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Journal articles on the topic 'Attention Deficit Disorder with Hyperactivity – etiology'

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Published: 4 June 2021

Last updated: 11 February 2022

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1

Castellanos, F. Xavier, and Judith L. Rapoport. "Etiology of Attention-Deficit Hyperactivity Disorder." Child and Adolescent Psychiatric Clinics of North America 1, no. 2 (1992): 373–84. http://dx.doi.org/10.1016/s1056-4993(18)30593-5.

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Kondekar, Santosh, Amit Rudrakar, and Sharan Subramanian. "Neonatal Head Injury as a Potential Etiology for Autism Spectrum Disorders and Attention Deficit Hyperactivity Disorder (ADHD)." Pediatric Education and Research 6, no. 2 (2018): 127–28. http://dx.doi.org/10.21088/per.2321.1644.6218.17.

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3

GÜL, Hesna, and Bedriye ÖNCÜ. "Etiology of Attention Deficit Hyperactivity Disorder: Genetical Factors." Turkiye Klinikleri Journal of Pediatrics 27, no. 3 (2018): 124–35. http://dx.doi.org/10.5336/pediatr.2018-61707.

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4

Shealy, Anne H. "Attention-Deficit Hyperactivity Disorder?Etiology, Diagnosis, and Management." Journal of Child and Adolescent Psychiatric Nursing 7, no. 2 (1994): 24–36. http://dx.doi.org/10.1111/j.1744-6171.1994.tb00193.x.

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Бариляк, Ирина Анатольевна, Николай Любомирович Бариляк, and Елена Анатольевна Карасева. "ATTENTION DEFICIENCY SYNDROME AND HYPERACTIVITY: ETIOLOGY, TYPES, PSYCHOLOGICAL WORK." Вестник Тверского государственного университета. Серия: Педагогика и психология, no. 3(52) (October 9, 2020): 88–94. http://dx.doi.org/10.26456/vtpsyped/2020.3.088.

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Представлены результаты теоретического исследования по проблеме синдрома дефицита внимания и гиперактивности. Рассматривается история изучения синдрома дефицита внимания и гиперактивности, теории этиологии синдрома, факторы и причины формирования, его классификация, методы психологической работы с расстройством. The article presents the results of a theoretical study of the problem of attention deficit hyperactivity disorder. The anamnesis of attention deficit hyperactivity disorder, the theory of the etiology of the syndrome, its classification, psychological work with the disorder were studi

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Riccio, Cynthia A., George W. Hynd, Morris J. Cohen, and Jose J. Gonzalez. "Neurological Basis of Attention Deficit Hyperactivity Disorder." Exceptional Children 60, no. 2 (1993): 118–24. http://dx.doi.org/10.1177/001440299306000205.

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Since the turn of the century, researchers have believed that attention deficit disorder (ADD) involves brain dysfunction. Many theories have been researched in an attempt to resolve the nature of brain dysfunction that has resulted in behaviors associated with ADD. In the past two decades, with an expansion of research technology, significant changes have occurred in the conceptualization of ADD—with or without hyperactivity—as well as in the neurological models relating to the etiology of this disorder. This article reviews these models and presents neuroanatomical, neurochemical, and neurop

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Freire Lucas, R. M. "Late onset adult attention deficit hyperactivity disorder." European Psychiatry 26, S2 (2011): 1718. http://dx.doi.org/10.1016/s0924-9338(11)73422-8.

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Attention-deficit hyperactivity disorder (ADHD) is a common, genetically transmitted neurological disorder, with onset in childhood, probably mediated in part by decreased brain dopaminergic functioning. Prevalence and natural history data suggest that of the 3–10% of children diagnosed with ADHD, one-to two-thirds continue to manifest appreciable ADHD symptoms as adults, interfering in academic, professional, affective and social life. Recently, several studies have challenged DSM-IV's requirement of ADHD onset before age 7 with the questioning of the validity of the age-at onset criterion.Ba

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8

Redmond, Sean M. "Language Impairment in the Attention-Deficit/Hyperactivity Disorder Context." Journal of Speech, Language, and Hearing Research 59, no. 1 (2016): 133–42. http://dx.doi.org/10.1044/2015_jslhr-l-15-0038.

