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Journal articles on the topic 'Atrial fibrillation'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

S., Sinha,, Kumari, S., and Mishra, K. "Molecular Docking Study of Crocetin with Interleukin–18 for the Treatment of Atrial Fibrillation." CARDIOMETRY, no. 24 (November 30, 2022): 379–84. http://dx.doi.org/10.18137/cardiometry.2022.24.379384.

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The most frequent arrhythmias are atrial fibrillation. Although the mechanisms underlying are not fully understood, they encompass postoperative and intraoperative phenomena like cardiac ischemia sympathetic activation and inflammation that ends up causing atrial fibrillation. Atrial fibrillation is frequently associated with the presence of pre-existing factors, making the atria susceptible to atrial fibrillation. There are various treatments have been used to manage the condition. However, they are sometimes ineffective, costly, and have side effects stressing the need to explore alternative

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2

Hulsmans, Maarten, Maximilian J. Schloss, I.-Hsiu Lee, et al. "Recruited macrophages elicit atrial fibrillation." Science 381, no. 6654 (2023): 231–39. http://dx.doi.org/10.1126/science.abq3061.

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Atrial fibrillation disrupts contraction of the atria, leading to stroke and heart failure. We deciphered how immune and stromal cells contribute to atrial fibrillation. Single-cell transcriptomes from human atria documented inflammatory monocyte and SPP1 + macrophage expansion in atrial fibrillation. Combining hypertension, obesity, and mitral valve regurgitation (HOMER) in mice elicited enlarged, fibrosed, and fibrillation-prone atria. Single-cell transcriptomes from HOMER mouse atria recapitulated cell composition and transcriptome changes observed in patients. Inhibiting monocyte migration

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3

Kahadi, Cik, and Ardian Rizal. "Atrial myopathy: a pathogenic mechanism linking atrial fibrillation and ischemic stroke." Heart Science Journal 6, no. 2 (2025): 3–10. https://doi.org/10.21776/ub.hsj.2025.006.02.2.

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Atrial myopathy (AM) has become a topic of study for many years. Atrial Myopathy is characterized as "any variety of architectural, structural, electrophysiological, or contractile abnormalities altering the atria, which can result in clinically significant manifestations," and is associated with atrial dysfunction and dilatation. Evidence shows that atrial myopathy (AM) contributes to atrial fibrillation and embolic strokes of unknown origin. Atrial myopathy or disease provides a substrate leading to atrial fibrillation (AF) and contributes to a chance of atrial thrombus development and, fina

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4

Wu, Shaohui, Guangchen Zou, Xu Liu, Weifeng Jiang, Mu Qin, and Daoliang Zhang. "Key Role of Left Atrial Appendage during Redo Ablation in a Case of Long-Standing Persistent Atrial Fibrillation." Case Reports in Cardiology 2020 (June 19, 2020): 1–4. http://dx.doi.org/10.1155/2020/9691584.

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Extrapulmonary vein focal sources have been recognized as the source of atrial fibrillation in some cases, and empiric electric isolation of the left atrial appendage has been proposed for long-standing persistent atrial fibrillation by some. Here, we present a case of redo ablation of long-standing persistent atrial fibrillation in which the left atrial appendage played a key role in maintaining AF during ablation, and atrial fibrillation was terminated by electrical isolation of the LAA. During the ablation, a rare phenomenon of half of the atria in atrial fibrillation while the other half o

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5

Hassanain Elsayed, Yasser Mohammed. "Sinusoidal Atrial Fibrillation (Yasser’s Fibrillation) and Partial SAN Function in COVID-19 Pneumonia; a New Cardiovascular Discovery Change in Atrial Fibrillation Directory." International Journal of Cardiovascular Medicine 3, no. 5 (2024): 01–08. http://dx.doi.org/10.31579/2834-796x/078.

