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Books on the topic 'Atherosclerosis: pathology'

Author: Grafiati

Published: 4 June 2021

Last updated: 15 February 2022

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1

Chlamydia atherosclerosis lesion: Discovery, diagnosis and treatment. Springer, 2007.

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2

J, Halpern M., ed. Lipid metabolism and its pathology: Proceedings of the IVth International Colloquium on Lipid Metabolism and its Pathology, Lisbon, 18-22 November 1984. Excerpta Medica, 1986.

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3

Cellular dysfunction in atherosclerosis and diabetes: Reports from bench to bedside. Romanian Academy Pub. House, 2004.

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4

Tracy, Richard E. The role of aging in atherosclerosis: The sequestration hypothesis. Kluwer Academic Publishers, 2003.

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5

Altschul Symposium (1st 1990 Saskatoon, Sask.). Atherosclerosis: Cellular and molecular interactions in the artery wall. Plenum Press, 1991.

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6

Yen, Ho Siew, ed. Atlas of coronary artery disease. Lippincott Raven, 1998.

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7

NATO Advanced Research Workshop on Progress, Problems, and Promises for an Effective Quantitative Evaluation of Atherosclerosis in Living and Autopsied Experimental Animals and Man (1990 Siena, Italy). Atherosclerotic plaques: Advances in imaging for sequential quantitative evaluation. Edited by Wissler Robert W. 1917- and North Atlantic Treaty Organization. Scientific Affairs Division. Plenum Press, 1991.

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8

S, Edgington Thomas, Ross Russell, and Silverstein Samuel C, eds. Perspectives in inflammation, neoplasia, and vascular cell biology: Proceedings of the Triton Biosciences-UCLA Symposium, held in Park City, Utah, February 2-8, 1985. Liss, 1987.

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9

Triton Biosciences-UCLA Symposium (1985 Park City, Utah). Perspectives in inflammation, neoplasia, and vascular cell biology: Proceedings of a Triton Biosciences-UCLA Symposium, held in Park City, Utah, February 2-8, 1985. Edited by Edgington Thomas S, Ross Russell, and Silverstein Samuel C. A.R. Liss, 1987.

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10

Tousoulis, Dimitris. Risk factors and vascular endothelium. Nova Science Publishers, 2011.

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11

Ockner, Robert K. Integration of metabolism, energetics, and signal transduction: Unifying foundations in cell growth and death, cancer, atherosclerosis, and Alzheimer disease. Kluwer Academic Publishers, 2004.

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12

General pathobiology. Appleton & Lange, 1994.

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13

Spontaneous and induced intima formation in blood vessels. R.G. Landes, 1995.

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14

Atherosclerosis: Pathology of the Vasculature in Live Patients. W.B. Saunders Company, 1999.

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15

Wilson, Peter W. F. Atlas of Atherosclerosis. 2nd ed. Current Medicine, 2000.

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16

M, Grundy Scott, and Current Medicine Inc, eds. Atlas of atherosclerosis. 4th ed. Current Medicine, 2005.

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17

Thubrikar, Mano J. Vascular Mechanics and Pathology. Springer, 2007.

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18

Carton, James. Vascular pathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0003.

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This chapter covers vascular pathology, including common arterial and venous diseases including atherosclerosis and its complications, hypertension, shock, chronic lower limb ischaemia, acute lower limb ischaemia, aortic dissection, abdominal aortic aneurysm, deep vein thrombosis, and varicose veins.

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19

Atherosclerosis: Risk Factors & Treatment. Mosby-Year Book, 1996.

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20

Carton, James. Vascular pathology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199591633.003.0003.

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Atherosclerosis 28Shock 30Hypertension 31Chronic lower limb ischaemia 32Acute lower limb ischaemia 33Aortic dissection 34Abdominal aortic aneurysm 35Varicose veins 36Deep vein thrombosis 37• An inflammatory disease of large- and medium-sized systemic arteries characterized by the formation of lipid-rich plaques in the vessel wall....

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21

(Editor), Valentin Fuster, Herbert C. Stary (Editor), a. Bleakley Chandler (Editor), et al., eds. Syndromes of Atherosclerosis: Correlations of Clinical Imaging and Pathology. Futura Publishing Company, 1996.

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22

Syndromes of atherosclerosis: Correlations of clinical imaging and pathology. Futura Pub. Co., 1996.

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23

Virgil, Brown W., ed. Atherosclerosis: Risk factors and treatment. Mosby, 1996.

