Relevant bibliographies by topics / Atrial fibrillation

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Atrial fibrillation'

Author: Grafiati
Published: 4 June 2021
Last updated: 28 September 2024

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Journal articles on the topic "Atrial fibrillation"

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S., Sinha,, Kumari, S., and Mishra, K. "Molecular Docking Study of Crocetin with Interleukin–18 for the Treatment of Atrial Fibrillation." CARDIOMETRY, no. 24 (November 30, 2022): 379–84. http://dx.doi.org/10.18137/cardiometry.2022.24.379384.

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The most frequent arrhythmias are atrial fibrillation. Although the mechanisms underlying are not fully understood, they encompass postoperative and intraoperative phenomena like cardiac ischemia sympathetic activation and inflammation that ends up causing atrial fibrillation. Atrial fibrillation is frequently associated with the presence of pre-existing factors, making the atria susceptible to atrial fibrillation. There are various treatments have been used to manage the condition. However, they are sometimes ineffective, costly, and have side effects stressing the need to explore alternative medicine. The alternative medicine which is now being explored for treating a variety of conditions is primarily involving botanical sources. However, the present research aims at exploring the potential targeting ability of crocetin, a phytochemical from saffron against Interleukin-18 which is an emerging target for atrial fibrillations using a computational approach with Autodock4. The results of the study revealed binding energy of -7.01 Kcal/mol for the most favorable docked confirmation which further suggests the potential of phytocompounds from saffron in treating and managing atrial fibrillation.
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Hulsmans, Maarten, Maximilian J. Schloss, I.-Hsiu Lee, Aneesh Bapat, Yoshiko Iwamoto, Claudio Vinegoni, Alexandre Paccalet, et al. "Recruited macrophages elicit atrial fibrillation." Science 381, no. 6654 (July 14, 2023): 231–39. http://dx.doi.org/10.1126/science.abq3061.

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Atrial fibrillation disrupts contraction of the atria, leading to stroke and heart failure. We deciphered how immune and stromal cells contribute to atrial fibrillation. Single-cell transcriptomes from human atria documented inflammatory monocyte and SPP1 + macrophage expansion in atrial fibrillation. Combining hypertension, obesity, and mitral valve regurgitation (HOMER) in mice elicited enlarged, fibrosed, and fibrillation-prone atria. Single-cell transcriptomes from HOMER mouse atria recapitulated cell composition and transcriptome changes observed in patients. Inhibiting monocyte migration reduced arrhythmia in Ccr2 −∕− HOMER mice. Cell-cell interaction analysis identified SPP1 as a pleiotropic signal that promotes atrial fibrillation through cross-talk with local immune and stromal cells. Deleting Spp1 reduced atrial fibrillation in HOMER mice. These results identify SPP1 + macrophages as targets for immunotherapy in atrial fibrillation.
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Wu, Shaohui, Guangchen Zou, Xu Liu, Weifeng Jiang, Mu Qin, and Daoliang Zhang. "Key Role of Left Atrial Appendage during Redo Ablation in a Case of Long-Standing Persistent Atrial Fibrillation." Case Reports in Cardiology 2020 (June 19, 2020): 1–4. http://dx.doi.org/10.1155/2020/9691584.

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Extrapulmonary vein focal sources have been recognized as the source of atrial fibrillation in some cases, and empiric electric isolation of the left atrial appendage has been proposed for long-standing persistent atrial fibrillation by some. Here, we present a case of redo ablation of long-standing persistent atrial fibrillation in which the left atrial appendage played a key role in maintaining AF during ablation, and atrial fibrillation was terminated by electrical isolation of the LAA. During the ablation, a rare phenomenon of half of the atria in atrial fibrillation while the other half of the atria in atrial flutter was seen.
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Lu, Zhibing, Benjamin J. Scherlag, Jiaxiong Lin, Guodong Niu, Kar-Ming Fung, Lichao Zhao, Muhammad Ghias, et al. "Atrial Fibrillation Begets Atrial Fibrillation." Circulation: Arrhythmia and Electrophysiology 1, no. 3 (August 2008): 184–92. http://dx.doi.org/10.1161/circep.108.784272.

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Wijffels, Maurits C. E. F., Charles J. H. J. Kirchhof, Rick Dorland, and Maurits A. Allessie. "Atrial Fibrillation Begets Atrial Fibrillation." Circulation 92, no. 7 (October 1995): 1954–68. http://dx.doi.org/10.1161/01.cir.92.7.1954.

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Medeiros de Vasconcelos, J. Tarcísio. "Obesidade e Fibrilação Atrial." Journal of Cardiac Arrhythmias 32, no. 3 (January 17, 2020): 153–54. http://dx.doi.org/10.24207/jca.v32i3.984_pt.