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Purpose Attention-deficit/hyperactivity disorder (ADHD) is a ubiquitous designation that affects the identification, assessment, treatment, and study of pediatric language impairments (LIs). Method Current literature is reviewed in 4 areas: (a) the capacity of psycholinguistic, neuropsychological, and socioemotional behavioral indices to differentiate cases of LI from ADHD; (b) the impact of co-occurring ADHD on children's LI; (c) cross-etiology comparisons of the nonlinguistic abilities of children with ADHD and specific LI (SLI); and (d) the extent to which ADHD contributes to educational an

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Biederman, J., and S. V. Faraone. "Current concepts on the neurobiology of Attention-Deficit/Hyperactivity Disorder." Journal of Attention Disorders 6, no. 1_suppl (2002): 7–16. http://dx.doi.org/10.1177/070674370200601s03.

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Attention-Deficit/Hyperactivity Disorder (ADHD) is an early onset, clinically heterogeneous disorder of inattention, hyperactivity, and impulsivity. In contrast to the widespread acceptance of ADHD as a childhood diagnosis, its prevalence in adults and its implications for clinical practice remain a source of controversy. Throughout the lifecycle, a key clinical feature observed in ADHD patients is comorbidity with Conduct, Depressive, Bipolar, and Anxiety disorders. Family studies consistently support the assertion that ADHD runs in families. Heritabillty data from twin studies of ADHD attrib

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Chaaya, Rony, and Diala El Khoury. "Attention-Deficit and Hyperactivity Disorder: A Disorder or a Fraud?" Global Journal of Health Science 11, no. 5 (2019): 100. http://dx.doi.org/10.5539/gjhs.v11n5p100.

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Attention Deficit Hyperactivity Disorder is a psychiatric and behavioral disorder marked by chronic inattention and/or hyperactivity-impulsivity that interferes with functioning or development. This disorder is caused by many dysfunctions in the brain especially in the prefrontal cortex. To date, numerous studies have attempted to unravel the biological pathways behind ADHD. Many environmental risk factors have been identified including lead exposure and prenatal alcohol and tobacco exposure. Specific mutations in genes affecting dopamine and norepinephrine release are also under investigation

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11

Millichap, J. G. "Etiologic Classification of Attention-Deficit/Hyperactivity Disorder." PEDIATRICS 121, no. 2 (2008): e358-e365. http://dx.doi.org/10.1542/peds.2007-1332.

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12

GLOD, C. "Attention deficit hyperactivity disorder throughout the lifespan: Diagnosis, etiology, and treatment." Journal of the American Psychiatric Nurses Association 3, no. 3 (1997): 89–92. http://dx.doi.org/10.1016/s1078-3903(97)90023-9.

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Glod, Carol A. "Attention Deficit Hyperactivity Disorder Throughout the Lifespan: Diagnosis, Etiology, and Treatment." Journal of the American Psychiatric Nurses Association 3, no. 3 (1997): 89–92. http://dx.doi.org/10.1177/107839039700300306.

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14

Aureli, A., D. Piancatelli, T. Del Beato, et al. "Holly's Story: Illustration of an Attention Deficit Hyperactivity Disorder Case." International Journal of Immunopathology and Pharmacology 21, no. 4 (2008): 1049–51. http://dx.doi.org/10.1177/039463200802100433.

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During the last 10 years a significant increase of childhood neuropsychiatric disorders, such as attention deficit hyperactivity disorder (ADHD), has been reported. ADHD is believed to have a multifactorial etiology including multiple genetic and prenatal environmental factors. For this reason, there has been a recent revival regarding the role of autoimmunity in this pathology. An ADHD combined-type patient born from a drug-addicted mother was studied. Neuropsychological tests according to the criteria of the 4th edition of the Diagnostic and Statistical Manual (DSM4) permitted us to make the

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15

Lindstrøm, Jon A. "Why Attention-Deficit/Hyperactivity Disorder Is Not a True Medical Syndrome." Ethical Human Psychology and Psychiatry 14, no. 1 (2012): 61–73. http://dx.doi.org/10.1891/1559-4343.14.1.61.

