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Relevant bibliographies by topics / Bacterial kidney disease (Fish disease)

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Academic literature on the topic 'Bacterial kidney disease (Fish disease)'

Author: Grafiati

Published: 4 June 2021

Last updated: 17 February 2022

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Journal articles on the topic "Bacterial kidney disease (Fish disease)"

1

Sergeenko, NV, EA Ustimenko, MG Eliseikina, AD Kuhlevskiy, EV Bochkova, and TV Ryazanova. "First report of bacterial kidney disease in coho salmon Oncorhynchus kisutch in Russia." Diseases of Aquatic Organisms 140 (June 18, 2020): 31–36. http://dx.doi.org/10.3354/dao03486.

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This paper describes the first case of bacterial kidney disease (BKD) to be identified in coho salmon Oncorhynchus kisutch in Russia. The fish in question was caught in Lake Bolshoi Vilyui on the Kamchatka Peninsula. The diseased fish had foci of granulomatous inflammation in the kidneys. The diagnosis was confirmed by isolating the bacterium Renibacterium salmoninarum from kidney tissue in pure culture, and by determining the partial 16S RNA gene sequence of the isolate. This is the first detection of this pathogen in the genus Oncorhynchus in Russia, and detection of BKD in coho salmon indicates that the pathogen is present in the natural fish populations of Kamchatka. Therefore, it will be necessary to conduct screening studies of mature salmon selected for artificial reproduction, for the presence of BKD signs and asymptomatic infection with R. salmoninarum, which will allow us to estimate the prevalence of the pathogen.

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Delghandi, Mohammad Reza, Mansour El-Matbouli, and Simon Menanteau-Ledouble. "Renibacterium salmoninarum—The Causative Agent of Bacterial Kidney Disease in Salmonid Fish." Pathogens 9, no. 10 (October 15, 2020): 845. http://dx.doi.org/10.3390/pathogens9100845.

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Renibacterium salmoninarum is one of the oldest known bacterial pathogens of fish. This Gram-positive bacterium is the causative agent of bacterial kidney disease, a chronic infection that is mostly known to infect salmonid fish at low temperatures. Externally, infected fish can display exophthalmia as well as blebs on the skin and ulcerations alongside haemorrhages at the base of the fins and alongside the lateral line. Internally, the kidney, heart, spleen and liver can show signs of swelling. Granulomas can be seen on various internal organs, as can haemorrhages, and the organs can be covered with a false membrane. Ascites can also accumulate in the abdominal cavity. The bacterium is generally cultivated on specialized media such as kidney disease medium-1 (KDM-1), KDM-2 and selective kidney disease medium (SKDM), and a diagnostic is performed using molecular tools such as PCRs or real-time quantitative PCRs (RT-qPCRs). Several virulence mechanisms have been identified in R. salmoninarum, in particular the protein p57 that is known to play a role in both agglutination and immunosuppression of the host’s defense mechanisms. Control of the disease is difficult; the presence of asymptomatic carriers complicates the eradication of the disease, as does the ability of the bacterium to gain entrance inside the eggs. Bacterin-killed vaccines have proven to be of doubtful efficacy in controlling the disease, and even more recent application of a virulent environmental relative of R. salmoninarum is of limited efficacy. Treatment by antibiotics such as erythromycin, azithromycin and enrofloxacin can be effective but it is slow and requires prolonged treatment. Moreover, antibiotic-resistant strains have been reported. Despite being known for a long time, there is still much to be discovered about R. salmoninarum, notably regarding its virulence mechanisms and its vaccine potential. Consequently, these gaps in knowledge continue to hinder control of this bacterial disease in aquaculture settings.

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Murwantoko, Murwantoko, Eka Diniarti, and Triyanto Triyanto. "Isolation, Characterization and Pathogenicity of Edwardsiella tarda a Causative Disease on Freshwater Fish in Yogyakarta." Jurnal Perikanan Universitas Gadjah Mada 21, no. 1 (August 2, 2019): 41. http://dx.doi.org/10.22146/jfs.39920.

