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Journal articles on the topic 'As markers of inflammation'

Author: Grafiati

Published: 4 June 2025

Last updated: 1 August 2025

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1

Johnkennedy, Nnodim, and Okafor Chibuzor Mercy. "Perspective of Inflammation and Inflammation Markers." Journal La Medihealtico 3, no. 1 (2022): 16–26. http://dx.doi.org/10.37899/journallamedihealtico.v3i1.620.

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Progression and occurrence of coronary heart disease can be attributed in part to the presence of inflammation. As a result of our understanding of inflammation's molecular underpinnings, we have identified markers that may also serve as new treatment targets for atherosclerosis. Individuals with and without a history of cardiovascular disease can benefit from monitoring their C-reactive protein (CRP) levels (CVD). The anti-inflammatory characteristics of statins have lately been explored, and they have been shown to significantly lower cardiovascular morbidity and death. C-reactive protein, a

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2

Kharitonov, Sergei A., and Peter J. Barnes. "Exhaled markers of inflammation." Current Opinion in Allergy and Clinical Immunology 1, no. 3 (2001): 217–24. http://dx.doi.org/10.1097/01.all.0000011017.58506.f1.

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3

BEHAR-COHEN, F. "Early markers of inflammation." Acta Ophthalmologica 90 (August 6, 2012): 0. http://dx.doi.org/10.1111/j.1755-3768.2012.3512.x.

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4

COSCAS, G. "Clinical markers of inflammation." Acta Ophthalmologica 90 (August 6, 2012): 0. http://dx.doi.org/10.1111/j.1755-3768.2012.3513.x.

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5

Kharitonov, Sergei A., and Peter J. Barnes. "Exhaled markers of inflammation." Current Opinion in Allergy and Clinical Immunology 1, no. 3 (2001): 217–24. http://dx.doi.org/10.1097/00130832-200106000-00004.

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6

Morozov, A., S. Zhukov, A. Morozova, et al. "Modern markers of inflammation." Vrach 33, no. 11 (2022): 47–52. http://dx.doi.org/10.29296/25877305-2022-11-08.

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7

Kuznetsova, L., О. Gopko, and L. Savchenko. "Markers of systemic inflammation in COPD patients with obesity." Ukraïnsʹkij žurnal medicini, bìologìï ta sportu 5, no. 3 (2020): 68–73. http://dx.doi.org/10.26693/jmbs05.03.068.

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8

Tsounis, Dimitrios, Georgios Bouras, Georgios Giannopoulos, Charalampos Papadimitriou, Dimitrios Alexopoulos, and Spyridon Deftereos. "Inflammation Markers in Essential Hypertension." Medicinal Chemistry 10, no. 7 (2014): 672–81. http://dx.doi.org/10.2174/1573406410666140318111328.

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9

Kusnir, P., M. Dohal, I. Porvaznik, and J. Mokry. "Serum Inflammation Markers in Tuberculosis." Acta Medica Martiniana 20, no. 3 (2020): 103–13. http://dx.doi.org/10.2478/acm-2020-0012.

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AbstractTuberculosis remains one of the leading infectious cause of death in the world. The goals of screening are to detect active tuberculosis early enough and to identify individuals eligible for preventive therapy to reduce a po tential co-infection by tuberculosis. Plasma/serum screening for selected potential biomarkers could represent a suitable method of tuberculosis diagnosis and treatment outcome. Furthermore, monitoring of tuberculosis treatment is crucial to clinical decision-making and besides the plasmatic concentration of administered antituberculosis drugs, the biomarkers appea

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10

Venge, P. "Soluble markers of allergic inflammation." Allergy 49, no. 1 (1994): 1–8. http://dx.doi.org/10.1111/j.1398-9995.1994.tb00765.x.

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11

Sherwood, Roy A. "Faecal markers of gastrointestinal inflammation." Journal of Clinical Pathology 65, no. 11 (2012): 981–85. http://dx.doi.org/10.1136/jclinpath-2012-200901.

