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Relevant bibliographies by topics / Ascites

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Academic literature on the topic 'Ascites'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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Journal articles on the topic "Ascites"

1

Sastry, Anand Sankar, Subash Ch Mahapatra, and Vidyasagar Dumpula. "Ascitic fluid analysis with special reference to serum ascites cholesterol gradient and serum ascites albumin gradient." International Journal of Research in Medical Sciences 5, no. 2 (2017): 429. http://dx.doi.org/10.18203/2320-6012.ijrms20170059.

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Background: Ascites being a common clinical problem with a vast spectrum of etiologies, less expensive and widely available biochemical parameters are required to differentiate ascites which can correlate with pathogenesis and pin point towards an etiology with high sensitivity and significant accuracy. Aims of the study were to determine the sensitivity, specificity and diagnostic efficacy of serum ascites albumin Gradient (SAAG) and that of ascitic fluid total protein (AFTP), evaluating their diagnostic role in identifying the etiology of ascites, to determine the diagnostic efficacy of Asci

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Joksimović, Zoran, and Dušan Bastać. "Ascites u cirozi jetre." Timocki medicinski glasnik 49, no. 3-4 (2024): 15–19. https://doi.org/10.5937/tmg2403069j.

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Ascit ili ascites (grč. ascίtes-askites-mešina) je medicinski izraz kojim se označava nakupljanje slobodne tečnosti u trbušnoj duplji. Javlja se kao jedan od znakova kod mnogih bolesti i poremećaja u trbušnoj duplji. Hepatički uzroci ascitesa: kod ciroze jetre, akutne insuficijencije jetre, alkoholnog hepatitisa, tromboze vene porte, Budd-Chiari sindroma, difuznih metastaza malignih tumora u jetri i venookluzivne bolesti. Nehepatički uzroci ascitesa su srčana insuficijencija, plućna hipertenzija, konstriktivni perikardit, nefrotski sindrom, karcinomatoza peritoneuma, tuberkuloza, akutni pankre

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Suman, Shanker, Divya Jyoti, Pramod Kumar Agrawal, and Bijoy Kumar Bhattacharya. "Clinicopathological correlation of serum ascites albumin gradient with ascitic fluid total protein in patients of ascites with portal hypertension attending a tertiary care hospital in Eastern Bihar, India." International Journal of Advances in Medicine 4, no. 3 (2017): 842. http://dx.doi.org/10.18203/2349-3933.ijam20172282.

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Background: The level of ascitic fluid total protein (AFTP) is used to differentiate between transudative and exudative ascites. Ascites patients having portal hypertension are considered to be transudative in nature. The traditional transudate/exudate system of ascitic fluid classification based on ascitic fluid total protein concentration is sometimes misleading in patients of ascites with portal hypertension. Now a days SAAG (serum ascites albumin gradient) has become more acceptable in differentiating patients presenting with ascites due to portal hypertension. The objective of this prospe

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Castaldo, G., M. Intrieri, G. Calcagno, et al. "Ascitic pseudouridine discriminates between hepatocarcinoma-derived ascites and cirrhotic ascites." Clinical Chemistry 42, no. 11 (1996): 1843–46. http://dx.doi.org/10.1093/clinchem/42.11.1843.

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Abstract Various biochemical indexes discriminate neoplastic from nonneoplastic ascites. However, within the latter group, the distinction between cirrhotic ascites and ascites caused by hepatocarcinoma (HC) is usually based on liver biopsy or cytology. HC-derived ascites is included in the group of nonneoplastic ascites because it is not associated with peritoneal spreading of neoplastic cells. In 54 cases of cirrhotic ascites and 17 cases of HC ascites, all histologically diagnosed, ascitic pseudouridine concentrations discriminated cirrhotic from HC ascites. For example, using the cutoff va

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Mi, Wei, Tetsu Fukunaga, and Yang Yu. "Observation on the Therapeutic Effect of Hypotonic Hyperthermic Intraperitoneal Chemotherapy in the Treatment of Advanced Gastric Cancer and Ovarian Cancer Accompanied by Ascites." Proceedings of Anticancer Research 8, no. 6 (2024): 251–56. http://dx.doi.org/10.26689/par.v8i6.8936.

