Relevant bibliographies by topics / Astrocyte neurodegeneration

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Astrocyte neurodegeneration'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Astrocyte neurodegeneration"

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Amuti, T., I. Ouko, S. Mukonjia, et al. "Role of heterogeneous astrocyte receptor expression in determining astrocytic response to neuronal disorders." Anatomy Journal of Africa 7, no. 1 (2018): 1169–74. http://dx.doi.org/10.4314/aja.v7i1.169490.

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Following neuronal disorders, astrocytes carry out either neuroprotection or neurodegeneration. Previous authors suggest that favoring of neurodegeneration or neuroprotection by astrocytes can be due to many factors such as the influence of cytokines following their binding on their receptors on astrocytes. These receptors have however been shown to be region specific and heterogeneous. Further, research exploiting their role and influence in determining astrocytic response remains partly elucidated. A review of previous and ongoing research on these receptors would be helpful in the disclosur
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Sulimai, Nurul, Jason Brown, and David Lominadze. "Fibrinogen Interaction with Astrocyte ICAM-1 and PrPC Results in the Generation of ROS and Neuronal Death." International Journal of Molecular Sciences 22, no. 5 (2021): 2391. http://dx.doi.org/10.3390/ijms22052391.

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Many neuroinflammatory diseases, like traumatic brain injury (TBI), are associated with an elevated level of fibrinogen and short-term memory (STM) impairment. We found that during TBI, extravasated fibrinogen deposited in vasculo-astrocyte interfaces, which was associated with neurodegeneration and STM reduction. The mechanisms of this fibrinogen-astrocyte interaction and its functional role in neurodegeneration are still unclear. Cultured mouse brain astrocytes were treated with fibrinogen in the presence or absence of function-blocking antibody or peptide against its astrocyte receptors int
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Miyazaki, Ikuko, and Masato Asanuma. "Neuron-Astrocyte Interactions in Parkinson’s Disease." Cells 9, no. 12 (2020): 2623. http://dx.doi.org/10.3390/cells9122623.

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Parkinson’s disease (PD) is the second most common neurodegenerative disease. PD patients exhibit motor symptoms such as akinesia/bradykinesia, tremor, rigidity, and postural instability due to a loss of nigrostriatal dopaminergic neurons. Although the pathogenesis in sporadic PD remains unknown, there is a consensus on the involvement of non-neuronal cells in the progression of PD pathology. Astrocytes are the most numerous glial cells in the central nervous system. Normally, astrocytes protect neurons by releasing neurotrophic factors, producing antioxidants, and disposing of neuronal waste
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Mitroshina, Elena, Elizaveta Kalinina, and Maria Vedunova. "Optogenetics in Alzheimer’s Disease: Focus on Astrocytes." Antioxidants 12, no. 10 (2023): 1856. http://dx.doi.org/10.3390/antiox12101856.

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Alzheimer’s disease (AD) is the most common form of dementia, resulting in disability and mortality. The global incidence of AD is consistently surging. Although numerous therapeutic agents with promising potential have been developed, none have successfully treated AD to date. Consequently, the pursuit of novel methodologies to address neurodegenerative processes in AD remains a paramount endeavor. A particularly promising avenue in this search is optogenetics, enabling the manipulation of neuronal activity. In recent years, research attention has pivoted from neurons to glial cells. This rev
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Mohn, Tal C., and Andrew O. Koob. "Adult Astrogenesis and the Etiology of Cortical Neurodegeneration." Journal of Experimental Neuroscience 9s2 (January 2015): JEN.S25520. http://dx.doi.org/10.4137/jen.s25520.

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As more evidence points to a clear role for astrocytes in synaptic processing, synaptogenesis and cognition, continuing research on astrocytic function could lead to strategies for neurodegenerative disease prevention. Reactive astrogliosis results in astrocyte proliferation early in injury and disease states and is considered neuroprotective, indicating a role for astrocytes in disease etiology. This review describes the different types of human cortical astrocytes and the current evidence regarding adult cortical astrogenesis in injury and degenerative disease. A role for disrupted astrogene
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Vicente-Acosta, Andrés, Alfredo Giménez-Cassina, Javier Díaz-Nido, and Frida Loria. "THE SONIC HEDGEHOG AGONIST SAG ATTENUATES MITOCHONDRIAL DYSFUNCTION AND DECREASES THE NEUROTOXOCITY INDUCED BY FRATAXIN-DEFICIENT ASTROCYTES." IBJ Plus 1, s5 (2022): 47. http://dx.doi.org/10.24217/2531-0151.22v1s5.00047.

