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Dissertations / Theses on the topic 'Atherosclerotic plague'

Author: Grafiati

Published: 4 June 2021

Last updated: 6 February 2022

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1

Shakya, Arvind. "Mechanism of matrix metalloproteinase-14 (mmp-14) regulation during atherosclerosis." Diss., Columbia, Mo. : University of Missouri-Columbia, 2006. http://hdl.handle.net/10355/4436.

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Thesis (Ph. D.)--University of Missouri-Columbia, 2006.<br>"December 2006" The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Vita. Includes bibliographical references.

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2

McCord, Barbara Norton. "Fatigue of atherosclerotic plaque." Diss., Georgia Institute of Technology, 1992. http://hdl.handle.net/1853/15890.

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3

Zhu, Chengcheng. "High resolution black blood magnetic resonance imaging of atherosclerotic plaque." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648792.

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4

Obaid, Daniel Rhys. "Coronary atherosclerotic plaque imaging." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608243.

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5

Lilledahl, Magnus Borstad. "Detection of Vulnerable Atherosclerotic Plaque." Doctoral thesis, Norwegian University of Science and Technology, Department of Electronics and Telecommunications, 2007. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-1506.

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<p>Heart attacks are the cause for the majority of deaths in the industrialized world. The underlying cause of most of these deaths is the rupture of vulnerable plaque. As of this day, no widely accepted clinical tool exists for detecting plaques that are prone to rupture, although several techniques are being investigated. This thesis gives an overview of detection modalities that have been proposed for detecting vulnerable plaque with special emphasis on three methods, diffuse reflection spectroscopy, thermography and multiphoton microscopy.</p>

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6

Calvert, Patrick Andrew. "Virtual-histology intravascular ultrasound in vulnerable atherosclerosis." Thesis, University of Cambridge, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609857.

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7

Loree, Howard Martin. "The mechanics of atherosclerotic plaque rupture." Thesis, Massachusetts Institute of Technology, 1992. http://hdl.handle.net/1721.1/17301.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Whitaker College of Health Sciences and Technology, 1992.<br>Includes bibliographical references (leaves 139-146).<br>by Howard M. Loree II.<br>Ph.D.

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8

Kim, Taehong. "Thermal study of vulnerable atherosclerotic plaque." [College Station, Tex. : Texas A&M University, 2007. http://hdl.handle.net/1969.1/ETD-TAMU-2541.

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9

Lee, Regent. "Biomarkers of coronary atherosclerotic plaque rupture." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:7f0136bf-ad55-4dc8-bcc3-1d55cb269ef8.

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Coronary atherosclerotic plaque rupture is a critical event during atherosclerosis disease progression. Clinical consequences of atherosclerotic plaque rupture vary from asymptomatic to acute arterial thrombosis, yet the mechanisms underpinning such divergent biological response remain poorly understood. Novel biological signatures of plaque rupture will confer further insights into the dynamic responses triggered by plaque rupture event(s), and may provide alternative strategies for modulation of this prevalent disease. This thesis aims to investigate the events that accompany coronary plaque

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10

Sun, Binjian. "Multicontrast MRI of Atherosclerotic Plaques: Acquisition, Characterization and Reconstruction." Diss., Georgia Institute of Technology, 2007. http://hdl.handle.net/1853/16291.

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Cardiovascular Disease (CVD) continues to be the leading cause of death in western countries according to the statistics update by the American Heart Association. Atherosclerosis is estimated to be responsible for a large portion of CVD and affects 60 million people in the United States. Accurate diagnosis is crucial for proper treatment planning. Currently, the clinical standard screening technique for diagnosing atherosclerosis is x-ray angiography, which reveals the residual lumen size. X-ray angiographic images possess good resolution and contrast, however, lumen size is not always a prope

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11

Song, Hannah. "Endothelial bone morphogenic protein 4 and bone morphogenic protein receptor II expression in inflammation and atherosclerosis." Diss., Atlanta, Ga. : Georgia Institute of Technology, 2007. http://hdl.handle.net/1853/28258.