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Objective: To observe the time of ascites disappearance, time to ascites recurrence, improvement rate in quality of life, and the effective rate of ascites treatment in patients with advanced gastric and ovarian cancer with ascites following hypotonic hyperthermic intraperitoneal chemotherapy. Methods: Forty patients with advanced gastric and ovarian cancer with ascites, treated in our hospital from January 2021 to August 2024, were selected as research subjects. They were divided into a treatment group and a reference group using a random number table method. The treatment group received hypo

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Sun, Juan, Yan-Xiang Chang, and Chun-Yan Niu. "Evaluation of ascitic soluble human leukocyte antigen-G for distinguishing malignant ascites from benign ascites." Tumor Biology 39, no. 11 (2017): 101042831772684. http://dx.doi.org/10.1177/1010428317726840.

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The overexpression of soluble human leukocyte antigen-G is associated with malignant tumours. The purpose of our study was to detect soluble human leukocyte antigen-G concentrations in ascites and to evaluate the value of ascitic soluble human leukocyte antigen-G for the diagnosis of malignant ascites. Enzyme-linked immunosorbent assay was used to detect soluble human leukocyte antigen-G levels in 64 patients with malignant ascites and 30 patients with benign ascites. Receiver operating characteristic curves were used to evaluate the diagnostic efficacy of ascitic soluble human leukocyte antig

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Kalmegh, Rohan, and Sameer Chaudhary. "Study of role of amniotic fluid total protein (AFTP) assessment in the diagnosis of tubercular ascites." International Journal of Advances in Medicine 5, no. 4 (2018): 909. http://dx.doi.org/10.18203/2349-3933.ijam20183117.

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Background: It is not economically feasible to estimate the serum and ascitic albumin level for calculating Serum Ascites Albumin Gradient (SAAG) in every suspected tubercular ascites patient. Ascites fluid total protein (AFTP) seems a cheaper and viable alternative. Authors attempted to compare the efficacy of Amniotic Fluid Total Protein (AFTP) against Serum Ascites Albumin Gradient (SAAG) in the diagnosis of Tubercular Ascites.Methods: Patients who were admitted to Medicine Wards, with the complaints of distension of abdomen, proved by ultrasound to have ascites, were included in the study.

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Yun, Jina, Ju-Sun Song, Jeong-Ju Yoo, et al. "Microbial and Immune Landscape of Malignant Ascites: Insights from Gut, Bladder, and Ascitic Fluid Analyses." Cancers 17, no. 8 (2025): 1280. https://doi.org/10.3390/cancers17081280.

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Background/Objectives: Malignant ascites frequently arises in advanced cancers with peritoneal metastasis and is associated with poor outcomes. Known mechanisms include lymphatic obstruction by tumor cells, increased vascular permeability, and sodium retention via the renin–angiotensin–aldosterone system; however, the pathogenesis remains not fully understood. We investigated whether gut and bladder microbiomes correlate with malignant ascites development or progression and whether the immune microenvironment in ascitic fluid is altered. Methods: We enrolled 66 histologically confirmed cancer

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Dede, Hysni, Rovena Roshi, Rovena Bode, Enver Roshi, and Jovan Basho. "The Role of Serum Ascites Albumin Gradient in the Differential Diagnosis of Ascites." Albanian Journal of Trauma and Emergency Surgery 6, no. 1 (2022): 933–38. http://dx.doi.org/10.32391/ajtes.v6i1.268.

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Introduction: Ascites is of Greek derivation (“askos”) and refers to a bag or sack. The word is a noun and describes pathologic fluid accumulation within the peritoneal cavity. Orientation in finding or excluding portal hypertension through examination of ascitic fluid is the first step towards an accurate diagnosis. Material and Methods: The aim of this study was to evaluate the role of SAAG (Serum Ascites Albumin Gradient) in the differential diagnosis between cirrhotic and malignant ascites. The SAAG is obtained by subtracting the value of serum albumin, the value of ascites albumin (

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Khan, Salman, Ihsan Ullah, Moeen ul Haq, Umar Badshah, and Maryam Nazir. "Comparison of Diagnostic Accuracy of Portal Serum Ascitic Albumin Gradient (SAAG) with Ascitic Fluid Total Protien (AFTP) Differentiating Portal Hypertension from Non-Portal in Patients with Ascites." Pakistan Journal of Medical and Health Sciences 15, no. 6 (2021): 1924–26. http://dx.doi.org/10.53350/pjmhs211561924.