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Critics of attention-deficit/hyperactivity disorder (ADHD) have repeatedly argued that there is no proof for the condition being symptomatic of an organic brain disease and that the current “ADHD epidemic” is an expression of medicalization. To this, the supporters of ADHD can retort that the condition is only defined as a mental disorder and not a physical disease. As such, ADHD needs only be a harmful mental dysfunction, which, like other genuine disorders, can have a complex and obscure etiology. This article argues that such a line of argument fails to save ADHD as a valid diagnostic categ

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Derks, Eske M., Conor V. Dolan, Jim J. Hudziak, Michael C. Neale, and Dorret I. Boomsma. "Assessment and Etiology of Attention Deficit Hyperactivity Disorder and Oppositional Defiant Disorder in Boys and Girls." Behavior Genetics 37, no. 4 (2007): 559–66. http://dx.doi.org/10.1007/s10519-007-9153-4.

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17

Lange, Klaus W. "Dietary factors in the etiology and therapy of attention deficit/hyperactivity disorder." Current Opinion in Clinical Nutrition & Metabolic Care 20, no. 6 (2017): 464–69. http://dx.doi.org/10.1097/mco.0000000000000415.

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18

Puddu, Giannina, Paula Rothhammer, and Francisco Rothhammer. "Genetic and Evolutionary Contributions to the Etiology of Attention Deficit Hyperactivity Disorder." Current Genetic Medicine Reports 5, no. 1 (2017): 54–57. http://dx.doi.org/10.1007/s40142-017-0114-9.

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19

Maag, John W., and Robert Reid. "Attention-Deficit Hyperactivity Disorder: A Functional Approach to Assessment and Treatment." Behavioral Disorders 20, no. 1 (1994): 5–23. http://dx.doi.org/10.1177/019874299402000104.

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Although numerous terms have been used to describe Attention-Deficit Hyperactivity Disorder (ADHD) during the past several decades, it has continued to be conceptualized from a biological or neurological perspective. Consequently, much research has accumulated investigating possible biological and neurological causes, developing better diagnostic criteria, and accumulating medication protocols. Despite voluminous amounts of research, little consensus has been reached as to the best way to define and diagnose ADHD. Consequently, it becomes difficult to generate and test hypotheses related to th

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SMITH, SHELLEY D., and COLLEEN A. MORRIS. "Planning studies of etiology." Applied Psycholinguistics 26, no. 1 (2005): 97–110. http://dx.doi.org/10.1017/s0142716405050083.

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Knowledge of the environmental and genetic etiologies of complex cognitive disorders can guide strategies for diagnosis, prevention, and therapy, but disentangling the various causes can be very challenging. Two basic approaches can be used in identifying genetic factors, a top-down approach, in which phenotypic descriptions are used to discover genes that influence those phenotypes, and a bottom-up approach, comparing the phenotypic effects of genes that are known to cause syndromes that include cognitive disabilities. Thorough characterization of phenotypes throughout development is critical

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Işık, Ümit, Faruk Kılıç, Evrim Aktepe, and Bilal Tanrıtanır. "The Relationship between Second-to-Fourth Digit Ratios, Attention-Deficit/Hyperactivity Disorder Symptoms, Aggression, and Intelligence Levels in Boys with Attention-Deficit/Hyperactivity Disorder." Psychiatry Investigation 17, no. 6 (2020): 596–602. http://dx.doi.org/10.30773/pi.2020.0003.