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24

Shor, Allan. Chlamydia Atherosclerosis Lesion: Discovery, Diagnosis and Treatment. Springer, 2016.

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25

1948-, Wilson Peter, and Current Medicine Inc, eds. Atlas of atherosclerosis: Risk factors and treatment. 3rd ed. Current Medicine, 2003.

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26

Atherosclerosis and arteriosclerosis: Human pathology and experimental animal methods and models. CRC Press, 1989.

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27

Atlas Of Atherosclerosis And Metabolic Syndrome. Springer, 2010.

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28

Titov. Phylogenetic theory of the general pathology. Pathogenesis of a civilization diseases. Atherosclerosis. Infra-M Academic Publishing House, 2013. http://dx.doi.org/10.12737/858.

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29

The Role of Aging in Atherosclerosis: The Sequestration Hypothesis. Springer, 2003.

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30

Tracy, R. E. The Role of Aging in Atherosclerosis: The Sequestration Hypothesis. Springer, 2014.

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31

N, Diana John, ed. Tobacco smoking and atherosclerosis: Pathogenesis and cellular mechanisms. Plenum Press, 1990.

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32

Seymour, Glagov, Newman William P, and Schaffer Sheldon A, eds. Pathobiology of the human atherosclerotic plaque. Springer-Verlag, 1990.

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33

(Editor), Robert W. Wissler, M. Gene Bond (Editor), Michele Mercuri (Editor), Piero Tanganelli (Editor), and Giorgio Weber (Editor), eds. Atherosclerotic Plaques: Advances in Imaging for Sequential Quantitative Evaluation (Nato Science Series: A:). Springer, 1992.

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34

(Editor), Sergey Fedoroff, Gary D. Burkholder (Editor), Avrum I. Gotlieb (Editor), and B. Lowell Langille (Editor), eds. Atherosclerosis: Cellular and Molecular Interactions in the Artery Wall (Altschul Symposia Series). Springer, 1991.

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35

Klingenberg, Roland, and Ulf Müller-Ladner. Mechanisms of inflammation. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0270.

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This chapter provides a brief summary of the immune pathogenesis of atherosclerosis, highlighting shared features with inflammatory pathways in rheumatoid arthritis (RA) described in detail in Chapter 25.4. RA constitutes a prototype autoimmune disease primarily affecting the joints but also the heart and vessels associated with increased cardiovascular mortality. Recent years have produced a wealth of novel insights into the diversity of immune cell types which either propagate or dampen inflammation in atherogenesis. Expansion of this inherent anti-inflammatory component carried by regulator

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36

Perspectives in inflammation, neoplasia, and vascular cell biology: Proceedings of a Triton Biosciences-UCLA Symposium, held in Park City, Utah, February ... symposia on molecular and cellular biology). A.R. Liss, 1987.

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37

S, Suri Jasjit, and Laxminarayan Swamy, eds. Angiography and plaque imaging: Advanced segmentation techniques. CRC Press, 2003.

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38

Alfonso, Fernando, and Javier Botas. Intracoronary Diagnostic Techniques. Cc de la Salud, 2003.

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39

Thornton, Clare, and Justin Mason. Vascular biology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0057.

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Vascular biology is the study of the physiology of the vasculature and how it may be the target for disease processes. An understanding of vascular biology is central to the study of rheumatic disease for three reasons: it is an integral part of a functioning immune system; it is the primary site of pathology in many conditions; and it is the site of the important secondary complications of chronic inflammation, endothelial dysfunction and atherosclerosis. Vascular biology requires a detailed knowledge of the anatomy and physiology of the vasculature and its constituent vessels. The multistep

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40

Thornton, Clare, and Justin Mason. Vascular biology. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199642489.003.0057_update_001.

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Vascular biology is the study of the physiology of the vasculature and how it may be the target for disease processes. An understanding of vascular biology is central to the study of rheumatic disease for three reasons: it is an integral part of a functioning immune system; it is the primary site of pathology in many conditions; and it is the site of the important secondary complications of chronic inflammation, endothelial dysfunction and atherosclerosis. Vascular biology requires a detailed knowledge of the anatomy and physiology of the vasculature and its constituent vessels. The multistep

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41

Krams, Robert, and Magnus Bäck, eds. The ESC Textbook of Vascular Biology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.001.0001.