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Edwarsiella tarda is a cosmopolitan bacterium and is a cause of Edwardsiellosis in various fish species. The bacterial infection causes large losses on aquaculture in Asia, especially Japan. This study was conducted to isolate and characterize E. tarda as causative disease in freshwater fishes, and to determine its pathogenicity to catfish (Pangasius sp.). Bacteria were isolated from kidney of diseased fishes on Tryptone Soya Agar medium. Identification was conducted based on morphological colonies, morphological cells and biochemical tests. Fulfillment of Koch Postulates was done by injecting bacteria intraperitoneally on 7-9 cm fishes at dose of 107 cfu/fish. Pathogenicity test was carried out by intraperitoneal injection at 104, 105, 106, and 107 cfu/fish to 7-9 cm-catfish (Pangasius sp.) and followed by observation of disease signs and mortality every six hours for 7 days. Pathogenicity was determined as Lethal Dosage (LD50) using Dragstedt Behrens method. In this research we have isolated three isolates E. tarda causing disease in fishes. The clinical signs of this disease were lose of pigmentation over the lession, swollen of stomach, haemorhage on fins , small cutaneous lesions, and necrotic on fins area. The LD50 of E. tarda isolate L2, L3, and N3 were 4.64 ± 0.35x105, 1.54 ± 0.07x105, and 1.13 ± 0.13x106 cfu/fish, respectively.

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White, Melvin Randall, Ching Ching Wu, and Sharon R. Albregts. "Comparison of Diagnostic Tests for Bacterial Kidney Disease in Juvenile Steelhead Trout (Oncorhynchus Mykiss)." Journal of Veterinary Diagnostic Investigation 7, no. 4 (October 1995): 494–99. http://dx.doi.org/10.1177/104063879500700412.

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In order to accurately diagnose bacterial kidney disease caused by Renibacterium salmoninarum in steelhead trout, kidney tissue from experimentally infected fish was evaluated using a commercially available enzyme-linked immunosorbent assay (ELISA) test kit, fluorescent antibody (FA) testing, bacteriologic culture, and histopathology. Seventy-five steelhead trout were randomly assigned to 1 of 4 groups and intraperitoneally inoculated with 0.15 ml saline ( n = 20), 1 × 1010 organisms/ml ( n = 18), 1 × 108 organisms/ml ( n = 18), or 1 × 106 organisms/ml ( n = 19) of R. salmoninarum. ELISA, FA, and bacteriologic culture were positive for R. salmoninarum from the kidney tissue of the 2 groups infected with the highest doses. Although the ELISA and FA tests were accurate when compared to the bacteriologic culture from the 2 groups infected with the higher doses of the organism, they were less sensitive at the lowest level of inoculum. Histopathology was not specific for this disease; however, all infected fish had a marked proliferative histiocytic interstitial nephritis, characterized by marked expansion of the renal hematopoietic tissue by histiocytes without tissue necrosis. Other microscopic findings included splenitis and myositis (at the injection site) of some fish.

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Kristianingrum, Yuli Purwandari, Bambang Sutrisno, Sitarina Widyarini, and Kurniasih Sugiyono. "Disease incidence of freshwater fish in the Special Region of Yogyakarta, Indonesia." BIO Web of Conferences 33 (2021): 06001. http://dx.doi.org/10.1051/bioconf/20213306001.

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The potential of freshwater farming in the Special Region of Yogyakarta is very high. Infectious and non-infectious diseases can cause significant economic losses. The main problem of infectious disease is caused by viruses, bacteria, fungi and parasites. This study was conducted to identify diseases that attack freshwater fish using data analysis on fish disease cases in the Laboratory of Pathology, Faculty of Veterinary Medicine, UGM in 2019. A total of 239 cases of fish diseases were examined by the Laboratory Diagnostic Co-assistance students in 2019 from the Special Region of Yogyakarta which includes Sleman district, Kulonprogo district, Yogyakarta City, Bantul district and Gunungkidul district. Based on data analysis, it showed that the highest fish sample was tilapia (52%), came from Sleman Regency (94%). The highest disease infection was caused by bacterial infections (54%) followed by parasite infections (35%). Macroscopic changes were observed in the liver (25%), skin (19%). and gills (26%). These changes included a yellow swollen liver, ulcer in the skin, fragile consistency and nodules in the surface of kidneys, attachment lamella and necrosis of gill lamellas. The parasite identification resulted in following parasite taxa/species Dactylogyrus., Trichodina sp., Gyrodactylus sp. and Ichtyopthirius sp. in the Parasitology Laboratory. The histopathological changes were identified using the the Hematoxillin & Eosin st aining technique, the organs showed dermatitis (18%), hepatitis (27%) and branchitis (23%). It can be concluded that in this research the highest cases of freshwater fish disease in Yogyakarta Special Region were caused by bacterial infections of tilapia from the Sleman district.