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Objective Observations of sex differences have led some scientists to doubt whether the neuroendocrine system is involved in attentiondeficit/hyperactivity disorder (ADHD) etiology. One of the interesting study subjects in this context is prenatal steroid hormone exposure. The aim of the present study was to replicate and extend previous work by addressing two research questions: 1) Are second-tofourth digit (2D:4D) ratios lower in ADHD than in controls? 2) Is there a correlation between 2D:4D ratios and symptoms of ADHD, aggression and intelligence scores in boys with ADHD?Methods The study i

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Polderman, T. J. C., R. A. Hoekstra, A. A. E. Vinkhuyzen, P. F. Sullivan, S. van der Sluis, and D. Posthuma. "Attentional switching forms a genetic link between attention problems and autistic traits in adults." Psychological Medicine 43, no. 9 (2012): 1985–96. http://dx.doi.org/10.1017/s0033291712002863.

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BackgroundAttention deficit hyperactivity disorder (ADHD) symptoms and autistic traits often occur together. The pattern and etiology of co-occurrence are largely unknown, particularly in adults. This study investigated the co-occurrence between both traits in detail, and subsequently examined the etiology of the co-occurrence, using two independent adult population samples.MethodData on ADHD traits (Inattention and Hyperactivity/Impulsivity) were collected in a population sample (S1, n = 559) of unrelated individuals. Data on Attention Problems (AP) were collected in a population-based family

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23

Baker, Judy. "Attention deficit disorder: A creation of the medical profession?" Journal of Psychologists and Counsellors in Schools 4 (November 1994): 65–80. http://dx.doi.org/10.1017/s1037291100001916.

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They can't sit stilly they don't pay attention to the teacher; they mess around and get into trouble; they try to get others into trouble; they are rude; they get mad when they don't get their way; and they say they can beat everybody up (Whalen & Henker, 1991, p.216). There would not be too many amongst us who have not had first hand experience with a child of this description in a class at some time during our teaching career. Whether this disorder is formally labelled or not, it is difficult to deny that these children exist, and as professionals we should acquaint ourselves with the de

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Боряк, Оксана, та Лариса Одинченко. "МОЛОДШІ ШКОЛЯРІ З ГІПЕРАКТИВНИМ РОЗЛАДОМ ІЗ ДЕФІЦИТОМ УВАГИ В УМОВАХ СУЧАСНОГО ІНКЛЮЗИВНОГО ПРОСТОРУ". Педагогічні науки: теорія, історія, інноваційні технології, № 5-6(99-100) (31 серпня 2020): 12–23. http://dx.doi.org/10.24139/2312-5993/2020.05-06/012-023.

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The article considers peculiarities of teaching, upbringing and development of children with special educational needs in the conditions of the modern educational space – an inclusive education institution. Among modern children of both preschool and primary school age, a special place is occupied by schoolchildren with hyperactivity disorder and attention deficit. The specificity of disorder manifestations has a negative impact on their behavior, causes certain difficulties during assimilation of the educational material, affects the level of formation of the necessary knowledge, skills and a

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Bekle, Bruna. "Review of Research on Teachers’ Knowledge and Attitudes About Attention-Deficit Hyperactivity Disorder (ADHD)." Australasian Journal of Special Education 25, no. 1-2 (2001): 67–85. http://dx.doi.org/10.1017/s1030011200024866.

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ADHD is a neuro‐developmental disorder that is diagnosed in 3 to 6% of the childhood population in a diversity of cultures and a variety of geographical locations. It presents as a persistent pattern of inattention and/or hyperactivity‐impulsivity, with boys being over‐represented by approximately 3 to 1. High levels of comorbidity between ADHD and a number of other disorders, including Oppositional Defiant Disorder, Conduct Disorder, and learning disabilities, have been identified. This review will examine the historical development of the understanding of ADHD, knowledge of its etiology, and

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Tuzun Mutluer, B., T. G. Yener Orum, and S. Sertcelik. "Incidence of Internet addiction in adult attention deficit hyperactivity disorder." European Psychiatry 41, S1 (2017): S396—S397. http://dx.doi.org/10.1016/j.eurpsy.2017.02.457.