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A fibrilação atrial ocupa uma discussão central hoje no universo das arritmias cardíacas. O aumento da expectativa de vida que trouxe um substancial aumento de sua prevalência na população geral1 implicou em um aprofundamento marcante na compreensão dos seus mecanismos eletrofisiológicos, na identificação de fatores determinantes ou de potencialização da sua ocorrência e obviamente no desenvolvimento de estratégias efetivas de tratamento e de prevenção das suas complicações. Desde o clássico estudo de Wijffels et al., publicado em 1995 (Atrial Fibrillation Begets Atrial Fibrillation)2 demonstrando experimentalmente que frequências atriais artificialmente impostas ao miocárdio atrial implicam em mudanças eletrofisiológicas marcantes determinantes da própria ocorrência da fibrilação atrial, ficou claro que a arritmia uma vez iniciada potencializa a sua própria ocorrência e um processo constante de retroalimentação, cujo desfecho final ao longo do tempo é a instalação da arritmia em uma forma permanente. Contudo para que a fibrilação atrial ocorra é necessária a presença de um ambiente eletrofisiológico adequado, consequente à presença de diversos elementos que agridem o miocárdio atrial sob o aspecto elétrico e estrutural.
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Adeyana, Sri, Haryadi Haryadi, and Chandra Wijaya. "Hubungan Kejadian Fibrilasi Atrium dengan Diameter Atrium Kiri pada Fibrilasi Atrium Valvular dan Fibrilasi Atrium Non-Valvular Di RSUD Arifin Achmad." Jurnal Ilmu Kedokteran 11, no. 1 (March 15, 2018): 31. http://dx.doi.org/10.26891/jik.v11i1.2017.31-38.

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Atrial Fibrillationis a kind of arrhythmia which has the most incidence. Based on its Etiology, atrial fibrillationcouldbe divided in to two, valvular and nonvalvular atrial fibrillation. This study was aimed toknow the correlation ofatrial fibrillation incidence between valvular and nonvalvular with its left atrium diameter in Arifin Achmad ProvinsiRiau’s General Hospital. This study was analytical and done by cross sectional approach with 185 patient. The datawere processing with computerize to univariate analysis and chi-square for bivarite analysis. From this study it can beconcluded that the most occurrence of atrial fibrillation was non valvular atrial fibrillation which was 76,8% with theetiology mostly of hypertension which was 41,5%. Valvular atrial fibrillation was mostly caused by mitral stenosiswhich was 37,2% and there were no correlation between the diameter of left atrium to the occurrence of valvular andnon valvular atrial fibrillation (p=0,273.)
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Yim, Jeffrey, and Andrew D. Krahn. "Postoperative Atrial Fibrillation Begets Atrial Fibrillation." JACC: Clinical Electrophysiology 10, no. 7 (July 2024): 1720–21. http://dx.doi.org/10.1016/j.jacep.2024.06.009.

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Algalarrondo, Vincent, and Fabrice Extramiana. "Autoimmune Atrial Fibrillation or Atrial Fibrillation–Induced Autoimmunity? A New Atrial Fibrillation Begets Atrial Fibrillation Pathway?" Circulation 148, no. 6 (August 8, 2023): 499–501. http://dx.doi.org/10.1161/circulationaha.123.063672.

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Opie, Lionel H. "Tedisamil in Coronary Disease: Additional Benefits in the Therapy of Atrial Fibrillation?" Journal of Cardiovascular Pharmacology and Therapeutics 8, no. 1_suppl (March 2003): S33—S37. http://dx.doi.org/10.1177/107424840300800105.

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Atrial fibrillation has recently come into clinical and research focus. In particular, ventricular rate control has been carefully compared with atrial rhythm control. Additionally, the recent discovery of atrial stunning has initiated clinical and research interest in atrial remodeling. Atrial fibrillation is more likely to occur when the atria are damaged by increased fibrosis. The ideal way to prevent atrial fibrillation and the risk of repetition is by tackling the root causes, such as ischemic heart disease, heart failure, and left ventricular hypertrophy. Tedisamil is an unusual antifibrillatory compound that has a novel mechanism of action by inhibiting the transient outward current (Ito) and the repolarizing potassium currents in the sinoatrial node. Tedisamil works acutely against atrial fibrillation. Importantly, atrial fibrillation is often caused by or related to cardiac ischemia, and conversely, ischemia is caused by the increased oxygen demand of atrial fibrillation. Hence, the double properties of tedisamil as a drug that both inhibits atrial fibrillation and acts in an anti-ischemic mode are an attractive basis for future clinical research.
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Dissertations / Theses on the topic "Atrial fibrillation"

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Donate, Puertas Rosa. "Omic approach to atrial fibrillation." Thesis, Lyon, 2017. http://www.theses.fr/2017LYSE1164.