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Rejeki, Sri, Triyanto Triyanto, and Murwantoko Murwantoko. "ISOLASI DAN IDENTIFIKASI BAKTERI Aeromonas sp. DARI LELE DUMBO (Clarias sp.) DI KABUPATEN NGAWI." Jurnal Perikanan Universitas Gadjah Mada 18, no. 2 (August 12, 2016): 55. http://dx.doi.org/10.22146/jfs.26917.

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African catfish (Clarias sp.) is one of important freshwater fish which prefer consumed by people due to many advantages. Aeromonas sp. bacteria are dangerous patogen for freshwater fishes. This objective of the research was to isolate, identify and determine pathogenicity of Aeromonas sp. bacteria from African catfish from District Ngawi. The samples of catfish with 20 - 25 cm length showing clinical signs disease were obtained from three sub districts. Bacteria were isolated from kidney and inoculated into GSP medium. Characterization and identification through morphology of bacterial colonies, cells and biochemical test. Postulate Koch was conducted to verify abaility to couse disease. Pathogenicity was analyzed by determination of value of Lethal Dosage-50 on catfish on 7 - 9 cm length. The result showed that the disease symptoms of fish infected by the bacteria were skin ulcer, abdominal swelling and kidney damage. Fifteen bacterial isolates were collected which five, six and four isolates were from Kecamatan Karang Jati, Geneng dan Paron.sub-district respectively. The result showed 12 isolates (GKJ1, GKJ3, GKJ4, GGN1, GGN2, GGN3, GGN4, GGN5, GGN6, GPR2, GPR3 and GPR4) were identified as Aeromonas hydrophila. Three isolates (GKJ2, GKJ5 and GPR1) were identified as A. salmonicida. Isolate of A. hydrophila GKJ1, GKJ4, GGN2, GGN5, GPR2 and GPR4 were virulent to African catfish with LD50 values of 1,55 x 105, 3,89 x 105, 7,24 x 105, 2,39 x 105, 6,61 x 104 and 1,95 x 104 cfu/fish.

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Rashid, MM, MA Hasan, K. Mostafa, and MA Islam. "Isolation of Aeromonas hydrophila from EUS Affected Shing Heteropneustes fossilis of a Fish Farm in Mymensingh." Progressive Agriculture 19, no. 1 (December 18, 2013): 117–24. http://dx.doi.org/10.3329/pa.v19i1.17362.

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Aeromonas hydrophila bacteria was isolated from the suspected EUS-affected shing fish, Heteropneustes fossilis (Bloch). The disease investigations were primarily based on clinical signs and subsequently confirmed by the isolation of bacterial pathogen Aeromonas hydrophila from lesion of liver and kidney. The A. hydrophila isolates were identified by a series of morphological, physiological and biochemical tests. The total bacterial load in liver, intestine and kidney were 1.67 × 104 to 6.46 × 108 CFU/g, 1.71 × 103 to 1.18 × 109 CFU/g and 1.47 × 104 to 3.70 × 108 CFU/g respectively.DOI: http://dx.doi.org/10.3329/pa.v19i1.17362 Progress. Agric. 19(1): 117 - 124, 2008

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AUSTIN, B., and J. N. RAYMENT. "Epizootiology of Renibacterium salmoninarum, the causal agent of bacterial kidney disease in salmonid fish." Journal of Fish Diseases 8, no. 6 (November 1985): 505–9. http://dx.doi.org/10.1111/j.1365-2761.1985.tb00965.x.

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Guz, Leszek, and Krzysztof Puk. "Molecular detection of Renibacterium salmoninarum in rainbow trout (Oncorhynchus mykiss) from Poland." Fisheries & Aquatic Life 28, no. 4 (December 1, 2020): 234–37. http://dx.doi.org/10.2478/aopf-2020-0028.

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Abstract Renibacterium salmoninarum causes bacterial kidney disease mainly in salmonid fish. Oligonucleotide primers incorporating R. salmoninarum unique sequences were designed to amplify a 501 bp region of the gene encoding a 57 kDa soluble extra-cellular protein. The primers did not amplify other wide varieties of aquatic or piscine bacteria Aeromonas salmonicida or Yersinia ruckeri. This assay provides a molecular description and definitive identification of R. salmoninarum in Poland.

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Meylani, Vita, and Rinaldi Rizal Putra. "DETEKSI BAKTERI GENUS VIBRIO SEBAGAI CAUSATIVE AGENT PADA IKAN LELE SANGKURIANG (CLARIAS GARIEPINUS VAR. SANGKURIANG) DI KOTA TASIKMALAYA." BIOLINK (Jurnal Biologi Lingkungan, Industri, Kesehatan) 5, no. 1 (August 8, 2018): 42. http://dx.doi.org/10.31289/biolink.v5i1.1689.