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Thesis (M. S.)--Biomedical Engineering, Georgia Institute of Technology, 2008.<br>Committee Chair: Hanjoong Jo; Committee Member: Ajit P. Yoganathan; Committee Member: Andrew P. Kowalczyk; Committee Member: David G. Harrison; Committee Member: Kathy K. Griendling

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12

Lee, Kelvin Sze Yen. "A genomic approach to atherosclerotic plaque vulnerability." Thesis, University of Newcastle Upon Tyne, 2012. http://hdl.handle.net/10443/1678.

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BACKGROUND: Macrophages in atherosclerotic plaques are believed to play a central role in plaque instability. However, the cell-specific molecular mechanisms involved are incompletely characterized. To address this, we performed genome-wide gene expression analyses using microarrays on RNA extracted from macrophage-rich regions of stable and unstable human atheromatous plaques. METHODS AND RESULTS: Plaques derived from carotid endarterectomy specimens (n=118) were designated as stable or unstable according to clinical, radiological and histopathological criteria. Macrophage-rich regions were e

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13

Moll, Robin John. "Detection of periodontal bacteria in atherosclerotic plaque." Thesis, University of Central Lancashire, 2016. http://clok.uclan.ac.uk/20464/.

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This study aims to examine the role infection plays in the pathogenesis of atherosclerosis through the molecular identification of latent bacterial species present in atherosclerotic plaque tissue from the carotid artery. Immunohistological examination of the atherosclerotic plaque tissue revealed widespread localisation of the bacterial cell wall constituent peptidoglycan (PGN). Targeted PCR amplification of the 16S rRNA gene permitted identification of a diverse collection of 16S rDNA sequences within atherosclerotic plaque samples. Thirteen of 21 (61.9%) plaques contained bacterial 16S rDNA

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14

Salem, Murtaza Karimjee. "An investigation into carotid atherosclerotic plaque instability." Thesis, University of Leicester, 2014. http://hdl.handle.net/2381/28979.

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Stroke is the leading cause of death and permanent neurological disability in the developed world and a significant burden on the NHS and wider economy. A third of all strokes are caused by thrombo-embolism from unstable carotid atherosclerotic plaques. The exact pathogenesis of plaque progression and instability is unknown. The aim of this thesis was to investigate carotid plaque instability from a clinical perspective and on a molecular level. Patients with spontaneous embolisation detected during Transcranial Doppler (TCD) monitoring were significantly more likely to have recent symptoms an

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15

Kojima, Yoji. "Progranulin Expression in Advanced Human Atherosclerotic Plaque." Kyoto University, 2009. http://hdl.handle.net/2433/124322.

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16

Flavall, Elizabeth A. "Localisation of antioxidants and oxidative markers within the atherosclerotic plaque." Thesis, University of Canterbury. Biological Sciences, 2008. http://hdl.handle.net/10092/1941.

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Atherosclerosis is a complex inflammatory disease in which oxidative stress is a major protagonist in the development and progression of the atherosclerotic plaque. All biochemical analysis studies of plaque over the past fifteen years have been carried out on whole plaque with no attempt to localise sites of differing biochemical conditions. This study set out to identify in oxidation levels and inflammatory markers in relation to spatial localisation within the plaque. Advanced plaque samples removed during endarectomy were obtained from the Christchurch Hospital Department of surgery and we

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17

White, Gemma. "The role of fractalkine (CX₃CL1) and its receptor (CX₃CR1) in vascular biology." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.670108.

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18

Arumuganainar, Ponnappan. "Automatic soft plaque detection from CTA." Thesis, Atlanta, Ga. : Georgia Institute of Technology, 2008. http://hdl.handle.net/1853/26690.

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Thesis (M. S.)--Biomedical Engineering, Georgia Institute of Technology, 2009.<br>Committee Chair: Tannenbaum, Allen; Committee Member: Skrinjar, Oskar; Committee Member: Yezzi, Anthony. Part of the SMARTech Electronic Thesis and Dissertation Collection.

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19

Molloy, Kevin John. "The role of collagenases in atherosclerotic plaque instability." Thesis, University of Leicester, 2004. http://hdl.handle.net/2381/29472.

