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Books on the topic 'Autonomic (sympathetic and parasympathetic'

Author: Grafiati

Published: 5 June 2025

Last updated: 1 August 2025

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1

Prouse, Darrel D. Fundamentals of the chiropractic diagnosis. 4th ed. Darrel D. Prouse, 1994.

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2

N, Attal, Jänig Wilfrid, and Schmidt Robert F. 1932-, eds. Reflex sympathetic dystrophy: Pathophysiological mechanisms and clinical implications. VCH Verlagsgesellschaft, 1992.

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3

Pichon, Aurélien. Homeostatic role of the parasympathetic nervous system in human behavior. Nova Science Publisher's, 2009.

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4

Kobayashi, Hiroyuki. Jiritsu shinkei o totonoeru akirameru kenkōhō. Kadokawa Shoten, 2013.

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5

M, Evans Joyce, and United States. National Aeronautics and Space Administration., eds. Validation of spectral analysis as a noninvasive tool to assess autonomic regulation of cardiovascular function: Final report. Center for Biomedical Engineering, Wenner-Gren Research Laboratory, University of Kentucky, 1996.

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6

Juris, Elena. Positive options for complex regional pain syndrome (CRPS): Self-help and treatment. Hunter House, an imprint of Turner Publishing Company, 2014.

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7

de Geus, Eco, Rene van Lien, Melanie Neijts, and Gonneke Willemsen. Genetics of Autonomic Nervous System Activity. Edited by Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.010.

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Large individual differences in the activity of the autonomic nervous system (ANS) play a key role in risk for cardiovascular disease. This chapter presents an overview of the measurement strategies that can be used to study ANS activity in samples that are sufficiently large to allow genetic analyses. Heart rate variability, in particular, respiratory sinus arrhythmia (RSA) is identified as the measure of choice to index parasympathetic activity, whereas preejection period (PEP) is the measure of choice to index sympathetic activity. Twin studies have demonstrated significant genetic contribu

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8

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 44-Year-Old Male with Subacute Onset of Syncope. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0032.

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Syncope in a patient with orthostatic hypotension (OH) may indicate autonomic dysfunction. The definition of OH is presented. Clinical features of parasympathetic and sympathetic function are discussed. The differential of acute autonomic dysfunction includes a number of conditions. An autoimmune etiology may occur autoimmune autonomic ganglionopathy. Serologic testing can assist in this diagnosis. If autoimmune immune modulating therapies may be indicated. Autonomic neuropathy may be a paraneoplastuc syndrome. Autonomic testing can also help with documenting autonomic neuropathy as well as th

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9

Fisch, Adam. Peripheral Nervous SystemAutonomic Nervous System. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199845712.003.0118.

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Chapter 6 discusses how to draw the peripheral nervous system, specifically the autonomic nervous system, including autonomic fiber arrangements, the parasympathetic nervous system, the sympathetic nervous system, the urinary system, and the cardiac reflex.

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10

Burton, Derek, and Margaret Burton. Integration and control: the nervous system. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198785552.003.0011.

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The complexity of fish behaviour and information processing indicates high levels of neural, anatomical and functional organization. Neural cells are conducting neurons and neuroglia with putative support and physiological roles. Neuronal conduction, synaptic transmission, reflexes and neuropils are factors in integrative activity and information processing. Fish nervous systems are organized into central (brain and spinal cord) and peripheral (including autonomic) components. Interestingly the structure and function of the fish optic tectum have been considered comparable to those of the tetr

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11

Colombetti, Giovanna, and Neil Harrison. From physiology to experience: Enriching existing conceptions of “arousal” in affective science. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198811930.003.0013.

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This chapter examines the notion of “arousal”, an influential notion in affective science referring to the degree of an individual’s “activation” or “excitement” during an emotional state. It considers this notion specifically in relation to interoception, defined broadly as “sensitivity to stimuli arising inside the organism.” “Physiological arousal” is distinguished from “experienced arousal” and it is argued that both need to be characterized more broadly than commonly done. Physiological arousal cannot be reduced to sympathetic activation, as it involves complex interactions between multip

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12

Langley, John Newport. Autonomic Nervous System: Part 1. Creative Media Partners, LLC, 2018.

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13

Reflex Sympathetic Dystrophy. Chapman & Hall, 1992.

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14

Langley, John Newport. The Autonomic Nervous System: Part 1. Franklin Classics Trade Press, 2018.

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15

Langley, John Newport. The Autonomic Nervous System: Part 1. Franklin Classics Trade Press, 2018.

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16

Evans, Griffith. Latent Syphilis and the Autonomic Nervous System. Elsevier Science & Technology Books, 2017.

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17

Janig, Wilfrid. Reflex Sympathetic Dystrophy: Pathophysiological Mechanisms and Clinical Implications. Chapman & Hall, 1992.

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18

Kla, Koffi. Autonomic Dysreflexia. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0071.

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Autonomic dysreflexia (AD), a potentially emergent clinical syndrome that can occur in patients with spinal cord injury, is characterized by a sudden rise in blood pressure caused by uncontrolled sympathetic activation of the autonomic system below the level of the lesion. When caring for spinal cord injury (SCI) patients, a pretreatment plan should be in place if an AD episode occurs. Physical manifestations of an AD episode can include headache, flushing, and diaphoresis above the lesion. Treatment should be aimed at identifying and eliminating the trigger stimulus and must be prompt to avoi

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19

Brandner, Brigitta. Explaining reflex sympathetic dystrophy. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0003.