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Introduction: Atrial fibrillation (AF) is one of the most common and probable serious arrhythmias and a hallmark of an increasing risk of pathological thrombus formation. Interestingly, normal sinus rhythm (NSR) is the standard rhythm in a normal person. The sino-atrial node (SAN) is the heart's pacemaker and generator of the normal electrical conduction system of the heart which allows for the generating of electrical impulses. In AF, the normal regular electrical impulses generated by the sinoatrial node are overwhelmed by disorganized electrical waves, usually originating from the roots of

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6

Lu, Zhibing, Benjamin J. Scherlag, Jiaxiong Lin, et al. "Atrial Fibrillation Begets Atrial Fibrillation." Circulation: Arrhythmia and Electrophysiology 1, no. 3 (2008): 184–92. http://dx.doi.org/10.1161/circep.108.784272.

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7

Wijffels, Maurits C. E. F., Charles J. H. J. Kirchhof, Rick Dorland, and Maurits A. Allessie. "Atrial Fibrillation Begets Atrial Fibrillation." Circulation 92, no. 7 (1995): 1954–68. http://dx.doi.org/10.1161/01.cir.92.7.1954.

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8

Adeyana, Sri, Haryadi Haryadi, and Chandra Wijaya. "Hubungan Kejadian Fibrilasi Atrium dengan Diameter Atrium Kiri pada Fibrilasi Atrium Valvular dan Fibrilasi Atrium Non-Valvular Di RSUD Arifin Achmad." Jurnal Ilmu Kedokteran 11, no. 1 (2018): 31. http://dx.doi.org/10.26891/jik.v11i1.2017.31-38.

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Atrial Fibrillationis a kind of arrhythmia which has the most incidence. Based on its Etiology, atrial fibrillationcouldbe divided in to two, valvular and nonvalvular atrial fibrillation. This study was aimed toknow the correlation ofatrial fibrillation incidence between valvular and nonvalvular with its left atrium diameter in Arifin Achmad ProvinsiRiau’s General Hospital. This study was analytical and done by cross sectional approach with 185 patient. The datawere processing with computerize to univariate analysis and chi-square for bivarite analysis. From this study it can beconcluded that

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9

Opie, Lionel H. "Tedisamil in Coronary Disease: Additional Benefits in the Therapy of Atrial Fibrillation?" Journal of Cardiovascular Pharmacology and Therapeutics 8, no. 1_suppl (2003): S33—S37. http://dx.doi.org/10.1177/107424840300800105.

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Atrial fibrillation has recently come into clinical and research focus. In particular, ventricular rate control has been carefully compared with atrial rhythm control. Additionally, the recent discovery of atrial stunning has initiated clinical and research interest in atrial remodeling. Atrial fibrillation is more likely to occur when the atria are damaged by increased fibrosis. The ideal way to prevent atrial fibrillation and the risk of repetition is by tackling the root causes, such as ischemic heart disease, heart failure, and left ventricular hypertrophy. Tedisamil is an unusual antifibr

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10

Medeiros de Vasconcelos, J. Tarcísio. "Obesidade e Fibrilação Atrial." Journal of Cardiac Arrhythmias 32, no. 3 (2020): 153–54. http://dx.doi.org/10.24207/jca.v32i3.984_pt.

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A fibrilação atrial ocupa uma discussão central hoje no universo das arritmias cardíacas. O aumento da expectativa de vida que trouxe um substancial aumento de sua prevalência na população geral1 implicou em um aprofundamento marcante na compreensão dos seus mecanismos eletrofisiológicos, na identificação de fatores determinantes ou de potencialização da sua ocorrência e obviamente no desenvolvimento de estratégias efetivas de tratamento e de prevenção das suas complicações. Desde o clássico estudo de Wijffels et al., publicado em 1995 (Atrial Fibrillation Begets Atrial Fibrillation)2 demonstr

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11

Jandrić-Kočič, Marijana. "The incidence of atrial fibrillation in type 2 diabetes mellitus patients." Sestrinska rec 23, no. 81 (2020): 20–24. http://dx.doi.org/10.5937/sestrec2081020j.

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Goal: Diabetes mellitus type 2 is a metabolic syndrome with systemic disorders of carbohydrate metabolism, fat and protein due to the absolute or relative lack of biologically active insulin. Atrial fibrillation is characterized by high frequency excitation of atria, consequent asynchronous atrial contraction, and irregular ventricular excitation. Diabetes predisposes fibrillation through metabolic abnormalities, structural, electrical, electromechanical, and autonomic remodeling of atrias. The study had to examine the incidence of atrial fibrillation in type 2 diabetes mellitus patients and t

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12

Yim, Jeffrey, and Andrew D. Krahn. "Postoperative Atrial Fibrillation Begets Atrial Fibrillation." JACC: Clinical Electrophysiology 10, no. 7 (2024): 1720–21. http://dx.doi.org/10.1016/j.jacep.2024.06.009.