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Introduction: Although majority of the cases of ascites have cirrhosis, there are 15% patients where there is a non-hepatic cause of fluid retention like malignancy, congestive heart failure and tuberculous peritonitis. Ascites is the most common complication of cirrhosis that leads to hospital admission. Objective:To compare the diagnostic Accuracy of Serum Ascitic Albumin Gradient (SAAG) and Ascitic Fluid Total Proteins in patients with ascites by taking Ultrasound abdomen & Pelvis as gold standard. There are international studies on the accuracy of SAAG in determining cause of ascites b

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Dissertations / Theses on the topic "Ascites"

1

Curutchet, Elisabeth. "Les ascites pancréatiques : à propos d'un cas d'ascite chez un malade porteur d'une pancréatite chronique." Bordeaux 2, 1989. http://www.theses.fr/1989BOR25134.

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Jeandroz-Madec, Véronique. "Les ascites tuberculeuses : à propos de dix observations, actualité et perspectives." Bordeaux 2, 1992. http://www.theses.fr/1992BOR23123.

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Benesse, Patrick. "Anastomose porto-cave intrahépathique par voie transjugulaire dans l'ascite réfractaire : à propos de 9 cas." Bordeaux 2, 1993. http://www.theses.fr/1993BOR2M192.

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Arthaud, Anne. "Physiopathologie et diagnostic biologique des ascites cirrhotiques et de leurs complications infectieuses." Paris 5, 1988. http://www.theses.fr/1988PA05P021.

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Serste, Thomas. "Identification de facteurs pronostiques délétères dans la cirrhose compliquée d'ascite réfractaire." Doctoral thesis, Universite Libre de Bruxelles, 2012. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209663.

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Cirrhosis is characterized by the progressive development of portal hypertension. Portal hypertension, associated with sodium retention, leads to the formation of ascites. Ascites may be difficult to treat and, thereby, become "refractory". To date, the mortality of patients with refractory ascites remains high. In order to offer patients adequate treatment, it is important to identify complications that may develop and the factors of good and poor prognosis affecting their survival. One can retain five specific complications of cirrhosis with ascites: hepatorenal syndrome, spontaneous bacteri

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Bell, Mary Siobhan. "Malignancy antigens of the Erlich ascites cell." Thesis, Queen's University Belfast, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.317049.

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Navarro-Martínez, Pau. "Genetic studies of ascites in broiler populations." Thesis, University of Edinburgh, 2003. http://hdl.handle.net/1842/15495.

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Continuous genetic improvement of growth and conformation traits in broiler populations has coincided with an increase in defects in heart and lung function. These defects have led to an increased incidence of metabolic disorders such as ascites (AS) (or pulmonary hypertension), a functional hypoxia. The incidence of AS in well-managed flocks is low, but it nonetheless causes important economic losses to the breeding industry and is an important issue from a welfare standpoint. The aim of this thesis was to study the genetics of AS-related quantitative traits. A low blood oxygen saturation (Sa

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Udwan, Khalil. "Role of oxidative stress in primary sodium retention and edema formation in nephrotic syndrome." Thesis, Paris 6, 2015. http://www.theses.fr/2015PA066331/document.

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Le syndrome néphrotique (SN) est causé par une altération glomérulaire, responsable d’une excrétion urinaire anormale de protéines plasmatiques, compliquée d’hypoalbuminémie. Le SN est toujours associé à une rétention rénale de Na + qui conduit à la génération d'ascite et / ou d'œdèmes. La pathogénie de la rétention de Na + et de la constitution d’œdèmes n’est pas entièrement élucidée. Dans notre étude, nous avons évalué le rôle possible des espèces réactives de l'oxygène (ROS) dans cette pathogénie. Notre étude dans le modèle de rat aminonucléoside puromycine (PAN) de SN fournit des éléments

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Tregouet, Patrick Thierry. "Étude d'un protocole de traitement des ascites cirrhotiques." Nantes, 1985. http://www.theses.fr/1985NANT3450.

