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Journal articles on the topic 'Autoimmune Uveitis'

Author: Grafiati

Published: 3 June 2025

Last updated: 31 July 2025

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1

Verma, Ashutosh, S. R. Rathinam, C. Gowri Priya, V. R. Muthukkaruppan, Brian Stevenson, and John F. Timoney. "LruA and LruB Antibodies in Sera of Humans with Leptospiral Uveitis." Clinical and Vaccine Immunology 15, no. 6 (2008): 1019–23. http://dx.doi.org/10.1128/cvi.00203-07.

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ABSTRACT Uveitis can be a serious complication of leptospirosis. Previous studies indicated that the leptospiral lipoproteins LruA and LruB are expressed in the eyes of uveitic horses and that antibodies directed against those proteins show in vitro cross-reactivity with components of equine lens, ciliary body, and/or retina. We now demonstrate that sera from a significant proportion of humans who have leptospiral uveitis also contain antibodies against LruA and LruB. Different categories of nonleptospiral uveitis and autoimmune uveitis were also screened; patients diagnosed with Fuchs uveitis

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Lorenz, Lea, Barbara Amann, Sieglinde Hirmer, Roxane L. Degroote, Stefanie M. Hauck, and Cornelia A. Deeg. "NEU1 is more abundant in uveitic retina with concomitant desialylation of retinal cells." Glycobiology 31, no. 7 (2021): 873–83. http://dx.doi.org/10.1093/glycob/cwab014.

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Abstract Desialylation of cell surface glycoproteins carried out by sialidases affects various immunological processes. However, the role of neuraminidase 1 (NEU1), one of the four mammalian sialidases, in inflammation and autoimmune disease is not completely unraveled to date. In this study, we analyzed the retinal expression of NEU1 in equine recurrent uveitis (ERU), a spontaneous animal model for autoimmune uveitis. Mass spectrometry revealed significantly higher abundance of NEU1 in retinal Müller glial cells (RMG) of ERU-diseased horses compared to healthy controls. Immunohistochemistry u

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Plekhanov, A. N., A. S. Fomina, O. P. Sverkunova, and J. V. Ivanova. "Autoimmune Uveitis. Review." Ophthalmology in Russia 16, no. 1 (2019): 5–11. http://dx.doi.org/10.18008/1816-5095-2019-1-5-11.

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For many years in ophthalmology there is a complex intractable problem associated with inflammation of the vascular membrane of the eye, the so-called uveitis. Experts around the world are trying to understand this heterogeneous pathology of the visual organ, in a short time, which can lead to a sharp decrease in visual acuity, the development of severe complications and further disability. The number of patients with inflammation of the vascular tract is growing rapidly every year, the age is younger. The clinical picture of this pathology has changed slightly. Often began to manifest itself

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Commodaro, Alessandra Gonçalves, Luciana de Deus Vieira de Moraes, Denise Vilarinho Tambourgi, Rubens Belfort Jr., Osvaldo Augusto Sant’Anna, and Luiz Vicente Rizzo. "Autoimmune uveitis: study of treatment therapies." Einstein (São Paulo) 8, no. 1 (2010): 117–21. http://dx.doi.org/10.1590/s1679-45082010rb1416.

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ABSTRACT Experimental autoimmune uveitis is an organ-specific T-cell mediated autoimmune disease characterized by inflammation and consequent destruction of the neural retina and adjacent tissues. Inflammation in experimental autoimmune uveitis may be induced in rodents by immunization with retinal antigens, such as interphotoreceptor retinoid-binding protein. We present a review of experimental studies that correlate primary immunobiological functions with this chronic disease and the possible use of molecules for the treatment of autoimmune uveitis.

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Muntean, Laura, and Simona Rednic. "TREATMENT OF UVEITIS ASSOCIATED WITH SYSTEMIC AUTOIMMUNE DISEASES IN ADULTS: A CONCISE OVERVIEW." Romanian Journal of Rheumatology 26, no. 4 (2017): 154–59. http://dx.doi.org/10.37897/rjr.2017.4.3.

