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Relevant bibliographies by topics / Autoimmune process

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Academic literature on the topic 'Autoimmune process'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Autoimmune process"

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Barker, Roger A., and Anthony P. Cahn. "Parkinson's Disease: An Autoimmune Process." International Journal of Neuroscience 43, no. 1-2 (1988): 1–7. http://dx.doi.org/10.3109/00207458808985773.

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Kozlov, V. A. "Tumorigenesis as an autoimmune process." Medical Immunology (Russia) 27, no. 4 (2025): 703–12. https://doi.org/10.15789/1563-0625-taa-3152.

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The data presented in the article reasonably indicate that the immune system responses during the development of autoimmune pathology and in the process of tumor growth are, in fact, autoimmune responses directed at autoantigens of various tissues and organs, and tumor-associated antigens. The latter, in addition to the tumor itself, are also expressed by the cells of normal organs and tissues. At the same time, the differences are characterized by insufficient activity of suppressor cells in the first variant, and enhanced activity in the second variant. One should take into account a potenti

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Герасун, Б. А., Р. Ю. Грицко, О. Б. Герасун, and Р. А. Копець. "New method for the treatment autoimmune process." Family Medicine, no. 2(64) (March 21, 2016): 47–51. http://dx.doi.org/10.30841/2307-5112.2(64).2016.101899.

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Fadeev, V. V., I. V. Shevchenko, and G. A. Melnichenko. "Autoimmune polyglandular syndromes." Problems of Endocrinology 45, no. 1 (2019): 47–54. http://dx.doi.org/10.14341/probl11708.

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Autoimmune polyglandular syndromes (APS) are the primary defeat of the autoimmune process of 2 peripheral endocrine glands and more, leading, as a rule, to their insufficiency, often combined with various organ-specific non-endocrine autoimmune diseases.

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Gopal, KVT, GRaghu Rama Rao, YHari Kishan Kumar, MV Appa Rao, P. Vasudev, and Srikant. "Vitiligo: A part of a systemic autoimmune process." Indian Journal of Dermatology, Venereology and Leprology 73, no. 3 (2007): 162. http://dx.doi.org/10.4103/0378-6323.32710.

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Munoz, A., R. Martinez, M. Leon, et al. "Study on autoimmune mechanisms in the neurological process." Annals of the Rheumatic Diseases 70, Suppl 2 (2011): A91. http://dx.doi.org/10.1136/ard.2010.149021.23.

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Galenok, F. A., and Ye A. Zhuk. "Autoimmune process markers in insulin-dependent diabetes mellitus." Problems of Endocrinology 43, no. 3 (2019): 13–16. http://dx.doi.org/10.14341/probl10387.

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Quantitative and functional parameters of the monocyte and В-cell immunity were assessed in patients with insulin-dependent diabetes over the course of disease. Studies of the monocyte component in subjects predisposed to disease and of the humoral one in those with clinical manifestations are valuable for predicting the autoimmune process. Signs predicting a poor outcome are increase of the count of Fc-positive cells, decrease of NBT reduction of monocytes in subjects without signs of the disease, high levels of immunoglobulin G in manifest disease, and increase of IgM and B-lymphopcyte count

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Chhabra, Seema, Biman Saikia, Yashwant Kumar, and RanjanaW Minz. "Anti-centriole antibody: An infectious or autoimmune process?" Indian Journal of Allergy, Asthma and Immunology 29, no. 2 (2015): 84. http://dx.doi.org/10.4103/0972-6691.178274.

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Stys, Peter K. "Multiple Sclerosis: Autoimmune Disease or Autoimmune Reaction?" Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 37, S2 (2010): S16—S23. http://dx.doi.org/10.1017/s0317167100022393.