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La fibrillation atriale (FA) est un problème de santé publique majeur dans le monde entier. Le remodelage électrique, structurel et neuronal est sous-jacent à la myopathie atriale. La pharmacothérapie actuelle est souvent inefficace en raison du manque de connaissance de la pathophysiologie de la FA.Pour comprendre comment se réalise le remodelage atrial, une approche Omique qui explore le transcriptome, l'épigénome (méthylome et microOme) et le génome de patients atteints de FA a été réalisée. Parallèlement, le phénotype de vieux rats spontanément hypertendus (SHRs) a été caractérisé et une étude pharmacologique avec la décitabine (5-Aza-2'-deoxycitidine) a été menée. Les patients atteint de FA présentent un profil transcriptomique et d'expression de miRNA alteré dans l'oreillete gauche (OG), soulignant le rôle important d'un processus de "œmorphogénèse de la structure anatomique". L'expression réduite de Pitx2 était inversement corrélée à la taille de l'OG et ne pouvait pas être expliquée ni par le facteur de transcription ni par la surexpression de Smyd2, une cible de miR-519b. Les SHRs, similairement aux observations chez l'homme, ont développé des arythmies dépendantes de l'âge associées au remodelage atrial et ventriculaire gauche. La FA a été trouvée associée à l'hyperméthylation du promoteur de Pitx2 à la fois chez l'homme et chez les SHRs. L'agent hyperméthylant décitabine a amélioré le profil arhytmique de l'ECG et les activités SOD, et la réduction de la fibrose dans le ventricule gauche des SHRs. En utillisant une approche NGS basée sur un panel personnalisé de 55 gènes candidats à la myopathie atriale dans une cohorte de 94 patients atteints de FA, 11 nouvelles variantes faux-sens potentiellement pathogènes impliqués dans le remodelage structurel ont été identifiés. Des études fonctionnelles de ces variants ont débuté. Trois patients sont également des porteurs de variantes dans les gènes connus de FA. Les résultats actuels suggèrent que 1) la régulation épigénétique peut jouer un rôle dans la pathophysiologie de la FA 2) les agents hypométhylants doivent être considérés comme une nouvelle thérapie de la FA 3) une approche Omique peut aider à découvrir de nouveaux mécanismes sous-jacents à la myopathie atriale
Atrial fibrillation (AF) is a major public health care problem worldwide. Electrical, structural, and neural remodeling underlie atrial myopathy. Current pharmacotherapy is often ineffective due to the lack of knowledge of AF pathophysiology. To understand how atrial remodeling occurs, an Omic approach that explore the transcriptome, epigenome (methylome and microOme) and genome of AF patients was performed. In parallel, ageing spontaneously hypertensive rats (SHRs) were phenotypically characterised and a pharmacological study with decitabine (5-Aza-2’-deoxycitidine) was conducted. AF patients presented an altered transcriptomic and microRNA expression profile in the left atria (LA), emphasizing the important role of an "anatomical structure morphogenesis" process. The Pitx2 reduced expression was inversely correlated with LA size, and could not be explained by transcriptor factor. Smyd2 is a target of miR-519b-3p. SHRs, similar to what is observed in humans, developed age-dependent arrhythmias associated with left atrial and ventricular remodeling. AF was found to be associated with Pitx2 promoter hypermethylation both in humans and in SHRs. The hypomethylating agent decitabine improved ECG arrhythmic profiles and superoxide dismutase activities, and reduced fibrosis in the left ventricle of SHRs. Using a next-generation sequencing approach based on a custom panel of 55 atrial myopathy candidate genes in a cohort of 94 AF patients, 11 novel potentially pathogenic missense variants involved in structural remodeling were identified. Functional studies of these variants have started. Three patients were also carriers of variants in known AF-causing genes. The present results suggest that 1) epigenetic regulation may play a role in the pathophysiology of AF 2) hypomethylating agents have to be considered as a new AF therapy 3) an Omic approach may help to uncover new mechanisms underlying atrial myopathy
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Wijffels, Maurits Christoffel Emile Franciscus. "Atrial fibrillation begets atrial fibrillation an experimental study in chronically instrumented goats /." Maastricht : Maastricht : Universiteit Maastricht ; University Library, Maastricht University [Host], 1996. http://arno.unimaas.nl/show.cgi?fid=6751.

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Vaizurs, Raja Sarath Chandra Prasad. "Atrial Fibrillation Signal Analysis." Scholar Commons, 2011. http://scholarcommons.usf.edu/etd/3386.