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Uveitis represents a heterogenous group of inflammatory ocular diseases which may cause vision loss. Association of uveitis with arthritis may occur in many systemic autoimmune diseases, including spondyloarthritis, juvenile idiopathic arthritis, Behҫet’s disease and sarcoidosis. Recent advances in the field of autoimmune diseases have provided new insights and developments of diverse therapies for noninfectious uveitis. However, the optimal treatment strategy for patients with uveitis remains to be defined. Topical and/or systemic corticosteroids are the first-line therapy of autoimmune uveit

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Alawneh, Khaldoon M., Omar A. Saleh, Mahmoud M. Smadi, et al. "Pattern of Uveitis in a Tertiary Hospital in North Jordan and the Impact of Behcet’s Disease." Journal of Ophthalmology 2023 (August 8, 2023): 1–6. http://dx.doi.org/10.1155/2023/2076728.

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Aim. The purpose of this study is to assess the prevalence of autoimmune-mediated uveitis in relation to other diseases and to describe the clinical patterns of uveitis in a single tertiary hospital in north Jordan. Methods. A cross-sectional retrospective review was performed. We included all patients diagnosed with uveitis in King Abdullah University Hospital (KAUH) ophthalmology clinic and/or patients referred to KAUH rheumatology clinics for evaluation of suspected autoimmune mediated uveitis or for difficult to treat uveitis. This included patients from January 2015 to January 2019. Data

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7

Ruggieri, Simona, Maria Antonia Frassanito, Rosanna Dammacco, and Silvana Guerriero. "TregLymphocytes in Autoimmune Uveitis." Ocular Immunology and Inflammation 20, no. 4 (2012): 255–61. http://dx.doi.org/10.3109/09273948.2012.681830.

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8

Horai, Reiko, and Rachel R. Caspi. "Cytokines in Autoimmune Uveitis." Journal of Interferon & Cytokine Research 31, no. 10 (2011): 733–44. http://dx.doi.org/10.1089/jir.2011.0042.

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9

WHITCUP, SCOTT M., and ROBERT B. NUSSENBLATT. "Treatment of Autoimmune Uveitis." Annals of the New York Academy of Sciences 696, no. 1 (2006): 307–18. http://dx.doi.org/10.1111/j.1749-6632.1993.tb17166.x.

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10

Caspi, Rachel R. "Mechanisms Underlying Autoimmune Uveitis." Drug Discovery Today: Disease Mechanisms 3, no. 2 (2006): 199–206. http://dx.doi.org/10.1016/j.ddmec.2006.05.006.

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11

Feltkamp, T. E. W. "Ocular autoimmune diseases (excluding experimental autoimmune uveitis)." Experimental Eye Research 55 (September 1992): 30. http://dx.doi.org/10.1016/0014-4835(92)90316-k.

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12

Klímová, Aneta, P. Seidler Štangová, J. Heissigerová, P. Svozílková, and T. Kučera. "Mycophenolate Mofetil and Cyclophosphamide Treatments Suppress Inflammation Intensity in an Experimental Model of Autoimmune Uveitis." Folia Biologica 60, no. 5 (2014): 228–34. http://dx.doi.org/10.14712/fb2014060050228.

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In human, autoimmune uveitis is a leading cause of visual disability and ranks with diabetic retinopathy as a major source of blind registrations in developed countries. Since most cases of non-infectious uveitis are considered to be autoimmune or at least immune-mediated, the management of such patients has rested on appropriate immunosuppression. Some patients, however, despite maximal immunotherapy, fail to respond or are seriously intolerant of the drug therapies. Since its establishment 20 years ago, the model of experimental autoimmune uveoretinitis has served as a useful template for no

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Shirinsky, I. V., and V. S. Shirinsky. "Autoimmune uveitis: risk factors and issues of immunopathogenesis." Medical Immunology (Russia) 27, no. 1 (2025): 7–20. https://doi.org/10.15789/1563-0625-aur-2956.