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ABSTRACT:Multiple sclerosis (MS) is traditionally considered an autoimmune inflammatory demyelinating disease of the central nervous system (CNS) with much knowledge available to support this view. However, this characterization implies that the primary event is an aberrant immune response directed at CNS antigens, promoting inflammation and later driving progressive axo-glial degeneration. Trials with potent anti-inflammatory agents and detailed neuropathological studies raise questions about this sequence of events. This hypothetical paper argues that MS may be primarily a “cytodegenerative”

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Mayorova, M. A., N. N. Petrova, U. I. Stroev, L. P. Churilov, and Y. Shoenfeld. "Interrelation of auto-immune process, endocrine disorders and depression." V.M. BEKHTEREV REVIEW OF PSYCHIATRY AND MEDICAL PSYCHOLOGY, no. 1 (April 2, 2020): 8–19. http://dx.doi.org/10.31363/2313-7053-2020-1-8-19.

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Disturbances in immune and endocrine regulation are closely interrelated with development of psychiatric diseases, including affective disorders. Depression may be a characteristic of at least 23 autoimmune diseases. Negative influence on the depression development have: an increased level of proinflammatory cytokines, impairment of T-regulatory cells, kynurenine pathway shift towards production of its potentially neurotoxic metabolites, overproduction of stress hormones, hyperleptinemia, hyperprolactinemia, hypothyroidism, as well as vitamin D, calcidiol and calcitriol deficiency. In this rev

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Books on the topic "Autoimmune process"

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Keith, Lierre. The vegetarian myth: Food, justice and sustainability. Flashpoint Press, 2009.

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Lawrenson, John. Pathogenesis: from acute rheumatic fever to rheumatic heart disease. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0280.

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Rheumatic heart disease is considered to be an autoimmune disease; the trigger of the process is a streptococcal throat infection which then initiates both a humeral and a cellular immune response in environmentally and genetically susceptible individuals.

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Marshall, Tarnya, and Rita Abdulkader. Anti-rheumatic drugs in pregnancy and lactation. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0097.

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Anti-rheumatic drugs in pregnancy and lactation are increasingly a common clinical dilemma. With the shift towards early, aggressive control of autoimmune diseases and with the advent of newer therapeutic agents, there is a need to understand the effects of these medicines in pregnancy and lactation, on fertility in both men and women, and on the process of spermatogenesis, in order to understand the risk of teratogenesis. Although there are some limited data available for the use of anti-rheumatic drugs in pregnancy and lactation, much of our knowledge is derived from animal models and from l

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Marshall, Tarnya, Rita Abdulkader, and Poonam Sharma. Antirheumatic drugs in pregnancy and lactation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0097_update_003.

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Antirheumatic drugs in pregnancy and lactation are increasingly a common clinical dilemma. With the shift towards early, aggressive control of autoimmune diseases and with the advent of newer therapeutic agents, there is a need to understand the effects of these medicines in pregnancy and lactation, on fertility in both men and women, and on the process of spermatogenesis, in order to understand the risk of teratogenesis. Although there are some limited data available for the use of antirheumatic drugs in pregnancy and lactation, much of our knowledge is derived from animal models and from lim

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Gay, Steffen, and Michel Neidhart. Epigenetics. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0039.

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In higher eukaryotic organisms epigenetic modifications are crucial for proper chromatin folding and thereby proper regulation of gene expression. Epigenetics include DNA methylation, histone modifications, and microRNAs. First described in tumors, the involvement of aberrant epigenetic modifications has been reported also in other diseases, i.e. metabolic, psychiatric, inflammatory, and autoimmune. Deregulation of epigenetic mechanisms occurred in patients with rheumatoid arthritis, systemic lupus erythematosus, and scleroderma. Many questions remain: e.g. what is the cause of these epigeneti

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Jayne, David. The patient with vasculitis. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0157.

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The definition of vasculitis syndromes relies on clinical, serological, and pathological descriptions. A number of primary syndromes are recognized, of presumed autoimmune aetiology, but vasculitis may also occur as a secondary disease process. The heterogeneity of clinical presentation, low specificity of many clinical features, and mimics of other diseases complicate vasculitis diagnosis. The kidney is the most common severe manifestation for small vessel vasculitis syndromes and the severity of renal involvement predicts end-stage renal failure risk and death. Suspicion of vasculitis is key

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Deveza, Leticia A., Changhai Ding, Xingzhong Jin, Xia Wang, Zhaohua Zhu, and David J. Hunter. Laboratory tests. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0019.