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The medical community had long been aware of a constellation of symptoms involving pain, bone atrophy, and trophic changes that could occur following a trauma; however, this paper was the first to unify and explain the syndrome. James Evans amalgamated other theories of the time and was the first to realize the fundamental role of the autonomic nervous system in explaining the pathophysiology of the condition, which he named ‘reflex sympathetic dystrophy’. Evans uses 57 cases histories to highlight the variety of noxious stimuli that may result in reflex sympathetic dystrophy, and goes onto ou

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20

Kraus, Walter Max 1889. Vegetative Neurology: The Anatomy, Physiology, Pharmacodynamics and Pathology of the Sympathetic and Autonomic Nervous Systems. Creative Media Partners, LLC, 2021.

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21

Kraus, Walter Max 1889. Vegetative Neurology: The Anatomy, Physiology, Pharmacodynamics and Pathology of the Sympathetic and Autonomic Nervous Systems. Creative Media Partners, LLC, 2021.

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22

Masson-Guipe, Emily M. The effects of exercise training on sympathetic and parasympathetic nervous system responses to a novel stressor in Sprague-Dawley rats. 1993.

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23

Pineles, Suzanne L., and Scott P. Orr. The Psychophysiology of PTSD. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0022.

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This chapter provides an overview of post-traumatic stress disorder (PTSD)-related psychophysiological research. Specific foci include psychophysiological reactivity to trauma-related stimuli and loud tones, conditioned fear acquisition and extinction, fear memory reconsolidation blockade, and the potential usefulness of psychophysiological measures in predicting PTSD development, maintenance, and treatment efficacy. A detailed discussion is provided on the contribution of reduced parasympathetic tone and increased sympathetic activity to the heightened psychophysiological reactivity associate

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24

Higier, Henryk, and Walter Max Kraus. Vegetative Neurology; the Anatomy, Physiology, Pharmaco-Dynamics and Pathology of the Sympathetic and Autonomic Nervous Systems. Creative Media Partners, LLC, 2018.

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25

El sínodrome de distrofia simpática refleja. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 2000.

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26

Abcejo, Arnoley S., and Jeffrey J. Pasternak. Neurogenic Shock. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0072.

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Neurogenic shock is a pathophysiologic state of systemic hypoperfusion characterized by a significant decrease in systemic vascular resistance secondary to loss of sympathetic tone. Neurogenic shock is most commonly seen in the setting of acute spinal cord injury (SCI) but can also occur following significant brain injury. Interruption of sympathetic fibers causes loss of basal vascular sympathetic tone, commonly allowing unopposed parasympathetic tone. As a result, severe hypotension and bradycardia can further exacerbate neurologic injury and organ perfusion. Understanding the physiologic an

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27

Kobayashi, Hiroyuki. Sutoresu ga kieru "shinai" kenkō hō. 2014.

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28

Newell-Price, John, Alia Munir, and Miguel Debono. Adrenal disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0188.

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This chapter reviews the clinical features, diagnosis, and treatment of three adrenal diseases: adrenal insufficiency, primary aldosteronism (hyperaldosteronism), and phaeochromocytoma. Adrenal insufficiency is a disorder characterized by impaired adrenocortical function. In primary adrenal insufficiency, destruction of the adrenal cortex results in a decreased production of glucocorticoids, mineralocorticoids, and/or androgens. Secondary adrenal insufficiency is due to disordered pituitary and hypothalamic function resulting in decreased secretion of adrenocorticotropic hormone or corticotrop

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29

Murray-Close, Dianna, Nicole L. Breslend, and Leigh Ann Holterman. Psychophysiology Indicators of Relational Aggression. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190491826.003.0009.

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Mounting evidence implicates psychophysiological processes in the development of relational aggression. This review discusses the state of the field regarding associations between physiological stress systems—including the sympathetic nervous system, the parasympathetic nervous system, and the hypothalamic-pituitary-adrenal axis—and relational aggression. The theoretical significance of these processes is discussed, and potential moderators of associations, such as functions of relational aggression, contextual risk, and gender, are considered. Finally, critical next steps in this research are

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30

Mason, Peggy. Spinal Cord. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0004.

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The spinothalamic and lemniscal pathways carry somatosensory information from the periphery into the brain while the corticospinal pathway carries motor commands from the brain to motoneurons of the spinal cord. Following these pathways through the spinal cord allows the student to infer lesion location from symptoms. To exemplify the clinical importance of sympathetic outputs from thoracic segments, Horner syndrome is described. Similarly, the common problems caused by spinal cord injury on sacral parasympathetic functions are stressed. The contributions of specific spinal segments to breathi

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31

Boyatzis, Richard E. The Science of Change. Oxford University PressNew York, NY, 2024. http://dx.doi.org/10.1093/9780197765142.001.0001.

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Abstract Change is ephemeral if it occurs at all. The Science of Change is about the quest for sustained, desired change. It uses 58 years of research from many fields on the multilevel fractal Intentional Change Theory (ICT). The ICT process has five phases with tipping points of the Positive or Negative Emotional Attractors (PEA and NEA) that move or stop the process. They are Ideal Self (shared vision), Real Self (norms), learning agenda, experimentation/practice, and resonant relationships. PEA and NEA are combinations of the Sympathetic and Parasympathetic Nervous Systems, Default Mode an

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