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Gastrointestinal (GI) symptoms including abdominal pain, bloating and diarrhoea are a relatively common reason for consulting a physician. They may be due to inflammatory bowel disease (inflammatory bowel disease; Crohn's disease, ulcerative colitis and indeterminate colitis), malignancy (colorectal cancer), infectious colitis or irritable bowel syndrome (IBS). Differentiation between these involves the use of clinical, radiological, endoscopic and serological techniques, which are invasive or involve exposure to radiation. Serological markers include C-reactive protein, erythrocyte sedimentat

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12

Kabutomori, Osamu, Yuzuru Kanakura, and Yoshinori Iwatani. "Inflammation markers and liver dysfunction." Annals of Hematology 84, no. 2 (2004): 136. http://dx.doi.org/10.1007/s00277-004-0967-3.

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13

Kabutomori, Osamu, Yuzuru Kanakura, and Yoshinori Iwatani. "Inflammation markers and liver dysfunction." Annals of Hematology 84, no. 6 (2005): 416. http://dx.doi.org/10.1007/s00277-005-1029-1.

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14

Lang, Hermann, and Siegfried Neumann. "Analytical procedures for inflammation markers." Fresenius' Zeitschrift für analytische Chemie 324, no. 3-4 (1986): 229. http://dx.doi.org/10.1007/bf00487874.

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15

Heber, David. "Oxidative Stress Markers and Inflammation." Nutrition Today 49, Supplement (2014): S4—S5. http://dx.doi.org/10.1097/01.nt.0000453844.83969.21.

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16

Takala, Annika, Irmeli Nupponen, Marja-Leena Kylänpää-Bäck, and Heikki Repo. "Markers of inflammation in sepsis." Annals of Medicine 34, no. 7 (2002): 614–23. http://dx.doi.org/10.1080/078538902321117841.

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17

Onuora, Sarah. "Obesity skews markers of inflammation." Nature Reviews Rheumatology 13, no. 6 (2017): 323. http://dx.doi.org/10.1038/nrrheum.2017.62.

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18

Venge, P. "Markers of the allergic inflammation." Journal of the European Academy of Dermatology and Venereology 5, no. 1 (1995): S182. http://dx.doi.org/10.1016/0926-9959(95)96525-d.

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19

Wu, Meng-Hsing, Kuei-Yang Hsiao, and Shaw-Jenq Tsai. "Endometriosis and possible inflammation markers." Gynecology and Minimally Invasive Therapy 4, no. 3 (2015): 61–67. http://dx.doi.org/10.1016/j.gmit.2015.05.001.

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20

Mousa, Ahmad, and Julia V. Deyeva. "Objective markers of allergic inflammation." OTORHINOLARYNGOLOGY, No1-2(6) 2023 (April 10, 2023): 30–37. http://dx.doi.org/10.37219/2528-8253-2023-1-30.

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Introduction: According to the results of modern research, allergic rhinitis (AR) affects 10% to 40% of the population. There is a need for an effective objective and dynamic method of assessing the quality of treatment of such patients. Purpose: to assess the relationship between the level of eosinophil cationic protein (ECB), VAS indicators, nasocytogram, Phadiatop inhalation allergy screening and disease activity in patients with allergic rhinitis. Methods and materials: Two research groups were created. The main group – 48 patients with allergic rhinitis in a state of exacerbation, the con

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21

Carra*, Brian Irving, and Rossella Donghiab. "Inflammation Markers and HCC Aggressiveness." Journal of Biomedical Research & Environmental Sciences 6, no. 4 (2025): 378–87. https://doi.org/10.37871/jbres2093.

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Background: Inflammation is thought to be important in the development and progression of Hepatocellular Carcinoma (HCC), but which inflammatory indices are more useful in clinical practice is not clear. Aims: Several inflammatory indices were examined with respect to Maximum Tumor Diameter (MTD), Portal Vein Thrombosis by Tumor (PVT) and survival. Results: Serum C-Reactive Protein (CRP) and Platelet Lymphocyte Ratio (PLR) each significantly increased with increasing MTD as did percent patients with higher Glasgow index; whereas albumin levels and HALP (Hemoglobin, Albumin, Lymphocytes and Pla

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22

Mills, Robert Coleman. "Breast Cancer Survivors, Common Markers of Inflammation, and Exercise: A Narrative Review." Breast Cancer: Basic and Clinical Research 11 (January 1, 2017): 117822341774397. http://dx.doi.org/10.1177/1178223417743976.