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Atrial fibrillation (AF) is the most common type of cardiac arrhythmia encountered in clinical practice and is associated with an increased mortality and morbidity. Identification of the sources of AF has been a goal of researchers for over 20 years. Current treatment procedures such as Cardio version, Radio Frequency Ablation, and multiple drugs have reduced the incidence of AF. Nevertheless, the success rate of these treatments is only 35-40% of the AF patients as they have limited effect in maintaining the patient in normal sinus rhythm. The problem stems from the fact that there are no methods developed to analyze the electrical activity generated by the cardiac cells during AF and to detect the aberrant atrial tissue that triggers it. In clinical practice, the sources triggering AF are generally expected to be at one of the four pulmonary veins in the left atrium. Classifying the signals originated from four pulmonary veins in left atrium has been the mainstay of signal analysis in this thesis which ultimately leads to correctly locating the source triggering AF. Unlike many of the current researchers where they use ECG signals for AF signal analysis, we collect intra cardiac signals along with ECG signals for AF analysis. AF Signal collected from catheters placed inside the heart gives us a better understanding of AF characteristics compared to the ECG. . In recent years, mechanisms leading to AF induction have begun to be explored but the current state of research and diagnosis of AF is mainly about the inspection of 12 lead ECG, QRS subtraction methods, spectral analysis to find the fibrillation rate and limited to establishment of its presence or absence. The main goal of this thesis research is to develop methodology and algorithm for finding the source of AF. Pattern recognition techniques were used to classify the AF signals originated from the four pulmonary veins. The classification of AF signals recorded by a stationary intra-cardiac catheter was done based on dominant frequency, frequency distribution and normalized power. Principal Component Analysis was used to reduce the dimensionality and further, Linear Discriminant Analysis was used as a classification technique. An algorithm has been developed and tested during recorded periods of AF with promising results.
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Anjos, Diana Sofia Pereira dos. "Ablation of atrial fibrillation." Master's thesis, Instituto de Ciências Biomédicas Abel Salazar, 2010. http://hdl.handle.net/10216/62150.

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Anjos, Diana Sofia Pereira dos. "Ablation of atrial fibrillation." Dissertação, Instituto de Ciências Biomédicas Abel Salazar, 2010. http://hdl.handle.net/10216/62150.

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An, Yoshimori. "Causes of death in Japanese patients with atrial fibrillation: The Fushimi Atrial Fibrillation Registry." Doctoral thesis, Kyoto University, 2020. http://hdl.handle.net/2433/245817.

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Scholten, Marcoen Franciscus. "Topics in atrial fibrillation management." [S.l.] : Rotterdam : [The Author] ; Erasmus University [Host], 2006. http://hdl.handle.net/1765/7636.

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Park, Helen Loreen. "Atrial fibrillation and cognitive impairment." Thesis, University of Newcastle Upon Tyne, 2004. http://hdl.handle.net/10443/765.

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Background In our aging population the burden of dementia is increasing, necessitating the urgent identification of treatable risk factors. Small cross-sectional studies demonstrate associations between nonvalvular atrial fibrillation (NVAF), silent cerebral infarction and decreased cognitive function, but there are few longitudinal studies in this area. This thesis reports the results of a prospective longitudinal cohort study of cognitive decline in people with recent-onset NVAF compared to controls. To inform the thesis, an extensive literature review was undertaken . This included searches on NVAF and cognitive decline, NVAF and silent infarction, epidemiology of NVAF, other risk factors for cognitive decline, epidemiology of cognitive decline and the neuropsychological tests included in the CAFE battery. Methods 362 people over 60, screenedi n primary care, underwent baseline assessmenitn cluding a battery of neuropsychological tests, repeated at 12 months (n=304). Cases (n=175) with recent-onset NVAF, were matched for age, sex and GP practice with controls in sinus rhythm. Data were compared using SPSS software (version 11) with both parametric and non-parametric analysis. Results Baseline characteristics, including cognitive function, were similar for cases and controls. There was wide variation between individuals in change in performance on the neuropsychological tests over 12 months, with some improving and some deteriorating for each sub-test. Cases (NVAF) significantly (p<0.05) deteriorated in four subtests measuring attention/ non-verbal memory, and significantly (p<0.05) improved in two subtests measuring verbal memory. Controls significantly (p<0.05) deteriorated and improved in the same sub-tests as cases, but significantly (p<0.05) deteriorated in another three subtests measuring attention/non-verbal memory, and significantly (p<0.05) improved in another six subtests. Treatment with warfarin or aspirin did not appear to be associated with change in cognitive status. Conclusions At baseline there was no significant difference in cognitive function between cases in NVAF and controls in sinus rhythm. At follow-up there was no consistent relationship between NVAF and cognitive decline over 12 months, nor any apparent effect of antithrombotic therapy. Explanations include true independence of NVAF and cognitive decline, or too short a follow-up period. An additional follow-up at 36 months is underway to explore this further.
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Marrouf, Nedal. "Mitochondrial DNA in atrial fibrillation." Thesis, St George's, University of London, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.415671.

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Själander, Sara. "Stroke prevention in atrial fibrillation." Doctoral thesis, Umeå universitet, Medicin, 2016. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-124951.