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Yan, Fu J. "Investigating the Therapeutic Efficacy of a Novel Inhibitor GAP-107B8 on Ovarian Cancer Cells." Thèse, Université d'Ottawa / University of Ottawa, 2011. http://hdl.handle.net/10393/20189.

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Ovarian cancers often develop resistance mechanisms against the standard platinum and taxane chemotherapy, which indicates the need for novel therapeutics to improve patient outcome. In vitro assays were performed to assess the effects and mechanism of action of a novel peptide, GAP-107B8, on ovarian cancer cell viability. Xenograft models were used to determine GAP-107B8’s effects on tumour burden in immune-incompetent mice. GAP-107B8 significantly reduced cell viability in ovarian cancer cell lines, although no synergistic effects with carboplatin were observed. This reduction in cell vi

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Books on the topic "Ascites"

1

Gins, Pere, Vicente Arroyo, Juan Rods, and Robert Schrier, eds. Ascites and Renal Dysfunction in Liver Disease. Blackwell Publishing Ltd, 2005. http://dx.doi.org/10.1002/9780470987476.

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Pere, Ginès, ed. Ascites and renal dysfunction in liver diseases. 2nd ed. BMJ Books, 2005.

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3

Gerbes, A. L. Ascites, hyponatremia, and hepatorenal syndrome: Progress in treatment. Karger, 2011.

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Sungaila, Ina. Spironolactone pharmacokinetics and pharmacodynamics in cirrhotics with ascites. National Library of Canada, 1990.

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J, Groszmann Roberto, and Grace Norman D, eds. Complications of portal hypertension: Esophagogastric varices and ascites. Saunders, 1992.

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Ontario. Ministry of Agriculture and Food. Ascites in meat-type chickens caused by right heart failure. s.n, 1989.

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Seebold, Anna-Maria. Untersuchungen zur zellulären Aufnahme des Erythromycins bei Ehrlich-Ascites-Tumorzellen. [s.n.], 1986.

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Pere, Ginès, ed. Ascites and renal dysfunction in liver diseases: Pathogenesis, diagnosis, and treatment. 2nd ed. Blackwell, 2005.

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9

V, Arroyo, ed. Ascites and renal dysfunction in liver disease: Pathogenesis, diagnosis, and treatment. Blackwell Science, 1999.

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10

Khomutovskiĭ, O. A. Ėlektronnai͡a︡ gistokhimii͡a︡ ret͡s︡eptorov kletochnykh membran. Nauk. dumka, 1986.

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Book chapters on the topic "Ascites"

1

Paredes, Angelo, and Arun J. Sanyal. "Ascites and Refractory Ascites." In Complications of Cirrhosis. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-13614-1_13.

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Schiro, Adam J. "Ascites." In Liver Disorders. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-30103-7_30.

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Garcia-Tsao, Guadalupe. "Ascites." In Sherlock's Diseases of the Liver and Biliary System. John Wiley & Sons, Ltd, 2018. http://dx.doi.org/10.1002/9781119237662.ch9.

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Bataller, RamóN, Vicente Arroyo, Gilles Pomier-Layrargues, Ziad Hassoun, Dominique Franco, and Sheila Sherlock. "Ascites." In Controversies in Surgery. Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-56777-3_9.

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Joshi, Rohit, and Hooi Wen Hong. "Ascites." In The MASCC Textbook of Cancer Supportive Care and Survivorship. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-90990-5_29.

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Garcia-Tsao, Guadalupe. "Ascites." In Textbook of Clinical Gastroenterology and Hepatology. Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781118321386.ch17.

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Cárdenas, Andrés, Isabel Graupera, and Pere Ginès. "Ascites." In Cirrhosis: A practical guide to management. John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781118412640.ch14.

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Becker, Gerhild. "Ascites." In Encyclopedia of Cancer. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-27841-9_35-3.

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Garcia-Tsao, Guadalupe. "Ascites." In Sherlock's Diseases of the Liver and Biliary System. Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444341294.ch10.