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In clinical practice, laboratory tests play a major role in the investigation of underlying diseases predisposing to osteoarthritis (OA) (e.g. metabolic and genetic causes) and in the differential diagnosis of other joint disorders, particularly autoimmune and crystal-related arthropathies. In terms of diagnosis and management of OA, there is still no recommendation in guidelines for the use of laboratory tests. However, an increasing number of potential applications of laboratory tests in OA are emerging. In this regard, research investigating the role of vitamin D and other substances such a

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Reintjes, Staci, and Susie Peterson. Rhinosinusitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0012.

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Rhinosinusitis is inflammation of the nasal passages and paranasal sinuses, commonly caused by allergies or viral infection. Sinusitis occurs after the development of rhinitis or inflammation of the nasal passages. Rhinitis is most commonly caused by allergens, but it also can be to the result of an infectious or autoimmune process. For rhinitis to progress to rhinosinusitis, there must be obstruction within the ostiomeatal complex, which is the draining center for the maxillary, anterior ethmoid, and frontal sinuses. History and physical exam are more specific than imaging for diagnosis. Comp

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van der Vlag, Johan, and Jo H. M. Berden. The patient with systemic lupus erythematosus. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0161.

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Systemic lupus erythematosus (SLE) is a systemic autoimmune disease with various clinical manifestations. The hallmark of SLE is the presence of antibodies against nuclear constituents, such as double-stranded (ds)DNA, histones, and nucleosomes. Local deposition of antinuclear antibodies in complex with nuclear autoantigens induces serious inflammatory conditions that can affect several tissues and organs, including the kidney.The levels of antinucleosome and anti-dsDNA antibodies seem to correlate with glomerulonephritis and these autoantibodies can often be detected years before the patient

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Eljaafari, Assia, and Pierre Miossec. Cellular side of acquired immunity (T cells). Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0049.

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The adaptive T-cell response represents the most sophisticated component of the immune response. Foreign invaders are recognized first by cells of the innate immune system. This leads to a rapid and non-specific inflammatory response, followed by induction of the adaptive and specific immune response. Different adaptive responses can be promoted, depending on the predominant effector cells that are involved, which themselves depend on the microbial/antigen stimuli. As examples, Th1 cells contribute to cell-mediated immunity against intracellular pathogens, Th2 cells protect against parasites,

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Book chapters on the topic "Autoimmune process"

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Ingram, W., Y. Kordasti, and G. J. Mufti. "MDS as an Autoimmune Process." In The Myelodysplastic Syndromes. Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0440-4_9.

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Russo, E., R. A. Jackson, F. Dotta, et al. "Type I Diabetes as a “Mendelian” and “Regulated” Immune Process." In The Molecular Biology of Autoimmune Disease. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75133-2_34.

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Ma, Bin, Chaomin Li, Brian T. Hopkins, et al. "Discovery and Process Development of BIIB068: A Reversible Bruton’s Tyrosine Kinase (BTK) Inhibitor for the Treatment of Autoimmune Diseases." In ACS Symposium Series. American Chemical Society, 2020. http://dx.doi.org/10.1021/bk-2020-1369.ch010.

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Sarukhan, Adelaida, Henri-Jean Garchon, and Claude Carnaud. "An Analysis of T cell receptor diversity in the NOD mouse: What can it tell us about the autoimmune process?" In Autoimmunity: Experimental Aspects. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78779-9_16.

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Ragbetli, Murat Cetin, and Aysenur Kaya. "Histopathological Changes in Diabetic Pancreas." In Current Multidisciplinary Approach to Diabetes Mellitus Occurrence Mechanism. Nobel Tip Kitabevleri, 2023. http://dx.doi.org/10.69860/nobel.9786053359104.3.

