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Journal articles on the topic 'Atherosclerosis – Risk factors'

Author: Grafiati

Published: 4 June 2021

Last updated: 28 January 2023

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1

FUJITA, Toshiro, and Katsuyuki ANDO. "Risk Factors of Atherosclerosis." Journal of Japan Atherosclerosis Society 23, no. 7-8 (1996): 423–27. http://dx.doi.org/10.5551/jat1973.23.7-8_423.

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2

Siscovick, David, Frits Rosendaal, and Alexander Reiner. "Hemostatic Risk Factors and Arterial Thrombotic Disease." Thrombosis and Haemostasis 85, no. 04 (2001): 584–95. http://dx.doi.org/10.1055/s-0037-1615638.

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SummaryThe pathogenesis of arterial thrombotic disease involves multiple genetic and environmental factors related to atherosclerosis and thrombosis. Acute thrombosis at the site of a ruptured, lipid-rich atherosclerotic plaque is the usual precipitating event in the transition from stable or subclinical atherosclerotic disease to acute myocardial infarction (MI), stroke, or peripheral arterial occlusion (1). Pathologic studies of coronary arteries in acute MI suggest that the acute thrombosis likely involves activation of both platelets and the coagulation system.

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3

D’Adamo, Ebe, Ornella Guardamagna, Francesco Chiarelli, et al. "Atherogenic Dyslipidemia and Cardiovascular Risk Factors in Obese Children." International Journal of Endocrinology 2015 (2015): 1–9. http://dx.doi.org/10.1155/2015/912047.

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Childhood obesity when associated with serum lipoprotein changes triggers atherosclerosis. Evidences suggest that the atherosclerotic process begins in childhood and that the extent of early atherosclerosis of the aorta and coronary arteries can be associated with lipoprotein levels and obesity. Furthermore, many studies in childhood demonstrate an important relationship between parameters of insulin sensitivity, body fat distribution, and the development of lipid abnormalities. This review focuses on the most recent findings on the relationship between obesity, dyslipidemia, and cardiovascula

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4

YASUDA, Hideaki, Hideyuki TSUBOI, and Takahito SONE. "Risk factors influencing carotid atherosclerosis." Choonpa Igaku 34, no. 1 (2007): 57–63. http://dx.doi.org/10.3179/jjmu.34.57.

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5

Kullo, Iftikhar J., Gerald T. Gau, and A. Jamil Tajik. "Novel Risk Factors for Atherosclerosis." Mayo Clinic Proceedings 75, no. 4 (2000): 369–80. http://dx.doi.org/10.4065/75.4.369.

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6

Kullo, Iftikhar J., and Christie M. Ballantyne. "Conditional Risk Factors for Atherosclerosis." Mayo Clinic Proceedings 80, no. 2 (2005): 219–30. http://dx.doi.org/10.4065/80.2.219.

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7

Dobroski, David R., and Joseph Loscalzo. "Thrombotic risk factors for atherosclerosis." Coronary Artery Disease 7, no. 12 (1996): 919–32. http://dx.doi.org/10.1097/00019501-199612000-00007.

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8

Leijdekkers, V. J., A. C. Vahl, J. J. M. Leenders, P. C. Huijgens, R. O. B. Gans, and J. A. Rauwerda. "Risk Factors for Premature Atherosclerosis." European Journal of Vascular and Endovascular Surgery 17, no. 5 (1999): 394–97. http://dx.doi.org/10.1053/ejvs.1998.0775.

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9

Rosanov, V., E. Shugaeva, A. Alexandrov, and Ch Pugoeva. "Alimentary risk factors of atherosclerosis." Atherosclerosis Supplements 2, no. 2 (2001): 112. http://dx.doi.org/10.1016/s1567-5688(01)80311-1.

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10

Papazafiropoulou, Athanasia, Nicholas Katsilambros, and Nicholas Tentolouris. "Novel Risk Factors for Atherosclerosis." Open Biomarkers Journal 1, no. 1 (2008): 36–47. http://dx.doi.org/10.2174/1875318300801010036.