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The primary aim of this thesis was to quantify the collagenase concentrations in carotid plaques and to relate them to markers to plaque instability.;Recent studies have shown that strain therapy decreases cardiovascular risk, even in patients with normal cholesterol levels. A further aim of this thesis was to observe the effects of statins on clinical and biochemical indicators of plaque instability.;Atherosclerotic plaques were collected from 159 patients undergoing carotid endarterectomy. The presence and timing of carotid territory symptoms was ascertained. Pre-operative embolisation was r

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20

Jaw, Jen Erh. "Intratracheal lipopolysaccharide exposure induced atherosclerotic plaque destabilization : a novel murine plaque vulnerability model." Thesis, University of British Columbia, 2016. http://hdl.handle.net/2429/58326.

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Rationale: Literature has implicated lung inflammation as a risk factor for acute cardiovascular (CV) diseases (CVD), but the underlying mechanisms linking lung injury with CVD are largely unknown. Plasma neutrophilic myeloperoxidase (MPO) is an inflammatory biomarker for acute CVD, but its pathological role in CVD is unclear. Hypothesis: A murine model of atheromatous plaque vulnerability can be established by intratracheal Lipopolysaccharide (LPS) exposure. Neutrophils and MPO may contribute to this process. Approaches: LPS (3 mg/kg) or saline (control) was instilled directly into the lun

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21

Schapira, Kitty Brigitte. "Atherosclerotic plaque stability: identification and validation of key regulators." [Maastricht] : Maastricht : Universitaire Pers Maastricht ; University Library, Universiteit Maastricht [host], 2007. http://arno.unimaas.nl/show.cgi?fid=9083.

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22

Golemati, Spiretta. "Motion analysis of atherosclerotic plaque from b-mode ultrasound." Thesis, Imperial College London, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.396015.

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23

Zainon, Rafidah Binti. "Spectral Micro-CT Imaging of Ex Vivo Atherosclerotic Plaque." Thesis, University of Canterbury. Physics and Astronomy, 2012. http://hdl.handle.net/10092/7165.

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The goal of this research was to demonstrate the potential of spectral CT for the discrimination of vulnerable atherosclerotic plaques. It was proposed that spectral CT has the potential to identify the presence of specific markers for vulnerable plaques: iron deposits and lipid core. A spectral micro-CT system incorporating the latest Medipix spectroscopic photon- counting detectors was commissioned for this purpose. Using spectroscopic methods developed with this system, it was possible to distinguish the presence of iron deposits and lipid core within ex vivo atherosclerotic plaques. Athero

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24

Weert, Thomas de. "Atherosclerotic carotid plaque assessment with multidetector computed tomography angiography." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2009. http://hdl.handle.net/1765/15123.

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25

Fukumitsu, Ryu. "Expression of Vasohibin-1 in Human Carotid Atherosclerotic Plaque." Kyoto University, 2017. http://hdl.handle.net/2433/226775.

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26

Chihara, Hideo. "In vivo detection of atherosclerotic plaque using non-contact and label-free near-infrared hyperspectral imaging." Kyoto University, 2016. http://hdl.handle.net/2433/217737.

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27

Bashiardes, Evy. "Gene polymorphisms, gene expression and atherosclerotic plaques." Thesis, Imperial College London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.420882.

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28

Biasiolli, Luca. "In-vivo MRI characterization of atherosclerotic plaques." Thesis, University of Oxford, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558196.

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Acute ischemic events associated with atherosclerosis are most often caused by rupture or erosion of unstable plaques. Clinical studies have demonstrated that in-vivo multi- contrast MRI can characterize plaque morphology and composition to evaluate the vulnerability of atherosclerotic plaques. The standard protocol for carotid imaging uses the Double-Inversion-Recovery (DIR) Fast-Spin-Echo (FSE) pulse sequence to acquire black-blood 2D high-resolution cross-sectional T\W, PDW and T2W images. With the addition of bright-blood Time-of-Flight images, it was demonstrated that in-vivo multi- contr

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29

Douglas, Graeham Rees. "Fibre microstructure and mechanics of atherosclerotic plaques." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/271511.