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Exercise may help positively improve inflammatory marker levels, therefore promoting better outcomes in breast cancer survivors. This narrative review is intended to provide an overview between inflammation and breast cancer, in addition to the effects exercise may have on common inflammatory markers that have been examined in both healthy populations and breast cancer survivors throughout the literature. The inconsistencies and gaps in the literature addressed may be important for future research to further understand the relationship between exercise and inflammation, as well as the underlyi

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23

Calder, P. C., N. Ahluwalia, R. Albers, et al. "A Consideration of Biomarkers to be Used for Evaluation of Inflammation in Human Nutritional Studies." British Journal of Nutrition 109, S1 (2013): S1—S34. http://dx.doi.org/10.1017/s0007114512005119.

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To monitor inflammation in a meaningful way, the markers used must be valid: they must reflect the inflammatory process under study and they must be predictive of future health status. In 2009, the Nutrition and Immunity Task Force of the International Life Sciences Institute, European Branch, organized an expert group to attempt to identify robust and predictive markers, or patterns or clusters of markers, which can be used to assess inflammation in human nutrition studies in the general population. Inflammation is a normal process and there are a number of cells and mediators involved. These

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24

Majid, A., and M. Fouad. "Serum visfatin, resistin levels and inflammation markers in psoriasis patients." Ukrainian Biochemical Journal 94, no. 6 (2023): 48–56. http://dx.doi.org/10.15407/ubj94.06.048.

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Psoriasis is a common chronic inflammatory skin condition that varies in severity. Psoriasis is associated with complex disorders, which incorporate metabolic syndrome, obesity and impaired glucose tolerance. Adipose tissue secretes several hormones and cytokines, in particular visfatin and resistin that could be involved in the development of psoriasis by acting as pro-inflammatory or immunoregulatory factors. The aim of this work was to evaluate the serum level of visfatin and resistin as well as of high-sensitivity C-reactive protein (hs-CRP) and erythrocyte sedimentation rate (ESR) in psor

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25

Schildkraut, Joellen M. "Invited Commentary: Relationship Between Ovulation and Markers of Systemic Inflammation Versus Markers of Localized Inflammation." American Journal of Epidemiology 189, no. 7 (2019): 671–73. http://dx.doi.org/10.1093/aje/kwz265.

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Abstract In this issue of the American Journal of Epidemiology, Huang et al. (Am J Epidemiol. 2020;189(7):660–670) report an inverse relationship between lifetime ovulatory years (LOY) and circulating levels of C-reactive protein (CRP), counter to their hypothesis. CRP is a nonspecific systemic marker of chronic inflammation, and there is evidence that there are other drivers of systemic inflammation as measured by CRP. There also is a body of evidence supporting the possibility that ovulation may be responsible for a localized inflammatory response. Because ovarian cancer is the fifth most co

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26

Rebrova, S. A. "Clinical significance of exhaled nitric oxide, nasal nitric oxide, peripheral blood eosinophiles as markers of airway inflammation in bronchial asthma and allergic rhinitis." Russian Otorhinolaryngology 22, no. 1 (2023): 79–85. http://dx.doi.org/10.18692/1810-4800-2023-1-79-85.

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Chronic airway inflammation plays an important role in the development and progression of bronchial asthma (BA) and allergic rhinitis (AR). The lack of correlation between the severity of clinical symptoms and the activity of inflammation determines the need to develop and introduce various biomarkers into clinical practice. Allergic ВА and AR are predominantly based on eosinophilic inflammation. Sufficiently studied markers of eosinophilic inflammation of the respiratory tract are peripheral blood eosinophils, exhaled nitric oxide (FeNO), nasal nitric oxide (nNO). FeNO increases in BA and AR,

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27

Saarni, Suoma E., Britt-Marie Loo, Samuli Saarni, et al. "CHRONIC INFLAMMATION IN SCHIZOPHRENIA – EFFECT OF OBESITY ON INFLAMMATION MARKERS." Schizophrenia Research 117, no. 2-3 (2010): 318. http://dx.doi.org/10.1016/j.schres.2010.02.539.

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28

Rodríguez-Hernández, Heriberto, Luis E. Simental-Mendía, Gabriela Rodríguez-Ramírez, and Miguel A. Reyes-Romero. "Obesity and Inflammation: Epidemiology, Risk Factors, and Markers of Inflammation." International Journal of Endocrinology 2013 (2013): 1–11. http://dx.doi.org/10.1155/2013/678159.

