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Relevant bibliographies by topics / Atheromatous plaque

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Academic literature on the topic 'Atheromatous plaque'

Author: Grafiati

Published: 4 June 2021

Last updated: 2 February 2022

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Journal articles on the topic "Atheromatous plaque"

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Hayashi, Junichi, Takashi Saito, and Katsuo Aizawa. "Photodynamic Diagnosis and Treatment for Atherosclerosis by an Endoscopic Approach." Diagnostic and Therapeutic Endoscopy 5, no. 3 (January 1, 1999): 191–95. http://dx.doi.org/10.1155/dte.5.191.

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The photosensitizer, mono-L-aspartyl chlorin e6 (NPe6), specifically accumulates in the atheromatous plaque. We detected the fluorescence spectra of NPe6 emitted from atheromatous plaques on the descending thoracic aorta by an angioscopic approach using the animal model of atherosclerosis. We also showed that a fluorescence spectrum peak at 675 nm was obtained laparoscopically only in parts of the abdominal aorta with an atheromatous plaque. By a fluorescence endoscope, atheromatous plaques on the carotid artery were recognized as reddish spots from outside the artery. In addition, we visualized specifically at the beating heart surface small coronary atherosclerosis using an epifluorescence stereoscope system.We examined the effects of photodynamic treatment with NPe6 on the atheromatous plaque. The change in the elastic framework in the atheromatous plaque after photodynamic treatment was evaluated using scanning electron microscopy. The destruction of the architecture of the elastic fiber network in the atheromatous plaque was revealed. We also studied the change in the lipid components of the atheromatous plaque using Fourier transform infrared (FTIR) microspectroscopy. FTIR microspectroscopic analysis showed a dissociation of ester bonds of cholesterol esters in the atheromatous plaque after photodynamic treatment. The framework of the atheromatous plaque and the lipids accumulated in the plaque could be destroyed following such treatment.

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Rath, SarojK, Manish Mukherjee, R. Kaushik, Sourav Sen, and Mukesh Kumar. "Periodontal pathogens in atheromatous plaque." Indian Journal of Pathology and Microbiology 57, no. 2 (2014): 259. http://dx.doi.org/10.4103/0377-4929.134704.

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UENO, Yuji, and Takao URABE. "Diagnosis for atheromatous aortic plaque." Neurosonology 27, no. 1 (2014): 1–2. http://dx.doi.org/10.2301/neurosonology.27.1.

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Gong, Hai-Ying, Xiao-Ke Shi, Heng-Quan Zhu, Xian-Zhong Chen, Jiang Zhu, and Bo-Wen Zhao. "Evaluation of carotid atherosclerosis and related risk factors using ultrasonic B-Flow technology in elderly patients." Journal of International Medical Research 48, no. 10 (October 2020): 030006052096122. http://dx.doi.org/10.1177/0300060520961224.

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Objective This study was performed to identify the risk factors for carotid atherosclerotic plaque formation using B-Flow ultrasound. Methods In total, 120 patients who underwent bilateral carotid ultrasound examination were enrolled in this cross-sectional study. The intima–media thickness was measured, and the risk factors for carotid atheromatous plaque formation were investigated. Results Age, sex, medical history of hypertension, coronary heart disease, and diabetes were risk factors for carotid atheromatous plaque formation. Multivariate logistic regression analysis revealed that the main risk factors for carotid atheromatous plaque formation were male sex, advanced age, a high hemoglobin concentration, a high red cell distribution width, and a high low-density lipoprotein cholesterol concentration. Conclusion The risk factors for carotid atheromatous plaque formation were basically the same as those for stroke. Early ultrasound examination of the carotid artery enables the identification of risk factors associated with stroke.

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Orzan, Marius, Roxana Hodas, Mihaela Dobra, Nora Rat, Monica Chitu, and Imre Benedek. "Original Research. Transluminal Contrast Attenuation Gradient Is Associated with Coronary Plaque Vulnerability — a Computed Tomography Angiography-based Study." Journal Of Cardiovascular Emergencies 3, no. 3 (September 26, 2017): 121–27. http://dx.doi.org/10.1515/jce-2017-0016.