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Histopathological changes in the diabetic pancreas are characterized by several key alterations that impact its structure and function. In type 1 diabetes mellitus (T1DM), autoimmune destruction of insulin-producing beta cells within the pancreatic islets results in their selective loss, termed insulitis. This process involves infiltration of immune cells such as T lymphocytes and macrophages into the islets, leading to progressive beta cell destruction and ultimately insulin deficiency. In contrast, type 2 diabetes mellitus (T2DM) is associated with insulin resistance and eventual beta cell d

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Cheng, Yougan, Ronny Straube, Abed E. Alnaif, Lu Huang, Tarek A. Leil, and Brian J. Schmidt. "Virtual Populations for Quantitative Systems Pharmacology Models." In Methods in Molecular Biology. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2265-0_8.

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AbstractQuantitative systems pharmacology (QSP) places an emphasis on dynamic systems modeling, incorporating considerations from systems biology modeling and pharmacodynamics. The goal of QSP is often to quantitatively predict the effects of clinical therapeutics, their combinations, and their doses on clinical biomarkers and endpoints. In order to achieve this goal, strategies for incorporating clinical data into model calibration are critical. Virtual population (VPop) approaches facilitate model calibration while faced with challenges encountered in QSP model application, including modelin

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"Immune Responses, Autoimmune Diseases, and Therapeutic Relevance of Immunity." In Progress, Pioneers and Process. WORLD SCIENTIFIC, 2018. http://dx.doi.org/10.1142/9789813270589_0008.

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Weetman, Anthony P. "Autoimmune thyroid disease." In Oxford Textbook of Endocrinology and Diabetes. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235292.003.3136.

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Along with neoplasia, autoimmunity is the most common cause of endocrine disease and, of this group of disorders, thyroid autoimmunity is the most frequent. Conversely, the autoimmune thyroid diseases are the most common organ-specific or nonorgan-specific autoimmune conditions affecting any site. This prevalence, the ease of access to the target organ, the often slow progression of disease, and the historical legacy of being the first distinctive autoimmune process to be defined, have ensured that there is now a reasonable understanding of the main factors involved in pathogenesis. This chapt

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Weetman, Anthony P. "Autoimmune Thyroid Disease." In Oxford Textbook of Endocrinology and Diabetes 3e, edited by John A. H. Wass, Wiebke Arlt, and Robert K. Semple. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198870197.003.0051.

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Along with neoplasia, autoimmunity is the most common cause of endocrine disease, and of this group of disorders, thyroid autoimmunity is the most frequent. Conversely, the autoimmune thyroid diseases are the most common organ-specific or non-organ-specific autoimmune conditions affecting any site. This prevalence, the ease of access to the target organ, the often slow progression of disease, and the historical legacy of being the first distinctive autoimmune process to be defined, have ensured that there is now a reasonable understanding of the main factors involved in pathogenesis. This chap

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Brody, Tom. "Immunosuppression, Drug-Induced Hypersensitivity Reactions, and Drug-Induced Autoimmune Reactions." In FDA's Drug Review Process and the Package Label. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-12-814647-7.00009-9.

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Conference papers on the topic "Autoimmune process"

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Ghanem, Fred, Purnima M. Kodate, Gerard M. Capellades, and Kirti M. Yenkie. "Optimal Design of Antibody Extraction Systems using Protein A Resin with Multicycling." In Foundations of Computer-Aided Process Design. PSE Press, 2024. http://dx.doi.org/10.69997/sct.170492.