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Obesity is a public health problem that has reached epidemic proportions with an increasing worldwide prevalence. The global emergence of obesity increases the risk of developing chronic metabolic disorders. Thus, it is an economic issue that increased the costs of the comorbidities associated. Moreover, in recent years, it has been demonstrated that obesity is associated with chronic systemic inflammation, this status is conditioned by the innate immune system activation in adipose tissue that promotes an increase in the production and release of pro-inflammatory cytokines that contribute to

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29

Nędzi-Góra, Małgorzata, Jolanta Kostrzewa-Janicka, and Renata Górska. "Markers of inflammation in periodontal diseases." Central European Journal of Immunology 3 (2013): 363–66. http://dx.doi.org/10.5114/ceji.2013.37746.

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30

Loos, Bruno G. "Systemic Markers of Inflammation in Periodontitis." Journal of Periodontology 76, no. 11-s (2005): 2106–15. http://dx.doi.org/10.1902/jop.2005.76.11-s.2106.

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31

Martínez, Vicente Bertomeu, and J. Ramón González-Juanatey. "Markers of Inflammation and Cardiovascular Disease." American Journal Cardiovascular Drugs 9, Supplement 1 (2009): 3–7. http://dx.doi.org/10.2165/1153161-s0-000000000-00000.

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32

ROSA, Antonella, Veronica DIURNI, Antonella PLACIDO, and Giuliano BERTAZZONI. "Markers of inflammation in atrial fibrillation." Acta Cardiologica 58, no. 1 (2003): 43–44. http://dx.doi.org/10.2143/ac.58.1.2005260.

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33

Pearson, Thomas A., George A. Mensah, R. Wayne Alexander, et al. "Markers of Inflammation and Cardiovascular Disease." Circulation 107, no. 3 (2003): 499–511. http://dx.doi.org/10.1161/01.cir.0000052939.59093.45.

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34

&NA;. "Inflammation Markers Signal Advancing Coronary Disease." Emergency Medicine News 26, no. 12 (2004): 45. http://dx.doi.org/10.1097/00132981-200412000-00033.

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35

A, Pearson T., Mensah G. A, and Alexander R. W. "MARKERS OF INFLAMMATION AND CARDIOVASCULAR DISEASE." Journal of Cardiopulmonary Rehabilitation 23, no. 3 (2003): 236–37. http://dx.doi.org/10.1097/00008483-200305000-00016.

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36

BHATIA, V. K. "Markers of inflammation in unstable angina." Heart 85, no. 6 (2001): 603–4. http://dx.doi.org/10.1136/heart.85.6.603.

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37

Lind, L. "Circulating markers of inflammation and atherosclerosis." Atherosclerosis 169, no. 2 (2003): 203–14. http://dx.doi.org/10.1016/s0021-9150(03)00012-1.

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38

Bartosik-Psujek, H., E. Belniak, and Z. Stelmasiak. "Markers of inflammation in cerebral ischemia." Neurological Sciences 24, no. 4 (2003): 279–80. http://dx.doi.org/10.1007/s10072-003-0156-5.

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39

Wang, Tracy Y., Wael A. AlJaroudi, and L. Kristin Newby. "Markers of Cardiac Ischemia and Inflammation." Cardiology Clinics 23, no. 4 (2005): 491–501. http://dx.doi.org/10.1016/j.ccl.2005.08.007.

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40

Tracy, Russell P. "Inflammation markers and coronary heart disease." Current Opinion in Lipidology 10, no. 5 (1999): 435–42. http://dx.doi.org/10.1097/00041433-199910000-00008.

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41

Goseva, Zlatica, Angelko Gjorcev, Dejan Dokic, Tatjana Caparoska, and Zoran Arsovski. "Markers of inflammation in asthmatic patients." World Allergy Organization Journal &NA; (November 2007): S69. http://dx.doi.org/10.1097/01.wox.0000301321.58461.ed.

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42

Dotsenko, V., and E. Kligunenko. "Influence of bupivacaine on inflammation markers." European Journal of Anaesthesiology 29 (June 2012): 132. http://dx.doi.org/10.1097/00003643-201206001-00432.