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We present recent advances in studying the mechanisms of susceptibility to immune-mediated uveitis (IM) and its pathogenesis. Animal models of human uveitis are described in details. Those include the best characterized models of experimental anterior uveitis (endotoxin-induced uveitis and experimental autoimmune anterior uveitis). As a result of these studies, some relevant transcription factors were detected, such as STAT3, Interferon regulatory factor 4, 8; regulatory proteins, e.g., suppressors of cytokine signaling 1, 3 (SOCS1, SOCS3) and cytokine signaling pathways that regulate the deve

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Deeg, Cornelia A., Albert J. Raith, Barbara Amann, et al. "CRALBP is a Highly Prevalent Autoantigen for Human Autoimmune Uveitis." Clinical and Developmental Immunology 2007 (2007): 1–6. http://dx.doi.org/10.1155/2007/39245.

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Cellular retinaldehyde binding protein (CRALBP) is an autoantigen in spontaneous equine recurrent uveitis. In order to test whether CRALBP contributes to human autoimmune uveitis, the specificity of antibodies from human uveitis patient's sera was first evaluated in two-dimensional (2D) Western blot analysis. Subsequent identification of the immunoreactive proteins by mass spectrometry resulted in the identification of CRALBP as a putative autoantigen. Additionally, sera from human uveitis and control patients were by Western blot using purified human recombinant CRALBP. Anti-CRALBP autoantibo

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15

Santini, Marija, Mirna Vučemilo, Ivan Jovanović, et al. "Vogt-Koyanagi-Harada syndrome." Infektološki glasnik 39, no. 4 (2020): 129–32. http://dx.doi.org/10.37797/ig.39.4.5.

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Vogt-Koyanagi-Harada syndrome is an autoimmune, multisystem disease and important cause of uveitis. In this case report we address the significance of reliable diagnostics and including autoimmune diseases in differential diagnosis of uveitis and meningitis syndrome.

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Caspi, Rachel, Reiko Horai, Jun Chen, et al. "Retina-specific T regulatory cells accumulate in the eye during autoimmune uveitis and act to control inflammation (115.22)." Journal of Immunology 186, no. 1_Supplement (2011): 115.22. http://dx.doi.org/10.4049/jimmunol.186.supp.115.22.

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Abstract Experimental autoimmune uveitis (EAU) induced in mice by immunization with the retinal antigen IRBP is a model of human autoimmune uveitis. We examined whether T regulatory (Treg) cells found in uveitic eyes are (i) IRBP specific, (ii) functionally suppressive, and (iii) may play a role in natural resolution of disease. EAU was induced in B10.RIII FoxP3-GFP reporter mice with IRBP161-180. IRBP-specific T cells were detected by staining with IRBP-MHC class II dimers. In uveitic eyes, progressive increase of FoxP3+ Treg to T effector (Teff) ratio correlated with resolution of disease. A

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17

Hara, Y., R. R. Caspi, B. Wiggert, C. C. Chan, G. A. Wilbanks, and J. W. Streilein. "Suppression of experimental autoimmune uveitis in mice by induction of anterior chamber-associated immune deviation with interphotoreceptor retinoid-binding protein." Journal of Immunology 148, no. 6 (1992): 1685–92. http://dx.doi.org/10.4049/jimmunol.148.6.1685.