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Atherosclerosis is characterised by the progressive growth of a plaque, where a fibrous cap covers a lipid-rich core. Rupture of this fibrous cap can lead to thrombosis and a heart attack or stroke. This dissertation considered the role of plaque microstructure in the mechanics and rupture risk of atherosclerotic plaques. Fibre structures in human atherosclerotic plaques were characterised through scanning electron microscopy, histology, and image processing. Local primary fibre orientation and the fibre dispersion were calculated. Plaque shoulder regions, where rupture is most frequent, had h

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30

Campbell, Ian Christopher. "Plaque erosion and murine plaque stability: a biomechanical examination of exceptions to the phenomenon of plaque rupture." Diss., Georgia Institute of Technology, 2013. http://hdl.handle.net/1853/47741.

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Atherosclerotic plaque disruption leading to thrombosis has traditionally been studied as a rupture of a thin fibrous cap over a lipid-laden necrotic core. However, two noteworthy categories of plaques that do not rupture have presented themselves: 1) in mice, plaque rupture is rare if not absent, and 2) in humans, some plaques erode and form a thrombus without rupturing. Current understanding of the biomechanical differences between plaques that rupture and those that do not is incomplete. In this research, we used patient-specific computational biomechanics tools to study differences among t

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31

TEUMA-MELAGO, Eric. "A FLUID STRUCTURE INTERACTION MODEL OF INTRACORONARY ATHEROSCLEROTIC PLAQUE RUPTURE." Doctoral diss., University of Central Florida, 2006. http://digital.library.ucf.edu/cdm/ref/collection/ETD/id/2359.

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Plaque rupture with superimposed thrombosis is the primary cause of acute coronary syndromes of unstable angina, myocardial infarction and sudden death. Although intensive studies in the past decade have shed light on the mechanism that causes unstable atheroma, none has directly addressed the clinical observation that most myocardial infarction (MI) patients have moderate stenoses (less than 50%). Considering the important role the arterial wall compliance and pulsitile blood flow play in atheroma rupture, fluid-structure interaction (FSI) phenomenon has been of interest in recent studies. In

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32

Yuan, Jianmin. "Three-dimensional quantitative magnetic resonance imaging of carotid atherosclerotic plaque." Thesis, University of Cambridge, 2017. https://www.repository.cam.ac.uk/handle/1810/267723.

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Stroke is one of the leading causes of death and disability worldwide with 20% of ischemic strokes attributed to carotid atherosclerosis. In recent years, morphological characteristics of atherosclerotic plaque such as a thin fibrous cap, large lipid-rich necrotic core, intraplaque haemorrhage and ulceration have shown correlations with subsequent clinical events. High resolution, multi-contrast magnetic resonance imaging (MRI) can qualitatively identify these features and monitor disease progression. Compared to traditional contrast weighted imaging, quantitative MRI could provide an objectiv

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33

Huang, Yuan. "Material fatigue as a possible mechanism of atherosclerotic plaque rupture." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608011.

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34

Rudd, James H. F. "Imaging atherosclerotic plaque inflammation with [18F]- fluorodeoxyglucose positron emission tomography." Thesis, University of Cambridge, 2003. https://www.repository.cam.ac.uk/handle/1810/243719.

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Inflammation is important in both the pathogenesis and outcome of atherosclerosis. Plaques containing numerous inflammatory cells, particularly macrophages, have a high risk of rupture, whereas those with fewer inflammatory cells are at lower risk. The current ‘gold standard’ technique for imaging atherosclerosis is x-ray contrast angiography, which provides high-resolution definition of the site and severity of luminal stenoses, but no information about plaque inflammation. Quantification of plaque inflammation is desirable both to predict risk of plaque rupture and to monitor the effects of

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35

Gerbaud, Edouard. "Multi-modality imaging of atherosclerotic plaque using optical coherence tomography." Thesis, Bordeaux, 2016. http://www.theses.fr/2016BORD0132/document.