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Background: The Framingham Study from 1991 showed a clear correlation between atrial fibrillation (AF) and ischemic stroke, where patients with AF had an almost fivefold increase in risk of stroke compared with patients without AF. Since then, several trials have evaluated different antithrombotic treatments to reduce the risk of stroke in patients with AF. Other trials have investigated factors that increase the risk of stroke in patients with AF and risk score systems have been developed to categorize patients into low or increased risk of stroke to help clinicians to decide which patients benefit from antithrombotic treatment and in whom it can be abstained, not to expose patients with low stroke risk to an increased risk of bleeding conferred by antithrombotic treatment. The aims of this thesis were: [1] to evaluate if a warfarin dosing algorithm can increase hit rate and decrease mean error compared with manually changed doses; [2] to assess the prevalence and net clinical benefit of aspirin as monotherapy for stroke prevention in AF; [3] to investigate the risk of thromboembolic and haemorrhagic complications within 30 days after electrical cardioversion (ECV) of AF in patients with and without oral anticoagulation (OAC) pre-treatment; and [4] to assess the proportion of patients discontinuing OAC after pulmonary vein isolation (PVI), identify factors predicting stroke after PVI and to investigate risk of complications after PVI with and without OAC. Materials and methods: All studies are retrospective and based on data from Swedish national quality registries. In paper I, data from Auricula was used to compare the resulting INR values after algorithmic warfarin dose suggestions and manually changed doses. In paper II data was extracted from the Swedish National Patient Register, the Dispensed Drugs Register and the Cause of Death Register. Patients with aspirin treatment were compared with patients without any antithrombotic treatment regarding risk of thromboembolic and haemorrhagic complications. In paper III data was collected from the Swedish National Patient Register and the Dispensed Drugs Register to examine risk of complications (thromboembolic and haemorrhagic events) within 30 days after cardioversion, comparing patients with and without oral anticoagulation pre-treatment. In paper IV data from six different Swedish national quality registries were used (Swedish Catheter Ablation Register, Auricula, Swedish National Patient Register, Dispensed Drugs Register, Cause of Death Register and Riksstroke). Patients undergoing pulmonary vein isolation (PVI) were investigated for adherence to guidelines regarding oral anticoagulation, predictors for stroke after PVI, as well as risk of ischemic stroke or intracranial haemorrhage after PVI in patients with and without treatment. Results: Paper I showed that a computerized dosing algorithm for warfarin in most cases perform as well or better compared with doses that have been changed manually, with a better hit-rate (0.72 vs. 0.67) and a lower mean error (0.44 vs. 0.48). Paper II showed that 32% of 182.678 patients with a diagnosis of AF were on monotherapy with aspirin for stroke prevention. A total of 115.185 patients were included, 58.671 with aspirin treatment and 56.514 without antithrombotic treatment at baseline. After stratification after CHA2DS2-VASc score and after multivariable adjustment, aspirin treatment did not confer a decrease in thromboembolic events. After propensity score mathcing, rate of ischemic stroke was 7.4%/year (95% CI 7.1-7.6) in aspirin treated patients and 6.6%/year (95% CI 6.4-6.9) in patients without antithrombotic treatment. In paper III 22.874 patients undergoing electrical cardioversion were included, 10.722 with and 12.152 without OAC pre-treatment. In patients with low stroke risk (CHA2DS2-VASc 0-1), no thromboembolic complication was seen within 30 days after cardioversion. In patients with CHA2DS2-VASc ≥2, the risk of thromboembolic complications was increased when no oral anticoagulation pre-treatment was used, results that remained after propensity score matching. No difference regarding haemorrhagic complications was seen. Paper IV included a total of 1585 patients undergoing PVI with a mean follow up of 2.6 years. Adherence to current guidelines regarding oral anticoagulation was good in patients with CHA2DS2-VASc ≥2. Previous ischemic stroke was a predictor for a new stroke after PVI. In patients with CHA2DS2-VASc ≥2 stroke risk was increased in patients discontinuing OAC compared to those continuing OAC (1,60%/year vs. 0.34%/year). Conclusion: Oral anticoagulation is still underutilized for prevention of stroke and systemic embolism in patients with atrial fibrillation. Patients with risk factors for stroke (CHA2DS2-VASc ≥2p) benefit from continuous oral anticoagulation treatment to prevent stroke, also in conjunction with electrical cardioversion and after pulmonary vein isolation. If warfarin is chosen, a computerised dosing algorithm can facilitate and standardize warfarin dosing and lead to better resulting INR values than manually changed doses. Aspirin should not be used for stroke prevention in patients with atrial fibrillation.
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Books on the topic "Atrial fibrillation"

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Natale, Andrea, and José Jalife, eds. Atrial Fibrillation. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-163-5.

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Rawles, John. Atrial Fibrillation. London: Springer London, 1992. http://dx.doi.org/10.1007/978-1-4471-1898-5.

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1958-, Martin David, ed. Atrial fibrillation. Boston: Blackwell Scientific Publications, 1994.

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Rawles, John. Atrial fibrillation. London: Springer-Verlag, 1992.

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Mainardi, Luca, Leif Sörnmo, and Sergio Cerutti. Understanding Atrial Fibrillation. Cham: Springer International Publishing, 2008. http://dx.doi.org/10.1007/978-3-031-01632-5.