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Abstract Immunization with bovine interphotoreceptor retinoid-binding protein induces autoimmune uveitis in B10.A mice. We have examined whether this soluble retina-specific Ag can induce anterior chamber-associated immune deviation when injected into the anterior chamber (AC) of the eye, and whether this deviant immune response has any effect on uveitis is susceptible mice. The results of these experiments indicate that interphotoreceptor retinoid-binding protein (IRBP) injected intracamerally altered the subsequent immune response of B10.A mice such that a) they were not able to develop IRBP

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18

Birchall, A., S. Byravan, P. Kumar, and A. Moorthy. "POS1275 A RETROSPECTIVE STUDY ON UVEITIS FLARES FOLLOWING COVID19 VACCINATION: SHARING EXPERIENCE FROM A TEACHING HOSPITAL COMBINED RHEUMATOLOGY AND UVEITIS CLINIC." Annals of the Rheumatic Diseases 81, Suppl 1 (2022): 974.1–974. http://dx.doi.org/10.1136/annrheumdis-2022-eular.4921.

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BackgroundUveitis is a sight threatening disease caused by inflammation of the uveal tract of the eye. Uveitis is a manifestation of many autoimmune conditions and is associated with seronegative axial spondyloarthritis, reactive arthritis, Behçet’s disease, inflammatory bowel disease, and psoriatic arthritis. Acute anterior uveitis is the most common presentation and is most commonly idiopathic or associated with the HLA-B27 gene (around 20% of cases). Studies have shown that anterior uveitis frequently recurs in patients after it has previously remitted. (1) Patients suffering from autoimmun

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Park, Un Chul, Tae Wan Kim, and Hyeong Gon Yu. "Immunopathogenesis of Ocular Behçet’s Disease." Journal of Immunology Research 2014 (2014): 1–13. http://dx.doi.org/10.1155/2014/653539.

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Behçet’s disease (BD) is a chronic recurrent systemic inflammatory disorder of unknown etiology characterized by oral and genital ulcerations, skin lesions, and uveitis. The ocular involvement of BD, or Behçet’s uveitis (BU), is characterized by panuveitis or posterior uveitis with occlusive retinal vasculitis and tends to be more recurrent and sight threatening than other endogenous autoimmune uveitides, despite aggressive immunosuppression. Although pathogenesis of BD is unclear, researches have revealed that immunological aberrations may be the cornerstone of BD development. General hypothe

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Rahman, Najiha, Harry Petrushkin, and Ameenat Lola Solebo. "Paediatric autoimmune and autoinflammatory conditions associated with uveitis." Therapeutic Advances in Ophthalmology 12 (January 2020): 251584142096645. http://dx.doi.org/10.1177/2515841420966451.

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Childhood uveitis comprises a collection of heterogenous ocular phenotypes which are associated with a diverse range of childhood autoimmune and autoinflammatory disorders. Of these genetic and/or acquired disorders, juvenile idiopathic arthritis is the most common, affecting 30-80% of children with uveitis. Up to a third of children with uveitis have ‘isolated’ idiopathic disease and do not have an associated systemic disease which manifests in childhood. However, uveitis may be the presenting manifestation of disease; thus, the apparently well child who presents with uveitis may have isolate

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Klaska, Izabela, and John Forrester. "Mouse Models of Autoimmune Uveitis." Current Pharmaceutical Design 21, no. 18 (2015): 2453–67. http://dx.doi.org/10.2174/1381612821666150316122928.

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22

Kamal, A., A. Bhan, and P. I. Murray. "Uveitis with autoimmune hepatic disorders." Ocular Immunology and Inflammation 9, no. 4 (2001): 267–72. http://dx.doi.org/10.1076/ocii.9.4.267.3960.

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23

Papotto, Pedro Henrique, Eliana Blini Marengo, Luiz Roberto Sardinha, Anna Carla Goldberg, and Luiz Vicente Rizzo. "Immunotherapeutic strategies in autoimmune uveitis." Autoimmunity Reviews 13, no. 9 (2014): 909–16. http://dx.doi.org/10.1016/j.autrev.2014.05.003.

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24

Wildner, Gerhild, Maria Diedrichs-Möhring, and Stephan R. Thurau. "Rat Models of Autoimmune Uveitis." Ophthalmic Research 40, no. 3-4 (2008): 141–44. http://dx.doi.org/10.1159/000119865.

