." Neurology Genetics 6, no. 4 (2020): e448. http://dx.doi.org/10.1212/nxg.0000000000000448.

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ObjectiveTo report a series of patients with cerebral arteriopathy associated with heterozygous variants in the casitas B-lineage lymphoma (CBL) gene and examine the functional role of the identified mutant Cbl protein. We hypothesized that mutated Cbl fails to act as a negative regulator of the RAS-mitogen-activated protein kinases (MAPK) signaling pathway, resulting in enhanced vascular fibroblast proliferation and migration and enhanced angiogenesis and collateral vessel formation.MethodsWe performed whole-exome sequencing in 11 separate families referred to Great Ormond Street Hospital, Lo

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49

McKenna, Mary Clare, Noel Fanning, and Simon Cronin. "Focal Cerebral Arteriopathy in Young Adult Patients With Stroke." Stroke 51, no. 5 (2020): 1596–99. http://dx.doi.org/10.1161/strokeaha.119.028343.

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Background and Purpose— Focal cerebral arteriopathy is monophasic inflammatory stenosis of the distal internal carotid artery or the proximal segment of the middle cerebral artery. It is one of the most common causes of acute arterial ischemic stroke in young children but is a less familiar entity for adult neurologists. Methods— We retrospectively reviewed stroke service radiology records at a tertiary referral center from January 2013 to December 2014. Focal cerebral arteriopathy was defined as nonprogressive unifocal and unilateral stenosis/irregularity of the distal internal carotid artery

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50

Velayudhan, Akhil M., Jyoti R. Chandran, Sukumarapillai Jayasree, Rajeshwary Pillay, and Vijay Kumar Bindu. "Pregnancy and Delivery in a Patient with CADASIL: A Case Report." Journal of South Asian Federation of Obstetrics and Gynaecology 12, no. 5 (2020): 326–27. http://dx.doi.org/10.5005/jp-journals-10006-1818.

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ABSTRACT Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) is an autosomal dominant arteriopathy, caused by mutations in a gene called Notch3 on long arm of chromosome 19. It is very rare in pregnancy. This case report is presented to create awareness regarding investigating patients presenting with such illness for a better perinatal outcome. How to cite this article: Velayudhan AM, Chandran JR, Jayasree S, et al. Pregnancy and Delivery in a Patient with CADASIL: A Case Report. J South Asian Feder Obst Gynae 2020;12(5):326–327.

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