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Friedreich’s ataxia (FRDA) is predominantly a neurodegenerative disease caused by the deficiency of a protein called frataxin (FXN). Although the main pathological alterations are observed in neurons, it is becoming clear that other non-neuronal cells such as astrocytes may be actively involved in the neurodegenerative process associated with the disease. Depending on the stimuli they respond to, astrocytes acquire different activation states in a process called astrogliosis. Neuroinflammatory stimuli induce the formation of A1 reactive astrocytes, which upregulate proinflammatory genes, being
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Preato, André Maciel, Ester da Silva Pinheiro, Tatiana Rosado Rosenstock, and Isaias Glezer. "The Relevance of Astrocytic Cell Culture Models for Neuroinflammation in Neurodegeneration Research." Neuroglia 5, no. 1 (2024): 27–49. http://dx.doi.org/10.3390/neuroglia5010003.

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Astrocytes are the predominant glial cells that provide essential support to neurons and promote microenvironment changes in neuropathological states. Astrocyte and astrocytic-like cell culture have substantially contributed to elucidating the molecular pathways involved in key glial roles, including those relevant to neurodevelopment, brain physiology and metabolism, which are not readily accessible with traditional approaches. The in vitro methodology has also been applied to neuroinflammatory and neurodegeneration contexts, revealing cellular changes involved in brain dysfunction. Astrocyte
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Sun, Yuanhong, Ali Winters, Linshu Wang, et al. "Metabolic Heterogeneity of Cerebral Cortical and Cerebellar Astrocytes." Life 13, no. 1 (2023): 184. http://dx.doi.org/10.3390/life13010184.

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Astrocytes play critical roles in regulating neuronal synaptogenesis, maintaining blood–brain barrier integrity, and recycling neurotransmitters. Increasing numbers of studies have suggested astrocyte heterogeneity in morphology, gene profile, and function. However, metabolic phenotype of astrocytes in different brain regions have not been explored. In this paper, we investigated the metabolic signature of cortical and cerebellar astrocytes using primary astrocyte cultures. We observed that cortical astrocytes were larger than cerebellar astrocytes, whereas cerebellar astrocytes had more and l
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Nassar, Ajmal, Triveni Kodi, Sairaj Satarker, et al. "Astrocytic MicroRNAs and Transcription Factors in Alzheimer’s Disease and Therapeutic Interventions." Cells 11, no. 24 (2022): 4111. http://dx.doi.org/10.3390/cells11244111.

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Astrocytes are important for maintaining cholesterol metabolism, glutamate uptake, and neurotransmission. Indeed, inflammatory processes and neurodegeneration contribute to the altered morphology, gene expression, and function of astrocytes. Astrocytes, in collaboration with numerous microRNAs, regulate brain cholesterol levels as well as glutamatergic and inflammatory signaling, all of which contribute to general brain homeostasis. Neural electrical activity, synaptic plasticity processes, learning, and memory are dependent on the astrocyte–neuron crosstalk. Here, we review the involvement of
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Valori, Chiara F., Agostino Possenti, Liliana Brambilla, and Daniela Rossi. "Challenges and Opportunities of Targeting Astrocytes to Halt Neurodegenerative Disorders." Cells 10, no. 8 (2021): 2019. http://dx.doi.org/10.3390/cells10082019.

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Neurodegenerative diseases are a heterogeneous group of disorders whose incidence is likely to duplicate in the next 30 years along with the progressive aging of the western population. Non-cell-specific therapeutics or therapeutics designed to tackle aberrant pathways within neurons failed to slow down or halt neurodegeneration. Yet, in the last few years, our knowledge of the importance of glial cells to maintain the central nervous system homeostasis in health conditions has increased exponentially, along with our awareness of their fundamental and multifaced role in pathological conditions
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Dissertations / Theses on the topic "Astrocyte neurodegeneration"

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MARTORANA, FRANCESCA. "Mitochondria as the core of neuroinflammation and neurodegeneration in models of neuronal and astrocytic dysfunction." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2020. http://hdl.handle.net/10281/259338.