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13

Algalarrondo, Vincent, and Fabrice Extramiana. "Autoimmune Atrial Fibrillation or Atrial Fibrillation–Induced Autoimmunity? A New Atrial Fibrillation Begets Atrial Fibrillation Pathway?" Circulation 148, no. 6 (2023): 499–501. http://dx.doi.org/10.1161/circulationaha.123.063672.

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14

Schotten, Ulrich, Sunniva de Haan, Hans-Ruprecht Neuberger, et al. "Loss of atrial contractility is primary cause of atrial dilatation during first days of atrial fibrillation." American Journal of Physiology-Heart and Circulatory Physiology 287, no. 5 (2004): H2324—H2331. http://dx.doi.org/10.1152/ajpheart.00581.2004.

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Atrial fibrillation (AF) induces a progressive dilatation of the atria which in turn might promote the arrhythmia. The mechanism of atrial dilatation during AF is not known. To test the hypothesis that loss of atrial contractile function is a primary cause of atrial dilatation during the first days of AF, eight goats were chronically instrumented with epicardial electrodes, a pressure transducer in the right atrium, and piezoelectric crystals to measure right atrial diameter. AF was induced with the use of repetitive burst pacing. Atrial contractility was assessed during sinus rhythm, atrial p

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15

Yashin, Sergei, and Yuri Shubik. "Atriopathy and atrial fibrillation. Part I." Vestnik of Saint Petersburg University. Medicine 17, no. 4 (2022): 254–71. http://dx.doi.org/10.21638/spbu11.2022.402.

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Atrial fibrillation is the most common supraventricular tachycardia, the incidence of which increases with age. This arrhythmia is the consequenсe of multiple cardiac and noncardiac pathology. The most significant side effects of atrial fibrillation are thromboembolic complications. It is generally accepted that the source of these embolisms is a thrombus in the left atrial appendage. However, the risk of thromboembolism also occurs after the onset of atrial rhythm, which can lead to a more complex hemostasis mechanism in this disease. There is no unambiguous definition of the relationship bet

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16

Mazur, Evgeniy S., Vera V. Mazur, Nikolay D. Bazhenov, Sergey V. Kolbasnicov, and Oksana V. Nilova. "Epicardial obesity and atrial fibrillation: emphasis on atrial fat depot." Obesity and metabolism 17, no. 3 (2020): 316–25. http://dx.doi.org/10.14341/omet12614.

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The studies, performed with MRI and CT, showed that the increase of fat, immediately adjacent to the myocardium (epicardial fat) is correlated more strongly with the risk of atrial fibrillation than the general or abdominal obesity. According to some studies, epicardial fat around the left atrium is a strong predictor of the development at atrial fibrillation. Also, the amount of the fat is associated with the effectiveness of cardioversion and the risk of developing thromboembolic stroke in patients with atrial fibrillation. The number of such works is small, since tomographic examinations ar

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17

Psychari, Stavroula N., Dionysios Tsoukalas, Dimitrios Varvarousis, et al. "Opposite relations of epicardial adipose tissue to left atrial size in paroxysmal and permanent atrial fibrillation." SAGE Open Medicine 6 (January 2018): 205031211879990. http://dx.doi.org/10.1177/2050312118799908.

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Objectives: Atrial fibrillation has been associated with obesity in epidemiological studies. Epicardial adipose tissue is an ectopic fat depot in the proximity of atria, with endocrine and inflammatory properties that is implicated in the pathophysiology of atrial fibrillation. Inflammation also has a role in atrial arrhythmogenesis. The aim of this study was to investigate the potential relations of epicardial adipose tissue to left atrial size and to adiponectin and the pro-inflammatory mediators, high-sensitivity C-reactive protein, and interleukin-6 in paroxysmal and permanent atrial fibri

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18

Leistad, E., G. Christensen, and A. Ilebekk. "Effects of atrial fibrillation on left and right atrial dimensions, pressures, and compliances." American Journal of Physiology-Heart and Circulatory Physiology 264, no. 4 (1993): H1093—H1097. http://dx.doi.org/10.1152/ajpheart.1993.264.4.h1093.