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Abstract The aim of this study was to demonstrate that the transluminal contrast attenuation gradient (TAG), a new CT imaging-derived marker of functional significance of a coronary stenosis, is directly associated with the vulnerability degree of atheromatous coronary plaques. Material and methods: This is a prospective study on 21 patients with 30 atheromatous plaques in the coronary arteries, who underwent cardiac computed tomography angiography (CCTA) for assessment of coronary plaques. Results: Twelve plaques were classified as vulnerable (40%) and 18 plaques (60%) as non-vulnerable. Plaques associated with a TAG value above 10 HU exhibited in a significantly higher proportion CCTA markers of plaque vulnerability, as compared to plaques in which the attenuation gradient was below 10 HU. TAG values >10 HU were associated with a higher amount of plaque volume (107.4 ± 91.2 mm3 vs. 56.0 ± 37.5 mm3, p = 0.009), necrotic core (32.5 ± 36.9 mm3 vs. 3.1 ± 3.2 mm3, p = 0.0003), and fibro-fatty tissue (17.7 ± 16.3 mm3 vs. 4.0 ± 2.6 mm3, p = 0.0002), as compared to those lesions with TAG values below 10 HU. Linear regression analysis revealed a significant correlation between TAG values and CCTA features of plaque instability: necrotic core (r = −0.73, p <0.0001), fibrofatty tissue (r = −0.63, p = 0.0002), and plaque volume (r = −0.48, p = 0.006). Conclusions: In patients with coronary artery disease, contrast attenuation gradient along the coronary plaques, determined by CCTA, correlates with CT markers of plaque vulnerability. Vulnerable coronary plaques are associated with a higher functional significance than the stable ones with a similar anatomic profile.

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Lamberti, Giuseppe, Francesco Gelsomino, Stefano Brocchi, Antonio Poerio, Barbara Melotti, Francesca Sperandi, Mauro Gargiulo, Claudio Borghi, Michelangelo Fiorentino, and Andrea Ardizzoni. "New disappearance of complicated atheromatous plaques on rechallenge with PD-1/PD-L1 axis blockade in non-small cell lung cancer patient: follow up of an unexpected event." Therapeutic Advances in Medical Oncology 12 (January 2020): 175883592091380. http://dx.doi.org/10.1177/1758835920913801.

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Atherosclerosis is considered an irreversible process, with crucial contribution of inflammation and immune cells. Impact of cancer immunotherapy on a partly immune-driven disease, such as atherosclerosis, is poorly understood, but preclinical models suggest its worsening on programmed death/ligand-1 (PD-1/PD-L1) inhibitors. In a previously reported cohort of 11 patients with non-small cell lung cancer (NSCLC) treated with nivolumab and pre-existing complicated atheromatous plaques, 3 patients had a dramatic radiologic reduction of aortic plaques while on nivolumab; of these 3, 2 died receiving no further treatment. The remaining patient was an 83-year-old woman with history of arterial hypertension and hypothyroidism who was diagnosed with locally advanced squamous NSCLC. At relapse, complicated aortic atheromatous plaques were demonstrated on scans. The patient was then treated with nivolumab obtaining stable disease at radiological assessment, which also demonstrated almost complete vanishing of aortic plaques. After relapse and interval treatment with chemotherapy, she experienced new development of aortic atheromatous plaques. At further relapse she received atezolizumab, which yielded disease response and new reduction in aortic plaques, until nearly complete resolution. The observation of a repeated improvement of atheromatous plaques on treatment with PD-1/PD-L1 inhibitors favors the protective role of T cells on atheromatous plaques that is impaired by PD-L1 expression by plaque-associated macrophages. Validation by independent and prospective observation is needed.

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Mureebe, Leila, and James F. McKinsey. "Infrainguinal Arterial Intervention: Is There a Role for an Atherectomy Device?" Vascular 14, no. 5 (September 2006): 313–18. http://dx.doi.org/10.2310/6670.2006.00053.