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Le malattie neurodegenerative sono caratterizzate da disfunzione e perdita di specifiche popolazioni neuronali in risposta ad invecchiamento o eventi tossici o traumatici. Tuttavia, recentemente, è diventato chiaro che la disfunzione astrocitica ha un ruolo importante nei processi degenerativi. Infatti, gli astrociti rappresentano la popolazione cellulare maggiore del Sistema Nervoso Centrale (SNC) e costituiscono l’elemento principale per la sua omeostasi. Alterazioni biochimiche e strutturali degli astrociti durante la neuroinfiammazione rappresentano una risposta fisiologica a danni a caric
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Shanaki, Bavarsad Mahsa. "Astrocyte-targeted production of IL-10 reduces the neuroinflammatory response associated to TBI and improves neurodegeneration." Doctoral thesis, Universitat Autònoma de Barcelona, 2020. http://hdl.handle.net/10803/670855.

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La inflamació és essencial en les respostes a les infeccions i danys perifèrics. Es poden obtenir beneficis de la inflamació si es controla durant un període de temps definit. La inflamació no regulada, sostinguda o excessiva és la causa principal de diferents neuropatologies. Al sistema nerviós central (SNC), la resposta neuroinflamatòria cerebral després d’una lesió cerebral traumàtica (TBI) s’ha caracteritzat en pacients i en diferents models animals experimentals com una de les principals causes de lesions secundàries que condueixen a la degeneració neuronal. La neuroinflamació es caracter
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Efremova, Liudmila [Verfasser]. "Development of neuron-astrocyte co-culture system for mechanistic and pharmacological studies in neurodegeneration / Liudmila Efremova." Konstanz : Bibliothek der Universität Konstanz, 2015. http://d-nb.info/1081016922/34.

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Wang, Yongchao. "THE ROLE OF ENDOPLASMIC RETICULUM STRESS IN ETHANOL-INDUCED NEURODEGENERATION." UKnowledge, 2019. https://uknowledge.uky.edu/pharmacol_etds/33.

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Heavy ethanol use causes neurodegeneration manifested by neuronal loss and dysfunction. It is becoming imperative to delineate the underlying mechanism to promote the treatment of ethanol-induced neurodegeneration. Endoplasmic reticulum (ER) stress is a hallmark and an underlying mechanism of many neurodegenerative diseases. This study aims to investigate the role of ER stress in ethanol-induced neurodegeneration. In experimental design, adult mice were exposed to binge ethanol drinking by daily gavage for 1, 5, or 10 days and the response of ER stress was examined. We found the induction of E
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Di, Malta Chiara. "The analysis of a mouse model of Lysosomal Storage Disorder uncovers a role for astrocyte dysfunction in neurodegeneration." Thesis, Open University, 2012. http://oro.open.ac.uk/54503/.

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Astrocytes are the most abundant cellular population in the brain and their role in neurodegenerative processes is becoming increasingly appreciated. In my PhD project, I investigated the contribution of astrocytes to neurodegeneration in Multiple Sulfatase Deficiency (MS D), a severe Lysosomal Storage Disorder (LSD) caused by mutations in the Sulfatase Modifying Factor 1 (SUMF1) gene. Using Cre/Lox mouse models, I found that astrocyte-specific deletion of Sumf1 in vivo induced severe lysosomal storage and autophagy dysfunction with consequential cytoplasmic accumulation of toxic substrates. L
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CARUCCI, NICOLA MARIA. "Upon the way through which astrocytes sense and react to a reduction of extracellular Nerve Growth Factor level : NGF and Astrocytes in neurodegenerative disease models." Doctoral thesis, Scuola Normale Superiore, 2021. http://hdl.handle.net/11384/104404.

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Lhuillier, Alice. "Identification de programmes d'activation macrophagique et microgliale dans les formes progressives de la sclérose en plaques." Phd thesis, Université Claude Bernard - Lyon I, 2014. http://tel.archives-ouvertes.fr/tel-01056829.