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The effects of atrial fibrillation on left and right atrial dimensions, pressures, and compliances were examined in two groups of seven barbiturate-anesthetized open-chest pigs. Atrial diameters and pressures were recorded during atrioventricular (AV) pace and thereafter during atrial fibrillation. Both rhythms were studied with constant ventricular rate after complete AV block. Left atrial maximal diameter, which appeared at the end of the atrial filling phase, decreased from 32.4 (28.9-36.7; median and 95% confidence interval) to 31.3 (28.4-35.7) mm after induction of atrial fibrillation. Th

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19

Carrero, Juan Jesus, Marco Trevisan, Manish M. Sood, et al. "Incident Atrial Fibrillation and the Risk of Stroke in Adults with Chronic Kidney Disease." Clinical Journal of the American Society of Nephrology 13, no. 9 (2018): 1314–20. http://dx.doi.org/10.2215/cjn.04060318.

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Background and objectivesPatients with CKD have a high risk of atrial fibrillation. Both CKD and atrial fibrillation are associated with higher risk of stroke and death. However, the effect of incident atrial fibrillation on stroke risk among patients with CKD is unknown.Design, setting, participants, & measurementsOur study included adults with CKD (eGFR<60 ml/min per 1.73 m2) without previously documented atrial fibrillation who had been in contact with health care in Stockholm, Sweden during 2006–2011. Incident atrial fibrillation was identified by administrative diagnostic codes in

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20

Aidietis, Audrius, Jūratė Barysienė, Germanas Marinskis, et al. "Factors influencing development of atrial fibrillation after implantation of cardiac pacemaker for sinus node dysfunction." Medicina 45, no. 3 (2009): 169. http://dx.doi.org/10.3390/medicina45030022.

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Objective. The aim of this study was to evaluate factors influencing the success of atrial fibrillation treatment associated with the sick sinus syndrome after pacemaker implantation. Methods and results. In 163 patients with sick sinus syndrome followed up after pacemaker implantation, statistical analysis showed that the recurrence of atrial fibrillation increased 2.8 times and 2.5 times when the left atrium or the right atrium, respectively, were increased by 1 cm (P=0.001). In addition, the recurrence of atrial fibrillation increased 2.5 times when the interventricular septum was thickened

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21

Cohen, Mitchell I., Jordan A. Cohen, Connor Shope, Lauren Stollar, and Lucas Collazo. "Ivabradine as a stabilising anti-arrhythmic agent for multifocal atrial tachycardia." Cardiology in the Young 30, no. 6 (2020): 899–902. http://dx.doi.org/10.1017/s1047951120001195.

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AbstractMultifocal atrial tachycardia has certain electrocardiographic similarities to atrial fibrillation. The mechanism of atrial fibrillation is heterogenous but in some cases may arise from a single ectopic driver with fibrillatory conduction to the rest of the atria. This has led to the speculation that multifocal atrial tachycardia may have a similar mechanistic unifocal site that disperses through the atrium in a fibrillatory pattern. Ivabradine has been reported to be efficacious in an adult with paroxysmal atrial fibrillation as well as in children with junctional or ectopic atrial ta

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22

Sharikov, Nikita L., S. M. Chibisov, O. N. Ragozin, and S. Sh Gasimova. "Variants of the anatomical structure of the coronary arteries and the left atrium remodeling in patients with different forms of atrial fibrillation." Clinical Medicine (Russian Journal) 96, no. 9 (2018): 809–13. http://dx.doi.org/10.18821/0023-2149-2018-96-9-809-813.