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Excision of atheromatous plaque is an attractive option for the minimally invasive treatment of peripheral arterial disease. Approved for use in 2003, the SilverHawk Plaque Exicison System (FoxHollow Technologies, Redwood City, CA) is a catheter-based plaque excision device allowing percutaneous removal of atheromatous material. This device represents the most recent generation of atherectomy tools. Overall experience with plaque debulking in the peripheral arteries spans almost two decades, and understanding of the technique continues to evolve. This article reviews the technology, current practices, and data on plaque excision.

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Uno, J., M. Kawashima, M. Miyazono, T. Nakamura, and H. Gi. "Intravascular Ultrasound with Percutaneous Transluminal Angioplasty for Supra-Aortic Arteries." Interventional Neuroradiology 4, no. 1_suppl (November 1998): 27–30. http://dx.doi.org/10.1177/15910199980040s103.

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One of the major complications during or after percutaneous transluminal angioplasty (PTA) is distal embolism. We performed intravascular ultrasound (IVUS) to assess the morphological characteristics of atheromatous plaques which caused stenosis of arteries. In 7 cases of ICA stenosis, IVUS demonstrated hyperechoic plaques which were considered to be fibrous. Mixed type of plaque was observed in one case of ICA stenosis and another one case of ICA stenosis had plaque which was hyperechoic with acoustic shadowing. In all cases of SCA stenosis, plaques were hypoechoic, indicating fatty plaque. Distal embolism occurred after PTA in the case of ICA stenosis which had a mixed type of plaque. It is important to evaluate plaque morphology to prevent distal embolism. PTA is considered to he contraindicated in cases of ICA stenosis having hypoechoic plaques or ulceration.

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Novikova, Olga A., Pavel P. Laktionov, and Andrey A. Karpenko. "The roles of mechanotransduction, vascular wall cells, and blood cells in atheroma induction." Vascular 27, no. 1 (August 29, 2018): 98–109. http://dx.doi.org/10.1177/1708538118796063.

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Background This paper describes and analyzes the cellular and molecular mechanisms underlying atherosclerosis development. In particular, the roles of monocytes/macrophages, smooth muscle cells, and vascular endothelium in the formation of stable/unstable atheromatous plaques, and the contributions of some processes to atheroma formation. Methods and results In this study we analyzed endothelium: function, dysfunction, and involvement into atherogenesis; cell proteins mediating mechanotransduction; proatherogenic role of monocytes; the role of macrophages in the development of unstable atheromatous plaques and smooth muscle cell origin in atherosclerosis. Smooth muscle cell phenotypic switching; their functioning; the ability to retain cholesterol and lipoproteins as well as secretion of pro- and anti-inflammatory molecules and extracellular matrix proteins, their response to extracellular stimuli secreted by other cells, and the effect of smooth muscle cells on the cells surrounding atheromatous plaques are fundamentally important for the insight into atherosclerosis molecular basis. Conclusion Atheromatous plaque transcriptome studies will be helpful in the identification of the key genes involved in atheroma transformation and development as well as discovery of the new targets for diagnosis and therapy.

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Sargolzaie, Naser, Nava Naghibi, Amin Khajavi, Amir Moeintaghavi, Mohammad Abbasi Tashnizi, Kiarash Ghazvini, and Farid Shiezadeh. "Quantitative Detection of Periodontopathogenic Bacteria in Atherosclerotic Plaques from Coronary Arteries by Real-Time PCR." Open Dentistry Journal 14, no. 1 (December 31, 2020): 724–30. http://dx.doi.org/10.2174/1874210602014010724.