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Garg, Sunir J. "Autoimmune Thyroid Disease and Uveitis." JAMA Ophthalmology 135, no. 6 (2017): 599. http://dx.doi.org/10.1001/jamaophthalmol.2017.0776.

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Klímová, Aneta, Michaela Brichová, Eva Říhová, Jarmila Heissigerová, and Petra Svozílková. "Immune-Mediated Intraocular Inflammation. Review." Czech and Slovak Ophthalmology 77, no. 4 (2021): 163–68. http://dx.doi.org/10.31348/2021/07.

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Immune mediated inflammatory diseases are categorized into autoimmune and autoinflammatory. Autoimmune etiology is represented by autoreactive lymphocytes or autoantibodies, e.g. primary Sjögren‘s syndrome or rheumatoid arthritis. Ocular specific diseases with presumed autoimmune origin are sympathetic ophthalmia or birdshot chorioretinopathy. Autoinflammatory diseases are caused by mutations in regulatory genes for specific immunity. Hereditary periodic fevers represent monogenic autoinflammatory diseases; eye specific is Blau syndrome also named sarcoidosis with early onset. This article rev

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Pan, Wang, Furong Pan, Yanrui Dong, and Jing’an Tong. "The Pathogenesis and Treatment of Autoimmune Uveitis." Journal of Contemporary Medical Practice 7, no. 2 (2025): 47–50. https://doi.org/10.53469/jcmp.2025.07(02).09.

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Uveitis is an inflammatory reaction that occurs in intraocular tissues. Among them, non-infectious uveitis is a T cell-mediated autoimmune inflammatory response, but its pathogenesis is not fully understood. This article reviews its pathogenesis and treatment progress.

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Horai, Reiko, Ru Zhou, So Jin Bing, et al. "Vitamin A deficiency impacts acquisition, but not expression, of autoimmunity to the neuroretina." Journal of Immunology 198, no. 1_Supplement (2017): 218.8. http://dx.doi.org/10.4049/jimmunol.198.supp.218.8.

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Abstract Vitamin A (VitA) and its derivative retinoic acid (RA) are essential for immunological responses. Acquisition of effector responses is impeded in VitA-deficient (VAD) mice, but little is known about maintenance and expression of previously acquired effector function in the VAD environment, or its impact on progression of autoimmune diseases. We examined this using two models of uveitis: experimental autoimmune uveitis (EAU) induced by active immunization and spontaneous uveitis in retina-specific T cell receptor transgenic (R161H) mice, and in the model of experimental autoimmune ence

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S, V., S. Ganu, S. Surendran, L. Biswas, M. Cb, and C. V. Marwaha. "SAT0538 CLINICAL PROFILE OF AUTOIMMUNE UVEITIS NOT ASSOCIATED WITH HLA B27: A RETROSPECTIVE SINGLE CENTER STUDY." Annals of the Rheumatic Diseases 79, Suppl 1 (2020): 1226.1–1227. http://dx.doi.org/10.1136/annrheumdis-2020-eular.5140.

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Background:Autoimmune uveitis is frequently referred to a rheumatologist for evaluation & treatment. While clinical characteristics of HLA B27 associated uveitis are well documented, scanty information is available on non HLA-B27 associated autoimmune uveitis. We have described the clinical characteristics of our patients of non-B27 uveitis and compare the same with patients of Asian Indian HLAB27 uveitis.Objectives:To describe and compare the clinical characteristics of non HLA-B27 associated autoimmune uveitis.Methods:Retrospective review of records from hospital’s electronic medical rec

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Visalakshy, Jyothi, Sandeep Surendran, Salil Ganu, et al. "Describing the Clinical and Laboratory Features and HLA-B Pattern of Adult-Onset Idiopathic Autoimmune Uveitis at a Tertiary Hospital in South India: A Cross-Sectional Study." Scientific World Journal 2022 (February 8, 2022): 1–9. http://dx.doi.org/10.1155/2022/5032881.