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One of the reasons leading to dilatation of the left atrium is atrial fibrillation. A retrospective analysis of 136 case histories of patients with various forms of “non-valvular” atrial fibrillation was performed, depending on the shape of atrial fibrillation, the patients were divided into 3 groups. In patients with atrial fibrillation in 62.5%, the source of the atrial branches was the envelope branch of the left coronary artery. Atrial branches originating from the right coronary artery system were identified in 35.8%. In men, atrial arteries occur significantly more often. The results dif

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23

Ioannou, Adam. "Atrial Fibrillation with Ventricular Pre-Excitation: A Diagnosis That Must Not Be Missed." Archives of Medical Case Reports and Case Study 5, no. 4 (2022): 01–03. http://dx.doi.org/10.31579/2692-9392/106.

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Wolff-Parkinson-White (WPW) syndrome is caused by an accessory pathway that communicates between the atria and ventricles known as the Bundle of Kent. The development of atrial fibrillation, can result in the atrial impulses all being conducted via the accessory pathway and result in a sinister, board complex, irregular tachycardia, with varying QRS morphology (known as pre-excited atrial fibrillation) Adenosine is a potent atrioventricular node blocker, which can be used in the treatment of supraventricular tachycardias, but also has diagnostic utility, particularly in differentiating between

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24

Melo, J. Q., J. P. Neves, L. M. Abecasis, P. Adragão, R. Ribeiras, and R. Seabra-Gomes. "Operative Risks of the Maze Procedure Associated with Mitral Valve Surgery." Cardiovascular Surgery 5, no. 1 (1997): 112–16. http://dx.doi.org/10.1177/096721099700500119.

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Twelve patients were operated on for mitral valve disease with concomitant chronic atrial fibrillation. Valve repair was performed in five patients and replacement in seven. Maze I and maze III procedures were applied in eight and four patients, respectively, and are compared. There was a regular rhythm in all maze I patients with a constant junctional rhythm in three and an alternating sinus and junctional rhythm in the remainder. In one case, part of the left atrium was in atrial fibrillation with the remaining atria in sinus rhythm. In the maze III group, one patient was always on a regular

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25

Fu, Fumin, Michael Pietropaolo, Lei Cui, et al. "Lack of authentic atrial fibrillation in commonly used murine atrial fibrillation models." PLOS ONE 17, no. 1 (2022): e0256512. http://dx.doi.org/10.1371/journal.pone.0256512.

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The mouse is a useful preclinical species for evaluating disease etiology due to the availability of a wide variety of genetically modified strains and the ability to perform disease-modifying manipulations. In order to establish an atrial filtration (AF) model in our laboratory, we profiled several commonly used murine AF models. We initially evaluated a pharmacological model of acute carbachol (CCh) treatment plus atrial burst pacing in C57BL/6 mice. In an effort to observe micro-reentrant circuits indicative of authentic AF, we employed optical mapping imaging in isolated mouse hearts. Whil

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26

Michaud, Gregory F., and William G. Stevenson. "Atrial Fibrillation." New England Journal of Medicine 384, no. 4 (2021): 353–61. http://dx.doi.org/10.1056/nejmcp2023658.

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27

Jacek, Grace A. "Atrial Fibrillation." Home Healthcare Now 39, no. 1 (2021): 6–12. http://dx.doi.org/10.1097/nhh.0000000000000946.

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28

Elizabeth, Hunt. "Atrial fibrillation." Nursing Standard 27, no. 1 (2012): 59–60. http://dx.doi.org/10.7748/ns.27.1.59.s54.

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29

Stevenson, Rebecca. "Atrial fibrillation." Nursing Standard 27, no. 14 (2012): 59–60. http://dx.doi.org/10.7748/ns.27.14.59.s53.

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30

Lewis, Richard V. "Atrial Fibrillation." Drugs 40, no. 6 (1990): 841–53. http://dx.doi.org/10.2165/00003495-199040060-00006.

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31

Hunt, Elizabeth. "Atrial fibrillation." Nursing Standard 27, no. 1 (2012): 59. http://dx.doi.org/10.7748/ns2012.09.27.1.59.c9272.

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32

Stevenson, Rebecca. "Atrial fibrillation." Nursing Standard 27, no. 14 (2012): 59. http://dx.doi.org/10.7748/ns2012.12.27.14.59.c9477.

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33

Witham, Dawn. "Atrial fibrillation." Nursing Standard 27, no. 32 (2013): 59. http://dx.doi.org/10.7748/ns2013.04.27.32.59.s54.