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Atherosclerosis continues to be the most significant pathology giving rise to cardiovascular disease worldwide. Vascular biology forms the basic armamentarium to address the underlying mechanisms. The ESC Textbook of Vascular Biology discusses these new developments and recent breakthroughs in the field, while also covering the major changes in the diagnosis, prevention, and treatment of atherosclerosis that have occurred in recent years. This book offers a compendium of topics written by the best scientists in Europe. The dissemination of the authors’ vast knowledge and expertise in a single

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42

London, Gerard M. Cardiovascular complications in end-stage renal disease patients. Edited by Jonathan Himmelfarb. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0268.

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Cardiovascular complications are the predominant cause of death in patients with end-stage renal disease (ESRD). The high incidence of cardiovascular complications results from pathology present before ESRD (generalized atherosclerosis, diabetes, hypertension) and an additive effect of multiple factors including haemodynamic overload and metabolic and endocrine abnormalities more or less specific to uraemia or its treatment modalities. These disorders are usually associated and can exacerbate each other. While ischaemic heart disease is a frequent cause of cardiac death, heart failure and sudd

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43

Raggi, Paolo, and Luis D’Marco. Imaging for detection of vascular disease in chronic kidney disease patients. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0116.

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The well-known severity of cardiovascular disease in patients suffering from chronic kidney disease (CKD) requires an accurate risk stratification of these patients in several clinical situations. Imaging has been used successfully for such purpose in the general population and it has demonstrated excellent potential among CKD patients as well. Two main forms of arterial pathology develop in patients with CKD: atherosclerosis, with accumulation of inflammatory cells, lipids, fibrous tissue and calcium in the subintimal space, and arteriosclerosis. The latter is characterized by accumulation of

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44

Kockx, Mark M. Spontaneous and Induced Intima Formation in Blood Vessels. Springer, 2013.

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45

Carotid Atherosclerotic Disease: Pathologic Basis for Treatment. Informa Healthcare, 2008.

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46

Guzik, Tomasz J., and Rhian M. Touyz. Vascular pathophysiology of hypertension. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755777.003.0019.

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Hypertension is a multifactorial disease, in which vascular dysfunction plays a prominent role. It occurs in over 30% of adults worldwide and an additional 30% are at high risk of developing the disease. Vascular pathology is both a cause of the disease and a key manifestation of hypertension-associated target-organ damage. It leads to clinical symptoms and is a key risk factor for cardiovascular disease. All layers of the vascular wall and the endothelium are involved in the pathogenesis of hypertension. Pathogenetic mechanisms, whereby vascular damage contributes to hypertension, are linked

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47

Jaff, Michael R. Endovascular Therapy for Atherosclerotic Renal Artery Stenosis: Present and Future. 2nd ed. Blackwell Publishing Limited, 2001.

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48

Ritchie, James, Darren Green, Constantina Chrysochou, and Philip A. Kalra. Renal artery stenosis. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0213.

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Renovascular disease refers to a narrowing of a main or branch renal artery. Consequences include loss of functional renal tissue and renovascular hypertension, with other manifestations depending on the underlying cause. Worldwide the most common cause is atherosclerotic narrowing, with other causal pathologies including fibromuscular disease (FMD) and inflammatory conditions. FMD occurs much more frequently in women than in men, and is associated with smoking but genetic predisposing factors are also suspected. In South East Asia, Takayasu arteritis is an important cause.Takayasu disease oft

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49

Shirodaria, Cheerag, and Sam Dawkins. Chronic stable angina. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0089.

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Chronic stable angina is a condition where patients experience symptoms of chest pain of a particular character (e.g. angina pectoris) on effort only, due to atherosclerotic coronary artery disease. The hallmarks of stable angina are as follows. First, there must be stable atherosclerotic coronary artery disease, resulting in luminal narrowing(s) in one or more of the major epicardial coronary arteries. Atheroma can be detected by using appropriate technology. Not all angina chest discomfort is due to atherosclerotic coronary disease—some is due to aortic stenosis, and some to hypertrophic car

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50

Covic, Adrian, Mugurel Apetrii, Luminita Voroneanu, and David J. Goldsmith. Vascular calcification. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0120_update_001.

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Vascular calcification (VC) is a common feature of patients with advanced CKD and it could be, at least in part, the cause of increased cardiovascular mortality in these patients. From a morphologic point of view, there are at least two types of pathologic calcium phosphate deposition in the arterial wall—namely, intima calcification (mostly associated with atherosclerotic plaques) and media calcification (associated with stiffening of the vasculature, resulting in significantly adverse cardiovascular outcomes). Although VC was viewed initially as a passive phenomenon, it appears to be a cell-

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