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<em>Sangkuriang catfish (Clarias gariepinus var Sangkuriang) is the main commodity that widely cultivated in Tasikmalaya City. However, farmers have difficulty because of the disease which causes death in fish. High mortality is suspected as a vibriosis disease by genus Vibrio because redness wounds on dead fish. The purpose of this study is to know the genus Vibrio which causes of disease in Sangkuriang catfish. 10 samples of fish were taken from Sangkuriang catfish pond culture in Kelurahan Kersanagara Tasikmalaya City which were potentially suspected of vibriosis disease. Isolation of bacteria were done on TCBS medium. Bacterial isolates were collected from fish lesion on the body surface, liver, and kidneys of catfish. Isolation were able to gained 21 isolates and then 5 isolates (VK1, VK5, VK7, VK17, and VK21) were selected based on colony morphology and Postulates Koch’s were tested. The results showed that the clinical symptoms of catfish infected by vibriosis were redness lesions/ulcers on the body surface, hemorrhagic, fluid inside stomach, and fin eroded with redness wound. Bacterial identification through biochemical test revealed the causative agent of catfish disease at brackish pond area were bacteria of the genus Vibrio (VK 1, VK 5, and VK 7), Vibrio vulnificus (VK 17 and VK 21).</em>

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Dissertations / Theses on the topic "Bacterial kidney disease (Fish disease)"

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Manfredi, Eugene Trent. "Immunodiagnostic methods for the detection of bacterial kidney disease in salmonid fishes /." Thesis, Connect to this title online; UW restricted, 1986. http://hdl.handle.net/1773/5282.

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Hamel, Owen Sprague. "The dynamics and effects of bacterial kidney disease in Snake River spring Chinook salmon (Oncorhynchus tshawytscha) /." Thesis, Connect to this title online; UW restricted, 2001. http://hdl.handle.net/1773/6364.

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Campos-Perez, Juan Jose. "The role of reactive oxygen and nitrogen species in the immune response of rainbow trout to Renibacterium salmoninarum." Thesis, University of Aberdeen, 1998. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU112258.

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The role of reactive oxygen and nitrogen intermediates in the immune response of rainbow trout to R.s. was investigated. The early events occurring when the pathogen interacted with trout macrophages were assessed in terms of the respiratory burst elicited. Live R.s. elicited a respiratory burst, which was enhanced by heat-killed microorganism. This phenomenon, though, was not observed using UV-killed bacteria. Both responses were enhanced when a combination of LPS and TNF was used to activate the macrophages prior to contact R.s. Further studies demonstrated that both compounds synergised to enhance superoxide (O2) production, and that this was correlated with the ability to kill the pathogen. Opsonisation of R.s. with serum factors also increased the respiratory burst, but no difference was found between normal serum and heat-inactivated serum. The role of NO in the immune response of rainbow trout is also studied. Though no evidence of NO production was found in vitro, i.p. injection of live R.s. produced higher NO levels in serum as compared to controls. Fish injected with a virulent strain showed higher levels of NO than controls and than fish injected with an avirulent strain and other strains of unknown virulence. Fish vaccinated with killed R.s. and FIA also showed a significant increase in NO levels, but only four days after vaccination, decreasing thereafter, at both doses of vaccine tested. Injected of Brivax II, an attenuated strain of Aeromonas salmonicida, did not produce a significant increase of NO. RT-PCR was used to detect the expression of the iNOS in different tissues of rainbow trout. iNOS expression was seen only in gill and kidney after i.p. injection. iNOS was detected in the gills 6 h after injecting live R.s. and the expression was still present at day 5. iNOS was detected in the kidney 24 h after injection but was switched off at day 3. After bath challenge with the bacterium, iNOS was expressed in gill, gut and kidney, but the expression varied in each fish. No iNOS expression was found in macrophages isolated from challenged fish.

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Mazur, Carl François. "Growth, incidence of bacterial kidney disease and immunological function of salmonids reared in captivity." Thesis, University of British Columbia, 1991. http://hdl.handle.net/2429/30127.