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ObjectiveIn this study, it was aimed to determine the internet use properties of Adult Attention Deficit Hyperactivity Disorder (ADHD) patients who were followed-up at Haydarpasa Numune Research and Training Hospital, Psychiatry Department; ADHD Outpatient Clinic.MethodThirty participants who were diagnosed with adult ADHD aged between 18–31 years rated their ADHD symptoms in childhood retrospectively, using Wender Utah Rating Scale. Patients rated current adult ADHD symptoms with the Adult ADHD DSM-IV-Based Diagnostic Screening and Rating Scale (DSRS) and severity of symptoms measured by Adul

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Darwish, Amira Hamed, Tarek Mohamed Elgohary, and Nahla A. Nosair. "Serum Interleukin-6 Level in Children With Attention-Deficit Hyperactivity Disorder (ADHD)." Journal of Child Neurology 34, no. 2 (2018): 61–67. http://dx.doi.org/10.1177/0883073818809831.

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Introduction: Attention-deficit hyperactivity disorder (ADHD) is a common neurobehavioral disorder in children, but its specific etiology and pathophysiology are still incompletely understood. Objectives: This case-control study aimed to measure the level of serum interleukin-6 (IL-6) as a predictor of the immunologic status in children with ADHD, and to study its correlation with severity of symptoms. Subjects and Methods: 60 ADHD children who met the Diagnostic and Statistical Manual of Mental Disorders, 5th Edition, criteria for ADHD and 60 control children were subjected to complete histor

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Plourde, Vickie, Michel Boivin, Mara Brendgen, Frank Vitaro, and Ginette Dionne. "Phenotypic and genetic associations between reading and attention-deficit/hyperactivity disorder dimensions in adolescence." Development and Psychopathology 29, no. 4 (2016): 1215–26. http://dx.doi.org/10.1017/s0954579416001255.

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AbstractMultiple studies have shown that reading abilities and attention-deficit/hyperactivity disorder symptoms, mainly inattention symptoms, are phenotypically and genetically associated during childhood. However, few studies have looked at these associations during adolescence to investigate possible developmental changes. The aim of the study is to examine the genetic and environmental etiology of the associations between inattention and hyperactivity reported by parents, and reading accuracy, reading speed, and word reading in a population-based twin sample (Quebec Newborn Twin Study). Pa

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Kapoor, Vidhi, Raffaele Ferri, Mark Stein, Chris Ruth, Jennifer Reed, and Lourdes Delrosso. "616 Restless sleep and other comorbidities in children with attention deficit hyperactivity disorder." Sleep 44, Supplement_2 (2021): A242. http://dx.doi.org/10.1093/sleep/zsab072.614.

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Abstract Introduction The etiology of restless sleep has recently been identified as secondary to various medical and sleep conditions. Parents of children with ADHD complain of restless sleep and sleep disturbances in their children. We aimed to assess restless sleep causes in our patient population with ADHD and establish the prevalence of restless sleep disorder (RSD), as recently defined. Methods A retrospective study of children with ADHD who underwent polysomnography was carried out. Diagnostic and descriptive information collected for each patient included age, sex, polysomnographic par

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Goulardins, Juliana B., Daniela Rigoli, Melissa Licari, et al. "Attention deficit hyperactivity disorder and developmental coordination disorder: Two separate disorders or do they share a common etiology." Behavioural Brain Research 292 (October 2015): 484–92. http://dx.doi.org/10.1016/j.bbr.2015.07.009.

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ORDAHAN, Banu, and Hatice UĞURLU. "The Role of Attention-Deficit/Hyperactivity Disorder in the Etiology of Osgood Schlatter Disease." Fiziksel Tıp ve Rehabilitasyon Bilimleri Dergisi 23, no. 3 (2020): 158–61. http://dx.doi.org/10.31609/jpmrs.2019-72299.

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Sheppard, D. "Tourette's and comorbid syndromes Obsessive compulsive and attention deficit hyperactivity disorder. a common etiology?" Clinical Psychology Review 19, no. 5 (1999): 531–52. http://dx.doi.org/10.1016/s0272-7358(98)00059-2.

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Sharma, Alok, and Justin Couture. "A Review of the Pathophysiology, Etiology, and Treatment of Attention-Deficit Hyperactivity Disorder (ADHD)." Annals of Pharmacotherapy 48, no. 2 (2013): 209–25. http://dx.doi.org/10.1177/1060028013510699.