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Introduction. There is a scarcity of information available on clinical and laboratory features of adult-onset idiopathic autoimmune uveitis. Therefore, we conducted a single centre descriptive cross-sectional study. Patients and Methods. A chart review of all patients with idiopathic autoimmune uveitis with onset after 18 years of age who were referred to the rheumatology department between January 2017 and December 2018 was performed. Their clinical features, demographic features, and HLA-B genotypes were documented and described. Results. Out of 210 patients referred to rheumatology, 66 were

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Okunuki, Yoko, Ryo Mukai, Takeshi Nakao, et al. "Retinal microglia initiate neuroinflammation in ocular autoimmunity." Proceedings of the National Academy of Sciences 116, no. 20 (2019): 9989–98. http://dx.doi.org/10.1073/pnas.1820387116.

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Autoimmune uveitis is a sight-threatening ocular inflammatory condition in which the retina and uveal tissues become a target of autoreactive immune cells. While microglia have been studied extensively in autoimmune uveitis, their exact function remains uncertain. The objective of the current study was to determine whether resident microglia are necessary and sufficient to initiate and amplify retinal inflammation in autoimmune uveitis. In this study, we clearly demonstrate that microglia are essential for initiating infiltration of immune cells utilizing a murine model of experimental autoimm

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Taniguchi, Ruth, Jason Devoss, James Moon, Marc Jenkins, and Mark Anderson. "Aire mediates peptide epitope-specific thymic deletion of self reactive T cells to prevent autoimmune uveitis (148.3)." Journal of Immunology 186, no. 1_Supplement (2011): 148.3. http://dx.doi.org/10.4049/jimmunol.186.supp.148.3.

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Abstract Aire is a transcription factor required for the expression of many tissue specific antigens in the thymus. In the absence of functional Aire protein, human patients and mice develop multi-organ autoimmune disease, including autoimmune uveitis, due to a defect in negative selection. The development of spontaneous uveitis in the Aire KO mouse model is dependent on the eye antigen, interphotoreceptor retinoid-binding antigen (IRBP), which has Aire-regulated expression the thymus. To study the mechanism by which Aire prevents IRBP-dependent autoimmune uveitis, two tetramer reagents were g

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Lee, Darren J., and Fauziyya Muhammad. "Mapping the generation and fate of Tregs associated with recovery of Autoimmune Disease." Journal of Immunology 202, no. 1_Supplement (2019): 192.17. http://dx.doi.org/10.4049/jimmunol.202.supp.192.17.

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Abstract Experimental autoimmune uveitis (EAU) is used to gain a better understanding of human autoimmune uveitis. Resolution of EAU is in part due to emergence of ocular antigen specific regulatory T cells (Post-EAU Tregs) that are found in the spleen of post-EAU mice. However, the stability of these cells and in what tissues these cells migrate to suppress disease is not known. Therefore, we assessed the stability of post-EAU Tregs, asked where these cells migrate to suppress disease, and determined when and where they emerge during EAU. FoxP3GFP-Cre; Rosa26stopfl/fl-Tom mice were used to id

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García-Aparicio, Á., L. Del Olmo Pérez, S. Sánchez Fernandez, et al. "AB1214 EPIDEMIOLOGY OF UVEITIS IN A SPANISH REGION: PREVALENCE AND ETIOLOGY, UVECAM PROJECT." Annals of the Rheumatic Diseases 79, Suppl 1 (2020): 1898.1–1898. http://dx.doi.org/10.1136/annrheumdis-2020-eular.1409.