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34

James, Natalie. "Atrial fibrillation." Nursing Standard 27, no. 46 (2013): 59. http://dx.doi.org/10.7748/ns2013.07.27.46.59.s48.

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35

Jolobe, OMP. "Atrial Fibrillation." Journal of the Royal College of Physicians of Edinburgh 30, no. 2 (2000): 180. http://dx.doi.org/10.1177/147827150003000221.

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36

Georgilis, Shenaz, and Carlene Byfield. "Atrial fibrillation." Nursing Made Incredibly Easy! 20, no. 5 (2022): 24–31. http://dx.doi.org/10.1097/01.nme.0000853772.24358.53.

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37

Spector, Peter. "Atrial Fibrillation." JACC: Clinical Electrophysiology 8, no. 5 (2022): 578–81. http://dx.doi.org/10.1016/j.jacep.2022.04.003.

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38

Callans, David J. "Atrial Fibrillation." Annals of Internal Medicine 149, no. 9 (2008): ITC5–1. http://dx.doi.org/10.7326/0003-4819-149-9-200811040-01005.

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39

Zimetbaum, Peter. "Atrial Fibrillation." Annals of Internal Medicine 153, no. 11 (2010): ITC6–1. http://dx.doi.org/10.7326/0003-4819-153-11-201012070-01006.

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40

Medi, Caroline, Graeme J. Hankey, and Saul B. Freedman. "Atrial fibrillation." Medical Journal of Australia 186, no. 4 (2007): 197–202. http://dx.doi.org/10.5694/j.1326-5377.2007.tb00862.x.

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41

Erol, Cetin. "Atrial Fibrillation." Anatolian Journal of Cardiology 26, no. 9 (2022): 672. http://dx.doi.org/10.5152/anatoljcardiol.2022.9.

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Zimetbaum, Peter. "Atrial Fibrillation." Annals of Internal Medicine 166, no. 5 (2017): ITC33. http://dx.doi.org/10.7326/aitc201703070.

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Baman, Jayson R., and Rod S. Passman. "Atrial Fibrillation." JAMA 325, no. 21 (2021): 2218. http://dx.doi.org/10.1001/jama.2020.23700.

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Riaz, Rehan, Nabeel Ahmad, and Shahid Abbas. "ATRIAL FIBRILLATION;." Professional Medical Journal 24, no. 12 (2017): 1852–59. http://dx.doi.org/10.29309/tpmj/2017.24.12.617.

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Introduction: Atrial Fibrillation (AF) is one of the most common arrhythmiaafter coronary artery bypass grafting (CABG). Many risk factors have been identified for thedevelopment of postoperative AF with varied level of evidence. AF is associated with increasedrisk of morbidity and mortality causing prolong hospital stay and utilizing more resources.Our study was aimed to identify high risk population for developing post-operative AF andto draw recommendations for its prevention. Objectives: Our objective was to determine thefrequency of Atrial Fibrillation (AF) and its predisposing factors in

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45

&NA;. "Atrial fibrillation." Drugs & Therapy Perspectives 6, no. 4 (1995): 9–12. http://dx.doi.org/10.2165/00042310-199506040-00004.

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Richardson, Rachael. "Atrial fibrillation." Nursing Standard 26, no. 26 (2012): 59. http://dx.doi.org/10.7748/ns2012.02.26.26.59.c8970.

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McCartney, David E., Oliver Lomas, and Thomas J. Cahill. "Atrial fibrillation." InnovAiT: Education and inspiration for general practice 8, no. 8 (2014): 485–92. http://dx.doi.org/10.1177/1755738014541425.

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48

Guha, Kaushik. "Atrial fibrillation." Clinical Medicine 19, no. 1 (2019): 90.1–90. http://dx.doi.org/10.7861/clinmedicine.19-1-90.

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Pradip Verma, Kunal, and Michael Wong. "Atrial fibrillation." Australian Journal of General Practice 48, no. 10 (2019): 694–99. http://dx.doi.org/10.31128/ajgp-12-18-4787.

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50

Futterman, Laurie G., and Louis Lemberg. "Atrial Fibrillation." American Journal of Critical Care 14, no. 5 (2005): 438–40. http://dx.doi.org/10.4037/ajcc2005.14.5.438.

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