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Pacific salmon reared commercially off of the Coast of British Columbia suffer great mortality losses to Bacterial Kidney Disease (BKD), caused by the diplobacillus bacterium Renibacterium salmqninarum. This thesis investigates the effects of environmental conditions on the growth performance and disease susceptibility of salmonids reared in captivity. I found that growth rate of chinook salmon was significantly higher in fish fed to 100 compared to 67 % of satiation during the first 175 days of saltwater rearing but not during the first winter. Feed coversion rate was significantly higher for fish fed at 100 % of satiation compared to 67 % of satiation and higher during the winter compared to summer and fall, irrespective of feeding level. Mortality rates were significantly higher during the summer than during the fall or winter, irrespective of experimental treatment. The last BKD sampling period (day 263) revealed that infection rates were directly proportional to stocking densities of 1.5 to 4 kg.m⁻₃. Hatchery-reared chinook salmon held in freshwater aquaria had significantly lower hematocrit and plasma cortisol concentration increases in response to increased stocking density than did their wild counterparts. Crowding of hatchery-reared and wild chinook salmon resulted in equally increased mortality rates for both groups of fish. Day 33 plasma cortisol concentrations in Atlantic salmon held at three stocking densities were directly proportional to stocking densities of 8 to 64 kg.m⁻₃. The ability of anterior kidney lymphocytes from these fish to produce antibody-producing cells was inversely proportional to the density at which the fish were held.
Land and Food Systems, Faculty of
Graduate

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Jansson, Eva. "Bacterial kidney disease in salmonid fish : development of methods to assess immune functions in salmonid fish during infection by Renibacterium salmoninarum /." Uppsala : Swedish Univ. of Agricultural Sciences (Sveriges lantbruksuniv.), 2002. http://epsilon.slu.se/avh/2002/91-576-6352-1.pdf.

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Densmore, Christine L. "Bacterial Kidney Disease and Its effect on the Salmonid Immune response." Diss., Virginia Tech, 1997. http://hdl.handle.net/10919/30488.

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Renibacterium salmoninarum, the etiological agent of bacterial kidney disease (BKD) of salmonid fish, is a pathogen of great concern among fisheries and the aquaculture industry worldwide. Previous investigations have indicated the pathogenesis of BKD is complex. It is a chronic, multisystemic, granulomatous disease with a number of potential immunomodulatory effects on the host. Given the current limitations for treatment and control of BKD, it is imperative that the pursuit of development of methods of prevention, namely management strategies and vaccination, be continued. To do so, the immunology of BKD must be elucidated in order to better understand and manipulate the associated immune responses to our advantage. This dissertation is composed of four chapters which relate to BKD and the associated immune responses of three species of susceptible salmonid fish as follows: Exogenous stress factors, through stress-induced immunosuppression, have been shown to influence BKD development in cultured salmonids. Chapter 1 examines the effects of two environmental stressors common to fish culture, overcrowding and overfeeding, as they affect BKD development and R. salmoninarumantigen prevalence among juvenile chinook salmon (Oncorhynchus tshawytscha) Immunomodulatory interaction between pathogen and host in BKD is widely reported and merits further investigation. Particularly, the immunological parameters affected and the role of the extracellular protein (ECP) of R. salmoninarum are of interest. Chapter 2 examines the in vivo immune response of rainbow trout (Oncorhynchus mykiss) following exposure to the ECP in terms of both humoral and cell-mediated immunological parameters, including the immune response against another bacterial pathogen. Chapter 3 addresses the in vitro effects of the ECP upon specific splenic immunocyte functions, phagocytosis and respiratory burst activity, in brook trout (Salvelinus fontinalis). The immune-complex mediated hypersensitivity reported to occur with BKD has considerable ramifications for control measures involving immunostimulation via antigen exposure. Further investigation is warranted to discern the significance and consistency of immunological hypersensivity in BKD pathogenesis. Chapter 4 examines the renal lesions, including immunopathologic changes and indications of immune-mediated disease, of brook trout exposed to R. salmoninarum.
Ph. D.

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Hulbig, Veronica A. "Developing a Model for Bacterial Kidney Disease in the Zebrafish, Danio rerio." Fogler Library, University of Maine, 2010. http://www.library.umaine.edu/theses/pdf/HulbigVA2010.pdf.

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Forsyth, Robert Bruce. "Stress proteins, phagocytes, and pathology in coho salmon with bacterial kidney disease." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0017/NQ48636.pdf.

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Rodgers, Christopher John. "Epidemiological studies of the bacterial fish pathogen Yersinia ruckeri." Thesis, University of Plymouth, 1991. http://hdl.handle.net/10026.1/1112.