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La sclérose en plaques (SEP) est une maladie neuro-inflammatoire chronique, première cause de handicap chez le jeune adulte. Actuellement, aucun traitement ne freine l'aggravation des symptômes liée aux formes progressives. Bien que connue, l'implication des macrophages et de la microglie dans la démyélinisation et l'atteinte axonale doit être plus finement caractérisée. Ce d'autant plus que la plasticité fonctionnelle de ces cellules suggère une réponse spécifique selon la pathologie, la localisation des lésions et le stade évolutif de la maladie. Ce travail de thèse a consisté en une caracté
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Stone, Rebecca. "Glutathione release from astrocytes; characterization and implications for neurodegeneration." Thesis, University College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.395567.

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Maezawa, Izumi. "Glia-regulated, apolipoprotein E specific mechanisms of neuroprotection and neurodegeneration /." Thesis, Connect to this title online; UW restricted, 2005. http://hdl.handle.net/1773/6340.

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Jay, Taylor Reagan. "The TREM2 Receptor Directs Microglial Activity in Neurodegeneration and Neurodevelopment." Case Western Reserve University School of Graduate Studies / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1560181547156823.

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Books on the topic "Astrocyte neurodegeneration"

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1954-, Schipper Hyman M., ed. Astrocytes in brain aging and neurodegeneration. R.G. Landes, 1998.

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Schipper, Hyman M. Astrocytes in Brain Aging and Neurodegeneration (Neuroscience Intelligence Unit Series). International Thomson Publishing Services, 1999.

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Benarroch, Eduardo E. Neuroscience for Clinicians. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190948894.001.0001.

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The aim of this book is to provide the clinician with a comprehensive and clinical relevant survey of emerging concepts on the organization and function of the nervous system and neurologic disease mechanisms, at the molecular, cellular, and system levels. The content of is based on the review of information obtained from recent advances in genetic, molecular, and cell biology techniques; electrophysiological recordings; brain mapping; and mouse models, emphasizing the clinical and possible therapeutic implications. Many chapters of this book contain information that will be relevant not only
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Book chapters on the topic "Astrocyte neurodegeneration"

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Brancaccio, Marco, Anne C. Wolfes, and Natalie Ness. "Astrocyte Circadian Timekeeping in Brain Health and Neurodegeneration." In Circadian Clock in Brain Health and Disease. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-81147-1_6.

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Kojima, K., H. Wekerle, H. Lassmann, T. Berger, and Ch Linington. "Induction of experimental autoimmune encephalomyelitis by CD4+ T cells specific for an astrocyte protein, Sl00ß." In Advances in Research on Neurodegeneration. Springer Vienna, 1997. http://dx.doi.org/10.1007/978-3-7091-6844-8_5.

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Gray, Michelle. "Astrocytes in Huntington’s Disease." In Neuroglia in Neurodegenerative Diseases. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-9913-8_14.

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Valori, Chiara F., Giulia Guidotti, Liliana Brambilla, and Daniela Rossi. "Astrocytes in Motor Neuron Diseases." In Neuroglia in Neurodegenerative Diseases. Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-9913-8_10.

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Meares, Gordon P., and Etty N. Benveniste. "Inflammation and the Pathophysiology of Astrocytes in Neurodegenerative Diseases." In Neuroinflammation and Neurodegeneration. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1071-7_4.

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Koob, Andrew O., and Paola Sacchetti. "Astrocytes and the Synucleinopathies." In Pathology, Prevention and Therapeutics of Neurodegenerative Disease. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-13-0944-1_8.

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Roginski, Raymond S. "A Model for the Expression of Different Glutamate Transporter Proteins from a Rat Astrocyte-Type Glutamate Transporter Gene." In Neurodegenerative Diseases. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-0209-2_4.

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Sue, W., T. Griffin, and Laura C. Stanley. "Glial Activation as a Common Denominator in Neurodegenerative Disease: A Hypothesis in Neuropathophysiology." In Biology and Pathology of Astrocyte-Neuron Interactions. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4757-9486-1_32.

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Roewe, Julian, Yasmin Riesinger, Ruven Wilkens, et al. "Co-cultures of Human-Induced Pluripotent Stem Cell-Derived Neurons, Astrocytes, and Microglia for Modeling Neurodegenerative Diseases." In Methods in Molecular Biology. Springer US, 2025. https://doi.org/10.1007/978-1-0716-4530-7_6.

