Relevant bibliographies by topics / Avian sarcoma leukosis virus (ASLV)

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Avian sarcoma leukosis virus (ASLV)'

Author: Grafiati
Published: 4 June 2021
Last updated: 4 February 2022

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Journal articles on the topic "Avian sarcoma leukosis virus (ASLV)"

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Dimcheff, Derek E., Sergei V. Drovetski, Mallika Krishnan, and David P. Mindell. "Cospeciation and Horizontal Transmission of Avian Sarcoma and Leukosis Virus gag Genes in Galliform Birds." Journal of Virology 74, no. 9 (2000): 3984–95. http://dx.doi.org/10.1128/jvi.74.9.3984-3995.2000.

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ABSTRACT In a study of the evolution and distribution of avian retroviruses, we found avian sarcoma and leukosis virus (ASLV) gag genes in 26 species of galliform birds from North America, Central America, eastern Europe, Asia, and Africa. Nineteen of the 26 host species from whom ASLVs were sequenced were not previously known to contain ASLVs. We assessed congruence between ASLV phylogenies based on a total of 110gag gene sequences and ASLV-host phylogenies based on mitochondrial 12S ribosomal DNA and ND2 sequences to infer coevolutionary history for ASLVs and their hosts. Widespread distribu
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Hejnar, Jiří, Anna Koslová, Pavel Trefil, et al. "CRISPR/Cas9 Editing of Viral Receptors and Biotechnological Approach to Host Resistance." Proceedings 50, no. 1 (2020): 22. http://dx.doi.org/10.3390/proceedings2020050022.

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Avian sarcoma and leukosis virus (ASLV), diversified into seven phylogenetically relativesubgroups (A, B, C, D, E, J, and K), present as either exogenous or endogenous viruses in domesticchicken. [...]
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Reinišová, Markéta, Filip Šenigl, Xueqian Yin, et al. "A Single-Amino-Acid Substitution in the TvbS1 Receptor Results in Decreased Susceptibility to Infection by Avian Sarcoma and Leukosis Virus Subgroups B and D and Resistance to Infection by Subgroup E In Vitro and In Vivo." Journal of Virology 82, no. 5 (2007): 2097–105. http://dx.doi.org/10.1128/jvi.02206-07.

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ABSTRACT The avian sarcoma and leukosis virus (ASLV) family of retroviruses contains five highly related envelope subgroups (A to E) thought to have evolved from a common viral ancestor in the chicken population. Three genetic loci in chickens determine the susceptibility or resistance of cells to infection by the subgroup A to E ASLVs. Some inbred lines of chickens display phenotypes that are somewhere in between either efficiently susceptible or resistant to infection by specific subgroups of ASLV. The tvb gene encodes the receptor for subgroups B, D, and E ASLVs. The wild-type TvbS1 recepto
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Barr, Stephen D., Jeremy Leipzig, Paul Shinn, Joe R. Ecker, and Frederic D. Bushman. "Integration Targeting by Avian Sarcoma-Leukosis Virus and Human Immunodeficiency Virus in the Chicken Genome." Journal of Virology 79, no. 18 (2005): 12035–44. http://dx.doi.org/10.1128/jvi.79.18.12035-12044.2005.

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ABSTRACT We have analyzed the placement of sites of integration of avian sarcoma-leukosis virus (ASLV) and human immunodeficiency virus (HIV) DNA in the draft chicken genome sequence, with the goals of assessing species-specific effects on integration and allowing comparison to the distribution of chicken endogenous retroviruses (ERVs). We infected chicken embryo fibroblasts (CEF) with ASLV or HIV and sequenced 863 junctions between host and viral DNA. The relationship with cellular gene activity was analyzed by transcriptional profiling of uninfected or ASLV-infected CEF cells. ASLV weakly fa
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Yin, Xueqian, Deborah C. Melder, William S. Payne, Jerry B. Dodgson, and Mark J. Federspiel. "Mutations in Both the Surface and Transmembrane Envelope Glycoproteins of the RAV-2 Subgroup B Avian Sarcoma and Leukosis Virus Are Required to Escape the Antiviral Effect of a Secreted Form of the TvbS3 Receptor †." Viruses 11, no. 6 (2019): 500. http://dx.doi.org/10.3390/v11060500.