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GILGER, JEFFREY W., BRUCE F. PENNINGTON, and JOHN C. DeFRIES. "A Twin Study of the Etiology of Comorbidity: Attention-deficit Hyperactivity Disorder and Dyslexia." Journal of the American Academy of Child & Adolescent Psychiatry 31, no. 2 (1992): 343–48. http://dx.doi.org/10.1097/00004583-199203000-00024.

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35

Frans, Emma Maria. "The Importance of Immune System Diseases in the Etiology of Attention-Deficit/Hyperactivity Disorder." Biological Psychiatry 81, no. 5 (2017): e39-e40. http://dx.doi.org/10.1016/j.biopsych.2016.12.016.

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Türkoğlu, Serhat, Battal Tahsin Somuk, Emrah Sapmaz, and Ayhan Bilgiç. "Effect of adenotonsillectomy on sleep problems, attention deficit hyperactivity disorder symptoms, and quality of life of children with adenotonsillar hypertrophy and sleep-disordered breathing." International Journal of Psychiatry in Medicine 54, no. 3 (2019): 231–41. http://dx.doi.org/10.1177/0091217419829988.

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Objective Chronic adenotonsillar hypertrophy is the most common etiologic reason for upper airway obstruction in childhood and has been found to be associated with a variety of psychiatric disorders and poor quality of life. In the present study, we investigated the impact of adenotonsillectomy on attention deficit hyperactivity disorder symptoms, sleep problems, and quality of life in children with chronic adenotonsillar hypertrophy. Methods The parents of children with chronic adenotonsillar hypertrophy filled out the Conners’s Parent Rating Scale-Revised Short form (CPRS-RS), Children’s Sle

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Peng, Chun-Zi, Julia D. Grant, Andrew C. Heath, Angela M. Reiersen, Richard C. Mulligan, and Andrey P. Anokhin. "Familial influences on the full range of variability in attention and activity levels during adolescence: A longitudinal twin study." Development and Psychopathology 28, no. 2 (2015): 517–26. http://dx.doi.org/10.1017/s0954579415001091.

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AbstractTo investigate familial influences on the full range of variability in attention and activity across adolescence, we collected maternal ratings of 339 twin pairs at ages 12, 14, and 16, and estimated the transmitted and new familial influences on attention and activity as measured by the Strengths and Weaknesses of Attention-Deficit/Hyperactivity Disorder Symptoms and Normal Behavior Scale. Familial influences were substantial for both traits across adolescence: genetic influences accounted for 54%–73% (attention) and 31%–73% (activity) of the total variance, and shared environmental i

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Biederman, J., T. Spencer, A. Lomedico, H. Day, C. R. Petty, and S. V. Faraone. "Deficient emotional self-regulation and pediatric attention deficit hyperactivity disorder: a family risk analysis." Psychological Medicine 42, no. 3 (2011): 639–46. http://dx.doi.org/10.1017/s0033291711001644.

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BackgroundAlthough deficient emotional self-regulation (DESR) is associated with attention deficit hyperactivity disorder (ADHD), little research investigates this association and little is known about its etiology. Family studies provide a method of clarifying the co-occurrence of clinical features, but no family studies have yet addressed ADHD and DESR in children.MethodSubjects were 242 children with ADHD and 224 children without ADHD. DESR was operationalized using an aggregate score ⩾180 and <210 in the anxious/depressed, attention and aggression scales (AAA profile) of the Child Behav

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Saedisomeolia, Ahmad, Mahsa Samadi, Fatemeh Gholami, et al. "Vitamin D’s Molecular Action Mechanism in Attention-Deficit/ Hyperactivity Disorder: A Review of Evidence." CNS & Neurological Disorders - Drug Targets 17, no. 4 (2018): 280–90. http://dx.doi.org/10.2174/1871527317666180501111627.