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Prà, Ilaria Dal, Ubaldo Armato, and Anna Chiarini. "Astrocytes’ Role in Alzheimer’s Disease Neurodegeneration." In Astrocyte - Physiology and Pathology. InTech, 2018. http://dx.doi.org/10.5772/intechopen.72974.

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Conference papers on the topic "Astrocyte neurodegeneration"

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Ravagnani, Felipe, Hellen Valerio, Jersey Maués, et al. "Omics profile of iPSC-derived astrocytes from Progressive Supranuclear Palsy (PSP) patients." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.414.

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Progressive Supranuclear Palsy (PSP) is a neurodegenerative tauopathy and, to date, the pathophysiological mechanisms in PSP that lead to Tau hyperphosphorylation and neurodegeneration are not clear. The development of a model using neural cell lines derived from patients has the potential to identify molecules and possible biomarkers. We developed a model of induced pluripotent stem cells iPSC-derived astrocytes to investigate the pathophysiology of PSP, particularly early events that might contribute to Tau hyperphosphorylation, applying an omics approach to detect differentially expressed g
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Chaplygina, A. V., V. I. Kovalev, D. Y. Zhdanova, and N. V. Bobkova. "CHEMICAL CONVERSION OF PRIMARY NEURONAL CULTURES." In NOVEL TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. Institute of information technology, 2022. http://dx.doi.org/10.47501/978-5-6044060-2-1.389-393.

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The paper discusses the use of direct chemical transformation of glial cells into neurons to solve the problems of neurodegenerative diseases. Original experimental data on the success-ful use of a conversion cocktail for astrocyte-neuronal conversion in a primary cell culture of the 5xFAD mouse hippocampus are presented.
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Chen, Yuanxin, James Mancuso, Zhen Zhao, Xuping Li, Zhong Xue, and Stephen T. C. Wong. "In vivo optical activation of astrocytes as a potential therapeutic strategy for neurodegenerative diseases." In SPIE BiOS, edited by Nikiforos Kollias, Bernard Choi, Haishan Zeng, et al. SPIE, 2013. http://dx.doi.org/10.1117/12.2004712.

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Okhalnikov, A. D., A. O. Motorina, M. S. Gavrish, and A. A. Babaev. "EVALUATION OF CHANGES IN THE MITOCHONDRIAL ARCHITECTURE IN ASTROCYTES IN AN IN VITRO MODEL OF ALZHEIMER’S DISEASE." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-354.

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Alzheimer’s disease is a neurodegenerative disease whose pathogenesis is inextricably linked with a long-term disruption of metabolic processes in neuronal and glial cells. According to the literature data, mitochondrial dysfunction contributes to the activation of various signaling mechanisms leading to reprogramming of cell metabolism, which contributes to the development of the disease.
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Patel, Minjal, and Nimish Acharya. "The Neuroprotective Role of Lipoxin A4 in Reinstating Blood Brain Barrier Integrity in Neuroinflammatory Disease Processes." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. https://doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.158_2024.

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Background: The blood-brain barrier (BBB), formed by the vascular endothelium, astrocytic foot processes, pericytes, is a highly selective barrier that is responsible for maintaining brain homeostasis and ultimately proper neuronal function. Disruption of the BBB, leading to increased BBB permeability, has been reported in several neurodegenerative diseases, including Alzheimer’s disease (AD) and traumatic brain injury (TBI).1 Loss of BBB integrity leads to the proliferation of pro-inflammatory cytokines, including TNFɑ, IL-1β, and IL-6.2 Moderate inflammation has a beneficial response in the
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Reports on the topic "Astrocyte neurodegeneration"

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Rumbaut Gil, Ana Laura. A Systemic Analysis of Vascular Dysfunction in Parkinson’s Disease: Reviewing the Roles of Astrocytes and COX-2. Florida International University, 2025. https://doi.org/10.25148/fiuurj.3.1.6.

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Although Parkinson’s Disease (PD) is the second most prevalent neurodegenerative disease in the U.S., the pathology remains an enigma. Many neuroinflammation hypotheses have been studied to explain its development. It’s shown that inflammatory markers such as COX-2 activity, which synthesizes the potent vasodilator PGE2, are overexpressed in PD. Additionally, astrocytes, regulators of inflammation in the brain and BBB, undergo important changes during PD. However, the vascular consequences that all this has for PD-led neurodegeneration are relatively unexplored. We conducted a systemic review
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