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Epidemiologic studies demonstrated that the classical cardiovascular risk factors explain only a part of the increased cardiovascular morbidity and mortality. Large scale studies have shown that novel cardiovascular risk factors, including increased plasma homocysteine, fibrinogen, C-reactive protein, uric acid levels, and increased white blood cells count as well as low adiponectin levels, might have a key role in the pathogenesis of the cardiovascular disease. This review examines recent literature data on the effect of novel risk factors on cardiovascular morbidity and mortality in healthy

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11

NAKAZAWA, Kohji, and Katsumi MURATA. "Epidemiological Study of the Risk Factors for Atherosclerosis." Journal of Japan Atherosclerosis Society 13, no. 6 (1986): 1433–36. http://dx.doi.org/10.5551/jat1973.13.6_1433.

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12

Dreval, A. K. "Prevention and treatment of diabetic microangiopathy: A lecture." Problems of Endocrinology 41, no. 6 (1995): 29–34. http://dx.doi.org/10.14341/probl11491.

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One of the main complications leading to high disability and mortality in patients with diabetes mellitus is atherosclerotic vascular disease (diabetic macroangiopathy). At the same time, the frequency of atherosclerosis among patients with diabetes mellitus is significantly affected by the so-called risk factors for atherosclerosis. In particular, the incidence of atherosclerosis among the general population and among diabetics increases with increased levels of cholesterol (cholesterol) in the blood, blood pressure, smoking and obesity. However, in patients with diabetes mellitus, mortality,

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13

Zurnic, Irena, Tamara Djuric, Igor Koncar, Aleksandra Stankovic, Dragan Dincic, and Maja Zivkovic. "Apolipoprotein E gene polymorphisms as risk factors for carotid atherosclerosis." Vojnosanitetski pregled 71, no. 4 (2014): 362–67. http://dx.doi.org/10.2298/vsp1404362z.

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Background/Aim. Atherosclerosis is still the leading cause of death in Western world. Development of atherosclerotic plaque involves accumulation of inflammatory cells, lipids, smooth muscle cells and extracellular matrix proteins in the intima of the vascular wall. Apolipoprotein E participates in the transport of exogenous cholesterol, endogenouly synthesized lipids and triglycerides in the organism. Apolipoprotein E gene has been identified as one of the candidate genes for atherosclerosis. Previous studies in different populations have clearly implicated apolipoprotein E genetic variation

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14

Fahed, Akl C., Joanna M. Gholmieh, and Sami T. Azar. "Connecting the Lines between Hypogonadism and Atherosclerosis." International Journal of Endocrinology 2012 (2012): 1–12. http://dx.doi.org/10.1155/2012/793953.

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Epidemiological studies show that atherosclerotic cardiovascular disease is a leading cause of morbidity and mortality worldwide and point to gender differences with ageing males being at highest risk. Atherosclerosis is a complex process that has several risk factors and mediators. Hypogonadism is a commonly undiagnosed disease that has been associated with many of the events, and risk factors leading to atherosclerosis. The mechanistic relations between testosterone levels, atherosclerotic events, and risk factors are poorly understood in many instances, but the links are clear. In this pape

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15

Saito, Yasushi. "Atherosclerosis and risk factors in eldery." Nippon Ronen Igakkai Zasshi. Japanese Journal of Geriatrics 45, no. 5 (2008): 470–73. http://dx.doi.org/10.3143/geriatrics.45.470.

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16

Dolidze, N., and N. Latsabidze. "Nontraditional risk factors for coronary atherosclerosis." European Journal of Cardiovascular Prevention & Rehabilitation 13, Supplement 1 (2006): S67. http://dx.doi.org/10.1097/00149831-200605001-00270.

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17

TOMONO, SHOICHI, MASAKO FURUYA, TAKASHI IIZUKA, NAHOKO SATOH, RIKURO HAYASHI, and SEIICHIRO KOGURE. "THE RISK FACTORS OF CORONARY ATHEROSCLEROSIS." KITAKANTO Medical Journal 35, no. 3Supplement (1985): 315–22. http://dx.doi.org/10.2974/kmj1951.35.3supplement_315.

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18

Skilton, M. R. "Intrauterine Risk Factors for Precocious Atherosclerosis." PEDIATRICS 121, no. 3 (2008): 570–74. http://dx.doi.org/10.1542/peds.2007-1801.

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19

Ridker, Paul M., Meir J. Stampfer, and Nader Rifai. "Novel Risk Factors for Systemic Atherosclerosis." JAMA 285, no. 19 (2001): 2481. http://dx.doi.org/10.1001/jama.285.19.2481.