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Antibody therapies are important in treating life-threatening ailments such as cancer and autoimmune diseases. Purity of the antibody is essential for successful applications and Protein A selective resin extraction is the standard step for antibody recovery. Unfortunately, such resins can cost up to 30% of the total cost of antibody production. Hence, the optimal design of this purification step becomes a critical factor in downstream processing to minimize the size of the column needed. An accurate predictive model, as a digital twin representing the purification process, is necessary where

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Abazova, Z. Kh, A. B. Ivanov, I. Kh Borukaeva, and K. Yu Shkhagumov. "Normobaric interval hypoxytherapy in the correction of neuroimmunoendocrine disorders in children and adolescents with autoimmune thyroiditis." In VIII Vserossijskaja konferencija s mezhdunarodnym uchastiem «Mediko-fiziologicheskie problemy jekologii cheloveka». Publishing center of Ulyanovsk State University, 2021. http://dx.doi.org/10.34014/mpphe.2021-3-6.

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130 children and adolescents aged 6 to 16 years with a diagnosis of autoimmune thy-roiditis (AIT) at the stage of hypothyroidism underwent a course of hypoxytherapy. The high ef-ficiency of the normobaric interval hypoxic training method in the treatment of hypothyroidism on the background of AIT is shown. An increase in the function and number of CD8+ cells after a course of hypoxytherapy prevents the progression of the autoimmune process and promotes the restoration of thyroid function, which in turn leads to positive dynamics in the neurological status of patients: improved indicators of me

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Sharma, Puja, Kevin Sheets, and Amrinder S. Nain. "The Influence of Polymeric Fiber Stiffness and Alignment on Cytoplasmic Bleb Dynamics and Migration of Glioblastoma Multiforme Cells." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80923.

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Cell migration is a tightly regulated phenomenon necessary for regular physiologic processes such as wound healing, immune response, embryonic development, growth, and regeneration [1–3]. Consequences of abnormal migratory behaviors include autoimmune diseases and metastasis during cancer progression [4, 5]. Described as one of the hallmarks of cancer, metastasis is a complex multistep process, and is responsible for 90% of cancer deaths in humans. A better understanding of the process of metastasis is of paramount importance in developing efficient cancer treatment therapies and drugs [6].

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Silveira, Wellington, Rafael Korb, Graçaliz Dimuro, and Rodrigo de Bem. "UniMRI: Unified Repository of Magnetic Resonance Images for Multiple Sclerosis Diagnosis." In Workshop de Visão Computacional. Sociedade Brasileira de Computação - SBC, 2021. http://dx.doi.org/10.5753/wvc.2021.18912.

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Multiple sclerosis is an autoimmune disease that affects the central nervous system, destroying myelin. To detect multiple sclerosis, you need to have MRI scans so you can see the areas where myelin has been damaged. This analysis is complex and costly due to the time required to assess injuries. The use of machine learning is desirable as these exams are taken periodically. However, the number of public databases present in the literature containing patients with multiple sclerosis is small when compared to the amount of data needed to train deep neural networks. Thus, the objective of this w

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Moraes, Marianna, Fabiano Abrantes, José Luiz Pedroso, and Orlando Graziani Povoas Barsottini. "Etiological evaluation of hypertrophic pachymeningitis in a tertiary general neurology department." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.665.

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Introduction: Hypertrophic pachymeningitis (HP) is the thickening of the cranial or spinal dura. This process causes cranial nerve palsy, vascular events (stroke and venous thrombosis), and intracranial hypertension. The diagnosis of HP is disclosed by magnetic resonance imaging, showing dural thickening and contrast enhancement. Several etiologies are possible for HP, but infection and autoimmune diseases are the most frequent. The gold standard for diagnosis is the dural biopsy, but cerebrospinal fluid (CSF), blood and other tissues analysis are essential guides to the correct diagnosis. A q

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Silva, Gustavo Figueiredo da, Caroline Figueiredo da Silva, Washigton Luiz Gomes de Medeiros Junior, and Marcus Vinícius Magno Gonçalves. "Anti -Iglon5 Syndrome: What we know so far? A non-systematic review." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.237.