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Objectives: Epidemiologic studies have suggested periodontitis as a risk factor for Coronary Artery Diseas (CAD). Detection of periopathogens in atheromatous plaque provides some evidence for the causal relationship between these two conditions. The aim of this study was to determine the presence and quantity of periopathogens in coronary atherosclerotic plaques in patients undergoing Coronary Artery Bypass Graft (CABG) surgery. Methods: 20 patients who were candidates for endarterectomy were enrolled in this study for the periodontal examination. Subgingival and coronary atherosclerotic plaque samples were then collected. Thereafter, quantitative detection of Aggregatibacter actinomycetemcomitans (A.a), Porphyromonas gingivali (P.g), and all bacteria detected by Real-Time PCR (RT-PCR) were measured. The correlation analysis was also used to evaluate the relationship between quantities of periopathogens in atherosclerotic and subgingival plaque samples. Results: A.a was detected in 13 patients (65%) with subgingival plaques and 4 patients (20%) with atherosclerotic plaques. In addition, P.g was found in 15 patients (75%) with subgingival and 10 patients (50%) with atherosclerotic plaques. A.a represented means of 2.7% and 10.04% of detected bacteria in both atherosclerotic and subgingival plaque samples, respectively. The mean of quantity of P.g was 10.85% and 12.87% of the detected bacteria obtained from atherosclerotic and subginigival samples, respectively. Correlation analysis showed a significant correlation between the quantities of A.a in the atherosclerotic and subgingival plaques, but such a significant relationship was not found for P.g. Conclusion: This study confirmed the detection of A.a and P.g in atheromatous plaque. The quantitative data suggested that periopathogens comprise a significant proportion of atherosclerotic plaque microbiome, which may consequently contribute to the development of CAD.

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Dissertations / Theses on the topic "Atheromatous plaque"

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Russell, David Alexander. "The histological and ultrasound characteristics of the unstable carotid atheromatous plaque." Thesis, University of Leeds, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.485774.

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Garry, Jennifer Mary Christina. "The effect of dietary n-3 and n-6 PUFA intake on atheromatous plaque lipid composition." Thesis, University of Southampton, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.367970.

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Didier, Romain. "Athérosclérose : approche translationnelle de la détection, caractérisation, et du traitement de la plaque athéromateuse." Thesis, Brest, 2018. http://www.theses.fr/2018BRES0030.

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L’athérosclérose est une pathologie artérielle chronique évoluant sur plusieurs années ou dizaines d’années. Sa traduction clinique est variable, pouvant rester longtemps asymptomatique, ou induire des symptômes d’effort directement lié à la réduction du calibre de l’artère (angor, claudication des membres inférieurs). Malgré cette évolution en apparence lente et progressive, les manifestations cliniques aiguës de cette pathologie sont souvent de survenue brutale. La formation de thrombus en regard des lésions athéromateuses dites déstabilisées représente la composante physiopathologique la plus fréquemment rencontrée. Cette complication aigüe de la plaque athéromateuse est responsable d’événements cardio-vasculaires majeurs impactant la morbi-mortalité, représentés par les syndromes coronariens aigus, les accidents vasculaires cérébraux ischémiques, et les ischémies aigues de membre inférieur. Après une revue des connaissances actuelles sur la plaque athéromateuse, nous étudierons à l’aide d’un modèle animal d’athérosclérose, son évolution, ses caractéristiques morphologiques, et nous testerons l’impact d’un traitement au long cours par statine en prévention primaire sur les plaques athéromateuses et sur les composants de la paroi vasculaire. Puis, dans une approche clinique, nous nous intéresserons à la durée optimale du traitement par double antiagrégant plaquettaire instauré après un traitement par angioplastie percutanée de plaque athéromateuse. Dans un second temps, nous détaillerons l’évolution des pratiques d’angioplastie coronaire en analysant les facteurs ayant contribué à limiter l’utilisation des stents non actifs (dit nus) au cours des 10 dernières années. Enfin, nous étudierons les principaux facteurs restant associés à la survenue d’un accident vasculaire cérébral post angioplastie percutanée, correspondant à l’une des principales complications majeures de ces procédures
Atherosclerosis is a chronic arterial disease that progresses over several years or decades. Its clinical translation is variable, and may remain asymptomatic for a long time, or induce symptoms of stress directly related to the reduction in size of the artery (angina, chronic limb ischemia). Despite this seemingly slow and progressive evolution, the acute clinical manifestations of this pathology often occur suddenly. Thrombus formation in regard to the “destabilized” atheromatous lesions is the most frequently physiopathological components. This acute complication of atheromatous plaque is responsible of major cardiovascular events impacting the morbi-mortality, mostly represented by acute coronary syndromes, ischemic strokes, and acute lower limb ischemia. After a review of current knowledge on atheromatous plaque, we will study using an animal model of atherosclerosis, its evolution, its morphological characteristics, and we will test the impact of a long-term statin treatment in primary prevention on atheromatous plaques and on vascular wall components. Then, in a clinical approach, we will look at the optimal duration of double platelet antiaggregant after angioplasty. In a second step, we will detail the evolution of coronary angioplasty practices by analyzing the main factors that have contributed to limiting the use of bare metal stent over the past 10 years. Finally, we will analyze the major factors still associated with the occurrence of a stroke after percutaneous angioplasty