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The subgroup A through E avian sarcoma and leukosis viruses ASLV(A) through ASLV(E) are a group of highly related alpharetroviruses that have evolved to use very different host protein families as receptors. We have exploited genetic selection strategies to force the replication-competent ASLVs to naturally evolve and acquire mutations to escape the pressure on virus entry and yield a functional replicating virus. In this study, evolutionary pressure was exerted on ASLV(B) virus entry and replication using a secreted for of its Tvb receptor. As expected, mutations in the ASLV(B) surface glycop
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Earp, Laurie J., Sue E. Delos, Robert C. Netter, Paul Bates, and Judith M. White. "The Avian Retrovirus Avian Sarcoma/Leukosis Virus Subtype A Reaches the Lipid Mixing Stage of Fusion at Neutral pH." Journal of Virology 77, no. 5 (2003): 3058–66. http://dx.doi.org/10.1128/jvi.77.5.3058-3066.2003.

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ABSTRACT We previously showed that the envelope glycoprotein (EnvA) of avian sarcoma/leukosis virus subtype A (ASLV-A) binds to liposomes at neutral pH following incubation with its receptor, Tva, at ≥22°C. We also provided evidence that ASLV-C fuses with cells at neutral pH. These findings suggested that receptor binding at neutral pH and ≥22°C is sufficient to activate Env for fusion. A recent study suggested that two steps are necessary to activate avian retroviral Envs: receptor binding at neutral pH, followed by exposure to low pH (W. Mothes et al., Cell 103:679-689, 2000). Therefore, we
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Munguia, Audelia, and Mark J. Federspiel. "Avian Sarcoma and Leukosis Virus Envelope Glycoproteins Evolve to Broaden Receptor Usage Under Pressure from Entry Competitors †." Viruses 11, no. 6 (2019): 519. http://dx.doi.org/10.3390/v11060519.

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The subgroup A through E avian sarcoma and leukosis viruses (ASLV(A) through ASLV(E)) are a group of highly related alpharetroviruses that have evolved their envelope glycoproteins to use different receptors to enable efficient virus entry due to host resistance and/or to expand host range. Previously, we demonstrated that ASLV(A) in the presence of a competitor to the subgroup A Tva receptor, SUA-rIgG immunoadhesin, evolved to use other receptor options. The selected mutant virus, RCASBP(A)Δ155–160, modestly expanded its use of the Tvb and Tvc receptors and possibly other cell surface protein
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Federspiel, Mark J. "Reverse Engineering Provides Insights on the Evolution of Subgroups A to E Avian Sarcoma and Leukosis Virus Receptor Specificity." Viruses 11, no. 6 (2019): 497. http://dx.doi.org/10.3390/v11060497.

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The initial step of retrovirus entry—the interaction between the virus envelope glycoprotein trimer and a cellular receptor—is complex, involving multiple, noncontiguous determinants in both proteins that specify receptor choice, binding affinity and the ability to trigger conformational changes in the viral glycoproteins. Despite the complexity of this interaction, retroviruses have the ability to evolve the structure of their envelope glycoproteins to use a different cellular protein as receptors. The highly homologous subgroup A to E Avian Sarcoma and Leukosis Virus (ASLV) glycoproteins bel
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Klucking, Sara, Heather B. Adkins, and John A. T. Young. "Resistance to Infection by Subgroups B, D, and E Avian Sarcoma and Leukosis Viruses Is Explained by a Premature Stop Codon within a Resistance Allele of the tvb Receptor Gene." Journal of Virology 76, no. 15 (2002): 7918–21. http://dx.doi.org/10.1128/jvi.76.15.7918-7921.2002.