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Background: The first report of Anti-IgLON5 syndrome was in 2014. AntiIgLON5 antibodies have a prevalence of 12 in 150,000 patients per year. However, considering the unreported patients, the prevalence can be much higher. Objectives: Provide an overview of the current knowledge of Anti- IgLON5 syndrome. Design and setting: Narrative review. Methods: Non- systematic review on Pubmed database. Results: The IgLON proteins are a family of cell adhesion molecules and the presence of antibodies against IgLON5 is crucial for the AntiIgLON5 Syndrome diagnosis. This syndrome has an expanded clinical s

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Coppetti, Liara Eickhoff, Lana Caroline Palaver Dall’Ago Dall’Ago, and Andressa Wierzbicki. "Guillain-Barre related to Covid-19." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.525.

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Background: COVID-19 is a disease that affects the nervous system as proven by the presence of symptoms such as headache, nausea, dizziness, myalgia, anosmia and ageusia - and can cause numerous neurological complications. These complications include Guillain-Barré Syndrome, an autoimmune polyneuropathy usually caused by a previous infectious process. Its main clinical manifestation is asthenia in the limbs, in an ascending form. Objectives: Review studies that suggest relation between COVID-19 infection and Guillain-Barré Syndrome. Methods: A systematic review was carried out in April of 2021

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Almeida, Rafael de, Lorena Dias Araujo, Sayuri Aparecida Hirayama, et al. "Neuromyelitis optica in childhood: a case report." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.474.

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Introduction: Neuromyelitis optica (NMO) is a relatively rare inflammatory disease that primarily affects the spinal cord and optic nerves. Objectives: To report an atypical case of NMO in the child, with brainstem syndrome. Case report: A 12-year-old patient, 20 days after Pfizer vaccination for severe acute respiratory syndrome coronavirus-19 (SARS-COV-19), started with asymmetrical ascending flaccid paraparesis, with global arreflexia, ataxia, dysphagia and dysarthria. On admission, after 2 months, thought of acute polyradiculoneuritis, immunoglobulin was administered without improvement. H

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Eldor, A., M. Bar-Ner, L. Wasserman, Y. matzner, Z. Fuks, and I. Viodavsky. "HEPARIN AND NON-ANTICOAGULANT HEPARINS INHIBIT HEPARANASE ACTIVITY IN NORMAL AND MALIGNANT CELLS:POSSIBLE THERAPEUTIC USE IN PREVENTION OF EXTRAVASATION AND DISSEMINATION OF BLOOD BORNE CELLS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643664.

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Degradation of vascular subendothelium occurs in_vivo during the process of inflammation and tumor invasion. Various observations suggest that the capacity of some blood-borne cells to extravasate may depend in part on their ability to express hepara-nase activity. Incubation of human platelets, human nc-utrophils, or highly metastatic mouse lymphoma cells with sulfate-labeled extracellular matrix (ECM) results in heparanase mediated release of labeled heparan sulfate cleavage fragments (0.5<Kav<0.85 on Sepharose 5B) (J. Clin.Invest. 74: 1842 and 76: 1306; Cancer Res. 43: 2704). The pres

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Slichter, Sherrill J. "PATHOPHYSIOLOGY OF THROMBOCYTOPENIA AND RESULTANT CLINICAL INDICATIONS FOR PLATELET TRANSFUSION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643996.

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Careful evaluation of platelet survival data in normal individuals and patients with thrombocytopeniasecondary to marrow aplasia has demonstrated that platelets are lost from circulation by two mechanisms a fixed fraction of platelets, amounting to approxi mately 7,100 platelets/ul/day, are lost randomly while the remaining platelets are removed by senescent mechanisms. At platelet counts of <100,000/ul, platelet survival becomes progressively shorter as the fixed platelet loss becomes a proportionately greater fraction of the circulating platelets. Thus, there is a direct relationship betw

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Reports on the topic "Autoimmune process"

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Gontar, I. P., O. I. Emelyanova, O. A. Rusanova, N. I. Emelyanov, A. N. Krasilnikov, and I. A. Zborovskaya. The sequence of involvement of the protein components of connective tissue in autoimmune inflammation in the scleroderma process. Academy of Natural Knowledge, 2019. http://dx.doi.org/10.18411/2070-7428-2019-1-29267.

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