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Belzacq, Tristan. "Modélisation numérique et expérimentale des interactions fluide structure en conduite sténosée : contribution à l'étude de la vulnérabilité de la plaque d'athérome carotidienne." Phd thesis, Ecole Nationale Supérieure des Mines de Saint-Etienne, 2012. http://tel.archives-ouvertes.fr/tel-00784003.

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La rupture de la plaque d'athérome carotidienne est la première cause des infarctus cérébraux. Pour prévenir ces accidents, l'endartérectomie carotidienne est le traitement le plus utilisé. La vulnérabilité de la plaque est en relation avec les efforts que le sang applique sur la plaque. Ces actions sont différentes suivant les propriétés constitutives, mécaniques et géométriques de la plaque. Plusieurs auteurs ont développé des modèles numériques de la plaque d'athérome carotidienne à partir desquels une analyse mécanique a permis de caractériser les déformations et les contraintes en lien avec la rupture de la plaque. Néanmoins, les caractéristiques d'une plaque vulnérable sont encore mal connues. Dans ce manuscrit, un modèle numérique de plaque d'athérome carotidienne est développé en interaction fluide-structure dans le but mieux comprendre comment les actions mécaniques du sang sur la plaque sont affectées par les propriétés mécaniques et géométriques de la plaque. Plusieurs résultats sont en concordance avec la littérature : la vulnérabilité de la plaque est associée à la sévérité de sténose et à l'épaisseur de la chape fibreuse. De plus une analyse de l'écoulement du sang, de la déformation de la plaque et des contraintes dans la plaque révèle que les effets de l'écoulement du sang sont amplifiés si la plaque est courte, si la pente en amont de sténose est raide ou si la morphologie de la plaque est irrégulière et asymétrique. Ces résultats offrent de nouvelles perspectives dans la compréhension de la vulnérabilité de la plaque.

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Books on the topic "Atheromatous plaque"

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Jardine, Alan G., and Rajan K. Patel. Lipid disorders of patients with chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0102.

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The risk of developing cardiovascular (CV) disease is increased in patients with chronic kidney disease (CKD) and although dyslipidaemia is a major contributory factor to the development of premature CV disease, the relationship is complex. Changes in lipid fractions are related to glomerular filtration rate and the presence and severity of proteinuria, diabetes, and other confounding factors. The spectrum of CV disease changes from lipid-dependent, atheromatous coronary disease in early CKD to lipid-independent, non-coronary disease, manifesting as heart failure, and sudden cardiac death in advanced and end-stage renal disease. Statin-based lipid-lowering therapy is proven to reduce coronary events across the spectrum of CKD. The relative reduction in overall CV events, however, diminishes as CKD progresses and the proportion of lipid-dependent coronary events declines. There is nevertheless a strong argument for the use of statin-based therapy across the spectrum of CKD. The argument is particularly strong for those patients with progressive renal disease who will eventually require transplantation, in whom preventive therapy should start as early as possible. The SHARP study established the benefits and endorses the use of lipid-lowering therapy in CKD 3-4 but uncertainty about the value of initiation of statin therapy in CKD 5 remains. There is, however, no rationale for stopping agents started earlier in the course of the illness for compelling indications, particularly in those who will ultimately be transplanted. The place of high-density lipoprotein-cholesterol raising and triglyceride lowering therapy needs to be assessed in trials. Modifying dyslipidaemia in CKD has demonstrated that lipid-dependent atheromatous cardiovascular disease is only one component of the burden of CV disease in CKD patients, that this is proportionately less in advanced CKD, and that modification of lipid profiles is only one part of CV risk management.