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20

Duffy, David L., Dianne L. O'Connell, Richard F. Heller, and Nicholas G. Martin. "Risk factors for atherosclerosis in twins." Genetic Epidemiology 10, no. 6 (1993): 557–62. http://dx.doi.org/10.1002/gepi.1370100638.

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21

&NA;. "Atherosclerosis risk factors increase VTE risk with tamoxifen." Inpharma Weekly &NA;, no. 1477 (2005): 21. http://dx.doi.org/10.2165/00128413-200514770-00047.

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&NA;. "Atherosclerosis risk factors increase VTE risk with tamoxifen." Reactions Weekly &NA;, no. 1041 (2005): 3. http://dx.doi.org/10.2165/00128415-200510410-00006.

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23

Scherbak, S. G., D. G. Lisovets, A. M. Sarana, et al. "BIOMARKERS OF CARDIOVASCULAR DISEASE." Physical and rehabilitation medicine, medical rehabilitation 1, no. 2 (2019): 60–76. http://dx.doi.org/10.36425/2658-6843-19190.

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Inflammation, cardiac remodeling, and fibrosis are potentially important pathways in the pathogenesis of cardiovascular diseases. Complications of atherosclerosis are one of the leading causes of death in the world. Effective prevention of cardiovascular disease by adequate control of major cardiovascular risk factors can provide substantial public health gains. However, detection and control of major cardiovascular risk factors continues to be a major challenge because of poor awareness of an individual's status. A solution to this problem is important for an early identification and appropri

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24

ZHOU, TIAN, YIMING ZHENG, JUHUI QIU, et al. "ENDOTHELIAL MECHANOTRANSDUCTION MECHANISMS FOR VASCULAR PHYSIOLOGY AND ATHEROSCLEROSIS." Journal of Mechanics in Medicine and Biology 14, no. 05 (2014): 1430006. http://dx.doi.org/10.1142/s0219519414300063.

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Vascular physiology and disease progression, such as atherosclerosis, are mediated by hemodynamic force generated from blood flow. The hemodynamic force exerts on vascular endothelial cells (ECs), which could perceive the mechanical signals and transmit them into cell interior by multiple potential shear sensors, collectively known as mechanotransduction. However, we do not understand completely how these shear-sensitive components orchestrate physiological and atherosclerotic responses to shear stress. In this review, we provide an overview of biomechanical mechanisms underlying vascular phys

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25

Negru, Robert D., Andrei Georgescu, Bogdan Mihnea Ciuntu, et al. "Periodontitis and Cardiovascular Risk." Revista de Chimie 68, no. 12 (2018): 2896–901. http://dx.doi.org/10.37358/rc.17.12.6002.

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Atherosclerosis is a major component of the cardiovascular diseases and is centered by inflammation but its well-known predictors do not explain some of the atherosclerotic vascular disease events, generating the need to look for independent additional risk factors. Periodontitis, a chronic infection produced by oral bacteria and affecting the supporting structures of the teeth, seems to be linked with atherosclerosis and cardiovascular disease through several mechanisms, like genetic susceptibility, systemic inflammation, infection, and the molecular mimicry, the association being worsened in

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26

Kim, Jong S., Yeon-Jung Kim, Sung-Ho Ahn, and Bum J. Kim. "Location of cerebral atherosclerosis: Why is there a difference between East and West?" International Journal of Stroke 13, no. 1 (2016): 35–46. http://dx.doi.org/10.1177/1747493016647736.

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Intracranial atherosclerosis is more prevalent in Asian patients, whereas extracranial atherosclerosis is more common in individuals from western countries. The reasons for this discrepancy remain unknown. We reviewed the relevant literature and discussed the currently available information. Although the study population, diagnostic modality, and risk factor definitions differ between studies, hypercholesterolemia is more correlated with extracranial atherosclerosis than intracranial atherosclerosis. The difference in hypercholesterolemia prevalence is one of the main reasons for racial differ

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27

Damjanovic, Tatjana, and Nada Dimkovic. "Dialysis modalities as risk factors in the development of atherosclerosis." Medical review 56, no. 1-2 (2003): 17–21. http://dx.doi.org/10.2298/mpns0302017d.