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Background:Epidemiologic studies about the distribution of the various types of uveitis and their etiology are important in helping the clinician to investigate, diagnose, and manage these pathologies. Approximating prevalence of uveitis is an important public health challenge, though studies on this purpose are scarce and of low quality. Therefore it is difficult to estimate accurately the impact of this disease in our days.Objectives:To estimate the prevalence of uveitis and to describe its etiologic and anatomical pattern by means of a population study carried out in a Spanish regionMethods

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Benyó, Fruzsina, Alexandra Farkas, Hajnalka Horváth, Zoltán Zsolt Nagy, and Zsuzsanna Szepessy. "Biológiai terápia szisztémás alkalmazása a szemészetben." Orvosi Hetilap 160, no. 44 (2019): 1744–50. http://dx.doi.org/10.1556/650.2019.31552.

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Abstract: We present herewith cases of non-infectious uveitis with biological treatment where the ocular complaints were the initial symptoms indicating a multi-organ autoimmune disease. The first case was a patient with panuveitis and Vogt–Koyanagi–Harada disease, the second case was also a panuveitic patient with sarcoidosis and the third case was a patient with intermediate uveitis and inflammatory bowel disease. In all cases, emerging new, biological therapy (adalimumab) was necessary to achieve permanent inactive period of uveitis and the autoimmune disease. Introducing systemic biologica

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Mattapallil, Mary, Phyllis Silver, Joseph Mattapallil, et al. "Uveitis-associated epitopes of retinal antigens are pathogenic in the humanized mouse model of EAU and identify autoaggressive T cells (93.1)." Journal of Immunology 184, no. 1_Supplement (2010): 93.1. http://dx.doi.org/10.4049/jimmunol.184.supp.93.1.

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Abstract Autoimmune uveitis affects 150,000 Americans annually and is a leading cause of blindness. Pathogenesis is thought to involve T cell mediated responses to retinal proteins, such as retinal arrestin (S-Ag). Susceptibility to autoimmune uveitis is strongly associated with HLA class I and class II, but relatively little is known about the nature of HLA class II alleles and about the epitopes that might be pathogenic in the context of different HLA class II molecules. Using a ‘humanized’ mouse model for experimental autoimmune uveitis (EAU) in HLA transgenic mice, we have identified permi

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Diogo, Leonardo Rodrigues Ferreira, and Manuela Chaves Marques Lopes. "Uveitis in Children with Autoimmune Diseases: An Update on Diagnosis, Complications, and Management." International Health Sciences Review 1, no. 3 (2025): 75–79. https://doi.org/10.70164/ihsr.v1i3.12.

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Pediatric uveitis, though less common than in adults, is a potentially sight-threatening condition often associated with autoimmune diseases such as juvenile idiopathic arthritis, Behçet’s disease, and tubulointerstitial nephritis and uveitis (TINU) syndrome. This integrative review aimed to explore the clinical manifestations, complications, and therapeutic strategies of autoimmune-related uveitis in children. A literature search was conducted in the PubMed database using selected MeSH terms, identifying 10 studies, of which 4 met the inclusion criteria for detailed analysis. The selected art

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Horai, Reiko. "Gut microbiota linked to autoimmune uveitis." Annals of Eye Science 2 (2018): 19. http://dx.doi.org/10.21037/aes.2017.03.02.

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Karmochkine, Marina. "Intravenous Immune Globulin in Autoimmune Uveitis." Annals of Internal Medicine 129, no. 12 (1998): 1078. http://dx.doi.org/10.7326/0003-4819-129-12-199812150-00030.

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DEWISPELAERE, R., D. LIPSKI, M. MAKHOUL, et al. "Adhesion molecules in experimental autoimmune uveitis." Acta Ophthalmologica 90 (August 6, 2012): 0. http://dx.doi.org/10.1111/j.1755-3768.2012.f077.x.

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Levy, Roger A., Francisco Assis de Andrade, and Ivan Foeldvari. "Cutting-Edge Issues in Autoimmune Uveitis." Clinical Reviews in Allergy & Immunology 41, no. 2 (2011): 214–23. http://dx.doi.org/10.1007/s12016-011-8267-x.

