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Books on the topic 'As markers of inflammation'

Author: Grafiati

Published: 4 June 2025

Last updated: 1 August 2025

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1

A, Buckwalter Joseph, Lotz Martin, and Stoltz J. F, eds. Osteoarthritis, inflammation, and degradation: A continuum. IOS Press, 2007.

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2

S, Axford John, and Jenner International Glycoimmunology Meeting, eds. Glycoimmunology 2. Plenum Press, 1998.

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3

Clausen, Björn E., and Jon D. Laman, eds. Inflammation. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6786-5.

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4

Górski, Andrzej, Hubert Krotkiewski, and Michał Zimecki, eds. Inflammation. Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-015-9702-9.

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5

Wang, Yong-Xiao, ed. Inflammation. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-55254-0.

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6

Pierangelo, Geppetti, and Holzer, Peter, Mag. rer. nat. Dr. phil., eds. Neurogenic inflammation. CRC Press, 1996.

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7

Winyard, Paul G., and Derek A. Willoughby. Inflammation Protocols. Humana Press, 2003. http://dx.doi.org/10.1385/1592593747.

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8

Miyasaka, Masayuki, and Kiyoshi Takatsu, eds. Chronic Inflammation. Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-56068-5.

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9

Zierhut, Manfred, Carlos Pavesio, Shigeaki Ohno, Fernando Orefice, and Narsing A. Rao, eds. Intraocular Inflammation. Springer Berlin Heidelberg, 2016. http://dx.doi.org/10.1007/978-3-540-75387-2.

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10

Man, Francis, and Simon J. Cleary, eds. Imaging Inflammation. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-23661-7.

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11

Wu, Alan H. B. Cardiac Markers. Humana Press, 2003. http://dx.doi.org/10.1385/1592593852.

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12

Jucker, Andreas H., and Yael Ziv, eds. Discourse Markers. John Benjamins Publishing Company, 1998. http://dx.doi.org/10.1075/pbns.57.

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13

Wu, Alan H. B., ed. Cardiac Markers. Humana Press, 1998. http://dx.doi.org/10.1007/978-1-4612-1806-7.

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14

Wu, Alan H. B., ed. Cardiac Markers. Humana Press, 2003. http://dx.doi.org/10.1007/978-1-59259-385-9.

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15

Ranger, Graham. Discourse Markers. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-70905-5.

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16

name, No. Cardiac markers. 2nd ed. Humana Press, Inc., 2003.

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17

Buckwalter, J. Osteoarthritis, Inflammation and Degradation: A Continuum. IOS Press, Incorporated, 2007.

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18

Axford, John S. Glycoimmunology 2. Springer, 2012.

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19

Elastase: A new marker for inflammatory diseases. GIT Verlag, 1985.

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20

Bienvenu, J., and J. A. Grimaud. Marker Proteins in Inflammation: Proceedings of the Third Symposium. Lyon, France, June 26-28, 1985. Mouton De Gruyter, 1986.

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21

Axford, John S. Glycoimmunology 2. Springer London, Limited, 2012.

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22

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

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23

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

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24

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

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25

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

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26

Montuschi, Paolo. New Perspectives in Monitoring Lung Inflammation: Analysis of Exhaled Breath Condensate. Taylor & Francis Group, 2004.

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27

Arnaud, P., and J. Bienvenu. Marker Proteins in Inflammation: Proceedings of the 2nd Symposium Lyon, France, June 27-30, 1983. Walter De Gruyter Inc, 1985.

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28

Suskind, Robert M., Jacques Bienvenu, Robert C. Allen, and Philippe Laurent. Marker Proteins in Inflammation: Proceedings of the Symposium Lyon, France, April 22-25 1981. de Gruyter GmbH, Walter, 2019.

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29

Grimaud, J. A., Jacques Bienvenu, and Philippe Laurent. Marker Proteins in Inflammation: Proceedings of the Third Symposium. Lyon, France, June 26-28 1985. De Gruyter, Inc., 2019.

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30

(Editor), Jacques Bienvenu, P. Laurent (Editor), and J. A. Grimaud (Editor), eds. Marker Proteins in Inflammation Vol. 3: Proceedings of the 3rd Symposium, Lyon, France, June 26-28, 1985. Walter de Gruyter, 1986.

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31

Edwards, Chris, and David Warwick. Rheumatology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757689.003.0012.

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A number of rheumatological diseases are manifest in the hand and wrist. Many are associated with considerable systemic inflammation. This produces chronic ill-health, poor wound healing, and osteoporosis. Attempts to eliminate inflammation at the earliest opportunity are vital. Multiple involved joints with high acute phase markers (CRP and erythrocyte sedimentation rate raised) and early morning stiffness are key features. Multi-disciplinary management, involving the rheumatologist and hand therapist is essential. With improving medical management, surgical problems are becoming far less com

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32

Popescu, Bogdan Florin Gh, Yong Guo, and Claudia Francesca Lucchinetti. Multiple Sclerosis: Pathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0081.

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The pathology of MS consists of areas of focal demyelination, known as plaques or lesions, characterized by inflammation, gliosis, and relative axonal preservation. Recent neuropathological studies have established that white matter lesions are heterogenous with respect to the targets of injury and mechanisms of demyelination, highlighting the need for the identification of surrogate clinical and/or paraclinical markers that would correlate with immunopatterns in the general MS population and for the design of novel therapeutic strategies specifically tailored to each immunopattern. Recent neu

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33

Ferran, Christiane. The Multiple Therapeutic Targets of A20. Springer, 2014.