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Background & Objective: Attention-deficit/hyperactivity disorder (ADHD) is a heterogeneous disorder characterized by hyperactivity, impulsivity and inattention. Children with ADHD have challenges with learning, behavior and psychosocial adjustments, sometimes retained into adulthood. The exact etiology of ADHD is unknown, and the pathophysiology of this disease is complex. Several hypotheses have been raised regarding ADHD pathogenesis, including serotonergic and catecholaminergic signalling pathway dysfunction, neurotropic-related factors, oxidative stress, or neuroinflammation. Vitamin D

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Donzelli, Gabriele, Annalaura Carducci, Agustin Llopis-Gonzalez, et al. "The Association between Lead and Attention-Deficit/Hyperactivity Disorder: A Systematic Review." International Journal of Environmental Research and Public Health 16, no. 3 (2019): 382. http://dx.doi.org/10.3390/ijerph16030382.

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The etiology of Attention-Deficit/Hyperactivity Disorder (ADHD) is complex and multifactorial. Although the development of ADHD symptoms remains to be elucidated, in recent years, epigenetic processes have emerged as candidate mechanisms. Lead is one of the most dangerous environmental pollutants, and it is suspected to be associated with ADHD. The aim of the present study was to review the epidemiological literature currently available on the relation between lead exposure and the diagnosis of ADHD. The PubMed and EMBASE databases were searched from 1 July 2018 up to 31 July 2018. The authors

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Amiri, Shahrokh, Ayyoub Malek, Majid Sadegfard, and Salman Abdi. "Pregnancy-Related Maternal Risk Factors of Attention-Deficit Hyperactivity Disorder: A Case-Control Study." ISRN Pediatrics 2012 (June 5, 2012): 1–5. http://dx.doi.org/10.5402/2012/458064.

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Background. The etiology of attention-deficit hyperactivity disorder (ADHD) is complex.This study was conducted to evaluate the pregnancy-related maternal risk factors of ADHD. Methods. 164 ADHD children attending to Child and Adolescent Psychiatric Clinics compared with 166 normal children selected in a random-cluster method from primary schools. ADHD rating scale and clinical interview based on Schedule for Affective disorders and Schizophrenia for School-Aged Children (K-SADS) were used to diagnose ADHD cases and to select the control group. Results. The mean maternal age at pregnancy, dura

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Willcutt, Erik G., Bruce F. Pennington, and John C. DeFries. "Twin study of the etiology of comorbidity between reading disability and attention-deficit/hyperactivity disorder." American Journal of Medical Genetics 96, no. 3 (2000): 293–301. http://dx.doi.org/10.1002/1096-8628(20000612)96:3<293::aid-ajmg12>3.0.co;2-c.

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Naboychenko, Evgeniya Sergeevna, and Ekaterina Feliksovna Abshilava. "ETIOLOGY, PATHOGENESIS AND CLINICAL MANIFESTATIONS OF ATTENTION DEFICIT HYPERACTIVITY DISORDER AT DIFFERENT PERIODS OF ONTOGENESIS." Pedagogical Education in Russia, no. 1 (2016): 183–87. http://dx.doi.org/10.26170/po16-01-32.

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Malow, Beth, and Angela Maxwell-Horn. "Sleep in Autism." Seminars in Neurology 37, no. 04 (2017): 413–18. http://dx.doi.org/10.1055/s-0037-1604353.

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AbstractAutism spectrum disorder (ASD) is a neurodevelopmental disorder that has increased in prevalence over the last several decades. A significant proportion of children with ASD have comorbid sleep disorders. The interplay between ASD and sleep is multifactorial and bidirectional. There is evidence for physiological differences in ASD that contribute to sleep problems, including sensory overresponsiveness (SOR) and abnormal melatonin production. Comorbidities associated with ASD (attention deficit hyperactivity disorder [ADHD], mood disorders) as well as medications used to treat these com

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Chou, Wen-Jiun, Tai-Ling Liu, Ray C. Hsiao, Yu-Min Chen, Chih-Cheng Chang, and Cheng-Fang Yen. "Caregiver-Attributed Etiologies of Children’s Attention-Deficit/Hyperactivity Disorder: A Study in Taiwan." International Journal of Environmental Research and Public Health 17, no. 5 (2020): 1652. http://dx.doi.org/10.3390/ijerph17051652.