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Mainardi, Luca, Leif Sörnmo, and Sergio Cerutti. Understanding Atrial Fibrillation. Cham: Springer International Publishing, 2008. http://dx.doi.org/10.1007/978-3-031-01633-2.

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Dan, Gheorghe-Andrei, Antoni Bayés de Luna, and John Camm, eds. Atrial Fibrillation Therapy. London: Springer London, 2014. http://dx.doi.org/10.1007/978-1-4471-5475-4.

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Natale, Andrea, and Antonio Raviele, eds. Atrial Fibrillation Ablation. Oxford, UK: Blackwell Publishing Ltd, 2007. http://dx.doi.org/10.1002/9780470692646.

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Yap, Yee Guan, and A. John Camm. Essentials of Atrial Fibrillation. Tarporley: Springer Healthcare Ltd., 2014. http://dx.doi.org/10.1007/978-1-907673-98-6.

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Lip, Gregory Y. H. Atrial fibrillation in clinical practice. London: Martin Dunitz, 2001.

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Book chapters on the topic "Atrial fibrillation"

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Padanilam, Benzy J., and Eric N. Prystowsky. "Epidemiology of Atrial Fibrillation The Rising Prevalence." In Atrial Fibrillation, 3–11. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-163-5_1.

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Wang, Qing K. "Atrial Fibrillation Genetic Considerations: The Basic Scientist's Perspective." In Atrial Fibrillation, 133–44. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-163-5_10.

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Stuglin, Carlo, and D. George Wyse. "Review of Recent Trials of Medical Therapy for Atrial Fibrillation." In Atrial Fibrillation, 147–68. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-163-5_11.

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Lowe, Boris S., Mina K. Chung, and Allan L. Klein. "Antithrombotic Treatment and Cardioversion of Patients with Atrial Fibrillation." In Atrial Fibrillation, 169–83. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-163-5_12.

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Baranchuk, Adrian, Carlos A. Morillo, Martin Thoenes, Rodolpho Ventura, and Stuart J. Connolly. "Current Role of Medical Therapy for Prevention or Termination of Atrial Fibrillation." In Atrial Fibrillation, 185–95. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-163-5_13.

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Shivkumar, Kalyanam. "Applied Cardiac Anatomy for Catheter Ablation of Atrial Fibrillation." In Atrial Fibrillation, 199–207. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-163-5_14.

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Cesario, David, Osamu Fujimura, Aman Mahajan, Noel G. Boyle, and Kalyanam Shivkumar. "Selection of Ablation Catheters, Energy Sources, and Power Delivery." In Atrial Fibrillation, 209–21. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-163-5_15.

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Oral, Hakan, and Fred Morady. "Anatomically Guided Catheter Ablation for Atrial Fibrillation." In Atrial Fibrillation, 223–36. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-163-5_16.

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Jais, Pierre, Mark O' Neill, Meleze Hocini, Frederic Sacher, Nicolas Derval, Jacques Clementy, and Michel Haissaguerre. "Chronic Atrial Fibrillation and Catheter Ablation." In Atrial Fibrillation, 237–44. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-163-5_17.

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Arruda, Mauricio, Claude Elayi, Jose Carlos Pachon, and Andrea Natale. "Hybrid Strategies for Ablation of Permanent AF." In Atrial Fibrillation, 245–53. Totowa, NJ: Humana Press, 2008. http://dx.doi.org/10.1007/978-1-59745-163-5_18.

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Conference papers on the topic "Atrial fibrillation"

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Therien, Aidan, Arielle Joasil, and Christine P. Hendon. "Cardiac Substrate Classification of Human Venoatrial Junction OCT Images." In CLEO: Applications and Technology, ATh3B.6. Washington, D.C.: Optica Publishing Group, 2024. http://dx.doi.org/10.1364/cleo_at.2024.ath3b.6.

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Atrial fibrillation is the most common arrhythmia in the United States. Radiofrequency ablation is a procedure during which lesions are placed within the left atrium to prevent these signals from conducting. Correctly targeting the substrate is a critical aspect of this procedure; however, there is no real-time guidance during this procedure. This research investigates the use of optical coherence tomography-guided feedback by classifying patches from images of the venoatrial junction.
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Arad, Erez, Lital Haim, Attia Guy, Ilan Davidov, and Ludmila Sidorenko. "Recent Biotechnological Approach to Genetically Determined Atrial Fibrillation." In 12th International Conference on Electronics, Communications and Computing. Technical University of Moldova, 2022. http://dx.doi.org/10.52326/ic-ecco.2022/bme.07.