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34

Ferran, Christiane. Multiple Therapeutic Targets of A20. Springer London, Limited, 2014.

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35

Ferran, Christiane. The Multiple Therapeutic Targets of A20. Springer, 2016.

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36

Ley, Klaus. Inflammation. WORLD SCIENTIFIC, 2017. http://dx.doi.org/10.1142/10028.

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37

Palmer, D. D. Inflammation. Kessinger Publishing, LLC, 2006.

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38

Wagenlehner, Florian M. E., Adrian Pilatz, Thomas Bschleipfer, Thorsten Diemer, and Wolfgang Weidner. Inflammation. Edited by Rob Pickard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0007.

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There is a consensus on the diagnostic and therapeutic manage¬ment of bacterial prostatitis (acute and chronic). However, increas¬ing antimicrobial resistance rates for quinolones pose problems for the future, especially for therapy of chronic bac¬terial prostatitis. In chronic prostatitis/chronic pelvic pain syndrome, the diag¬nostic approach currently points more and more to an individu¬alized phenotypic assessment, in an effort to direct multimodal management towards improvement of specific symptom domains. Most therapy trials for single agents in CP/CPPS have been nega¬tive, therefore stra

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39

Garthwaite, Mary. Inflammation. Edited by Rob Pickard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0008.

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This section provides key knowledge on the pathogenesis and natural history of epididymitis and scrotal abscess, as well as providing a framework for the assessment, investigation, and management of patients presenting with these conditions. The information provided links to the knowledge and clinical skills requirement of Intermediate stage topics ‘Urinary Tract Infection’ and ‘Emergency Urology’, within the syllabus of the United Kingdom (UK) Intercollegiate Surgical Curriculum Project (ISCP) Urology Curriculum (https://www.iscp.ac.uk/). There is a lack of data concerning the incidence and p

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40

Greco, Francesco, and Paolo Fornara. Inflammation. Edited by Rob Pickard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0009.

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Fournier’s gangrene (FG) defines a life-threatening necrotizing fasciitis of the external genitalia and perineum with a peak incidence at 50 years of age. It is associated with a mixed bacterial infection with Escherichia coli and Bacteroides spp. predominating. The diagnosis is made clinically with symptoms of skin necrosis, swelling, pain, crepitus, and feculent odour, and local and systemic signs of severe sepsis. Initial resuscitation with fluid replacement, oxygen therapy, and broad-spectrum empirical antibiotics should be rapidly followed by complete and aggressive surgical debridement w

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41

Allison, A. C. Inflammation. Springer, 2013.

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42

Górski, Andrzej, Hubert Krotkiewski, and Michal Zimecki. Inflammation. Springer London, Limited, 2013.

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43

Vane, John R., and Sergio H. Ferreira. Inflammation. Brand: Springer, 2011.

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44

(Editor), Andrzej Górski, Hubert Krotkiewski (Editor), and Michal Zimecki (Editor), eds. Inflammation. Springer, 2001.

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45

Górski, Andrzej, Hubert Krotkiewski, and Michal Zimecki. Inflammation. Springer, 2010.

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46

DeAugustinas, M., and A. Kiely. Endophthalmitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0017.

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Endophthalmitis refers to inflammation of both the anterior and posterior intraocular chambers and their structures. This vision-threatening condition occurs in three principal scenarios: penetrating ocular trauma, after intraocular surgery, and in systemically infected (often immunocompromised) patients. Endophthalmitis presents with marked intraocular inflammation, often with hypopyon. Patients report pain and significant vision loss out of proportion to typical post-operative complaints. It is distinguished from uveitis by both history and slit lamp examination. Endophthalmitis is an ophtha

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47

Speer, Thimoteus, and Danilo Fliser. Abnormal endothelial vasomotor and secretory function. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0113.

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The endothelium plays a crucial role in the maintenance of vascular integrity and function. Nitric oxide produced by endothelial cells is a key player, inducing relaxation of vascular smooth muscle cells, inhibition of vascular inflammation, and prevention of coagulatory activation. Chronic kidney disease (CKD) is characterized by deterioration of different protective endothelial properties, collectively described as endothelial dysfunction. Several factors such as methylarginines, modified lipoproteins, and other substances that accumulate may be involved in the pathogenesis of endothelial dy

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48

Twisk, Jos, and Isabel Ferreira. Physical activity, physical fitness, and cardiovascular health. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199232482.003.0025.

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The incidence of morbidity and mortality related to CVD is rather low in a paediatric population. Studies investigating the relationship between physical activity, physical fitness, and cardiovascular health in children and adolescents are therefore mostly limited to CVD risk factors as outcome measures. For this reason, this chapter will focus on the association of physical activity and physical fitness with CVD risk factors in children and adolescents. These risk factors can be divided into the so-called traditional CVD risk factors; that is, lipoproteins [total cholesterol, low-density lipo

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49

Srivastava, S. Epigenetic Markers (Disease Markers). Ios Press, 2007.

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50

Warren, George. Markers. Globe Fearon Co, 1985.

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