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Introduction Atherosclerosis is a major risk factor for increased cardiovascular morbidity and mortality in dialysis patients. First clinical symptoms are usually associated with initial atherosclerotic changes of blood vessels. The aim of this study was ultrasound evaluation of intimae media thickness (IMT) of carotid arteries in dialysis patients and its correlation with certain risk factors. Patients and methods IMT was measured in 45 dialysis patients with no signs of cardiovascular diseases:15 continuous ambulatory peritoneal dialysis (CAPD) patients, 30 haemodialysis (HD) patients and in

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28

Thorbjornsdottir, P., S. T. Sigurdarson, S. Bodvarsson, G. Thorgeirsson, and G. J. Arason. "The correlation of C3 to atherosclerosis and atherosclerosis risk factors." Molecular Immunology 46, no. 14 (2009): 2843–44. http://dx.doi.org/10.1016/j.molimm.2009.05.255.

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29

Bugger, Heiko, and Andreas Zirlik. "Anti-inflammatory Strategies in Atherosclerosis." Hämostaseologie 41, no. 06 (2021): 433–42. http://dx.doi.org/10.1055/a-1661-0020.

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AbstractAtherosclerotic vascular disease and its related complications are the major cause of mortality in Western societies. Atherosclerosis is a chronic inflammatory disease of the arterial wall triggered by traditional and nontraditional risk factors and mediated by inflammatory and immune responses. Recent clinical trials provided compelling evidence corroborating that atherosclerosis is an inflammatory disease and demonstrated efficacy of anti-inflammatory interventions in reducing cardiovascular events and mortality. Traditional risk factors drive vascular inflammation, further justifyin

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30

Lechner, Katharina, Clemens von Schacky, Amy L. McKenzie, et al. "Lifestyle factors and high-risk atherosclerosis: Pathways and mechanisms beyond traditional risk factors." European Journal of Preventive Cardiology 27, no. 4 (2019): 394–406. http://dx.doi.org/10.1177/2047487319869400.

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Despite major efforts to reduce atherosclerotic cardiovascular disease (ASCVD) burden with conventional risk factor control, significant residual risk remains. Recent evidence on non-traditional determinants of cardiometabolic health has advanced our understanding of lifestyle–disease interactions. Chronic exposure to environmental stressors like poor diet quality, sedentarism, ambient air pollution and noise, sleep deprivation and psychosocial stress affect numerous traditional and non-traditional intermediary pathways related to ASCVD. These include body composition, cardiorespiratory fitnes

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31

Raman Parajuli, Sudhir, Bishwonath Yadav, Prahlad Karki, Paricha Upadhyaya, and Shivendra Jha. "An Autopsy Study of Atherosclerotic Changes in Coronary Arteries at B.P. Koirala Institute of Health Sciences." Annapurna Journal of Health Sciences 1, no. 1 (2021): 4–8. http://dx.doi.org/10.52910/ajhs.5.

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Introduction: Atherosclerosis is a giant killer and the incidence of atherosclerosis in coronary arteries is rapidly increasing. The study was designed to assess the atherosclerotic lesions in coronary artery and to correlate the risk factors related to prevalence of atherosclerosis. Methods: Heart from 100 medico legal autopsy cases ranging between ages 15 to 35 years which came to BPKIHS Dharan were taken for this study and processed for coronary arteries using conventional technique. They were then studied,and grading was done based on Modified American Heart Association (AHA) classif

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32

Ferns, Gordon A. A. "New and emerging risk factors for CVD." Proceedings of the Nutrition Society 67, no. 2 (2008): 223–31. http://dx.doi.org/10.1017/s0029665108007064.

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Morphological and immunocytochemical studies have elucidated the complex processes involved in atherogenesis. The notion of plaque instability has emerged from this work and underscored the importance of inflammation in determining clinical complications associated with atherosclerosis, such as acute coronary syndrome. Cells of the immune system have been detected within atherosclerotic lesions and auto-antibodies directed against modified LDL and heat-shock proteins have been identified in the blood of individuals with atherosclerosis. The use of risk ‘engines’, e.g. the Framingham coronary r

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33

An, Jihyun, Hyung-Don Kim, Seon-Ok Kim, et al. "Cervicocerebral atherosclerosis and its hepatic and coronary risk factors in patients with liver cirrhosis." Clinical and Molecular Hepatology 28, no. 1 (2022): 67–76. http://dx.doi.org/10.3350/cmh.2021.0202.