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Lee, Richard W., Lindsay B. Nicholson, H. Nida Sen, et al. "Autoimmune and autoinflammatory mechanisms in uveitis." Seminars in Immunopathology 36, no. 5 (2014): 581–94. http://dx.doi.org/10.1007/s00281-014-0433-9.

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Giuliari, Gian Paolo, Ama Sadaka, and David M. Hinkle. "Biologic agents in experimental autoimmune uveitis." International Ophthalmology 34, no. 1 (2013): 145–56. http://dx.doi.org/10.1007/s10792-013-9756-0.

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Lim, Lyndell L., John D. Scarborough, Jennifer E. Thorne, et al. "Uveitis in Patients with Autoimmune Hepatitis." American Journal of Ophthalmology 147, no. 2 (2009): 332–38. http://dx.doi.org/10.1016/j.ajo.2008.08.019.

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Efe, Cumali, Ersan Ozaslan, Tugrul Purnak, Zeynep Ozbalkan, and Emin Altıparmak. "Uveitis in Patients With Autoimmune Hepatitis." American Journal of Ophthalmology 149, no. 4 (2010): 684–85. http://dx.doi.org/10.1016/j.ajo.2009.12.016.

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Selmi, Carlo. "Diagnosis and classification of autoimmune uveitis." Autoimmunity Reviews 13, no. 4-5 (2014): 591–94. http://dx.doi.org/10.1016/j.autrev.2014.01.006.

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Mesquida, Marina, Blanca Molins, Víctor Llorenç, Maite Sáinz de la Maza, and Alfredo Adán. "Targeting interleukin-6 in autoimmune uveitis." Autoimmunity Reviews 16, no. 10 (2017): 1079–89. http://dx.doi.org/10.1016/j.autrev.2017.08.002.

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Caspi, Rachel R., Phyllis B. Silver, Dror Luger, et al. "Mouse Models of Experimental Autoimmune Uveitis." Ophthalmic Research 40, no. 3-4 (2008): 169–74. http://dx.doi.org/10.1159/000119871.

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Mase, Oliver, Mustafa Qasem, and Nicholas Beare. "Systematic review of studies comparing infliximab and adalimumab in autoimmune uveitis." BMJ Open Ophthalmology 8, no. 1 (2023): e001303. http://dx.doi.org/10.1136/bmjophth-2023-001303.

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Abstract:

ObjectiveThis study aimed to review effectiveness studies comparing two biological anti-tumour necrosis factor agents, adalimumab (ADA) and infliximab (IFX), in the management of autoimmune uveitis.MethodsA systematic search was conducted across PubMed, Scopus, Web of Science and Google Scholar from 2014 until February 2022. The search included the following keywords “Adalimumab”, “Infliximab”, “Autoimmune”, “Anterior”, “Intermediate”, “Posterior”, “Panuveitis”, “Refractory” and “Uveitis”. Primary studies comparing both ADA and IFX in a population of autoimmune uveitis patients were considered

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Davydova, G. A., T. A. Lisitsyna, L. A. Kovaleva, E. S. Sorozhkina, A. A. Zaitseva, and A. A. Baisangurova. "Clinical Variations of Uveitis in Immuno-Inflammatory Diseases. Review of the Literature. Part 1." Ophthalmology in Russia 19, no. 3 (2022): 465–74. http://dx.doi.org/10.18008/1816-5095-2022-3-465-474.

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Non-infectious uveitis is one of the main and insufficiently studied causes of disability and blindness in patients with immuno-inflammatory diseases. Uveitis associated with spondyloarthritis, Behcet’s disease, juvenile idiopathic arthritis, systemic sarcoidosis and Vogt-Koyanagi-Harada syndrome are described more often and better than others, but the pathogenesis of different variants of their course is not well understood. Also, there remains a need to study the clinical and pathogenetic features of uveitis in rare autoimmune inflammatory diseases. Despite the currently existing diagnostic

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