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The most prevalent persistent arrhythmia in cardiology is atrial fibrillation. Former atrial fibrillation which appears without any underlying reason was called „lone atrial fibrillation“. Due to new biotechnological methods in electrophysiology, like mapping, unusual conducting mechanisms were stabilized. Due to new biotechnological methods of DNA analysis recently the reason is detected. This is a genetically determined atrial fibrillation. The aim of this study is to analyse what are the most common mutations which lead to atrial fibrillation. Material and methods. This is a systematic review study. The sources of information which were analysed are mostly from google scholar and web of science. From 2000 sources, several sources were filtered out by the keywords and remained 14 sources on which is based this review study. Results. More than 70 genes are recently detected which lead to atrial fibrillations. Majority of them are mutations of the genes which encode the transport proteins of the heart’s conductive system. The most common mutations that lead to genetically determined atrial fibrillation occure in KCNQ1, KCNA5 and 6q14–16. Conclusions. Before starting treatment of lone atrial fibrillation, a genetical test should be done in order to stabilize the type of the underlying mutation. This is a tactical step in taking the decision on treatment strategy by antiarrhytmic drugs or ablation. So ablatogenoics is the best solution for patients with genetically determined atrial fibrillation.
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Gupta, Raghav, Yara Assadi, Shaniece Nicole Lawrence, Erika Jeanie Pitsker, Michael Scott Jr Bickford, and Victoria Amber Saniko. "Potential Mechanisms for New Onset Atrial Fibrillation in COVID-19 Patients." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. http://dx.doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.201_2024.

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Introduction: This study shows the prevalence of New-Onset Atrial Fibrillation in COVID-19 patients and highlights the most prevalent explanatory pathologic theories for the correlation. Methods: The authors carried out a literature review over PubMed using the keywords “atrial fibrillation” and “long-term COVID.” 48 articles were reviewed. Articles relating to new onset of atrial fibrillation in COVID-19 patients were included while articles posted before 2020, not related to COVID and atrial fibrillation were excluded. Results: The prevalence of atrial fibrillation in COVID patients is often reported to be around 17%, however 44% of patients within Intensive Care units (ICU) have atrial fibrillation with COVID. COVID-19 is proposed to lead to development of atrial fibrillation through dysregulation of the ACE2 receptor, increase in T-cell activation, and increasing the thrombocytes within the heart. Leading to increased edema and blockage of the electrophysiology of the heart. Discussions: Atrial fibrillation constitutes a significant risk factor for patients hospitalized for COVID-19, especially in severe cases like in the ICU. This finding resembles similar arrhythmias found in other viruses like Middle East respiratory syndrome (MERS). The prevalence of arrhythmias with viral infections shows the need to monitor the cardiac rhythms of patients with viral infections. Conclusions: COVID-19 poses a significant risk of leading to atrial fibrillation in severe cases. However, the pathophysiology has not been confirmed. Future work needs to confirm the pathophysiology of COVID-19 and atrial fibrillation to establish better treatments for patients and show the need to monitor patients’ hospitalized for viral infection cardiac rhythm.
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Karim, R., R. Mohiaddin, and D. Rueckert. "Left atrium segmentation for atrial fibrillation ablation." In Medical Imaging, edited by Michael I. Miga and Kevin R. Cleary. SPIE, 2008. http://dx.doi.org/10.1117/12.771023.

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Vaizurs, R. S. C. P., R. Sankar, and F. Leonelli. "Atrial fibrillation source identification." In 2011 33rd Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 2011. http://dx.doi.org/10.1109/iembs.2011.6091091.

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Bonizzi, P., V. Zarzoso, and O. Meste. "Atrio-Ventricular Junction behaviour during Atrial Fibrillation." In 2007 34th Annual Computers in Cardiology Conference. IEEE, 2007. http://dx.doi.org/10.1109/cic.2007.4745547.

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Funamoto, Kenichi, Ryo Koizumi, Toshiyuki Hayase, Muneichi Shibata, and Tomoyuki Yambe. "Hemodynamic Changes in the Left Atrium due to Atrial Fibrillation." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53817.

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The left atrium (LA), which connects four pulmonary veins (PVs) to the left ventricle (LV), has a characteristic shape called the left atrial appendage (LAA) under the left PV. Atrial fibrillation (AF) is a heart disease, by which irregular electrical signals with high-frequency contraction (> 400 bpm) occur in the LA. Although AF itself is not fatal, it may cause thrombus formation, resulting to cerebral infarction. In this study, hemodynamics in the LA with/without AF was investigated by means of fluid-structure interaction simulation.
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Salzmann-Djufri, M., T. Giessler, S. Rohrbach, F. Knapp, L. Ling, S. Vogt, N. Mirow, A. Böning, and B. Niemann. "New-Onset Atrial Fibrillation—Metabolic Markers, Cytokines, and Remodeling Anticipating Paroxysmal Atrial Fibrillation." In 49th Annual Meeting of the German Society for Thoracic and Cardiovascular Surgery. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1705421.

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Bellini, Chiara, and Elena S. Di Martino. "Constitutive Models for the Passive Porcine Atria at the Healthy Stage and After Ventricular Tachypacing." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80115.