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Les technologies d'imagerie intravasculaire c’est à dire l’échographie endo-coronaire (IVUS)et la tomographie par cohérence optique (IV-OCT) sont des outils précieux pour aider audiagnostic de la plaque d’athérosclérose et guider les gestes thérapeutiques.Le chapitre 1 présente les inconvénients de la coronarographie dans la pratique clinique.Dans plusieurs circonstances, l’IVUS et l’OCT ont clairement une valeur supplémentaire quandils sont utilisés comme un outil diagnostique en cas d'ambiguïtés angiographiques. Nousavons récemment rapporté la première observation utilisant l’OCT pour décrir

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36

Khan, Tania Nur. "Design and Performance of a Localized Fiber Optic, Near-Infrared Spectroscopic Prototype Device for the Detection of the Metabolic Status of "Vulnerable Plaque": in-vitro Investigation of Human Carotid Plaque." Digital WPI, 2003. https://digitalcommons.wpi.edu/etd-dissertations/11.

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INTRODUCTION: The“vulnerable plaque" is defined as the“precursor lesion" that ultimately ends in acute coronary thrombi (clots) that create a heart attack. Macrophages and inflammatory cells, found preferentially in vulnerable plaque, sustain their activity in the plaque through anaerobic metabolism and lactate production. The ultimate goal is to assess anaerobic metabolism in-vivo by measuring tissue pH and lactate concentration in atherosclerotic plaques using optical spectroscopy. The proposed in-vitro optical probe design, experimental method, and spectroscopic data analysis methodology a

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37

Miyachi, Shigeru, Takashi Izumi, Noriaki Matsubara, Osamu Hososhima, Yuko Tsurumi, and Arihito Tsurumi. "Virtual Histology Analysis of Carotid Atherosclerotic Plaque: Plaque Composition at the Minimum Lumen Site and of the Entire Carotid Plaque." Wiley-Blackwell, 2013. http://hdl.handle.net/2237/17694.

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38

Zhu, Ting F. (Ting Fredrick). "Estimation of nonlinear mechanical properties of atherosclerotic plaques." Thesis, Massachusetts Institute of Technology, 2005. http://hdl.handle.net/1721.1/33907.

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Thesis (S.M.)--Massachusetts Institute of Technology, Dept. of Mechanical Engineering, 2005.<br>Includes bibliographical references (leaves 57-59).<br>A numerical method has been developed to estimate the mechanical properties of atherosclerotic plaques by combining genetic algorithm with finite element methods. Plaque images derived from optical coherence tomography were employed to construct finite element models which were subsequently used in conjunction with a genetic algorithm to determine the parameters in a nonlinear constitutive model. A new multi-frame scheme is introduced to better

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39

Sun, Binjian. "Multicontrast MRI of atherosclerotic plaques acquistion, characterization and reconstruction /." Available online, Georgia Institute of Technology, 2007, 2007. http://etd.gatech.edu/theses/available/etd-06192007-160035/.

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Thesis (Ph. D.)--Biomedical Engineering, Georgia Institute of Technology, 2008.<br>Robert C. Long, Jr., Committee Member ; Raymond P. Vito, Committee Member ; W. Robert Taylor, Committee Member ; John N. Oshinski, Committee Co-Chair ; Don P. Giddens, Committee Chair.

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40

Laskar, Amit. "Cell response to imaging contrast agents suggested for atherosclerotic plaque imaging." Doctoral thesis, Linköpings universitet, Experimentell patologi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-92003.

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Oxysterols are the major cytotoxic components of oxidized low-density lipoprotein (OxLDL) that accumulate in atherosclerotic plaques. Their uptake by macrophages ensue foam cell formation, atherogenesis and plaque progression. Magnetic resonance imaging (MRI) has grown as a modality to track such intra-plaque developments by using intracellular contrast agents. The focus of this study was to evaluate the effects of two contrast agents; manganese based mangafodipir (TeslascanTM) and iron based super-paramagnetic iron oxide nanoparticles (SPION, ResovistTM) on cell functions and examined their i

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41

Pugh, Karen. "Protease-activated receptor-1 as potential target in atherosclerotic plaque stabilisation." Thesis, University of Strathclyde, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.431780.

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42

Carson, Kevin George Stephen. "Pathways of atherosclerotic plaque destabilisation and rupture in an animal model." Thesis, University of Bristol, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.431821.