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A new medium, designated Ribose Ornithine Deoxycholate agar (ROD), was developed and used in field trials at two fish farms where fish were known to have ERM. The medium indicated that Yersinia ruckeri could occur in faeces four to six weeks before appearing in the kidney. Fxurther epidemiological studies dealing with fish condition, performance, disease signs and water isolation are presented. These factors are discussed in relation to husbandry and management practices. The minimum uihibitory concentration (MIC) values for 124 strains of F. ruckeri were detemuned. The effect of oxolinic acid, oxytetracycline and a potentiated sulphonamide, on growth patterns over a 72 h period, was also determined. Results showed the bactericidal or bacteriostatic nature of each antimicrobial agent. It was possible to increase the MIC for oxoUnic acid, oxytetracycline and a potentiated sulphonamide usmg an in vitro technique. Attempts to decrease resistance to oxolinic acid were unsuccessful. However, it was possible to reduce the MIC's for o^etracycline and a potentiated sulphonamide. The recovery of Y. ruckeri, after artificial challenge, was less from the faecal and kidney material of a vaccinated group of fish compared with a non-vaccinated group. An E L I S A technique indicated that although there was no detectable serum antibody there was a local mucosal response i n vaccinated fish. Uptake of F. ruckeri antigen was demonstrated in vaccinated and non-vaccinated fish using an immunoperoxidase technique. However, only vaccinated fish appeared to take up the antigen by an active process. A survey of salmonid farms showed that there was a tendency for those sites where ERM had been diagnosed, irrespective of vaccination, to be larger table farms with production ranging up to over 200 tonnes p:a. Certain environmental and husbandry factors were reported as coinciding with the appearance of F. ruckeri. A slight tendency to consider that vaccines had failed i n some way was also indicated. This and the use of antimicrobial agents are discussed.

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Turgut, Emine. "Characterisation and detection of Renibacterium salmoninarum cultured in vivo and in vitro." Thesis, University of Stirling, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.250258.

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Books on the topic "Bacterial kidney disease (Fish disease)"

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Moffitt, Christine A. FDA approved registration of erythromycin for treatment of bacterial kidney disease (BKD) in juvenile and adult chinook salmon: Annual report, reporting period: year 1, 10 March 1989 - 9 March 1990. Portland, OR: U.S. Dept. of Energy, Bonneville Power Administration, Division of Fish and Wildlife, 1991.

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O'Connor, Glenda. Use of ELISA for monitoring bacterial kidney disease in naturally spawning chinook salmon. Salem, Or: Oregon Dept. of Fish and Wildlife, 2006.

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Kaattari, S. L. Development of a vaccine for bacterial kidney disease in salmon: Final report. Portland, Or: U.S. Dept. of Energy, Bonneville Power Administration, Division of Fish & Wildlife, 1991.

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Bruno, David W. Bacterial kidney disease. Aberdeen: Department of Agriculture and Fisheries for Scotland, Marine Laboratory, 1988.

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A, Austin D., ed. Bacterial fish pathogens: Disease in farmed and wild fish. 2nd ed. New York: E. Horwood, 1993.

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A, Austin D., ed. Bacterial fish pathogens: Disease in farmed and wild fish. Chichester: E. Horwood, 1987.

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Bullock, G. L. Bacterial kidney disease of salmonid fishes caused by Renibacterium salmoninarum. Washington, D.C: U.S. Dept. of the Interior, Fish and Wildlife Service, Research and Development, 1988.

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Stephen, Kaattari, United States. Bonneville Power Administration. Division of Fish and Wildlife., and Oregon State University. Dept. of Microbiology., eds. Development of a vaccine for bacterial kidney disease in salmon: Annual report FY 1984. Portland, Or: U.S. Dept. of Energy, Bonneville Power Administration, Division of Fish & Wildlife, 1985.

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Bruno, D. W. Bacterial Kidney Disease (Aquaculture Information Series: 3. 1988). The Stationery Office Books (Agencies), 1988.

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Austin, D. A., and B. Austin. Bacterial Fish Pathogens: Disease of Farmed and Wild Fish. Springer, 2010.

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Book chapters on the topic "Bacterial kidney disease (Fish disease)"

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Elliott, Diane G., Gregory D. Wiens, K. Larry Hammell, and Linda D. Rhodes. "Vaccination against Bacterial Kidney Disease." In Fish Vaccination, 255–72. Chichester, UK: John Wiley & Sons, Ltd, 2014. http://dx.doi.org/10.1002/9781118806913.ch22.

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Tittel, André P., Christoph Heuser, Natalio Garbi, and Christian Kurts. "Dendritic Cells Orchestrate Innate Immunity against Bacterial Kidney Infection." In Innate Immunity: Resistance and Disease-Promoting Principles, 112–19. Basel: S. KARGER AG, 2013. http://dx.doi.org/10.1159/000346530.