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Background/Aims: We aimed to investigate the silent atherosclerotic burden of cervicocephalic vessels in cirrhotic patients compared with the general population, as well as the relevant risk factors including coronary parameters.Methods: This study included 993 stroke-free patients with liver cirrhosis (LC) who underwent magnetic resonance angiography (MRA) of the head and neck as a pre-liver transplant assessment and 6,099 health checkup participants who underwent MRA examination. The two cohorts were matched for cerebrovascular risk factors, and the prevalence of atherosclerosis in major int

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34

Lloyd, Guy W., and Graham Jackson. "Premenopausal risk factors for coronary artery disease." British Menopause Society Journal 8, no. 3 (2002): 108–11. http://dx.doi.org/10.1258/136218002100321802.

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The premenopausal period is a time when the risk of coronary events for women is low. It is, however, a key period during which the atherosclerotic process, the leading cause of death in women, becomes established. The role of female sex hormones in protecting premenopausal women from overt atherosclerosis is widely accepted. Proof of a direct oestrogen, atheroprotective effect, however, continues to prove elusive. The specific risk factors are diabetes, hypertension, lipid abnormalities, smoking, body habitus, inflammatory disease and prothrombotic tendencies. Reproductive factors do not appe

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35

Svyrydova, N. K., N. O. Kravchuk, and K. F. Trinus. "Atherosclerotic vascular lesions in patients with chronic brain ischemia." East European Journal of Neurology, no. 6(6) (December 20, 2015): 13–22. http://dx.doi.org/10.33444/2411-5797.2015.6(6).13-22.

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Atherosclerotic cardiovascular disease is the leading cause of mortality worldwide. Evidence suggests that the risk of atherosclerosis complications associated not only with local changes within atherosclerotic plaque (such as the state necrotic core or fibrous cap), but also with the state of blood or systemic factors. Circulating levels of cytokines, prothrombotic factors or acute phase reactants may play a role in the occurrence of acute stroke in the presence of affected vessels, even in the absence of stenosis. For example, increased serum biomarkers such as C-reactive protein, cytokines

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36

Ershova, A. I., S. A. Boytsov, О. M. Drapkina, and Т. V. Balakhonova. "ULTRASOUND MARKERS OF PREMANIFEST ATHEROSCLEROSIS OF CAROTID AND FEMORAL ARTERIES IN ASSESSMENT OF CARDIOVASCULAR RISK." Russian Journal of Cardiology, no. 8 (September 9, 2018): 92–98. http://dx.doi.org/10.15829/1560-4071-2018-8-92-98.

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More than a half of cardiovascular events occur in low to moderate cardiovascular risk patients if assessed based on the traditional risk factors. At the same time, ultrasound examination of arteries makes it possible to reveal atherosclerosis even at early stages of its development. High prevalence of subclinical atherosclerosis in low to moderate risk patients, which is a realization of traditional and “unknown” risk factors, makes ultrasound examination a useful method for risk stratification. The review is focused on ultrasound markers of atherosclerosis with association to traditional ris

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37

Pizov, A. V., N. A. Pizov, O. A. Skachkova, and N. V. Pizova. "Endothelial dysfunction as early predictor of atherosclerosis." Medical alphabet 4, no. 35 (2020): 28–33. http://dx.doi.org/10.33667/2078-5631-2019-4-35(410)-28-33.

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Cardiovascular diseases in Russia are leading in the structure of total mortality. Atherosclerosis is considered a progressive inflammatory systemic disease. The role of endothelium in the development of the atherosclerotic process is described in detail. The main functions of endotheliocytes are normal and in various pathological conditions. The main markers of endothelial dysfunction are presented. Data on the development of the atherosclerotic process in time, risk factors are presented. Local and systemic risk factors for atherosclerosis are highlighted. Own data on the frequency of occurr

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38

Fruchart, J. C. "New Risk Factors for Atherosclerosis and Patient Risk Assessment." Circulation 109, no. 23_suppl_1 (2004): III—15—III—19. http://dx.doi.org/10.1161/01.cir.0000131513.33892.5b.

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39

Poredoš, Pavel, and Mateja Kaja Ježovnik. "Markers of preclinical atherosclerosis and their clinical relevance." Vasa 44, no. 4 (2015): 247–56. http://dx.doi.org/10.1024/0301-1526/a000439.