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Book chapters on the topic "Atheromatous plaque"

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Dartsch, P. C., G. Bauriedel, B. Höfling, and E. Betz. "Cell culture of human atheromatous plaque material." In Interventional Cardiology and Angiology, 115–25. Heidelberg: Steinkopff, 1989. http://dx.doi.org/10.1007/978-3-662-12114-6_15.

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Campbell, G. R., and J. H. Campbell. "Microscopic and Ultramicroscopic Methods of Identifying and Quantitating Cells in Diffuse Fibrous Arterial Thickenings and Atheromatous Plaque." In Atherosclerotic Plaques, 213–28. New York, NY: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4757-0438-9_23.

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Iwasaki, Masaru, Kihachirou Kamiya, and Akira Ueno. "Fundamental Research in Laser Angioplasty (1): Effects of Nd:YAG, Argon-Ion and Excimer Lasers on Human Aortic Wall with or Without Atheromatous Plaque." In LASER Optoelectronics in Medicine, 451–54. Berlin, Heidelberg: Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-72870-9_119.

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Bihari-Varga, M. "Humoral factors of direct influence on the formation and regression of atheromatous plaques." In Atherosclerosis and Cardiovascular Disease, 417–23. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-0731-7_54.

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Heinle, H., D. Kling, and E. Betz. "Metabolism of Fibromuscular and Atheromatous Plaques in an Experimental Model: Causal Mechanisms for the Development of Intimal Necrosis." In Current Topics in Pathology, 193–221. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-76849-1_7.

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Kashihara, M., S. Ueda, and K. Matsumoto. "Effects of Blood Flow on Development of Atheromatous Plaques at an Experimental Inter-Carotid Anastomosis (Model Carotid Bifurcation)." In Role of Blood Flow in Atherogenesis, 205–8. Tokyo: Springer Japan, 1988. http://dx.doi.org/10.1007/978-4-431-68399-5_31.

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"Atheromatous Plaque." In Encyclopedia of Clinical Neuropsychology, 278. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_5541.

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Ryan, Nicola, Nieves Gonzalo, and Javier Escaned. "Intracoronary imaging." In ESC CardioMed, edited by William Wijns, 1360–71. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0334.

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Intracoronary imaging allows detailed in vivo assessment of atheromatous plaques in patients who present with stable coronary artery disease. Although atheromatous plaques have been classified as stable and unstable or vulnerable, based on their histological and imaging features, plaque type and clinical presentation are not mutually exclusive. Longitudinal studies using intracoronary imaging have allowed assessment of the prognostic relevance of atheromatous plaques and their modification by various therapeutic strategies.

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Cilla, Myriam, Estefanía Peña, and Miguel A. Martínez. "Atheromatous plaque initiation and growth: a multiphysical process explored by an in silico mass transport model." In Biomechanics of Coronary Atherosclerotic Plaque, 335–60. Elsevier, 2021. http://dx.doi.org/10.1016/b978-0-12-817195-0.00015-9.

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Fox, Keith A. A., and Rajesh K. Kharbanda. "Management of acute coronary syndrome." In Oxford Textbook of Medicine, 2911–35. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199204854.003.161305_update_003.

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The acute coronary syndrome (ACS) is precipitated by an abrupt change in an atheromatous plaque, resulting in increased obstruction to perfusion and ischaemia or infarction in the territory supplied by the affected vessel. The clinical consequences of plaque rupture can range from an entirely silent episode, through to unstable symptoms of ischaemia without infarction, to profound ischaemia complicated by progressive infarction, heart failure, and risk of sudden death. Clinical presentation with an ACS identifies a patient at high risk of further cardiovascular events requiring a defined acute and long-term management strategy....

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Conference papers on the topic "Atheromatous plaque"

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Woodburn, Kathryn W., Stuart W. Young, Q. Fan, David Kessel, and Richard A. Miller. "Selective uptake of texaphyrins in atheromatous plaque." In Photonics West '96, edited by R. Rox Anderson. SPIE, 1996. http://dx.doi.org/10.1117/12.239994.