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43

Jenkins, Nicholas Paul. "Metalloproteinases and foam-cell macrophage phenotypes in atherosclerotic plaques." Thesis, University of Bristol, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.535202.

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44

Sim, Alisia Mara. "Detection of calcification in atherosclerotic plaques using optical imaging." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/33151.

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PET imaging, using the bone tracer Na18F, allows the non-invasive location of atherosclerotic plaques that are at risk of rupture. However, the spatial resolution of PET is only 4-5 mm, limiting the mechanistic information this technique can provide. In this thesis, the use of fluorescence and Raman imaging to elucidate the mechanism of micro-calcification within atherosclerotic plaques has been investigated. A number of fluorescent probes to detect fluoride and calcium have been synthesised. One of the fluoride probes has been shown to be selective for fluoride however, the concentration of f

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45

Dawson, Andrew. "Carotid atherosclerotic plaque instability as assessed by texture analysis of ultrasonic images." Thesis, McGill University, 2013. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=114382.

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BACKGROUND: Internal carotid artery stenosis is a major etiological factor causing cerebrovascular events. Carotid endarterectomy (CEA) has been shown to reduce the risk of future cerebrovascular events. It is increasingly recognized that in addition to stenosis, plaque morphology plays an important role in determining plaque stability. The evolution of imaging modalities such as ultrasound and of computer-assisted image analysis has led to the development of objective, reproducible methods to assess plaque morphology. Previous studies have identified differences in plaque morphology between a

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46

Mawhin, Marie-Anne. "Role of neutrophils and leukotrienes in atherosclerotic plaque destabilisation : implication of endotoxemia." Thesis, Strasbourg, 2017. http://www.theses.fr/2017STRAJ034/document.

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La déstabilisation de la plaque d’athérosclérose reste de nos jours un problème majeur, malgré les progrès récents dans sa compréhension. Les neutrophiles sont des acteurs puissants de l’immunité innée capables d’altérer les plaques. Un chimio-attractant majeur des neutrophiles, le leucotriène B4, pourrait être un des contributeurs potentiels de la déstabilisation des plaques en particulier dans l’endotoxémie, elle-même associée aux accidents cardiovasculaires. L’objectif de ce travail a été de définir le rôle du leucotriène B4 dans l’attraction des neutrophiles dans la plaque au cours de l’en

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47

Baba, Osamu. "MicroRNA-33 Deficiency Reduces the Progression of Atherosclerotic Plaque in ApoE-/- Mice." Kyoto University, 2014. http://hdl.handle.net/2433/188657.

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48

Masoodzadehgan, Nazanin. "PET/MR imaging of atherosclerotic plaque and tumor using dual modality SPIOS." Diss., Georgia Institute of Technology, 2014. http://hdl.handle.net/1853/54253.

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Early stage disease diagnosis still remains a challenge despite much efforts to develop novel imaging and diagnostic techniques. Nanoparticles used as molecular imaging contrast agents with multifunctionality and flexibility provide a platform for targeting the specific disease biomarkers and integration of imaging modalities. In this work, we developed a simplified method for synthesis of radiolabeled targeted super paramagnetic iron oxide nanoparticles (SPIOs). This method takes advantage of the chelator BAT that is conjugated to the PEG before the coating process begins. The effect of nanop

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49

Jashari, Fisnik. "Carotid artery disease : plaque features and vulnerability." Doctoral thesis, Umeå universitet, Kardiologi, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-111048.

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Background: Atherosclerosis is an important cause of stroke. Ultrasound offers the convenience of real-time and detailed assessment of carotid plaque features as well as arterial wall thickening and composition. Evaluation of these features is important for determining patients’ risk of suffering vascular events and also contributes to selecting the best treatment strategy. Methods: Using ultrasound data analysis we have determined plaque features in the bifurcation and internal carotid artery (ICA), including: surface plaque irregularities, calcification, echogenicity (grey scale median-GSM)

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50

Bicknell, Colin David. "Embolic potential of human carotid atherosclerotic plaques during endoluminal intervention." Thesis, Imperial College London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.422309.

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