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The aim of this survey study was to examine the etiologies of attention-deficit/hyperactivity disorder (ADHD) attributed by caregivers of Taiwanese children with ADHD, particularly factors affecting such attribution. This study had 400 caregivers of children with ADHD as participants. We examined the caregiver-attributed etiologies of ADHD and factors affecting such attribution. Caregivers completed the self-report questionnaire to rate how likely they perceived various etiologies of ADHD to be; the Affiliate Stigma Scale for the level of affiliate stigma; and the short Chinese version of the

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Lange, Klaus W., Ewelina Makulska-Gertruda, Jakob Reisinger, Thomas-A. Sontag, and Joachim Hauser. "Dietary omega-3 fatty acids and locomotor activity in an animal model of attention deficit hyperactivity disorder (ADHD)." Functional Foods in Health and Disease 3, no. 6 (2013): 223. http://dx.doi.org/10.31989/ffhd.v3i6.52.

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Background: Attention deficit hyperactivity disorder (ADHD) is the most frequent behavioral disorder of childhood and is characterized by hyperactivity, attention problems and impulsivity. Pharmacological and behavioral therapies have been shown to be effective. In addition, the role of dietary compounds in the etiology and possibly the treatment of ADHD has attracted increasing attention. For example, the lack of dietary essential fatty acids has been suggested to be associated with symptoms of ADHD in humans.Objective: To investigate the effects of omega-3 polyunsaturated fatty acids (n-3 PU

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Liu, Lu, Jia Cheng, Haimei Li, Li Yang, Qiujin Qian, and Yufeng Wang. "The possible involvement of genetic variants of NET1 in the etiology of attention-deficit/hyperactivity disorder comorbid with oppositional defiant disorder." Journal of Child Psychology and Psychiatry 56, no. 1 (2014): 58–66. http://dx.doi.org/10.1111/jcpp.12278.

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Mamenko, M. Ye. "Attention Deficit Hyperactivity Disorder in Children: Current Views on the Etiology, Pathogenesis, Approaches to the Correction." CHILD`S HEALTH, no. 5.65 (September 29, 2015): 7–13. http://dx.doi.org/10.22141/2224-0551.5.65.2015.74874.

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Makulska-Gertruda, Ewelina, Joachim Hauser, Thomas A. Sontag, and Klaus W. Lange. "The effects of dietary omega-3 polyunsaturated fatty acid supplementation on attention and impulsivity in an animal model of attention deficit/hyperactivity disorder (ADHD)." Functional Foods in Health and Disease 4, no. 7 (2014): 285. http://dx.doi.org/10.31989/ffhd.v4i7.4.

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Background: Attention deficit/hyperactivity disorder (ADHD) is one of the commonest psychiatric disorders in children and adolescents. The main symptoms of ADHD are hyperactivity, inattention and impulsivity. Both etiology and neurobiological basis of ADHD are unknown. In this context, long-chain polyunsaturated fatty acids (LC-PUFAs), especially omega-3 (n-3) PUFAs, have become a focus of interest. The symptoms of ADHD have been suggested to be associated with a deficiency of n-3 PUFAs. In addition, the impact of a supply of dietary n-3 PUFAs in the treatment of ADHD has frequently been discu

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Levy, Amanda M., Peristera Paschou, and Zeynep Tümer. "Candidate Genes and Pathways Associated with Gilles de la Tourette Syndrome—Where Are We?" Genes 12, no. 9 (2021): 1321. http://dx.doi.org/10.3390/genes12091321.

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Gilles de la Tourette syndrome (GTS) is a childhood-onset neurodevelopmental and -psychiatric tic-disorder of complex etiology which is often comorbid with obsessive-compulsive disorder (OCD) and/or attention deficit hyperactivity disorder (ADHD). Twin and family studies of GTS individuals have shown a high level of heritability suggesting, that genetic risk factors play an important role in disease etiology. However, the identification of major GTS susceptibility genes has been challenging, presumably due to the complex interplay between several genetic factors and environmental influences, l

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