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43

Soraya Amali, Zavaleta-Muniz, and Maravilla-Dominguez Maria Aurora. "Diabetes: Its Complications and Inflammation Markers." Journal of Biomedical Research & Environmental Sciences 3, no. 12 (2022): 1538–40. http://dx.doi.org/10.37871/jbres1631.

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Type 2 Diabetes (T2DM) is a chronic inflammatory metabolic disease of high prevalence around the world. The elevated inflammation markers such as Neutrophil Lymphocyte Ratio (NLR), and Platelet Lymphocyte Ratio (PLR) are associated with macrovascular and microvascular complications and have been described as a predictors of mortality in patients with renal damage, atherosclerosis and cardiovascular diseases. The chronic inflammation state and the oxidative stress associated with aging could accelerate the microvascular complications in diabetic patients.

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44

Virani, Salim S., Venkateshwar R. Polsani, and Vijay Nambi. "Novel markers of inflammation in atherosclerosis." Current Atherosclerosis Reports 10, no. 2 (2008): 164–70. http://dx.doi.org/10.1007/s11883-008-0024-0.

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45

KESTELOOT, H. "Alcohol intake and markers of inflammation." European Heart Journal 25, no. 23 (2004): 2075–76. http://dx.doi.org/10.1016/j.ehj.2004.09.029.

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46

Ugge, Henrik, Mary K. Downer, Jessica Carlsson, et al. "Circulating inflammation markers and prostate cancer." Prostate 79, no. 11 (2019): 1338–46. http://dx.doi.org/10.1002/pros.23842.

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47

Floc'h, Johann Le, Winston Tan, Ravindra S. Telang, et al. "Markers of cochlear inflammation using MRI." Journal of Magnetic Resonance Imaging 39, no. 1 (2013): 150–61. http://dx.doi.org/10.1002/jmri.24144.

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48

Mashnafi, Sultan, Sabine Baumgartner, Ronald P. Mensink, et al. "A Transient Inflammatory Response Induced by Lipopolysaccharide Infusion Lowers Markers of Endogenous Cholesterol and Bile Acid Synthesis in Healthy Normocholesterolemic Young Men." Biomedicines 11, no. 1 (2023): 126. http://dx.doi.org/10.3390/biomedicines11010126.

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Inflammation is associated with changes in plasma lipids, lipoproteins, and cholesterol efflux capacity (CEC). It is unknown if the changes in lipids and lipoproteins during inflammation are related to changes in cholesterol absorption, synthesis, and bile acid synthesis. We, therefore, examined the effects of acute lipopolysaccharide (LPS)-induced transient systemic inflammation on lipids, lipoproteins, CEC, and markers of cholesterol metabolism. We also evaluated whether markers for cholesterol metabolism at baseline predict the intensity of the inflammatory response. Eight healthy young sub

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49

Bohn, Bruno, Curtis Tilves, Yingan Chen, et al. "Associations of gut microbiota features and circulating metabolites with systemic inflammation in children." BMJ Open Gastroenterology 11, no. 1 (2024): e001470. http://dx.doi.org/10.1136/bmjgast-2024-001470.

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ObjectiveGut microbes and microbe-dependent metabolites (eg, tryptophan-kynurenine-serotonin pathway metabolites) have been linked to systemic inflammation, but the microbiota-metabolite-inflammation axis remains uncharacterised in children. Here we investigated whether gut microbiota features and circulating metabolites (both microbe-dependent and non-microbe-dependent metabolites) associated with circulating inflammation markers in children.MethodsWe studied children from the prospective Gen3G birth cohort who had data on untargeted plasma metabolome (n=321 children; Metabolon platform), gut

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50

Inflammatory, markers: An Overview. "Inflammatory markers: An Overview." Science World a monthly e magazine 2, no. 11 (2022): 1845–49. https://doi.org/10.5281/zenodo.7339579.

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Inflammation is the body’s innate response to injury or insult, including infection, trauma, surgery, burns, and cancer. Certain proteins are released into the bloodstream during inflammation; if their concentrations increase or decrease by at least 25%, they can be used as systemic inflammatory markers. Although there are many inflammatory markers, also known as acute phase reactants, those most commonly measured in clinical practice are C-reactive protein (CRP), erythrocyte sedimentation rate (ESR), and procalcitonin (PCT). PCT is a newer marker of inflammation that may, in certain cas

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