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Abstract. The estimation of risk for atherosclerotic and cardiovascular events based only on the presence of classical risk factors is often insufficient. Therefore, efforts have been made to find markers that indicate the presence of preclinical disease in individual subjects: blood markers of atherosclerosis and preclinical deterioration of the arterial wall. Elevated levels of several inflammatory mediators have been found in subjects with atherosclerosis. Increased basal levels of cytokines, the cell adhesion molecules, selectins and acute-phase reactants such as high sensitive C-reactive

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40

Shenkova, N. N., G. A. Chumakova, N. G. Veselovskaya, E. S. Osipova, and A. V. Ott. "Risk factors of subclinical atherosclerosis of obesity women." CardioSomatics 8, no. 2 (2017): 44–50. http://dx.doi.org/10.26442/cs45328.

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Research objective: study the metabolic risk factors (RF) of subclinical atherosclerosis of obese women. Material and methods. 89 women have been included in research, 50.6±6.6 years old with abdominal obesity and waist circumference >80 cm without coronary heart disease and atherosclerosis of other localisation. According to the results of the ultrasound study of brachiocephalic arteries (BCA), two groups were formed for comparison: Group 1 (n=60) without atherosclerosis BCA, Group 2 (n=29) with atherosclerosis BCA. Definitions of classical and additional metabolic RF, of adipokines of vis

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41

NAKAMURA, Tadashi. "Visceral Fat and Risk Factors for Atherosclerosis." Internal Medicine 43, no. 12 (2004): 1095–96. http://dx.doi.org/10.2169/internalmedicine.43.1095.

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42

Hasegawa, Takashi, Toyoko Negishi, and Miyako Deguchi. "WBC Count, Atherosclerosis and Coronary Risk Factors." Journal of Atherosclerosis and Thrombosis 9, no. 5 (2002): 219–23. http://dx.doi.org/10.5551/jat.9.219.

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43

Suciu, Claudia Floriana, Robert Adrian Dumbrava, Maria Andrada Jiga, Liviu Cristescu, and Andreea Varga. "Shared risk factors for atherosclerosis and arteriosclerosis." Romanian Journal of Medical Practice 15, no. 2 (2020): 174–77. http://dx.doi.org/10.37897/rjmp.2020.2.9.

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44

Hutchinson, Richard G., Robert L. Watson, C. Edward Davis, et al. "Racial Differences in Risk Factors for Atherosclerosis." Angiology 48, no. 4 (1997): 279–90. http://dx.doi.org/10.1177/000331979704800401.

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45

Leinonen, Maija. "Chlamydia pneumoniaeand Other Risk Factors for Atherosclerosis." Journal of Infectious Diseases 181, s3 (2000): S414—S416. http://dx.doi.org/10.1086/315624.

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Oghagbon, E. K., A. B. Okesina, and S. A. Adebisi. "Awareness of atherosclerosis risk factors in Nigeria." Journal of the Royal Society for the Promotion of Health 124, no. 4 (2004): 180–83. http://dx.doi.org/10.1177/146642400412400411.

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47

Burgess, Jefferey L., Margaret Kurzius-Spencer, Richard D. Gerkin, James L. Fleming, Wayne F. Peate, and Matthew Allison. "Risk Factors for Subclinical Atherosclerosis in Firefighters." Journal of Occupational and Environmental Medicine 54, no. 3 (2012): 328–35. http://dx.doi.org/10.1097/jom.0b013e318243298c.

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48

Misra, Anoop. "Risk Factors for Atherosclerosis in Young Individuals." European Journal of Cardiovascular Risk 7, no. 3 (2000): 215–29. http://dx.doi.org/10.1177/204748730000700309.

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49

Crouse, J. R., J. F. Toole, W. M. McKinney, et al. "Risk factors for extracranial carotid artery atherosclerosis." Stroke 18, no. 6 (1987): 990–96. http://dx.doi.org/10.1161/01.str.18.6.990.

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50

ZOLER, MITCHEL L. "Registry Shakes Up Risk Factors for Atherosclerosis." Internal Medicine News 43, no. 15 (2010): 1–12. http://dx.doi.org/10.1016/s1097-8690(10)70761-7.

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