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Dar, Gowhar Hamid, Rouf Ahmad Bhat, Azra N. Kamili, Mohammad Z. Chishti, Humaira Qadri, Rubiya Dar, and Mohammad Aneesul Mehmood. "Correlation Between Pollution Trends of Freshwater Bodies and Bacterial Disease of Fish Fauna." In Fresh Water Pollution Dynamics and Remediation, 51–67. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-8277-2_4.

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Morris, David J., and Alexandra Adams. "PCR and in Situ Hybridisation of Tetracapsula Bryosalmonae (PKX), the Causative Agent of Proliferative Kidney Disease." In Reviews: Methods and Technologies in Fish Biology and Fisheries, 299–313. Dordrecht: Springer Netherlands, 2002. http://dx.doi.org/10.1007/978-94-017-2315-2_12.

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Austin, B. "Developments in vaccination against fish bacterial disease." In Infectious Disease in Aquaculture, 218–43. Elsevier, 2012. http://dx.doi.org/10.1533/9780857095732.2.218.

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Birkbeck, Thomas Henry. "Role of Probiotics in Fish Disease Prevention." In Current Trends in the Study of Bacterial and Viral Fish and Shrimp Diseases, 390–416. WORLD SCIENTIFIC, 2004. http://dx.doi.org/10.1142/9789812565709_0014.

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Yusufi, A. N. K., M. W. Khan, S. A. Khan, S. Priyamvada, and F. N. Khan Yusufi. "Dietary Fish Oil Protects Against Gentamicin, Cisplatin, Uranyl Nitrate, and Nitric Oxide Donor/Metabolite-Induced Nephrotoxicity and Oxidative Damage in Rat Kidney." In Fish and Fish Oil in Health and Disease Prevention, 305–20. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-12-802844-5.00028-2.

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"Whirling Disease: Reviews and Current Topics." In Whirling Disease: Reviews and Current Topics, edited by LINDA STATON, DAVE ERDAHL, and MANSOUR EL-MATBOULI. American Fisheries Society, 2002. http://dx.doi.org/10.47886/9781888569377.ch21.

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<EM>ABSTRACT </EM>Two potential therapeutants, fumagillin, an antibiotic derived from the fungus <em>Aspergillus fumagatus</em>, and TNP-470, a superactive analog of fumagillin, were tested for efficacy to prevent <em>Myxobolus cerebralis </em>infection in rainbow trout. The study was conducted at the Wild Trout Research Laboratory in Bozeman, Montana utilizing rainbow trout <em>Oncorhynchus mykiss </em>fry (~2.0 g). Treatment groups included negative controls (no treatment, no exposure), positive controls (no treatment, exposure), fumagillin top-dressed on feed, fumagillin incorporated in feed, and TNP-470 incorporated in feed. The exposure dose was 1000 triactinomyxons (TAMs) per fish for 2 h. Medicated feed treatment was initiated 24 h after exposure to TAMs. All treated groups received medicated feed for 10 d, with the exception of a single TNP-470 treatment group that was fed for 26 d. Five replicate tanks of fish were used for each treatment group. Presence and level of <em>Myxobolus cerebralis </em>infection were determined by histology, spore counts, and electron microscopy evaluation conducted 150 d postexposure. Hematology samples were also collected to evaluate potential toxic effects of treatment. Spore count and histological evaluation indicated that fumagillin and TNP-470 treatment was not efficacious in preventing or reducing <em>Myxobolus cerebralis </em>infection. Although fumagillin and TNP-470 administered for 10 d or 26 d did result in a reduction in spore numbers, results were not significantly different than observed in positive controls. Furthermore, histological scores were similar for all treatment groups and the positive controls. Although electron microscopy revealed spore deformation in both fumagillin and TNP-470 treated groups, no treatment group was effective in preventing <em>Myxobolus cerebralis </em>infection. Fumagillin treatment and TNP-470 fed for 10 d did not appear to negatively impact fish performance. However, toxicity was observed in fish fed TNP-470 for 26 d. These fish were observed to be lethargic 30 d pe, and blood samples revealed low hematocrits, severely decreased lymphocytes, and reduced numbers of blast cells. Histological evaluation revealed abnormal cytology in both the kidney and thymus. Based on study results, fumagillin and TNP-470 did not appear to be effective therapeutants for use in the prevention or control of <em>Myxobolus cerebralis </em>infection in rainbow trout.