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ABSTRACT Here we present the first molecular characterization of the defect associated with an avian sarcoma and leukosis virus (ASLV) receptor resistance allele, tvb r. We show that resistance to infection by subgroups B, D, and E ASLV is explained by the presence of a single base pair mutation that distinguishes this allele from tvb s1, an allele which encodes a receptor for all three viral subgroups. This mutation generates an in-frame stop codon that is predicted to lead to the production of a severely truncated protein.
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Klucking, Sara, Asha S. Collins, and John A. T. Young. "Avian Sarcoma and Leukosis Virus Cytopathic Effect in the Absence of TVB Death Domain Signaling." Journal of Virology 79, no. 13 (2005): 8243–48. http://dx.doi.org/10.1128/jvi.79.13.8243-8248.2005.

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ABSTRACT The cytopathic effect (CPE) seen with some subgroups of avian sarcoma and leukosis virus (ASLV) is associated with viral Env activation of the death-promoting activity of TVB (a tumor necrosis factor receptor-related receptor that is most closely related to mammalian TNF-related apoptosis-inducing ligand [TRAIL] receptors) and with viral superinfection leading to unintegrated viral DNA (UVD) accumulation, which is presumed to activate a cellular DNA damage response. In this study, we employed cells that express signaling-deficient ASLV receptors to demonstrate that an ASLV CPE can be
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Dissertations / Theses on the topic "Avian sarcoma leukosis virus (ASLV)"

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Al, Andary Elsy. "Étude comparative des processus intégratifs des rétrovirus aviaires et porcins." Phd thesis, Université Claude Bernard - Lyon I, 2011. http://tel.archives-ouvertes.fr/tel-00866109.

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Les rétrovirus sont des virus à ARN, enveloppés présents dans de nombreuses espèces animales de rente, chez les animaux de compagnie et chez l'homme. Une des particularités des rétrovirus concerne l'intégration du génome viral au sein du génome de la cellule infectée; cette intégration est réalisée par une enzyme virale, l'intégrase. Le projet de cette thèse vise à mieux comprendre le fonctionnement de cette enzyme notamment en identifiant des facteurs cellulaires interagissant avec celle-ci, facteurs qui pourraient être des agents favorisant le processus intégratif ou, au contraire, des agent
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Banks, Jennifer Dawn. "Characterization of a minimal avian leukosis-sarcoma virus packaging signal /." Thesis, Connect to this title online; UW restricted, 1999. http://hdl.handle.net/1773/11528.

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Ronfort, Corinne. "Etude des structures rétrovirales endogènes dans des cellules de poules et d'un exemple d'interaction avec des vecteurs rétroviraux dérivés des ASLV (Avian Sarcoma Leukemia Viruses)." Lyon 1, 1992. http://www.theses.fr/1992LYO10214.

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Les structures retrovirales endogenes (ou ev) sont des sequences retrovirales integrees dans les genomes cellulaires et qui se transmettent a la descendance des sujets comme des genes cellulaires. Le travail de these a consiste, en premier lieu, a etudier les loci ev qui sont presents au sein d'une souche de poules leghorn doree. Par cartographie de restriction, 4 loci endogenes ont ete identifies, 2 d'entre eux representent des formes non encore decrites. Tous sont deletes pour une partie plus ou moins importante de leur genome et aucun n'est capable de produire des particules virales infecti
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Book chapters on the topic "Avian sarcoma leukosis virus (ASLV)"

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Kotier, M. "Activation and Regulation of Avian Sarcoma Leukemia Virus (ASLV) Protease." In Proteases of Retroviruses, edited by Vladimír Kostka. De Gruyter, 1989. http://dx.doi.org/10.1515/9783110862782-016.

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"ASLV (avian sarcoma-leukosis virus)." In Encyclopedia of Genetics, Genomics, Proteomics and Informatics. Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6754-9_1224.

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