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Woodburn, Kathryn W., Fan Qing, David Kessel, and Stuart W. Young. "Photoeradication and imaging of atheromatous plaque with texaphyrins." In BiOS '97, Part of Photonics West. SPIE, 1997. http://dx.doi.org/10.1117/12.275065.

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Gmitro, Arthur F., Andrew L. Alexander, Carolyn M. Davenport, and G. H. Manriquez. "Optimum illumination wavelength for fluorescence spectroscopy of atheromatous plaque." In OE/LASE '90, 14-19 Jan., Los Angeles, CA, edited by Abraham Katzir. SPIE, 1990. http://dx.doi.org/10.1117/12.17583.

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Chan, Kwok-Leung. "Movement analysis of atheromatous carotid plaque in ultrasonic image sequence." In Medical Imaging 1996, edited by Eric A. Hoffman. SPIE, 1996. http://dx.doi.org/10.1117/12.237874.

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Selvathi, D., and N. Emimal. "Statistical modeling for the characterization of atheromatous plaque in Intravascular Ultrasound images." In 2012 International Conference on Devices, Circuits and Systems (ICDCS 2012). IEEE, 2012. http://dx.doi.org/10.1109/icdcsyst.2012.6188729.

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Gijsen, Frank, Anna Ten Have, Jolanda Wentzel, and Antonius Van Der Steen. "Temperature Measurement of Advanced Murine Atherosclerotic Plaques." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176307.

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Ischaemic heart disease is most frequently caused by coronary atherosclerosis, of which the vulnerable plaque is one of the developmental stages. Rupture of a vulnerable plaque with superimposed thrombosis frequently leads to acute coronary syndromes. The major components of a vulnerable plaque are a lipid-rich, atheromatous core, and a thin fibrous cap with macrophage and macrophage infiltration (Schaar et al., 2004). After the first paper suggesting the possibility of thermographic detection of vulnerable plaques (Casscells et al., 1996), intracoronary thermography as a vulnerable plaque detection technique has been investigated. Increased metabolic activity of macrophages is suggested as the main reasons for the increased temperatures (ten Have et al., 2005).

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Golemati, S., S. Lehareas, N. N. Tsiaparas, A. Gastounioti, A. Chatziioannou, K. S. Nikita, and D. N. Perrea. "Toward recognizing the vulnerable asymptomatic atheromatous plaque from B-mode ultrasound: the importance of the morphology of the plaque shoulder." In 2014 IEEE International Ultrasonics Symposium (IUS). IEEE, 2014. http://dx.doi.org/10.1109/ultsym.2014.0596.

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Schwarzmaier, Hans-Joachim, Matthias P. Heintzen, Mathias Zumdick, Raimund Kaufmann, and Myron L. Wolbarsht. "Changes in optical density of normal vessel wall and lipid atheromatous plaque after Nd:YAG laser irradiation." In Optics, Electro-Optics, and Laser Applications in Science and Engineering, edited by Steven L. Jacques. SPIE, 1991. http://dx.doi.org/10.1117/12.44096.

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Schwarzmaier, Hans-Joachim, Thomas Hennig, Peter Betz, Raimund Kaufmann, and Myron L. Wolbarsht. "Optical density of healthy human arterial vessel wall and atheromatous plaque as a basis for pulsed laser angioplasty." In OE/LASE '90, 14-19 Jan., Los Angeles, CA, edited by Abraham Katzir. SPIE, 1990. http://dx.doi.org/10.1117/12.17507.

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Ballard, John R., Andrew J. Casper, Dalong Liu, Alyona Haritonova, Islam A. Shehata, Mitchell Troutman, and Emad S. Ebbini. "Dual-mode ultrasound arrays for image-guided targeting of atheromatous plaques." In 12TH INTERNATIONAL SYMPOSIUM ON THERAPEUTIC ULTRASOUND. AIP, 2012. http://dx.doi.org/10.1063/1.4769929.

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