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Sivakamavalli, Jeyachandran, Kiyun Park, Ihn–Sil Kwak, and Vaseeharan Baskaralingam. "Bacterial Disease Control Methods in Shrimp (Penaeus, 1798) Farming Sector in Asian Countries." In Arthropods [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.93680.

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Aquaculture industry produces the enormous amount of sea foods (fish, shrimp, planktons, etc.) with enriched quantity of proteins, essential amino acids, essential fatty acids, and micronutrients and also possesses the medicinal values. This production industry is very important to meet out the need of the global population. Recently, different culture practices for aquatic culturing organisms were developed in practices, where the risk of infection and diseases outbreak also increased which leads to the production loss to the aquatic sector. Several conventional methods are used to prevent the diseases probiotics, antibiotics, plants, immmunostimulants, proteins, immune proteins enhancement, nanoparticles, etc. At the same time, these treatment techniques also have merits and demerits to execute into the practical platform. For instance, chemical or antibiotics treatment into the culture system leads to the some adverse effects in culturing organisms, environment, and also consumer. In this chapter, various diseases caused by the bacterial strains and its control strategies in the shrimp farming industry to enhance the aquaculture are discussed.

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Ellis, A. E. "Chapter 14 Development of the immune response in relation to bacterial disease in the growing fish." In Microbial Ecology in Growing Animals, 314–27. Elsevier, 2005. http://dx.doi.org/10.1016/s1877-1823(09)70047-5.

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Conference papers on the topic "Bacterial kidney disease (Fish disease)"

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"Modelling control options for a disease with hidden sub-clinical infection: bacterial kidney disease in Scottish aquaculture." In 19th International Congress on Modelling and Simulation. Modelling and Simulation Society of Australia and New Zealand (MSSANZ), Inc., 2011. http://dx.doi.org/10.36334/modsim.2011.b2.murray.

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Reports on the topic "Bacterial kidney disease (Fish disease)"

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Kaatari, S., P. Turaga, and G. Wiens. Development of a vaccine for bacterial kidney disease in salmon. [und Renibacterium salmoninarum]. Test accounts, August 1989. http://dx.doi.org/10.2172/5301998.

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Kaattari, Stephen L. Development of a Vaccine for Bacterial Kidney Disease in Salmon, 1985 Annual Report. Office of Scientific and Technical Information (OSTI), June 1986. http://dx.doi.org/10.2172/760068.

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Kaattari, Stephen L. Development of a Vaccine for Bacterial Kidney Disease in Salmon, 1986 Annual Report. Office of Scientific and Technical Information (OSTI), June 1987. http://dx.doi.org/10.2172/760069.

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Kaattari, Stephen. Development of a Vaccine for Bacterial Kidney Disease in Salmon, 1987 Annual Report. Office of Scientific and Technical Information (OSTI), June 1988. http://dx.doi.org/10.2172/760071.

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Kaattari, Stephen L. Development of a Vaccine for Bacterial Kidney Disease in Salmon, 1988 Final Report. Office of Scientific and Technical Information (OSTI), August 1989. http://dx.doi.org/10.2172/760072.

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Kaattari, Stephen L. Development of a Vaccine for Bacterial Kidney Disease in Salmon, 1984 Annual Report. Office of Scientific and Technical Information (OSTI), June 1985. http://dx.doi.org/10.2172/760067.

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Kaattari, Stephen L. ELISA-Based Segregation of Adult Spring Chinook Salmon for Control of Bacterial Kidney Disease: Annual Report 1991. Office of Scientific and Technical Information (OSTI), February 1993. http://dx.doi.org/10.2172/920640.

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Kaattari, Stephen L., and James R. Winton. ELISA-Based Segregation of Adult Spring Chinook Salmon for Control of Bacterial Kidney Disease, Annual Report FY 1989. Office of Scientific and Technical Information (OSTI), December 1989. http://dx.doi.org/10.2172/920196.

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Winton, James R., and Stephen L. Kaattari. ELISA-Based Segregation of Adult Spring Chinook Salmon for Control of Bacterial Kidney Disease, Annual Report FY 1990. Office of Scientific and Technical Information (OSTI), December 1990. http://dx.doi.org/10.2172/5208467.

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Moffitt, Christine A. FDA Approved Registration of Erythromycin for Treatment of Bacterial Kidney Disease (BKD) in Juvenile and Adult Chinook Salmon : Annual Report, Reporting Period March 10, 1989 to March 9, 1990. Office of Scientific and Technical Information (OSTI), April 1991. http://dx.doi.org/10.2172/5392957.

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