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Books on the topic 'Bakers asthma; allergic diseases'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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1

Namazy, Jennifer A., and Michael Schatz, eds. Asthma, Allergic and Immunologic Diseases During Pregnancy. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-03395-8.

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2

Gern, James E. Contemporary diagnosis and management of allergic diseases and asthma. 3rd ed. Newton, Pa: Handbooks in Health Care, 1999.

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3

W, Busse W., ed. Contemporary diagnosis and management of allergic diseases and asthma. 2nd ed. Newton, Pa: Handbooks in Health Care, 1998.

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4

Gern, James E. Contemporary diagnosis and management of allergic diseases and asthma. 5th ed. Newtown, Pa: Handbooks in Health Care Co., 2008.

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5

Gern, James E. Contemporary diagnosis and management of allergic diseases and asthma. Newton, Pa: Handbooks in Health Care, 1996.

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6

Busse, W. W. (William W.), ed. Contemporary diagnosis and management of allergic diseases and asthma. 6th ed. Newtown, Pa: Handbooks in Health Care Co., 2009.

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7

International, Conference on Asthma (5th 1997 Ischia Italy). Asthma and allergic diseases: Physiology, immunopharmacology, and treatment : fifth international symposium. San Diego: Academic Press, 1998.

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8

Pereira, Celso. Allergic diseases - highlights in the clinic, mechanisms and treatment. Rijeka: InTech, 2012.

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9

B, Kay A., ed. Allergy and allergic diseases. 2nd ed. Malden, Mass: Blackwell, 2008.

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10

Robinson, D. S., ed. Immunological Mechanisms in Asthma and Allergic Diseases. S. Karger AG, 2000. http://dx.doi.org/10.1159/isbn.978-3-318-00611-7.

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11

Immunological Mechanisms in Asthma and Allergic Diseases. Muenchen: Karger, 2004.

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12

(Editor), Gianni Marone, K. Frank Austen (Editor), Stephen T. Holgate (Editor), A. Barry Kay (Editor), and Lawrence M. Lichtenstein (Editor), eds. Asthma and Allergic Diseases: Physiology, Immunopharmacology and Treatment. Academic Press, 1998.

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13

Asthma and Allergic Diseases: Physiology, Immunopharmacology and Treatment. Academic Press, 1998.

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14

B, Kay A., ed. Allergy and allergic diseases. Malden, MA: Distributor, Blackwell Science, 1997.

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15

Gern, James E., and William W. Busse. Contemporary Diagnosis and Management of Allergic Diseases and Asthma. 4th ed. Handbooks in Health Care Company, 2003.

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16

Gern, James E. Contemporary Diagnosis and Management of Allergic Diseases and Asthma. Handbooks in Health Care, 2003.

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17

Immunological Mechanisms in Asthma and Allergic Diseases (Chemical Immunology). Not Avail, 2000.

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18

Denburg, MD Judah A. Allergy and Allergic Diseases: The New Mechanisms and Therapeutics. Humana Press, 2010.

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19

Asthma, Allergic and Immunologic Diseases During Pregnancy: A Guide to Management. Springer, 2018.

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20

M, Weiler J., ed. Allergic and respiratory disease in sports medicine. New York: M. Dekker, 1997.

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21

Cullinan, Paul, and Joanna Szram. Occupational lung disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0142.

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Some occupational lung diseases are defined by their clinical or pathological nature (e.g. occupational asthma or mesothelioma), while others are defined by their specific etiology (e.g. silicosis, farmer’s lung). Most fall into one of three categories. The first is airways disease, including occupational asthma (induced by a workplace agent), work-exacerbated asthma (preexisting asthma provoked by one or more agents at work), and irritant-induced asthma (initiated by a single, toxic exposure to a respiratory irritant); COPD and obliterative bronchiolitis may arise from workplace exposures, and around 10% of lung cancers have an occupational etiology. The second is parenchymal diseases, incorporating the many types of pneumoconiosis, differentiated by the dust that caused them, and the many types of extrinsic allergic alveolitis (or hypersensitivity pneumonia) categorized by the occupations in which they arise. The third is pleural diseases comprising pleural plaques, diffuse pleural thickening, and mesothelioma.
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22

Jolly, Elaine, Andrew Fry, and Afzal Chaudhry, eds. Respiratory medicine. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199230457.003.0018.

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Chapter 18 covers the basic science and clinical topics relating to respiratory medicine which trainees are required to learn as part of their basic training and demonstrate in the MRCP. The chapter starts with an introduction to the respiratory system, before covering respiratory defence and physiology, respiratory investigations, respiratory failure, pneumonia, tuberculosis, cystic fibrosis, bronchiectasis, pleural effusion, chronic obstructive pulmonary Disease, adult respiratory distress syndrome, asthma , fungal lung diseases, pulmonary embolism , lung cancer, pulmonary fibrosis, extrinsic allergic alveolitis, occupational lung diseases, sarcoidosis, Cor pulmonale and pulmonary hypertension, pneumothorax, cough and haemoptysis, pulmonary eosinophilia, and obstructive sleep apnoea.
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23

Kosmidis, Chris, David W. Denning, and Eavan G. Muldoon. Fungal disease in cystic fibrosis and chronic respiratory disorders. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0037.

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A range of fungal disease syndromes affect patients with chronic respiratory diseases and cystic fibrosis (CF). Invasive aspergillosis is increasingly recognized in seriously ill patients with chronic obstructive pulmonary disease, especially after high-dose steroids. Chronic pulmonary aspergillosis affects patients with pre-existing cavities or bullae, such as those with previous tuberculosis or atypical mycobacterial disease, bullous emphysema, sarcoidosis, pneumothorax, or treated lung cancer. In addition, fungi have become one of the most important trigger agents for asthma, and allergic bronchopulmonary aspergillosis may complicate up to 3.5% of cases of asthma and up to 15% of cases of CF, starting in childhood. CF patients are commonly colonized with fungal organisms, although the impact of such colonization on outcome is not clear. Aspergillus is the most common mould isolated from CF patients. Distinguishing between colonization and infection remains challenging. Candida is thought to be of no clinical significance; however, it has been associated with decline in lung function.
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