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Kbalid, Sakib Karim, and Guadalupe Garcia-Tsao. "Ascites." In Portal Hypertension. Humana Press, 2005. http://dx.doi.org/10.1007/978-1-59259-885-4_19.

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Conference papers on the topic "Ascites"

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Cooper, P. C., D. R. Triger, H. Kennedy, R. G. Malia, and F. E. Preston. "FIBRINOLYSIS AND HAEMOSTASIS DURING ASCITES RECIRCULATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643061.

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The use of ascites recirculation in liver disease removes litres of incapacitating fluid and the patient is reinfused with the concentrated ascites, a rich source of albumin. Ascites is thought to contain plasminogen activator (PA) which may further affect deranged haemostasis in these patients. We have examined the effect that ascites recirculation has on levels of tPA, fibrinogen, FDP and platelet count on samples taken pre and approximately 4 hours into ascites recirculation. Using plasminogen rich fibrin plates we were able to demonstrate PA in unfractioned ascitic fluid (N=10, mean diamet

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Arora, Rahul D. "Definition, etiopathogenesis, management and role of flouroquinolone prophylaxis in prevention of spontaneous bacterial peritonitis complicating malignant ascites." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685345.

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Background: Malignancy related ascites encompasses multiple etiologies which include peritoneal carcinomatosis, hepatic synthetic dysfunction due to parenchymal involvement by the tumour, transcoeloemic metastasis and chylous ascites due to lymphatic obstruction. Primary Cancer type, liver metastasis and serum albumin have been listed as independent prognostic markers in malignant ascites. Spontaneous Bacterial Peritonitis is usually seen as a complication of decompensated chronic liver disease due to translocation of bacteria or haematogenous dissemination from a distant focus of infection. T

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Wajid, M., G. Michaeli, Z. Birner, A. Sharif, S. Khan, and O. Rahman. "Ascites in Sarcoidosis." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a3254.

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Amoun, T., M. Bogart, D. J. Markley, A. Hussain, and C. V. Shah. "Recognizing Urinary Ascites as a Rare Entity of Abdominal Ascites in Cirrhotic Patient." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5139.

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Muslim, Ihsanuddin Mohamed, and Manorama Gadde. "1591 Urinary ascites in children." In Royal College of Paediatrics and Child Health, Abstracts of the RCPCH Conference–Online, 15 June 2021–17 June 2021. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2021. http://dx.doi.org/10.1136/archdischild-2021-rcpch.736.

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Edianto, Deri, Isnayu Jurimal, and Muhammad Rizki Yaznil. "Cylous Ascites in Gynaecology Malignancy." In International Conference of Science, Technology, Engineering, Environmental and Ramification Researches. SCITEPRESS - Science and Technology Publications, 2018. http://dx.doi.org/10.5220/0010070504330435.

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Bevan, Katherine, and Claire Stark-Toller. "130 Measuring serum-ascites albumin gradient to optimize management of ascites in a hospice setting." In Accepted Oral and Poster Abstract Submissions, The Palliative Care Congress 1 Specialty: 3 Settings – home, hospice, hospital 25 – 26 March 2021 | A virtual event, hosted by Make it Edinburgh Live, the Edinburgh International Conference Centre’s hybrid event platform. British Medical Journal Publishing Group, 2021. http://dx.doi.org/10.1136/spcare-2021-pcc.148.

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Nagy, J. A., and H. F. Devorak. "FIBRINOGEN INFLUX AND FIBRIN ACCUMULATION IN ASCITES TUMORS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643668.

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Fibrin gel matrix organizes solid tumors into discrete cell nests and providesa provisional matrix for mature stroma generation. Fibrin deposits result fromlocal extravasation of plasma fibrinogen followed by extravascular coagulationand crosslinking. Unlike solid tumors, ascites tumor cells in body cavities grow in suspension, and are not envelopedin fibrin gel. The lack of fibrin gel in ascites tumors has several possible explanations: 1. Peritoneal wall blood vessels are impermeable to molecules as large as fibrinogen. 2. Fibrinogen leaks from peritoneal vessels, but is clotted to fibrin an

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Wilhelm, O., R. Hafter, and H. Graeff. "FIBRONECTIN-FIBRIN-COMPLEXES : AN IMMUNOASSAY TO EVALUATE THEIR FUNCTION IN COAGULATION DISORDERS AND TUMOR SITUATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643194.