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Experimental measures of the mechanical response to planar biaxial loads and observations of the local arrangement of fibers were combined to generate average constitutive models for the atria at the healthy stage and after ventricular tachypacing. These models improve the understanding of the changes in the mechanical behavior of the atria induced by atrial fibrillation.
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Ivanko, K., N. Ivanushkina, and Y. Karplyuk. "Atrial electrical activity extraction for atrial fibrillation assessment." In 2016 IEEE 36th International Conference on Electronics and Nanotechnology (ELNANO). IEEE, 2016. http://dx.doi.org/10.1109/elnano.2016.7493046.

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Reports on the topic "Atrial fibrillation"

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Gliklich, Richard E., Michelle B. Leavy, and Fang Li. Standardized Library of Atrial Fibrillation Outcome Measures. Agency for Healthcare Research and Policy (AHRQ), November 2018. http://dx.doi.org/10.23970/ahrqepcwhitepaperharmonization.

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Sonaglioni, Andrea, Antonino Bruno, Gian Luigi Nicolosi, and Michele Lombardo. Echocardiographic assessment of left atrial mechanics in patients with atrial fibrillation undergoing electrical cardioversion. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, September 2024. http://dx.doi.org/10.37766/inplasy2024.9.0037.

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Shen, Ting, Bo Zhuang, Guanghe Li, Yumei Jiang, Xiaoling Liu, Yishan Jin, Guangyu Wang, Liang Zheng, and Yuqin Shen. Cardiac rehabilitation for atrial fibrillation recurrence, a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2020. http://dx.doi.org/10.37766/inplasy2020.6.0003.

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Cui, Zhaorui, Yahui Wang, and Yanchen Zhu. Chinese herbal medicine for catheter ablation of atrial fibrillation. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2022. http://dx.doi.org/10.37766/inplasy2022.5.0096.

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Yang, Shengyi, Min Huang, Ge Ge, Junyue Yang, Yingchao Tan, Jieqiong Guan, Wenjing Song, and Lina Wang. Atrial fibrillation burden and risk of stroke: a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2020. http://dx.doi.org/10.37766/inplasy2020.10.0032.

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Sanders, Gillian D., Angela Lowenstern, Ethan Borre, Ranee Chatterjee, Adam Goode, Lauren Sharan, Nancy M. Allen LaPointe, et al. Stroke Prevention in Patients With Atrial Fibrillation: A Systematic Review Update. Agency for Healthcare Resarch and Quality (AHRQ), October 2018. http://dx.doi.org/10.23970/ahrqepccer214.

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Pshezhetskiy, Dmitry, Tanveer Alam, and Heba Alshaker. Unsynchronised Cardioversion as a Cause of Ventricular Tachycardia in a Patient with Atrial Fibrillation. Nature Library, November 2020. http://dx.doi.org/10.47496/nl.ccr.2020.01.02.

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Background: Synchronised cardioversion (SC) is used to terminate tachycardic arrhythmia by applying electric current to the thorax. SC is synchronised to the R wave of the cardiac cycle and ventricular tachycardia (VT) or ventricular fibrillation (VF) can occur if an electrical shock is provided in a nonsynchronised way. Case Presentation: Here we present a case of a 66-year-old man who had elective cardioversion for atrial fibrillation worsened by severe left ventricular impairment. A manual defibrillator was used for the cardioversion, which, after the first synchronised shock, reverted to defibrillator mode. An unsynchronised shock was administered and induced VT, which was reverted to sinus rhythm with a defibrillation shock. Conclusion: When using manual defibrillator for SC, the machine needs to be set to a synchronised mode. The synchronisation to the R wave needs to be checked before every shock.
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Velichkova, Anna, Arman Postadjian, Kiril Karamfiloff, and Milena Staneva. Anticoagulation in Atrial Fibrillation - the Choice of Bulgarian Physicians in 2015 and 2017. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, August 2021. http://dx.doi.org/10.7546/crabs.2021.08.15.

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Gao, Zheng, He-Kai Shi, Wei Xu, Xiuxiu Su, Zhengzhao Guan, Nuojin Guo, and Huijie Ma. Hyperuricemia increases the risk of atrial fibrillation: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, December 2021. http://dx.doi.org/10.37766/inplasy2021.12.0092.

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Oh, SangHyeon, Seoyong Choi, and Jee-Eun Chung. Comparative efficacy and safety of reduced dose of DOACs in patients with atrial fibrillation. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2022. http://dx.doi.org/10.37766/inplasy2022.8.0073.

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Review question / Objective: To compare the risk of stroke/systemic embolism (S/SE), mortality and bleeding in AF patients with reduced-dose DOACs. Rationale: Although each DOAC has its dose reduction criteria, many physicians still prefer to prescribe the reduced-dose DOACs, regardless of label adherence. However, inappropriate administration of DOACs is an important clinical problem because patients may not benefit from the recommended DOAC dose to prevent stroke and systemic embolism. Therefore, this study aims to investigate the risk of stroke/systemic embolism (S/SE) and mortality in AF patients with reduced-dose DOACs. Condition being studied: Adult patients with AF taking DOACs or Warfarin.
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