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Fibronectin and fibronectin degradation products interact with fibrin(ogen) by non-covalent and by factor XIII catalyzed covalent binding. In certain physiological or pathophysiological situations these complexes may be of considerable importance. It was the aim of this study to accumulate more information of the function of this proteininteraction. Immunoadsorption in combination with Westernblotting with different monoclonal and polyclonal antibodies demonstrate several fibronectin-fibrin(ogen)-complexes in tumor ascites. For further information we established an immunoassay to measure these

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Walinjkar, Sagar, Anil Arora, Ashish Kumar, Praveen Sharma, and Naresh Bansal. "IDDF2020-ABS-0221 Ascitic fluid cholesterol is a useful marker to rule out malignant ascites." In Abstracts of the International Digestive Disease Forum (IDDF), 22–23 November 2020, Hong Kong. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2020. http://dx.doi.org/10.1136/gutjnl-2020-iddf.129.

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Reports on the topic "Ascites"

1

Wideman, Jr., Robert F., Nicholas B. Anthony, Avigdor Cahaner, Alan Shlosberg, Michel Bellaiche, and William B. Roush. Integrated Approach to Evaluating Inherited Predictors of Resistance to Pulmonary Hypertension Syndrome (Ascites) in Fast Growing Broiler Chickens. United States Department of Agriculture, 2000. http://dx.doi.org/10.32747/2000.7575287.bard.

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Background PHS (pulmonary hypertension syndrome, ascites syndrome) is a serious cause of loss in the broiler industry, and is a prime example of an undesirable side effect of successful genetic development that may be deleteriously manifested by factors in the environment of growing broilers. Basically, continuous and pinpointed selection for rapid growth in broilers has led to higher oxygen demand and consequently to more frequent manifestation of an inherent potential cardiopulmonary incapability to sufficiently oxygenate the arterial blood. The multifaceted causes and modifiers of PHS make

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Yang, Ming, Youwei Wu, Yiwen Qiu, and Wentao Wang. Comparision between Transjugular Intrahepatic Portosystemic Shunt and Endoscopic Therapy for Prevention of Variceal Re-bleeding: A Meta-Analysis of Randomized Controlled Trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.11.0142.

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Review question / Objective: The efficacy and safety of placement of transjugular intrahepatic portosystemic shunts (TIPS) in patients with cirrhosis and acute variceal bleeding is controversial. Eligibility criteria: (1) RCTs that compared TIPS and ET for the prevention of variceal rebleeding in patients with cirrhosis; (2) TIPS and ET (EVL or injection therapy) were after randomization; (3) patients in the included studies were aged 18 years old or older; (4) The prognosis that assessed the effects of treatments included at least one of the following outcomes: the overall mortality, the rate

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Ma, Yuan, Xiaowu Qin, Jiaoling Shi, Jun Zhang, Huiying Li, and Kewei Sun. Umbilical therapy of herbal medicine for ascites in patients with cirrhosis: A protocol for systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.8.0041.

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Leonard, Talayna, Robert Lemme, Cati Kral, et al. High-Percentage of Early Resectable Pancreatic Ductal Adenocarcinoma is Unidentified on Abdominal CT Obtained for Unrelated Diagnosis. Science Repository, 2021. http://dx.doi.org/10.31487/j.aco.2021.02.03.

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Abstract:

Objective: Pancreatic ductal adenocarcinoma (PDAC) has the best survival when detected early with 5-year survival near 40% for small, resectable PDAC. We evaluate the undiagnosed PDAC imaging features on routine CT and their impact on resectability. Methods: 76 of the screened 134 CTs from 1/1/2012 to 12/31/2018 using our tumor registry were obtained prior to PDAC diagnosis for other indications at least one month before presentation. Each cross-sectional study was reviewed for features of early PDAC: pancreatic mass, pancreatic ductal dilatation, perivascular/peripancreatic soft-tissue infilt

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